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Libros sobre el tema "The immune response of T lymphocyte cell"

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Publicado: 10 de marzo de 2023
Última modificación: 31 de julio de 2025

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1

Alexander, Michael A. Immune-based cancer treatment: The T lymphocyte response. CRC Press, 2011.

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2

Alexander, Michael A. Immune-based cancer treatment: The T lymphocyte response. CRC Press, 2011.

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3

Marc, Feldmann, and Mitchison N. Avrion, eds. Immune regulation. Humana Press, 1985.

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4

Liu, Yang. The costimulatory pathway for T cell response. R.G. Landes, 1994.

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5

Rook, G. A. W. 1946- and Lightman Stafford L, eds. Steroid hormones and the T-cell cytokine profile. Springer, 1997.

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6

Miami Bio/Technology Winter Symposium (1990 Miami, Fla.). Advances in gene technology: The molecular biology of immune diseases and the immune reponse : proceedings of the 1990 Miami Bio/Technology Winter Symposia. IRL Press, 1990.

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7

B, Schook Lawrence, Tew John G, and International RES Symposium (1987 : Richmond, Va.), eds. Antigen presenting cells: Diversity, differentiation, and regulation : proceedings of a symposium held in Richmond, Virginia, March 26-29, 1987. Liss, 1988.

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8

1943-, Watson James D., and Marbrook John, eds. Recognition and regulation in cell-mediated immunity. M. Dekker, 1985.

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9

Z, Atassi M., and Abbott Laboratories, eds. Immunobiology of proteins and peptides IV: T-cell recognition and antigen presentation. Plenum Press, 1987.

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10

Na, Songqing, and Chandrasekar Venkataraman Iyer. Effector CD4+ T cells in health and disease 2007. Transworld Research Network, 2007.

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11

R, Bock Gregory, Goode Jamie, and Novartis Foundation, eds. Generation and effector functions of regulatory lymphocytes. John Wiley, 2003.

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12

International Conference on Lymphocyte Activation and Immune Regulation (9th 2002 Newport Beach, Calif.). Lymphocyte activation and immune regulation IX: Homeostasis and lymphocyte traffic. Kluwer Academic/Plenum Publishers, 2002.

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13

Sudhir, Gupta, ed. Mechanisms of lymphocyte activation and immune regulation XI: B cell biology. Springer, 2007.

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14

Brouwenstijn, Nathalie. Characterization of the T-cell mediated immune response to renal cell carcinoma. University of Leiden], 1998.

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15

Rao, Samhita Anand. Deconstructing T cell transcriptional heterogeneity and clonal dynamics in response to immune checkpoint blockade. [publisher not identified], 2022.

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16

Meding, Sally Joanna. CD4(plus) T cell effector mechanisms in the protective immune response to Plasmodium chabaudi chabaudi. Brunel University, 1991.

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17

Sudhir, Gupta, and International Conference on Mechanisms of Lymphocyte Activation and Immune Regulation (5th : 1994 : Newport Beach, Calif.), eds. Mechanisms of lymphocyte activation and immune regulation V: Molecular basis of signal transduction. Plenum Press, 1994.

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18

Constandinou, Christothea Maria. Clinical phenotypic aspects of T-cell lymphomas and Hodgkin's disease,its association with the Epstein-Barr virus, and its influence on the immune response. University of Wolverhampton, 2002.

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19

Mitchison, N. A. Immune Regulation. Island Press, 1985.

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20

Breban, Maxime, and Hill Gaston. Immune mechanisms: adaptive immunity. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0008.

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The role of adaptive immunity (i.e. the involvement of B and T lymphocytes) in the pathogenesis of axial spondyloarthritis has been investigated in both human disease and relevant animal models. Studies of B cell responses have not generally implicated an autoantibody in the disease, but there are abnormalities of antibody responses, particularly increased titres of antibodies to various gut bacteria. T cells are critical to the disease in animal models other than those where overexpression of a cytokine is engineered, suggesting that they are the drivers of the inflammatory response. There is
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21

Mucosal Immunology: Intraepithelial Lymphocytes (Advances in Host Defense Mechanisms). Raven Pr, 1994.

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22

Voll, Reinhard E., and Barbara M. Bröker. Innate vs acquired immunity. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0048.

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The innate and the adaptive immune system efficiently cooperate to protect us from infections. The ancient innate immune system, dating back to the first multicellular organisms, utilizes phagocytic cells, soluble antimicrobial peptides, and the complement system for an immediate line of defence against pathogens. Using a limited number of germline-encoded pattern recognition receptors including the Toll-like, RIG-1-like, and NOD-like receptors, the innate immune system recognizes so-called pathogen-associated molecular patterns (PAMPs). PAMPs are specific for groups of related microorganisms
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23

Moerdler, Scott, and Xingxing Zang. PD-1/PDL-1 Inhibitors as Immunotherapy for Ovarian Cancer. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190248208.003.0010.

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Programmed death 1 (PD-1), a member of the B7-CD28 immunoglobulin superfamily, and its ligands PD-L1/PD-L2 inhibit T-cell activation. They also play a key role in the tumor microenvironment, allowing for cancer immune escape. PD-1 is induced on a variety of immune cells, including tumor-infiltrating lymphocytes (TILs), while PD-L1 is found on many types of solid tumors including ovarian cancer and some TILs. The use of immunocheckpoint inhibitors like anti-PD-1 and anti-PD-L1 therapies has been shown to reactivate the immune system to attack tumor cells. Ovarian cancers have been shown to be r
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24

Muthukumar, Thangamani, Darshana Dadhania, Choli Hartono, and Manikkam Suthanthiran. Immunology, sensitization, and histocompatibility. Edited by Jeremy R. Chapman. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0279.

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Allograft rejection of the histo-incompatible allograft involves a highly orchestrated action of multiple cell types and mediators, with lymphocytes responsible for the identification of the foreignness of the allograft. The immune response directed against the donor is primarily, but not exclusively, directed at the donor’s major histocompatibility complex region class I and class II proteins. This chapter describes the immunobiology of the T cell and the role of human leucocyte antigens in clinical transplantation, thus identifying the targets for manipulation of the immune response by immun
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25

Atassi, M. Zouhair, and Howard L. Bachrach. Immunobiology of Proteins and Peptides IV: T-Cell Recognition and Antigen Presentation. Springer London, Limited, 2012.

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26

Atassi, M. Zouhair. Immunobiology of Proteins and Peptides Iv: T-Cell Recognition And Antigen Presentation. Springer, 2012.

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27

Goode, Jamie A., Gregory R. Bock, and Novartis Foundation Symposium Staff. Generation and Effector Functions of Regulatory Lymphocytes. Wiley & Sons, Incorporated, John, 2008.

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28

Tsai, Ching-Wei, Sanjeev Noel, and Hamid Rabb. Pathophysiology of Acute Kidney Injury, Repair, and Regeneration. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199653461.003.0030.

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Acute kidney injury (AKI), regardless of its aetiology, can elicit persistent or permanent kidney tissue changes that are associated with progression to end-stage renal disease and a greater risk of chronic kidney disease (CKD). In other cases, AKI may result in complete repair and restoration of normal kidney function. The pathophysiological mechanisms of renal injury and repair include vascular, tubular, and inflammatory factors. The initial injury phase is characterized by rarefaction of peritubular vessels and engagement of the immune response via Toll-like receptor binding, activation of
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29

Marincola, Francesco M., and Dirk Nagorsen. Analyzing T Cell Responses: How to Analyze Cellular Immune Responses Against Tumor Associated Antigens. Springer, 2010.

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30

Marincola, Francesco M., and Dirk Nagorsen. Analyzing T Cell Responses: How to Analyze Cellular Immune Responses Against Tumor Associated Antigens. Springer London, Limited, 2006.

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31

Gupta, Sudhir, and J. John Cohen. Mechanisms of Lymphocyte Activation and Immune Regulation VI: Cell Cycle and Programmed Cell Death in the Immune System. Springer, 2013.

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32

Alt, Frederick W., Fritz Melchers, Sudhir Gupta, Max D. Cooper, and Klaus Rajewsky. Mechanisms of Lymphocyte Activation and Immune Regulation XI: B Cell Biology. Springer, 2010.

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33

Kassiotis, George, and Adrian Liston. Regulatory T Cells: Methods and Protocols. Humana Press, 2016.

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34

Foundation, Novartis. Generation and Effector Functions of Regulatory Lymphocytes (Novartis Foundation Symposia). Wiley, 2003.

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35

Lightman, S., and G. A. W. Rook. Steroid Hormones and the T-Cell Cytokine Profile. Springer London, Limited, 2012.

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36

Lightman, S., and G. A. W. Rook. Steroid Hormones and the T-Cell Cytokine Profile. Springer London, Limited, 2011.

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37

Go, Cynthia. IL-2 gene regulation during T cell activation and anergy induction. 1992.

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38

McKisic, Maureen Denise. Characterization of ova-reactive and alloreactive CD4+ T cell subsets. 1992.

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39

(Editor), Sudhir Gupta, Frederick W. Alt (Editor), Max D. Cooper (Editor), Fritz Melchers (Editor), and Klaus Rajewsky (Editor), eds. Mechanisms of Lymphocyte Activation and Immune Regulation XI: B Cell Biology (Advances in Experimental Medicine and Biology). Springer, 2007.

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40

Cellular basis of immune modulation: Proceedings of the 19th International Leukocyte Culture Conference held at Banff Springs Hotel, Banff, Alberta, May 8-12, 1988 (Progress in leukocyte biology). A.R. Liss, 1988.

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41

(Editor), Sudhir Gupta, Eugene Butcher (Editor), and William E. Paul (Editor), eds. Lymphocyte Activation and Immune Regulation IX: Homeostasis and Lymphocyte Traffic (Advances in Experimental Medicine and Biology). Springer, 2007.

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42

Fassò, Marcella. Analysis of the CD4⁺ T cell immune response in vivo using CD4 up-regulation as an activation marker: Normal versus autoimmune peripheral CD4⁺ T cell response development. 1999.

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43

Atassi, M. Zouhair, and Howard L. Bachrach. Immunobiology of Proteins and Peptides (Advances in Experimental Medicine and Biology). Springer, 1988.

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44

Howard, Colin R. Arenaviruses. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0032.

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There are few groups of viral zoonoses that have attracted such widespread publicity as the arenaviruses, particularly during the 1960’s and 1970’s when Lassa emerged as a major cause of haemorrhagic disease in West Africa. More than any other zoonoses, members of the family are used extensively for the study of virus-host relationships. Thus the study of this unique group of enveloped, single-stranded RNA viruses has been pursued for two quite separate reasons. First, lymphocytic choriomeningitis virus (LCM) has been used as a model of persistent virus infections for over half a century; its
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45

Killer cell dynamics: Mathematical and computational approaches to immunology. Springer, 2007.

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46

Killer Cell Dynamics: Mathematical and Computational Approaches to Immunology. Springer London, Limited, 2007.

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47

Wodarz, Dominik. Killer Cell Dynamics: Mathematical and Computational Approaches to Immunology. Springer, 2010.

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48

Colbert, Robert A., and Paul Bowness. Immune mechanisms: HLA-B27. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0006.

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HLA-B27 is present in the majority of patients with ankylosing spondylitis (AS). Although we have learned a considerable amount about the natural immunologic function of HLA class I proteins, this has not provided a definitive mechanism of AS pathogenesis. While HLA-B27 is adept at presenting antigenic peptides to CD8+ T cells, ‘arthritogenic’ peptides targeted by a cross-reactive T or natural killer cell response have not been described, nor have autoreactive T cells been found. Newer concepts have evolved based on the propensity of HLA-B27 to ‘misbehave’, both inside cells and on the cell su
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49

Wodarz, Dominik. Killer Cell Dynamics: Mathematical and Computational Approaches to Immunology (Interdisciplinary Applied Mathematics). Springer, 2006.

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50

Eljaafari, Assia, and Pierre Miossec. Cellular side of acquired immunity (T cells). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0049.

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The adaptive T-cell response represents the most sophisticated component of the immune response. Foreign invaders are recognized first by cells of the innate immune system. This leads to a rapid and non-specific inflammatory response, followed by induction of the adaptive and specific immune response. Different adaptive responses can be promoted, depending on the predominant effector cells that are involved, which themselves depend on the microbial/antigen stimuli. As examples, Th1 cells contribute to cell-mediated immunity against intracellular pathogens, Th2 cells protect against parasites,
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