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Letteratura scientifica selezionata sul tema "Sodium/calcium exchanger (NCX)"

Autore: Grafiati

Pubblicato: 4 giugno 2021

Ultima modifica: 18 febbraio 2022

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Articoli di riviste sul tema "Sodium/calcium exchanger (NCX)"

Chovancova, Barbora, Veronika Liskova, Petr Babula, and Olga Krizanova. "Role of Sodium/Calcium Exchangers in Tumors." Biomolecules 10, no. 9 (August 31, 2020): 1257. http://dx.doi.org/10.3390/biom10091257.

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The sodium/calcium exchanger (NCX) is a unique calcium transport system, generally transporting calcium ions out of the cell in exchange for sodium ions. Nevertheless, under special conditions this transporter can also work in a reverse mode, in which direction of the ion transport is inverted—calcium ions are transported inside the cell and sodium ions are transported out of the cell. To date, three isoforms of the NCX have been identified and characterized in humans. Majority of information about the NCX function comes from isoform 1 (NCX1). Although knowledge about NCX function has evolved

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Chernysh, Olga, Madalina Condrescu, and John P. Reeves. "Sodium-dependent inactivation of sodium/calcium exchange in transfected Chinese hamster ovary cells." American Journal of Physiology-Cell Physiology 295, no. 4 (October 2008): C872—C882. http://dx.doi.org/10.1152/ajpcell.00221.2008.

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High concentrations of cytosolic Na+ ions induce the time-dependent formation of an inactive state of the Na+/Ca2+ exchanger (NCX), a process known as Na+-dependent inactivation. NCX activity was measured as Ca2+ uptake in fura 2-loaded Chinese hamster ovary (CHO) cells expressing the wild-type (WT) NCX or mutants that are hypersensitive (F223E) or resistant (K229Q) to Na+-dependent inactivation. As expected, 1) Na+-dependent inactivation was promoted by high cytosolic Na+ concentration, 2) the F223E mutant was more susceptible than the WT exchanger to inactivation, whereas the K229Q mutant wa

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Roome, Chris J., Emmet M. Power, and Ruth M. Empson. "Transient reversal of the sodium/calcium exchanger boosts presynaptic calcium and synaptic transmission at a cerebellar synapse." Journal of Neurophysiology 109, no. 6 (March 15, 2013): 1669–80. http://dx.doi.org/10.1152/jn.00854.2012.

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Abstract (sommario):

The sodium/calcium exchanger (NCX) is a widespread transporter that exchanges sodium and calcium ions across excitable membranes. Normally, NCX mainly operates in its “forward” mode, harnessing the electrochemical gradient of sodium ions to expel calcium. During membrane depolarization or elevated internal sodium levels, NCX can instead switch the direction of net flux to expel sodium and allow calcium entry. Such “reverse”-mode NCX operation is frequently implicated during pathological or artificially extended periods of depolarization, not during normal activity. We have used fast calcium im

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Smith, L., and J. B. Smith. "Activation of adenylyl cyclase downregulates sodium/calcium exchanger of arterial myocytes." American Journal of Physiology-Cell Physiology 269, no. 6 (December 1, 1995): C1379—C1384. http://dx.doi.org/10.1152/ajpcell.1995.269.6.c1379.

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Chronic elevation of adenosine 3',5'-cyclic monophosphate (cAMP) is known to inhibit the proliferation of cultured vascular smooth muscle cells. The present findings show that the activation of adenylyl cyclase with forskolin decreased Na+/Ca2+ exchanger (NCX) mRNA and activity. Fetal bovine serum restored NCX transcript and activity. The changes in NCX transcript preceded the changes in NCX activity. Incubation of low-passage immortalized myocytes with forskolin plus 3-isobutyl-1-methylxanthine (IBMX), which inhibits cAMP phosphodiesterase, decreased NCX mRNA by 60% in 6 h and 80% in 24 h. Af

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Münch, Götz, Kai Rosport, Christine Baumgartner, Zhongmin Li, Silvia Wagner, Andreas Bültmann, and Martin Ungerer. "Functional alterations after cardiac sodium-calcium exchanger overexpression in heart failure." American Journal of Physiology-Heart and Circulatory Physiology 291, no. 2 (August 2006): H488—H495. http://dx.doi.org/10.1152/ajpheart.01324.2005.

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The sodium-calcium exchanger (NCX) is discussed as one of the key proteins involved in heart failure. However, the causal role and the extent to which NCX contributes to contractile dysfunction during heart failure are poorly understood. NCX overexpression was induced by infection with an adenovirus coding for NCX, which coexpressed green fluorescence protein (GFP) (AdNCX) by ex vivo gene transfer to nonfailing and failing rabbit cardiomyocytes. Myocardial gene transfer in rabbits in vivo was achieved by adenoviral delivery via aortic cross-clamping. Peak cell shortening of cardiomyocytes was

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Torrente, Angelo G., Rui Zhang, Audrey Zaini, Jorge F. Giani, Jeanney Kang, Scott T. Lamp, Kenneth D. Philipson, and Joshua I. Goldhaber. "Burst pacemaker activity of the sinoatrial node in sodium–calcium exchanger knockout mice." Proceedings of the National Academy of Sciences 112, no. 31 (July 20, 2015): 9769–74. http://dx.doi.org/10.1073/pnas.1505670112.

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In sinoatrial node (SAN) cells, electrogenic sodium–calcium exchange (NCX) is the dominant calcium (Ca) efflux mechanism. However, the role of NCX in the generation of SAN automaticity is controversial. To investigate the contribution of NCX to pacemaking in the SAN, we performed optical voltage mapping and high-speed 2D laser scanning confocal microscopy (LSCM) of Ca dynamics in an ex vivo intact SAN/atrial tissue preparation from atrial-specific NCX knockout (KO) mice. These mice lack P waves on electrocardiograms, and isolated NCX KO SAN cells are quiescent. Voltage mapping revealed disorga

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Haug-Collet, K., B. Pearson, R. Webel, R. T. Szerencsei, R. J. Winkfein, P. P. M. Schnetkamp, and N. J. Colley. "Cloning and Characterization of a Potassium-Dependent Sodium/Calcium Exchanger in Drosophila." Journal of Cell Biology 147, no. 3 (November 1, 1999): 659–70. http://dx.doi.org/10.1083/jcb.147.3.659.

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Sodium/calcium(-potassium) exchangers (NCX and NCKX) are critical for the rapid extrusion of calcium, which follows the stimulation of a variety of excitable cells. To further understand the mechanisms of calcium regulation in signaling, we have cloned a Drosophila sodium/calcium-potassium exchanger, Nckx30C. The overall deduced protein topology for NCKX30C is similar to that of mammalian NCKX, having five membrane-spanning domains in the NH2 terminus separated from six at the COOH-terminal end by a large intracellular loop. We show that NCKX30C functions as a potassium-dependent sodium/calciu

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de Ruijter, Wouter, Ger J. M. Stienen, Jan van Klarenbosch, and Jacob J. de Lange. "Negative and Positive Inotropic Effects of Propofol via L-type Calcium Channels and the Sodium-Calcium Exchanger in Rat Cardiac Trabeculae." Anesthesiology 97, no. 5 (November 1, 2002): 1146–55. http://dx.doi.org/10.1097/00000542-200211000-00019.

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Background Conflicting opinions are present in the literature regarding the origin of the negative inotropic effect of propofol on the myocardium. This study aims to resolve these discrepancies by investigating the inotropic effects of propofol the L-type calcium channels and the sodium-calcium exchanger (NCX). Methods The effect of 20 microg/ml propofol on force development was determined in rat cardiac trabeculae at different pacing frequencies and different extracellular calcium concentrations. Postrest potentiation, sodium withdrawal during quiescence, and the NCX inhibitor KB-R7943 were u

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Padín, Juan-Fernando, José-Carlos Fernández-Morales, Román Olivares, Stefan Vestring, Juan-Alberto Arranz-Tagarro, Enrique Calvo-Gallardo, Ricardo de Pascual, Luís Gandía, and Antonio G. García. "Plasmalemmal sodium-calcium exchanger shapes the calcium and exocytotic signals of chromaffin cells at physiological temperature." American Journal of Physiology-Cell Physiology 305, no. 2 (July 15, 2013): C160—C172. http://dx.doi.org/10.1152/ajpcell.00016.2013.

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The activity of the plasmalemmal Na+/Ca2+ exchanger (NCX) is highly sensitive to temperature. We took advantage of this fact to explore here the effects of the NCX blocker KB-R7943 (KBR) at 22 and 37°C on the kinetics of Ca2+ currents ( ICa), cytosolic Ca2+ ([Ca2+]c) transients, and catecholamine release from bovine chromaffin cells (BCCs) stimulated with high K+, caffeine, or histamine. At 22°C, the effects of KBR on those parameters were meager or nil. However, at 37°C whereby the NCX is moving Ca2+ at a rate fivefold higher than at 22°C, various of the effects of KBR were pronounced, namely

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Loffing, Johannes, Dominique Loffing-Cueni, Victor Valderrabano, Lea Kläusli, Steven C. Hebert, Bernard C. Rossier, Joost G. J. Hoenderop, René J. M. Bindels, and Brigitte Kaissling. "Distribution of transcellular calcium and sodium transport pathways along mouse distal nephron." American Journal of Physiology-Renal Physiology 281, no. 6 (December 1, 2001): F1021—F1027. http://dx.doi.org/10.1152/ajprenal.0085.2001.

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First published August 15, 2001; 10.1152/ajprenal. 00085.2001.—The organization of Na+ and Ca2+ transport pathways along the mouse distal nephron is incompletely known. We revealed by immunohistochemistry a set of Ca2+ and Na+transport proteins along the mouse distal convolution. The thiazide-sensitive Na+-Cl− cotransporter (NCC) characterized the distal convoluted tubule (DCT). The amiloride-sensitive epithelial Na+ channel (ENaC) colocalized with NCC in late DCT (DCT2) and extended to the downstream connecting tubule (CNT) and collecting duct (CD). In early DCT (DCT1), the basolateral Ca2+-e

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Più fonti

Tesi sul tema "Sodium/calcium exchanger (NCX)"

Jeffs, Graham J. "The effect of sodium/calcium exchanger 3 (NCX3) knockout on neuronal survival following global cerebral ischaemia in mice." University of Western Australia. School of Biomedical, Biomolecular and Chemical Sciences, 2007. http://theses.library.uwa.edu.au/adt-WU2008.0063.

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Cerebral ischaemia is a leading cause of disability and death world-wide. The only effective treatments are thrombolytic therapy (plasminogen activator; tPA) and hypothermia (33?C). However, tPA has limited clinical application due to its short therapeutic time window and its specific application in thrombo-embolic stroke. Moderate hypothermia (33?C) is only being used following cardiac arrest in comatose survivors. Hence more treatments are urgently required. The first step in developing new treatments is the identification and characterisation of a potential therapeutic target. Since brain d

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Reilly, Louise. "Palmitoylation of the cardiac sodium-calcium exchanger." Thesis, University of Dundee, 2014. https://discovery.dundee.ac.uk/en/studentTheses/37d8a92d-1536-4a05-85f6-a45f9c41a489.

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Sher, Anna. "Modelling local calcium dynamics and the sodium/calcium exchanger in ventricular myocytes." Thesis, University of Oxford, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.670114.

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Bossuyt, Julie. "Sodium-calcium exchange and caveolins." MU has:, 2002. http://wwwlib.umi.com/cr/mo/fulltext?p3052149.

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Han, C. (Chunlei). "Intracellular calcium stores and sodium-calcium exchanger in cardiac myocytes:experimental and computer simulation study." Doctoral thesis, University of Oulu, 2001. http://urn.fi/urn:isbn:9514265912.

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Abstract Cytosolic Ca2+, [Ca2+]I , has a key role in intracellular signalling during excitation-contraction coupling (E-C coupling) in cardiac myocytes. The sarcoplasmic reticulum (SR) is a main intracellular Ca2+ store and the Na+-Ca2+ exchanger (NaCaX) is a major mechanism to extrude Ca2+ for balancing the Ca2+ influx via L-type Ca2+ channels during excitation. Furthermore, [Ca2+]I also affects the configuration of the action potential (AP). The present study, by combination of animal experiments and computer simulations, investigated the roles of [Ca2+]I, SR and NaCaX in car

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Hung, Hsiao-Yu. "Spatial organization of sodium calcium exchanger and caveolin-3 in developing mammalian ventricular cardiomyocytes." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/791.

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In adult cardiomyocytes, the established mechanism of excitation-contraction coupling is calcium-induced calcium release (CICR) mediated by L-type Ca2+ channels (Cav1.2). Briefly, membrane depolarization opens voltage-gated Cav1.2 to allow for the influx of extracellular Ca2+ into the cytosol. This small sarcolemmal (SL) Ca2+ influx is necessary for triggering a larger release of Ca2+ from the intracellular Ca2+ storage site, the sarcoplasmic reticulum (SR), through the SR Ca2+ release channel also known as the ryanodine receptor (RyR). RyR-mediated release of SR Ca2+ effectively raises the

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Modgi, Amol Polo-Parada Luis. "The role of sodium-calcium exchanger in the electrical activity of embryonic chicken heart." Diss., Columbia, Mo. : University of Missouri--Columbia, 2008. http://hdl.handle.net/10355/6671.

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The entire thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file; a non-technical public abstract appears in the public.pdf file. Title from PDF of title page (University of Missouri--Columbia, viewed on September 25, 2009). Thesis advisor: Dr. Luis Polo Parada. Includes bibliographical references.

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Elliott, Elspeth B. A. "Investigation of factors affecting the sodium/calcium exchanger in a rabbit model of left ventricular dysfunction." Thesis, University of Glasgow, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.433518.

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Guan, Yinzheng. "Blebbistatin Protects Rodent Myocytes from Death in Primary Culture via Inhibiting the Sodium/ Calcium Exchanger and the L-type Calcium Channel." Master's thesis, Temple University Libraries, 2011. http://cdm16002.contentdm.oclc.org/cdm/ref/collection/p245801coll10/id/150014.

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Physiology<br>M.S.<br>Introduction: Cardiac disease is a leading cause of mortabity and morbidity in the developed countries. Cultured cardiac myocytes are widely used for exploring the underlying pathophysiology of cardiac disease. Rodents, especially mice with transgenes or gene ablation, have become popular animal models for heart disease research. However, it has been long recognized that rodent myoyctes die during long-term primary culture, which limits the use of genetically altered myocytes for signaling studies. Blebbistatin (BLB), a myosin II ATPase inhibitor, has been used to protect

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Yuan, Jiaqi. "Investigations of macromolecules and small biomolecules by solution NMR: applications to the intracellular loop structure of the sodium-calcium exchanger and metabolite identification methods." The Ohio State University, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=osu1542630633935356.

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Più fonti

Libri sul tema "Sodium/calcium exchanger (NCX)"

(Editor), Jonathan Lytton, Paul P. M. Schnetkamp (Editor), Larry V. Hryshko (Editor), and M. P. Blaustein (Editor), eds. Cellular and Molecular Physiology of Sodium-Calcium Exchange: Proceedings of the Fourth International Conference (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2002.

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Cellular and Molecular Physiology of Sodium-Calcium Exchange: Proceedings of the Fourth International Conference (Annals of the New York Academy of Sciences, V. 976). New York Academy of Sciences, 2002.

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Andre, Herchuelz, and New York Academy of Sciences, eds. Sodium-calcium exchange and the plasma membrane Ca2+-ATPase in cell function: Fifth international conference. Boston, Mass: Blackwell Pub. on behalf of the New York Academy of Sciences, 2007.

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Herchulez, Mordecai P. Blaustein, Jonathan Lytton, and Kenneth D. Philipson. Sodium-Calcium Exchange and the Plasma Membrane Ca2+-ATPase in Cell Function: Fifth International Conference (Annals of the New York Academy of Sciences). Blackwell Publishing Limited, 2007.

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Capitoli di libri sul tema "Sodium/calcium exchanger (NCX)"

Wang, Jian, Andrew Lindsley, Tony Creazzo, Srinagesh V. Koushik, and Simon J. Conway. "Role of the Sodium-calcium Exchanger (NCX-1) within Splotch (Sp2h) Myocardial Failure." In Cardiovascular Development and Congenital Malformations, 193–95. Malden, Massachusetts, USA: Blackwell Publishing Ltd, 2007. http://dx.doi.org/10.1002/9780470988664.ch48.

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Roome, Chris J., and Ruth M. Empson. "The Contribution of the Sodium-Calcium Exchanger (NCX) and Plasma Membrane Ca2+ ATPase (PMCA) to Cerebellar Synapse Function." In Advances in Experimental Medicine and Biology, 251–63. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_21.

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Yang, Hyun, Kyung-Chul Choi, Eui-Man Jung, Beum-Soo An, Sang-Hwan Hyun, and Eui-Bae Jeung. "Expression and Regulation of Sodium/Calcium Exchangers, NCX and NCKX, in Reproductive Tissues: Do They Play a Critical Role in Calcium Transport for Reproduction and Development?" In Advances in Experimental Medicine and Biology, 109–21. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_10.

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Hurtado, Cecilia, Thane G. Maddaford, and Grant N. Pierce. "Cardiac Sodium–Calcium Exchanger Expression." In Genes and Cardiovascular Function, 43–56. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4419-7207-1_5.

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Scorziello, Antonella, Claudia Savoia, Agnese Secondo, Francesca Boscia, Maria Josè Sisalli, Alba Esposito, Annalisa Carlucci, et al. "New Insights in Mitochondrial Calcium Handling by Sodium/Calcium Exchanger." In Advances in Experimental Medicine and Biology, 203–9. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_17.

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Yang, Ya-Chi, and Lung-Sen Kao. "Regulation of Sodium-Calcium Exchanger Activity by Creatine Kinase." In Advances in Experimental Medicine and Biology, 163–73. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_14.

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Scorziello, Antonella, Claudia Savoia, Agnese Secondo, Francesca Boscia, Maria Josè Sisalli, Alba Esposito, Annalisa Carlucci, et al. "Erratum To: Chapter 17 New Insights in Mitochondrial Calcium Handling by Sodium/Calcium Exchanger." In Advances in Experimental Medicine and Biology, E1. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_38.

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Rojas, Héctor, Claudia Colina, Magaly Ramos, Gustavo Benaim, Erica Jaffe, Carlo Caputo, and Reinaldo Di Polo. "Sodium-Calcium Exchanger Modulates the L-Glutamate Cai 2+ Signalling in Type-1 Cerebellar Astrocytes." In Advances in Experimental Medicine and Biology, 267–74. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_22.

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Levitsky, Dmitri O., and Masayuki Takahashi. "Interplay of Ca2+ and Mg2+ in Sodium-Calcium Exchanger and in Other Ca2+-Binding Proteins: Magnesium, Watchdog That Blocks Each Turn if Able." In Advances in Experimental Medicine and Biology, 65–78. Boston, MA: Springer US, 2012. http://dx.doi.org/10.1007/978-1-4614-4756-6_7.

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Millour, Marie, Laurent Lescaudron, Alexander Kraev, and Dmitri O. Levitsky. "Expression of Sodium-Calcium Exchanger Genes in Heart and Skeletal Muscle Development. Evidence for a Role of Adjacent Cells in Regulation of Transcription and Splicing." In Signal Transduction and Cardiac Hypertrophy, 105–23. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0347-7_9.

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Atti di convegni sul tema "Sodium/calcium exchanger (NCX)"

Zheng, Yun-Min, Lin Mei, Ling Dong, and Yong-Xiao Wang. "Genetic Evidence For Essential Role Of Sodium-Calcium Exchanger In Development Of Pulmonary Hypertension." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a4750.

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Balasubramaniam, Sona Lakshme, Anilkumar Gopalakrishnapillai, Nicholas J. Petrelli, and Sonali P. Barwe. "Abstract 3931: Sodium-calcium exchanger-1 regulates the epithelial phenotype and is lost in renal cancers." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3931.

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Zou, Yong, Liang Zhao, Gongming Xin, and Lin Cheng. "Effect of Metallic Ion on the Formation of Calcium Carbonate Fouling." In 2010 14th International Heat Transfer Conference. ASMEDC, 2010. http://dx.doi.org/10.1115/ihtc14-22312.

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Calcium carbonate (CaCO3) is the most common fouling adhering on the surface of heat exchanger. But metallic ion in natural water could affect the crystalline type of calcium carbonate. In this study, the effect of sodium ion, magnesium ion and aluminum ion on crystalline type and morphology of CaCO3 were reported. The experimental results indicate that the addition of sodium ion has no obvious role for changing the crystalline type of CaCO3, only calcite was obtained and the lattice parameter of calcite has a little variation depending on the concentration of sodium ion. However, the addition

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Shestakova, N. N., and D. A. Belinskaia. "Structure of the Cation- And Ligand-Binding Sites of Human Sodium-Calcium Exchanger According to Homology Modeling." In Mathematical Biology and Bioinformatics. Pushchino: IMPB RAS - Branch of KIAM RAS, 2020. http://dx.doi.org/10.17537/icmbb20.2.

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Izadi, M., D. K. Aidun, P. Marzocca, and H. Lee. "The Experimental Investigation of Fouling Phenomenon in Heat Exchangers by Heat Transfer Resistance Monitoring (HTRM) Method." In ASME 2009 International Mechanical Engineering Congress and Exposition. ASMEDC, 2009. http://dx.doi.org/10.1115/imece2009-12524.

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The aim of this paper is to describe a monitoring system for fouling phenomenon in tubular heat exchangers. This system is based on a physical model of the fouling resistance. A mathematical model of the fouling resistance is developed based on the applied thermal heat, the inside heat transfer coefficient, and geometrical characteristics of the heat exchanger under consideration. The resulting model is a function of measured quantities such as water and tube wall temperatures, fluid flow velocities, and some physical properties of the fluid flowing inside the tubes such as viscosity, conducti

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Izadi, M., D. K. Aidun, P. Marzocca, and H. Lee. "Effect of Surface Roughness on Fouling of Calcium Carbonate: An Experimental Investigation." In ASME 2010 International Mechanical Engineering Congress and Exposition. ASMEDC, 2010. http://dx.doi.org/10.1115/imece2010-40623.

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The effect of surface roughness on the fouling behavior of calcium carbonate is experimentally investigated. The real operating conditions of a tubular heat exchanger are simulated by performing prolonged experiments with duration of 3 to 7 days. The solution used is a mixture of sodium bicarbonate and calcium chloride in de-ionized water with the concentration of 0.4 g/l of each. An on-line fouling evaluation system was developed such that the fouling resistance for a selected solution could be measured in real time. The experiments are repeated with the same procedure for 90/10 Cu/Ni tubes w

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