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Literatura académica sobre el tema "Puberty"

Autor: Grafiati

Publicado: 4 de junio de 2021

Última modificación: 12 de abril de 2025

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Artículos de revistas sobre el tema "Puberty"

Dira, I. Komang Prayoga Ariguna, I. Made Arimbawa y I. Made Darma Yuda. "Hubungan status nutrisi dengan gangguan pubertas pada remaja panti asuhan di Kota Denpasar". Intisari Sains Medis 14, n.º 3 (31 de octubre de 2023): 1016–22. http://dx.doi.org/10.15562/ism.v14i3.1829.

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Background: Puberty is a transition period between childhood and adulthood influenced by various complex factors. Nutrition is one of the most important factors affecting pubertal development. Overweight or obese children are more likely to enter puberty earlier. Severe primary or secondary malnutrition can also delay the onset and progression of puberty. This study aims to determine the relationship between nutritional status and pubertal disorders in Denpasar city orphanage children. Methods: This study was conducted using an analytic research design with a cross-sectional design to determine the relationship between nutritional status and pubertal disorders in Denpasar city orphanage children. This study involved all children in Denpasar city orphanages, Bali, between November and December 2022. Subjects who met the inclusion and exclusion criteria were included in the study. Data were analyzed using SPSS version 25.0 for Windows. Results: This study involved 160 adolescents who had reached the age of puberty in an orphanage in Denpasar City. The sample was classified as having normal puberty, 93.75%, and as many as 10 research samples were found to have puberty disorders (6.25%). The samples were classified as well-nourished (75.00%) and malnutrition (25.00%). This study shows that nutritional status has a significant relationship with pubertal disorders, with a p-value of 0.003 and a prevalence ratio of 7.00 and CI95% (1.90-25.79). Conclusion: This study found that there is a significant relationship between nutritional status and the occurrence of puberty disorders. Adolescents with malnutrition nutritional status have a seven times greater risk of experiencing puberty disorders compared to adolescents with good nutritional status. Latar Belakang: Pubertas merupakan masa transisi antara masa anak-anak dengan dewasa yang di pengeruhi oleh berbagai faktor kompleks. Nutrisi merupakan salah satu faktor terpenting yang mempengaruhi perkembangan pubertas. Anak yang kelebihan berat badan atau obesitas lebih cenderung memasuki masa pubertas lebih awal. Malnutrisi primer atau sekunder yang parah juga dapat menunda permulaan dan perkembangan pubertas. Penelitian ini bertujuan untuk mengetahui hubungan antara status nutrisi dengan gangguan pubertas pada anak panti asuhan kota Denpasar. Metode: Penelitian ini dilakukan menggunakan rancangan penelitian analitik dengan desain potong lintang untuk mengetahui hubungan antara status nutrisi dengan gangguan pubertas pada anak panti asuhan kota Denpasar. Penelitian ini melibatkan semua anak pada panti asuhan kota Denpasar, Bali, diantara periode penelitian November hingga Desember 2022. Subjek yang memenuhi kriteria inklusi dan eksklusi dimasukkan ke dalam penelitian. Hasil: Penelitian ini melibatkan 160 remaja yang telah mencapai usia pubertas di Panti Asuhan yang terletak di Kota Denpasar. Sampel tergolong memiliki pubertas normal 93,75%, sebanyak 10 sampel penelitian ditemukan mengalami gangguan pubertas (6,25%). Sampel tergolong memiliki gizi baik (75,00%) dan malnutrisi (25,00%). Penelitian ini menunjukkan status nutrisi memiliki hubungan signifikan dengan gangguan pubertas dengan nilai p sebesar 0,003 dan prevalence ratio sebesar 7,00 dan IK95% (1,90-25,79). Simpulan: Penelitian ini menemukan bahwa terdapat hubungan yang signifikan antara status nutrisi dengan terjadinya gangguan pubertas. Remaja dengan status nutrisi malnutrisi memiliki risiko tujuh kali lebih besar untuk mengalami gangguan pubertas dibandingkan dengan remaja dengan status nutrisi baik.

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Begum, Poly, Dipti Rani Saha y Md Kamrul Hassan. "Precocious Puberty A Case Report". Bangladesh Journal of Obstetrics & Gynaecology 30, n.º 2 (30 de diciembre de 2016): 109–12. http://dx.doi.org/10.3329/bjog.v30i2.30906.

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The parents of a 04-year-old girl bring her to a Gynaecologist because of breast development, appearance of pubic hair and periodic per vaginal bleeding. Her medical history is unremarkable. The parents are of average height, and the mother reports first menstruating when she was 11 years old. At physical examination, the girl is 100 cm tall , weighs 17 kg, and has a bodymass index of 17. Her pubertal development is classified as Tanner stage 3 breast development and Tanner stage 2 pubic hair development. She was diagnosed as a case of precocious puberity. Appearance of secondary sexual development before the age of 9 in a male child and before the age of 8 in a female child is called precocious puberty. When the cause of precocious puberty is premature activation of the hypothalamic-pituitary axis, it is called central or complete precocious puberty and she was a case of central precocious puberty. After proper consult she was treated by GnRHa suppressor of pituitary till 11 years of age.Bangladesh J Obstet Gynaecol, 2015; Vol. 30(2) : 109-112

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Klump, K. L., K. M. Culbert, J. D. Slane, S. A. Burt, C. L. Sisk y J. T. Nigg. "The effects of puberty on genetic risk for disordered eating: evidence for a sex difference". Psychological Medicine 42, n.º 3 (22 de agosto de 2011): 627–37. http://dx.doi.org/10.1017/s0033291711001541.

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BackgroundDifferences in genetic influences on disordered eating are present across puberty in girls. Heritability is 0% before puberty, but over 50% during and after puberty. Emerging data suggest that these developmental differences may be due to pubertal increases in ovarian hormones. However, a critical piece of evidence is lacking, namely, knowledge of genetic influences on disordered eating across puberty in boys. Boys do not experience increases in ovarian hormones during puberty. Thus, if pubertal increases in genetic effects are present in boys, then factors in addition to ovarian hormones may drive increases in heritability in girls. The current study was the first to examine this possibility in a sample of 1006 male and female twins from the Michigan State University Twin Registry.MethodDisordered eating was assessed with the Minnesota Eating Behavior Survey. Pubertal development was assessed with the Pubertal Development Scale.ResultsNo significant differences in genetic influences on disordered eating were observed in males across any developmental stage. Heritability was 51% in boys during pre-puberty, puberty and young adulthood. By contrast, in girls, genetic factors accounted for 0% of the variance in pre-puberty, but 51% of the variance during puberty and beyond. Sex differences in genetic effects were only significant during pre-puberty, as the best-fitting models constrained heritability to be equal across all males, pubertal females and young adult females.ConclusionsThe results highlight sex-specific effects of puberty on genetic risk for disordered eating and provide indirect evidence of a role for ovarian hormones and/or other female-specific factors.

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Sánchez-Garrido, M. A., J. M. Castellano, F. Ruiz-Pino, D. Garcia-Galiano, M. Manfredi-Lozano, S. Leon, A. Romero-Ruiz, C. Diéguez, L. Pinilla y M. Tena-Sempere. "Metabolic Programming of Puberty: Sexually Dimorphic Responses to Early Nutritional Challenges". Endocrinology 154, n.º 9 (10 de junio de 2013): 3387–400. http://dx.doi.org/10.1210/en.2012-2157.

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Body energy stores and metabolic cues influence the onset of puberty. However, the pubertal impact of early nutritional challenges has been only fragmentarily addressed. We evaluated here the consequences, in terms of pubertal timing and hormonal markers, of various nutritional manipulations during pre- or postnatal maturation in rats of both sexes. Males and females were submitted to gestational undernutrition (UNG) or peripubertal (SUB) subnutrition or were raised in large (LL; underfeeding) or small (SL; overfeeding) litters. In addition, groups of UNG, LL, and SL rats were fed on a high-fat diet (HFD) after weaning. Postnatal overfeeding resulted in higher body weights (BWs) during pubertal transition in both sexes, but only SL males displayed overtly advanced external signs of puberty. Postnatal underfeeding persistently decreased BW gain during puberty, yet the magnitude of pubertal delay was greater in LL males. In contrast, regardless of postnatal nutrition, HFD tended to advance the onset of puberty in females but did not alter pubertal timing in males. Likewise, SUB females displayed a marked delay in BW gain and puberty onset, whereas despite similar reduction in BW, SUB males showed normal timing of puberty. These sex divergences were also detected in various hormonal and metabolic indices so that postnatal overnutrition consistently increased LH, FSH, leptin, and insulin levels only in pubertal females, whereas HFD decreased gonadotropin levels in SL females but increased them in SL males. Notably, UNG rats did not show signs of delayed puberty but displayed a striking sex dimorphism in serum insulin/glucose levels, regardless of the diet, so that only UNG males had signs of presumable insulin resistance. Our data disclose important sex differences in the impact of various early nutritional challenges on the timing of puberty, which may help to explain the different trends of altered puberty and related comorbidities between sexes.

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Lindhardt Johansen, Marie, Casper P. Hagen, Mikkel G. Mieritz, Ole D. Wolthers, Carsten Heuck, Jørgen Holm Petersen y Anders Juul. "Pubertal Progression and Reproductive Hormones in Healthy Girls With Transient Thelarche". Journal of Clinical Endocrinology & Metabolism 102, n.º 3 (23 de diciembre de 2016): 1001–8. http://dx.doi.org/10.1210/jc.2016-2871.

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Abstract Context: Detailed evaluation of pubertal progression in girls from longitudinal studies is sparse, and the phenomenon of transient thelarche (TT), defined as the appearance, regression, and subsequent reappearance of breast buds, in healthy girls remains undescribed. Objective: To describe TT in terms of pubertal progression, growth, genotypes, and reproductive hormones and to apply new puberty nomograms for breast stages, pubic hair, and menarche. Design: A prospective, longitudinal population-based study. Patients or Other Participants: Ninety-eight healthy Danish schoolchildren (Caucasian girls) followed longitudinally as part of the COPENHAGEN Puberty Study were included in the evaluation of TT. A total of 1466 girls from 2 cross-sectional studies were included in the creation of the puberty nomograms. Intervention(s): None. Main Outcome Measure(s): Pubertal progression, specifically thelarche, reproductive hormones, genotype, and growth. Results: Twelve of 98 (12%) girls experienced TT. A larger proportion of girls with TT entered puberty by the pubarche pathway (50%) compared with girls with normal progression (15.4%), P = 0.014. Girls with TT progressed through puberty normally when evaluated using puberty nomograms. Reproductive hormones and growth velocity were lower at the first (transient) thelarche than the second (permanent) thelarche. Conclusion: TT is a frequent phenomenon that appears to be a peripheral occurrence independent of central puberty. It does not appear to affect subsequent pubertal progression as evaluated by our new puberty nomograms.

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Manotas, María Carolina, Daniel Mauricio González, Camila Céspedes, Catalina Forero y Adriana Patricia Rojas Moreno. "Genetic and Epigenetic Control of Puberty". Sexual Development 16, n.º 1 (14 de octubre de 2021): 1–10. http://dx.doi.org/10.1159/000519039.

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Puberty is a complex transitional phase in which reproductive capacity is achieved. There is a very wide variation in the age range of the onset of puberty, which follows a familial, ethnic, and sex pattern. The hypothalamic-pituitary-gonadal axis and several genetic, environmental, and nutritional factors play an important role in the onset of and throughout puberty. Recently, there has been significant progress in identifying factors that affect normal pubertal timing. Different studies have identified single nucleotide polymorphisms (SNPs) that affect pubertal timing in both sexes and across ethnic groups. Single genes are implicated in both precocious and delayed puberty, and epigenetic mechanisms have been suggested to affect the development and function of the GnRH neuronal network and responsiveness of end organs. All these factors can influence normal puberty timing, precocious puberty, and delayed puberty. The objective of this review is to describe recent findings related to the genetic and epigenetic control of puberty and highlight the need to deepen the knowledge of the regulatory mechanisms of this process in the normal and abnormal context.

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Xu, Lu, Ming Li, Jinhua Yin, Hong Cheng, Miao Yu, Xiaoyuan Zhao, Xinhua Xiao y Jie Mi. "Change of Body Composition and Adipokines and Their Relationship with Insulin Resistance across Pubertal Development in Obese and Nonobese Chinese Children: The BCAMS Study". International Journal of Endocrinology 2012 (2012): 1–10. http://dx.doi.org/10.1155/2012/389108.

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A transient increase in insulin resistance (IR) is a component of puberty. We investigated the impact of body composition and adipokines on IR during puberty in Chinese children. This study included 3223 schoolchildren aged 6–18 years. IR was calculated using homeostasis model assessment (HOMA-IR). We revealed that body mass index (BMI) and waist circumference increased gradually during puberty in both genders, while fat-mass percentage (FAT%) increased steadily only in girls. Change of leptin showed striking sexual dimorphisms: in girls leptin increased steadily during puberty, whereas in boys, after a transient rise at the beginning of puberty, leptin declined by Tanner staging even in those overweight or obese. Inversely, adiponectin level decreased significantly during puberty. In both genders, HOMA-IR started to increase at the beginning of puberty, peaked in the middle, and revised at late puberty in overweight/obesity boys while it stayed high till the end of puberty in girls and normal weight boys. Multivariate regression analysis revealed that leptin presented a stronger indicator of HOMA-IR than anthropometric measures during puberty. Our results demonstrated that gender-specific FAT% and leptin changed with pubertal development. Leptin emerged as a stronger predictor of IR than traditional anthropometric indices, suggesting a prominent role in the development of pubertal IR.

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Vazquez, M. J., I. Velasco y M. Tena-Sempere. "Novel mechanisms for the metabolic control of puberty: implications for pubertal alterations in early-onset obesity and malnutrition". Journal of Endocrinology 242, n.º 2 (agosto de 2019): R51—R65. http://dx.doi.org/10.1530/joe-19-0223.

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Puberty is driven by sophisticated neuroendocrine networks that timely activate the brain centers governing the reproductive axis. The timing of puberty is genetically determined; yet, puberty is also sensitive to numerous internal and external cues, among which metabolic/nutritional signals are especially prominent. Compelling epidemiological evidence suggests that alterations of the age of puberty are becoming more frequent; the underlying mechanisms remain largely unknown, but the escalating prevalence of obesity and other metabolic/feeding disorders is possibly a major contributing factor. This phenomenon may have clinical implications, since alterations in pubertal timing have been associated to adverse health outcomes, including higher risk of earlier all-cause mortality. This urges for a better understanding of the neurohormonal basis of normal puberty and its deviations. Compelling evidence has recently documented the master role of hypothalamic neurons producing kisspeptins, encoded by Kiss1, in the neuroendocrine pathways controlling puberty. Kiss1 neurons seemingly participate in transmitting the regulatory actions of metabolic cues on pubertal maturation. Key cellular metabolic sensors, as the mammalian target of rapamycin (mTOR), AMP-activated protein kinase (AMPK) and the fuel-sensing deacetylase, SIRT1, have been recently shown to participate also in the metabolic modulation of puberty. Recently, we have documented that AMPK and SIRT1 operate as major molecular effectors for the metabolic control of Kiss1 neurons and, thereby, puberty onset. Alterations of these molecular pathways may contribute to the perturbation of pubertal timing linked to conditions of metabolic stress in humans, such as subnutrition or obesity and might become druggable targets for better management of pubertal disorders.

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Voordouw, Jasper J., Mirjam M. van Weissenbruch y Henriette A. Delemarre-van de Waal. "Intrauterine Growth Retardation and Puberty in Girls". Twin Research 4, n.º 5 (1 de octubre de 2001): 299–306. http://dx.doi.org/10.1375/twin.4.5.299.

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AbstractSome, albeit not all studies on the relationship between intrauterine growth retardation (IUGR) and female pubertal development have found an earlier and rapidly progressing puberty as well as concomitant disorders of related functional systems such as polycystic ovary syndrome and short stature. These pubertal changes are part of a growing list of IUGR-related diseases, which includes non-insulin dependent diabetes mellitus and coronary heart disease. A pulsatile release of gonadotropin releasing hormone is thought to be a conditio-sinne-qua-non for the initiation of puberty. In the absence of prospective studies on gonadotropin releasing hormone pulse patterns in IUGR-children other markers of pubertal development such as age at menarche have been deployed. From these studies it is not clear, however, whether the findings of an earlier onset of puberty in IUGR-girls merely reflect a more rapid progression of puberty. Both the role for IUGR and the mechanisms behind the onset of puberty are still elusive. Assuming a connection between IUGR and pubertal development, parallels can be drawn between hypotheses on the longterm consequences of IUGR and hypotheses on the initiation of puberty. For example, the somatometer concept proposes a role for fat mass in the initiation of puberty, which is compatible with the hypothesis on non-skeletal catch-up growth after IUGR. The debate on the origins of puberty and the role of IUGR mainly focuses on nature and nurture. Judgmentally, studies in mono- and dizygotic twins discordant for birth weight may be of particular help.

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Tesis sobre el tema "Puberty"

Bovard, Joshua Maschio. "Does competitive swimming during puberty affect lung development in pubertal females?" Thesis, University of British Columbia, 2017. http://hdl.handle.net/2429/62896.

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Whether the large lungs of competitive swimmers result from intensive swim training or genetic endowment has been widely debated. Given that peak growth velocities for the lungs occur during puberty, this longitudinal study aimed to determine if competitive swimming during puberty affected lung development. Female swimmers (n=11) and healthy controls (n=10) aged 11-14 years old were assessed before and after one competitive swimming season. Pulmonary function testing included lung volumes, spirometry, diffusion capacity (DL,CO), and maximal inspiratory (PIMAX) and expiratory (PEMAX) pressures. Ventilatory constraints, including end-expiratory lung volume (EELV), expiratory flow limitation (EFL), and utilization of ventilatory capacity (V̇E/V̇ECAP), were assessed during an incremental cycling test. Despite being of similar age (p=0.10), maturational development (p=0.27), and height (p=0.38) as controls, swimmers had a larger total lung capacity (p<0.01), forced vital capacity (p<0.01), and peak expiratory flow (p=0.03). Although DL,CO was greater in swimmers (p=0.01), there was no difference when expressed relative to alveolar volume (p=0.20). Both PIMAX (p=0.06) and PEMAX (p<0.001) were greater in swimmers. Swimmers and controls achieved a similar relative maximal oxygen consumption (p=0.32) and experienced similar ventilatory constraints as characterized by EELV (p=0.18), severity (p=0.95) and prevalence (p=0.71) of EFL, and V̇E/V̇ECAP (p=0.95). Changes over time were similar between groups (p>0.05). Pubertal female swimmers already had larger lung capacities, higher flows, and greater indices of respiratory muscle strength, but similar ventilatory constraints while cycling. One competitive swimming season did not further accentuate this enhanced function or alter exercise ventilatory mechanics, suggesting that competitive swimming during puberty did not affect lung development.
Education, Faculty of
Kinesiology, School of
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Cohen, Robin Zoe. "Puberty and scizophrenia". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0003/MQ40801.pdf.

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Jeffreys, Renee M. Ph D. "Physical Activity and Pubertal Onset: Longitudinal Analysis of the Puberty Study Cohorts". University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1406820304.

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Lee, Bo Yeon. "Action of manganese on puberty". Diss., Texas A&M University, 2003. http://hdl.handle.net/1969.1/5871.

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Manganese (Mn) is considered important for normal growth and reproduction. Because Mn can cross the blood brain barrier and accumulate in the hypothalamus, and because it has been suggested that infants and children are potentially more sensitive to Mn than adults, we wanted to determine the effects of Mn exposure on puberty-related hormones and the onset of puberty, and discern the site and mechanism of Mn action. We demonstrated that the central administration of manganese chloride (MnCl2) stimulated luteinizing hormone (LH) release in prepubertal rats. Incubation of medial basal hypothalamus (MBH) in vitro showed this effect was due to a Mn-induced stimulation of luteinizing hormone releasing hormone (LHRH). Further demonstration that this is a hypothalamic site of action was shown by in vivo blockade of LHRH receptors and the lack of a direct pituitary action of Mn to stimulate LH release in vitro. Chronic supplementation of low dose of MnCl2 caused elevated serum levels of LH, follicle stimulating hormone (FSH) and estradiol or testosterone. Importantly, Mn supplementation advanced the timing of puberty in both sexes. We investigated the mechanism by which Mn induces LHRH/LH release from the hypothalamus. Blocking the NMDA receptor, IGF1 receptor, or inhibiting nitric oxide synthase in vivo was ineffective in altering Mn-induced LH release. Dose-response, pharmacological blockade and nitrite assessments indicated that the lowest doses of Mn used stimulated LHRH release, but did not induce nitric oxide (NO) production, while only the highest dose of Mn stimulated NO. Conversely, a dose-dependent inhibition of Mn-induced LHRH release was observed in the presence of ODQ, a specific blocker of soluble guanylyl cyclase. Furthermore, Mn stimulated the release of cyclic GMP (cGMP) and LHRH from the same MBH, and a protein kinase G (PKG) inhibitor, KT5823, blocked Mn-induced LHRH release. Collectively, these data demonstrate that Mn can stimulate specific puberty-related hormones both acutely and chronically, and furthermore, suggest that low levels of Mn facilitate the normal onset of puberty. The principal action of Mn within the hypothalamus is to facilitate the activation of guanylyl cyclase, which subsequently stimulates the cGMP/PKG pathway resulting in the stimulation of prepubertal LHRH secretion.

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Balzer, Ben William Robert. "NOVEL WAYS OF ASSESSING PUBERTY". Thesis, The University of Sydney, 2019. https://hdl.handle.net/2123/21771.

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Puberty involves profound anthropometric and hormonal changes that have been associated with changes in mood, behaviour and other health risks that emerge in adolescence. However, there is little longitudinal research in the effects of puberty on adolescents. The overarching aim of this thesis was to demonstrate novel ways of studying puberty and how these improve our understanding of the longitudinal changes that occur over this period. Oestradiol is popularly associated with changes in mood and depression and testosterone with aggression and risk taking. Chapters 3 and 4 reviewed the effects of oestradiol on female, and testosterone on male adolescent mood and behaviour. In both reviews there were inconsistent cross-sectional data, and limited longitudinal data, supporting such associations. In Chapter 5, text messaging reminders for study compliance was studied. Specimen collection correlated with text message reply time. Liquid chromatography-tandem mass spectrometry assays on urine and serum specimens quantified oestradiol, testosterone and luteinising hormone levels in 97% of urine and 95% of serum specimens (Chapter 6). Hormone changes over one year were not linear, with frequent urine sampling offering a more nuanced description of puberty hormone change. Self-rated Tanner stage and other subjective measures of puberty (adolescent- and parent-rated) were compared with longitudinal changes in serum hormones in Chapter 7. Positive longitudinal associations were observed between these subjective measures and hormone changes. Chapter 8 identifies foot length growth as a novel marker for early puberty changes. Positive longitudinal relationships were observed between foot length, height, weight, Tanner stage and serum sex steroids. Foot length offers a practical, novel and cost-effective marker of early puberty. This thesis provides novel insights into puberty and adolescence, particularly how this life transition is studied. The importance of longitudinal research to determine the true effects of puberty hormone changes on adolescents is highlighted, and data are provided to show how this can be feasibly achieved.

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Kim, Kenneth. "Family structure, puberty and reproductive development". Thesis, University of Sheffield, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.266838.

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Mobley, Stacey Lloyd. "Calcium kinetics in girls during puberty /". The Ohio State University, 2000. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488191667183775.

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Paczkowski, Melissa Jeanne. "Effects of experimental fascioliasis on puberty and comparison of mounting activity by radiotelemetry in pubertal and gestating beef heifers". Texas A&M University, 2004. http://hdl.handle.net/1969.1/2796.

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Angus-sired heifers were allotted by age (mean=4 mo), BW (mean=135 kg), and sire (n=4) to either a control (n=10) or infected group (n=11; 600 metacercariae of Fasciola hepatica, intraruminally) to test our hypothesis that puberty is delayed by experimental fascioliasis. Blood samples were collected biweekly for analysis of steroid hormone concentrations. At 2-wk intervals, BW was recorded, and samples were collected for analysis of liver enzymes and serum proteins and fecal egg counts. A radiotelemetry system (HeatWatch??) was used to detect estrus and ovulation was confirmed by an elevation in serum progesterone (P4) after estrus. Heifers were artificially inseminated (AI) at the second observed estrus. Serum γ-glutamyl transpeptidase (GGT) and aspartate aminotransferase (AST) increased (p<0.0008) between day 0 and 112 in the infected group. Serum estradiol (E2) and P4 concentrations did not differ (p>0.1) between treatment groups. Mean age at puberty was 10 days later (p>0.1) in the infected group. Conception rate did not differ between control and infected heifers. The HeatWatch?? data were used to compare mounting activity during estrus in pubertal and gestating heifers. Mean duration of estrus was longer (p<0.01) for the second than for the pubertal estrus, though total mount duration and number of mounts did not differ. Number of mounts at second estrus was greater (p<0.05) for heifers that conceived (n=9). Mean duration of estrus and total mount duration at second estrus were not associated with pregnancy outcome. Estrus events were detected in all nine heifers during pregnancy (total=73). A majority (75%) of the interestrus intervals during gestation was <17 d. Number of mounts (p=0.035) and total duration of mounts (p=0.022) at second estrus were predictive of number of mounts during gestation. Experimental infection of Fasciola hepatica did not alter serum steroid hormone concentration or delay pubertal development in heifers. Estrus duration was longer for the second estrus compared to the pubertal estrus, and the number of mounts received during the second estrus was greater in heifers that did conceive to AI. Estrus events were detected in each heifer during pregnancy; however, a normal interestrus interval occurred in only 10% of the estrus events.

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Patterson, Jennifer Lynne. "Factors influencing onset of puberty in gilts". Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ60485.pdf.

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Bridges, Nicola Anne. "The endocrine and physical events of puberty". Thesis, University of Southampton, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.295696.

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Libros sobre el tema "Puberty"

Munch, Edvard. Edvard Munch: Pubertet/puberty. [Oslo]: Orfeus Publishing, 2012.

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International Conference on the Control of the Onset of Puberty (3rd 1989 Amsterdam, Netherlands). Control of the onset of puberty III: Proceedings of the 3rd International Conference of the onset of puberty ... Editado por Delemarre-van de Waal, H. A. Amsterdam: Excerpta Medica, 1989.

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Kumanov, Philip y Ashok Agarwal, eds. Puberty. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6.

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Wright, Susan Elliot. Puberty. London: Wayland, 2007.

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Ciallella, Mia. Puberty! New York, NY: The author, 2016.

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Bouvattier, Claire y Catherine Pienkowski, eds. Early Puberty. Paris: Springer Paris, 2016. http://dx.doi.org/10.1007/978-2-8178-0543-6.

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Dietrich, Jennifer E., ed. Female Puberty. New York, NY: Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4939-0912-4.

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Schofield, Nell. Puberty blues. Sydney: Currency Press, 2004.

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Appelbaum, Heather L., ed. Abnormal Female Puberty. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-27225-2.

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Levete, Sarah. Sex and puberty. London: Wayland, 2009.

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Capítulos de libros sobre el tema "Puberty"

Uenoyama, Yoshihisa, Naoko Inoue, Nahoko Ieda, Vutha Pheng, Kei-ichiro Maeda y Hiroko Tsukamura. "Maturation and Physiology of Hypothalamic Regulation of the Gonadal Axis". En Puberty, 1–11. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_1.

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Léger, Juliane y Jean-Claude Carel. "Precocious Puberty". En Puberty, 137–54. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_10.

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Lee, Peter A. y Christopher P. Houk. "Constitutional Delayed Puberty". En Puberty, 155–68. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_11.

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Passby, Lauren C., Kavitha Rozario y Jyothis T. George. "Pubertal Dysfunction: A Disorder of GnRH Pulsatility". En Puberty, 169–81. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_12.

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Kumanov, Philip. "Pubertal Gynecomastia". En Puberty, 183–96. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_13.

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Thappa, Devinder Mohan y Munisamy Malathi. "Pubertal Acne". En Puberty, 197–209. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_14.

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Majzoub, Ahmad y Edmund Sabanegh. "Adolescent Varicocele". En Puberty, 211–28. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_15.

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Gupta, Sajal, Elizabeth Pandithurai y Ashok Agarwal. "Polycystic Ovary Syndrome in Adolescent Girls". En Puberty, 229–45. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_16.

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Svinarov, Dobrin A. "Growth Hormone and Steroid Assays’ Problems in Childhood and Puberty". En Puberty, 247–61. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_17.

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Evans, Yolanda N. "Psychosocial Development of Adolescents With and Without Deviations". En Puberty, 263–72. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-32122-6_18.

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Actas de conferencias sobre el tema "Puberty"

Enachescu, Vladimir-Aurelian, Catalin Gheorghe Bejinariu y Costin Petcu. "ADAPTING THE SCHOOL ORGANIZATION TO PRECOCIOUS PUBERTY - MULTIPLE IMPLICATIONS OF AN INCREASINGLY PRESENT PHENOMENON". En 17th annual International Conference of Education, Research and Innovation, 5400–5406. IATED, 2024. https://doi.org/10.21125/iceri.2024.1320.

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Prawismo, Sang Aji, Lilik Bintartik y M. Anas Thohir. "Development of Interactive Multimedia “Komsiklopedia” to Improve Understanding of Puberty Material of Elementary School". En 2024 10th International Conference on Education and Technology (ICET), 97–101. IEEE, 2024. https://doi.org/10.1109/icet64717.2024.10778468.

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Chen, Wen-Qi y Xiao-Yu Xin. "Bra Sizing for Adolescent Girls Based on Pubertal Tanner Staging Self-Reporting". En 17th Textile Bioengineering and Informatics Symposium, 539–45. Hung Hom, Hong Kong: Textile Bioengineering and Informatics Society Limited (TBIS), 2024. https://doi.org/10.52202/076989-0065.

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Tuteja, Geetanjali, S. Unmesh, S. Shree y S. Rudra. "Juvenile granulosa cell tumor". En 16th Annual International Conference RGCON. Thieme Medical and Scientific Publishers Private Ltd., 2016. http://dx.doi.org/10.1055/s-0039-1685332.

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The differential diagnosis for precocious puberty in a young female includes peripheral causes. This case report documents a rare cause of isosexual precocious puberty, a juvenile granulosa cell tumour of the ovary–and a brief literature review. A one year-old baby girl presented with mass abdomen, vaginal discharge and rapid onset of pubertal development. She underwent an exploratory laparotomy for tumour resection. Pathology reported a juvenile granulosa cell tumour of the ovary. Early stage granulosa cell tumor surgically treated has good prognosis. Adjuvant chemotherapy is not indicated in this setting.

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Vink, NM, DS Postma, JP Schouten, JG Rosmalen y HM Boezen. "Asthma Incidence and Transition through Puberty." En American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6231.

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Nurfadhilah, Erry Utomo y Amos Neolaka. "Puberty Hypercontent Book, Expert and Community Responses". En 5th Asian Education Symposium 2020 (AES 2020). Paris, France: Atlantis Press, 2021. http://dx.doi.org/10.2991/assehr.k.210715.020.

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Rašková, Miluše. "Communication About Puberty Among Middle-School-Aged Children". En 8th International Conference on Education and Educational Psychology. Cognitive-crcs, 2017. http://dx.doi.org/10.15405/epsbs.2017.10.18.

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Attanasi, M., A. Mian, E. R. Van Meel, S. M. Blaauwendraad, V. W. Jaddoe y L. Duijts. "Role of sex and puberty on respiratory outcomes". En ERS International Congress 2022 abstracts. European Respiratory Society, 2022. http://dx.doi.org/10.1183/13993003.congress-2022.3384.

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Bartošová, Michaela, Miluše Rašková y Alena Vavrdová. "ACHIEVED KNOWLEDGE OF PREPUBESCENTS ABOUT PUBERTY IN HUNGARY". En 14th annual International Conference of Education, Research and Innovation. IATED, 2021. http://dx.doi.org/10.21125/iceri.2021.0616.

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Informes sobre el tema "Puberty"

LeMarchand, Loic. Nutritional and Genetic Determinants of Early Puberty. Fort Belvoir, VA: Defense Technical Information Center, junio de 2005. http://dx.doi.org/10.21236/ada437668.

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Haberland, Nicole y Debbie Rogow. iMatter: Teaching about Puberty, Gender, and Fairness. Population Council, 2015. http://dx.doi.org/10.31899/pgy9.1067.

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Xu, Dan, Xueying Zhou, Junfei Wang, Xi Cao y Tao Liu. The Value of Urinary Gonadotropins in the Diagnosis of Central Precocious Puberty: A Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, diciembre de 2021. http://dx.doi.org/10.37766/inplasy2021.12.0076.

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Review question / Objective: Precocious puberty is defined as the onset of secondary sexual characteristics before the age of 8 years in girls and 9 years in boys. It can be differentiated into central precocious puberty (CPP) and peripheral precocious puberty, and it is more common in girls than in boys. CPP may result in a decreased final adult height, an early age at menarche, and psychological and health problems in adulthood. Gonadotropin-releasing hormone (GnRH) GnRH stimulation test has been indispensable in the diagnosis of CPP. GnRH stimulation test is not only invasive, time-consuming and expensive, but also sometimes difficult to have patients cooperate. Nocturnal urinary LH and FSH can represent gonadotropin excretion in children with normal and early puberty. And urinary sample collection and evaluation are more convenient, more acceptable, cheaper, and noninvasive. This meta-analysis aims to assess the value of first-voided urinary luteinizing hormone (LH) and the ratio of urinary luteinizing hormone and follicle-stimulating hormone (FSH) in the diagnosis of female CPP and to compare the accuracy between urinary gonadotropins and serum GnRH-stimulated gonadotropins.

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Graves, Kody L., Bethany Mordhorst, Nicole R. Oldfather, Elane C. Wright, Benjamin J. Hale, Aileen F. Keating, Kenneth J. Stalder y Jason W. Ross. Identification of Measures Predictive of Age at First Puberty. Ames (Iowa): Iowa State University, enero de 2013. http://dx.doi.org/10.31274/ans_air-180814-22.

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Levavi-Sivan, Berta, Serge I. Doroshov, Frank A. Chapman, Gad Degani y Joel P. Van Eenennaam. Control of Synchronization of Puberty in the Russian Sturgeon. United States Department of Agriculture, septiembre de 2012. http://dx.doi.org/10.32747/2012.7699841.bard.

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Yaron, Zvi, Abigail Elizur, Martin Schreibman y Yonathan Zohar. Advancing Puberty in the Black Carp (Mylopharyngodon piceus) and the Striped Bass (Morone saxatilis). United States Department of Agriculture, enero de 2000. http://dx.doi.org/10.32747/2000.7695841.bard.

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Both the genes and cDNA sequences encoding the b-subunits of black carp LH and FSH were isolated, cloned and sequenced. Sequence analysis of the bcFSHb and LHb5'flanking regions revealed that the promoter region of both genes contains canonical TATA sequences, 30 bp and 17 bp upstream of the transcription start site of FSHb and LHb genes, respectively. In addition, they include several sequences of cis-acting motifs, required for inducible and tissue-specific transcriptional regulation: the gonadotropin-specific element (GSE), GnRH responsive element (GRE), half sites of estrogen and androgen response elements, cAMP response element, and AP1. Several methods have been employed by the Israeli team to purify the recombinant b subunits (EtOH precipitation, gel filtration and lentil lectin). While the final objective to produce pure recombinantGtH subunits has not yet been achieved, we have covered much ground towards this goal. The black carp ovary showed a gradual increase in both mass and oocyte diameter. First postvitellogenic oocytes were found in 5 yr old fish. At this age, the testes already contained spermatozoa. The circulating LH levels increased from 0.5 ng/ml in 4 yr old fish to >5ng/ml in 5 yr old fish. In vivo challenge experiments in black carp showed the initial LH response of the pituitary to GnRH in 4 yr old fish. The response was further augmented in 5 yr old fish. The increase in estradiol level in response to gonadotropic stimulation was first noted in 4 yr old fish but this response was much stronger in the following year. In vivo experiments on the FSHb and LHb mRNA levels in response to GnRH were carried out on common carp as a model for synchronom spawning cyprinids. These experiments showed the prevalence of FSHP in maturing fish while LHP mRNA was prevalent in mature fish, especially in females. The gonadal fat-pad was found to originate from the retroperitoneal mesoderm and not from the genital ridge, thus differing from that reported in certain amphibians This tissue possibly serves as the major source of sex steroids in the immature black carp. However, such a function is taken over by the developing gonads in 4 yr old fish. In the striped bass, we described the ontogeny of the neuro-endocrine parameters along the brain-pituitary-gonadal axis during the first four years of life, throughout gonadal development and the onset of puberty. We also described the responsiveness of the reproductive axis to long-term hormonal manipulations at various stages of gonadal development. Most males reached complete sexual maturity during the first year of life. Puberty was initiated during the third year of life in most females, but this first reproductive cycle did not lead to the acquisition of full sexual maturity. This finding indicates that more than one reproductive cycle may be required before adulthood is reached. Out of the three native GnRHs present in striped bass, only sbGnRH and cGnRH II increased concomitantly with the progress of gonadal development and the onset of puberty. This finding, together with data on GtH synthesis and release, suggests that while sbGnRH and cGnRH II may be involved in the regulation of puberty in striped bass, these neuropeptides are not limiting factors to the onset of puberty. Plasma LH levels remained low in all fish, suggesting that LH plays only a minor role in early gonadal development. This hypothesis was further supported by the finding that experimentally elevated plasma LH levels did not result in the induction of complete ovarian and testicular development. The acquisition of complete puberty in 4 yr old females was associated with a rise in the mRNA levels of all GtH subunit genes, including a 218-fold increase in the mRNA levels of bFSH. mRNA levels of the a and PLH subunits increased only 11- and 8-fold, respectively. Although data on plasma FSH levels are unavailable, the dramatic increase in bFSH mRNA suggests a pivotal role for this hormone in regulating the onset and completion of puberty in striped bass. The hormonal regulation of the onset of puberty and of GtH synthesis and release was studied by chronic administration of testosterone (T) and/or an analog of gonadotropin-releasing hormone (G). Sustained administration of T+G increased the mRNA levels of the PLH subunit to the values characteristic of sexually mature fish, and also increased the plasma levels of LH. However, these changes did not result in the acceleration of sexual maturation. The mRNA levels of the bFSH subunit were slightly stimulated, but remained about 1/10 of the values characteristic of sexually mature fish. It is concluded that the stimulation of FSH gene expression and release does not lead to the acceleration of sexual maturity, and that the failure to sufficiently stimulate the bFSH subunit gene expression may underlie the inability of the treatments to advance sexual maturity. Consequently, FSH is suggested to be the key hormone to the initiation and completion of puberty in striped bass. Future efforts to induce precocious puberty in striped bass should focus on understanding the regulation of FSH synthesis and release and on developing technologies to induce these processes. Definite formulation of hormonal manipulation to advance puberty in the striped bass and the black carp seems to be premature at this stage. However, the project has already yielded a great number of experimental tools of DNA technology, slow-release systems and endocrine information on the process of puberty. These systems and certain protocols have been already utilized successfully to advance maturation in other fish (e.g. grey mullet) and will form a base for further study on fish puberty.

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Yaron, Zvi, Martin P. Schreibman, Abigail Elizur y Yonathan Zohar. Advancing Puberty in the Black Carp (Mylopharyngodon Piceus) and the Striped Bass (Morone Saxatilis). United States Department of Agriculture, agosto de 1993. http://dx.doi.org/10.32747/1993.7568102.bard.

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The black carp (bc)GtH IIb cDNA was amplified and isolated, cloned and sequenced. Comparison of the bcGtH IIb deduced a.a. sequence with that of GtH IIb from other teleosts revealed high homology to cyprinid species and a lower homology to salmonid or perciform fish. The gene coding for the GtH IIb was isolated and sequenced. Three bc recombinant phages which hybridized to the goldfish GtH Ib cDNA probe were isolated and are currently being characterized. The region coding for the mature GtH IIb was expressed in a bacterial expression vector resulting in the production of a recombinant protein. In vitro folding resulted in a protein only 1.3% of which displaced the native common carp GtH II in a RIA. Therefore, the common carp GtH RIA was utilized for the physiological studies at the current phase of the project. Two non-functional sites were identified along the brain-pituitary gonadal axis in the immature black carp. The pituitary is refractory to GnRH stimulation due to a block proximal to the activation of PKA and PKC probably at the level of GnRH receptors. The gonads, although capable of producing steroids, are refractory to gonadotropic stimulation but do respond to cAMP antagonists, indicating a block at the GtH receptor level. Attempts to advance puberty in 2 and 3 y old black carp showed that testosterone (T) stimulates GtH synthesis in the pituitary and increases its sensitivity to GnRh. A 2 month treatment combining T+GnRH increased the circulating GFtH level in 3 y old fish. Addition of domperidone to such a treatment facilitated both the accumulation of GtH in the pituitary and its response to GnRH. The cDNA of striped bass GtH a, Ib and IIb subunits were amplified, isolated, cloned and sequenced, and their deduced a.a. sequences were compared with those of other teleosts. A ribonuclease protection assay was developed for a sensitive and simultaneous determination of all GtH subunits, and of b-actin mRNAs of the striped bass. GnRH stimulated dramatically the expression of the a and GtH IIb subunits but the level of GtH Ib mRNA increased only moderately. These findings suggest that GtH-II, considered in salmonids to be involved only in final stages of gametogenesis, can be induced by GnRH to a higher extent than GtH-I in juvenile striped bass. The native GtH II of the striped bass was isolated and purified, and an ELISA for its determination was developed. The production of all recombinant striped bass GtH subunits is in progress using the insect cell (Sf9) culture and the BAC-TO-BAC baculovirus expression system. A recombinant GtH IIb subunit has been produced already, and its similarity to the native subunit was confirmed. The yield of the recombinant glycoprotein can reach 3.5 mg/ml after 3 days culture. All male striped bass reach puberty after 3 y. However, precocious puberty was discovered in 1 and 2 y old males. Females become vitellogenic during their 4th year. In immature 2 y old females, T treatment elevates the pituitary GtH II content while GnRH only potentiates the effect. However, in males GnRH and not T affects GtH accumulation in the pituitary. Neither GnRH, nor T treatment resulted in gonadal growth in 2 y old striped bass, indicating that either the accumulated GtH II was not released, or if released, the gonads were refractory to GtH stimulation, similar to the situation in the immature black carp. In 3 y old female striped bass, 150 day GnRHa treatment resulted in an increase in GSI, while T treatment, with or without GnRHa, resulted in a decrease in oocyte diameter, similar to the effect seen in the black carp. Further attempts to advance puberty in both fish species should take into account the positive effect of T on pituitary GtH and its negative effect of ovarian growth.

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Díaz, Julia A. Calderón, Jeffrey L. Vallet, Clay Lents, Danny Nonneman, Jeremy Miles, Elaine Wright, Lea Rempel et al. Optimal Dietary Energy and Protein for Gilt Development: Age at Puberty, Ovulation Rate, and Reproductive Tract Traits. Ames (Iowa): Iowa State University, enero de 2015. http://dx.doi.org/10.31274/ans_air-180814-1338.

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Luo, Pan, Xing Chen, Yuan He, Jianping Liu, Ying Liu, Fangyang Guo, Yunhai Chen, Meiying Ao y Qian Liu. Application and research status of Chinese medicine compound formulae for the treatment of precocious puberty in children. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, noviembre de 2023. http://dx.doi.org/10.37766/inplasy2023.11.0010.

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Chen, Xing, Yuan He, Jianping Liu, Ying Liu, Heyun Nie, Xinyu Yu, Ci'en Gong, Ying Wu, Meiying Ao y Qian Liu. Evaluation of Chinese medicine prescriptions for intervention in precocious puberty based on clinical cases and network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, mayo de 2024. http://dx.doi.org/10.37766/inplasy2024.5.0032.

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