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Artículos de revistas sobre el tema "Tumourigenesis"

Autor: Grafiati
Publicado: 4 de junio de 2021
Última modificación: 6 de septiembre de 2023

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1

Peiser, J., A. Smith, B. Bapat, and H. Stern. "Colorectal tumourigenesis." Surgical Oncology 3, no. 4 (August 1994): 195–201. http://dx.doi.org/10.1016/0960-7404(94)90034-5.

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2

Warfel, Noel A., and Wafik S. El-Deiry. "p21WAF1 and tumourigenesis." Current Opinion in Oncology 25, no. 1 (January 2013): 52–58. http://dx.doi.org/10.1097/cco.0b013e32835b639e.

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3

Chang, Xiaotian, and Kehua Fang. "PADI4 and tumourigenesis." Cancer Cell International 10, no. 1 (2010): 7. http://dx.doi.org/10.1186/1475-2867-10-7.

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4

Reincke, M., F. Beuschlein, M. Slawik, and K. Borm. "Molecular adrenocortical tumourigenesis." European Journal of Clinical Investigation 30 (December 2000): 63–68. http://dx.doi.org/10.1046/j.1365-2362.2000.0300s3063.x.

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5

Hickman, J. "Apoptosis and tumourigenesis." Current Opinion in Genetics & Development 12, no. 1 (February 1, 2002): 67–72. http://dx.doi.org/10.1016/s0959-437x(01)00266-0.

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6

BUCKLEY, I. "Tumourigenesis: A malignant scenario." Cell Biology International Reports 15, no. 7 (July 1991): 545–49. http://dx.doi.org/10.1016/0309-1651(91)90001-y.

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7

Heijmans, J., N. V. J. A. Büller, E. Hoff, A. A. Dihal, T. van der Poll, M. A. D. van Zoelen, A. Bierhaus, et al. "Rage signalling promotes intestinal tumourigenesis." Oncogene 32, no. 9 (April 2, 2012): 1202–6. http://dx.doi.org/10.1038/onc.2012.119.

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8

Fu, K., F. Lloyd, C. Forrest, B. Klopcic, and I. Lawrance. "P036 SPARC affects colorectal tumourigenesis." Journal of Crohn's and Colitis 7 (February 2013): S24—S25. http://dx.doi.org/10.1016/s1873-9946(13)60059-8.

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9

Froldi, Francesca, Milán Szuperák, and Louise Y. Cheng. "Neural stem cell derived tumourigenesis." AIMS Genetics 2, no. 1 (2015): 13–24. http://dx.doi.org/10.3934/genet.2015.1.13.

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10

Kishida, S., and K. Kadomatsu. "Involvement of midkine in neuroblastoma tumourigenesis." British Journal of Pharmacology 171, no. 4 (January 24, 2014): 896–904. http://dx.doi.org/10.1111/bph.12442.

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11

Ernst, Matthias, and Tracy L. Putoczki. "Stat3: Linking inflammation to (gastrointestinal) tumourigenesis." Clinical and Experimental Pharmacology and Physiology 39, no. 8 (July 25, 2012): 711–18. http://dx.doi.org/10.1111/j.1440-1681.2011.05659.x.

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12

Hickman, John A. "Suppression of tumourigenesis by cell death." Toxicology 226, no. 1 (September 2006): 12–13. http://dx.doi.org/10.1016/j.tox.2006.05.019.

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13

Tominaga, Y. "Mechanism of parathyroid tumourigenesis in uraemia." Nephrology Dialysis Transplantation 14, no. 90001 (January 1, 1999): 63–65. http://dx.doi.org/10.1093/ndt/14.suppl_1.63.

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14

Thornalley, Paul J., and Naila Rabbani. "Glyoxalase in tumourigenesis and multidrug resistance." Seminars in Cell & Developmental Biology 22, no. 3 (May 2011): 318–25. http://dx.doi.org/10.1016/j.semcdb.2011.02.006.

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15

Junien, Claudine. "Beckwith-wiedemann syndrome, tumourigenesis and imprinting." Current Opinion in Genetics & Development 2, no. 3 (January 1992): 431–38. http://dx.doi.org/10.1016/s0959-437x(05)80154-6.

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16

Junien, Claudine. "Beckwith-Wiedemann syndrome, tumourigenesis and imprinting." Current Biology 2, no. 6 (June 1992): 321. http://dx.doi.org/10.1016/0960-9822(92)90888-h.

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17

Frappier, Lori. "Role of EBNA1 in NPC tumourigenesis." Seminars in Cancer Biology 22, no. 2 (April 2012): 154–61. http://dx.doi.org/10.1016/j.semcancer.2011.12.002.

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18

Poulard, Coralie, Juliette Rambaud, Emilie Lavergne, Julien Jacquemetton, Jack-Michel Renoir, Olivier Trédan, Sylvie Chabaud, Isabelle Treilleux, Laura Corbo, and Muriel Le Romancer. "Role of JMJD6 in Breast Tumourigenesis." PLOS ONE 10, no. 5 (May 7, 2015): e0126181. http://dx.doi.org/10.1371/journal.pone.0126181.

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19

Wilkinson, Simon, and Kevin M. Ryan. "Autophagy: an adaptable modifier of tumourigenesis." Current Opinion in Genetics & Development 20, no. 1 (February 2010): 57–64. http://dx.doi.org/10.1016/j.gde.2009.12.004.

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20

Quail, Daniela F., Gabrielle M. Siegers, Michael Jewer, and Lynne-Marie Postovit. "Nodal signalling in embryogenesis and tumourigenesis." International Journal of Biochemistry & Cell Biology 45, no. 4 (April 2013): 885–98. http://dx.doi.org/10.1016/j.biocel.2012.12.021.

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21

Atkinson, Stuart P., and W. Nicol Keith. "Epigenetic control of cellular senescence in disease: opportunities for therapeutic intervention." Expert Reviews in Molecular Medicine 9, no. 7 (March 2007): 1–26. http://dx.doi.org/10.1017/s1462399407000269.

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AbstractUnderstanding how senescence is established and maintained is an important area of study both for normal cell physiology and in tumourigenesis. Modifications to N-terminal tails of histone proteins, which can lead to chromatin remodelling, appear to be key to the regulation of the senescence phenotype. Epigenetic mechanisms such as modification of histone proteins have been shown to be sufficient to regulate gene expression levels and specific gene promoters can become epigenetically altered at senescence. This suggests that epigenetic mechanisms are important in senescence and further
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22

Bai, Xiao-Hui, Hae-Ra Cho, Serisha Moodley, and Mingyao Liu. "XB130—A Novel Adaptor Protein: Gene, Function, and Roles in Tumorigenesis." Scientifica 2014 (2014): 1–9. http://dx.doi.org/10.1155/2014/903014.

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Several adaptor proteins have previously been shown to play an important role in the promotion of tumourigenesis. XB130 (AFAP1L2) is an adaptor protein involved in many cellular functions, such as cell survival, cell proliferation, migration, and gene and miRNA expression. XB130’s functional domains and motifs enable its interaction with a multitude of proteins involved in several different signaling pathways. As a tyrosine kinase substrate, tyrosine phosphorylated XB130 associates with the p85αregulatory subunit of phosphoinositol-3-kinase (PI3K) and subsequently affects Akt activity and its
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23

Wu, X., X. Liu, N. Lan, X. Zheng, Y. Chen, Z. Cai, P. Lan, and X. Wu. "P069 CD73 promotes colitis-associated tumourigenesis in mice." Journal of Crohn's and Colitis 14, Supplement_1 (January 2020): S170. http://dx.doi.org/10.1093/ecco-jcc/jjz203.198.

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Abstract Background Patients with inflammatory bowel disease (IBD) are at a higher risk of developing colitis-associated colorectal cancer. The aim of the present study was to investigate the role of CD73 in IBD-associated tumourigenesis. Methods A mouse model of colitis-associated tumourigenesis (CAT) induced by azoxymethane and dextran sulphate sodium (AOM/DSS) was successfully constructed. Model mice were injected with CD73 inhibitor or adenosine receptor agonist. Colon length, body weight loss and tumour formation were assessed macroscopically. Measurement of inflammatory cytokines and RNA
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24

Aranha, M. M., P. M. Borralho, P. Ravasco, I. B. Moreira da Silva, L. Correia, A. Fernandes, M. E. Camilo, and C. M. P. Rodrigues. "NF-?B and apoptosis in colorectal tumourigenesis." European Journal of Clinical Investigation 37, no. 5 (May 2007): 416–24. http://dx.doi.org/10.1111/j.1365-2362.2007.01801.x.

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25

Levy, Andy. "Molecular and Trophic Mechanisms of Pituitary Tumourigenesis." Hormone Research in Paediatrics 76, s1 (2011): 2–6. http://dx.doi.org/10.1159/000329114.

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26

Ioannou, Savvas, and Michael Voulgarelis. "Toll-Like Receptors, Tissue Injury, and Tumourigenesis." Mediators of Inflammation 2010 (2010): 1–9. http://dx.doi.org/10.1155/2010/581837.

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Toll-like receptors (TLRs) belong to a class of molecules known as pattern recognition receptors, and they are part of the innate immune system, although they modulate mechanisms that impact the development of adaptive immune responses. Several studies have shown that TLRs, and their intracellular signalling components, constitute an important cellular pathway mediating the inflammatory process. Moreover, their critical role in the regulation of tissue injury and wound healing process as well as in the regulation of apoptosis is well established. However, interest in the role of these receptor
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27

Nemenoff, Raphael A., and Robert A. Winn. "Role of nuclear receptors in lung tumourigenesis." European Journal of Cancer 41, no. 16 (November 2005): 2561–68. http://dx.doi.org/10.1016/j.ejca.2005.08.015.

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28

Chatterjee, Aniruddha, Euan J. Rodger, and Michael R. Eccles. "Epigenetic drivers of tumourigenesis and cancer metastasis." Seminars in Cancer Biology 51 (August 2018): 149–59. http://dx.doi.org/10.1016/j.semcancer.2017.08.004.

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29

Sidhu, Stan, Christine Gicquel, Christopher P. Bambach, Peter Campbell, Christopher Magarey, Bruce G. Robinson, and Leigh W. Delbridge. "Clinical and molecular aspects of adrenocortical tumourigenesis." ANZ Journal of Surgery 73, no. 9 (September 2003): 727–38. http://dx.doi.org/10.1046/j.1445-2197.2003.02746.x.

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30

Biswas, S., D. Holyoake, and T. S. Maughan. "Molecular Taxonomy and Tumourigenesis of Colorectal Cancer." Clinical Oncology 28, no. 2 (February 2016): 73–82. http://dx.doi.org/10.1016/j.clon.2015.11.001.

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31

Lebelo, Maphuti T., Anna M. Joubert, and Michelle H. Visagie. "Warburg effect and its role in tumourigenesis." Archives of Pharmacal Research 42, no. 10 (August 31, 2019): 833–47. http://dx.doi.org/10.1007/s12272-019-01185-2.

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32

Koutsi, Aikaterini, Angeliki Papapanagiotou, and Athanasios G. Papavassiliou. "Thrombomodulin: From haemostasis to inflammation and tumourigenesis." International Journal of Biochemistry & Cell Biology 40, no. 9 (January 2008): 1669–73. http://dx.doi.org/10.1016/j.biocel.2007.06.024.

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33

Jäger, Richard, and Howard O. Fearnhead. "“Dead Cells Talking”: The Silent Form of Cell Death Is Not so Quiet." Biochemistry Research International 2012 (2012): 1–8. http://dx.doi.org/10.1155/2012/453838.

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After more than twenty years of research, the molecular events of apoptotic cell death can be succinctly stated; different pathways, activated by diverse signals, increase the activity of proteases called caspases that rapidly and irreversibly dismantle condemned cell by cleaving specific substrates. In this time the ideas that apoptosis protects us from tumourigenesis and that cancer chemotherapy works by inducing apoptosis also emerged. Currently, apoptosis research is shifting away from the intracellular events within the dying cell to focus on the effect of apoptotic cells on surrounding t
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34

Heiliger, Katrin-Janine, Julia Hess, Donata Vitagliano, Paolo Salerno, Herbert Braselmann, Giuliana Salvatore, Clara Ugolini, et al. "Novel candidate genes of thyroid tumourigenesis identified in Trk-T1 transgenic mice." Endocrine-Related Cancer 19, no. 3 (March 26, 2012): 409–21. http://dx.doi.org/10.1530/erc-11-0387.

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For an identification of novel candidate genes in thyroid tumourigenesis, we have investigated gene copy number changes in aTrk-T1transgenic mouse model of thyroid neoplasia. For this aim, 30 thyroid tumours fromTrk-T1transgenics were investigated by comparative genomic hybridisation. Recurrent gene copy number alterations were identified and genes located in the altered chromosomal regions were analysed by Gene Ontology term enrichment analysis in order to reveal gene functions potentially associated with thyroid tumourigenesis. In thyroid neoplasms fromTrk-T1mice, a recurrent gain on chromos
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35

Michel, E., C. Rohrer Bley, M. P. Kowalewski, S. K. Feldmann, and I. M. Reichler. "Prolactin - to be reconsidered in canine mammary tumourigenesis?" Veterinary and Comparative Oncology 12, no. 2 (June 28, 2012): 93–105. http://dx.doi.org/10.1111/j.1476-5829.2012.00337.x.

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36

Chang, F., S. Syrjänen, A. Tervahauta, and K. Syrjänen. "Tumourigenesis associated with the p53 tumour suppressor gene." British Journal of Cancer 68, no. 4 (October 1993): 653–61. http://dx.doi.org/10.1038/bjc.1993.404.

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37

Loveridge, Carolyn J., Sarah Slater, Kirsteen J. Campbell, Noor A. Nam, John Knight, Imran Ahmad, Ann Hedley, et al. "BRF1 accelerates prostate tumourigenesis and perturbs immune infiltration." Oncogene 39, no. 8 (November 18, 2019): 1797–806. http://dx.doi.org/10.1038/s41388-019-1106-x.

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AbstractBRF1 is a rate-limiting factor for RNA Polymerase III-mediated transcription and is elevated in numerous cancers. Here, we report that elevated levels of BRF1 associate with poor prognosis in human prostate cancer. In vitro studies in human prostate cancer cell lines demonstrated that transient overexpression of BRF1 increased cell proliferation whereas the transient downregulation of BRF1 reduced proliferation and mediated cell cycle arrest. Consistent with our clinical observations, BRF1 overexpression in a Pten-deficient mouse (PtenΔ/ΔBRF1Tg) prostate cancer model accelerated prosta
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38

Gonzalez-Meljem, Jose Mario, John Richard Apps, Helen Christina Fraser, and Juan Pedro Martinez-Barbera. "Paracrine roles of cellular senescence in promoting tumourigenesis." British Journal of Cancer 118, no. 10 (April 19, 2018): 1283–88. http://dx.doi.org/10.1038/s41416-018-0066-1.

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39

Reed, Karen R., Simon J. Tunster, Madeleine Young, Adam Carrico, Rosalind M. John, and Alan R. Clarke. "Entopic overexpression ofAscl2does not accelerate tumourigenesis in ApcMinmice." Gut 61, no. 10 (December 3, 2011): 1435–38. http://dx.doi.org/10.1136/gutjnl-2011-300842.

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40

SATO, Fumiaki, Shunsaku SASAKI, Fumitoshi CHINO, and Daiji ENDOH. "Tumourigenesis by partial body X-irradiation in mice." Japanese Journal of Veterinary Science 50, no. 6 (1988): 1161–68. http://dx.doi.org/10.1292/jvms1939.50.1161.

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41

Muşat, Mădălina, Damian G. Morris, Márta Korbonits, and Ashley B. Grossman. "Cyclins and their related proteins in pituitary tumourigenesis." Molecular and Cellular Endocrinology 326, no. 1-2 (September 15, 2010): 25–29. http://dx.doi.org/10.1016/j.mce.2010.03.017.

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42

Miyazawa, K. "Phosphoinositide 5-phosphatases: how do they affect tumourigenesis?" Journal of Biochemistry 153, no. 1 (September 26, 2012): 1–3. http://dx.doi.org/10.1093/jb/mvs107.

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43

Shirakawa, R., and H. Horiuchi. "Ral GTPases: crucial mediators of exocytosis and tumourigenesis." Journal of Biochemistry 157, no. 5 (March 20, 2015): 285–99. http://dx.doi.org/10.1093/jb/mvv029.

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44

Gardiner, Jennifer R., Yuichi Shima, Ken-ichirou Morohashi, and Amanda Swain. "SF-1 expression during adrenal development and tumourigenesis." Molecular and Cellular Endocrinology 351, no. 1 (March 2012): 12–18. http://dx.doi.org/10.1016/j.mce.2011.10.007.

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45

Angel, Peter. "AP-1-dependent gene expression during skin tumourigenesis." European Journal of Cancer Supplements 4, no. 6 (June 2006): 4. http://dx.doi.org/10.1016/j.ejcsup.2006.04.004.

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46

Ma, Yanlei, Peng Zhang, Feng Wang, Jianjun Yang, Zhe Yang, and Huanlong Qin. "The relationship between early embryo development and tumourigenesis." Journal of Cellular and Molecular Medicine 14, no. 12 (December 2010): 2697–701. http://dx.doi.org/10.1111/j.1582-4934.2010.01191.x.

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47

Diener, Kerrilyn R., Eleanor F. Need, Grant Buchanan та John D. Hayball. "TGF-β signalling and immunity in prostate tumourigenesis". Expert Opinion on Therapeutic Targets 14, № 2 (8 січня 2010): 179–92. http://dx.doi.org/10.1517/14728220903544507.

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48

Myant, Kevin. "COLGENES - Defining novel mechanisms critical for colorectal tumourigenesis." Impact 2017, no. 10 (November 25, 2017): 12–14. http://dx.doi.org/10.21820/23987073.2017.10.12.

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49

Myant, Kevin. "COLGENES - Defining novel mechanisms critical for colorectal tumourigenesis." Impact 2018, no. 7 (October 15, 2018): 74–76. http://dx.doi.org/10.21820/23987073.2018.7.74.

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50

Radford, I. R. "Chromosomal rearrangement as the basis for human tumourigenesis." International Journal of Radiation Biology 80, no. 8 (August 2004): 543–57. http://dx.doi.org/10.1080/09553000412331283489.

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