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Letteratura scientifica selezionata sul tema "IL-4"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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Articoli di riviste sul tema "IL-4"

1

Usha, Prof. "IL-4 and IL-6 in Bronchial Asthma Does IL-6 Plays More Important Role than IL-4? A Preliminary Study." Journal of Medical Science And clinical Research 05, no. 04 (2017): 20446–50. http://dx.doi.org/10.18535/jmscr/v5i4.115.

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2

Kelly-Welch, A., E. M. Hanson, and A. D. Keegan. "Interleukin-4 (IL-4) Pathway." Science Signaling 2005, no. 293 (2005): cm9. http://dx.doi.org/10.1126/stke.2932005cm9.

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3

Borish, Larry. "IL-4 and IL-13 Dual Antagonism." American Journal of Respiratory and Critical Care Medicine 181, no. 8 (2010): 769–70. http://dx.doi.org/10.1164/rccm.201002-0147ed.

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4

Otani, Yusuke, and Takumi Takizawa. "Anti-IL-4/IL-13 antibody (Dupilumab)." Nihon Shoni Arerugi Gakkaishi. The Japanese Journal of Pediatric Allergy and Clinical Immunology 37, no. 3 (2023): 240–47. http://dx.doi.org/10.3388/jspaci.37.240.

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5

Schindler, C., H. Kashleva, A. Pernis, R. Pine, and P. Rothman. "STF-IL-4: a novel IL-4-induced signal transducing factor." EMBO Journal 13, no. 6 (1994): 1350–56. http://dx.doi.org/10.1002/j.1460-2075.1994.tb06388.x.

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6

Gause, W. C., T. Takashi, J. D. Mountz, F. D. Finkelman, and A. D. Steinberg. "Activation of CD 4-, CD 8- thymocytes with IL 4 vs IL 1 + IL 2." Journal of Immunology 141, no. 7 (1988): 2240–45. http://dx.doi.org/10.4049/jimmunol.141.7.2240.

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Abstract Thymocytes from C57BL/6 mice were highly purified to obtain the CD 4-, CD 8- subpopulation which constitutes only 5% of all thymocytes. Substantial proliferation was induced in vitro with either IL-1 + IL-2 or with IL-4 in the presence of PMA. IL-1 and IL-2 synergized in inducing proliferation of these purified CD 4-, CD 8- thymocytes whereas neither synergized with IL-4. In order to determine whether stimulation with IL-1 + IL-2 acted via IL-4 or vice versa, cultures were treated reciprocally with affinity-purified anti-IL-2 or anti-IL-4 antibodies. Cultures with IL-4 were inhibited

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7

Ho, S. N., R. T. Abraham, A. Nilson, B. S. Handwerger, and D. J. McKean. "Interleukin 1-mediated activation of interleukin 4 (IL 4)-producing T lymphocytes. Proliferation by IL 4-dependent and IL 4-independent mechanisms." Journal of Immunology 139, no. 5 (1987): 1532–40. http://dx.doi.org/10.4049/jimmunol.139.5.1532.

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Abstract The role of IL 1 in the activation of IL 4-producing murine T cell clones was investigated by using a calcium ionophore (ionomycin) or a phorbol ester (12-O-tetradecanoylphorbol 13-acetate; TPA) as T cell receptor-independent costimuli. The use of these pharmacologic agents to investigate IL 1-mediated T cell activation revealed two distinct mechanisms of activation. IL 1 in combination with ionomycin (iono/rIL 1) stimulated a proliferative response that was associated with the production of IL 4 as measured by lymphokine bioassay and mRNA studies. Furthermore, inhibition of this prol

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8

Atamas, S. P., J. Choi, V. V. Yurovsky, and B. White. "An alternative splice variant of human IL-4, IL-4 delta 2, inhibits IL-4-stimulated T cell proliferation." Journal of Immunology 156, no. 2 (1996): 435–41. http://dx.doi.org/10.4049/jimmunol.156.2.435.

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Abstract Alternative splicing of mRNA can generate protein isoforms that are preferentially expressed in different tissues or during different states of cell differentiation or activation. Protein isoforms may have different functions. In this study, we cloned, expressed, and tested functional effects of a naturally occurring splice variant of human IL-4, called IL-4 delta 2. In IL-4 delta 2, the second exon of IL-4 is omitted by alternative splicing, with exons 1, 3, and 4 joined in an open reading frame. We found that IL-4 delta 2 RNA is expressed in the PBMC of all donors tested, usually in

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9

NAKANISHI, KENJI. "Interleukin cascade of IL-4,IL-5 and IL-2." Japanese Journal of Clinical Immunology 13, no. 5 (1990): 438–40. http://dx.doi.org/10.2177/jsci.13.438.

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10

Noben-Trauth, N., L. D. Shultz, F. Brombacher, J. F. Urban, H. Gu, and W. E. Paul. "An interleukin 4 (IL-4)-independent pathway for CD4+ T cell IL-4 production is revealed in IL-4 receptor-deficient mice." Proceedings of the National Academy of Sciences 94, no. 20 (1997): 10838–43. http://dx.doi.org/10.1073/pnas.94.20.10838.

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Tesi sul tema "IL-4"

1

Dawson, Charlotte Helen. "STAT 6 and IL-4 signalling." Thesis, University of Sheffield, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.245716.

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2

Nieuwenhuizen, Natalie. "Immune and allergic responses to Anisakis pegreffii, with focus on the roles of IL-4, IL-13 and the IL-4 receptor alpha." Doctoral thesis, University of Cape Town, 2007. http://hdl.handle.net/11427/3115.

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Includes bibliographical references (p. 117-127).<br>The fish-parasitizing nematode Anisakis pegreffii induces gastrointestinal disease and allergy when ingested by humans, and can cause occupational allergy in seafood processing workers. The present study examines immune and allergic responses to A. pegreffii in wildtype and gene deficient mice, with special focus on interleukin(IL)-4, IL-13, and the IL-4 receptor alpha (IL-4Rα). Experimental murine models of Anisakis infection, Anisakis-induced anaphylaxis and Anisakis-induced dermatitis were established in order to gain insight into the imm

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3

Oksuz, Samet. "Targeting IL-4 locus for epigenetic reprogramming." University of Cincinnati / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1423581203.

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4

Govender, Melissa. "Investigating the role of IL-4/IL-13 signalling through the IL-4 receptor alpha (IL-4Rα) on keratinocytes in murine models of Leishmania major and Schistosoma mansoni". Doctoral thesis, University of Cape Town, 2017. http://hdl.handle.net/11427/24890.

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Keratinocytes represent the major cell type in the skin. During cutaneous leishmaniasis (CL) and schistosomiasis, the skin is important during the parasite life cycle. While Th1 immunity is required to control CL, protection during schistosomiasis requires Th2 immunity. Paradoxically, Th2 characteristic IL-4 secreted early during L. major infection in mice, can drive a Th1 response by instructing dendritic cells to produce IL-12. Additionally, keratinocytes at the site of L. major infection in C57BL/6 mice, were postulated to be the source of the IL-4. We investigated if IL-4/IL-13 signalling

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5

Мозгова, Юлія Анатоліївна, Юлия Анатольевна Мозговая та Yuliia Anatoliivna Mozghova. "Динаміка вмісту IL-4 та IL-6 при хронічному тонзиліті в дітей". Thesis, Сумський державний університет, 2013. http://essuir.sumdu.edu.ua/handle/123456789/32359.

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Хронічне запалення піднебінних мигдаликів є розповсюдженим захворюванням дитячого віку, що несприятливо впливає на зростаючий організм. На хронічний тонзиліт страждає більше 2-3% дітей раннього віку, 6-7% - дошкільного та 8-9% школярів. При цитуванні документа, використовуйте посилання http://essuir.sumdu.edu.ua/handle/123456789/32359

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Pizzuti, Lucas. "Síntese de 4-(fur-2-il)- e 4-(tien-2-il)-pirimidinas a partir de β-Alcoxivinil trifluormetil cetonas". Universidade Federal de Santa Maria, 2005. http://repositorio.ufsm.br/handle/1/10401.

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Conselho Nacional de Desenvolvimento Científico e Tecnológico<br>The cyclocondensation of the 1,1,1-trifluoro-4-methoxy-4-(2-heteroaryl)-3-buten-2-ones (heteroaryl = furyl and thienyl) with urea and amidines (acetamidine, benzamidine, guanidine, 1H-pyrazole-1-carboxamidine and 2-methyl-2-thiopseudourea) for synthesis of two 4-(2-heteroaryl)-6-trifluoromethylpyrimidinones and a series of ten 4-(2-heteroaryl)-6-trifluoromethylpyrimidines is reported. The reaction of the 1,1,1-trifluoro-4-methoxy-4-(2-heteroaryl)-3-buten-2-ones with urea was carried out in the presence of boron trifluoride ethera

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7

Cirocco, Robert E. "Cytokine analisys in atlantic bottlenose dolphins: molecular characterization of IL-4, IL-6, and IL-10." FIU Digital Commons, 2001. http://digitalcommons.fiu.edu/etd/2370.

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The health status of wild and captive Atlantic Bottlenose dolphins (Tersiops truncatis) is difficult to ascertain. Mass strandings of these animals have been attributed to pollutants, as well as bacterial infections. Using human Enzyme Linked Immuno-Assays (ELISA) for immunological cytokines, I measured soluble cytokine levels with respect to their health status. In a retrospective analysis of dolphin sera, there was a trend of higher cytokine levels in “sick” animals. I cultured dolphin lymphocytes in the presence of a mitogen (PHA), a super antigen (Staph-A), Lipopolysaccharide (LPS), and a

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Vale, Mariana Lima. "Atividade analgesica das interleucinas 4, 10 e 13 (IL-4, IL-10 e il-13) na dor inflamatoria experimental : papel de celulas residentes e citocinas." reponame:Repositório Institucional da UFC, 2000. http://www.repositorio.ufc.br/handle/riufc/2569.

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VALE, Mariana Lima. Atividade analgesica das interleucinas 4, 10 e 13 (IL-4, IL-10 e il-13) na dor inflamatoria experimental : papel de celulas residentes e citocinas. 2000. 140 f. Dissertação (Mestrado em Farmacologia) - Universidade Federal do Ceará. Faculdade de Medicina, 2000.<br>Submitted by denise santos (denise.santos@ufc.br) on 2012-05-04T12:19:28Z No. of bitstreams: 1 2000_dis_mlvale.pdf: 664941 bytes, checksum: a8df00b6a53f2d6632ebe816657800aa (MD5)<br>Approved for entry into archive by Eliene Nascimento(elienegvn@hotmail.com) on 2012-05-04T12:36:24Z (GMT) No. of bitstreams: 1 2000_

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9

Toor, Iqbal Singh. "Investigating the role of eosinophils in cardiac remodelling following myocardial infarction." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/31187.

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Myocardial infarction (MI) occurs following acute thrombotic occlusion of a coronary artery, and triggers a robust inflammatory response. Within hours, neutrophils are recruited to the infarcted myocardium followed by the infiltration of pro-inflammatory Ly6Chi monocytes. Transition from the pro-inflammatory macrophage phenotype (M1) to an anti-inflammatory, pro-resolution phenotype (M2-like) is critical to successful infarct healing. Interventions that polarize macrophages towards an anti-inflammatory 'M2-like' phenotype improve infarct healing in the experimental MI mouse model and reduce su

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10

Dheda, Keertan Unkha Jairam. "The expression and role of IL-4 and IL-4(delta)2 in tuberculosis with and without HIV co-infection." Thesis, University College London (University of London), 2006. http://discovery.ucl.ac.uk/1445411/.

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Background: Tuberculosis progresses despite potent Thl responses. A putative explanation is the presence of a subversive Th2 response. However interpretation is confounded by a novel cytokine, IL-452, a splice variant and inhibitor of IL-4. Methods: The expression of Thl cytokines (IFN-y), IL-452, Th2 cytokines (IL-4) and sCD30 was investigated in whole blood, lung lavage and mononuclear cell cultures from donors with TB, TB-HIV co-infection, and matched controls. Results: After validation of a fluorogenic real-time RT-PCR assay, the half-life of IL- 4 mRNA, but not IL-452, was found to be pro

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Libri sul tema "IL-4"

1

Mutin, U. G. Lichnai︠a︡ voĭna mekhanika Il-4. Rosa, 2012.

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2

1948-, Spits Hergen, ed. IL-4: Structure and function. CRC Press, 1992.

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3

Kotelnikov, Vladimir. Il-4: "vozdushnye kreĭsera" Stalina. I︠A︡uza, 2009.

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4

Lazara, Silvia Ferri de, Giulia D'Amaro Valle, and Enoch Battagin. Cos'è il contemporaneo? 4: What is the contemporary? 4. CLEUP, 2012.

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5

Giuseppe, Verdi. Il trovatore: Dramma in 4 parti. Gremese Editore, 1988.

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Giuseppe, Verdi. Il Trovatore: Dramma in 4 parti. Gremese Editore, 1988.

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7

Trevisi, Giuseppe. Il delitto Fanin: 4 novembre 1948. Il Mulino, 1998.

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8

Natacha, Godeau, ed. Shrek 4: Il était une fin. Hachette jeunesse, 2010.

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9

Chang, Il-hong. Sanyuhwa: Chang Il-hong 4·3 changp'yŏn sosŏl. Wŏlin, 2017.

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Hua, Yu. 4-wŏl 3-il sakŏn: Wi Hwa sosŏl. Munhak Tongne, 2010.

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Capitoli di libri sul tema "IL-4"

1

Ranasinghe, Charani, Sreeja Roy, Zheyi Li, Mayank Khanna, and Ronald J. Jackson. "IL-4 and IL-13 Receptors." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101978.

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2

Ranasinghe, Charani, Sreeja Roy, Zheyi Li, Mayank Khanna, and Ronald J. Jackson. "IL-4 and IL-13 Receptors." In Encyclopedia of Signaling Molecules. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4614-6438-9_101978-1.

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3

Pelaia, Girolamo, Alessandro Vatrella, and Rosario Maselli. "Anti-IL-4/IL-13 Biologics." In Asthma: Targeted Biological Therapies. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-46007-9_6.

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4

Borish, Larry, John W. Steinke, Marion Kasaian, and Samuel J. Goldman. "IL-4- and IL-13-directed approaches." In New Drugs and Targets for Asthma and COPD. KARGER, 2010. http://dx.doi.org/10.1159/000320808.

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5

Schroeder, John T. "Diagnostic Components: T Helper Cell Cytokines (IL-4, IL-5, IL-9, IL-10, IL-13, IL-17)." In Encyclopedia of Medical Immunology. Springer New York, 2014. http://dx.doi.org/10.1007/978-1-4614-9194-1_298.

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6

Keegan, Achsah, Keats Nelms, and William E. Paul. "The IL-4 Receptor — Signaling Mechanisms." In Advances in Experimental Medicine and Biology. Springer US, 1994. http://dx.doi.org/10.1007/978-1-4899-0987-9_21.

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Clair, R. P. "Saturday, September 4, 2010—Chicago, IL." In Zombie Seed and the Butterfly Blues. SensePublishers, 2013. http://dx.doi.org/10.1007/978-94-6209-308-9_60.

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Clair, R. P. "Saturday, September 4, 2010—Chicago, IL." In Zombie Seed and the Butterfly Blues. SensePublishers, 2013. http://dx.doi.org/10.1007/978-94-6209-308-9_61.

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9

Shirokaze, J., K. Yanagida, K. Shudo, K. Konomoto, K. Kamiya, and K. Sagara. "IL-4 Production Using Macroporous Microcarrier." In Animal Cell Technology: Developments Towards the 21st Century. Springer Netherlands, 1995. http://dx.doi.org/10.1007/978-94-011-0437-1_141.

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Mieli, Eugenio, Andrea Maria Francesco Valli, and Claudio Maccone. "Il calcolo del 4° parametro fl." In La galassia vivente. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-65654-5_9.

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Atti di convegni sul tema "IL-4"

1

Chaker, Adam, Iannis Hara, Kumar Mithilesh Jha, et al. "Interleukin-4 (IL-4) Rezeptor alpha Blockade schützt vor IL-4-bedingtem Geruchsverlust bei Mäusen." In 94. Jahresversammlung Deutsche Gesellschaft für Hals-Nasen-Ohren-Heilkunde, Kopf- und Hals-Chirurgie e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1766938.

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2

Chaker, Adam, Iannis Hara, Kumar Mithilesh Jha, et al. "Interleukin-4 (IL-4) receptor alpha blockade protects from IL-4 driven loss of smell in mice." In 94th Annual Meeting German Society of Oto-Rhino-Laryngology, Head and Neck Surgery e.V., Bonn. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1767594.

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3

Kalinauskaitė-Žukauskė, Virginija, Andrius Januškevičius, Ieva Janulaitytė, and Kęstutis Malakauskas. "IL-4, IL-8, IL-13, IL-17, IL-33 serum levels changes in acute allergic asthma model." In Abstracts from the 17th ERS Lung Science Conference: ‘Mechanisms of Acute Exacerbation of Respiratory Disease’. European Respiratory Society, 2019. http://dx.doi.org/10.1183/23120541.lungscienceconference-2019.pp237.

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Muchtar, R. Masri. "294 Polymorphism promotor gene of il-4 and il-4 level at graves disease patients." In LUPUS 2017 & ACA 2017, (12th International Congress on SLE &, 7th Asian Congress on Autoimmunity). Lupus Foundation of America, 2017. http://dx.doi.org/10.1136/lupus-2017-000215.294.

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Haque, S. Jaharul, Rikhia Chakraborty, Pankaj Sharma, et al. "Antioxidant-Mediated Negative Regulation Of IL-4/IL-13 Signaling." In American Thoracic Society 2011 International Conference, May 13-18, 2011 • Denver Colorado. American Thoracic Society, 2011. http://dx.doi.org/10.1164/ajrccm-conference.2011.183.1_meetingabstracts.a2794.

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Kalinauskaitė-Žukauskė, Virginija, Andrius Januškevičius, Ieva Janulaitytė, and Kęstutis Malakauskas. "LSC - 2019 - IL-4, IL-8, IL-13, IL-17, IL-33 serum levels changes in acute allergic asthma model." In ERS International Congress 2019 abstracts. European Respiratory Society, 2019. http://dx.doi.org/10.1183/13993003.congress-2019.pa4083.

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Butova, Tatiana, Mykhailo Kuzhko, Dmytro Butov, and Nataliya Piriatinskaa. "Association between IL-2,IL-4 gene polymorphisms and pulmonary MDRTB." In ERS International Congress 2017 abstracts. European Respiratory Society, 2017. http://dx.doi.org/10.1183/1393003.congress-2017.pa3035.

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Yadav, Aravind, Rakesh Naidu, and Gopala Krishnan Govindasamy. "IL-4, IL-13 And IL-4RA Gene Polymorphisms And The Risk For Developing Idiopathic Nonallergic Rhinitis." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4191.

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Kozlowski, Mateusz, Iwona Rotter, Agnieszka Turon-Skrzypinska, et al. "#741 The utility of IL-4, IL-7 and IL-9 in the diagnosis of endometrial cancer." In ESGO 2023 Congress. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/ijgc-2023-esgo.252.

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10

Pereira, Clayrton de Barros, Yuri Felix Pedra, and Renata Dellalibera-Joviliano. "Profile of IL-1 beta, INF-gamma, IL-4 and IL-17 in patients with COVID-19." In III SEVEN INTERNATIONAL MULTIDISCIPLINARY CONGRESS. Seven Congress, 2023. http://dx.doi.org/10.56238/seveniiimulti2023-175.

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The immune response to SARS-CoV-2 is mediated by several soluble chemotactic factors, including cytokines whose levels vary at different stages and types of infections. Clinical evidence shows that "cytokine storm," in which the body has high levels of several pro-inflammatory cytokines, is common in several patients with severe COVID-19. Thus, the aim of this study was to evaluate the inflammatory response mediated by the cytokines IL-1 Beta, INF-Gama, IL-4 and IL-17 in patients with COVID. Sandwich-type immunoenzymatic assays (ELISA sandwich) were performed to determine the serum cytokine co

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Rapporti di organizzazioni sul tema "IL-4"

1

Cao, Xianling, Xuanyou Zhou, Naixin Xu, Songchang Chang, and Chenming Xu. Association of IL-4 and IL-10 Polymorphisms with Preterm Birth Susceptibility: A Systematic Review and Meta-Analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.4.0044.

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Review question / Objective: The aim of our systematic review and meta-analysis was to summarize the effects of IL-4 and IL-10 gene polymorphism and clarify their possible association with PTB. Condition being studied: World Health Organization (WHO) defines preterm birth (PTB) as babies born alive before 37 weeks of pregnancy are completed. The new estimates show that the prevalence of PTB during 2014 ranged from 8.7% to13.4% of all live births, about 15 million preterm babies born each year. Besides, PTB is the leading cause of death worldwide for children below 5 years of age. Babies born p

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2

Gilroy, Jill M. Overexpression of IL-4 Signaling Pathway to Inhibit Breast Tumor Growth. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada403390.

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3

Gooch, Jennifer L. Overexpression of IL-4 Signaling Pathway to Inhibit Breast Tumor Growth. Defense Technical Information Center, 1999. http://dx.doi.org/10.21236/ada390684.

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4

Gooch, Jennifer. Overexpression of IL-4 Signaling Pathway to Inhibit Breast Tumor Growth. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada384372.

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5

Sachdev, Deepali. Role of IRS-1 Phosphorylation in IGF-1 and IL-4 Signaling in Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada396609.

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Sachdev, Deepali. Role of IRS-1 Phosphorylation in IGF-1 and IL-4 Signaling in Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada407504.

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Sachdev, Deepali. Role of IRS-1 Phosphorylation in IGF-1 and IL-4 Signaling in Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada418308.

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ปาลกะ, ธนาภัทร. สารกดการกระตุ้นอินฟลามาโซมเพื่อบำบัดโรคอักเสบด้วยตนเอง : รายงานการวิจัย. จุฬาลงกรณ์มหาวิทยาลัย, 2015. https://doi.org/10.58837/chula.res.2015.47.

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Abstract (sommario):

กลุ่มโรคที่เกิดจากการอักเสบด้วยตนเอง (autoinflammatory diseases) เป็นโรคที่เกิดจาการอักเสบที่ไม่เกิดร่วมกับการติดเชื้อมีหลากหลายรูปแบบ โดยเป็นกลุ่มโรคที่มีสาเหตุมาจากปัจจัยทางพันธุกรรมและ/หรือจากความผิดปกติของระบบเมแทบอลิสม โรคเก๊าท์ (gout) จัดเป็นโรคหนึ่งในกลุ่มโรคนี้ ซึ่งมีการตกตะกอนเป็นผลึกของเกลือยูเรต (monosodium urate) ที่เป็นผลมาจากการมีปริมาณกรดยูริกในเลือดสูงเกินปกติ สาเหตุหลักของอาการอักเสบเกิดจากการหลั่งไซโตไคน์ที่เกี่ยวข้องกับการอักเสบ โดยเฉพาะอินเตอร์ลิวคิน 1 บีต้า (interleukin 1 β; IL-1β) และอินเตอร์ลิวคิน 18 (IL-18) ผ่านการกระตุ้นอินฟลามาโซม (Inflammasome) อินฟลามาโซมเป็นสารประก

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ปุญญโชต, สุทธาสินี, ฉัตรศรี เดชะปัญญา та สุพจน์ วัฒนะพันธ์ศักดิ์. การใช้เซลล์เพาะเลี้ยงเยื่อบุมดลูกสุกรทดสอบความแตกต่างการตอบสนองต่อพีอาร์อาร์เอสไวรัสสายพันธุ์ชนิดรุนแรงต่างกัน : รายงานฉบับสมบูรณ์. จุฬาลงกรณ์มหาวิทยาลัย, 2015. https://doi.org/10.58837/chula.res.2015.76.

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Abstract (sommario):

กลไกและพยาธิกำเนิดของการติดต่อโรคพีอาร์อาร์เอสจากแม่สู่ลูกยังไม่มีคำอธิบายที่แน่ชัดเนื่องจากยังขาดข้อมูลถึงความสามารถของการติดเชื้อที่ระบบสืบพันธุ์ เพื่อการศึกษาดังกล่าวด้วยการใช้เซลล์เยื่อบุมดลูกสุกรแบบเพาะเลี้ยงเบื้องตันและทำการตรวจสอบการเปลี่ยนแปลงรูปร่างของเซลล์ (ซีพีอี) การตอบสนองของระบบภูมิคุ้มกันที่มีมาแต่กำเนิดโดยเฉพาะการต้านทาน และการหลั่งไซโตคายน์ชนิดอักเสบเมื่อได้รับเชื้อไวรัสพีอาร์อาร์เอสจากสายพันธุ์ยูเอส หรืออียู ทางด้านท่อหรือด้านฐานเปรียบเทียบกัน การเปลี่ยนแปลงการปรากฏของโปรตีน และยีนของตัวรับชนิด CD151 CD163 และSn ต่อเชื้อไวรัสพีอาร์อาร์เอสซึ่งเป็นตัวการสำคัญที่ทำให้เซลล์สามารถ

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Suksom, Daroonwan, Jettanong Klaewsongkram, Wannaporn Tongtako, and Nutdanai Jaronsukwimal. Effect of acute exercises, chronic exercise training and vitamin C supplementation on physiological change and symptoms in allegic rhinitis patients. Chulalongkorn University, 2013. https://doi.org/10.58837/chula.res.2013.72.

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The aim of this present study was to investigate an effect of acute exhaustive and moderate intensities exercises, exercise training and vitamin C supplementation on physiological change and rhinitis symptoms in allergic rhinitis patients. There were two studies: Study I : The subjects, aged18-45 years old, were divided into 2 groups: 14 healthy individuals and 13 allergic rhinitis (AR) patients. They were assigned to perform the strenuous exercise on treadmill by using Bruce protocol until they were exhausted. Two weeks later, they were assigned to perform a moderate exercise by walking on tr

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