Bibliografie tematiche / Fetal size/growth

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Letteratura scientifica selezionata sul tema "Fetal size/growth"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 20 febbraio 2023

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Articoli di riviste sul tema "Fetal size/growth"

Harrington, Kevin, and Stuart Campbell. "Fetal size and growth." Current Opinion in Obstetrics and Gynecology 5, no. 2 (April 1993): 186???194. http://dx.doi.org/10.1097/00001703-199304000-00004.

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Kirchengast, Sylvia, and Beda Hartmann. "Association patterns of fetal head dimensions, postcranial body growth and neonatal size." Anthropologischer Anzeiger 77, no. 2 (April 30, 2020): 173–81. http://dx.doi.org/10.1127/anthranz/2020/1137.

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Merialdi, M., L. E. Caulfield, N. Zavaleta, A. Figueroa, K. A. Costigan, F. Dominici, and J. A. Dipietro. "Fetal growth in Peru: comparisons with international fetal size charts and implications for fetal growth assessment." Ultrasound in Obstetrics and Gynecology 26, no. 2 (July 22, 2005): 123–28. http://dx.doi.org/10.1002/uog.1954.

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Spencer, J. A. D., T. C. Chang, S. C. Robson, and S. Gallivan. "Fetal size and growth in Bangladeshi pregnancies." Ultrasound in Obstetrics and Gynecology 5, no. 5 (May 1, 1995): 313–17. http://dx.doi.org/10.1046/j.1469-0705.1995.05050313.x.

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Frusca, T., S. Parolini, A. Dall'Asta, W. A. Hassan, A. Vitulo, A. Gillett, D. Pasupathy, and C. C. Lees. "Fetal size and growth velocity in chronic hypertension." Pregnancy Hypertension 10 (October 2017): 101–6. http://dx.doi.org/10.1016/j.preghy.2017.06.007.

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McCarthy, Elizabeth A., and Susan P. Walker. "International fetal growth standards: one size fits all." Lancet 384, no. 9946 (September 2014): 835–36. http://dx.doi.org/10.1016/s0140-6736(14)61416-1.

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Friedrich, M. J. "International Standards for Newborn Size and Fetal Growth." JAMA 312, no. 15 (October 15, 2014): 1503. http://dx.doi.org/10.1001/jama.2014.13252.

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Keshavarz, Elham, Marjan Rustazade Sheikhyusefi, Ensi Khalili Pouya, Masoumeh Mirzamoradi, Mehdi Khazaei, Yashar Moharamzad, and Morteza Sanei Taheri. "Association Between Fetal Thymus Size and Intrauterine Growth Restriction." Journal of Diagnostic Medical Sonography 38, no. 2 (December 14, 2021): 120–26. http://dx.doi.org/10.1177/87564793211054747.

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Objective: The objective of this study was to evaluate the association between reduced fetal thymus size and intrauterine growth restriction (IUGR). This study was devised to determine the association between thymus size and any abnormal Doppler indices within the fetal umbilical artery (UA), as well as the middle cerebral artery (MCA). Materials and Methods: Forty-six pregnancies between 20 and 38 weeks of gestation with IUGR and 46 normal pregnancies within similar gestational age (GA) range were included. The transverse diameter of fetal thymus was measured. In the IUGR group, the fetal umb

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Fulford, A. J. C., S. E. Moore, S. E. Arifeen, L. Å. Persson, L. M. Neufeld, Y. Wagatsuma, and A. M. Prentice. "Disproportionate early fetal growth predicts postnatal thymic size in humans." Journal of Developmental Origins of Health and Disease 4, no. 3 (March 7, 2013): 223–31. http://dx.doi.org/10.1017/s2040174413000044.

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Prenatal events can affect neonatal thymus size and adult immune function. The causal insults are unknown, although fetal nutrient restriction is suspected. We used ultrasound at three time points during pregnancy (14, 19 and 30 weeks) to measure the growth of six fetal dimensions in rural Bangladeshi women participating in the Maternal and Infant Nutrition Interventions, Matlab study. Postnatal ultrasound was used to calculate thymic index (TI) at birth, 2, 6 and 12 m. Of the 3267 women recruited, 2861 participated by providing data at least at one fetal biometry and one TI time point. Patter

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Schwartz, Nadav, Mary Sammel, Hayley Quant, Rita Leite, and Samuel Parry. "379: Early placental size helps predict fetal growth restriction." American Journal of Obstetrics and Gynecology 208, no. 1 (January 2013): S166—S167. http://dx.doi.org/10.1016/j.ajog.2012.10.544.

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Più fonti

Tesi sul tema "Fetal size/growth"

Westerway, Susan Lyn Campbell. "Ultrasonic assessment of fetal size and growth." University of Sydney, 2006. http://hdl.handle.net/2123/2626.

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Doctor of Philosophy (Medicine) This work investigates a number of issues. Firstly it examines ultrasonic fetal biometry, the parameters and techniques for accurate measuring and reviews the procedure adopted for graph formation and application of regression analysis for a mathematical model to describe the relationship between fetal size and weeks of gestation. Next it establishes new Australian fetal measurement charts for the crown rump length, head circumference and abdominal circumference, based on an Australian population, to replace the charts currently in use that are over 20 year

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Westerway, Susan Lyn Campbell. "Ultrasonic assessment of fetal size and growth." Thesis, The University of Sydney, 2005. http://hdl.handle.net/2123/2626.

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This work investigates a number of issues. Firstly it examines ultrasonic fetal biometry, the parameters and techniques for accurate measuring and reviews the procedure adopted for graph formation and application of regression analysis for a mathematical model to describe the relationship between fetal size and weeks of gestation. Next it establishes new Australian fetal measurement charts for the crown rump length, head circumference and abdominal circumference, based on an Australian population, to replace the charts currently in use that are over 20 years old and relate to middle class wh

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Baird, Janis. "Birth size, blood pressure and glucose tolerance in twins : testing the fetal origins hypothesis." Thesis, University of Southampton, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.341624.

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Vaughn, Mathew Alan. "Characterization of intra-litter variation on myogenic development and myogenic progenitor cell response to growth promoting stimuli." Diss., Kansas State University, 2016. http://hdl.handle.net/2097/34595.

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Doctor of Philosophy Department of Animal Sciences and Industry John M. Gonzalez This series of studies focuses on the impact of intra-litter variation on fetal myogenesis, and the ability of porcine progenitor cells to respond to growth promoting stimuli. In study 1, the smallest (SM), median (ME), and largest (LG) male fetuses from each litter were selected for muscle morphometric analysis from gilts at d-60 ± 2 and 95 ± 2 of gestation. On d-60 and 95 of gestation LG fetuses had greater whole muscle cross-sectional area (CSA) than ME and SM fetuses, and ME fetuses had greater whole

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Bennini, Junior João Renato 1978. "Estimativa do peso do recem-nascido por meio de medidas ultrassonograficas bidimensionais e do volume da coxa fetal." [s.n.], 2009. http://repositorio.unicamp.br/jspui/handle/REPOSIP/313463.

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Orientadores: Cleisson Fabio Andrioli Peralta, Ricardo Barini Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas Made available in DSpace on 2018-11-27T11:44:05Z (GMT). No. of bitstreams: 1 BenniniJunior_JoaoRenato_M.pdf: 1809698 bytes, checksum: 399e6ab502353af527e35953428d5e09 (MD5) Previous issue date: 2009 Resumo: Introdução: Alguns estudos demonstram que a predição do peso fetal usando a volumetria dos membros fetais é mais precisa do que quando se usam medidas bidimensionais (2D). Até hoje, somente o método multiplanar foi utilizado para

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Dreyer, Carlien. "Fruit set and fruit size studies on ‘Forelle’ and ‘Abate Fetel’ pear (Pyrus communis L.)." Thesis, Stellenbosch : Stellenbosch University, 2013. http://hdl.handle.net/10019.1/79870.

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Thesis (MScAgric)--Stellenbosch University, 2013. ENGLISH ABSTRACT: Maintaining constant high yields in „Abate Fetel‟ and „Forelle‟ orchards in South Africa is challenging. Improving productivity in these orchards could be achieved by increasing fruit set and fruit size. Fruit size is an important marketing and quality parameter and has a significant effect on the economic value of fruit. Various protocols to improve fruit set are used by South African producers but these are not well researched. We therefore evaluated different combinations of plant growth regulators including gibberellic

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Hewitt, Damien Phillip. "Impact of glucocorticoids on placental growth and vascularisation." University of Western Australia. School of Anatomy and Human Biology, 2007. http://theses.library.uwa.edu.au/adt-WU2007.0195.

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[Truncated abstract] Glucocorticoids are critical for the maturation of the fetus late in pregnancy. Indeed, clinical administration of glucocorticoids is used to accelerate fetal lung maturation in mothers at risk of pre-term delivery. Increased glucocorticoid exposure, however, can have detrimental effects on fetal and placental growth and increase the risk of disease in later life. Many studies have focused on the effect of an increase in the transplacental passage of glucocorticoids on both fetal growth and subsequent postnatal development. But there is a growing body of evidence to sugges

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Mariet, Anne-Sophie. "Influence de l’exposition au bruit et à la pollution de l’air en milieu urbain sur la survenue de complications et d’issues défavorables de la grossesse." Thesis, Bourgogne Franche-Comté, 2020. https://nuxeo.u-bourgogne.fr/nuxeo/site/esupversions/b8bd098d-4e78-4f44-b87c-01a65906c7a1.

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La grossesse constitue une période de vulnérabilité où la survenue de complications et d’issues défavorables de la grossesse (CIDG) peut avoir des conséquences majeures sur le devenir de la mère et/ou du nouveau-né. De multiples facteurs en sont à l’origine. Cependant, il reste encore une part non expliquée de CIDG, pour laquelle l’environnement est suspecté de jouer un rôle.Ce travail de thèse est inclus dans le programme PreCEE (Pregnancy Combined Environmental Exposure) et a pour objectif d’étudier l’influence des expositions environnementales aux pollutions sonore et atmosphérique sur la s

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Cavallin, Mara. "Physiopathologie moléculaire et cellulaire des anomalies du développement du cortex cérébral : le syndrome d'Aicardi WDR81 mutations cause extreme microcephaly and impair mitotic progression in human fibroblasts and Drosophila neural stem cells TLE1, a key player in neurogenesis, a new candidate gene for autosomal recessive postnatal microcephaly Mutations in TBR1 gene leads to cortical malformations and intellectual disability Aicardi syndrome: Exome, genome and RNA-sequencing of a large cohort of 19 patients failed to detect the genetic cause Recurrent RTTN mutation leading to severe microcephaly, polymicrogyria and growth restriction Recurrent KIF2A mutations are responsible for classic lissencephaly Recurrent KIF5C mutation leading to frontal pachygyria without microcephaly Rare ACTG1 variants in fetal microlissencephaly De novo TUBB2B mutation causes fetal akinesia deformation sequence with microlissencephaly: An unusual presentation of tubulinopathy A novel recurrent LIS1 splice site mutation in classic lissencephaly Further refinement of COL4A1 and COL4A2 related cortical malformations Prenatal and postnatal presentations of corpus callosum agenesis with polymicrogyria caused By EGP5 mutation Delineating FOXG1 syndrome from congenital microcephaly to hyperkinetic encephalopathy Delineating FOXG1 syndrome: From congenital microcephaly to hyperkinetic encephalopathy." Thesis, Sorbonne Paris Cité, 2019. https://wo.app.u-paris.fr/cgi-bin/WebObjects/TheseWeb.woa/wa/show?t=2213&f=18201.

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Les malformations du cortex cérébral (MDC) représentent une cause importante de handicap et d'épilepsie pharmaco-résistante. Le séquençage à haut débit a permis une amélioration considérable de l'identification des bases moléculaires des MDC non syndromiques. Toutefois, certaines formes, notamment les MDC complexes, demeurent inexpliquées. Mon projet de thèse a pour objectif de progresser dans la compréhension des MDC complexes en utilisant deux modèles : les microlissencéphalies (MLIS) et le syndrome d'Aicardi (AIC), une forme syndromique particulière associant des malformations de l'oeil et

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Libri sul tema "Fetal size/growth"

Wilton, Niall, Brian J. Anderson, and Bruno Marciniak. Anatomy, physiology, and pharmacology in paediatric anaesthesia. Edited by Jonathan G. Hardman and Neil S. Morton. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199642045.003.0069.

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Anaesthesia for children is tempered by changes that occur during both growth and development. Drug dose is affected by size and clearance maturation processes as well as the changing body composition that occurs with age. All organ systems undergo these maturation changes and most are complete within the first few years of life. Normal physiological variables in infancy and childhood are quite different from adults. The central nervous, cardiovascular, and respiratory systems are particularly important. Cerebral immaturity and plasticity impacts sensitivity to drugs, pain responses, and behav

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S, Zagon Ian, and Slotkin Theodore A, eds. Maternal substance abuse and the developing nervous system. San Diego: Academic Press, 1992.

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Zagon, Ian S., and Theodore A. Slotkin. Maternal Substance Abuse and the Developing Nervous System. Elsevier Science & Technology Books, 2012.

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Capitoli di libri sul tema "Fetal size/growth"

Snow, M. H. L. "Effect of genome on size at birth." In Fetal Growth, 3–12. London: Springer London, 1989. http://dx.doi.org/10.1007/978-1-4471-1707-0_1.

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Snow, Michael H. L. "Control of Embryonic Growth Rate and Fetal Size in Mammals." In Human Growth, 67–82. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4613-2101-9_4.

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Hargitai, Beata. "Diagnostic Criteria of Fetal Growth Abnormalities and Interpretation of Postmortem Size and Weight Measurements." In Practical Manual of Fetal Pathology, 69–75. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-42492-3_6.

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Ulrich, Magda M. W. "Fetal Wound Healing." In Textbook on Scar Management, 3–9. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_1.

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AbstractFirst- and second-trimester fetal skin wounds are known to heal without scarring.Research has excluded factors like the sterile uterine environment as the cause of scarless repair, and it is believed that scarless healing is an intrinsic property of early fetal skin. However, increasing wound size and induction of the inflammatory reaction can evoke a scar response in the fetus.For decades, research is performed to elucidate the mechanisms responsible for scarless healing in fetuses. Much research has been performed in animal studies, and several mechanisms have been proposed to be involved such as the microenvironment and the extracellular matrix, a reduced inflammatory response, differences in growth factor profile, and differences in fibroblast phenotype.It is clear that the wound healing process leading to scarless healing cannot be attributed to just one factor or mechanism but will be the result of a complex of interconnected processes.This chapter describes some of the possible mechanisms which may play a role in scarless healing.

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Hirst, Jane E., and Aris T. Papageorghiou. "Fetal growth." In Oxford Textbook of Obstetrics and Gynaecology, edited by Sabaratnam Arulkumaran, William Ledger, Lynette Denny, and Stergios Doumouchtsis, 133–40. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198766360.003.0010.

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Fetal growth is a complex and highly orchestrated process. Monitoring fetal growth is one of the cornerstones of the evaluation of fetal well-being. This chapter explores the global epidemiology of poor fetal growth, the controversies around the definition of the condition, and the scientific validity of the multitude of growth charts and approaches to fetal growth currently in use around the world. In 2014, the accepted approaches to fetal growth were challenged by the findings of the international INTERGROWTH-21st Project. This major international project demonstrated that the growth of babies in utero and the size of babies around the world are remarkably similar if maternal environmental, social, and medical conditions are relatively optimal. From this study, the first standards for fetal and newborn growth have been produced. The management of babies detected to be large for gestational age (LGA) also remains controversial and a clinical challenge. With increasing rates of maternal obesity and gestational diabetes, a growing proportion of babies are born LGA or detected to be LGA in utero. Other than the treatment for gestational diabetes, the evidence for effective interventions to prevent the development of LGA during pregnancy is limited. Key issues regarding the management of LGA include the most appropriate timing and mode of delivery to prevent shoulder dystocia and birth trauma.

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Dutta, Dilip. "How to Assess Fetal Size and Growth Rate Abnormalities?" In Manual of Fetal Medicine, 33. Jaypee Brothers Medical Publishers (P) Ltd., 2009. http://dx.doi.org/10.5005/jp/books/10466_4.

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Snow, Michael H. L. "Embryonic growth and the manipulation of fetal size." In The Physiology of Human Growth, 1–10. Cambridge University Press, 1989. http://dx.doi.org/10.1017/cbo9780511896811.002.

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Enkin, Murray, Marc J. N. C. Keirse, James Neilson, Caroline Crowther, Lelia Duley, Ellen Hodnett, and Justus Hofmeyr. "Assessment of fetal growth, size, and well-being." In Guide to Effective Care in Pregnancy and Childbirth, 79–92. Oxford University Press, 2000. http://dx.doi.org/10.1093/med/9780192631732.003.0012.

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Butler, Gary. "Child and adolescent growth." In Oxford Textbook of Endocrinology and Diabetes, 989–1006. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199235292.003.7005.

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The growth of a human being from a single cell to a fully mature individual is a remarkable process and something that is subject to a large number of influences across the whole growth period. Growth before birth is actually the most rapid and probably the least understood phase, but a detailed description of antenatal events is beyond this chapter. Size at birth, however, is dependent on a number of factors, primarily maternal, in particular the wellbeing of the fetoplacental unit and its level of functioning. This unit is markedly affected in maternal undernutrition, which translates into significant deleterious effects on fetal growth. Probably as important as placental function is maternal size. Small maternal size will constrain growth even when the fetus is potentially of a genetically large size. Lastly, fetal factors are important themselves. Genetic or endocrine disturbances may constrain fetal growth, but these are secondary to maternal effects. In paediatric practice we are concerned about postnatal growth. It is useful to think of growth in three separate phases: infancy, childhood, and puberty (1). The infancy phase is largely nutrition dependent and lasts for 1–2 years. After this, the childhood phase, which is predominantly growth hormone-driven takes over, and continues until the pubertal or adolescent phase. This final phase is under the influence of the sex steroids and the speed of this phase determines the timing and rate of acceleration of the pubertal growth spurt, and the cessation of growth. It is very helpful to consider the different influences on each phase when presented with the diagnostic challenge of a child with abnormal growth (2).

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Maršál, Karel, and Bertil Sundén. "The development of ultrasound in obstetrics and gynaecology in Sweden." In Ultrasound in Clinical Diagnosis. Oxford University Press, 2011. http://dx.doi.org/10.1093/oso/9780199602070.003.0011.

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In the field of obstetrics, the advent of diagnostic ultrasound was most welcome because of the obvious lack of a non-invasive method providing information on the fetus in utero. The subsequent very fast and widespread use of ultrasound in clinical obstetrics was vindication that the method fulfilled the expectations and that it literally ‘opened a window into the uterus’. Ultrasound enabled direct examinations of fetal anatomy, measurements of fetal size and growth, and recording of intrauterine activities. Nowadays, 97 % of all pregnant women in Sweden undergo at least one ultrasound examination during their pregnancy. The early positive results reported from the application of ultrasound in cardiology and neurosurgery at Lund University elicited interest to test the method on pregnant women at the Department of Obstetrics and Gynecology in Lund. In 1957, Alf Sjövall, then professor in obstetrics and gynaecology, discussed over a lunch-table with neurosurgeon Lars Leksell his very first experience of diagnosing subdural hematoma using ultrasound. Professor Sjövall asked then Bertil Sundén, who worked at his department, to investigate early pregnancies with the Krautkrämer echoscope belonging to Leksell. The aim was to examine whether it would be possible to detect echoes from the fetus in early pregnancies and to differentiate it from myomatous enlargements of the uterus and from ovarian tumours. The Krautkrämer echoscope offered only an A-mode display of ultrasound signals so no tangible results were obtained as the origin of the echoes could not be identified. At that time, it was unknown whether or not ultrasound might have any harmful effects on embryonic tissue and therefore these first investigations in early pregnancies were done on patients admitted for interruption of pregnancy. After that Ian Donald published the first description of an echoscope generating a two dimensional display in 1958, Bertil Sundén went on a three-week visit to Professor Donald in Glasgow. There he met electronic engineer Tom G. Brown, employed by Smiths Industrial Division in Glasgow, who had built Donald’s machine. During his stay in Glasgow Sundén performed several investigations on obstetric and gynaecological patients using Brown’s equipment that was the only one of its type.

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Atti di convegni sul tema "Fetal size/growth"

Yalcin, Huseyin C., Huseyin E. Salman, and Reema Y. Kamal. "Assessment of Human Fetal Left Heart Hemodynamics during Prenatal Development." In Qatar University Annual Research Forum & Exhibition. Qatar University Press, 2021. http://dx.doi.org/10.29117/quarfe.2021.0086.

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The hemodynamic forces and wall shear stresses (WSS) play an important role during the fetal heart development. Abnormal levels of flow-driven shear stress can deteriorate the proper functioning of the cells responsible for the growth and remodeling of the heart and lead to congenital heart defects (CHDs). Hypoplastic left heart syndrome (HLHS) is a critical CHD with severely underdeveloped left ventricle and responsible for 25-40% of all neonatal cardiac deaths. To characterize the main differences between the healthy and HLHS fetal hearts in terms of morphology, flow behavior, and WSS levels

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Wang, H. T., C. J. Li, G. C. Ji, and G. J. Yang. "Influence of Annealing Treatment on the Microstructure and Microhardness of Cold Sprayed Nanostructured FeAl Coating." In ITSC2011, edited by B. R. Marple, A. Agarwal, M. M. Hyland, Y. C. Lau, C. J. Li, R. S. Lima, and A. McDonald. DVS Media GmbH, 2011. http://dx.doi.org/10.31399/asm.cp.itsc2011p1049.

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Abstract In the present study, a nanostructured FeAl coating was prepared by cold spraying of ball milled powder. Annealing treatment was applied to the coating to investigate its effect on the phase structure, grain size and microhardness of the cold-sprayed nanostructured FeAl coating. The results showed that the FeAl phase was kept unchangeable when the coating annealed at the temperature above 500°C. Annealing temperature significantly influenced the microstructure and microhardness of cold-sprayed FeAl coating. With raising annealing temperature, the lamellar structure in the as-sprayed c

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Rapporti di organizzazioni sul tema "Fetal size/growth"

Spencer, Thomas E., Elisha Gootwine, Arieh Gertler, and Fuller W. Bazer. Placental lactogen enhances production efficiency in sheep. United States Department of Agriculture, December 2005. http://dx.doi.org/10.32747/2005.7586543.bard.

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The key objectives of this BARD project were to: (1) study long-term effects of immunization of prepubertal ewes against recombinant ovine placental lactogen (roPL) on subsequent birth weights of their lambs and their milk production; (2) optimize the anti-roPL immunization protocol using adjuvant preparations acceptable to producers and regulatory agencies; and (3) determine the physiological mechanism(s) whereby immunization against oPL increases fetal growth and development and mammogenesis. These objectives were based on key findings from a previous BARD project that: (a) immunization of e

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