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Letteratura scientifica selezionata sul tema "Eotaxin"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 28 gennaio 2023

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Articoli di riviste sul tema "Eotaxin"

1

Varlamov, E. E., T. V. Vinogradova, A. A. Chuslyaeva, T. S. Okuneva, and A. N. Pampura. "BIOMARKERS ALLERGIC INFLAMATION AND SEVERITY OF ATOPIC DERMATITIS AT CHILDREN." Russian Journal of Allergy 9, no. 5 (December 15, 2012): 31–35. http://dx.doi.org/10.36691/rja694.

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Background.To establish the relationship between the serum concentrations of IL4, IL5, eotaxin, eotaxin2, ECP and the severity of atopic dermatitis in children. Methods. The study included 40 children with atopic dermatitis, patients were divided into two clusters with KMeans method, index SCORAD was taken as a criterion of clustering. All patients were conducted to determine the concentration of IL4, IL5, eotaxin, eotaxin2, ECP. Results. The SCORAD index was significantly higher in the second cluster in comparison to the first one. In second cluster the level of eotaxin2 was significantly hig

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2

Badewa, A. P., C. E. Hudson, and A. S. Heiman. "Regulatory Effects of Eotaxin, Eotaxin-2, and Eotaxin-3 on Eosinophil Degranulation and Superoxide Anion Generation1." Experimental Biology and Medicine 227, no. 8 (September 2002): 645–51. http://dx.doi.org/10.1177/153537020222700814.

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Eosinophilic leukocytes have been implicated as primary effector cells in inflammatory and allergic diseases. When activated by cytokines, human eosinophils secrete and produce a variety of proinflammatory or tissue damaging substances. Although well known for their chemoattractant effects, little is known about the precise contribution of the eosinophil-selective chemokines, eotaxin, eotaxin-2, and eotaxin-3 to the effector functions of eosinophils. This forms the central focus of these investigations for which clone 15-HL-60 human eosinophilic cells were used as the in vitro model. Investiga

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3

Haley, Kathleen J., Mary E. Sunday, Yolanda Porrata, Colleen Kelley, Anne Twomey, Aliakbar Shahsafaei, Benjamin Galper, Larry A. Sonna, and Craig M. Lilly. "Ontogeny of the eotaxins in human lung." American Journal of Physiology-Lung Cellular and Molecular Physiology 294, no. 2 (February 2008): L214—L224. http://dx.doi.org/10.1152/ajplung.00086.2007.

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The ontogeny of the C-C chemokines eotaxin-1, eotaxin-2, and eotaxin-3 has not been fully elucidated in human lung. We explored a possible role for eotaxin in developing lung by determining the ontogeny of eotaxin-1 (CCL11), eotaxin-2 (CCL24), eotaxin-3 (CCL26), and the eotaxin receptor, CCR3. We tested discarded surgical samples of developing human lung tissue using quantitative RT-PCR (QRT-PCR) and immunostaining for expression of CCL11, CCL24, CCL26, and CCR3. We assessed possible functionality of the eotaxin-CCR3 system by treating lung explant cultures with exogenous CCL11 and analyzing t

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4

Varlamov, E. E., O. G. Elisyutina, T. V. Vinogradova, E. S. Fedenko, and A. N. Pampura. "AGE-RELATED PATHOGENTIC SPECIFICITY OF CYTOKINE PROFILE IN ATOPIC DERMATITIS PATIENTS." Russian Journal of Allergy 13, no. 4-5 (December 15, 2016): 37–42. http://dx.doi.org/10.36691/rja358.

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Background. In this article the results of own research of the cytokine profile in atopic dermatitis (AD) adults and children depending on age are provided. Materials and methods. 88 patients with AD (54 children aged from 1 year to 16 years old and 24 adults aged from 21 years to 56 years old) were included in the study. Cytokine profile of a number of cytokines: IL-4, IL-5, IL-22, IL-31, IL-33, and transforming growth factor TGFβ1, eotaxin, eotaxin2 was examined. Results. Significant differences in IL-22, CC chemokines (eotaxin and eotaxin-2) and TGFβ levels depending on the age and severity

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Diny, Nicola, Xuezhou Hou, Jobert G. Barin, Monica V. Talor, Noel R. Rose, and Daniela Cihakova. "The eotaxin-CCR3 pathway is required for eosinophil trafficking to the heart in eosinophilic myocarditis." Journal of Immunology 196, no. 1_Supplement (May 1, 2016): 117.10. http://dx.doi.org/10.4049/jimmunol.196.supp.117.10.

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Abstract Cardiac manifestations are a major cause of morbidity and mortality in patients with eosinophil-associated diseases. Eosinophils have been proposed to play a pathologic role in the heart. Nevertheless, the pathways that recruit eosinophils to the heart have not been described. In IFNγ−/− IL-17A−/− mice, induction of experimental autoimmune myocarditis results in a Th-2-driven, eosinophilic inflammation. IFNγ−/− IL-17A−/− mice had much higher expression of the eosinophil-attracting chemokines eotaxin-1 (Ccl11) and eotaxin-2 (Ccl24) in the heart than wildtype mice. Genetic ablation of t

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6

Rothenberg, Marc E. "Eotaxin." American Journal of Respiratory Cell and Molecular Biology 21, no. 3 (September 1999): 291–95. http://dx.doi.org/10.1165/ajrcmb.21.3.f160.

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7

Jo, Jung Hyun, Yong-Tae Kim, Ho Soon Choi, Ho Gak Kim, Hong Sik Lee, Young Woo Choi, Dong Uk Kim, et al. "KG 4/2015: A randomized, controlled, multicenter, open-label phase III clinical trial of GV1001 with gemcitabine/capecitabine in previous untreated, eotaxin-high patients with advanced pancreatic ductal adenocarcinoma." Journal of Clinical Oncology 39, no. 15_suppl (May 20, 2021): 4020. http://dx.doi.org/10.1200/jco.2021.39.15_suppl.4020.

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4020 Background: In the TeloVac study, GV1001 with Gemcitabine/capecitabine (G/C) did not show increased overall survival (OS) than G/C in patients (pts) with advanced pancreatic ductal adenocarcinoma (PDA). But cytokine examination suggested high serum eotaxin level may predict improved survivals in pts received GV1001 with G/C. This phase III trial was designed to assess the efficacy of GV1001 with G/C for previous untreated eotaxin-high Korean pts with advanced PDA. Methods: Eligible pts with histologically proven locally advanced and metastatic PDA (except peritoneal carcinomatosis), age &

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Beal, Dominic R., David M. Stepien, Sudha Natarajan, Jiyoun Kim, and Daniel G. Remick. "Reduction of eotaxin production and eosinophil recruitment by pulmonary autologous macrophage transfer in a cockroach allergen-induced asthma model." American Journal of Physiology-Lung Cellular and Molecular Physiology 305, no. 11 (December 1, 2013): L866—L877. http://dx.doi.org/10.1152/ajplung.00120.2013.

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We sought to investigate the effects of cockroach allergen (CRA) exposure on the lung macrophage population to determine how different macrophage phenotypes influence exacerbation of disease. CRA exposure caused significantly reduced expression of CD86 on lung macrophages. These effects were not systemic, as peritoneal macrophage CD86 expression was not altered. To investigate whether naïve macrophages could reduce asthma-like pulmonary inflammation, autologous peritoneal macrophages were instilled into the airways 24 h before the final CRA challenge. Pulmonary inflammation was assessed by mea

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9

Forssmann, Ulf, Mariagrazia Uguccioni, Pius Loetscher, Clemens A. Dahinden, Hanno Langen, Marcus Thelen, and Marco Baggiolini. "Eotaxin-2, a Novel CC Chemokine that Is Selective for the Chemokine Receptor CCR3, and Acts Like Eotaxin on Human Eosinophil and Basophil Leukocytes." Journal of Experimental Medicine 185, no. 12 (June 16, 1997): 2171–76. http://dx.doi.org/10.1084/jem.185.12.2171.

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A novel human CC chemokine consisting of 78 amino acids and having a molecular mass of 8,778.3 daltons (VVIPSPCCMF FVSKRIPENR VVSYQLSSRS TCLKAGVIFT TKKGQQ SCGD PKQEWVQRYM KNLDAKQKKA SPRARAVA) was isolated together with three minor COOH-terminally truncated variants with 73, 75, and 76 residues. The new chemokine was termed eotaxin-2 because it is functionally very similar to eotaxin. In terms of structure, however, eotaxin and eotaxin-2 are rather distant, they share only 39% identical amino acids and differ almost completely in the NH2-terminal region. Eotaxin-2 induced chemotaxis of eosinoph

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10

Struyf, Sofie, Paul Proost, Dominique Schols, Erik De Clercq, Ghislain Opdenakker, Jean-Pierre Lenaerts, Michel Detheux, et al. "CD26/Dipeptidyl-Peptidase IV Down-Regulates the Eosinophil Chemotactic Potency, But Not the Anti-HIV Activity of Human Eotaxin by Affecting Its Interaction with CC Chemokine Receptor 3." Journal of Immunology 162, no. 8 (April 15, 1999): 4903–9. http://dx.doi.org/10.4049/jimmunol.162.8.4903.

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Abstract Chemokines attract and activate distinct sets of leukocytes. The CC chemokine eotaxin has been characterized as an important mediator in allergic reactions because it selectively attracts eosinophils, Th2 lymphocytes, and basophils. Human eotaxin has a penultimate proline, indicating that it might be a substrate for dipeptidyl-peptidase IV (CD26/DPP IV). In this study we demonstrate that eotaxin is efficiently cleaved by CD26/DPP IV and that the NH2-terminal truncation affects its biological activity. CD26/DPP IV-truncated eotaxin(3–74) showed reduced chemotactic activity for eosinoph

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Più fonti

Tesi sul tema "Eotaxin"

1

Pope, Samuel M. "Specific Role of Eotaxin-1 and Eotaxin-2 in Allergic Pulmonary Eosinophilia." Cincinnati, Ohio : University of Cincinnati, 2004. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=ucin1098472372.

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Benarafa, Charaf. "Equine eotaxin : cloning, expression and biological activity." Thesis, Royal Veterinary College (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.394942.

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Watanabe, Kimiko. "Eotaxin -1, -2 and -3 generation by different cell types." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.404880.

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Mariano, Nadia Sabrina. "Estudo da resposta inflamatoria pulmonar alergica em ratos expostos a enterotoxina estafilococica do tipo A (SEA)." [s.n.], 2009. http://repositorio.unicamp.br/jspui/handle/REPOSIP/308954.

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Orientadores: Edson Antunes, Ivani Aparecida de Souza<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas<br>Made available in DSpace on 2018-08-14T02:35:58Z (GMT). No. of bitstreams: 1 Mariano_NadiaSabrina_M.pdf: 621386 bytes, checksum: b01cd4d186fa15a5e8ea3c7657df604a (MD5) Previous issue date: 2009<br>Resumo: O Staphylococcus aureus é um tipo de bactéria gram-positiva que produz e secreta uma série de enterotoxinas com propriedade imunomoduladoras. Entretanto, pouco é conhecido sobre os mecanismos envolvidos na exacerbação do influxo celular observ

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Bodman, Lee Graham. "The effect of glycosylation on the biological activity of eotaxin and related chemokines." Thesis, Imperial College London, 2004. http://hdl.handle.net/10044/1/11889.

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Ghaffar, Omar. "Constitutive and cytokine-stimulated expression of eotaxin by human airway smooth muscle cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape11/PQDD_0024/MQ50776.pdf.

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Menzies-Gow, Andrew Neil. "The roles of interleukin-5 and eotaxin in oesinophil recruitment, maturation and activation." Thesis, Imperial College London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.429161.

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8

Kirchem, Antje. "Investigation of the signalling pathways coupled to the eotaxin receptor CCR3 in human eosinophils." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.406094.

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9

Konikoff, Michael R. "Non-Invasive Biomarkers of Eosinophilic Esophagitis: Blood Eosinophil Level, Eosinophil-Derived Neurotoxin, and Eotaxin-3." Cincinnati, Ohio : University of Cincinnati, 2006. http://www.ohiolink.edu/etd/view.cgi?acc_num=ucin1148043525.

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Thesis (M.S.)--University of Cincinnati, 2006.<br>Advisor: Dr. James E. Heubi. Title from electronic thesis title page (viewed June 3, 2009). Includes abstract. Keywords: Eosinophilic esophagitis; biomarker; eosinophil; eosinophil-derived neurotoxin; eotaxin-3. Includes bibliographical references.

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10

Lieschke, Simone [Verfasser]. "Untersuchung der Rolle von Eotaxin-1 nach ischämischem Schlaganfall im Mausmodell: Neuroprotektion, Neuroregeneration oder destruktiv-proinflammatorische Rolle? / Simone Lieschke." Göttingen : Niedersächsische Staats- und Universitätsbibliothek Göttingen, 2021. http://d-nb.info/1232492825/34.

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Capitoli di libri sul tema "Eotaxin"

1

Conroy, Dolores M., and Timothy J. Williams. "Eotaxin in Disease." In Chemokines in Disease, 123–38. Totowa, NJ: Humana Press, 1999. http://dx.doi.org/10.1007/978-1-59259-706-2_8.

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Williams, T. J., and P. J. Jose. "Role of Eotaxin and Related CC Chemokines in Allergy and Asthma." In Immunological Mechanisms in Asthma and Allergic Diseases, 166–77. Basel: KARGER, 2000. http://dx.doi.org/10.1159/000058825.

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Mitchell, Tracey J., and Timothy J. Williams. "The role of eotaxin and related CC-chemokines in asthma and allergy." In Inflammatory Processes: Molecular Mechanisms and Therapeutic Opportunities, 1–12. Basel: Birkhäuser Basel, 2000. http://dx.doi.org/10.1007/978-3-0348-8468-6_1.

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Williams, Timothy John, and James Edward Pease. "Eotaxins (CCL11, CCL24, CCL26)." In Encyclopedia of Signaling Molecules, 1554–58. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101627.

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Williams, Timothy John, and James Edward Pease. "Eotaxins (CCL11, CCL24, CCL26)." In Encyclopedia of Signaling Molecules, 1–5. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101627-1.

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6

"Eotaxin." In Encyclopedia of Genetics, Genomics, Proteomics and Informatics, 617. Dordrecht: Springer Netherlands, 2008. http://dx.doi.org/10.1007/978-1-4020-6754-9_5409.

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7

"Eotaxin." In Encyclopedia of Immunotoxicology, 298. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-642-54596-2_200451.

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8

Enna, S. J., and David B. Bylund. "Eotaxin." In xPharm: The Comprehensive Pharmacology Reference, 1. Elsevier, 2007. http://dx.doi.org/10.1016/b978-008055232-3.61690-3.

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9

Vaddi, Krishna, Margaret Keller, and Robert C. Newton. "Eotaxin." In The Chemokine FactsBook, 135–37. Elsevier, 1997. http://dx.doi.org/10.1016/b978-012709905-7/50024-5.

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"Eotaxin-1." In Encyclopedia of Signaling Molecules, 1553. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101126.

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Atti di convegni sul tema "Eotaxin"

1

Murphy, GE, NI Pitman, M. Kurowska-Stolarska, P. Kewin, D. Xu, C. McSharry, NC Thomson, and MC Shepherd. "Murine Airway Eosinophilia Following IL-33 Administration Is Eotaxin Dependent." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4253.

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2

Farghly, Saher, Atef Farouk El-Karn, Mahmoud Farouk Sherif, Mohamed Ismail Seddik, Safaa Abdelgayed, and Mohammed Abdelghany. "Serum periostin and eotaxin-2 as promising biomarkers in asthmatic smokers." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa1135.

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3

Lababidi, RR, JL Cane, and M. Bafadhel. "P54 Eosinophil migration is enhanced towards il-5 and eotaxin in copd." In British Thoracic Society Winter Meeting 2017, QEII Centre Broad Sanctuary Westminster London SW1P 3EE, 6 to 8 December 2017, Programme and Abstracts. BMJ Publishing Group Ltd and British Thoracic Society, 2017. http://dx.doi.org/10.1136/thoraxjnl-2017-210983.196.

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4

Gela, Anele, Gopinath Kasetty, Sandra Jovic, Maria Ekoff, Gunnar Nilsson, Sven Kjellstrom, James Pease, and Arne Egesten. "Eotaxin-3 exerts innate host defense activities that are modulated by mast cell proteases." In Annual Congress 2015. European Respiratory Society, 2015. http://dx.doi.org/10.1183/13993003.congress-2015.pa4032.

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Yocum, G. T., J. J. Hwang, and C. W. Emala. "6-shogaol, A Component of Ginger, Inhibits Human Airway Smooth Muscle Eotaxin Expression In Vitro." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4399.

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Lee, AH, SE Wenzel, JB Trudeau, and AT Mustovich. "Increased Eotaxin-2 mRNA in Asthmatic Bronchial Epithelial Cells Ex Vivo: Correlations with Clinical/Inflammatory Parameters." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a5412.

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Smith, Steven G., Haruki Imoaka, Karen J. Howie, Rick Watson, Roma Sehmi, and Gail M. Gauvreau. "The Effects Of Low Dose Peroxisome Proliferator-Activated Receptor (PPAR) Agonists On Eotaxin-Induced Eosinophil Chemotaxis." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4356.

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Couillard, Simon, James Melhorn, Akul Singhania, Daniel Horowitz, Ratko Djukanović, Christpher H. Woelk, and Timothy S. C. Hinks. "Correlation of eotaxin-3 gene expression and other IL-13-induced genes in patients with asthma." In ERS International Congress 2021 abstracts. European Respiratory Society, 2021. http://dx.doi.org/10.1183/13993003.congress-2021.pa825.

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Naik, Chetan, John Trudeau, Sally E. Wenzel, and Amy Lee. "Increased Expression Of Eotaxin-2 In The Airway Epithelium Correlates With Airway Eosinophilia And Severity Of Asthma." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a4443.

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Huang, M., J. Zhang, and Z. Chen. "Exposure to Ambient Particles Amplifies the Eotaxin-1 Release from Alternatively Activated Macrophages in Allergen-Sensitized Mice." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a3797.

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