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Articoli di riviste sul tema "Pathogenesis"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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1

Tanashyan, M. M., K. V. Antonova, N. E. Spryshkov, A. А. Panina, O. V. Lagoda, and E. P. Shchukina. "Diabetic Polyneuropathy: from Pathogenesis to Pathogenetic Therapy." Effective Pharmacotherapy 20, no. 8 (2024): 54–62. http://dx.doi.org/10.33978/2307-3586-2024-20-8-54-62.

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Abstract (sommario):

The growing worldwide prevalence of prediabetes and diabetes mellitus (DM) has led to an increase in the incidence of related chronic complications, the most common of which is diabetic polyneuropathy (DPN). The development of DPN is caused by chronic hyperglycemia and potentially modifiable cardiovascular risk factors, including elevated triglyceride levels, body mass index, smoking and hypertension. Social determinants of health are also risk factors for DPN. The review presents data on the pathophysiology of DPN with consideration of the causes of the sequence of symptoms. Discussed the mec

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2

Orazov, M. R., V. E. Radzinsky, E. D. Dolgov, and Yu G. Abramashvili. "Pathogenesis and pathogenetic options in endometriosis management." Voprosy ginekologii, akušerstva i perinatologii 22, no. 1 (2023): 92–104. http://dx.doi.org/10.20953/1726-1678-2023-1-92-104.

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This article provides a literature review on the etiopathogenesis of endometriosis. Particular attention was paid to genetic and molecular biological aspects of endometriosis. The issues of conservative management of patients with confirmed endometriosis were also considered. The use of dienogest in the treatment of patients with endometriosis was substantiated. Key words: endometriosis, etiopathogenesis, genetic and molecular determinants, dienogest

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3

Strokov, I. A., and V. V. Oganov. "Pathogenesis, evaluation and pathogenetic therapy of diabetic polyneuropathy." Neurology, Neuropsychiatry, Psychosomatics 13, no. 3 (2021): 99–106. http://dx.doi.org/10.14412/2074-2711-2021-3-99-106.

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The increase in the number and life expectancy of patients with diabetes mellitus (DM) worldwide determines the high prevalence of late complications of diabetes, including diabetic polyneuropathy (DPN), the most common type of polyneuropathy. Oxidative stress is considered the main reason for the cellular pathology development in diabetes mellitus, which determines the use of antioxidants for the DPN treatment. Alpha-lipoic acid (ALA), a natural fat-soluble antioxidant, is the most effective drug for reducing DPN symptoms. Furthermore, the symptom-modifying effect of ALA has been shown in num

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4

Roy, Saumendu Deb. "Pathogenesis of COVID-19." INDIAN RESEARCH JOURNAL OF PHARMACY AND SCIENCE 7, no. 4 (2020): 2354–58. http://dx.doi.org/10.21276/irjps.2020.7.3.3.

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5

Galante, Iv. "A. Bakk & Gh. Tamasescu Ueber die Aetiologie und Pathogenese der Epilepsie und deren Therapie. Wiener Med. W. No. 17, 1933." Kazan medical journal 29, no. 5-6 (2022): 489. http://dx.doi.org/10.17816/kazmj89620.

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Abstract (sommario):

Bakk and Tamasescu (A. Bakk Gh. Tamasescu Ueber die Aetiologie und Pathogenese der Epilepsie und deren Therapie. Wiener Med. W. No. 17, 1933) give a good concise overview of the etiology, pathogenesis and treatment of epilepsy. Epilepsy is an organic syndrome with a different etiology and multiple pathogenetic factors, which explains that the methods of treatment for epilepsy vary enormously.

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6

Kim, Young Kyoon, and Younsuck Koh. "Pathogenesis." Tuberculosis and Respiratory Diseases 50, no. 5 (2001): 525. http://dx.doi.org/10.4046/trd.2001.50.5.525.

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7

Witz, Craig, and Robert Schenken. "Pathogenesis." Seminars in Reproductive Medicine 15, no. 03 (1997): 199–208. http://dx.doi.org/10.1055/s-2008-1068749.

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8

A, Abdelmoktader. "Mycobacterial Tuberculosis Epidemiology and Pathogenesis." Virology & Immunology Journal 4, no. 4 (2020): 1–7. http://dx.doi.org/10.23880/vij-16000259.

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Mycobacterium tuberculosis (MTB) is an acid fast bacterium (AFB); it has tough cell wall and circular chromosome. It is transmitted through the airborne route and cause tuberculosis (TB). The distribution of tuberculosis is not uniform across the globe; about 80% of the population in many Asian and African countries and it is the second most common cause of death from infectious disease after HIV. Organisms deposited mainly in the upper lung zones, kidneys and bones. In persons with intact cell-mediated immunity (CMI), collections of activated T cells and macrophages form granulomas that limit

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9

Gollnick, Harald P. M., Christos C. Zouboulis, Hirohiko Akamatsu, Ichiro Kurokawa, and Anja Schulte. "Pathogenesis and Pathogenesis Related Treatment of Acne." Journal of Dermatology 18, no. 9 (1991): 489–99. http://dx.doi.org/10.1111/j.1346-8138.1991.tb03122.x.

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10

HAYASHI, Koichi. "Pathogenesis of Hypertension -Kidney as a Pathogenetic Organ of Hypertension." Internal Medicine 40, no. 2 (2001): 153–56. http://dx.doi.org/10.2169/internalmedicine.40.153.

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11

Bespalov, Vladimir G., and Elizaveta I. Kovalevskaya. "Modern conceptions of etiology and pathogenesis of benign breast disease: the possibilities of pathogenetic treatment." Gynecology 21, no. 1 (2019): 52–58. http://dx.doi.org/10.26442/20795696.2019.1.190217.

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Abstract (sommario):

Relevance. The review article is devoted to modern conceptions of etiology and pathogenesis of benign breast disease (BBD), a common female pathology, as well as the pathogenetic treatment of BBD. Aim. Substantiation of the new possibilities of the pathogenetic treatment of BBD. Materials and methods. It was conducted the analysis of domestic and world literature in the following areas: etiology, pathogenesis, treatment of BBD, prevention of breast cancer (BC). Results. Dozens of etiological factors of various natures cause BBD: genetic, reproductive, hormonal, gynecological diseases, extragen

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12

Bilalova, K. A., and L. A. Yusupova. "ROSACEA: FEATURES OF PATHOGENESIS AND THERAPY." European Journal of Natural History, no. 2 2022 (2022): 18–21. http://dx.doi.org/10.17513/ejnh.34252.

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13

Tuor, Paula, and Jenkins Zhao. "Pathogenesis of Brain: Autism Spectrum Disorders." Neuroscience and Neurological Surgery 2, no. 2 (2018): 01–02. http://dx.doi.org/10.31579/2578-8868/029.

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Autism spectrum disorders (ASDs) affect as many as 1 in 45 children and are characterized by deficits in sociability and communication, as well as stereotypic movements. Many children also show severe anxiety.

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14

Syabbalo, Nightingale. "Severe Neutrophilic Asthma: Pathogenesis and Treatment." Journal of Thoracic Disease and Cardiothoracic Surgery 3, no. 1 (2022): 01–13. http://dx.doi.org/10.31579/2693-2156/030.

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Abstract (sommario):

Asthma is a common chronic airway disease affecting about 358 million people worldwide, and an estimated 7 million children globally. Approximately 10% of patients with asthma have severe refractory disease, which is difficult to control on high doses of inhaled corticosteroids and other modifiers. Among these, are patients with severe neutrophilic asthma. Neutrophilic asthma is a severe phenotype of asthma, characterized by frequent exacerbations, persistent airway obstruction, and poor lung function. Immunopathologically, it is characterized by the presence of high levels of neutrophils in t

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15

O.A., Babadjanov, and Pulatova S.X. "ACNE: ETIOLOGY, PATHOGENESIS AND MODERN THERAPY." International Journal of Medical Sciences And Clinical Research 03, no. 03 (2023): 46–50. http://dx.doi.org/10.37547/ijmscr/volume03issue03-07.

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16

Smith, Laurie D., and Thomas A. Ficht. "Pathogenesis ofBrucella." Critical Reviews in Microbiology 17, no. 3 (1990): 209–30. http://dx.doi.org/10.3109/10408419009105726.

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17

Garcia, Sonia Arely, Vincent Y. Ng, Masahiro Iwamoto, and Motomi Enomoto-Iwamoto. "Osteochondroma Pathogenesis." American Journal of Pathology 191, no. 12 (2021): 2042–51. http://dx.doi.org/10.1016/j.ajpath.2021.08.003.

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18

Haidar, Ruvin. "Microbial Pathogenesis." International Journal of Health Sciences and Research 11, no. 12 (2021): 217–19. http://dx.doi.org/10.52403/ijhsr.20211228.

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For a pathogenic microbe to cause disease in a susceptible host, it must gain access to that host first. The pathogenicity of a microbe is determined by the virulence factors alongside other innate mechanisms. Apart from the initiation of infection, these virulence factors also enable the pathogenic microorganism to survive in the new environment within the susceptible host. They also enable the pathogenic microorganism to invade the host, colonize, and evade the host defense mechanisms. These virulence factors include; invasins, capsules, siderophores, adhesins, enzymes, endotoxins, and exoto

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19

Horwitz, Ralph I., Burton H. Singer, Allison Hayes-Conroy, et al. "Biosocial Pathogenesis." Psychotherapy and Psychosomatics 91, no. 2 (2022): 73–77. http://dx.doi.org/10.1159/000521567.

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20

Johnson, Cydney, Virginia Hargest, Valerie Cortez, Victoria Meliopoulos, and Stacey Schultz-Cherry. "Astrovirus Pathogenesis." Viruses 9, no. 1 (2017): 22. http://dx.doi.org/10.3390/v9010022.

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21

Eyre, David, and David J. Schurman. "Pathogenesis/Homeostasis." Clinical Orthopaedics and Related Research 427 (October 2004): S104. http://dx.doi.org/10.1097/01.blo.0000144978.73030.b7.

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22

Milner, Danny A. "Malaria Pathogenesis." Cold Spring Harbor Perspectives in Medicine 8, no. 1 (2017): a025569. http://dx.doi.org/10.1101/cshperspect.a025569.

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23

Scott, J. R. "Scrapie pathogenesis." British Medical Bulletin 49, no. 4 (1993): 778–91. http://dx.doi.org/10.1093/oxfordjournals.bmb.a072646.

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24

Moayeri, Mahtab, Stephen H. Leppla, Catherine Vrentas, Andrei P. Pomerantsev, and Shihui Liu. "Anthrax Pathogenesis." Annual Review of Microbiology 69, no. 1 (2015): 185–208. http://dx.doi.org/10.1146/annurev-micro-091014-104523.

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25

Fauci, Anthony S. "Cellular Pathogenesis." AIDS Research and Human Retroviruses 7, no. 2 (1991): 202–15. http://dx.doi.org/10.1089/aid.1991.7.202.

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26

Miller, L., M. Good, and G. Milon. "Malaria pathogenesis." Science 264, no. 5167 (1994): 1878–83. http://dx.doi.org/10.1126/science.8009217.

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27

FRITSCH, P. "WS102 Pathogenesis." Journal of the European Academy of Dermatology and Venereology 9 (September 1997): S100. http://dx.doi.org/10.1016/s0926-9959(97)89260-4.

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28

Ross, Christopher A. "Polyglutamine Pathogenesis." Neuron 35, no. 5 (2002): 819–22. http://dx.doi.org/10.1016/s0896-6273(02)00872-3.

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29

Karumanchi, S. Ananth, and Marshall D. Lindheimer. "Preeclampsia Pathogenesis." Hypertension 51, no. 4 (2008): 991–92. http://dx.doi.org/10.1161/hypertensionaha.107.100735.

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30

Paumgartner, G., and T. Sauerbruch. "Gallstones: pathogenesis." Lancet 338, no. 8775 (1991): 1117–21. http://dx.doi.org/10.1016/0140-6736(91)91972-w.

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31

Forrester, J. "Uveitis: pathogenesis." Lancet 338, no. 8781 (1991): 1498–501. http://dx.doi.org/10.1016/0140-6736(91)92309-p.

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32

Nowak, Martin A. "AIDS Pathogenesis." Journal of Acquired Immune Deficiency Syndromes & Human Retrovirology 10, Supplement (1995): S6. http://dx.doi.org/10.1097/00042560-199510001-00002.

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33

Buller, R. M., and G. J. Palumbo. "Poxvirus pathogenesis." Microbiological Reviews 55, no. 1 (1991): 80–122. http://dx.doi.org/10.1128/mmbr.55.1.80-122.1991.

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34

Buller, R. M., and G. J. Palumbo. "Poxvirus pathogenesis." Microbiological Reviews 55, no. 1 (1991): 80–122. http://dx.doi.org/10.1128/mr.55.1.80-122.1991.

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35

Edlin, Gordon. "AIDS Pathogenesis." Nature Biotechnology 6, no. 2 (1988): 217. http://dx.doi.org/10.1038/nbt0288-217b.

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36

Jackson, Alan C. "Rabies pathogenesis." Journal of Neurovirology 8, no. 4 (2002): 267–69. http://dx.doi.org/10.1080/13550280290100725.

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37

Nowak, Martin, and Robert M. May. "AIDS pathogenesis." AIDS 7 (January 1992): S3—S18. http://dx.doi.org/10.1097/00002030-199201001-00002.

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38

Nowak, Martin, and Robert M. May. "AIDS pathogenesis." AIDS 7 (January 1993): S3—S18. http://dx.doi.org/10.1097/00002030-199301001-00002.

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39

Ross, Christopher A., Jonathan D. Wood, Gabriele Schilling, et al. "Polyglutamine pathogenesis." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 354, no. 1386 (1999): 1005–11. http://dx.doi.org/10.1098/rstb.1999.0452.

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Abstract (sommario):

An increasing number of neurodegenerative disorders have been found to be caused by expanding CAG triplet repeats that code for polyglutamine. Huntington's disease (HD) is the most common of these disorders and dentato-rubral-pallidoluysian atrophy (DRPLA) is very similar to HD, but is caused by mutation in a different gene, making them good models to study. In this review, we will concentrate on the roles of protein aggregation, nuclear localization and proteolytic processing in disease pathogenesis. In cell model studies of HD, we have found that truncated N-terminal portions of huntingtin (

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40

Cantor, Jerome O., and Gerard M. Turino. "COPD Pathogenesis." Chest 155, no. 2 (2019): 266–71. http://dx.doi.org/10.1016/j.chest.2018.07.030.

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41

Dushku, Nicholas. "Pterygia Pathogenesis." Archives of Ophthalmology 119, no. 5 (2001): 695. http://dx.doi.org/10.1001/archopht.119.5.695.

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42

Ahn, Christine S., and William W. Huang. "Rosacea Pathogenesis." Dermatologic Clinics 36, no. 2 (2018): 81–86. http://dx.doi.org/10.1016/j.det.2017.11.001.

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43

Mykola, Kolesnyk, Stepanova Natalia, Driianska Victoria, Rudenko Ada, Kruglikov Vadim, and Lebid Larisa. "Pathogenesis of pyelonephritis: what we know and what not." Ukrainian Journal of Nephrology and Dialysis 3(31) (September 27, 2011): 34–46. https://doi.org/10.5281/zenodo.3352575.

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The aim of this work was an attempt to assess the state address some of the problems of the pathogenesis of pyelonephritis based on analysis of modern scientific literature on the basis of own research.

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44

Rohilla, Lakshita, Karandeep Singh, and Abhijit Garg. "Intranasal Malignantmelanoma: Pathogenesis Epidemiology and Clinical Insight." International Journal of Science and Research (IJSR) 12, no. 9 (2023): 740–44. http://dx.doi.org/10.21275/sr23907100157.

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45

K.Z., Kadirov, and Isroilov R.I. "ETHIO-PATHOGENESIS OF POUND DESTROYMENT IN CHILDREN." American Journal of Medical Sciences and Pharmaceutical Research 6, no. 2 (2024): 75–82. http://dx.doi.org/10.37547/tajmspr/volume06issue02-10.

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Infectious diseases are one of the most serious problems because they are the most widespread and the leading cause of death. Although significant advances have been made over the past decades in addressing this problem, this statement is still true today. Sepsis is the leading cause of death in critical care patients. It develops in 750000 people each year and more than 210000 of them die. Progress in the study of the pathophysiology and genetic basis of the individual response to sepsis has changed the common understanding of this syndrome, and some therapeutic interventions used in recent y

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46

Borozdenko, Denis A., Vladislava I. Bogorodova, Nina M. Kiseleva, and Vadim V. Negrebetsky. "Parkinson’s disease: epidemiology and pathogenesis." Medical Journal of the Russian Federation 27, no. 2 (2021): 183–94. http://dx.doi.org/10.17816/0869-2106-2021-27-2-183-194.

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This review presents data on the etiology, epidemiology, and pathogenesis of Parkinsons disease from National Center for Biotechnology Information (NCBI), eLibrary, CyberLeninka, and from monographs and textbooks. The prevalence, classification, genetic variability, main pathogenetic links, and potential disease development mechanisms are described. Both classic Parkinsons disease and variable manifestations of parkinsonism are considered. The factors that contribute to disease progression and inhibit its development are described. The main hypotheses of the pathogenetic mechanisms of Parkinso

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47

Brown, Noah A., and Bryan L. Betz. "Ameloblastoma: A Review of Recent Molecular Pathogenetic Discoveries." Biomarkers in Cancer 7s2 (January 2015): BIC.S29329. http://dx.doi.org/10.4137/bic.s29329.

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Ameloblastoma is an odontogenic neoplasm whose molecular pathogenesis has only recently been elucidated. The discovery of recurrent activating mutations in FGFR2, BRAF, and RAS in a large majority of ameloblastomas has implicated dysregulation of MAPK pathway signaling as a critical step in the pathogenesis of this tumor. Some degree of controversy exists regarding the role of mutations affecting the sonic hedgehog (SHH) pathway, specifically Smoothened (SMO), which have been postulated to serve as either an alternative pathogenetic mechanism or secondary mutations. Here, we review recent adva

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48

Plamen Todorov Titorenkov and Milena Dimitrova Encheva. "Asthma, COPD, the "asthma-COPD" syndrome and severe asthma - relatives or "incidental strangers"?" World Journal of Advanced Research and Reviews 24, no. 3 (2024): 1354–63. https://doi.org/10.30574/wjarr.2024.24.3.3828.

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Objective: Airways obstructive diseases (asthma, COPD, asthma-COPD overlap (ACO) and severe asthma) are different entities characterized by airflow obstruction. They may share common pathogenetic mechanisms allowing new therapeutic options for patients. Methods: A review of the current literature is performed to investigate the common pathogenetic mechanisms among these medical conditions. Results: Different specific cytokones are involved in both asthma and COPD pathogenesis, while ACO shares some of these cytokines placing the condition between the two entities. The cytokines regulate accumu

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Plamen, Todorov Titorenkov, and Dimitrova Encheva Milena. "Asthma, COPD, the "asthma-COPD" syndrome and severe asthma - relatives or "incidental strangers"?" World Journal of Advanced Research and Reviews 24, no. 3 (2024): 1354–63. https://doi.org/10.5281/zenodo.15193400.

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Abstract (sommario):

<strong>Objective</strong>: Airways obstructive diseases (asthma, COPD, asthma-COPD overlap (ACO) and severe asthma) are different entities characterized by airflow obstruction. They may share common pathogenetic mechanisms allowing new therapeutic options for patients. <strong>Methods</strong>: A review of the current literature is performed to investigate the common pathogenetic mechanisms among these medical conditions. <strong>Results</strong>: Different specific cytokones are involved in both asthma and COPD pathogenesis, while ACO shares some of these cytokines placing the condition betw

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Varghese, Smitha Ancy. "Secondary lymphedema: Pathogenesis." Journal of Skin and Sexually Transmitted Diseases 3 (April 6, 2021): 7–15. http://dx.doi.org/10.25259/jsstd_3_2020.

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Secondary lymphedema follows an acquired defect in the lymphatic system. The common causes leading to a defective lymphatic function include infection, inflammation, malignancy, trauma, obesity, immobility, and therapeutic interventions. Understanding the pathogenesis of lymphedema is of prime importance in offering effective treatment. The pathogenetic mechanisms such as lymphatic valvular insufficiency, obliteration/ disruption of lymphatic vessels, and decreased lymphatic contractility aggravate lymphatic hypertension and lymphstasis. Accumulation of lymph, interstitial fluid, proteins, and

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