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Artykuły w czasopismach na temat „Acute Hypotension”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Novosel, Dragan. "Acute Pancreatitis Mimicking Acute STEMI Infarction." Open Access Journal of Cardiology 7, no. 1 (2023): 1–4. http://dx.doi.org/10.23880/oajc-16000183.

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A 54-year-old male with the history of pancreatitis, arterial hypertension and diabetes was presented to cardiologist with epigastrial pain, inferior STEMI and hypotension. Initial laboratory findings were delayed because of hemolytic serum that caused administration of invasive procedures for ruling out myocardial infarction. The condition causing hypotension was necrotic pancreatitis. This case report highlights the importance of thinking of other not very likely diagnoses in patients with STEMI infarction and the importance of obtaining laboratory findings in time.

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Collette, Sabine L., Maarten Uyttenboogaart, Noor Samuels, et al. "Hypotension during endovascular treatment under general anesthesia for acute ischemic stroke." PLOS ONE 16, no. 6 (2021): e0249093. http://dx.doi.org/10.1371/journal.pone.0249093.

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Objective The effect of anesthetic management (general anesthesia [GA], conscious sedation, or local anesthesia) on functional outcome and the role of blood pressure management during endovascular treatment (EVT) for acute ischemic stroke is under debate. We aimed to determine whether hypotension during EVT under GA is associated with functional outcome at 90 days. Methods We retrospectively collected data from patients with a proximal intracranial occlusion of the anterior circulation treated with EVT under GA. The primary outcome was the distribution on the modified Rankin Scale at 90 days.

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Klijn, Eva, A. B. Johan Groeneveld, Michel E. van Genderen, Michiel Betjes, Jan Bakker, and Jasper van Bommel. "Peripheral Perfusion Index Predicts Hypotension during Fluid Withdrawal by Continuous Veno-Venous Hemofiltration in Critically Ill Patients." Blood Purification 40, no. 1 (2015): 92–98. http://dx.doi.org/10.1159/000381939.

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Aim: Peripheral perfusion may predict harmful hypovolemic hypotension during fluid withdrawal by continuous veno-venous hemofiltration (CVVH) in critically ill patients with acute kidney injury. Methods: Twenty-three critically ill AKI patients were subjected to progressive fluid withdrawal. Systemic hemodynamics and peripheral perfusion index (PPI) by pulse oximetry, forearm-to-fingertip skin temperature gradient (Tskin-diff) and tissue oxygen saturation (StO2, near infra-red spectroscopy) were measured. Results: Most hemodynamic values decreased with fluid withdrawal, particularly in the hyp

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4

Westhoff, Timm H., Stefanie Scheid, Markus Tölle, et al. "A physiogenomic approach to study the regulation of blood pressure." Physiological Genomics 23, no. 1 (2005): 46–53. http://dx.doi.org/10.1152/physiolgenomics.00077.2005.

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Several vasoregulatory systems including the renin-angiotensin system, sympathetic vasoregulation, and cytokine release have been studied extensively. The aim of the present study was to establish a physiogenomic screening model for differentially expressed genes in the regulation of blood pressure that might give a hint as to new vasoregulatory mechanisms. We induced acute hypotension in normotensive rats, assuming that vasoregulatory systems will counteract hypotension. Microarray transcriptome analysis was performed from kidneys 6 h after the induction of acute hypotension. The results were

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Sedaghattalab, Moslem, and Amir Hossein Doustimotlagh. "Acute Secondary Adrenal Insufficiency Misdiagnosed as Acute Cholecystitis." Case Reports in Endocrinology 2021 (November 30, 2021): 1–3. http://dx.doi.org/10.1155/2021/8318747.

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Hypopituitarism refers to insufficient secretion of the pituitary hormones. Patients with acute adrenocorticotropic hormone (ACTH) deficiency may be presented with fatigue, dizziness, orthostatic hypotension, hypoglycemia, nausea, vomiting, or nonspecific abdominal pain. This study described an unusual case of hypopituitarism in a patient who presented with general abdominal pain, abdominal tenderness, nausea, vomiting, hypotension, and hypoglycemia. At first, the patient was admitted with the diagnosis of acute cholecystitis, but after treatment of hypopituitarism and adrenal insufficiency, h

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Lizano-Díez, Irene, Stephen Poteet, Adrià Burniol-Garcia, and Mónica Cerezales. "The burden of perioperative hypertension/hypotension: A systematic review." PLOS ONE 17, no. 2 (2022): e0263737. http://dx.doi.org/10.1371/journal.pone.0263737.

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Study objective Our goal is to review the outcomes of acute hypertensive/hypotensive episodes from articles published in the past 10 years that assessed the short- and long-term impact of acute hypertensive/hypotensive episodes in the perioperative setting. Methods We conducted a systematic peer review based upon PROSPERO and Cochrane Handbook protocols. The following study characteristics were collected: study type, author, year, population, sample size, their definition of acute hypertension, hypotension or other measures, and outcomes (probabilities, odds ratio, hazard ratio, and relative r

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7

Pettersen, Jacqueline A., Julia Keith, Fuqiang Gao, J. David Spence, and Sandra E. Black. "CADASIL accelerated by acute hypotension." Neurology 88, no. 11 (2017): 1077–80. http://dx.doi.org/10.1212/wnl.0000000000003717.

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Objective:To underline the importance of blood pressure regulation in cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL) and to describe changes that occur in the veins in this condition, specifically venous collagenosis associated with leukoaraiosis.Methods:Case report with neuroimaging and pathologic data.Results:A 61-year-old man with genetically confirmed CADASIL was initially lucid following a motor vehicle accident but subsequently became hypotensive (60/40 mm Hg) due to an open femur fracture and required intubation. Multiple new white m

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Horne, R. S., P. J. Berger, G. Bowes, and A. M. Walker. "Effect of sinoaortic denervation on arousal responses to hypotension in newborn lambs." American Journal of Physiology-Heart and Circulatory Physiology 256, no. 2 (1989): H434—H440. http://dx.doi.org/10.1152/ajpheart.1989.256.2.h434.

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To examine whether hypotension reflexly initiates arousal from sleep and the mechanisms involved, we subjected sleeping lambs to hypotensive stimuli of 1-min duration, before and after sinoaortic denervation (SAD). In intact lambs, hypotension increased the probability of arousal from both quiet sleep (QS) and rapid-eye-movement (REM) sleep. Hypotension resulted in nonarousal in 42% (QS) and 47% (REM) of tests. Arousal time was significantly longer in REM (34.9 +/- 1.8 s, means +/- SE) than in QS (26.0 +/- 1.8 s). Arterial saturation of O2 (SO2) and PO2 measured at the point of arousal were un

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9

Cernacek, Peter, Duncan J. Stewart, and Mortimer Levy. "Plasma endothelin-1 response to acute hypotension induced by vasodilating agents." Canadian Journal of Physiology and Pharmacology 72, no. 9 (1994): 985–91. http://dx.doi.org/10.1139/y94-137.

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Cyclic GMP (cGMP) dependent vasodilating agents (natriuretic peptides, nitric oxide) inhibit secretion of endothelin-1 (ET-1) in cultured endothelial cells. However, in circulatory conditions associated with acute hypotension, a marked increase in plasma ET-1 has repeatedly been observed. Therefore, after administration of cGMP-dependent agents in hypotensive dose, the net effect of these opposing influences on ET-1 release may shed light on the mechanisms determining circulating levels of this peptide. We have studied the effect of a hypotensive dose of atrial natriuretic peptide (n = 16), 8-

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10

De la Cruz-Vigo, F., R. Galvez-Valdovinos, F. Sanchez-Bustos, R. Pardo-Garcia, and J. Cruz-Leiva. "Abdominal pain and acute hypotension." Postgraduate Medical Journal 71, no. 836 (1995): 363–64. http://dx.doi.org/10.1136/pgmj.71.836.363.

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Fitts, Douglas A., Jeannine R. Lane, Elizabeth M. Starbuck, and Chi-Pei Li. "Drinking and blood pressure during sodium depletion or ANG II infusion in chronic cholestatic rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 276, no. 1 (1999): R23—R31. http://dx.doi.org/10.1152/ajpregu.1999.276.1.r23.

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After a chronic ligation of the common bile duct (BDL), Long-Evans rats are hypotensive and have elevated saline intake during both sodium-depleted and nondepleted conditions. We tested whether BDL rats have exaggerated hypotension during sodium depletion or an elevated dipsogenic response to angiotensin II (ANG II) that might help to explain the saline intake. After 4 wk of BDL, rats were hypotensive at baseline and developed exaggerated hypotension during acute furosemide-induced diuresis. Without saline to drink, BDL rats increased water intake during depletion equal to sham-ligated rats. H

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12

Resta, T. C., R. D. Russ, M. P. Doyle, J. M. Martinez, and B. R. Walker. "Cardiovascular responses to hemorrhage during acute and chronic hypoxia." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 267, no. 3 (1994): R619—R627. http://dx.doi.org/10.1152/ajpregu.1994.267.3.r619.

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Previous work from our laboratory had demonstrated attenuation of systemic vasoreactivity to pressor agents in rats after acute or chronic exposure to hypoxia. Therefore we hypothesized that hemorrhage of acutely hypoxic (12% O2) or chronically hypoxic (barometric pressure 380 mmHg for 3 wk) rats would deter the normal increase in total peripheral resistance (TPR) and thus decrease the ability to maintain blood pressure. Progressive hemorrhage (2% of blood volume per min) was performed under conditions of either normoxia or acute hypoxia in conscious rats. In control animals, the increase in T

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13

Schiltz, Jennifer C., Gloria E. Hoffman, Edward M. Stricker, and Alan F. Sved. "Decreases in arterial pressure activate oxytocin neurons in conscious rats." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 273, no. 4 (1997): R1474—R1483. http://dx.doi.org/10.1152/ajpregu.1997.273.4.r1474.

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Hemorrhage and nonhypotensive hypovolemia are known to increase plasma levels of oxytocin (OT) and vasopressin (VP) in rats. The present experiments demonstrated that secretion of OT and VP also are stimulated by acute drug-induced hypotension. Injection of hydralazine abruptly decreased arterial blood pressure in conscious rats and induced Fos expression, a marker of neuronal activation, within OT and VP neurons in the hypothalamus. Hydralazine also elicited substantial increases in plasma levels of both OT and VP. Injection of chlorisondamine similarly elicited acute hypotension and increase

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14

Koyama, Yuhei, and Shinsuke Hamaguchi. "Challenging treatment of severe hypotension following tracheal intubation in a patient with primary hyperparathyroidism: A case report." Medicine 103, no. 35 (2024): e39510. http://dx.doi.org/10.1097/md.0000000000039510.

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Rationale: Primary hyperparathyroidism (PHPT), which is characterized by increased parathyroid hormone secretion, typically manifests as hypercalcemia and hypertension. Here, we report a case of severe hypotension following tracheal intubation during anesthesia induction in a patient with PHPT, in contrast to the expected hypertensive response. Patient concerns: A 52-year-old man presented with nausea after eating, leg pain when walking, and headaches. Diagnosis: Based on the blood test and computed tomography results, he was diagnosed with PHPT. Interventions: The patient underwent parathyroi

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Resta, T. C., J. M. Resta, and B. R. Walker. "Role of endogenous opioids and serotonin in the hemodynamic response to hemorrhage during hypoxia." American Journal of Physiology-Heart and Circulatory Physiology 269, no. 5 (1995): H1597—H1606. http://dx.doi.org/10.1152/ajpheart.1995.269.5.h1597.

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Previous studies from our laboratory indicate that acute but not chronic hypoxia decreases the hemorrhage volume required to elicit reflex hypotension. Furthermore, chronically hypoxic animals exhibit an elevated hypotensive threshold during both normoxia and hypoxia compared with control animals. Because reports suggest that opioid and serotonergic mechanisms may be involved in mediating the sympathoinhibition that occurs with hemorrhage, we hypothesized that opioid and/or serotonergic systems are stimulated during hemorrhage under conditions of acute hypoxia and suppressed after chronic expo

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16

Ngamprasertwong, Pornswan, Mounira Habli, Anne Boat, et al. "Maternal Hypotension during Fetoscopic Surgery: Incidence and Its Impact on Fetal Survival Outcomes." Scientific World Journal 2013 (2013): 1–7. http://dx.doi.org/10.1155/2013/709059.

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In this retrospective cohort study, we aimed to determine the incidence of intraoperative maternal hypotension during fetoscopic surgery for twin-twin transfusion syndrome (TTTS) and to evaluate the impact of intraoperative hypotension on fetal survival. A total of 328 TTTS patients with recipient twin cardiomyopathy who underwent fetoscopic surgery under epidural anesthesia were included. The exposure of interest was maternal medical therapy with nifedipine for the treatment of fetal cardiomyopathy. We found that intraoperative hypotension occurred in 53.4% (175/328 patients). There was no st

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17

Cardozo, Diogo, and Denise de Souza Destro. "Exercise order in resistance training – a brief review of the acute effects on cardiovascular response in the post-exercise period." Research, Society and Development 11, no. 13 (2022): e272111335489. http://dx.doi.org/10.33448/rsd-v11i13.35489.

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This study aims to review the main effects of the performance order of resistance exercises on cardiovascular response in strength training (ST) sessions. To do so, a search was carried out in PubMed, BIREME, and Google Scholar databases using as descriptors: 'order', 'resistance training order', 'resistance exercise order', 'blood pressure' 'hypotension', 'effect hypotensive', 'post-exercise hypotension' and 'cardiovascular responses'. After applying the inclusion/exclusion criteria, six studies were considered in this review. The results suggest that ST can be considered as a non-pharmacolog

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Lee, Subin, Misoon Lee, Sang-Hyun Kim, and Jiyoung Woo. "Intraoperative Hypotension Prediction Model Based on Systematic Feature Engineering and Machine Learning." Sensors 22, no. 9 (2022): 3108. http://dx.doi.org/10.3390/s22093108.

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Arterial hypotension is associated with incidence of postoperative complications, such as myocardial infarction or acute kidney injury. Little research has been conducted for the real-time prediction of hypotension, even though many studies have been performed to investigate the factors which affect hypotension events. This forecasting problem is quite challenging compared to diagnosis that detects high-risk patients at current. The forecasting problem that specifies when events occur is more challenging than the forecasting problem that does not specify the event time. In this work, we challe

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Sullivan, D. Matthew, John A. Watts, and Jeffrey A. Kline. "Biventricular cardiac dysfunction after acute massive pulmonary embolism in the rat." Journal of Applied Physiology 90, no. 5 (2001): 1648–56. http://dx.doi.org/10.1152/jappl.2001.90.5.1648.

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Cardiac dysfunction has been documented in vivo after acute massive pulmonary embolism (AMPE). The present study tests whether intrinsic ventricular dysfunction occurs in rat hearts isolated after AMPE. AMPE was induced in spontaneously breathing ketamine-xylazine-anesthetized rats by thrombus infusion until mean arterial blood pressure (MAP) was ∼40% of basal measurement. A hypotensive control group underwent controlled blood withdrawal to produce MAP ∼40% of basal levels. Shams underwent identical surgical and anesthesia preparation but without pulmonary embolization. Hearts were perfused in

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Du Pont-Thibodeau, G., N. Robitaille, F. Gauvin, et al. "Incidence of hypotension and acute hypotensive transfusion reactions following platelet concentrate transfusions." Vox Sanguinis 110, no. 2 (2015): 150–58. http://dx.doi.org/10.1111/vox.12329.

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Sung, Victor. "Acute Respiratory Failure Complicating Intracranial Hypotension." Headache: The Journal of Head and Face Pain 47, no. 9 (2007): 1340–41. http://dx.doi.org/10.1111/j.1526-4610.2007.00924.x.

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Kim, Hyun-Ah, and Hyung Lee. "Orthostatic hypotension in acute cerebellar infarction." Journal of Neurology 263, no. 1 (2015): 120–26. http://dx.doi.org/10.1007/s00415-015-7945-7.

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Kim, Hyun-Ah, Hyon-Ah Yi, and Hyung Lee. "Orthostatic hypotension in acute vestibular neuritis." Journal of the Neurological Sciences 358, no. 1-2 (2015): 479–80. http://dx.doi.org/10.1016/j.jns.2015.08.1547.

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Saites, Victoria, and Krzysztof Laudanski. "Acute Hypotension After 50% Dextrose Injections." A & A Case Reports 6, no. 10 (2016): 296–98. http://dx.doi.org/10.1213/xaa.0000000000000299.

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SHAMMAS, AMBER, and ANGELA P. CLARK. "Trendelenburg Positioning to Treat Acute Hypotension." Clinical Nurse Specialist 21, no. 4 (2007): 181–87. http://dx.doi.org/10.1097/01.nur.0000280485.03389.52.

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Capaldi, L. M. "Orthostatic hypotension following acute intracerebral haemorrhage." Age and Ageing 34, no. 1 (2005): 86–88. http://dx.doi.org/10.1093/ageing/afh241.

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Piccal, S., S. Picardo, F. Parisi, and G. Rizzoni. "Acute hypotension during continuous arteriovenous haemofiltration." Pediatric Nephrology 4, no. 3 (1990): 300. http://dx.doi.org/10.1007/bf00857681.

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Weiss, Avraham, Ehud Grossman, Yichayaou Beloosesky, and Joseph Grinblat. "Orthostatic Hypotension in Acute Geriatric Ward." Archives of Internal Medicine 162, no. 20 (2002): 2369. http://dx.doi.org/10.1001/archinte.162.20.2369.

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Besmertis, Lavrentios, David C. Bonovich, and J. Claude Hemphill. "The Role of Hypotension in Secondary Brain Injury after Intracerebral Hemorrhage." Stroke 32, suppl_1 (2001): 358. http://dx.doi.org/10.1161/str.32.suppl_1.358-d.

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P108 Objective: Hypotension is known to exacerbate secondary brain injury and worsen outcome after head trauma and ischemic stroke. Optimal blood pressure management after spontaneous intracerebral hemorrhage (ICH) remains a point of debate. The purpose of this study was to determine the incidence and impact on outcome of hypotension early after ICH. Methods: Medical records were reviewed for all 89 patients admitted with acute non-traumatic ICH to the University of California, San Francisco and San Francisco General Hospital during 1997. Complete information was available in 75 patients, who

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Charles, Christopher J., Miriam T. Rademaker, and A. Mark Richards. "Urocortin 1 modulates the neurohumoral response to acute nitroprusside-induced hypotension in sheep." Clinical Science 112, no. 9 (2007): 485–91. http://dx.doi.org/10.1042/cs20060303.

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In addition to haemodynamic actions, Ucn1 (urocortin 1) has been reported to affect a number of hormonal systems; however, it remains unclear whether Ucn1 modulates circulating hormones under physiological conditions. Accordingly, in the present study, we have examined the effects of Ucn1 on haemodynamics, hormones and renal indices in normal conscious sheep subjected to a nitroprusside-induced hypotensive stimulus designed to alter hormonal levels within the physiological range. Ucn1 administration did not alter the haemodynamic response to nitroprusside-induced hypotension. However, compared

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Kesler, Natalie V., Curt C. Bay, Brook Chavarria, et al. "25 Enteral Acetaminophen Induced Hypotension: Getting It Right vs Being Right." Journal of Burn Care & Research 45, Supplement_1 (2024): 21. http://dx.doi.org/10.1093/jbcr/irae036.025.

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Abstract Introduction Fevers in burn injured patients is a common phenomenon and is typically managed with enteral acetaminophen (APAP) in our center. While hypotension is a known side effect of IV APAP, it has not been reported with enteral administration. However, burn nurses report episodes of hypotension in critically ill patients following APAP that are reproducible and report hesitancy to give additional doses, fearing a cause-and effect relationship. Conversely, the medical team views the relationship as temporal in nature and believes continued use of APAP an appropriate therapy to tre

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Gaudin, Xavier P., Jacob C. Wochna, Timothy W. Wolff, et al. "Incidence of intraoperative hypotension in acute traumatic spinal cord injury and associated factors." Journal of Neurosurgery: Spine 32, no. 1 (2020): 127–32. http://dx.doi.org/10.3171/2019.7.spine19132.

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OBJECTIVEThe importance of maintaining mean arterial pressure (MAP) > 85 mm Hg for patients with acute spinal cord injury (SCI) is well documented, because systemic hypotension greatly increases the risk of secondary SCI. Current literature focuses on the ICU setting; however, there is a paucity of data describing the changes in MAP in the operating room (OR). In the present study, the authors investigated the incidence of intraoperative hypotension for patients with acute traumatic SCI as well as any associated factors that may have impacted these findings.METHODSThis retrospective study w

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Renjen, Pushpendra Nath, Dinesh Mohan Chaudhari, Nidhi Goyal, and Kamal Ahmed. "Severe acute orthostatic headache: spontaneous intracranial hypotension (SIH)." BMJ Case Reports 14, no. 6 (2021): e243179. http://dx.doi.org/10.1136/bcr-2021-243179.

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The most common cause of spontaneous intracranial hypotension headache is a cerebrospinal fluid (CSF) leakage, but the underlying mechanisms remain unknown. Intracranial hypotension is characterised by diffuse pachymeningeal enhancement on cranial MRI features, low CSF pressure and orthostatic headaches mostly caused by the dural puncture. We report a 31-year-old woman who presented to our services with reports of continuous severe bifrontal headache, which increased on sitting up and resolved on lying down. MRI of the cervical and lumbosacral spine showed signs of CSF leak; hence, patient was

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McDowall, D. Gordon. "CEREBRAL BLOOD FLOW AND METABOLISM IN ACUTE CONTROLLED HYPOTENSION: IMPLICATIONS FOR HYPOTENSIVE ANAESTHESIA." Acta Medica Scandinavica 215, S678 (2009): 97–103. http://dx.doi.org/10.1111/j.0954-6820.1984.tb08667.x.

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Lombardi, F., K. Lucarelli, L. Frassanito, et al. "ROLE OF THE MACHINE LEARNING–DERIVED HYPOTENSION INDEX (HPI) TO CONTAIN INTRAOPERATIVE HYPOTENSION DURING TRANSCATHETER EDGE TO EDGE REPAIR PROCEDURES." European Heart Journal Supplements 26, Supplement_2 (2024): ii117. http://dx.doi.org/10.1093/eurheartjsupp/suae036.291.

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Abstract Introduction Percutaneous endovascular valvular interventions can result in profound hemodynamic instability, elevated burden of intraoperative hypotension (IOH) and related postoperative complications: ischemic stroke, acute kidney injury and increased mortality. Machine learning(ML),a branch of Artificial intelligence (AI), can analyze large volumes of data, find associations and allowing predictive rather than reactive interventions. The Hypotension Prediction Index (HPI)a ML derived algorithm,provides a unitless number from 0 to 100, that increases accordingly to the risk of devel

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Kersey, Cooper B., Fitsumberhan Medhane, Andrew M. Pattock, et al. "Cardiac Arrest during Transesophageal Echocardiogram (TEE) due to Acute Right Ventricular Failure." Case Reports in Cardiology 2021 (December 24, 2021): 1–4. http://dx.doi.org/10.1155/2021/7427127.

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The case of a patient who suffered cardiac arrest while undergoing transesophageal echocardiography (TEE) is presented here. A 75-year-old man with moderate right ventricular (RV) dysfunction and pulmonary hypertension became bradycardic and hypotensive after receiving propofol for procedural sedation. His profound hypotension ultimately led to a pulseless electrical activity (PEA) cardiac arrest. TEE images captured immediately prior to cardiac arrest show a severely dilated and hypokinetic RV, consistent with acute right ventricular failure. This case highlights the potentially fatal consequ

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Aebli, Nikolaus, Jörg Krebs, Gerard Davis, Mark Walton, Michael J. A. Williams, and Jean-Claude Theis. "Fat Embolism and Acute Hypotension During Vertebroplasty." Spine 27, no. 5 (2002): 460–66. http://dx.doi.org/10.1097/00007632-200203010-00005.

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Noble, R. Joe, and Morton E. Tavel. "Management of Acute Myocardial Infarction With Hypotension." Chest 116, no. 5 (1999): 1464–67. http://dx.doi.org/10.1378/chest.116.5.1464.

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SIMPSON, D. L., W. R. MACRAE, J. A. W. WILDSMITH, and B. A. B. DALE. "Acute beta-adrenoreceptor blockade and induced hypotension." Anaesthesia 42, no. 3 (1987): 243–48. http://dx.doi.org/10.1111/j.1365-2044.1987.tb03034.x.

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Kleopa, K. A., and K. Natsiopoulos. "Acute cervical radiculopathies in spontaneous intracranial hypotension." Journal of Neurology 256, no. 3 (2009): 499–501. http://dx.doi.org/10.1007/s00415-009-0095-z.

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Ogoh, Shigehiko, Romain Lericollais, Ai Hirasawa, Sadayoshi Sakai, Hervé Normand, and Damian M. Bailey. "Regional redistribution of blood flow in the external and internal carotid arteries during acute hypotension." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 306, no. 10 (2014): R747—R751. http://dx.doi.org/10.1152/ajpregu.00535.2013.

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The present study examined to what extent an acute bout of hypotension influences blood flow in the external carotid artery (ECA) and the corresponding implications for blood flow regulation in the internal carotid artery (ICA). Nine healthy male participants were subjected to an abrupt decrease in arterial pressure via the thigh-cuff inflation-deflation technique. Duplex ultrasound was employed to measure beat-to-beat ECA and ICA blood flow. Compared with the baseline normotensive control, acute hypotension resulted in a heterogeneous blood flow response. ICA blood flow initially decreased fo

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Sirait, Robert H. "The Incidence of Intraoperative Hypotension and the Importance of Timely Detection." International Blood Research & Reviews 15, no. 1 (2024): 10–17. http://dx.doi.org/10.9734/ibrr/2024/v15i1330.

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Aims: Intraoperative hypotension (IOH) is a common condition faced by anesthesiologists intraoperatively which poignanting patients undergoing surgery under general and neuraxial anesthesia. Its occurrence is associated with dangerous morbid situations found perioperatively that can lead into fatal complications, such as acute kidney failure, direct myocardial injury, and even can end in mortality. Despite advanced closed hemodynamic monitoring and protocols utilizing goal directed therapy, recent trend of management is remaining reactive. Anesthesiologists tend to intervene when the episode o

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El-Mas, Mahmoud M., Jian Zhang, and Abdel A. Abdel-Rahman. "Upregulation of vascular inducible nitric oxide synthase mediates the hypotensive effect of ethanol in conscious female rats." Journal of Applied Physiology 100, no. 3 (2006): 1011–18. http://dx.doi.org/10.1152/japplphysiol.01058.2005.

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Previous reports from our laboratory have shown that ethanol elicits hypotension in female but not in male rats and that this effect of ethanol is estrogen dependent (El-Mass MM and Abdel-Rahman AA. Alcohol Clin Exp Res 23: 624–632, 1999; El-Mass MM and Abdel-Rahman AA. Clin Exp Hypertens 21: 1429–1445, 1999). In the present study, we tested the hypothesis that ethanol lowers blood pressure in female rats via upregulation of the inducible nitric oxide synthase (iNOS) in vascular tissues. The effects of pretreatment with NG-nitro-l-arginine (NOARG; nonselective nitric oxide synthase inhibitor)

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DIACONU, Camelia C., Denisa BUCUR, Daniel BERCEANU, and Ovidiu Gabriel BRATU. "Orthostatic arterial hypotension." Romanian Journal of Medical Practice 12, no. 2 (2017): 12–15. http://dx.doi.org/10.37897/rjmp.2017.1.2.

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Orthostatic hypotension is a persistent decrease of systolic blood pressure by at least 20 mm Hg and/or diastolic blood pressure by at least 10 mm Hg in the first 3 minutes upon standing. Orthostatic hypotension is more common in the elderly patients, being one of relatively frequent causes of hospitalization in the elderly population. Orthostatic hypotension may be primary or secondary, acute or chronic. Its etiology is represented in most cases by the primary or secondary autonomic failure. Clinical manifestations of orthostatic hypotension occur after the transition from supine to standing,

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Schadt, J. C., and R. R. Gaddis. "Renin-angiotensin system and opioids during acute hemorrhage in conscious rabbits." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 258, no. 2 (1990): R543—R551. http://dx.doi.org/10.1152/ajpregu.1990.258.2.r543.

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We measured changes in plasma renin activity (PRA) and used angiotensin-converting enzyme blockade with captopril to evaluate the role of the renin-angiotensin system during hemorrhage and after opioid receptor blockade in conscious rabbits. The increase in PRA after nonhypotensive hemorrhage was not statistically significant. PRA increased sixfold after a hypotensive hemorrhage to a mean arterial pressure less than 40 mmHg. This increase was statistically significant. Captopril altered the hemodynamic response to hemorrhage. The normal increase in vascular resistance early in hemorrhage was r

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Ohtake, P. J., J. K. Walker, and D. B. Jennings. "Renin-angiotensin system stimulates respiration during acute hypotension but not during hypercapnia." Journal of Applied Physiology 74, no. 3 (1993): 1220–28. http://dx.doi.org/10.1152/jappl.1993.74.3.1220.

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We reported that intravenous infusion of angiotensin II (ANG II) stimulated ventilation (VE) in conscious dogs. Other studies in our laboratory have demonstrated that increases in respiration occurred in association with activation of the renin-angiotensin system during acute hypotension and during hypercapnia. Therefore, in conscious dogs (n = 5), we examined the effects of ANG II receptor blockade with intravenous saralasin (0.5 micrograms.kg-1.min-1) on respiratory responses during progressive nitroprusside-induced hypotension and during the ventilatory response to increased inspired fracti

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Terasako, K. "Perioperative Plasma Concentrations of Atrial and Brain Natriuretic Peptides in Patients Undergoing Hip Arthroplasty." Anaesthesia and Intensive Care 30, no. 5 (2002): 588–90. http://dx.doi.org/10.1177/0310057x0203000507.

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Acute hypotension, transient hypoxaemia and elevation of pulmonary artery pressure are well known to occur during cemented arthroplasty. The aim of this prospective clinical study was to characterize the relationship between plasma concentrations of atrial and brain natriuretic peptides (ANP, BNP), and changes in blood pressure in patients undergoing hip arthroplasty. Elevated ANP and BNP levels may be markers of inadequate myocardial reserve. We measured plasma ANP and BNP levels before the operation and 20 minutes after the cementing in 18 patients (54–90 yr). We defined a hypotensive respon

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Pozzi, A., C. Cirelli, A. Merlo, et al. "C26 SIDE EFFECTS OF SGLT–2 INHIBITORS IN PATIENTS WITH HEART FAILURE: A META–ANALYSIS OF RANDOMIZED CONTROLLED TRIALS." European Heart Journal Supplements 25, Supplement_D (2023): D11—D12. http://dx.doi.org/10.1093/eurheartjsupp/suad111.025.

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Abstract Background sodium–glucose cotransoporter–2 inhibitors (SGLT–2Is) improve prognosis in heart failure patients both with reduced (HFrEF) and preserved ejection fraction (HFpEF). However, these drugs can have some side effects. Purpose: estimate the relative risk of side effects in HF patients treated with SGLT–2Is irrespectively from left ventricular EF and setting (acute and chronic HF). Methods 4 randomized controlled trials (RCTs) enrolling patients with HFrEF 3 RCTs enrolling acute HF and 3 RCTs enrolling HFpEF were included. Among side effects, urinary infection, genital infection,

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Hayrab, Chuker. "Acute and Long-Term Outcomes of Hypertensive Crisis and Hypertensive Emergency in Patients Accompanied by Orthostatic Hypotension." Journal of Advances in Medicine and Medical Research 35, no. 18 (2023): 9–15. http://dx.doi.org/10.9734/jammr/2023/v35i185116.

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Introduction: Hypertensive crisis and hypertensive emergency are urgent medical conditions characterized by severely elevated blood pressure levels, often exceeding systolic values of 180 mmHg and/or diastolic values of 120 mmHg. The acute outcomes of hypertensive crisis and hypertensive emergency in patients with orthostatic hypotension include an increased risk of cerebrovascular events, such as ischemic and hemorrhagic strokes, as well as myocardial infarction. Review of Literature: Orthostatic hypotension has been associated with various adverse outcomes, including syncope, stroke, C

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Lear, Christopher A., Joanne O. Davidson, Lindsea C. Booth, et al. "Biphasic changes in fetal heart rate variability in preterm fetal sheep developing hypotension after acute on chronic lipopolysaccharide exposure." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 307, no. 4 (2014): R387—R395. http://dx.doi.org/10.1152/ajpregu.00110.2014.

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Perinatal exposure to infection is highly associated with adverse outcomes. Experimentally, acute, severe exposure to gram-negative bacterial lipopolysaccharide (LPS) is associated with increased fetal heart rate variability (FHRV). It is unknown whether FHRV is affected by subclinical infection with or without acute exacerbations. We therefore tested the hypothesis that FHRV would be associated with hypotension after acute on chronic exposure to LPS. Chronically instrumented fetal sheep at 0.7 gestation were exposed to a continuous low-dose LPS infusion ( n = 12, 100 ng/kg over 24 h, followed

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