Gotowe bibliografie tematyczne / Acid peptic disorders

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji

Gotowa bibliografia na temat „Acid peptic disorders”

Autor: Grafiati
Data publikacji: 2 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Acid peptic disorders"

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Kale-Pradhan, Pramodini B., Heather K. Landry, and William T. Sypula. "Esomeprazole for Acid Peptic Disorders." Annals of Pharmacotherapy 36, no. 4 (2002): 655–63. http://dx.doi.org/10.1345/aph.1a104.

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OBJECTIVE: To review the pharmacology, pharmacokinetics, clinical efficacy, and safety of esomeprazole, a new proton-pump inhibitor (PPI). DATA SOURCES: A MEDLINE search (1966–March 2001) was conducted for relevant literature using the terms esomeprazole, Nexium, and H199/18. Abstracts from the XXXII Nordic Meeting of Gastroenterology and journal articles provided by AstraZeneca were reviewed. STUDY SELECTION AND DATA EXTRACTION: All available studies on the pharmacology, pharmacokinetics, clinical efficacy, and safety of esomeprazole were reviewed. DATA SYNTHESIS: Esomeprazole is a new PPI an
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Kuwayama, Hajime. "ADVANCES IN TREATING ACID PEPTIC DISORDERS." Journal of Gastroenterology and Hepatology 15, no. 12 (2000): H31. http://dx.doi.org/10.1046/j.1440-1746.2000.015012h31.x.

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Nasir, M., and AFM M. Rahman. "Proton Pump Inhibitors : The Controversies to Consider." Journal of Medical Science & Research 19, Number 2 (2012): 33–37. http://dx.doi.org/10.47648/jmsr.2012.v1902.06.

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Heartburn is ailing the masses. By one estimate. 20% of the Western population experiences acid reflux at least once a week and shelling out buckets of cash S25.6 billion worldwide in 2008 to alleviate those hot, sour. acidic pains in the chest. Proton pump inhibitors (PPIs) am one of the most commonly prescribed classes of medications in the primary care setting and are considered a major advance in the treatment of acid-peptic diseases. Since the introduction of omeprazok in 1989. several other PPIs have become available in combating various acid-peptic disorders, including gastroesophageal
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Nasir, M., and AFM M. Rahman. "Proton Pump Inhibitors : The Controversies to Consider." Journal of Medical Science & Research 19, Number 2 (2012): 33–37. http://dx.doi.org/10.47648/jmsr.2012.v1902.07.

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Heartburn is ailing the masses. By one estimate. 20% of the Western population experiences acid reflux at least once a week and shelling out buckets of cash S25.6 billion worldwide in 2008 to alleviate those hot, sour. acidic pains in the chest. Proton pump inhibitors (PPIs) am one of the most commonly prescribed classes of medications in the primary care setting and are considered a major advance in the treatment of acid-peptic diseases. Since the introduction of omeprazok in 1989. several other PPIs have become available in combating various acid-peptic disorders, including gastroesophageal
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Gustavsson, S., and L. Engstrand. "Future Strategies in Acid-peptic Disorders: Concluding Remarks." Scandinavian Journal of Gastroenterology 23, sup142 (1988): 114–16. http://dx.doi.org/10.3109/00365528809091725.

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Saltiel, Emmanuel. "Peptic Acid Disorders: Developing a Disease Management Program." Journal of Managed Care Pharmacy 2, no. 5 (1996): 569–75. http://dx.doi.org/10.18553/jmcp.1996.2.5.569.

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Parthasarathy, Srinivasan. "Vonoprazan – A New Drug for Acid Peptic Disorders." SBV Journal of Basic, Clinical and Applied Health Science 7, no. 3 (2024): 103–4. http://dx.doi.org/10.4103/sbvj.sbvj_42_24.

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CLEARFIELD, H. R. "Challenges in acid/peptic disorders: a symposium overview." Alimentary Pharmacology & Therapeutics 5 (March 31, 2007): 1–4. http://dx.doi.org/10.1111/j.1365-2036.1991.tb00743.x.

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LEWIS, J. H. "Idiopathic gastric acid hypersecretion: treatment implications for refractory acid/peptic disorders." Alimentary Pharmacology & Therapeutics 5 (March 31, 2007): 15–24. http://dx.doi.org/10.1111/j.1365-2036.1991.tb00745.x.

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Islam, M. M. Shahin Ul. "“Potassium Competitive Acid Blocker”- new Weapon to Combat Acid Peptic Disorders." Bangabandhu Sheikh Mujib Medical College Journal 2, no. 2 (2023): 72–73. http://dx.doi.org/10.3329/bsmmcj.v2i2.69830.

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Rozprawy doktorskie na temat "Acid peptic disorders"

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Minnie, Jacqueline Louise. "An education intervention on prescribing patterns of drugs for acid-related disorders in a clinic setting : a case study / Jacqueline Louise Minnie." Thesis, North-West University, 2007. http://hdl.handle.net/10394/1562.

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Janse, van Rensburg Hendrika Nicolien. "A longitudinal study of the usage of acid reducing medicine using a medicine claims database / Hendrika Nicolien Janse van Rensburg." Thesis, North-West University, 2007. http://hdl.handle.net/10394/1903.

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Acid-related disorders are common, chronic conditions that have considerable impact on a patient's quality of life. In a study conducted by Majumdar et al. (2003:2411) the prevalence of chronic acid-related disorders was 2.3%. Acid-related disorders represent a major financial consideration with respect to the costs of drug prescribing (Whitaker, 1998:6). Health care cost increases each year. This leads to an increased interest in economic evaluation of health care and medical technologies (Anell & Svarvar, 2000:175). Health care providers no longer make treatment decisions independent of the
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Ramraj, Varun. "Exploiting whole-PDB analysis in novel bioinformatics applications." Thesis, University of Oxford, 2014. http://ora.ox.ac.uk/objects/uuid:6c59c813-2a4c-440c-940b-d334c02dd075.

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The Protein Data Bank (PDB) is the definitive electronic repository for experimentally-derived protein structures, composed mainly of those determined by X-ray crystallography. Approximately 200 new structures are added weekly to the PDB, and at the time of writing, it contains approximately 97,000 structures. This represents an expanding wealth of high-quality information but there seem to be few bioinformatics tools that consider and analyse these data as an ensemble. This thesis explores the development of three efficient, fast algorithms and software implementations to study protein struct
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Van, Rensburg Hendrika Nicolien Janse. "A longitudinal study of the usage of acid reducing medicine using a medicine claims database / H.N. Janse van Rensburg." Thesis, 2007. http://hdl.handle.net/10394/1903.

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Książki na temat "Acid peptic disorders"

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Arnold, Berstad, ed. Management of peptic ulcer disease and acid-related disorders: Clinical aspects of antacids in the 1990s : Bermuda, November 15-16, 1991. Raven Press, 1992.

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Peptic ulcer disease and other acid-related disorders. Academic Research Associates, 1991.

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Clearfield, Harris R. Acid-Peptic Disorders of the Upper Gastrointestinal Tract: Diagnosis and Management. Handbooks in Health Care, 1995.

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Freston, James W. Diseases of the Gastroesophageal Mucosa: The Acid-Related Disorders. Humana Press, 2012.

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Freston, James W. Diseases of the Gastroesophageal Mucosa: The Acid-Related Disorders. Humana Press, 2001.

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(Editor), Henry Parkman, and Robert S. Fisher (Editor), eds. The Clinician's Guide to Acid/Peptic Disorders and Motility Disorders of the Gastrointestinal Tract (The Clinician's Guide to GI Series). Slack Incorporated, 2006.

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Modlin, Irvin M., and George Sachs. Acid Related Diseases: Biology and Treatment. 2nd ed. Lippincott Williams & Wilkins, 2004.

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Sedlack, Robert E., Conor G. Loftus, Amy S. Oxentenko, and Thomas R. Viggiano. Gastroenterology. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199755691.003.0210.

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Part 1 reviews the major portions of the gastrointestinal system (esophagus, stomach, small intestine, colon, and pancreas), their function (motility, acid production, enzymatic function, and absorption), and various disorders associated with them (dysmotility, ulceration, malabsorption, inflammation, and dysplasia). Symptoms, diagnostic testing, and treatment of common gastrointestinal conditions, such as gastroesophageal reflux disease, peptic ulcer disease, diarrhea, constipation, inflammatory bowel disease, and pancreatitis, are reviewed.
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Wójcik-Gładysz, Anna. Ghrelin – hormone with many faces. Central regulation and therapy. The Kielanowski Institute of Animal Physiology and Nutrition, Polish Academy of Sciences, 2020. http://dx.doi.org/10.22358/mono_awg_2020.

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Discovered in 1999, ghrelin, is one of the peptides co-creating the hypothalamicgastrointestinal axis, otherwise known as the brain-gut axis. Ghrelin participates in many physiological processes and spectrum of its activity is still being discovered. This 28 amino acid peptide ‒ a product of the ghrl gene, was found in all vertebrates and is synthesized and secreted mainly from enteroendocrine X/A cells located in the gastric mucosa of the stomach. Expression of the ghrelin receptor has been found in many nuclei of the hypothalamus involved in appetite regulation. Therefore it’s presumed that
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Części książek na temat "Acid peptic disorders"

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Kovacs, Thomas O. G. "Acid-Peptic Disorders." In Gut Instincts. CRC Press, 2024. http://dx.doi.org/10.1201/9781003524489-11.

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Lakshmanan, Mageshwaran. "Drugs Used in Acid Peptic Disorders." In Introduction to Basics of Pharmacology and Toxicology. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-33-6009-9_34.

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"Acid Peptic Disorders." In Yamada's Handbook of Gastroenterology. John Wiley & Sons, 2013. http://dx.doi.org/10.1002/9781118572610.ch21.

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Qadeer, Mohammed A., and Gary W. Falk. "Acid Peptic Disorders." In Current Clinical Medicine. Elsevier, 2010. http://dx.doi.org/10.1016/b978-1-4160-6643-9.00065-5.

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Daneshmend, Tawfique, and Alasdair Dow. "Gastroenterological disorders." In Perioperative Medicine. Oxford University PressOxford, 2006. http://dx.doi.org/10.1093/oso/9780199211739.003.0030.

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Abstract A history of acid-peptic ulceration of the upper digestive tract affecting the duodenum, stomach, or oesophagus will often be obtained in patients presenting for elective or emergency surgery unrelated to the upper GI tract. Surgery for acid-peptic disease itself (elective surgery for peptic ulcer, gastro-oesophageal reflux disease; emergency surgery for perforated peptic ulcer or oesophagus; or surgery for acute GI bleeding) is not the purpose of this chapter. The management of these patients is addressed in standard surgical texts. Chronic peptic ulceration should now be rare. Since
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Vikraman Pillai, R. "Acid Peptic Disease." In PG Textbook of Pediatrics: Infections and Systemic Disorders (Volume 2). Jaypee Brothers Medical Publishers (P) Ltd., 2015. http://dx.doi.org/10.5005/jp/books/12854_100.

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"Gastric Physiology and Acid-Peptic Disorders." In Modern Surgical Care. CRC Press, 2006. http://dx.doi.org/10.3109/9781420016581-19.

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Boarder, Michael, Jane Dixon, David Newby, Phyllis Navti, and Tyra Zetterström. "Upper gastrointestinal tract disorders." In Pharmacology for Pharmacy and the Health Sciences. Oxford University Press, 2016. http://dx.doi.org/10.1093/9780198728832.003.0012.

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This chapter focuses on upper gastrointestinal tract disorders. The commonly experienced symptoms of heartburn, acid regurgitation, and indigestion (dyspepsia) arise from disturbances of acid secretion in the stomach and the consequent damage to the mucosal lining of the oesophagus, stomach, or duodenum. Conditions affecting the upper gastrointestinal tract, like gastro-oesophageal reflux disease and peptic ulcer disease, are among the most prevalent illnesses in Western countries. The chapter discusses the causes and significance of nausea and vomiting. It cites that the main targets for anti
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Aridome, Kuniaki. "Benign diseases of the stomach." In Oesophagus and Stomach, edited by Matthias Reeh, Jakob R. Izbicki, Samiran Nundy, and Dirk J. Gouma. Oxford University PressOxford, 2023. http://dx.doi.org/10.1093/med/9780192863591.003.0005.

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Abstract Most benign diseases of the stomach are attributed to functional dyspepsia and acid-related disorders such as peptic ulcer disease. Dyspepsia is also the main manifestation of these acid-related conditions of the stomach. Functional dyspepsia comprises three subtypes: postprandial distress syndrome, epigastric pain syndrome, and a subtype with overlapping postprandial distress syndrome and epigastric pain syndrome features. Peptic ulcer disease is characterized by a non-malignant lesion in the gastrointestinal mucosa that may be acute, subacute, or chronic. Proton pump inhibitors sign
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Garg, Dr Gopal. "Antiulcer Agents." In Edited Book of Pharmacology-III [According to Latest Syllabus of B. Pharm-VI Semester of Pharmacy Council of India]. Iterative International Publishers, Selfypage Developers Pvt Ltd, 2024. http://dx.doi.org/10.58532/nbennurphch2.

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Antiulcer agents are essential medications used to manage and treat conditions like peptic ulcers, gastroesophageal reflux disease (GERD), and other acid-related disorders of the gastrointestinal tract. These agents are classified into several categories based on their mechanisms of action: proton pump inhibitors (PPIs), H2-receptor antagonists (H2RAs), antacids, mucosal protective agents, antibiotics, and prostaglandin analogues. PPIs, such as omeprazole and lansoprazole, work by irreversibly inhibiting the hydrogen-potassium ATPase enzyme, significantly reducing gastric acid secretion. H2RAs
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Streszczenia konferencji na temat "Acid peptic disorders"

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Bauer, K. A., B. L. Kass, M. Bednarek, M. Kloczewiak, J. Hawiger, and R. D. Rosenberg. "DETECTION OF FACTOR X ACTIVATION IN HUMANS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643828.

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The activation of factor X by factor VII/VIIa-tissue factor or factor IXa plays a pivotal role in the hemostatic mechanism. This reaction results in the liberation of a peptide from the zymogen for which we have developed a sensitive and specific radioimmunoassay (RIA), The native peptide was purified from activated human factor X by hydroxylapatite chromatography and reverse-phase high pressure liquid chromatography (HPLC). Gel filtration experiments demonstrated that the peptide was not physically associated with the enzyme. A 15 amino acid peptide with the COOH-terminal sequence of the acti
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Kibitkina, A. A., E. R. Vasilevskaya, N. A. Ilin, and G. S. Tolmacheva. "CHANGES IN MILK OF TPH2 HET MICE UNDER EXPOSURE TO STRESS." In NOVEL TECHNOLOGIES IN MEDICINE, BIOLOGY, PHARMACOLOGY AND ECOLOGY. LLC Institute Information Technologies, 2024. http://dx.doi.org/10.47501/978-5-6044060-4-5.41-46.

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The relationship between neuroregulatory factors of the brain and the composition of breast milk is an interesting and little-studied topic. The article is aimed at studying changes in the chemical composition of the milk of mice prone to the development of affective disorders (heterozygous for TPH2) under the influence of stress and without stress relative to the milk of ordinary mice. Raman spectroscopy (wavelength 785 nm, focal length 50x) was used for the study. Het mouse milk has a less rich protein and peptide profile, but the range of fatty acids, especially under stress, increases.
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Müller, E., and A. Henschen. "HUMAN PLASMA FIBRINOGEN MOLECULAR WEIGHT VARIANTS:CHARACTERIZATION BY HIGH-PERFORMANCE LIQUID CHROMATOGRAPHY AND IDENTIFICATION BY SEQUENCE ANALYSIS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643328.

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Human plasma has been shown to contain several fibrinogen variants which differ from each other in molecular weight. The three most common forms, with molecular weights of 340 kD, 305 kD and 270 kD, have been reported to change considerably in their relative amounts during certain pathophysiological processes such as acute phase reactions, disseminated intravascular coagulation, fibrinolytic disorders, liver disease and cancer. Though it has been suggested that the differences in molecular weight are due to degradation of one or both, respectively, of the carboxy-terminal parts of the Aα-chain
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Lazarević, Marija. "COGNITIVE, HEMATOPOIETIC AND GASTROINTESTINAL IMPAIRMENTS CAUSED BY VITAMIN B12 DEFICIENCY – PATOGENESIS, TREATMENT AND PATIENT CARE." In Global Challenges Through the Prism of Rural Development in the Sector of Agriculture and Tourism GIRR 2024. Academy of Applied Studies Šabac, 2025. https://doi.org/10.46793/girr25.287l.

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Many studies have investigated the pathogenetic association of cognitive, hematopoietic, and gastrointestinal impairments with low vitamin B12 concentrations. The aim of our study is to point out the importance of maintaining vitamin B12 concentrations in the body, in order to preserve cognition, but also other vital parameters of the body. For a systematic literature search on studies on the association of cognitive, hematopoietic and gastrointestinal impairments with vitamin B12 deficiency, we used we used the databases: Web of Science, PubMed, Google Scholar. The search keywords included “v
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Askew, Michael J., Gary B. Schneider, Kristina J. Grecco, Jason Hsu, Emily Mugler, and Donald A. Noe. "Effect of Pharmaceutical Bone Growth Stimulation With Novel Anabolic Peptides: Biomechanical and Bone Density Measurements in a Rat Model." In ASME 2003 International Mechanical Engineering Congress and Exposition. ASMEDC, 2003. http://dx.doi.org/10.1115/imece2003-43044.

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Pharmaceutical bone growth stimulation holds promise for prevention and treatment bone disorders, and the enhancement of fracture healing. Bone growth hormones have begun to have limited clinical use, but can illicit adverse side effects. Recent studies have shown that short peptides (less than 15 amino acids) derived from the protein sequence of Vitamin D Binding Protein (DBP), can enhance bone formation (osteogenesis). These peptides may have potential as controllable bone growth stimulators without the adverse side effects and cost of bone growth hormones. Rats, injected every other day for
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Scheefers-Borchel, U., and G. Muller-Berghaus. "A NEW FIBRIN-SPECIFIC ANTIBODY DISCRIMINATING BETWEEN FIBRIN AND FIBRINOGEN IS DIRECTED AGAINST THE SYNTHETIC PEPTIDE LEU-ILE-ASP-GLY-LYS-MET." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643771.

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To determine soluble fibrin in blood of patients with coagulation disorders we produced monoclonal antibodies which distinct fibrin from fibrinogen and other blood constituents. Fibrin-specific monoclonal antibodies were obtained by immunizing mice with the synthetic hexapeptide Leu-Ile-Asp-Gly-Lys-Met which was covalently linked to KLH via its C-terminus. Several of the monoclonal antibodies which reacted with the hexapeptide also reacted with batroxobin-induced desAA-fibrin and thrombin-induced desAABB-fibrin, but not with fibrinogen. No reaction was observed with plasmin-induced fibrinogeno
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Homes, W. E., H. R. Lijnen, L. Nelles, C. Kluft та D. Collen. "AN ALANINE INSERTION IN α2-ANTIPLASMIN ‘ENSCHEDE’ ABOLISHES ITS PLASM IN INHIBITORY ACTIVITY". У XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642897.

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Congenital deficiencies of the fibrinolytic inhibitor α2antiplasmin (α2AP) may result in bleeding disorders. An abnormal a AP (α2AP‘Enschede’) is known. 2 siblings with 3% functional activity and normal antigen level have parents with 50% activity and normal antigen. The protein interacts normally with the lysine-binding site(s) of plasmin(ogen) but does not inhibit plasmin irreversibly. α2AP Enschede is a plasmin substrate that like the normal protein releases a M 8,000 peptide upon reaction with plasmin. In the present study, Southern blot analysis, using an α2AP cDNA probe showed a restrict
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Asen, Nancy, and Rotimi Aluko. "Functional Properties of Enzymatic Pea Protein Hydrolysates That Inhibit in vitro Activities of Acetylcholinesterase and Butyrylcholinesterase." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/ktht4252.

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Overview: Alzheimer’s disease (AD) is a neurodegenerative disorder prevalent among the aged population with morbidity and mortality rate of ~ 12%. Research has linked the cause of this disorder to the loss of acetylcholine through excessive activities of acetylcholinesterase (AChE) and butyrylcholinesterase (BChE). Therefore, a promising therapeutic approach for AD treatment is the inhibition of AChE/BChE activities. Common features of an AD brain include low levels of acetylcholine, the presence of amyloid-β peptides deposits and severe oxidative stress triggered by lipid peroxidation and for
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Antonarakis, E. "The Molecular Genetics of Hemophilia A Stylianos." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643980.

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Hemophilia A is a common X linked hereditary disorder of blood coagulation due to deficiency of factor 8. The gene for factor 8 has been cloned and characterized (Nature 312:326-342, 1984). It is divided into 26 exons and 25 introns and spans 186 kb of DNA. The CGNA is 9 kb and codes for 2351 amino acids. The first 19 amino acids comprise the secretory leader peptide and the mature excreted polypeptide consists of 2332 amino acids. The nucleotide sequence of the exons and the exon-intron junctions is known and the complete amino acid sequence has been deducedSeveral laboratories have used clon
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