Bibliografías temáticas / PO2

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Literatura académica sobre el tema "PO2"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 10 de febrero de 2022

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Artículos de revistas sobre el tema "PO2"

1

Hogan, M. C., D. E. Bebout, P. D. Wagner y J. B. West. "Maximal O2 uptake of in situ dog muscle during acute hypoxemia with constant perfusion". Journal of Applied Physiology 69, n.º 2 (1 de agosto de 1990): 570–76. http://dx.doi.org/10.1152/jappl.1990.69.2.570.

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We investigated the relationships among maximal O2 uptake (VO2max), effluent venous PO2 (PvO2), and calculated mean capillary PO2 (PCO2) in isolated dog gastrocnemius in situ as arterial PO2 (PaO2) was progressively reduced with muscle blood flow held constant. The hypothesis that VO2max is determined in part by peripheral tissue O2 diffusion predicts proportional declines in VO2max and PCO2 if the diffusing capacity of the muscle remains constant. The inspired O2 fraction was altered in each of six dogs to produce four different levels of PaO2 [22 +/- 2, 29 +/- 1, 38 +/- 1, and 79 +/- 4 (SE) Torr]. Muscle blood flow, with the circulation isolated, was held constant at 122 +/- 15 ml.100 g-1.min-1 while the muscle worked maximally (isometric twitches at 5-7 Hz) at each of the four different values of PaO2. Arterial and venous samples were taken to measure lactate, pH, PO2, PCO2, and muscle VO2. PCO2 was calculated using Fick's law of diffusion and a Bohr integration procedure. VO2max fell progressively (P less than 0.01) with decreasing PaO2. The decline in VO2max was proportional (R = 0.99) to the fall in both muscle PvO2 and calculated PCO2 while the calculated muscle diffusing capacity was not different among the four conditions. Fatigue developed more rapidly with lower PaO2, although lactate output from the muscle was not different among conditions. These results are consistent with the hypothesis that resistance to O2 diffusion in the peripheral tissue may be a principal determinant of VO2max.
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2

Franchini, K. G., I. A. Cestari y E. M. Krieger. "Restoration of arterial blood oxygen tension increases arterial pressure in sinoaortic-denervated rats". American Journal of Physiology-Heart and Circulatory Physiology 266, n.º 3 (1 de marzo de 1994): H1055—H1061. http://dx.doi.org/10.1152/ajpheart.1994.266.3.h1055.

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The objective of the present study was to analyze whether the hypoxemia produced by chemoreceptor elimination influences the arterial pressure level after sinoaortic denervation (SAD) in rats. Hypoxemia and hypercapnia were observed in acute (1 day) and chronic (20 days) SAD rats [arterial PO2 (PaO2) = 65 +2- 1.6 and 71 +2- 2.2 mmHg and arterial PCO2 (PaCO2) = 46 +/- 1.3 and 37 +/- 1.8 mmHg, respectively] compared with control rats (PaO2 = 85 +/- 1.6 mmHg, PaCO2 = 31 +/- 1.07 mmHg). Increasing inspired PO2 (PIO2) from 138 mmHg (room air) to 155 mmHg restored the PaO2 of SAD rats to control levels (acute = 81 +/- 2.21 mmHg, chronic = 85 +/- 2.35 mmHg). PaO2. restoration produced pronounced elevation of mean arterial pressure (MAP) of acute (from 121 +/- 4 to 147 +/- 3.5 mmHg) and chronic (from 121 +/- 3 to 134 +/- 3.5 mmHg) SAD rats. Progressive stepwise increase of PIO2 (from 138 to 175, 210, and 235 mmHg) produced no additional elevation of MAP of acute (113 +/- 4, 137 +/- 5, 143 +/- 5, and 147 +/- 5 mmHg) and chronic (111 +/- 3.6, 131 +/- 7.4, 130 +/- 8.7, and 130 +/- 7 mmHg) SAD rats. Otherwise, the arterial pressure of control rats remained unchanged to progressive stepwise increase of PIO2 (118 +/- 5, 117 +/- 4, 118 +/- 4, 116 +/- 4 mmHg). These data suggest that the elimination of chemoreceptors in SAD rats produces hypoxemia responsible for hypotensive influences that counteract the pressor effects produced by baroreceptor elimination.
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3

Roca, J., M. C. Hogan, D. Story, D. E. Bebout, P. Haab, R. Gonzalez, O. Ueno y P. D. Wagner. "Evidence for tissue diffusion limitation of VO2max in normal humans". Journal of Applied Physiology 67, n.º 1 (1 de julio de 1989): 291–99. http://dx.doi.org/10.1152/jappl.1989.67.1.291.

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We recently found [at approximately 90% maximal O2 consumption (VO2max)] that as inspiratory PO2 (PIO2) was reduced, VO2 and mixed venous PO2 (PVO2) fell together along a straight line through the origin, suggesting tissue diffusion limitation of VO2max. To extend these observations to VO2max and directly examine effluent venous blood from muscle, six normal men cycled at VO2max while breathing air, 15% O2 and 12% O2 in random order on a single day. From femoral venous, mixed venous, and radial arterial samples, we measured PO2, PCO2, pH, and lactate and computed mean muscle capillary PO2 by Bohr integration between arterial (PaO2) and femoral venous PO2 (PfvO2). VO2 and CO2 production (VCO2) were measured by expired gas analysis, VO2max averaged 61.5 +/- 6.2 (air), 48.6 +/- 4.8 (15% O2), and 38.1 +/- 4.1 (12% O2) ml.kg-1.min-1. Corresponding values were 16.8 +/- 5.6, 14.4 +/- 5.0, and 12.0 +/- 5.0 Torr for PfVO2; 23.6 +/- 3.2, 19.1 +/- 4.2, and 16.2 +/- 3.5 Torr for PVO2; and 38.5 +/- 5.4, 30.3 +/- 4.1, and 24.5 +/- 3.6 Torr for muscle capillary PO2 (PmCO2). Each of the PO2 variables was linearly related to VO2max (r = 0.99 each), with an intercept not different from the origin. Similar results were obtained when the subjects were pushed to a work load 30 W higher to ensure that VO2max had been achieved. By extending our prior observations 1) to maximum VO2 and 2) by direct sampling of femoral venous blood, we conclude that tissue diffusion limitation of VO2max may be present in normal humans. In addition, since PVO2, PfVO2, and PmCO2 all linearly relate to VO2max, we suggest that whichever of these is most readily obtained is acceptable for further evaluation of the hypothesis.
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4

Norton, J. M. "A visual aid for teaching ventilation-perfusion relationships." Advances in Physiology Education 24, n.º 1 (diciembre de 2000): 38–42. http://dx.doi.org/10.1152/advances.2000.24.1.38.

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To help students understand the concept of the ventilation-perfusion ratio (VA/Q) and the effects that VA/Q mismatching has on pulmonary gas exchange, a "sliding rectangles" visual aid was developed to teach VA/Q relationships. Adjacent rectangles representing "ventilation" and "perfusion" are slid past one another so that portions of the ventilation and perfusion rectangles are not touching, illustrating the concepts of dead-space ventilation (VD) and shunt flow (QS). The portion of the ventilation bar representing VD is further subdivided into anatomical and alveolar VD and used to show the effects of alveolar dead space on the PO2 (PAO2) and PCO2 of alveolar air (PACO2); movement away from the "ideal" point). Similarly, the portion of the perfusion bar representing QS is used to define anatomical and physiological shunts and the effect of shunts on the PO2 (PaO2) and PCO2 of arterial blood (PaCO2). The genesis of the PAO2-PaO2 (A-a) PO2 difference as well as the effects of VA/Q mismatching and diffusion abnormalities can all be discussed with this visual aid. This approach has greatly assisted some students in mastering this traditionally difficult area of respiratory physiology.
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5

Demchenko, Ivan T., Yuriy I. Luchakov, Alexander N. Moskvin, Diana R. Gutsaeva, Barry W. Allen, Edward D. Thalmann y Claude A. Piantadosi. "Cerebral Blood Flow and Brain Oxygenation in Rats Breathing Oxygen under Pressure". Journal of Cerebral Blood Flow & Metabolism 25, n.º 10 (23 de marzo de 2005): 1288–300. http://dx.doi.org/10.1038/sj.jcbfm.9600110.

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Hyperbaric oxygen (HBO2) increases oxygen tension (PO2) in blood but reduces blood flow by means of O2-induced vasoconstriction. Here we report the first quantitative evaluation of these opposing effects on tissue PO2 in brain, using anesthetized rats exposed to HBO2 at 2 to 6 atmospheres absolute (ATA). We assessed the contribution of regional cerebral blood flow (rCBF) to brain PO2 as inspired PO2 (PiO2) exceeds 1 ATA. We measured rCBF and local PO2 simultaneously in striatum using collocated platinum electrodes. Cerebral blood flow was computed from H2 clearance curves in vivo and PO2 from electrodes calibrated in vitro, before and after insertion. Arterial PCO2 was controlled, and body temperature, blood pressure, and EEG were monitored. Scatter plots of rCBF versus pO2 were nonlinear ( R2 = 0.75) for rats breathing room air but nearly linear ( R2 = 0.88–0.91) for O2 at 2 to 6 ATA. The contribution of rCBF to brain PO2 was estimated at constant inspired PO2, by increasing rCBF with acetazolamide (AZA) or decreasing it with N-nitro-l-arginine methyl ester (l-NAME). At basal rCBF (78 mL/100 g min), local PO2 increased 7- to 33-fold at 2 to 6 ATA, compared with room air. A doubling of rCBF increased striatal PO2 not quite two-fold in rats breathing room air but 13- to 64-fold in those breathing HBO2 at 2 to 6 ATA. These findings support our hypothesis that HBO2 increases PO2 in brain in direct proportion to rCBF.
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6

Lahiri, S., W. L. Rumsey, D. F. Wilson y R. Iturriaga. "Contribution of in vivo microvascular PO2 in the cat carotid body chemotransduction". Journal of Applied Physiology 75, n.º 3 (1 de septiembre de 1993): 1035–43. http://dx.doi.org/10.1152/jappl.1993.75.3.1035.

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To understand the interplay between microcirculatory control and carotid body (CB) function, we simultaneously measured carotid body microvascular PO2 (CBM PO2) and chemosensory activity in the cat in vivo under several experimental conditions. Cats were anesthetized with pentobarbital sodium, paralyzed, and artificially ventilated. CBs were exposed, and steady-state CBM PO2 was measured by the O2-dependent quenching of the phosphorescence of Pd-meso-tetra-(4-carboxyphenyl)porphine, which was administered intravenously. A few fibers of the carotid sinus nerve were used to record chemosensory discharges. At arterial PO2 (PaO2) of 103.4 +/- 4.1 Torr, CBM PO2 was 52.5 +/- 3.6 Torr (n = 9). Graded lowering of PaO2 from 160 to 50 Torr resulted in nearly proportional decreases in CBM PO2, but at lower PaO2 the decrease in CBM PO2 became more substantial. As PaO2 decreased, chemosensory discharge increased in parallel with CBM PO2. Hypercapnia and hypocapnia did not significantly change the relationship between PaO2 and CBM PO2, although the chemosensory discharge responded significantly. CBM PO2 and chemosensory discharge were not affected by hemorrhagic hypotension until arterial blood pressure fell below approximately 50 Torr and then CBM PO2 decreased and chemosensory discharge increased. The lack of a significant effect of hemorrhagic hypotension indicated that O2 delivery to CB was almost independent of the systemic blood pressure. Taken together, the observations suggest that CB microcirculation and PO2 are subject to control by intrinsic mechanisms and that CBM PO2 is compatible with oxidative metabolism playing a role in O2 chemoreception during hypoxia.
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7

Bickler, P. E., L. Litt, D. L. Banville y J. W. Severinghaus. "Effects of acetazolamide on cerebral acid-base balance". Journal of Applied Physiology 65, n.º 1 (1 de julio de 1988): 422–27. http://dx.doi.org/10.1152/jappl.1988.65.1.422.

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Acetazolamide (AZ) inhibition of brain and blood carbonic anhydrase increases cerebral blood flow by acidifying cerebral extracellular fluid (ECF). This ECF acidosis was studied to determine whether it results from high PCO2, carbonic acidosis (accumulation of H2CO3), or lactic acidosis. Twenty rabbits were anesthetized with pentobarbital sodium, paralyzed, and mechanically ventilated with 100% O2. The cerebral cortex was exposed and fitted with thermostatted flat-surfaced pH and PCO2 electrodes. Control values (n = 14) for cortex ECF were pH 7.10 +/- 0.11 (SD), PCO2 42.2 +/- 4.1 Torr, PO2 107 +/- 17 Torr, HCO3- 13.8 +/- 3.0 mM. Control values (n = 14) for arterial blood were arterial pH (pHa) 7.46 +/- 0.03 (SD), arterial PCO2 (PaCO2) 32.0 +/- 4.1 Torr, arterial PO2 (PaO2) 425 +/- 6 Torr, HCO3- 21.0 +/- 2.0 mM. After intravenous infusion of AZ (25 mg/kg), end-tidal PCO2 and brain ECF pH immediately fell and cortex PCO2 rose. Ventilation was increased in nine rabbits to bring ECF PCO2 back to control. The changes in ECF PCO2 then were as follows: pHa + 0.04 +/- 0.09, PaCO2 -8.0 +/- 5.9 Torr, HCO3(-)-2.7 +/- 2.3 mM, PaO2 +49 +/- 62 Torr, and changes in cortex ECF were as follows: pH -0.08 +/- 0.04, PCO2 -0.2 +/- 1.6 Torr, HCO3(-)-1.7 +/- 1.3 mM, PO2 +9 +/- 4 Torr. Thus excess acidity remained in ECF after ECF PCO2 was returned to control values. The response of intracellular pH, high-energy phosphate compounds, and lactic acid to AZ administration was followed in vivo in five other rabbits with 31P and 1H nuclear magnetic resonance spectroscopy.(ABSTRACT TRUNCATED AT 250 WORDS)
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8

Wimberley, P. D., K. Grønlund Pedersen, J. Olsson y O. Siggaard-Andersen. "Transcutaneous carbon dioxide and oxygen tension measured at different temperatures in healthy adults." Clinical Chemistry 31, n.º 10 (1 de octubre de 1985): 1611–15. http://dx.doi.org/10.1093/clinchem/31.10.1611.

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Abstract Transcutaneous carbon dioxide tension (tc-pco2) at 37, 39, 41, 43, and 45 degrees C, and transcutaneous oxygen tension (tc-po2) at 41, 43, and 45 degrees C were measured simultaneously in 10 healthy adults during hyperventilation and inhalation of O2/CO2 gas. Nine electrodes were applied to each subject: Five CO2 electrodes, one O2 electrode, and three combined O2/CO2 electrodes. The CO2 electrodes had negligible temperature coefficients in the calibration gases, but the O2 electrodes showed an increase in po2 of 4.5% per degree C. With skin application, tc-pco2 increased approximately 4% per degrees C between 37 and 45 degrees C, which is close to the anaerobic temperature coefficient of pco2 in blood. The tc-po2 increases on the skin with increasing temperature appeared to be more dependent on changes in blood flow in skin, but in the temperature range 43 to 45 degrees C, tc-po2 showed the expected decrease in the temperature coefficient with increasing po2. The correlation between transcutaneous and capillary pco2 was close at all transcutaneous electrode temperatures, even 37 degrees C, provided the skin was preheated (via the electrode) to 45 degrees C. For tc-po2, an electrode temperature of at least 43 degrees C was necessary to produce a reasonable correlation between tc-po2 and capillary po2. The combined O2/CO2 electrodes measured slightly higher pco2 values than the single CO2 electrodes, but there were no differences in po2 readings, stabilization time, imprecision, or electrode drift between the two electrode types. The imprecision (CV, %) of tc-pco2 and tc-po2 measurements was approximately twice that of the corresponding capillary blood-gas measurements.
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9

FORGUE, JEAN, BERNARD BURTIN y JEAN-CHARLES MASSABUAU. "MAINTENANCE OF OXYGEN CONSUMPTION IN RESTING SILURUS GLANIS AT DIFFERENT LEVELS OF AMBIENT OXYGENATION". Journal of Experimental Biology 143, n.º 1 (1 de mayo de 1989): 305–19. http://dx.doi.org/10.1242/jeb.143.1.305.

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The mechanisms of adaptation that allow the teleost Silurus glanis to maintain its resting oxygen consumption constant when the O2 partial pressure (PO2) m the inspired water (PIO2) varied between 40 and 3kPa were studied at 13 °C. Steadystate values of oxygen consumption, ventilatory and circulatory flow rates, PO2 in the inspired and expired water, PO2 and O2 concentration in the arterial and venous blood, haematocrit and acid--base status in the arterial blood were determined after 1-day exposures at selected PIO2 values. Whole-blood O2-binding characteristics were also determined. The key adaptation after 1 day of acclimation was maintenance of oxygen consumption by ventilatory adjustment with no change in blood flow rate or pH (no Bohr effect). At each PIO2 value (i) the ventilatory adjustment was minimal as the O2 extraction coefficient from water always remained around 80–90 % and (ii) PaO2 stayed constant at about 2kPa. Data are compared with previous results in crayfish and other teleosts. It is concluded that the principle of a constant O2 status in themilieu intérieur -- independent of large changes in PIO2 for a given state of activity -- should be valid in many crustaceans and teleosts.
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10

Gutierrez, G., N. Lund, A. L. Acero y C. Marini. "Relationship of venous PO2 to muscle PO2 during hypoxemia". Journal of Applied Physiology 67, n.º 3 (1 de septiembre de 1989): 1093–99. http://dx.doi.org/10.1152/jappl.1989.67.3.1093.

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Anesthetized mechanically ventilated rabbits were subjected to progressive hypoxemia (n = 7) to determine the relationship of venous PO2 (PvO2) to skeletal muscle PO2 (PtiO2). Measures of arterial PO2 (PaO2), right atrial PO2 [(PvO2)RA], and hindlimb PO2 [(PvO2)limb], were obtained from the carotid artery, right atrium, and inferior vena cava, just above the level of the iliac bifurcation. Biceps femoris muscle PtiO2 was measured with a surface O2 microelectrode having eight measuring points. PaO2 was decreased from 90.3 +/- 5.4 to 26.8 +/- 0.8 Torr in five consecutive steps, followed by reoxygenation to 105.6 +/- 10.5 (SE) Torr. Measurements were obtained after each decrement in PaO2. A total of 128 measures of PtiO2 were obtained per experimental stage. The mean and distribution of the muscle PtiO2 histogram were determined. Measurements were compared with analysis of variance and the Newman-Keuls post hoc method. (PvO2)limb had similar values as the average muscle PtiO2 (PtiO2) for PaO2 values greater than 52.1 +/- 4.3 Torr, where (PvO2)limb became greater than PtiO2 (P less than 0.05). The lowest measures of (PvO2)limb and PtiO2 were 15.9 +/- 0.7 and 4.0 +/- 0.1 Torr, respectively (P less than 0.01). The PtiO2 histograms showed no evidence of increased microvascular heterogeneity with hypoxemia. We conclude that in hypoxemia PvO2 is greater than muscle PtiO2. This difference may be related to the establishment of significant physicochemical O2 gradients from erythrocyte to tissue cell.
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Tesis sobre el tema "PO2"

1

Hodges, Alastair N. H. "Effect of hyperbaric oxygen on venous PO2, transcutaneous PO2, and VO2max in a normobaric environment". Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=30175.

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Purpose. The purpose was to examine venous PO2, transcutaneous tissue PO2 (PtcO 2), and VO2max in a normobaric environment following a single HBO2 treatment. Methods. Ten moderately trained (VO2max = 57.6 mL·kg-1·min -1) males volunteered for the study. Baseline testing included measures of VO2max, PtcO2, and anthropometry. Subjects received two HBO2 treatments, which consisted of breathing 95% oxygen at 2.5 ATA for 90 min. Following the first HBO2 treatment (6.0 +/- 1.0 min), subjects performed a VO2max test. Following the second HBO2 treatment, leg and chest PtcO2 and venous PO2 were monitored for 60 min. Results . VO2max, running time, and peak La were not altered (p < 0.05) post-HBO2 treatment. Leg PtcO2 was lower (p < 0.05) and chest PtcO2 was unchanged following the HBO2 treatment compared to baseline values. Venous PO2 was lower in the first 3 min post-HBO2 treatment than subsequent values, but no other differences were found (p < 0.05). Conclusion. The results of this study show that a single HBO 2 treatment at 2.5 ATA for 90 min does not elevate venous PO2, PtcO2, or VO2max in a normobaric, normoxic environment.
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2

Parpaleix, Alexandre. "Imagerie biphotonique de la Po2 intracérébrale : une mesure de l’activité neuronale". Thesis, Paris 5, 2013. http://www.theses.fr/2013PA05T072/document.

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L’imagerie fonctionnelle cérébrale détecte les changements hémodynamiques induits par un stimulus pour déterminer les zones d’activation neuronale. Plus particulièrement, l’imagerie BOLD en IRMf détecte les changements d’oxygénation du sang grâce aux propriétés paramagnétiques de la déoxyhémoglobine. L’oxygène n’est donc pas uniquement un substrat énergétique pour le tissu neuronal, il joue également un rôle majeur dans l’imagerie noninvasive du cerveau humain. Au cours de ma thèse, j’ai tout d’abord participé à la mise au point d’une nouvelle technique non-invasive d’imagerie de l’oxygène dans le cerveau d’animaux anesthésiés. Couplant un nouveau senseur phosphorescent de l’oxygène (Finikova et al., 2008) et la microscopie biphotonique, cette approche permet à la fois de cartographier l’oxygène en 3D avec une résolution spatiale et temporelle jusqu’alors inégalée, mais aussi de suivre simultanément l’oxygène et le flux sanguin dans les capillaires cérébraux au repos ou lors d’une activation neuronale (Lecoq et al., 2011). Tirant profit des nouvelles possibilités de cette technique, nous avons alors démontré: • la présence d’un shunt artério-veineux uniquement basé sur la diffusion de l’oxygène. Ce résultat, obtenu chez le rat dans la couche la plus superficielle du bulbe olfactif: la couche du nerf (ONL), confirme que l’oxygène ne diffuse pas uniquement à partir des capillaires et démontre que les artérioles contribuent significativement à l’oxygénation du tissu cérébral. Il démontre également qu’il n’est pas possible de déterminer ni la Po2 capillaire ni la Po2 tissulaire à partir de la Po2 veineuse. • l’existence de transitoires de Po2 associés à chaque globule rouge dans le compartiment capillaire, appelés EATs (erythrocyte-associated transients) (Hellums, 1977; Cabrales and Intaglietta, 2007). En bref, de part leur diamètre supérieur à celui de la lumière d’un capillaire, les globules rouges passent un à un dans la lumière des capillaires, laissant entre eux un espace de plasma. Cependant, la faible solubilité de l’oxygène dans le plasma crée une barrière à la diffusion, ce qui se traduit par une inhomogénéité de la Po2 capillaire: celle-ci est élevée au bord du globule rouge et décroit avec la distance pour atteindre un minimum à mi-distance entre deux globule rouges. Poursuivant l’étude des EATs (Parpaleix et al., 2013), nous avons observé les points suivants: • La Po2 tissulaire dans l’environnement immédiat d’un capillaire peut être déterminée à partir de la Po2 vasculaire à mi-distance entre deux érythrocytes. Ce résultat est intéressant en ce qu’il permettra d’effectuer des mesures non invasives de Po2 tissulaire, utile notamment chez l’animal éveillé. • L’amplitude des EATs est si large (35 mmHg en moyenne) que la Po2 capillaire moyenne ne reflète en rien la saturation en oxygène de l’hémoglobine. • Une empreinte filtrée des EATs vasculaires est détectable dans le tissu (_5 mmHg d’amplitude). • Au cours d’une stimulation neuronale, une diminution de la Po2 capillaire moyenne peut être détectée avant l’hyperémie fonctionnelle, un résultat jusqu’à présent controversé dans le domaine de l’imagerie BOLD en IRMf, mais important en ce que ce dip pourrait être un rapporteur très résolutif de l’activation neuronale. Parmi les questions restant en suspens et pouvant être étudiées finement avec notre approche, j’en citerai une principale: quel est le poids des différents facteurs (métaboliques, présynaptiques ou post-synaptiques) et du flux sanguin dans l’établissement de la Po2 cérébrale au repos?
In humans, functional mapping of brain activity mainly relies on the increase of cerebral blood flow (CBF) triggered by neuronal activation. This neurovascular coupling provides energy substrates such as oxygen and glucose to the activated area. The steady state concentration of oxygen, as well as its dynamics upon neuronal activation, have been investigated with numerous methods, however, none of them provided highly resolute measurements in depth. During my PhD, we combined a phosphorescence quenching approach with two-photon microscopy to detect, in depth and with a micrometer spatial resolution scale, the emission of phosphorescence by PtP-C343, a new oxygen nano-sensor designed for two-photon excitation. We first characterized the technique and then reported two biological results, using the olfactory bulb (OB) glomerulus as a model to study oxygen concentration, at rest and upon odor stimulation. We found an arterio-venous shunt, purely based on diffusion, in the superficial nerve layer of the OB, confirming the role of arterioles in brain oxygenation. Simultaneous measurements of Po2 and blood flow allowed us to reveal the presence of erythrocyte-associated transients (EATs), i.e. Po2 fluctuations that are associated with individual erythrocytes. Pursuing the investigation of EAT characteristics, we found that in capillaries, Po2 at mid-distance between two erythrocytes is at equilibrium with, and thus reports Po2 in the nearby neuropil. Finally, we could observe that even in capillaries, a small oxygen initial dip can be detected prior to functional hyperemia, upon odor activation
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3

Apeke, Kodjo Séna. "Modélisation ubiquiste pour l'interaction d'échelles : application à la prédiction de la réponse d'une tumeur sous traitement en radiothérapie". Thesis, Brest, 2018. http://www.theses.fr/2018BRES0086/document.

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Les travaux présentés dans le cadre de cette thèse ont porté sur la modélisation mathématique de la réponse d’une tumeur en traitement par la radiothérapie. Le but étant de fournir aux médecins un outil numérique d’aide pour diagnostiquer le cancer. Comme par exemple, suivre l’évolution du volume de la tumeur pendant et après le traitement, réadapter les stratégies thérapeutiques, etc. Dans un premier temps, nous avons proposé un modèle discret stochastique basé sur une approche multiéchelle. Dans ce contexte, nous nous sommes concentrés sur trois différentes échelles de modélisation tumorale : l’échelle microscopique (les cellules dans un voxel), l’échelle mésoscopique (population de cellules dans un voxel) et l’échelle macroscopique (tissu tumoral), avec des interfaces de transition entre ces trois échelles. Au niveau cellulaire, la description est basée sur des probabilités de transfert de phase dans le cycle cellulaire. À l’échelle mésoscopique, nous représentons les populations de cellules selon les différentes étapes d’un cycle cellulaire. Enfin, à l’échelle macroscopique, la description tumorale est basée sur l’utilisation des images médicales PET FDG. Ces trois échelles existent naturellement : les données biologiques sont collectées au niveau macroscopique mais le comportement pathologique de la tumeur est basé sur un cycle cellulaire anormal à l’échelle microscopique. L’introduction d’une échelle mésoscopique a été essentielle pour réduire l’écart entre les deux extrêmes, en termes de transition entre eux. Nous utilisons le modèle multiéchelle discret pour prédire l’évolution temporelle du nombre de cellules tumorales. Par contre, ce modèle n’est pas bien adapté pour prédire l’évolution du volume de la tumeur. Aussi, avons-nous proposé dans un second temps, un deuxième modèle qui est biomécanique et basé sur une équation d’advection réaction. Enfin, les modèles discrets multiéchelle et biomécanique ont été associés pour former un modèle hybride. En effet, le modèle discret est utilisé pour estimer les trajectoires des pressions partielles d’oxygène dans l’environnement tumoral, ces pressions sont ensuite mises en entrée du modèle continu (biomécanique) pour la prédiction de l’évolution du volume tumoral
The work presented in this thesis focused on the mathematical modeling of tumor response during treatment by radiotherapy. The goal was to provide for doctors a digital tool to help cancer diagnose. For example, monitoring tumor volume during and after treatment, rehabilitating therapeutic strategies, etc. In a first step, we proposed a discrete stochastic model based on a multiscale approach. In this context, we focused on three different scales of tumor modeling :microscopic scale (cells in a voxel), mesoscopic scale (cell population in a voxel) and macroscopic scale (tumor tissue), with transitional interfaces between these three scales. At the cellular level, the description was based on probabilities of phase transfer in the cellular cycle. At the mesoscopic scale, we represented cell populations according to the differents stages of a cell cycle. Finally, on a macroscopic scale, tumor description was based on the use of FDG PET medical images.These three scales naturally exist : the biological data were collected at the macroscopic level but the pathological behavior of the tumor is based on an abnormal cell cycle at the microscopic scale. Introduction of a mesoscopic scale was essential to reduce the gap between the two extremes, in terms of transition between them. We used the discrete multiscale model to predict the temporal evolution of the tumor cells number. On the other hand, this model was not well adapted to predict the tumor volume evolution. Thus, we had proposed a second model which was biomechanical and based on an advection reaction equation. Finally, the discrete multiscale and the biomechanical models had been combined to form a hybrid model. Indeed, the discrete model was used to estimate the oxygen partial pressures trajectories, in the tumor environment. These pressures were then input to the continuous (biomechanical) model for the tumor volume evolution prediction
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Shah, Habiba. "PO2 dependence of oxygen consumption in skeletal muscle of hypertensive and normotensive rats". VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/5000.

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Human essential hypertension affects over 75 million people in the United States, and can lead to death due to its several serious health complications such as hypertension-related cardiovascular disease. The purpose of this research was to understand how hypertension could cause physiological changes to the microcirculation, specifically the PO2 dependence of oxygen consumption (VO2) in skeletal muscle of normotensive and hypertensive rats. The Spontaneously Hypertensive Rat (SHR) strain was used as the diseased model, and Wistar-Kyoto (WKY) rats were used as controls to conduct this study. The SHR strain develops hypertension between 5-6 weeks after birth with an average systolic blood pressure of 150 mmHg. By arresting blood flow using an objective-mounted inflatable airbag, PO2 measurements were obtained along with an oxygen disappearance curve (ODC), which was used to calculate VO2 over various ranges of physiological PO2 values. PO2 and VO2 curves were analyzed based on Hill’s equation to fit the data and describe the PO2 dependence of VO2. When compared to the healthy Wistar-Kyoto rats, the SHRs exhibited a higher Vmax, or maximum rate of oxygen consumption. The average maximal rate of consumption by the hypertensive animal models could be a consequence of a “mitochondrial uncoupling” or some disconnect in the mitochondrial oxygen consumption and the normal corresponding ATP production. In conclusion, this project demonstrated that in situ muscle tissue from hypertensive and normotensive rats had a PO2 dependence of oxygen consumption over a wide range of physiological PO2 values and the hypertensive rats consumed oxygen at a higher maximal rate.
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Liles, Alexander C. "PO2 DEPENDENCE OF OXYGEN CONSUMPTION IN SKELETAL MUSCLE OF DIABETIC AND NON-DIABETIC RATS". VCU Scholars Compass, 2017. http://scholarscompass.vcu.edu/etd/4865.

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Abstract PO2 DEPENDENCE OF OXYGEN CONSUMPTION IN SKELETAL MUSCLE OF DIABETIC AND NON-DIABETIC RATS By: Alexander C. Liles A thesis submitted in partial fulfillment of the requirements for the degree of Master of Science at the Medical College of Virginia Campus, Virginia Commonwealth University Virginia Commonwealth University, 2017 Advisor: Roland N. Pittman, Ph.D. Department of Physiology and Biophysics Type 2 diabetes mellitus (T2DM) is a major medical problem around the world, affecting nearly 6% of the world’s population. This study was an attempt to better understand physiological changes the disease may cause to the microcirculation and more specifically, to assess the dependence of oxygen consumption in skeletal muscle of a diabetic animal model. The spinotrapezius muscles of Goto-Kakizaki (G-K) and Wistar control rats were used to measure interstitial using phosphorescence quenching microscopy. The G-K rats spontaneously develop T2DM and serve as an appropriate model for the disease in humans. By rapidly arresting blood flow in the tissue and observing the resulting changes, an oxygen disappearance curve (ODC) was created. The ODC was used to calculate oxygen consumption rate (VO2) over the physiological range of PO2 values. The resulting VO2 vs PO2 curves were analyzed using Hill’s equation to fit the data and obtain values of several key parameters to quantitatively describe the PO2 dependence of oxygen consumption. When compared to healthy control rats, the G-K rats exhibited a significantly higher Vmax, or maximum rate of oxygen consumption, compared to the Wistar rats. The two rat sub-strains had similar values for P50, which indicates the PO2 at half maximal consumption. The overall higher maximal rate of consumption by the diseased animals could be explained by some disconnect in the consumption of oxygen by the mitochondria and the normal corresponding production of ATP. In conclusion, it was demonstrated that in situ muscle tissue from both diabetic and non-diabetic rats had a PO2 dependence of oxygen consumption over a wide range of PO2 values and the muscles of diabetic animals consumed oxygen at a higher maximal rate.
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Schönleber, Monika M. "Studies of polymeric membranes modifed for amperometric H2O2 and pO2 sensing with needle-type electrodes". Thesis, Queen Mary, University of London, 2010. http://qmro.qmul.ac.uk/xmlui/handle/123456789/594.

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Materials used for medical devices are far from ideal for the body, so polymers are applied at the outermost region to counteract the body's natural defences. Component-based failures such as delamination and biocompatibility-based failures such as membrane fouling and degradation still remain a signifi cant challenge. This study focuses on the surface properties and modi cation of polyurethane and silicone rubber as coating material for amperometric sensing devices. E ffective synthesis of polyurethane, as well as surface modifi cation techniques performed on polymers already attached to sensing devices, are proposed. Phase inversion resulted in increased soft segment content on the surface (confi rmed by FTIR with a decreased C=O/C=C ratio). It is proposed that such an optimised polymer surface enhances the yield of further surface modi cation, such as hydroxylation (using potassium peroxodisulphate) and sulphonation (employing sodium hydride, triisobutylaluminium and 1,3 propane sultone). A novel method to generate an SO3-derivatised PU surface was proposed. Additionally, successful synthesis of silicone rubber for induced permeability of H2O2 was demonstrated. The physical and chemical properties of these modifi ed polymers were examined and evaluated via FTIR, SEM, TGA and contact angle measurements. The biocompatibility of modifi ed polymers was confi rmed with retarded protein adsorption; cytotoxicity testing showed that polymers were non-toxic to cells. Steady state amperometry on polymer modifi ed needle-type electrodes showed enhanced performance with surface modifi ed polymers to oxygen and H2O2, both of which are potential biological assay targets. Synthesised Prussian Blue (redox mediator) on platinum surfaces showed that the electrochemical response to H2O2 was increased threefold; and in combination with sulphonated polyurethane, interfering current responses could be successfully eliminated.
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Wodey, Eric. "Role de la po2 dans la toxicite hepatique de l'halothane : etude sur primoculture d'hepatocyte de rat". Rennes 1, 1992. http://www.theses.fr/1992REN1M131.

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Valente, Ana Cláudia dos Santos [UNESP]. "Valores sanguíneos de pH, pCO2, pO2, e HCO3 e proteinograma sérico de cabritos de raça Saanem do nascimento aos quatro meses de idade". Universidade Estadual Paulista (UNESP), 2002. http://hdl.handle.net/11449/89291.

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Made available in DSpace on 2014-06-11T19:23:59Z (GMT). No. of bitstreams: 0 Previous issue date: 2002Bitstream added on 2014-06-13T20:30:48Z : No. of bitstreams: 1 valente_acs_me_botfmvz.pdf: 263551 bytes, checksum: 388383d471ed3ce7de30083585f88248 (MD5)
Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
O presente trabalho teve por objetivo avaliar os valores de pH, pO2, pCO2 e HCO3- sangüíneos e proteinograma sérico de cabritos da raça Saanen, do nascimento aos quatro meses de idade. Foram utilizados vinte cabritos, filhos de cabras livres do vírus da Artrite Encefalite Caprina (CAEV), cujos partos foram acompanhados. O colostro, obtido da própria mãe, foi oferecido por meio de mamadeira em volume correspondente à 20% do peso corporal, dividido em cinco mamadas, sendo a primeira uma hora após o nascimento e as demais a cada seis horas, até completarem 24 horas. Para a realização do fracionamento eletroforético das proteínas séricas foram obtidas amostras de sangue antes de cada administração de colostro, às 48 e 72 horas e aos 7, 15, 30, 60, 90 e 120 dias. Foram colhidas, ainda, amostras de sangue total para a realização da hemogasometria em todos estes momentos e imediatamente após o nascimento. A eletroforese foi realizada utilizando-se gel de agarose. Os resultados obtidos mostraram que, ao nascimento, os cabritos da raça Saanen apresentaram primariamente quadro de acidose respiratória. As concentrações séricas das frações albumina e betaglobulina aumentaram em função da idade, ao passo que ocorreu declínio da fração alfaglobulina. A concentração das gama globulinas diminuiu após 24 horas da última administração de colostro voltando a aumentar apenas aos 30 dias de idade.
The objective of the present study was to evaluate the blood pH, pO2, pCO2 and HCO3- values, and the serum protein concentrations in Saanen kids from birth to four months of age. The study used 20 kids, borne from CAEV free goats. The kids were fed with colostrum from their own mother, in a total volume corresponding to 20% of their body weight. The whole volume was divided in five parts: the first given one hour after birth; the others in intervals of 6 hours until 24 hours of age. Samples of blood for electrophoretic fractionation were obtained before each colostrum feeding; at 48 and 72 hours; and at 7, 15, 30, 60, 90 and 120 days of life. Blood samples for the hemogasometry were obtained in the same moments and few minutes after birth. The electrophoresis was performed on agarose gel. Results demonstrated that Saanen kids present a metabolic acidosis at birth. The albumin and beta-globulin concentrations raise with the age while the alfa-globulin declines. The gama-globulin concentration decreases 24 hours after the last colostrum feeding and raises again by 30 days of age.
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Valente, Ana Cláudia dos Santos. "Valores sanguíneos de pH, pCO2, pO2, e HCO3 e proteinograma sérico de cabritos de raça Saanem do nascimento aos quatro meses de idade /". Botucatu : [s.n.], 2002. http://hdl.handle.net/11449/89291.

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Orientador: Raimundo Souza Lopes
Resumo: O presente trabalho teve por objetivo avaliar os valores de pH, pO2, pCO2 e HCO3- sangüíneos e proteinograma sérico de cabritos da raça Saanen, do nascimento aos quatro meses de idade. Foram utilizados vinte cabritos, filhos de cabras livres do vírus da Artrite Encefalite Caprina (CAEV), cujos partos foram acompanhados. O colostro, obtido da própria mãe, foi oferecido por meio de mamadeira em volume correspondente à 20% do peso corporal, dividido em cinco mamadas, sendo a primeira uma hora após o nascimento e as demais a cada seis horas, até completarem 24 horas. Para a realização do fracionamento eletroforético das proteínas séricas foram obtidas amostras de sangue antes de cada administração de colostro, às 48 e 72 horas e aos 7, 15, 30, 60, 90 e 120 dias. Foram colhidas, ainda, amostras de sangue total para a realização da hemogasometria em todos estes momentos e imediatamente após o nascimento. A eletroforese foi realizada utilizando-se gel de agarose. Os resultados obtidos mostraram que, ao nascimento, os cabritos da raça Saanen apresentaram primariamente quadro de acidose respiratória. As concentrações séricas das frações albumina e betaglobulina aumentaram em função da idade, ao passo que ocorreu declínio da fração alfaglobulina. A concentração das gama globulinas diminuiu após 24 horas da última administração de colostro voltando a aumentar apenas aos 30 dias de idade.
Abstract: The objective of the present study was to evaluate the blood pH, pO2, pCO2 and HCO3- values, and the serum protein concentrations in Saanen kids from birth to four months of age. The study used 20 kids, borne from CAEV free goats. The kids were fed with colostrum from their own mother, in a total volume corresponding to 20% of their body weight. The whole volume was divided in five parts: the first given one hour after birth; the others in intervals of 6 hours until 24 hours of age. Samples of blood for electrophoretic fractionation were obtained before each colostrum feeding; at 48 and 72 hours; and at 7, 15, 30, 60, 90 and 120 days of life. Blood samples for the hemogasometry were obtained in the same moments and few minutes after birth. The electrophoresis was performed on agarose gel. Results demonstrated that Saanen kids present a metabolic acidosis at birth. The albumin and beta-globulin concentrations raise with the age while the alfa-globulin declines. The gama-globulin concentration decreases 24 hours after the last colostrum feeding and raises again by 30 days of age.
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Larby, Line y Annie Lundberg. "Primary Study of the Phase Relationship in the MgO-"V2O3" System at 1873 K and pO2=10-11 atm". Thesis, KTH, Skolan för industriell teknik och management (ITM), 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-231901.

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Vanadium can be found in some iron ores as an impurity. Since vanadium is a valuable alloy element there lies an interest in development of a stable and economic way for extraction of it. For this, knowledge about the thermodynamics behind the V-O system combined with other elements, like Mg, is indispensable. A transitional metal like vanadium exists in multiple valences which makes it difficult to study. When vanadium oxide is mixed with other metal oxides the vanadium tends to concentrate in spinels. To investigate the types of phases formed in the Mg-O-V system at 1873 K and an oxygen partial pressure of 10-11 atm, multiple samples containing MgO and V2O3 in different proportions were equilibrated under these conditions. The samples were examined using SEM/EDS and XRD. A three, one and two phase region were found during analysis. In the three phase region MgV2O4, Mg2VO4 and an unidentified solid solution were found when the fraction of V2O3 was 0,234 and 0,448. In the one phase region only MgV2O4 was found at a V2O3 fraction interval of 0,500-0,530. Lastly in the two phase region MgV2O4 and V2O3 was found within the V2O3 fraction interval of 0,549-0,799. In conclusion when there was a higher amount of V2O3 more vanadium rich spinels were formed until a certain point where the spinel was saturated.
Vanadin finns i vissa typer av järnmalm som ett spårämne. Eftersom vanadin är ett värdefullt legeringsämne finns det ett stort intresse av att hitta ett hållbart och ekonomiskt sätt att utvinna detta. För att göra det krävs kännedom om termodynamiken bakom V-O-systemet kombinerat med andra ämnen som Mg. Övergångsmetaller som vanadin existerar i ett flertal valenser vilket kan göra det svårt att undersöka dem. När vanadinoxid blandas med andra metalloxider tenderar vanadin att samlas i spineller. För att undersöka vilka faser som bildas i Mg-O-V-systemet vid 1873 K och ett syrepartialtryck på 10-11 atm läts en serie prover bestående av olika andelar MgO och V2O3 nå jämvikt under dessa förhållanden. Analys av dessa prover gjordes med SEM/EDS och XRD. Analysen visade att det existerade ett tre-, en- och tvåfasområde i serien. I trefasområdet hittades MgV2O4, Mg2VO4 och en oidentifierad fast lösning vid V2O3- fraktionen 0,234 och 0,448. I enfasområdet hittades MgV2O4 vid V2O3-fraktionen 0,500-0,530. Till sist hittades MgV2O4 och V2O3 i tvåfasområdet vid V2O3-fraktionen 0,549-0,799. Utifrån detta drogs slutsatsen att mer vanadinrika spineller bildades då fraktionen V2O3 ökar till en viss gräns efter vilken spinellfasen blev mättad.
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Libros sobre el tema "PO2"

1

Pol Pot. San Diego, Calif: Lucent Books, 2005.

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Stefoff, Rebecca. Pol Pot. New York: Chelsea House, 1991.

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Mihovec, Janez. Pot pod noge 1. Ljubljana: Mladinska knjiga, 2004.

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Romero, Vicente. Pol Pot: El último verdugo. Barcelona, España: Planeta, 1998.

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Short, Philip. Pol Pot: Anatomy of a Nightmare. 2a ed. New York: Henry Holt and Company, 2006.

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Bazhanov, Boris. Ḣȯkmdarlar: Stalin, Pol Pot, Săddam Ḣu̇sei̐n. Baky: I̐azychy, 1993.

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Kiernan, Ben. How Pol Pot came to power. London: Verso, 1985.

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Poochie-poo: A pop-up book. Oxford: David Fickling Books, 2003.

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Pol Pot: Anatomy of a nightmare. New York: Henry Holt, 2005.

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Dreyfus, Paul. Pol Pot, le bourreau du Cambodge. [Paris]: Stock, 2000.

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Capítulos de libros sobre el tema "PO2"

1

Rooth, G., U. Ewald y F. Caligara. "Transcutaneous Po2 and Pco2 Monitoring at 37°C Cutaneous Po2 and Pco2". En Continuous Transcutaneous Monitoring, 23–32. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1927-6_4.

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Altmeyer, Peter, Klaus Hoffmann y Markus Stücker. "Transkutane Po2- und Pco2-Messung". En Kutane Mikrozirkulation, 73–119. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-642-60728-8_3.

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Kessler, M. y J. Höper. "Potentiometric Polarographic PO2 Electrode (PO2-PPE)". En Oxygen Transport to Tissue XI, 137–40. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5643-1_17.

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Böning, Dieter, Michael I. Lindinger, Damian M. Bailey, Istvan Berczi, Kameljit Kalsi, José González-Alonso, David J. Dyck et al. "Arterial PO2". En Encyclopedia of Exercise Medicine in Health and Disease, 93. Berlin, Heidelberg: Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_4065.

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Steinacker, J. M., K. Röcker y R. E. Wodick. "Arterieller pO2 bei Laufbandbelastung". En Sportmedizin — Kursbestimmung, 336–39. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-72571-5_76.

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Doyle, D. John. "Predicted Arterial PO2 (PREDO2)". En Computer Programs in Clinical and Laboratory Medicine, 41–44. New York, NY: Springer New York, 1989. http://dx.doi.org/10.1007/978-1-4612-3576-7_7.

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Schachinger, Harald y Christian Kolz. "Effectiveness of Combined Transcutaneous PO2/PcO2 Monitoring of Newborns". En Continuous Transcutaneous Monitoring, 101–4. Boston, MA: Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-1927-6_18.

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Arquint, Ph, B. H. van der Schoot y N. F. de Rooij. "Combined Blood Gas Sensor for pO2, pCO2 and pH". En Micro Total Analysis Systems, 191–94. Dordrecht: Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0161-5_18.

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Lübbers, D. W. "The Relationship Between Tissue Oxygen Pressure, Skin Surface PO2, and Transcutaneous PO2". En Clinical Oxygen Pressure Measurement, 3–13. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71226-5_1.

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Tenbrinck, R., W. Schairer, G. J. van Daal, M. H. Kuypers, G. F. J. Steeghs y B. Lachmann. "Evaluation of a Heparin-Coated PO2 Electrode for Continuous Intravasal PO2 Monitoring". En Oxygen Transport to Tissue XI, 157–62. Boston, MA: Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5643-1_20.

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Actas de conferencias sobre el tema "PO2"

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Israr, Ali, Siyan Zhao, Kyna McIntosh, JaeKyun Kang, Zachary Schwemler, Eric Brockmeyer, Mark Baskinger y Moshe Mahler. "Po2". En SIGGRAPH '15: Special Interest Group on Computer Graphics and Interactive Techniques Conference. New York, NY, USA: ACM, 2015. http://dx.doi.org/10.1145/2782782.2792489.

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"Session PO2: PO2 — Digital signal processing". En TELSIKS 2011 - 2011 10th International Conference on Telecommunication in Modern Satellite, Cable and Broadcasting Services. IEEE, 2011. http://dx.doi.org/10.1109/telsks.2011.6143258.

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Magnet, Friederike Sophie, Daniel Majorski, Jens Callegari, Sarah Bettina Schwarz, Wolfram Windisch, Jan Hendrik Storre y Claudia Schmoor. "Capillary PO2 does not adequately reflect arterial PO2 in hypoxemic COPD patients". En ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa2215.

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Tanase, D., K. Tang, P. J. French, N. Komen, G. Kleinrensink, J. Jeekel, J. F. Lange, J. J. van Veen y A. Draaijer. "PO2 and PCO2 Measurements for the Prevention and Early Detection of Anastomotic Leakage". En ASME 2008 3rd Frontiers in Biomedical Devices Conference. ASMEDC, 2008. http://dx.doi.org/10.1115/biomed2008-38046.

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Anastomotic leakage is the main, yet unsolved reason for mortality in abdominal surgery. Every year patients die because of anastomotic leakage after surgery. In the case of colorectal surgery, statistics show an incidence rate of 10–13% and a mortality rate of 32%. The aim of this work is to reduce the complications by improving the feedback to the surgeon during the operation and to minimise the fatalities by developing an early warning system. Currently, there are no peroperative methods to avoid anastomotic leakage or any validated early postoperative parameters for leakage detection. Therefore, an objective aid to detect leakage during surgery and recovery is of utmost importance.
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oude Egbrink, Mirjam G. A., Geert Jan Tangelder, Dick W. Slaaf y Robert S. Reneman. "IN VIVO CHANGES IN SYSTEMIC PCO2 AND PO2 INFLUENCE THE THROMBOEMBOLIC REACTION FOLLOWING WALL PUNCTURE IN VENULES BUT NOT IN ARTERIOLES". En XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643180.

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Changes in pH and PCO2 influence the aggregation of blood platelets in response to various agents in vitro. In the present study intravital video-microscopy was used to investigate whether changes in systemic blood gas values influence the thromboembolic reaction in vivo as induced by vessel wall injury.The microtrauma was induced by puncturing the walls of microvessels in the rabbit mesentery (diameter range: 20-40 μm) with glass micropipets (tip diameters: 6-8 μm). The thromboembolic reactions were compared in two groups of anesthetized rabbits. The control group was ventilated to keep the blood gas values within normal ranges (means: pH=7.40, pCO2=32.9 mmHg, pO2=104.7 mmHg). The experimental group breathed spontaneously (mean blood gas values: pH=7.34, pCO2=50.5 mmHg, pO2=48.1 mmHg). The pCO2 and pO2 values were significantly different between both groups.In arterioles and venules of both groups bleeding and thrombus formation started immediately following wall puncture. Bleeding times were short (medians between 1.0 and 2.6 s). Parts of the thrombi started to embolize between 11.4 and 18.2 s following wall puncture (medians). In the control group embolization continued for 101 s in the arterioles and 17 s in the venules; during these periods 6 and 1 emboli were produced, respectively (all median values). In the experimental group the duration of embolization in the arterioles was 143 s in which period 7.5 emboli were produced, values not significantly different from control. In the venules of the experimental group embolization and hence platelet reaction went on uninhibited during the whole observation period of 600 s and 30 emboli were produced. Fluid dynamic factors cannot explain the differences in thromboembolic reaction between the control and experimental venules; vessel diameters and red blood cell velocities were not significantly different between both groups. Therefore, it is likely that the change in thromboembolic reaction in the venules results from the changes in systemic PCO2 and/or pO2. The different reactions in arterioles and venules in response to the altered systemic blood gas values might arise from different reactions in the vessel walls.
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Haubner, F., F. Pohl, M. Jakob y Y. Reinders. "Imaging von pO2 und pH auf bestrahlte orale Plattenepithelkarzinomzellen". En Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1640034.

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"Session PO2: Digital signal processing, computer systems and Internet technologies". En TELSIIKS 2009 - 2009 9th International Conference on Telecommunication in Modern Satellite, Cable, and Broadcasting Services. IEEE, 2009. http://dx.doi.org/10.1109/telsks.2009.5339460.

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Mao Li, Yan Li y Peng Wen. "Tumor-induced effects on PO2 distribution in a normal tissue". En 2012 IEEE-EMBS International Conference on Biomedical and Health Informatics (BHI). IEEE, 2012. http://dx.doi.org/10.1109/bhi.2012.6211594.

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Finta, Robert y Patrick Currier. "Kalman filter for improved PO2 management in closed circuit rebreathers". En IEEE SOUTHEASTCON 2013. IEEE, 2013. http://dx.doi.org/10.1109/secon.2013.6567427.

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Haubner, F., F. Pohl, M. Jakob y Y. Reinders. "Imaging of pO2 and pH on irradiated oral squamous carcinoma cells". En Abstract- und Posterband – 89. Jahresversammlung der Deutschen Gesellschaft für HNO-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn – Forschung heute – Zukunft morgen. Georg Thieme Verlag KG, 2018. http://dx.doi.org/10.1055/s-0038-1640035.

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Informes sobre el tema "PO2"

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Johnson, T. M., W. A. Gerth y D. G. Southerland. 1.3 ATA PO2 N2-O2 Decompression Table Validation. Fort Belvoir, VA: Defense Technical Information Center, septiembre de 2000. http://dx.doi.org/10.21236/ada383688.

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Long, Edwin T. y M. J. Fennewald. Manned Evaluation of the Carleton 1.3 ATA PO2 Primary Electronics Assembly With the MK 16 Underwater Breathing Apparatus. Fort Belvoir, VA: Defense Technical Information Center, marzo de 2000. http://dx.doi.org/10.21236/ada383699.

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Gerth, Wayne A. y Thomas M. Johnson. Development and Validation of 1.3 ATA PO2-in-He Decompression Tables for the MK 16 MOD 1 UBA. Fort Belvoir, VA: Defense Technical Information Center, agosto de 2002. http://dx.doi.org/10.21236/ada407841.

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Schneider, Stefan, Ashwani Vij, Jeffrey A. Sheehy, Thorsten Schroer y Karl O. Christe. In Pursuit of the PO2+ Cation. The Reaction of KPO2F2 and SbF5 Leads to an Eight-Membered, Antimony-Oxygen-Phosphorus-Bridged Ring. Fort Belvoir, VA: Defense Technical Information Center, diciembre de 2000. http://dx.doi.org/10.21236/ada408567.

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Nelson, O. D. Portable exhausters POR-004 SKID B, POR-005 SKID C, POR-006 SKID D storage plan. Office of Scientific and Technical Information (OSTI), septiembre de 1997. http://dx.doi.org/10.2172/341248.

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Gellens, R. POP URL Scheme. RFC Editor, agosto de 1998. http://dx.doi.org/10.17487/rfc2384.

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Chacha, Tenzi y Matalie M. Howard. Soda Pop Punk. Ames: Iowa State University, Digital Repository, 2014. http://dx.doi.org/10.31274/itaa_proceedings-180814-1022.

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Mayer, Michael F. Double Spin Asymmetry in (pol)d((pol)e,e'p)n. Office of Scientific and Technical Information (OSTI), mayo de 2013. http://dx.doi.org/10.2172/1432782.

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Sánchez-Prieto, José. Silenciamiento sináptico por cannabinoides. Sociedad Española de Bioquímica y Biología Molecular (SEBBM), julio de 2016. http://dx.doi.org/10.18567/sebbmdiv_anc.2016.07.1.

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Pikin A., A. Kponou, J. Ritter y V. Zajic. Pepper Pot Emittance Meter. Office of Scientific and Technical Information (OSTI), julio de 2006. http://dx.doi.org/10.2172/1061835.

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