Bibliografías temáticas / Pain sensitization

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Literatura académica sobre el tema "Pain sensitization"

Autor: Grafiati

Publicado: 25 de enero de 2025

Última modificación: 31 de julio de 2025

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Artículos de revistas sobre el tema "Pain sensitization"

Song, Soo Youn, Ye Won Jung, WonKyo Shin, et al. "Endometriosis-Related Chronic Pelvic Pain." Biomedicines 11, no. 10 (2023): 2868. http://dx.doi.org/10.3390/biomedicines11102868.

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Endometriosis, which is the presence of endometrial stroma and glands outside the uterus, is one of the most frequently diagnosed gynecologic diseases in reproductive women. Patients with endometriosis suffer from various pain symptoms such as dysmenorrhea, dyspareunia, and chronic pelvic pain. The pathophysiology for chronic pain in patients with endometriosis has not been fully understood. Altered inflammatory responses have been shown to contribute to pain symptoms. Increased secretion of cytokines, angiogenic factors, and nerve growth factors has been suggested to increase pain. Also, alte

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Aguggia, Marco, M. G. Saracco, M. Cavallini, G. Bussone, and P. Cortelli. "Sensitization and pain." Neurological Sciences 34, S1 (2013): 37–40. http://dx.doi.org/10.1007/s10072-013-1382-0.

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Melkani, Indu, Prashant Kumbhar, Pankaj Parasher, et al. "Mechanism and preclinical models of neuropathic pain: An update." Trends in Immunotherapy 9, no. 1 (2025): 23–44. https://doi.org/10.54963/ti.v9i1.974.

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Neuropathic pain is a debilitating condition that is a product of nervous system damage or dysfunction. Since the drugs prescribed by the physician provide partial pain relief to the patients. Hence, current updates for its treatment are required. It is a global concern as neuropathic pain happens in many diseased conditions like cancer, trauma, surgery and diabetics, etc. Developed as well as developing countries are both trying to find suitable medicine. Understanding the mechanisms behind it can be crucial for the effective treatment and management of neuropathic pain. Central sensitization

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Ramalingam, Thangamani, Pooja Desai, Dhruvi Ghoghari, Vidhi Jethva, and Rushvi Shah. "Neurophysiology of pain education knowledge, pain disability, patient satisfaction and central sensitization in chronic musculoskeletal pain." IP Journal of Surgery and Allied Sciences 4, no. 4 (2023): 137–41. http://dx.doi.org/10.18231/j.jsas.2022.026.

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Chronic musculoskeletal pains are multifaceted, and Central sensitization is a potential pathophysiological mechanism underlying a group of chronic musculoskeletal pain disorders. Neurophysiology of pain education knowledge and patient satisfaction levels of chronic musculoskeletal pain subjects may contribute to central nervous system sensitization. Hence, the aim of the study was to evaluate the impact of neurophysiology of pain education knowledge and patient satisfaction levels on central sensitization in large population of patient with chronic musculoskeletal pain. The study included 200

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Austin, Publishing Group. "Central Sensitization in Patients Attending Physical Therapy for Musculoskeletal Disorders." Physical Medicine and Rehabilitation - International 8, no. 5 (2021): 1194. https://doi.org/10.26420/physmedrehabilint.2021.1194.

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Abstract Study Design: Survey study. Objective: To determine what percentage of patients attending physical therapy with musculoskeletal pain present with central sensitization and which patient factors may be predictive of central sensitization. Background: Treating pain, especially chronic pain is clinically challenging. It has been suggested that pain be sub-classified as either nociceptive, peripheral neuropathic or central sensitization, to aid clinical decision-making to inform the treatment approach for specific pain cond

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Coderre, Terence J., and Joel Katz. "Peripheral and central hyperexcitability: Differential signs and symptoms in persistent pain." Behavioral and Brain Sciences 20, no. 3 (1997): 404–19. http://dx.doi.org/10.1017/s0140525x97251484.

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This target article examines the clinical and experimental evidence for a role of peripheral and central hyperexcitability in persistent pain in four key areas: cutaneous hyperalgesia, referred pain, neuropathic pain, and postoperative pain. Each suggests that persistent pain depends not only on central sensitization, but also on inputs from damaged peripheral tissue. It is instructive to think of central sensitization as comprised of both an initial central sensitization and an ongoing central sensitization driven by inputs from peripheral sources. Each of these factors, initial sensitization

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Lee, Jaesung, Seohyun Chung, Minkyu Hwang, Yeongkag Kwon, Seung Hyun Han, and Sung Joong Lee. "Estrogen Mediates the Sexual Dimorphism of GT1b-Induced Central Pain Sensitization." Cells 12, no. 5 (2023): 808. http://dx.doi.org/10.3390/cells12050808.

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We have previously reported that the intrathecal (i.t.) administration of GT1b, a ganglioside, induces spinal cord microglia activation and central pain sensitization as an endogenous agonist of Toll-like receptor 2 on microglia. In this study, we investigated the sexual dimorphism of GT1b-induced central pain sensitization and the underlying mechanisms. GT1b administration induced central pain sensitization only in male but not in female mice. Spinal tissue transcriptomic comparison between male and female mice after GT1b injection suggested the putative involvement of estrogen (E2)-mediated

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Koca, Tuba Tülay, Hasan Göğebakan, and Gözde Çetin. "CENTRAL SENSITIZATION IN RHEUMATOID ARTHRITIS. A CROSS-SECTIONAL ANALYTIC STUDY." Central Asian Journal of Medical Hypotheses and Ethics 6, no. 1 (2025): 56–62. https://doi.org/10.47316/cajmhe.2025.6.1.06.

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Abstract Aim: Here, we analyzed the relationship between central sensitization (CS) and neuropathic pain in disease activity in patients with Rheumatoid arthritis (RA). Materials and Methods: In our study, 116 RA patients diagnosed according to the American College of Rheumatology/European League Against Rheumatism 2010 criteria were included. Central sensitization inventory (CSI) was used for central sensitization. All patients were questioned regarding central sensitization syndromes. Self-Leeds assessment of neuropathic symptom and signs (S-LANNS) and pain-detect questionnaire were used for

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De Meulemeester, Kayleigh, Mira Meeus, Robby De Pauw, Barbara Cagnie, Hannah Keppler, and Dorine Lenoir. "Suffering from chronic tinnitus, chronic neck pain, or both: Does it impact the presence of signs and symptoms of central sensitization?" PLOS ONE 18, no. 8 (2023): e0290116. http://dx.doi.org/10.1371/journal.pone.0290116.

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Chronic subjective tinnitus is a prevalent symptom, which has many similarities with chronic pain. Central sensitization is considered as a possible underlying mechanism of both symptoms. Central sensitization has already been investigated in chronic pain populations but not in patients with chronic subjective tinnitus. Therefore, the main objective of this cross-sectional study was to compare signs and symptoms, indicative for central sensitization, in tinnitus patients with and without chronic idiopathic neck pain, patients with chronic idiopathic neck pain only, and healthy controls. Also,

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AKULINUSHKINA, EKATERINA Y., SVETLANA P. YAKUPOVA, EDUARD Z. YAKUPOV, LARISA V. IVANOVA, TATYANA A. BRAGINA, and NIKOLAI I. MAKSIMOV. "CENTRAL SENSITIZATION IN PSORIATIC ARTHRITIS." Bulletin of Contemporary Clinical Medicine 16, no. 5 (2023): 16–21. http://dx.doi.org/10.20969/vskm.2023.16(5).16-21.

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Introduction. Chronic pain consists of nociceptive, neuropathic and nociplastic components. Nociplastic pain based on central sensitization. Patients with central sensitization may experience severe pain even with no any signs of clinical and laboratory inflammation, and describe it in descriptors of neuropathic pain. The prevalence and influence of central sensitization in clinical and laboratory activity, the development of mood disorders in patients with psoriatic arthritis are not explored well. Aim. To assess the prevalence of central sensitization in patients with psoriatic arthritis, to

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Tesis sobre el tema "Pain sensitization"

McCormick, Emma, and Magdalena Sjöwall. "Central sensitization in orofacial pain." Thesis, Malmö högskola, Odontologiska fakulteten (OD), 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-19833.

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Syfte. Att retrospektivt undersöka relationen mellan central sensitisering i det orofacialaområdet och refererad smärta, som kliniskt fynd, samt psykosociala faktorer hos patienter medDC/TMD-muskeldiagnosen myofasciell smärta med refererad smärta (MPR). Studien syftadeäven till att undersöka skillnader gällande psykosociala faktorer mellan patienter somdiagnostiserats med DC/TMD muskeldiagnoserna myofasciell smärta med refererad smärta(MPR), lokal myalgi (LM) och patienter med orofacial smärta eller käkdysfunktion men ejkäkmuskeldiagnos (WMD) som kontrollgrupper.Material och metod. Information

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Seitz, Viola [Verfasser]. "Sensitization and inhibition of pain / Viola Seitz." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2020. http://d-nb.info/1209130866/34.

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Schrepf, Andrew David. "Inflammation and central pain sensitization in Interstitial Cystitis/Bladder Pain Syndrome." Diss., University of Iowa, 2015. https://ir.uiowa.edu/etd/1900.

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Central sensitization refers to abnormal pain modulation present which is characterized by non-aversive or mildly aversive stimuli promoting feelings of pain. Many conditions referred to as Functional Somatic Syndromes (FSS)s are characterized by abnormal pain modulation, including pain in areas of the body not thought to be related to the specific FSS with which the patient has been diagnosed. Interstitial Cystitis/Bladder Pain Syndrome (IC/BPS) is a diagnosis of exclusion characterized by pelvic pain and urologic symptoms that shares many environmental and psychosocial correlates with FSSs.

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Janke, Elizabeth Amy. "Psychosocial Correlates of Sensitization in Chronic Pain: An Exploratory Analysis." Ohio University / OhioLINK, 2004. http://www.ohiolink.edu/etd/view.cgi?ohiou1108061243.

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Asiedu, Marina N. "Spinal Sensitization Mechanisms Promoting Pain: Gabaergic Disinhibition and Pkmζ-Mediated Plasticity". Diss., The University of Arizona, 2012. http://hdl.handle.net/10150/232496.

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As a major public health problem affecting more that 76.5 million Americans, chronic pain is one main reason why people seek medical attention. It is a pathological nervous system disorder that persists for months or years. Sensitization of nociceptive neurons in the dorsal horn of the spinal cord is crucial in the development of allodynia and hyperalgesia. The work presented in this thesis will focus on spinal protein kinase M zeta (PKMζ)-mediated plasticity and GABAergic disinhibition as spinal amplification mechanisms that orchestrate persistent changes in the dorsal horn of the spinal cord

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Warwick, Charles A. "The role of complement component C5a in nociceptive sensitization." Diss., University of Iowa, 2017. https://ir.uiowa.edu/etd/5676.

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The complement system is a principal component of innate immunity. Recent studies have underscored the importance of C5a and other complement components in inflammatory and neuropathic pain, although the underlying mechanisms are largely unknown. In particular, it is unclear how the complement system communicates with nociceptors and which ion channels and receptors are involved. Here we demonstrate that inflammatory thermal and mechanical hyperalgesia induced by complete Freund’s adjuvant were accompanied by C5a upregulation and were markedly reduced by C5a receptor (C5aR1) knockout (KO) or t

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Mickle, Aaron David. "Pain sensitization by parathyroid hormone-related peptide via convergent phosphoregulation of TRPV1." Diss., University of Iowa, 2014. https://ir.uiowa.edu/etd/5970.

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The neurobiological mechanisms underlying chronic pain in bone-metastasized breast and prostate cancer are not well understood although it is hypothesized that factors released in the tumor microenvironment may modulate sensory nociceptive sensory nerve fibers innervating the bone increasing pain sensation. Advanced metastatic breast and prostate cancer cells secrete high levels of parathyroid hormone-related peptide (PTHrP), which plays a critical role in metastasis to bones and subsequent tumor growth. PTHrP can activate parathyroid hormone receptor 1 (PTH1R), which signaling can activate ei

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Öjstedt, Erik, and Simon Pankalla. "Clinical Assessment of Disturbed Central Pain Modulation in Orofacial Pain." Thesis, Malmö universitet, Odontologiska fakulteten (OD), 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-19798.

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Syfte. Studiens syfte var att retrospektivt undersöka vilka kliniska variabler, bedömda under specialistundersökning av orofacial smärta, som kan förutsäga närvaro av en störd central smärtmodulering (DCPM). Material och metod. DC/TMD-data hämtades ur patientjournaler från 86 patienter som undersökts på Orofaciala smärtenheten på Malmö Universitet under perioden september 2012 till och med december 2013. Undersökta variabler omfattade smärtintensitet, smärtutbredning, smärtrelaterad nedsatthet, psykosociala variabler, refererad smärta samt kliniska fynd under somatosensoriska undersökningar. B

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King, Jacqlyn. "Sensorimotor Abnormalities in Chronic Subacromial Pain: The Influence of Sex, Contribution of Pain, and Utility of Using the Contralateral Limb as a Control." Thesis, University of Oregon, 2018. http://hdl.handle.net/1794/23190.

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Patients with subacromial pain syndrome (SPS) display a number of sensorimotor deficits including alterations in pain processing, poor proprioception, and weakness at the symptomatic limb. The primary purpose of this dissertation was to explore whether the aforementioned deficits: (1) can be quantified by using the non-involved limb as a measure of control, (2) are purely localized to the symptomatic limb or represent a more generalized deficit, (3) are influenced by the presence of subacromial pain, and (4) present similarly in male and female patients. Here, we utilized modern clinical techn

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Bettini, Elizabeth, and Elizabeth Bettini. "Central Sensitization and Associated Factors in Adolescents With Joint Hypermobility and Dysautonomia." Diss., The University of Arizona, 2016. http://hdl.handle.net/10150/622947.

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Background: Postural Orthostatic Tachycardia Syndrome (POTS) is a disorder of the autonomic nervous system that has high association with chronic pain syndromes such as fibromyalgia, migraine disorders, and chronic abdominal pain in adolescents with the diagnosis. Many of these disorders are characterized as central sensitization disorders, or pathological pain memory mediated by neural plasticity. Ehlers Danlos Syndrome Type 3 (EDS-3), also called joint hypermobility syndrome (JHS) is a genetic disorder of the connective tissue that causes joint laxity and is also highly associated with ch

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Libros sobre el tema "Pain sensitization"

Veldhuijzen, Dieuwke S., Henriët van Middendorp, and Andrea W. M. Evers. Stress and Sensitization in Chronic Pain. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780190627898.003.0005.

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Stress and sensitization are central concepts in chronic pain. Both can be a consequence and a contributor to the pain experience. This chapter describes the psychobiology of stress and sensitization within a multilevel perspective, indicating the impact of various forms of stress and sensitization on multiple psychoneurobiological processes (i.e., autonomic, endocrine, immune, and central processes) related to chronic pain. As a result of disordered stress regulation, sensitization may occur as a mechanism that explains how acute pain problems can become chronic and how acute pain problems ca

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Schaible, Hans-Georg, and Rainer H. Straub. Pain neurophysiology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0059.

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Physiological pain is evoked by intense (noxious) stimuli acting on healthy tissue functioning as a warning signal to avoid damage of the tissue. In contrast, pathophysiological pain is present in the course of disease, and it is often elicited by low-intensity stimulation or occurs even as resting pain. Causes of pathophysiological pain are either inflammation or injury causing pathophysiological nociceptive pain or damage to nerve cells evoking neuropathic pain. The major peripheral neuronal mechanism of pathophysiological nociceptive pain is the sensitization of peripheral nociceptors for m

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Sandkühler, Jürgen. Making the link from “central sensitization” to clinical pain. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0047.

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The landmark paper discussed in this chapter is ‘Central sensitization: Implications for the diagnosis and treatment of pain’, published by C. J. Woolf in 2011. The phrase ‘central sensitization’ is often used as an umbrella term for all kinds of central nervous system (CNS) mechanisms contributing to pain hypersensitivity. The International Association for the Study of Pain (IASP) defines ‘central sensitization’ as the ‘increased responsiveness of nociceptive neurons in the CNS’. In the CNS, highly distinct mechanisms contribute to pain hypersensitivity depending upon pain aetiology and disea

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Pak, Daniel J., and Neel Mehta. Pain Anatomy and Physiology. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190217518.003.0001.

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This chapter focuses on pain anatomy and physiology to provide a comprehensive review of the mechanisms of nociception for preparation for the ABA Pain Medicine (PM) Examination. It reviews the anatomy of pain pathways (particularly the spinothalamic sensory tract), and the process of pain conduction from peripheral nociceptors to the cerebral cortex. It also reviews the different mechanisms of sensitization and inhibition at peripheral nociceptors (manifested as primary and secondary hyperalgesia), the spinal cord (wind-up and sensitization of second-order neurons) and supraspinal structures,

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Goudie, A. J., and M. W. Emmett-Oglesby. Psychoactive Drugs: Tolerance and Sensitization. Humana Press, 2013.

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Goudie, A. J., and M. W. Emmett-Oglesby. Psychoactive Drugs: Tolerance and Sensitization. Humana Press, 1989.

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Welechew, Edward. Treatment of pain in burns patients. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199234721.003.0009.

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Pain from burns has three components: background, breakthrough, and procedural pain. Central sensitization is an important component of the ongoing pain. Early management of pain, prior to the arrival at hospital is essential. Multimodal treatment including opiates will be necessary and psychological support is key. Procedural pain is of high intensity and short duration, and will require a combination of pharmacological and non-pharmacological methods of analgesia. Central sensitization and opiate tolerance are common problems in burns patients.

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Jarrell, John F. History of Gynecological Treatment of Women's Pelvic Pain and the Recent Emergence of Pain Sensitization. Elsevier Science & Technology, 2024.

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Jarrell, John F. History of Gynecological Treatment of Women's Pelvic Pain and the Recent Emergence of Pain Sensitization. Elsevier Science & Technology Books, 2024.

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Price, Chane, Zahid Huq, Eellan Sivanesan, and Constantine Sarantopoulos. Pain Pathways and Pain Physiology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190457006.003.0001.

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Pain is a multidimensional sensory experience that is mediated by complex peripheral and central neuroanatomical pathways and mechanisms. Typically, noxious stimuli activate specific peripheral nerve terminals onto Aδ‎ and C nerve fibers that convey pain and generate signals that are relayed and processed in the spinal cord and then conveyed via the spinothalamic tracts to the contralateral thalamus and from there to the brain. Acute pain is self-limited and resolves with the healing process, but conditions of extensive injury or inflammation sensitize the pain pathways and generate aberrant,

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Capítulos de libros sobre el tema "Pain sensitization"

Jenkinson, Robert H. "Central Sensitization." In Pain. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-319-99124-5_11.

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Kam, Peter, Ian Power, Michael J. Cousins, and Philip J. Siddal. "Pain Sensitization." In Principles of Physiology for the Anaesthetist. CRC Press, 2020. http://dx.doi.org/10.1201/9780429288210-71.

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Vardeh, Daniel, and Julian F. Naranjo. "Peripheral and Central Sensitization." In Pain Medicine. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-43133-8_4.

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Bennett, David L. H. "NGF, Sensitization of Nociceptors." In Encyclopedia of Pain. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_2721.

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Gebhart, Gerald F., and Bin Feng. "Sensitization of Visceral Nociceptors." In Encyclopedia of Pain. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_3937.

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Ray, Albert L. "Neuroplasticity, Sensitization, and Pain." In Treatment of Chronic Pain by Integrative Approaches. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-1821-8_2.

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Ray, Albert L. "Neuroplasticity, Sensitization, and Pain." In Comprehensive Treatment of Chronic Pain by Medical, Interventional, and Integrative Approaches. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-1560-2_71.

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Simone, Donald A. "Spinothalamic Tract Neurons, Central Sensitization." In Encyclopedia of Pain. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_4170.

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Nicholas, Michael K. "Psychology of Pain, Sensitization, Habituation, and Pain." In Encyclopedia of Pain. Springer Berlin Heidelberg, 2013. http://dx.doi.org/10.1007/978-3-642-28753-4_3641.

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Arendt-Nielsen, Lars. "Central Sensitization in Humans: Assessment and Pharmacology." In Pain Control. Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-46450-2_5.

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Actas de conferencias sobre el tema "Pain sensitization"

Isci, Hakan, Polat Şendur, Damla Gezegen, Ramazan Ünal, and Nima Heidari. "Customized Pilot Cervical Spine Protection Orthosis Development." In Vertical Flight Society 80th Annual Forum & Technology Display. The Vertical Flight Society, 2024. http://dx.doi.org/10.4050/f-0080-2024-1076.

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Pilots and crew of rotary-wing aircraft can be exposed to inertial and task position stressors that generate pain. Repeated painful exposures with or without tissue damage are precursors to pain sensitization and chronic pain. Chronic pain leads to reduced operational readiness and long-term medical treatment. This study investigated protection orthosis for unrecoverable effects on the cervical spine by heavy helmets and accessories. A user-customized product has been developed and customization has been intended to be done with multi-body dynamic modeling and testing. Although there are many

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Ume, K., N. Seki, Y. Naganawa, T. Hyodo, K. Satoh, and Y. Kuriki. "Influence of Thermal History on Corrosion Resistance of Duplex Stainless Steel Linepipe." In CORROSION 1986. NACE International, 1986. https://doi.org/10.5006/c1986-86155.

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Abstract Using NK CR22® duplex stainless steel of 22%Cr-5.5%Ni-3%Mo, the extensive research has been carried out to analyze influences of various types of thermal cycles on corrosion resistance. Special attention was paid to the resistance to pitting corrosion in the weld heat affected zone (HAZ) through the complicated metallurgical aspect. The optimum range of welding heat input exists for the improvement of pitting corrosion resistance in the HAZ. Lower heat input brings about the deterioration of the resistance near the fusion line, higher one on the contrary degrades the HAZ apart from th

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Schall, Donald C. "New High Solids Acrylic Technologies for High Performance Topcoats: Alternatives to Acrylic Urethane Coatings." In CORROSION 1990. NACE International, 1990. https://doi.org/10.5006/c1990-90553.

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Abstract Acrylic urethanes have set a standard for high performance topcoats by virtue of their long lasting resistance and appearance properties. Such a high level of performance has made acrylic urethanes attractive to an ever increasing share of various coatings markets, including maintenance coatings. Naturally, this technology is not without its shortcomings, not the least of which are the health concerns associated with the isocyanate resins (HDI prepolymers) used in these coatings. These urethane paint vapors and mists can irritate the respiratory tract and repeated overexposure can lea

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Cruz, AR, F. Sales, J. Chaves Carvalho, E. Oliveira, and L. Agualusa. "B395 Pain interventional treatment after episiotomy sensitization." In ESRA Abstracts, 39th Annual ESRA Congress, 22–25 June 2022. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/rapm-2022-esra.471.

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Narcisse, Darryl, Robert Benkowski, Arman Fijany, and Samarendra K. Mohanty. "Pain modulation by multi-characteristic opsin sensitization of inhibitory network of brain." In Optogenetics and Optical Manipulation 2021, edited by Samarendra K. Mohanty, Anna W. Roe, and Shy Shoham. SPIE, 2021. http://dx.doi.org/10.1117/12.2586107.

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Pettersen, Pernille Steen, Tuhina Neogi, Karin Magnusson, et al. "THU0417 THE SEVERITY OF STRUCTURAL AND INFLAMMATORY FEATURES OF HAND OSTEOARTHRITIS ASSOCIATE WITH PERIPHERAL PAIN SENSITIZATION." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.3960.

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Pettersen, Pernille Steen, Tuhina Neogi, Marthe Gløersen, et al. "THU0418 NEUROPATHIC-LIKE PAIN IN PERSONS WITH HAND OSTEOARTHRITIS AND ASSOCIATIONS WITH PERIPHERAL AND CENTRAL SENSITIZATION." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.4060.

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Sena, Débora de Jesus, Maria Nathalia Gabriela Rocha Pontes de Oliveira, Maria Roberta Melo Pereira Soares, Luís Fábio Barbosa Botelho, and ALESSANDRA DE SOUSA BRAZ. "PRESENCE OF NEUROPATHIC PAIN AND/OR CENTRAL SENSITIZATION IN PATIENTS WITH ANKYLOSING SPONDYLITIS FOLLOWED AT A UNIVERSITY HOSPITAL." In XLI Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2024. https://doi.org/10.47660/cbr.2024.2144.

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Oliveira, Maria Nathalia Gabriela Rocha Pontes de, Debora de Jesus Sena, Ana Karla Guedes de Melo, Danielle Christinne Soares do Egypto, Luís Fábio Barbosa Botelho, and ALESSANDRA DE SOUSA BRAZ. "PRESENCE OF NEUROPATHIC PAIN AND/OR CENTRAL SENSITIZATION IN PATIENTS WITH ANKYLOSING SPONDYLITIS FOLLOWED AT A UNIVERSITY HOSPITAL." In XLI Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2024. https://doi.org/10.47660/cbr.2024.2143.

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Bordvik, D. H., P. Steen Pettersen, I. Kjeken, M. Gløersen, T. Neogi, and I. K. Haugen. "AB0358 SLEEP PROBLEMS ARE ASSOCIATED WITH HIGHER PAIN INTENSITY AND CENTRAL SENSITIZATION IN PEOPLE WITH HAND OSTEOARTHRITIS: DATA FROM THE NOR-HAND LONGITUDINAL STUDY." In EULAR 2024 European Congress of Rheumatology, 12-15 June. Vienna, Austria. BMJ Publishing Group Ltd and European League Against Rheumatism, 2024. http://dx.doi.org/10.1136/annrheumdis-2024-eular.4441.

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