Bibliografías temáticas / Monoamine

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Literatura académica sobre el tema "Monoamine"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 26 de julio de 2025

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Artículos de revistas sobre el tema "Monoamine"

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Zhou, Shi-Sheng, Yi-Ming Zhou, Da Li, and Qiang Ma. "Early Infant Exposure to Excess Multivitamin: A Risk Factor for Autism?" Autism Research and Treatment 2013 (2013): 1–8. http://dx.doi.org/10.1155/2013/963697.

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Autism, a neurodevelopmental disorder that affects boys more than girls, is often associated with altered levels of monoamines (serotonin and catecholamines), especially elevated serotonin levels. The monoamines act as both neurotransmitters and signaling molecules in the gastrointestinal and immune systems. The evidence related to monoamine metabolism may be summarized as follows: (i) monoamine neurotransmitters are enzymatically degraded/inactivated by three mechanisms: oxidative deamination, methylation, and sulfation. The latter two are limited by the supply of methyl groups and sulfate, r
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Aksoz, Begum E., and Erkan Aksoz. "Vital Role of Monoamine Oxidases and Cholinesterases in Central Nervous System Drug Research: A Sharp Dissection of the Pathophysiology." Combinatorial Chemistry & High Throughput Screening 23, no. 9 (2020): 877–86. http://dx.doi.org/10.2174/1386207323666200220115154.

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Background: Monoamine oxidase and cholinesterase enzymes are very critical enzymes that regulate the level of neurotransmitters such as acetylcholine and monoamines. Monoamine neurotransmitters and acetylcholine play a very important role in many physiological events. An increase or decrease in the amount of these neurotransmitters is observed in a wide range of central nervous system pathologies. Balancing the amount of these neurotransmitters is important in improving the progression of these diseases. Inhibitors of monoamine oxidase and cholinesterase enzymes are important in symptomatic th
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Anlauf, Martin, Rolf Eissele, Martin K. H. Schäfer, et al. "Expression of the Two Isoforms of the Vesicular Monoamine Transporter (VMAT1 and VMAT2) in the Endocrine Pancreas and Pancreatic Endocrine Tumors." Journal of Histochemistry & Cytochemistry 51, no. 8 (2003): 1027–40. http://dx.doi.org/10.1177/002215540305100806.

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The uptake of monoamines into the secretory granules of monoamine-storing neuroendocrine cells is mediated by vesicular monoamine transporter protein 1 or 2 (VMAT1 or VMAT2). This study analyzed the expression of VMAT1 and VMAT2 in endocrine cells of normal human and monkey pancreas. The expression of VMAT1 and VMAT2 was also examined in infants with hyperinsulinemic hypoglycemia and in adults with pancreatic endocrine tumors (PETs). Using immunohistochemistry (IHC) and in situ hybridization (ISH), we demonstrated the mutually exclusive expression of VMAT1 in endocrine cells of the duct system
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Murtazina, Alya R., Nadegda S. Bondarenko, Tatiana S. Pronina, et al. "A Comparative Analysis of CSF and the Blood Levels of Monoamines As Neurohormones in Rats during Ontogenesis." Acta Naturae 13, no. 4 (2021): 89–97. http://dx.doi.org/10.32607/actanaturae.11516.

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According to the literature, the cerebrospinal fluid (CSF) in the cerebral ventricles contains numerous neuron-derived physiologically active substances that can function as neurohormones and contribute to volume neurotransmission in the periventricular region of the brain. This study was aimed at carrying out a comparative analysis of CSF and the blood levels of monoamines in rats during ontogenesis as an indicator of age-related characteristics of monoamine transport to body fluids and their function as neurohormones in volume neurotransmission in the periventricular region of the brain. We
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Hollander, Eric. "Parkinson's Disease, Tourette Syndrome, and the Changing Nature of Depression: The Dog Days of Summer." CNS Spectrums 13, no. 8 (2008): 643–44. http://dx.doi.org/10.1017/s1092852900013729.

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Since the development of selective serotonin reuptake inhibitors there has been a widening of the definition of depression and a decrease in the placebo-drug difference in controlled studies. In the early 1960s, ~33% of depressed patients improved with placebo and 66% with active compounds and current controlled studies suggest that the situation has certainly not improved. The Sequenced Treatment Alternatives to Relieve Depression Study found that response rates to new compounds after the failure of the first antidepressant are low. The monoamine hypothesis of depression was formulated in the
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Belmaker, Robert H. "The Future of Depression Psychopharmacology." CNS Spectrums 13, no. 8 (2008): 682–87. http://dx.doi.org/10.1017/s1092852900013766.

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ABSTRACTAlong with the development of selective sertonin reuptake inhibitors there has been a tremendous widening of the definition of depression and an impressive decrease in the placebodrug difference in controlled studies. In the early 1960s, about one third of depressed patients improved with placebo and two thirds with active compounds. Current controlled studies suggest that the situation has certainly not improved. The Sequenced Treatment Alternatives to Relieve Depression Study found that response rates to new compounds after the failure of the first antidepressant are low. The monoami
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Mooslehner, Katrin A., Pok Man Chan, Weiming Xu, et al. "Mice with Very Low Expression of the Vesicular Monoamine Transporter 2 Gene Survive into Adulthood: Potential Mouse Model for Parkinsonism." Molecular and Cellular Biology 21, no. 16 (2001): 5321–31. http://dx.doi.org/10.1128/mcb.21.16.5321-5331.2001.

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ABSTRACT We have created a transgenic mouse with a hypomorphic allele of the vesicular monoamine transporter 2 (Vmat2) gene by gene targeting. These mice (KA1) have profound changes in monoamine metabolism and function and survive into adulthood. Specifically, these animals express very low levels of VMAT2, an endogenous protein which sequesters monoamines intracellularly into vesicles, a process that, in addition to being important in normal transmission, may also act to keep intracellular levels of the monoamine neurotransmitters below potentially toxic thresholds. Homozygous mice show large
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Cai, Lu, Chao Wang, Xiao-kui Huo, et al. "Effect of Alkaloids Isolated fromPhyllodium pulchellumon Monoamine Levels and Monoamine Oxidase Activity in Rat Brain." Evidence-Based Complementary and Alternative Medicine 2016 (2016): 1–6. http://dx.doi.org/10.1155/2016/6826175.

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Phyllodium pulchellum(P. pulchellum) is a folk medicine with a significant number of bioactivities. The aim of this study was to investigate the effects displayed by alkaloids fractions, isolated from the roots ofP. pulchellum, on neurotransmitters monoamine levels and on monoamine oxidase (MAO) activity. Six alkaloids, which had indolealkylamine orβ-carboline skeleton, were obtained by chromatographic technologies and identified by spectroscopic methods such as NMR and MS. After treatment with alkaloids ofP. pulchellum, the reduction of DA levels (54.55%) and 5-HT levels (35.01%) in rat brain
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Nilsson, G. E., A. A. Alfaro, and P. L. Lutz. "Changes in turtle brain neurotransmitters and related substances during anoxia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 259, no. 2 (1990): R376—R384. http://dx.doi.org/10.1152/ajpregu.1990.259.2.r376.

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Freshwater turtles (Pseudemys scripta elegans) were exposed to 0.5-13 h of anoxia at 25 degrees C, whereupon the brain concentrations of 14 amino acid and monoamine neurotransmitters and related substances were measured. Monoamines are of particular interest, because their synthesis and (in part) degradation require molecular oxygen. During anoxia, the level of the inhibitory transmitter gamma-aminobutyric acid (GABA) increased (2.3-fold after 13 h) and the level of the excitatory transmitter Glu fell. Furthermore, anoxia caused increases in the levels of Ala (14 times after 13 h), Tau, Gly, a
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Nishimura, Mitsuhiro, Kohji Sato, Tomoya Okada, et al. "Ketamine Inhibits Monoamine Transporters Expressed in Human Embryonic Kidney 293 Cells." Anesthesiology 88, no. 3 (1998): 768–74. http://dx.doi.org/10.1097/00000542-199803000-00029.

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Background Ketamine has been characterized as having psychotomimetic and sympathomimetic effects. These symptoms have raised the possibility that ketamine affects monoaminergic neurotransmission. To elucidate the relation between ketamine and monoamine transporters, the authors constructed three cell lines that stably express the norepinephrine, dopamine, and serotonin transporters and investigated the effects of ketamine on these transporters. Methods Human embryonic kidney cells were transfected using the Chen-Okayama method with the human norepinephrine, rat dopamine, and rat serotonin tran
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Tesis sobre el tema "Monoamine"

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Mullan, Elaine L. "Inhibitors of monoamine oxidase." Thesis, Queen's University Belfast, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.337116.

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Kaushal, Setu. "Characterization of Three Putative Monoamine Oxidase Genes in Caenorhabditis elegans." Ohio University / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=ohiou1218747926.

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Fitzgerald, Julia. "Monoamine oxidase in neuronal cell death." Thesis, Nottingham Trent University, 2008. http://irep.ntu.ac.uk/id/eprint/51/.

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Monoamine oxidase (MAO) is an oxidative enzyme that deaminates a variety of amine substrates, including the neurotransmitter dopamine. The enzymatic reaction requires molecular oxygen and produces hydrogen peroxide as a by-product. MAO is localised in the outer mitochondrial membrane and exists as two isoforms, MAO-A and MAO-B, which are differentially expressed in the body and differ in their substrate and inhibitor specificities. Previous studies have suggested that MAO-generated reactive oxygen species (ROS) contribute to oxidative stress in the cell and can directly inhibit electron transp
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Stenström, Anders. "Intra- and extraneuronal monoamine oxidase (MAO)." Umeå : [University of Umeå], 1986. http://catalog.hathitrust.org/api/volumes/oclc/15239401.html.

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Yeomanson, K. B. "An immunochemical analysis of monoamine oxidase profiles." Thesis, Nottingham Trent University, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254741.

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Maurel, Agnès. "Monoamine oxydases rénales et cardiaques : implications physiopathologiques." Paris 7, 2004. http://www.theses.fr/2004PA077127.

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Wu, Bo. "Structure-function relationships in monoamine oxidase B /." Digital version accessible at:, 1998. http://wwwlib.umi.com/cr/utexas/main.

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Ruchala, Iwona. "EXPANDING MONOAMINE TRANSPORTERS PHARMACOLOGY USING CALCIUM CHANNELS." VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/5032.

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Research in drug development meets many challenges including lengthy, complex and costly procedures to identify novel pharmacotherapies. In our lab, we developed a method for fast screening of small molecules that interact with monoamine transports – dopamine and serotonin (DAT, SERT). These membrane proteins play important roles in brain neurotransmission responsible for cognition, motion and pleasure. Dysfunction in dopaminergic and serotonergic systems result in neurological disorders such as depression, Attention Deficit Hyperactivity Disorder (ADHD), schizophrenia and addiction. DAT and S
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Saiyudthong, Somrudee. "Mechanisms underlying the antidepressant properties of St. John's wort (Hypericum perforatum)." Thesis, University of Nottingham, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.275319.

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Backwell, Colette. "Labels for the active site of monoamine oxidase." Thesis, Queen's University Belfast, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.336043.

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Libros sobre el tema "Monoamine"

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Binda, Claudia, ed. Monoamine Oxidase. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-2643-6.

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Oreland, Lars, and Brian A. Callingham, eds. Monoamine Oxidase Enzymes. Springer Vienna, 1987. http://dx.doi.org/10.1007/978-3-7091-8901-6.

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National Institutes of Health (U.S.), ed. Low monoamine (LMA) diet. U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1989.

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1939-, Oreland L., Callingham B. A, and Workshop on Amine Oxidases (2nd : 1986 : Biomedical Center, Uppsala University), eds. Monoamine oxidase enzymes: Review and overview. Springer-Verlag, 1987.

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Szelenyi, I., ed. Inhibitors of Monoamine Oxidase B. Birkhäuser Basel, 1993. http://dx.doi.org/10.1007/978-3-0348-6348-3.

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F, Tipton Keith, ed. Amine oxidases: Function and dysfunction : proceedings of the 5th International Amine Oxidase Workshop, Galway, Ireland, August 22-25, 1992. Springer-Verlag, 1994.

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H, Yasuhara, ed. Monoamine oxidase: Basic and clinical aspects. VSP, 1993.

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1938-, Lieberman A. N., ed. Monoamine oxidase inhibitors in neurological diseases. M. Dekker, 1994.

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Ebrahimi, Soltan Ahmed. Characterisation of guinea pig liver monoamine oxidase. University of Portsmouth, School of Pharmacy and Biomedical Science, 1995.

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H, Kennedy Sidney, ed. Clinical advances in monoamine oxidase inhibitor therapies. American Psychiatric Press, 1994.

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Capítulos de libros sobre el tema "Monoamine"

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Morgan, Michael M., MacDonald J. Christie, Thomas Steckler, et al. "Monoamine Depletion." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_3405.

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Morgan, Michael M., MacDonald J. Christie, Thomas Steckler, et al. "Monoamine Hypotheses." In Encyclopedia of Psychopharmacology. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-540-68706-1_3406.

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Rey, Jose A. "Monoamine Oxidase." In Encyclopedia of Clinical Neuropsychology. Springer New York, 2011. http://dx.doi.org/10.1007/978-0-387-79948-3_1742.

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Rey, Jose A. "Monoamine Oxidase." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-56782-2_1742-2.

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Edmondson, Dale E., and Claudia Binda. "Monoamine Oxidases." In Subcellular Biochemistry. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-10-7757-9_5.

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Rey, Jose A. "Monoamine Oxidase." In Encyclopedia of Clinical Neuropsychology. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-57111-9_1742.

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Gainetdinov, Raul R., and Marc G. Caron. "Monoamine Transporters." In Neurotransmitter Transporters. Humana Press, 2002. https://doi.org/10.1007/978-1-59259-158-9_5.

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Singer, T. P. "Perspectives in MAO: past, present, and future." In Monoamine Oxidase Enzymes. Springer Vienna, 1987. http://dx.doi.org/10.1007/978-3-7091-8901-6_1.

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Tipton, K. F., Anne-Marie O’Carroll, and J. M. McCrodden. "The catalytic behaviour of monoamine oxidase." In Monoamine Oxidase Enzymes. Springer Vienna, 1987. http://dx.doi.org/10.1007/978-3-7091-8901-6_2.

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Callingham, B. A., and M. A. Barrand. "Some properties of semicarbazide-sensitive amine oxidases." In Monoamine Oxidase Enzymes. Springer Vienna, 1987. http://dx.doi.org/10.1007/978-3-7091-8901-6_3.

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Actas de conferencias sobre el tema "Monoamine"

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Verlinden, Heleen. "Monoamine receptors and swarming behaviour in locusts." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.95192.

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Serra, Silvia, Eugenio Uriarte, Lourdes Santana, Giovanna Delogu, and Maria Matos. "Synthesis of New Possible Monoamine Oxidase Inhibitors." In The 14th International Electronic Conference on Synthetic Organic Chemistry. MDPI, 2010. http://dx.doi.org/10.3390/ecsoc-14-00378.

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Serra, Silvia, Eugenio Uriarte, Lourdes Santana, Giovanna Delogu, and Maria Matos. "Synthesis of new possible monoamine oxidase inhibitors." In The 14th International Electronic Conference on Synthetic Organic Chemistry. MDPI, 2010. http://dx.doi.org/10.3390/ecsoc-14-00474.

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Ma, Peixiang, Shanshan Wang, Feng Qu, and Yulin Deng. "Determination of Monoamine Oxidase Activity by Capillary Electrophoresis Method." In 2007 IEEE/ICME International Conference on Complex Medical Engineering. IEEE, 2007. http://dx.doi.org/10.1109/iccme.2007.4382056.

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"Monoamine signaling gene networks unraveled in mouse social stress model." In Bioinformatics of Genome Regulation and Structure/ Systems Biology. institute of cytology and genetics siberian branch of the russian academy of science, Novosibirsk State University, 2020. http://dx.doi.org/10.18699/bgrs/sb-2020-172.

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Yurttas, Leyla, Asaf Evren, Demokrat Nuha, Sam Dawbaa, and Begüm Sağlık. "New thiazolylhydrazone derivatives as potent monoamine oxidase and aromatase inhibitors." In 7th International Electronic Conference on Medicinal Chemistry. MDPI, 2021. http://dx.doi.org/10.3390/ecmc2021-11582.

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Yan, Shaomin, and Guang Wu. "Distributions of amino acids in the primary structure of monoamine oxidase family." In International Conference on Biomedical and Intelligent Systems (IC-BIS 2022), edited by Ahmed El-Hashash. SPIE, 2022. http://dx.doi.org/10.1117/12.2660263.

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Bayer, A., E. Zrenner, H. J. Thiel, S. Ried, and D. Schmidt. "Early detection of anticonvulsant drug toxicity by colour vision tests." In Noninvasive Assessment of the Visual System. Optica Publishing Group, 1991. http://dx.doi.org/10.1364/navs.1991.md10.

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Phenytoin and Carbamazepine, especially in combination or after repeated exposure to toxic blood concentrations, can produce neurological and mental syndroms. Possible mechanisms of impaired neurological and mental function include neuronal damage, or disturbance of folic acid, monoamine or hormonal metabolism. Phenytoin and Carbamazepine may also impair the ocular system. The most common symptoms are diplopia, nystagmus and blurred vision. Cerebellar or vestibulo-ocular dysfunction is suspected as mechanism of these ocular adverse effects.
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Kamruzzaman, A. S. M. "Integration of midgut-fat body-gonadal axis inPeriplaneta americana: Monoamine and peptide regulation." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.94434.

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Khidiyatova, Irina, Gulnara Akhmadeeva, Irina Gilyazova, et al. "THE ROLE OF MONOAMINE METABOLISM SYSTEM GENES IN NEUROPSYCHOLOGICAL MANIFESTATIONS OF PARKINSON'S DISEASE." In XVI International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2020. http://dx.doi.org/10.29003/m1317.sudak.ns2020-16/494-495.

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Informes sobre el tema "Monoamine"

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Jacobs, Barry L. Central Postsynaptic Actions of Monoamine Neurotransmitters in Behaving Animals. Defense Technical Information Center, 1997. http://dx.doi.org/10.21236/ada325701.

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Gluck, Martin R. Parkinson's Disease: The Link Between Monoamine Oxidase and Mitochondrial Respiration. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada612171.

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Shih, Jean C., and Bogdan Z. Olenyuk. Monoamine Oxidase A: A Novel Target for Progression and Metastasis of Prostate Cancer. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada612725.

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Shih, Jean C., and Bogdan Z. Olenyuk. Monoamine Oxidase A: A Novel Target for Progression and Metastasis of Prostate Cancer. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada593309.

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Yin, Ziqian, Haoyang Xue, Youjia Qiu, et al. Safety and efficacy of vesicular monoamine transporter 2 inhibitors for the treatment of Huntington's disease: a review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.10.0004.

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Mincher, Bruce Jay. Radiation chemistry of the branched-chain monoamide di-ethylhexyl-isobutyramide. Office of Scientific and Technical Information (OSTI), 2016. http://dx.doi.org/10.2172/1406976.

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Arias Quiroz, Kevin Josué, and Ariel Enrique Pastrana Arias. Efectos en el sistema nervioso de recién nacidos por consumo de cocaína en la vida intrauterina. Universidad de Cartagena, 2023. https://doi.org/10.32997/11227/18440.

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Si bien en la actualidad no es que ese vean muchos casos de niños afectados por el consumo de la droga durante la vida intra uterina, pero cada vez se es más frecuente por lo cual es un tema de investigación importante. Para que se hagan de cuanto está aumentando el consumo en mujeres en edad fértil algún tipo de sustancia en Colombia, el Observatorio de Drogas de Colombia (ODC) reportó un aumentó en un 3,5% en el consumo de sustancias como marihuana, pasta básica de coca, éxtasis o heroína en las personas entre los 15 y 65 años. Desde hace muchos años se han descrito numerosos efectos secunda
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