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Articoli di riviste sul tema "IL-4"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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1

Usha, Prof. "IL-4 and IL-6 in Bronchial Asthma Does IL-6 Plays More Important Role than IL-4? A Preliminary Study." Journal of Medical Science And clinical Research 05, no. 04 (2017): 20446–50. http://dx.doi.org/10.18535/jmscr/v5i4.115.

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2

Kelly-Welch, A., E. M. Hanson, and A. D. Keegan. "Interleukin-4 (IL-4) Pathway." Science Signaling 2005, no. 293 (2005): cm9. http://dx.doi.org/10.1126/stke.2932005cm9.

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3

Borish, Larry. "IL-4 and IL-13 Dual Antagonism." American Journal of Respiratory and Critical Care Medicine 181, no. 8 (2010): 769–70. http://dx.doi.org/10.1164/rccm.201002-0147ed.

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4

Otani, Yusuke, and Takumi Takizawa. "Anti-IL-4/IL-13 antibody (Dupilumab)." Nihon Shoni Arerugi Gakkaishi. The Japanese Journal of Pediatric Allergy and Clinical Immunology 37, no. 3 (2023): 240–47. http://dx.doi.org/10.3388/jspaci.37.240.

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5

Schindler, C., H. Kashleva, A. Pernis, R. Pine, and P. Rothman. "STF-IL-4: a novel IL-4-induced signal transducing factor." EMBO Journal 13, no. 6 (1994): 1350–56. http://dx.doi.org/10.1002/j.1460-2075.1994.tb06388.x.

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6

Gause, W. C., T. Takashi, J. D. Mountz, F. D. Finkelman, and A. D. Steinberg. "Activation of CD 4-, CD 8- thymocytes with IL 4 vs IL 1 + IL 2." Journal of Immunology 141, no. 7 (1988): 2240–45. http://dx.doi.org/10.4049/jimmunol.141.7.2240.

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Abstract Thymocytes from C57BL/6 mice were highly purified to obtain the CD 4-, CD 8- subpopulation which constitutes only 5% of all thymocytes. Substantial proliferation was induced in vitro with either IL-1 + IL-2 or with IL-4 in the presence of PMA. IL-1 and IL-2 synergized in inducing proliferation of these purified CD 4-, CD 8- thymocytes whereas neither synergized with IL-4. In order to determine whether stimulation with IL-1 + IL-2 acted via IL-4 or vice versa, cultures were treated reciprocally with affinity-purified anti-IL-2 or anti-IL-4 antibodies. Cultures with IL-4 were inhibited

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7

Ho, S. N., R. T. Abraham, A. Nilson, B. S. Handwerger, and D. J. McKean. "Interleukin 1-mediated activation of interleukin 4 (IL 4)-producing T lymphocytes. Proliferation by IL 4-dependent and IL 4-independent mechanisms." Journal of Immunology 139, no. 5 (1987): 1532–40. http://dx.doi.org/10.4049/jimmunol.139.5.1532.

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Abstract The role of IL 1 in the activation of IL 4-producing murine T cell clones was investigated by using a calcium ionophore (ionomycin) or a phorbol ester (12-O-tetradecanoylphorbol 13-acetate; TPA) as T cell receptor-independent costimuli. The use of these pharmacologic agents to investigate IL 1-mediated T cell activation revealed two distinct mechanisms of activation. IL 1 in combination with ionomycin (iono/rIL 1) stimulated a proliferative response that was associated with the production of IL 4 as measured by lymphokine bioassay and mRNA studies. Furthermore, inhibition of this prol

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8

Atamas, S. P., J. Choi, V. V. Yurovsky, and B. White. "An alternative splice variant of human IL-4, IL-4 delta 2, inhibits IL-4-stimulated T cell proliferation." Journal of Immunology 156, no. 2 (1996): 435–41. http://dx.doi.org/10.4049/jimmunol.156.2.435.

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Abstract Alternative splicing of mRNA can generate protein isoforms that are preferentially expressed in different tissues or during different states of cell differentiation or activation. Protein isoforms may have different functions. In this study, we cloned, expressed, and tested functional effects of a naturally occurring splice variant of human IL-4, called IL-4 delta 2. In IL-4 delta 2, the second exon of IL-4 is omitted by alternative splicing, with exons 1, 3, and 4 joined in an open reading frame. We found that IL-4 delta 2 RNA is expressed in the PBMC of all donors tested, usually in

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9

NAKANISHI, KENJI. "Interleukin cascade of IL-4,IL-5 and IL-2." Japanese Journal of Clinical Immunology 13, no. 5 (1990): 438–40. http://dx.doi.org/10.2177/jsci.13.438.

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10

Noben-Trauth, N., L. D. Shultz, F. Brombacher, J. F. Urban, H. Gu, and W. E. Paul. "An interleukin 4 (IL-4)-independent pathway for CD4+ T cell IL-4 production is revealed in IL-4 receptor-deficient mice." Proceedings of the National Academy of Sciences 94, no. 20 (1997): 10838–43. http://dx.doi.org/10.1073/pnas.94.20.10838.

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11

Guida, Pier Luigi. "Il nuovo PMBOK "4"." PROJECT MANAGER (IL), no. 2 (June 2010): 15–20. http://dx.doi.org/10.3280/pm2010-002006.

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12

Pan, Ping-Ying, and Paul Rothman. "IL-4 receptor mutations." Current Opinion in Immunology 11, no. 6 (1999): 615–20. http://dx.doi.org/10.1016/s0952-7915(99)00026-6.

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13

Strait, Richard T., Suzanne C. Morris, Kristi Smiley, Joseph F. Urban, and Fred D. Finkelman. "IL-4 Exacerbates Anaphylaxis." Journal of Immunology 170, no. 7 (2003): 3835–42. http://dx.doi.org/10.4049/jimmunol.170.7.3835.

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14

Pearce, Edward J., Joao Pedras Vasconcelos, Laura Rosa Brunet, and Elizabeth A. Sabin. "IL-4 in Schistosomiasis." Experimental Parasitology 84, no. 2 (1996): 295–99. http://dx.doi.org/10.1006/expr.1996.0116.

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15

de Moraes-Pinto, M. I., G. S. Vince, B. F. Flanagan, C. A. Hart, and P. M. Johnson. "Il-4 and Il-4 receptors in human pregnancy tissues at term." Placenta 17, no. 5-6 (1996): A52. http://dx.doi.org/10.1016/s0143-4004(96)90268-4.

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16

Jeannin, Pascale, Yves Delneste, Jean-Pierre Aubry, et al. "Thiols Decrease Human IL-4 Production and IL-4-lnduced Immunoglobulin Synthesis." International Archives of Allergy and Immunology 113, no. 1-3 (1997): 329–30. http://dx.doi.org/10.1159/000237591.

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17

Jung, Thomas, Kathrin Wagner, Christine Neumann, and Christoph H. Heusser. "Enhancement of human IL-4 activity by soluble IL-4 receptorsin vitro." European Journal of Immunology 29, no. 3 (1999): 864–71. http://dx.doi.org/10.1002/(sici)1521-4141(199903)29:03<864::aid-immu864>3.0.co;2-t.

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18

Reich, Adam, Justyna Szczęch, and Dominik Samotij. "Biologics for Itch: IL-4/IL-13, IL-31, IL-17, and IL-23 Antagonists." Current Dermatology Reports 6, no. 4 (2017): 263–72. http://dx.doi.org/10.1007/s13671-017-0204-7.

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19

Vélez, C., D. Williamson та M. Koncurat. "IL-1β, IL-2, IL-4 and IL-10 profile during porcine gestation". Placenta 51 (березень 2017): 116. http://dx.doi.org/10.1016/j.placenta.2017.01.065.

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20

Rubin, J. T., A. Brumfield, and H. T. Lotze. "OIL-BASED DELIVERY OF IL-2, IL-4, IL-12, and IL-15." Journal of Immunotherapy 16, no. 3 (1994): 243. http://dx.doi.org/10.1097/00002371-199410000-00044.

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21

Rasha, A.H. Alathary, A. Abass Rusul, Taqi Mansour Al muhtaser Seenaa, and A. Al-fahham Ali. "Biochemistry, Pathophysiology and Clinical Importance of IL-4: A Review Article." INTERNATIONAL JOURNAL OF HEALTH & MEDICAL RESEARCH 04, no. 01 (2025): 44–47. https://doi.org/10.5281/zenodo.14676811.

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Abstract (sommario):

Interleukin-4 (IL-4) is one of the most important immunomodulatory cytokines. Although it is thought of purely as acting on the differentiation of T helper cells, IL-4 can polarize macrophages. That is why knowledge of the biochemical pathways and molecular mechanisms of IL-4 action is needed. Various biological activities have been ascribed to IL-4, including immune responses, inflammation, and tissue homeostasis. This wide range of activities is likely to be based on defined pathophysiological features in diseases within the nervous system and allergic disorders. Hence, this review attempts

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22

Hamna, Tariq1* Aniqa Amir1 Muhammad Saleem1 Kainat Ramzan2* Tuba Aslam1 Mehmooda Asif1. "In-Depth In-Silico Functional, And Structural Screening Of IL-4 Gene Variants Linked with Pink Eye Infection." International Journal of Scientific Research and Technology 1, no. 11 (2024): 213–29. https://doi.org/10.5281/zenodo.14234050.

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Conjunctival inflammation affects millions globally and can be triggered by microbial infections, allergens, or genetic factors. This study focuses on specific SNPs in the IL-4 gene, linked to pink eye susceptibility. Using <em>In-Silico</em> tools, 15 SNPs were identified as potentially damaging to IL-4 protein, with the R139G, A118G, R112C, and R139W variants showing probable damage on high RMSD values, as highlighted by SAVESV6.1. Structural analysis using HOPE predicted that the R139W mutation may be structurally bigger than the IL-4 wild type, while other variants were dama

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23

Jiang, Hong, Miera B. Harris, and Paul Rothman. "IL-4/IL-13 signaling beyond JAK/STAT." Journal of Allergy and Clinical Immunology 105, no. 6 (2000): 1063–70. http://dx.doi.org/10.1067/mai.2000.107604.

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24

Minton, Kirsty. "What 'drives' IL-4 versus IL-13 signalling?" Nature Reviews Immunology 8, no. 3 (2008): 167. http://dx.doi.org/10.1038/nri2283.

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25

Zurawski, S. M., F. Vega, E. L. Doyle, and G. Zurawski. "The receptors for IL-4 and IL-13." Cytokine 6, no. 5 (1994): 580. http://dx.doi.org/10.1016/1043-4666(94)90320-4.

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26

Baranova, N. I., B. A. Molotilov, L. A. Ashchina та N. A. Shkurova. "Role of IL-1β, TNF-α, IL-10, IL-17, and IL-4 gene polymorphisms in the pathogenesis of chronic rhinosinusitis with nasal polyps". Russian Medical Inquiry 6, № 2 (2022): 57–61. http://dx.doi.org/10.32364/2587-6821-2022-6-2-57-61.

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Background: chronic rhinosinusitis with nasal polyps (CRSwNP) is a multifactorial disease, those mechanisms are not fully understood. A significant role of cytokines, e.g., interleukin (IL)-1β, tumor necrosis factor (TNF) α, IL-10, IL-17А, IL-4, and their genes, in the pathogenesis and predisposition to CRSwNP and treatment efficacy is established. Aim: to investigate IL-1β (Т-31С), TNF-α (G-308A), IL-10 (G-1082A), IL-17(G-197A), and IL-4 (С-589Т) gene polymorphisms and their role in CRSwNP pathogenesis. Patients and Methods: this open-label, prospective, randomized study enrolled 100 patients

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27

KOPF, MANFRED, GRAHAM LE GROS, ANTHONY J. COYLE, MARIE KOSCO-VTLBOIS, FRANK BROMBACHER, and Georges Kohler. "Immune Responses of IL-4, IL-5, IL-6 Deficient Mice." Immunological Reviews 148, no. 1 (1995): 45–69. http://dx.doi.org/10.1111/j.1600-065x.1995.tb00093.x.

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28

DAVID, MURIEL, JACQUES BERTOGLIO та JOSIANE PIERRE. "TNF-α Potentiates IL-4/IL-13-Induced IL-13Rα2 Expression". Annals of the New York Academy of Sciences 973, № 1 (2002): 207–9. http://dx.doi.org/10.1111/j.1749-6632.2002.tb04633.x.

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29

Vélez, Carolina, Mariángeles Clauzure, Delia Williamson, Mirta A. Koncurat, Tomás A. Santa-Coloma та Claudio Barbeito. "IL-1β, IL-2 and IL-4 concentration during porcine gestation". Theriogenology 128 (квітень 2019): 133–39. http://dx.doi.org/10.1016/j.theriogenology.2019.01.017.

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30

Le Gros, G., S. Z. Ben-Sasson, R. Seder, F. D. Finkelman, and W. E. Paul. "Generation of interleukin 4 (IL-4)-producing cells in vivo and in vitro: IL-2 and IL-4 are required for in vitro generation of IL-4-producing cells." Journal of Experimental Medicine 172, no. 3 (1990): 921–29. http://dx.doi.org/10.1084/jem.172.3.921.

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T cell populations derived from naive mice produce very small amounts of interleukin 4 (IL-4) in response to stimulation on anti-CD3-coated dishes. IL-4 production by such cells is mainly found among large- and intermediate-sized T cells and is dependent upon IL-2. Injection of anti-IgD into mice, a stimulus that leads to striking increases in serum levels of IgG1 and IgE, causes a striking increase in the IL-4-producing capacity of T cells. This increase is first observed 4 d after injection of anti-IgD. IL-4 production by T cells from anti-IgD-injected donors is mainly found among large- and

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31

Furusu, A., M. Miyazaki, T. Koji, et al. "Involvement of IL-4 in human glomerulonephritis: an in situ hybridization study of IL-4 mRNA and IL-4 receptor mRNA." Journal of the American Society of Nephrology 8, no. 5 (1997): 730–41. http://dx.doi.org/10.1681/asn.v85730.

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Interleukin-4 (IL-4) has been recently implicated in the pathogenesis of glomerulonephritis. However, the expression of IL-4 and IL-4 receptor (IL-4R) in human kidney has not been fully determined. Nonradioactive in situ hybridization was used to examine the expression of IL-4 mRNA and IL-4R mRNA in tissues from normal kidneys and specimens from a variety of human kidney diseases. In normal glomeruli, a few mesangial cells and cells of the Bowman's capsule weakly expressed IL-4 and IL-4R mRNA, whereas in diseased glomeruli both mRNA types were strongly expressed in resident glomerular cells, i

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32

Ben-Sasson, S. Z., G. Le Gros, D. H. Conrad, F. D. Finkelman, and W. E. Paul. "IL-4 production by T cells from naive donors. IL-2 is required for IL-4 production." Journal of Immunology 145, no. 4 (1990): 1127–36. http://dx.doi.org/10.4049/jimmunol.145.4.1127.

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Abstract Utilizing a sensitive and selective assay for IL-4, it was shown that lymph node T cells from naive mice could produce small amounts of this lymphokine in response to anti-CD3 antibodies adsorbed to culture dishes. The capacity of these cells to produce IL-4 in response to plate-bound anti-CD3 was substantially enhanced by the addition of IL-2 to the culture and was strikingly inhibited by monoclonal anti-IL-2 antibody. Thus, IL-2 appears to be essential for IL-4 production by anti-CD3 antibody-stimulated T cells from naive mice. The effect of IL-2 was not mediated either by preferent

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33

Martínez-Saldaña, Ma. Consolación. "CYPERMETHRIN CAUSE ALLERGIC INFLAMMATORY RESPONSE IN RAT LUNG." World Journal of Pharmaceutical Science and Research 2, no. 6 (2023): 263–76. https://doi.org/10.5281/zenodo.10421769.

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Cypermethrin is a type 2 photosensitive liposoluble pyrethroid for agricultural and domestic use, has a half-life in the ambient air for days, its metabolism in human liver generate a cyan group (CN), an alcohol fraction (dimethylcyclopropanecarboxylic acid) (DCCA), and 3-acid phenoxybenzoic acid (3-PBA) with reactive oxygen species (ROS) properties, there are scarce studies when is inhaled. The objective of this work was to evaluate the structural changes and activation of allergic inflammatory response in lower airway components and alveoli induced by inhaled exposure to low doses of cyperme

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34

Ho, I.-Cheng, Mark H. Kaplan, Laurie Jackson-Grusby, Laurie H. Glimcher, and Michael J. Grusby. "Marking IL-4-producing cells by knock-in of the IL-4 gene." International Immunology 11, no. 2 (1999): 243–47. http://dx.doi.org/10.1093/intimm/11.2.243.

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35

Jeannin, P., Y. Delneste, S. Lecoanet-Henchoz, et al. "Thiols decrease human interleukin (IL) 4 production and IL-4-induced immunoglobulin synthesis." Journal of Experimental Medicine 182, no. 6 (1995): 1785–92. http://dx.doi.org/10.1084/jem.182.6.1785.

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N-Acetyl-L-cysteine (NAC) is an antioxidant precursor of intracellular glutathione (GSH), usually given in human as a mucolytic agent. In vitro, NAC and GSH have been shown to act on T cells by increasing interleukin (IL) 2 production, synthesis and turnover of IL-2 receptors, proliferation, cytotoxic properties, and resistance to apoptosis. We report here that NAC and GSH decrease in a dose-dependent manner human IL-4 production by stimulated peripheral blood T cells and by T helper (Th) 0- and Th2-like T cell clones. This effect was associated with a decrease in IL-4 messenger RNA transcript

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36

Nakashima, H., K. Miyake, Y. Inoue, et al. "Association between IL-4 genotype and IL-4 production in the Japanese population." Genes & Immunity 3, no. 2 (2002): 107–9. http://dx.doi.org/10.1038/sj.gene.6363830.

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37

Kubo, Masato, Masakatsu Yamashita, Ryo Abe, et al. "CD28 Costimulation Accelerates IL-4 Receptor Sensitivity and IL-4-Mediated Th2 Differentiation." Journal of Immunology 163, no. 5 (1999): 2432–42. http://dx.doi.org/10.4049/jimmunol.163.5.2432.

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Abstract The development of Th1 and Th2 cells is determined by the type of antigenic stimulation involved in the initial cell activation step. Evidence indicates that costimulatory signals, such as those delivered by CD28, play an important role in Th2 development, but little is known about how CD28 costimulation contributes to Th2 development. In this study, TCR cross-linking was insufficient for Th2 development, while the addition of CD28 costimulation drastically increased Th2 generation through the IL-4-mediated pathway. Th2 generation following CD28 costimulation was not simply explained

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38

Konenkov, Konenkov V. I., Koroleva E. G. Koroleva, Orlov N. B. Orlov та ін. "Anti-inflammatory activity of serum cytokines (IL-4, IL-10, IL-13) and the natural IL-1β receptor antagonist (IL-1Ra) in women with uterine myoma". Akusherstvo i ginekologiia 10_2018 (31 жовтня 2018): 80–85. http://dx.doi.org/10.18565/aig.2018.10.80-85.

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39

Dubin, Celina, Ester Del Duca, and Emma Guttman-Yassky. "The IL-4, IL-13 and IL-31 pathways in atopic dermatitis." Expert Review of Clinical Immunology 17, no. 8 (2021): 835–52. http://dx.doi.org/10.1080/1744666x.2021.1940962.

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40

Klatka, Janusz, and Jacek Tabarkiewicz. "Dendritic Cells, IL-2, IL-4 and IL-12 in Laryngeal Cancer." Otolaryngology–Head and Neck Surgery 143, no. 2_suppl (2010): P214—P215. http://dx.doi.org/10.1016/j.otohns.2010.06.420.

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41

Cicuttini, F. M., K. A. Byron, D. Maher, A. M. Wootton, K. D. Muirden, and J. A. Hamilton. "Serum IL-4, IL-10 and IL-6 levels in inflammatory arthritis." Rheumatology International 14, no. 5 (1995): 201–6. http://dx.doi.org/10.1007/bf00262298.

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42

Huang, H., J. Hu-Li, H. Chen, S. Z. Ben-Sasson, and W. E. Paul. "IL-4 and IL-13 production in differentiated T helper type 2 cells is not IL-4 dependent." Journal of Immunology 159, no. 8 (1997): 3731–38. http://dx.doi.org/10.4049/jimmunol.159.8.3731.

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Abstract CD4+ T cell differentiation into cells capable of producing IL-4 and IL-13 (Th2 cells) requires the presence of IL-4 and is STAT-6 dependent. Here we show that IL-4 is not required for IL-4 or IL-13 production by Th2 cells. Anti-IL-4 or anti-IL-4R Ab did not diminish IL-4 production by Th2 cells in response to TCR-mediated stimulation, nor did IL-4 enhance IL-4 production in response to stimulation of Th2 cells with limiting amounts of Ag. Th2 cells prepared from IL-4 knockout mice were capable of producing IL-13 mRNA in response to stimulation with immobilized anti-CD3. IL-4 did not

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43

Park, Yong Jin, та He Ro Yoon. "Expression of IL-4, IL-6, GM-CSF and IFN-γ mRNAs in Nasal Polyps". Journal of Clinical Otolaryngology Head and Neck Surgery 10, № 1 (1999): 53–60. http://dx.doi.org/10.35420/jcohns.1999.10.1.53.

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44

Perkins, Charles, Marsha Wills-Karp, and Fred D. Finkelman. "IL-4 induces IL-13–independent allergic airway inflammation." Journal of Allergy and Clinical Immunology 118, no. 2 (2006): 410–19. http://dx.doi.org/10.1016/j.jaci.2006.06.004.

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45

McCormick, Sarah M., and Nicola M. Heller. "Commentary: IL-4 and IL-13 receptors and signaling." Cytokine 75, no. 1 (2015): 38–50. http://dx.doi.org/10.1016/j.cyto.2015.05.023.

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46

Keegan, Achsah D. "IL-4 and IL-13: From “supe” to nuts." Cytokine 75, no. 1 (2015): 1–2. http://dx.doi.org/10.1016/j.cyto.2015.06.001.

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47

Krymskaya, V., G. Vass, R. Z. Syed, R. A. Panettieri, and A. Haczku. "IL-4 and IL-13 induce fibroblast-myofibroblast differentiation." Journal of Allergy and Clinical Immunology 115, no. 2 (2005): S120. http://dx.doi.org/10.1016/j.jaci.2004.12.493.

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48

Murata, Takashi, Nicholas I. Obiri, and Raj K. Puri. "Human ovarian-carcinoma cell lines express IL-4 and IL-13 receptors: Comparison between IL-4- and IL-13-induced signal transduction." International Journal of Cancer 70, no. 2 (1997): 230–40. http://dx.doi.org/10.1002/(sici)1097-0215(19970117)70:2<230::aid-ijc15>3.0.co;2-m.

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49

Chasserio, Stephanie, Philippe Pailot, and Corinne Poroli. "4. L’entrepreneuriat est-il genré ?" Regards croisés sur l'économie 19, no. 2 (2016): 62. http://dx.doi.org/10.3917/rce.019.0062.

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50

Jauregui, Paula. "IL-4 promotes immunotherapy resistance." Nature Immunology 25, no. 12 (2024): 2169. http://dx.doi.org/10.1038/s41590-024-02032-3.

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