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Articoli di riviste sul tema "Protective Immunity"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 25 luglio 2025

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1

Burger, Eva. "Paracoccidioidomycosis Protective Immunity." Journal of Fungi 7, no. 2 (2021): 137. http://dx.doi.org/10.3390/jof7020137.

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Protective immunity against Paracoccidioides consists of a stepwise activation of numerous effector mechanisms that comprise many cellular and soluble components. At the initial phase of non-specific innate immunity, resistance against Paracoccidioides comes from phagocytic polymorphonuclear neutrophils, natural killer (NK) cells and monocytes, supplemented by soluble factors such as cytokines and complement system components. Invariant receptors (Toll-like receptors (TLRs), Dectins) which are present in cells of the immune system, detect patterns present in Paracoccidioides (but not in the ho

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2

Hoerauf, Achim, and Cathy Steel. "3.2 PROTECTIVE IMMUNITY—VACCINES." American Journal of Tropical Medicine and Hygiene 71, no. 5_suppl (2004): 34–36. http://dx.doi.org/10.4269/ajtmh.2004.71.5_suppl.0700034.

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3

Cox, R. A., and D. M. Magee. "Protective immunity in coccidioidomycosis." Research in Immunology 149, no. 4-5 (1998): 417–28. http://dx.doi.org/10.1016/s0923-2494(98)80765-7.

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4

Huang, Shou-Jie, Xiao-Hui Liu, Jun Zhang, and Mun-Hon Ng. "Protective immunity against HEV." Current Opinion in Virology 5 (April 2014): 1–6. http://dx.doi.org/10.1016/j.coviro.2013.10.003.

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5

Zinkernagel, Rolf M. "Immunological memory ≠ protective immunity." Cellular and Molecular Life Sciences 69, no. 10 (2012): 1635–40. http://dx.doi.org/10.1007/s00018-012-0972-y.

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6

Lin, Wen-Hsuan, and Diane E. Griffin. "138 Understanding protective immunity." JAIDS Journal of Acquired Immune Deficiency Syndromes 65 (April 2014): 58. http://dx.doi.org/10.1097/01.qai.0000446718.70725.4b.

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7

Watson, J. D. "Leprosy: understanding protective immunity." Immunology Today 10, no. 7 (1989): 218–21. http://dx.doi.org/10.1016/0167-5699(89)90253-3.

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8

Watanabe, Mineo, and Masaaki Nagai. "Role of Systemic and Mucosal Immune Responses in Reciprocal Protection against Bordetella pertussis and Bordetella parapertussis in a Murine Model of Respiratory Infection." Infection and Immunity 71, no. 2 (2003): 733–38. http://dx.doi.org/10.1128/iai.71.2.733-738.2003.

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ABSTRACT The roles of systemic humoral immunity, cell-mediated immunity, and mucosal immunity in reciprocal protective immunity against Bordetella pertussis and Bordetella parapertussis were examined by using a murine model of respiratory infection. Passive immunization with serum from mice infected with B. pertussis established protective immunity against B. pertussis but not against B. parapertussis. Protection against B. parapertussis was induced in mice that had been injected with serum from mice infected with B. parapertussis but not from mice infected with B. pertussis. Adoptive transfer

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9

Blutt, Sarah E., Kelly L. Warfield, Mary K. Estes, and Margaret E. Conner. "Differential Requirements for T Cells in Viruslike Particle- and Rotavirus-Induced Protective Immunity." Journal of Virology 82, no. 6 (2008): 3135–38. http://dx.doi.org/10.1128/jvi.01727-07.

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ABSTRACT Correlates of protection from rotavirus infection are controversial. We compared the roles of B and T lymphocytes in protective immunity induced either by intranasally administered nonreplicating viruslike particles or inactivated virus or by orally administered murine rotavirus. We found that protection induced by nonreplicating vaccines requires CD4+ T cells and CD40/CD40L. In contrast, T cells were not required for short-term protective immunity induced by infection, but both T-cell-dependent and -independent mechanisms contributed to long-term maintenance of protection. Our findin

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10

Mokaya, Jolynne, Derick Kimathi, Teresa Lambe, and George M. Warimwe. "What Constitutes Protective Immunity Following Yellow Fever Vaccination?" Vaccines 9, no. 6 (2021): 671. http://dx.doi.org/10.3390/vaccines9060671.

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Yellow fever (YF) remains a threat to global health, with an increasing number of major outbreaks in the tropical areas of the world over the recent past. In light of this, the Eliminate Yellow Fever Epidemics Strategy was established with the aim of protecting one billion people at risk of YF through vaccination by the year 2026. The current YF vaccine gives excellent protection, but its use is limited by shortages in supply due to the difficulties in producing the vaccine. There are good grounds for believing that alternative fractional dosing regimens can produce strong protection and overc

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11

Doolan, Denise L., and Stephen L. Hoffman. "Pre–erythrocytic–stage immune effector mechanisms in Plasmodium spp. infections." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 352, no. 1359 (1997): 1361–67. http://dx.doi.org/10.1098/rstb.1997.0121.

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The potent protective immunity against malaria induced by immunization of mice and humans with radiation–attenuated Plasmodium spp. sporozoites is thought to be mediated primarily by T–cell responses directed against infected hepatocytes. This has led to considerable efforts to develop subunit vaccines that duplicate this protective immunity, but a universally effective vaccine is still not available and in vitro correlates of protective immunity have not been established. Contributing to this delay has been a lack of understanding of the mechanisms responsible for the protection. There are no

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12

Igarashi, Ikuo, Reiko Suzuki, Seiji Waki, et al. "Roles of CD4+ T Cells and Gamma Interferon in Protective Immunity against Babesia microtiInfection in Mice." Infection and Immunity 67, no. 8 (1999): 4143–48. http://dx.doi.org/10.1128/iai.67.8.4143-4148.1999.

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ABSTRACT Babesia microti produces a self-limiting infection in mice, and recovered mice are resistant to reinfection. In the present study, the role of T cells in protective immunity against challenge infection was examined. BALB/c mice which recovered from primary infection showed strong protective immunity against challenge infection. In contrast, nude mice which failed to control the primary infection and were cured with an antibabesial drug did not show protection against challenge infection. Treatment of immune mice with anti-CD4 monoclonal antibody (MAb) diminished the protective immunit

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13

Roy, Sreeja, Clare M. Williams, Julian Pardo, Danushka K. Wijesundara, and Yoichi Furuya. "Impact of Pre-Existing Immunity on Live Attenuated Influenza Vaccine-Induced Cross-Protective Immunity." Vaccines 8, no. 3 (2020): 459. http://dx.doi.org/10.3390/vaccines8030459.

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The efficacy of the intranasally (i.n.) delivered live attenuated influenza vaccine (LAIV) is variable and, in some seasons, suboptimal. In this study, we report that LAIV exhibits cross-protective efficacy in mice, potentially associated with cellular immunity as opposed to antigen-specific antibody responses. However, pre-exposure to the intramuscularly (i.m.) delivered inactivated influenza vaccine (IIV) severely impaired LAIV-induced cross-protection against heterologous challenge, potentially by inhibiting replication of LAIV. Our findings suggest that pre-existing immunity afforded by II

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14

Raeven, René H. M., Naomi van Vlies, Merijn L. M. Salverda, et al. "The Role of Virulence Proteins in Protection Conferred by Bordetella pertussis Outer Membrane Vesicle Vaccines." Vaccines 8, no. 3 (2020): 429. http://dx.doi.org/10.3390/vaccines8030429.

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The limited protective immunity induced by acellular pertussis vaccines demands development of novel vaccines that induce broader and longer-lived immunity. In this study, we investigated the protective capacity of outer membrane vesicle pertussis vaccines (omvPV) with different antigenic composition in mice to gain insight into which antigens contribute to protection. We showed that total depletion of virulence factors (bvg(-) mode) in omvPV led to diminished protection despite the presence of high antibody levels. Antibody profiling revealed overlap in humoral responses induced by vaccines i

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15

Stingl, G. "PL04.3 Protective and Non-Protective Immunity in STIs." Sexually Transmitted Infections 89, Suppl 1 (2013): A4.3—A4. http://dx.doi.org/10.1136/sextrans-2013-051184.0011.

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16

Harvill, Eric T., Manuel Osorio, Crystal L. Loving, Gloria M. Lee, Vanessa K. Kelly, and Tod J. Merkel. "Anamnestic Protective Immunity to Bacillus anthracis Is Antibody Mediated but Independent of Complement and Fc Receptors." Infection and Immunity 76, no. 5 (2008): 2177–82. http://dx.doi.org/10.1128/iai.00647-07.

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ABSTRACT The threat of bioterrorist use of Bacillus anthracis has focused urgent attention on the efficacy and mechanisms of protective immunity induced by available vaccines. However, the mechanisms of infection-induced immunity have been less well studied and defined. We used a combination of complement depletion along with immunodeficient mice and adoptive transfer approaches to determine the mechanisms of infection-induced protective immunity to B. anthracis. B- or T-cell-deficient mice lacked the complete anamnestic protection observed in immunocompetent mice. In addition, T-cell-deficien

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17

Montgomery, Christopher P., Melvin Daniels, Fan Zhao, Maria-Luisa Alegre, Anita S. Chong, and Robert S. Daum. "Protective Immunity against Recurrent Staphylococcus aureus Skin Infection Requires Antibody and Interleukin-17A." Infection and Immunity 82, no. 5 (2014): 2125–34. http://dx.doi.org/10.1128/iai.01491-14.

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ABSTRACTAlthough many microbial infections elicit an adaptive immune response that can protect against reinfection, it is generally thought thatStaphylococcus aureusinfections fail to generate protective immunity despite detectable T and B cell responses. No vaccine is yet proven to preventS. aureusinfections in humans, and efforts to develop one have been hampered by a lack of animal models in which protective immunity occurs. Our results describe a novel mouse model of protective immunity against recurrent infection, in whichS. aureusskin and soft tissue infection (SSTI) strongly protected a

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18

Silva, Ediane B., Andrew Goodyear, Marjorie D. Sutherland, et al. "Correlates of Immune Protection following Cutaneous Immunization with an Attenuated Burkholderia pseudomallei Vaccine." Infection and Immunity 81, no. 12 (2013): 4626–34. http://dx.doi.org/10.1128/iai.00915-13.

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ABSTRACTInfections with the Gram-negative bacteriumBurkholderia pseudomallei(melioidosis) are associated with high mortality, and there is currently no approved vaccine to prevent the development of melioidosis in humans. Infected patients also do not develop protective immunity to reinfection, and some individuals will develop chronic, subclinical infections withB. pseudomallei. At present, our understanding of what constitutes effective protective immunity againstB. pseudomalleiinfection remains incomplete. Therefore, we conducted a study to elucidate immune correlates of vaccine-induced pro

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19

Goldman, Gustavo H., Yves Delneste, and Nicolas Papon. "Fungal Polysaccharides Promote Protective Immunity." Trends in Microbiology 29, no. 5 (2021): 379–81. http://dx.doi.org/10.1016/j.tim.2021.02.004.

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20

Parodi, Cecilia, Angel Marcelo Padilla, and Miguel Angel Basombrío. "Protective immunity against Trypanosoma cruzi." Memórias do Instituto Oswaldo Cruz 104, suppl 1 (2009): 288–94. http://dx.doi.org/10.1590/s0074-02762009000900038.

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21

Mulupuri, Prasad, Tania Gourley, Adam Popkowski, et al. "Protective Immunity in Transplant Recipients." FASEB Journal 22, S2 (2008): 532. http://dx.doi.org/10.1096/fasebj.22.2_supplement.532.

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22

ROSS, A. CATHARINE. "Vitamin A and Protective Immunity." Nutrition Today 27, no. 4 (1992): 18–26. http://dx.doi.org/10.1097/00017285-199207000-00005.

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23

Lee, Chi-Jen, Lucia H. Lee, and Carl E. Frasch. "Protective Immunity of Pneumococcal Glycoconjugates." Critical Reviews in Microbiology 29, no. 4 (2003): 333–49. http://dx.doi.org/10.1080/713608018.

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24

Bachmann, Martin F., and Manfred Kopf. "Balancing protective immunity and immunopathology." Current Opinion in Immunology 14, no. 4 (2002): 413–19. http://dx.doi.org/10.1016/s0952-7915(02)00363-1.

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25

Huster, Katharina M., Christian Stemberger, and Dirk H. Busch. "Protective immunity towards intracellular pathogens." Current Opinion in Immunology 18, no. 4 (2006): 458–64. http://dx.doi.org/10.1016/j.coi.2006.05.008.

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26

Kapil, Parul, and Tod J. Merkel. "Pertussis vaccines and protective immunity." Current Opinion in Immunology 59 (August 2019): 72–78. http://dx.doi.org/10.1016/j.coi.2019.03.006.

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27

Nutman, Thomas B. "Protective immunity in lymphatic filariasis." Experimental Parasitology 68, no. 2 (1989): 248–52. http://dx.doi.org/10.1016/0014-4894(89)90106-9.

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28

Hewitt, Eric W., and Gillian E. Dugan. "Virus subversion of protective immunity." Current Allergy and Asthma Reports 4, no. 5 (2004): 365–70. http://dx.doi.org/10.1007/s11882-004-0085-2.

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29

Dranoff, Glenn. "Targets of Protective Tumor Immunity." Annals of the New York Academy of Sciences 1174, no. 1 (2009): 74–80. http://dx.doi.org/10.1111/j.1749-6632.2009.04938.x.

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30

Nilsson, Jan. "Regulating Protective Immunity in Atherosclerosis." Circulation Research 96, no. 4 (2005): 395–97. http://dx.doi.org/10.1161/01.res.0000159183.88730.79.

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31

Fikrig, E., F. S. Kantor, S. W. Barthold, and R. A. Flavell. "Protective immunity in lyme borreliosis." Parasitology Today 9, no. 4 (1993): 129–31. http://dx.doi.org/10.1016/0169-4758(93)90176-g.

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32

Ponomarenko, D. G., M. V. Kostyuchenko, E. L. Rakitina, et al. "Quantitative analysis of protective T cell immunity against brucellosis." Medical Immunology (Russia) 26, no. 1 (2023): 211–20. http://dx.doi.org/10.15789/1563-0625-qao-2604.

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The results of study relationship between antigen reactivity of T-lymphocyte population under ex vivo conditions and the intensity of protective post-vaccination immunity to causative agent of brucellosis are presented. Тaking into account the peculiarities of immunopathogenesis brucellosis and prevailing role of adaptive T-cell immunity to protect against the causative agent of infection, possibility predictive evaluation of protective immunity against brucellosis using CAST-tests is considered as the most important aspect of brucellosis problems. There is an obvious need for an ex vivo corre

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33

Seo, Sang Heui, Malik Peiris, and Robert G. Webster. "Protective Cross-Reactive Cellular Immunity to Lethal A/Goose/Guangdong/1/96-Like H5N1 Influenza Virus Is Correlated with the Proportion of Pulmonary CD8+ T Cells Expressing Gamma Interferon." Journal of Virology 76, no. 10 (2002): 4886–90. http://dx.doi.org/10.1128/jvi.76.10.4886-4890.2002.

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ABSTRACT A/Goose/Guangdong/1/96-like H5N1 influenza viruses now circulating in southeastern China differ genetically from the H5N1 viruses transmitted to humans in 1997 but were their precursors. Here we show that the currently circulating H9N2 influenza viruses provide chickens with cross-reactive protective immunity against the currently circulating H5N1 influenza viruses and that this protective immunity is closely related to the percentage of pulmonary CD8+ T cells expressing gamma interferon (IFN-γ). In vivo depletion of T-cell subsets showed that the cross-reactive immunity was mediated

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34

Tomic, Adriana, Andrew J. Pollard, and Mark M. Davis. "Systems Immunology: Revealing Influenza Immunological Imprint." Viruses 13, no. 5 (2021): 948. http://dx.doi.org/10.3390/v13050948.

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Understanding protective influenza immunity and identifying immune correlates of protection poses a major challenge and requires an appreciation of the immune system in all of its complexity. While adaptive immune responses such as neutralizing antibodies and influenza-specific T lymphocytes are contributing to the control of influenza virus, key factors of long-term protection are not well defined. Using systems immunology, an approach that combines experimental and computational methods, we can capture the systems-level state of protective immunity and reveal the essential pathways that are

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35

Hogarth, Philip J., Mark J. Taylor, and Albert E. Bianco. "IL-5-Dependent Immunity to Microfilariae Is Independent of IL-4 in a Mouse Model of Onchocerciasis." Journal of Immunology 160, no. 11 (1998): 5436–40. http://dx.doi.org/10.4049/jimmunol.160.11.5436.

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Abstract Th2 lymphocyte responses under the control of IL-4 and IL-5 are frequently associated with protective responses to parasitic helminths. Studies on the role of these cytokines in acquired resistance to parasitic nematodes indicate that, in the case of gastrointestinal nematodes, immunity is mediated by IL-4, while immunity to tissue-dwelling nematodes is dependent on IL-5. Here we investigate the role of IL-5 and eosinophils in protective immunity to Onchocerca microfilariae in IL-4-deficient mice. In the absence of IL-4, and despite the up-regulation of Th1-type responses, immunity re

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Blander, S. J., and M. A. Horwitz. "Vaccination with the major secretory protein of Legionella induces humoral and cell-mediated immune responses and protective immunity across different serogroups of Legionella pneumophila and different species of Legionella." Journal of Immunology 147, no. 1 (1991): 285–91. http://dx.doi.org/10.4049/jimmunol.147.1.285.

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Abstract In a previous study, we demonstrated that immunization of guinea pigs with the major secretory protein (MSP) of Legionella pneumophila, serogroup 1 induced humoral and cell-mediated immune responses to MSP and protective immunity against lethal aerosol challenge with this serogroup of L. pneumophila. Although serogroup 1 L. pneumophila cause most cases of Legionnaires' disease, other serogroups of L. pneumophila and species of Legionella cause many cases. In this study, we have examined if immunization with MSP induces humoral and cell-mediated immune responses and protective immunity

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Msema Bwire, George, and Kennedy Daniel Mwambete. "Immunological Perspectives of sub-Saharan Populations under Prophylaxis against Malaria." Immunology and Inflammation Diseases Therapy 3, no. 1 (2019): 01–03. http://dx.doi.org/10.31579/2637-8876/013.

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Immunity is the state of protection against infectious disease conferred either through an immune response generated by immunization or previous infection. Generated immune responses may be long lasting even lifelong or gives immediate, but short-lived protection. In malaria-endemic areas, young children and pregnant women are particularly susceptible to malaria, but with exposures protective immunity against malaria develop although sterile immunity is never achieved. Assuring protection to vulnerable populations, prophylaxis against malaria is advocated to pregnancy women and sickle cell dis

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Sun, Lu, Lufei Wang, Bethany B. Moore, et al. "IL-17: Balancing Protective Immunity and Pathogenesis." Journal of Immunology Research 2023 (August 12, 2023): 1–9. http://dx.doi.org/10.1155/2023/3360310.

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The biological role of interleukin 17 (IL-17) has been explored during recent decades and identified as a pivotal player in coordinating innate and adaptive immune responses. Notably, IL-17 functions as a double-edged sword with both destructive and protective immunological roles. While substantial progress has implicated unrestrained IL-17 in a variety of infectious diseases or autoimmune conditions, IL-17 plays an important role in protecting the host against pathogens and maintaining physiological homeostasis. In this review, we describe canonical IL-17 signaling mechanisms promoting neutro

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Darby, Matthew G., Alisha Chetty, Dunja Mrjden, et al. "Pre-conception maternal helminth infection transfers via nursing long-lasting cellular immunity against helminths to offspring." Science Advances 5, no. 5 (2019): eaav3058. http://dx.doi.org/10.1126/sciadv.aav3058.

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Maternal immune transfer is the most significant source of protection from early-life infection, but whether maternal transfer of immunity by nursing permanently alters offspring immunity is poorly understood. Here, we identify maternal immune imprinting of offspring nursed by mothers who had a pre-conception helminth infection. Nursing of pups by helminth-exposed mothers transferred protective cellular immunity to these offspring against helminth infection. Enhanced control of infection was not dependent on maternal antibody. Protection associated with systemic development of protective type

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Wu, Shipo, Zhe Zhang, Rui Yu, et al. "Intramuscular Delivery of Adenovirus Serotype 5 Vector Expressing Humanized Protective Antigen Induces Rapid Protection against Anthrax That May Bypass Intranasally Originated Preexisting Adenovirus Immunity." Clinical and Vaccine Immunology 21, no. 2 (2013): 156–64. http://dx.doi.org/10.1128/cvi.00560-13.

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ABSTRACTDeveloping an effective anthrax vaccine that can induce a rapid and sustained immune response is a priority for the prevention of bioterrorism-associated anthrax infection. Here, we developed a recombinant replication-deficient adenovirus serotype 5-based vaccine expressing the humanized protective antigen (Ad5-PAopt). A single intramuscular injection of Ad5-PAopt resulted in rapid and robust humoral and cellular immune responses in Fisher 344 rats. Animals intramuscularly inoculated with a single dose of 108infectious units of Ad5-PAopt achieved 100% protection from challenge with 10

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Kollmann, Tobias R., Arnaud Marchant, and Sing Sing Way. "Vaccination strategies to enhance immunity in neonates." Science 368, no. 6491 (2020): 612–15. http://dx.doi.org/10.1126/science.aaz9447.

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Neonates are particularly susceptible to infection. This vulnerability occurs despite their responsiveness to most vaccines. However, current vaccines do not target the pathogens responsible for most of the severe neonatal infections, and the time it takes to induce protective pathogen-specific immunity after vaccination limits protection in the first days to weeks of life. Alternative strategies include using vaccines to broadly stimulate neonatal immunity in a pathogen-agnostic fashion or vaccinating women during pregnancy to induce protective antibodies that are vertically transferred to of

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Zarling, Stasya, and Urszula Krzych. "Increased apoptosis of intrahepatic central memory CD8 T cells in the absence of IL-15 is attributed to the loss of long-lasting protection against malaria infection (P3334)." Journal of Immunology 190, no. 1_Supplement (2013): 134.20. http://dx.doi.org/10.4049/jimmunol.190.supp.134.20.

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Abstract Ag-specific memory T cell responses elicited by infections or vaccinations are inextricably linked to long-lasting protective immunity. Studies of protective immunity amongst residents of malaria endemic areas indicate that memory responses to Plasmodia antigens are not adequately developed or maintained. In contrast, multiple exposures to radiation-attenuated Plasmodia sporozoites (γ-spz) induce long-lasting protective immunity to experimental sporozoite challenge. In the P. berghei (Pb) γ-spz mouse model, lasting protection is associated with the presence of intrahepatic CD8 T cells

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Li, Zhuofan, Yiwen Zhao, Yibo Li, and Xinyuan Chen. "Adjuvantation of Influenza Vaccines to Induce Cross-Protective Immunity." Vaccines 9, no. 2 (2021): 75. http://dx.doi.org/10.3390/vaccines9020075.

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Influenza poses a huge threat to global public health. Influenza vaccines are the most effective and cost-effective means to control influenza. Current influenza vaccines mainly induce neutralizing antibodies against highly variable globular head of hemagglutinin and lack cross-protection. Vaccine adjuvants have been approved to enhance seasonal influenza vaccine efficacy in the elderly and spare influenza vaccine doses. Clinical studies found that MF59 and AS03-adjuvanted influenza vaccines could induce cross-protective immunity against non-vaccine viral strains. In addition to MF59 and AS03

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Tang, Jinyi, Arka Chaudhuri, Panke Qu, et al. "Respiratory mucosal immunity against SARS-CoV-2 after vaccination and infection." Journal of Immunology 212, no. 1_Supplement (2024): 1559_5071. http://dx.doi.org/10.4049/jimmunol.212.supp.1559.5071.

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Abstract The current COVID-19 mRNA vaccine is effective in systemic immune memory but lacks in generating mucosal anti-SARS-CoV-2 immunity. While prior SARS-CoV-2 infection establishes respiratory immunity, its strain-specific nature and limited duration pose challenges for sustained protection. Recent research has shown that hybrid immunity, a combination of vaccination and infection, results in a more robust, durable, and broadly reactive immune response in circulation compared to vaccination or infection alone. However, the characteristics and protective mechanisms of hybrid immunity in the

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Sparber, Florian, and Salomé LeibundGut-Landmann. "Interleukin-17 in Antifungal Immunity." Pathogens 8, no. 2 (2019): 54. http://dx.doi.org/10.3390/pathogens8020054.

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The field of IL-17 biology has received much attention over the last decade owing to the pathogenic role of this cytokine in psoriasis and other autoinflammatory disorders and the successful implementation of IL-17-targeting therapies in patients suffering from these diseases. IL-17-mediated pathologies are contrasted by the important host beneficial effects of this cytokine. IL-17 is essential for regulating microbial colonization in barrier tissues. Rare congenital defects in the IL-17 pathway exemplify the relevance of IL-17 in protective immunity against the opportunistic fungal pathogen C

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Chen, Chen, Erin J. van Schaik, Anthony E. Gregory, et al. "Chemokine Receptor 7 Is Essential for Coxiella burnetii Whole-Cell Vaccine-Induced Cellular Immunity but Dispensable for Vaccine-Mediated Protective Immunity." Journal of Infectious Diseases 220, no. 4 (2019): 624–34. http://dx.doi.org/10.1093/infdis/jiz146.

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Abstract Background Protective immunity against Coxiella burnetii infection is conferred by vaccination with virulent (PI-WCV), but not avirulent (PII-WCV) whole-cell inactivated bacterium. The only well-characterized antigenic difference between virulent and avirulent C. burnetii is they have smooth and rough lipopolysaccharide (LPS), respectively. Methods Mice were vaccinated with PI-WCV and PII-WCV. Humoral and cellular responses were evaluated using protein chip microarrays and ELISpots, respectively. Dendritic cell (DC) maturation after stimulation with PI-WVC and PII-WVC was evaluated us

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Shodiyeva, Dildora G'iyosovna, and Xushnud Toshtemir o'g'li Xoljigitov. "HUMAN IMMUNITY." GOLDEN BRAIN 1, no. 5 (2023): 174–80. https://doi.org/10.5281/zenodo.7677069.

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<em>The purpose of our scientific work was about the protective functions of the human body against foreign microorganisms and foreign genes and the specific features of this system. we studied immune damage.</em>

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Rey-Ladino, Jose, Kasra M. Koochesfahani, Michelle L. Zaharik, Caixia Shen, and Robert C. Brunham. "A Live and Inactivated Chlamydia trachomatis Mouse Pneumonitis Strain Induces the Maturation of Dendritic Cells That Are Phenotypically and Immunologically Distinct." Infection and Immunity 73, no. 3 (2005): 1568–77. http://dx.doi.org/10.1128/iai.73.3.1568-1577.2005.

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ABSTRACT The intracellular bacterial pathogen Chlamydia trachomatis is a major cause of sexually transmitted disease worldwide. While protective immunity does appear to develop following natural chlamydial infection in humans, early vaccine trials using heat-killed C. trachomatis resulted in limited and transient protection with possible enhanced disease during follow-up. Thus, immunity following natural infection with live chlamydia may differ from immune responses induced by immunization with inactivated chlamydia. To study this differing immunology, we used murine bone marrow-derived dendri

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Kawabata, Yuko, Heiichiro Udono, Kiri Honma, et al. "Merozoite Surface Protein 1-Specific Immune Response Is Protective against Exoerythrocytic Forms of Plasmodium yoelii." Infection and Immunity 70, no. 11 (2002): 6075–82. http://dx.doi.org/10.1128/iai.70.11.6075-6082.2002.

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ABSTRACT One of the difficulties in developing an effective malaria vaccine is the antigenic change of the parasite during the life cycle. It is desirable that vaccine-induced protective immunity be effective at different stages of parasite development. Merozoite surface protein 1 (MSP1) is a candidate vaccine antigen against blood-stage malaria, but it is also expressed in the exoerythrocytic forms. It was not known, however, whether the anti-MSP1 immune response is effective against the liver-stage malaria parasite. We generated a recombinant protein of MSP1 fused to heat-shock cognate prote

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Sparber, Florian, and Salomé LeibundGut-Landmann. "Interleukin-17 in Antifungal Immunity." Pathogens 8, no. 2 (2019): 54. https://doi.org/10.5281/zenodo.13534890.

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(Uploaded by Plazi for the Bat Literature Project) The field of IL-17 biology has received much attention over the last decade owing to the pathogenic role of this cytokine in psoriasis and other autoinflammatory disorders and the successful implementation of IL-17-targeting therapies in patients suffering from these diseases. IL-17-mediated pathologies are contrasted by the important host beneficial effects of this cytokine. IL-17 is essential for regulating microbial colonization in barrier tissues. Rare congenital defects in the IL-17 pathway exemplify the relevance of IL-17 in protective i

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