Gotowe bibliografie tematyczne / Adenosine deaminase and Metabolism

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Adenosine deaminase and Metabolism”

Autor: Grafiati
Data publikacji: 3 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Adenosine deaminase and Metabolism"

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Shatova, O. P., Eu V. Butenko, Eu V. Khomutov, D. S. Kaplun, I. Eu Sedakov, and I. I. Zinkovych. "Metformin impact on purine metabolism in breast cancer." Biomeditsinskaya Khimiya 62, no. 3 (2016): 302–5. http://dx.doi.org/10.18097/pbmc20166203302.

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Large-scale epidemiological and clinical studies have demonstrated the efficacy of metformin in oncology practice. However, the mechanisms of implementation of the anti-tumor effect of this drug there is still need understanding. In this study we have investigated the effect of metformin on the activity of adenosine deaminase and respectively adenosinergic immunosuppression in tumors and their microenvironment. The material of the study was taken during surgery of breast cacer patients receiveing metformin, and also patients which did not take this drug. The adenosine deaminase activity and su
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Kocic, G., J. Nikolic, T. Jevtovic-Stoimenov, et al. "L-Arginine Intake Effect on Adenine Nucleotide Metabolism in Rat Parenchymal and Reproductive Tissues." Scientific World Journal 2012 (2012): 1–4. http://dx.doi.org/10.1100/2012/208239.

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L-arginine is conditionally essetcial amino acid, required for normal cell growth, protein synthesis, ammonia detoxification, tissue growth and general performance, proposed in the treatment of men sterility and prevention of male impotence. The aim of the present paper was to estimate the activity of the enzymes of adenine nucleotide metabolism:5′-nucleotidase (5′-NU), adenosine deaminase (ADA), AMP deaminase, and xanthine oxidase (XO), during dietary intake of L-arginine for a period of four weeks of male Wistar rats. Adenosine concentration in tissues is maintained by the relative activitie
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Nishiyama, Akira, Shoji Kimura, Hong He, et al. "Renal interstitial adenosine metabolism during ischemia in dogs." American Journal of Physiology-Renal Physiology 280, no. 2 (2001): F231—F238. http://dx.doi.org/10.1152/ajprenal.2001.280.2.f231.

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The present study was conducted to determine the metabolism of renal interstitial adenosine under resting conditions and during ischemia. By using a microdialysis method with HPLC-fluorometric analysis, renal interstitial concentrations of adenosine, inosine, and hypoxanthine were assessed in pentobarbital-anesthetized dogs. Average basal renal interstitial concentrations of adenosine, inosine, and hypoxanthine were 0.18 ± 0.04, 0.31 ± 0.05, and 0.35 ± 0.05 μmol/l, respectively. Local inhibition of adenosine kinase with iodotubercidin (10 μmol/l in perfusate) or inhibition of adenosine deamina
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Schrader, W. P., and C. A. West. "Localization of adenosine deaminase and adenosine deaminase complexing protein in rabbit heart. Implications for adenosine metabolism." Circulation Research 66, no. 3 (1990): 754–62. http://dx.doi.org/10.1161/01.res.66.3.754.

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Schrader, W. P., C. A. West, and N. L. Strominger. "Localization of adenosine deaminase and adenosine deaminase complexing protein in rabbit brain." Journal of Histochemistry & Cytochemistry 35, no. 4 (1987): 443–51. http://dx.doi.org/10.1177/35.4.3546489.

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Adenosine deaminase and adenosine deaminase complexing protein have been localized in rabbit brain. Brains fixed in paraformaldehyde or in Clarke's solution were blocked coronally. Blocks from brains fixed in paraformaldehyde were either frozen in liquid nitrogen or embedded in paraffin. Tissue fixed in Clarke's solution was embedded in paraffin. Sections from each block were stained by the peroxidase-antiperoxidase method for adenosine deaminase or complexing protein using affinity-purified goat antibodies. Adenosine deaminase and complexing protein did not co-localize. Adenosine deaminase wa
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Ashish, Jain, Pandey* Chetan, Sharma Pandey Archana, Dutta Pritha, and Jain RJ. "Effect of 24 Hours Storage at Different Temperature on Pleural Fluid ADA (Adenosine Deaminase) Level." International Journal of Pharmacy and Biological Sciences-IJPBS 13, no. 2 (2023): 113–16. https://doi.org/10.5281/zenodo.8129772.

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Adenosine deaminase, an enzyme produced from lymphocyte and involved in purine metabolism has been extensively studied as a biochemical marker in pleural fluid during investigation for tropical pulmonary eosinophilia. The test assay, low priced, quick, barely forwarded, and can be performed in most laboratories its major physiological role is related to the proliferation and distinction of lymph cell. Pleural fluid of tuberculate start is generally lymph cell preponderant. Different cut off values of Adenosine Deaminase ranging from 30-100 IU/ L have been used in various studies with differing
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Schrader, W. P., C. A. West, U. H. Rudofsky, and W. A. Samsonoff. "Subcellular distribution of adenosine deaminase and adenosine deaminase-complexing protein in rabbit kidney: implications for adenosine metabolism." Journal of Histochemistry & Cytochemistry 42, no. 6 (1994): 775–82. http://dx.doi.org/10.1177/42.6.8189039.

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We evaluated the age-related distribution of adenosine deaminase (ADA) and adenosine deaminase-complexing protein (CP) in rabbit kidney by immunohistochemical staining procedures. Paraffin- or resin-embedded tissue from rabbits < 1 week-4 years of age were stained by the peroxidase-anti-peroxidase (PAP) method for ADA and CP. With the exception of neonates, the qualitative staining pattern of each protein remained generally constant with age. In the cortex, distal tubules, blood vessels, histiocytes, and epithelial cells lining Bowman's capsule stained for ADA. Proximal tubules and glomerul
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Dyakova, M. E., K. B. Vladimirov, D. S. Esmedlyaeva, and P. K. Yablonskiy. "Enzymes of purine metabolism — biomarkers for the diagnostics of tuberculous pleurisy in patients with HIV infection." HIV Infection and Immunosuppressive Disorders 15, no. 1 (2023): 32–40. http://dx.doi.org/10.22328/2077-9828-2023-15-1-32-40.

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The objective of the study was to evaluate the information content of determining the activity of adenosine deaminase and adenosine deaminase-2 in the diagnosis of tuberculous pleurisy in patients with HIV infection.Materials and methods. A total of 378 patients with pleural effusion were retrospectively examined. In 215 cases, tuberculous pleurisy was detected (TP); and 163 patients had non-tuberculous pleural effusion (non-TP). As much as 27 patients in the TP group were HIV co-infected (TP/HIV+), the remaining 188 patients were HIV — negative (TP/HIV–). In all the patients, the activity of
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Jenuth, Jack P., Ellen R. Mably, and Floyd F. Snyder. "Modelling of purine nucleoside metabolism during mouse embryonic development. Relative routes of adenosine, deoxyadenosine, and deoxyguanosine metabolism." Biochemistry and Cell Biology 74, no. 2 (1996): 219–25. http://dx.doi.org/10.1139/o96-022.

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The individual activities for adenosine kinase, deoxyadenosine kinase, adenosine deaminase, deoxyguanosine kinase, and purine nucleoside phosphorylase were determined during days 7 to 13 of mouse embryonic development. Adenosine deaminase increased 74-fold between days 7 and 9; deoxyadenosine kinase increased 5.4-fold during the same interval. Adenosine kinase, deoxyguanosine kinase, and purine nucleoside phosphorylase exhibited less than 2-fold changes in activity between days 7 and 13. Using Michaelis constants for each enzyme and the maximal velocities determined from enzyme assay, the rela
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Hohl, Charlene M. "AMP deaminase in piglet cardiac myocytes: effect on nucleotide metabolism during ischemia." American Journal of Physiology-Heart and Circulatory Physiology 276, no. 5 (1999): H1502—H1510. http://dx.doi.org/10.1152/ajpheart.1999.276.5.h1502.

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The purpose of this study was to examine in situ regulation of AMP deaminase in newborn piglet cardiac myocytes and to determine its role in nucleotide metabolism during ischemia. When a rapid deenergization paradigm was used to assay AMP deaminase, enzyme activity depended on the hormonal and metabolic status of cells just before deenergization. Inosine 5′-monophosphate (IMP) formation was increased 150% in deenergized myocytes pretreated with phorbol 12-myristate 13-acetate (PMA; EC50= 4.7 × 10−8M). This effect was 90% blocked with the protein kinase C (PKC) inhibitor staurosporine. In addit
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Rozprawy doktorskie na temat "Adenosine deaminase and Metabolism"

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Wu, Joe. "HIF-1α in the Heart: Provision of Ischemic Cardioprotection and Remodeling of Nucleotide Metabolism". Digital Commons @ East Tennessee State University, 2014. https://dc.etsu.edu/etd/2450.

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In our studies we found that stabilized expression of HIF-1α in heart led to better recovery of function and less tissue death after 30 minutes of global ischemia, via mechanisms that preserve the mitochondrial polarization. Our group previously showed that HIF-1α conferred ischemic tolerance by allowing cardiomyocytes to use fumarate as an alternative terminal electron acceptor to sustain anaerobic mitochondrial polarization. The source of fumarate was identified as the purine nucleotide cycle (PNC). Here we discovered that HIF-1α upregulates AMP deaminase 2 (AMPD2), the entry point to the PN
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Bona, Karine Santos de. "Efeito do extrato de Syzygium cumini e alterações provocadas pela síndrome metabólica sobre parâmetros bioquímicos e inflamatórios." Universidade Federal de Santa Maria, 2013. http://repositorio.ufsm.br/handle/1/3846.

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Coordenação de Aperfeiçoamento de Pessoal de Nível Superior<br>Metabolic syndrome (MetS) is a complex disorder represented by a set of cardiovascular risk factors, including endothelial dysfunction, oxidative stress and inflammation. Syzygium cumini has hypoglycemic, anti-inflammatory, antipyretic, hypolipidemic and antioxidant properties, besides antiviral and anticarcinogenic action. Considering the importance of MetS in the current economic and social context, morbidity and complications following this pathology, the aim of this study was to assess biochemical and inflammatory parameters in
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Abbott, C. M. "Adenosine deaminase in the wasted mouse." Thesis, University of Reading, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.374695.

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Molaudzi, Mulalo. "The usefulness of the adenosine deaminase assay for diagnosing tuberculosis pleuritis in immunocompromised patients at Dr George Mukhari tertiary laboratory, Pretoria." Thesis, University of Limpopo (Medunsa Campus), 2012. http://hdl.handle.net/10386/671.

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Thesis (MSc (Med)(Microbiology)) -- University of Limpopo, 2012.<br>Mycobacterium tuberculosis is the most common cause of death world-wide and its incidence has been steadily increasing, which is more evident when comparing the global tuberculosis (T8) incidence of 9.24 million in 2006 to 9.27 million cases in 2007. African countries are the second most affected by the epidemic and South Africa is among the 22 highest burden countries most affected by T8 with a very high number of cases relative to the total population. The early diagnosis of tuberculosis and screening of contacts is the corn
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SIGNA, SARA. "NETosis Dysregulation in Adenosine Deaminase 2 deficiency (DADA2)." Doctoral thesis, Università degli studi di Genova, 2022. http://hdl.handle.net/11567/1080132.

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Introduction: Deficiency of Adenosine deaminase 2 (DADA2) is a monogenic autoinflammatory disorder presenting a broad spectrum of clinical manifestations, including vasculitis, immunodeficiency and hematologic disease. The genetic mutations in ADA2 gene have been associated to an insufficient ADA2 activity leading to reduction in deamination of adenosine to deoxyadenosine, and a consequent accumulation of extracellular adenosine. The pathogenic mechanisms investigated so far have elucidated a skewed polarization from the M2 macrophage subtype to the proinflammatory M1 subtype with an increased
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Dinjens, Winandus Nicolaas Maria. "Distribution of adenosine deaminase complexing protein in normal and neoplastic cells." Maastricht : Maastricht : Rijksuniversiteit Limburg ; University Library, Maastricht University [Host], 1988. http://arno.unimaas.nl/show.cgi?fid=5412.

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Björkegren, Emma Katia Madeleine. "Development of gene therapy for the treatment of adenosine deaminase deficiency." Thesis, University College London (University of London), 2005. http://discovery.ucl.ac.uk/1444352/.

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Adenosine deaminase (ADA) severe combined immunodeficiency (SCED) is a life-threatening condition resulting from lack of the ADA enzyme. Consequences include immunodeficiency and non-immunological symptoms such as neurological abnormalities. Bone marrow transplantation (BMT) from a haploidentical donor usually results in complete restoration of immune function. However, the majority of patients do not have a matched donor and are therefore treated with enzyme replacement therapy (PEG-ADA). This treatment is not always fully effective, it is expensive and needs to be administered throughout lif
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Samee, Abdus. "An analysis of the ADAT1 (adenosine deaminase, tRNA- specific 1) gene in cancer." Thesis, Keele University, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.530765.

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Svenningsson, Per. "Striatal adenosine A₂A receptors /." Stockholm, 1998. http://diss.kib.ki.se/search/diss.se.cfm?19981015sven.

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Voigt, Michael D. "The diagnostic value of adenosine deaminase activity in the ascitic fluid of patients with tuberculous peritonitis." Master's thesis, University of Cape Town, 1988. http://hdl.handle.net/11427/25882.

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This study was designed to: 1. Examine the diagnostic value of ADA levels in ascitic fluid, 2. Establish the sensitivity and specificity of this test in the diagnosis of tuberculous peritonitis, in a large number of patients, 3. Establish levels of adenosine deaminase activity which give the best discriminatory information in patients with ascites. 4. Determine what conditions may give rise to false positive or false negative results. 5. Finally, the study was designed to assess the relative diagnostic accuracy of previously used biochemical and haematological data, such as ascites total prote
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Książki na temat "Adenosine deaminase and Metabolism"

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L, Tritsch George, New York Academy of Sciences., and Conference on Adenosine Deaminase in Disorders of Purine Metabolism and in Immune Deficiency (1984 : New York, N.Y.), eds. Adenosine deaminase in disorders of purine metabolism and in immune deficiency. New York Academy of Sciences, 1985.

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Kate, Johannes ten. Adenosine deaminase complexing protein as a marker in cancer studies. [s.n.], 1985.

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Chu, Peter Pui Tak. Retroviral-mediated human adenosine deaminase gene transfer into human hematopoietic progenitor and stem cells. National Library of Canada, 1995.

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I, Stefanovich, and Okyayuz-Baklouti I, eds. Role of adenosine in cerebral metabolism and blood flow. VNU Science Press, 1987.

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W, Stone T., ed. Adenosine in the nervous system. Academic Press, 1991.

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F, Eccleston J., and Royal Society (Great Britain), eds. Nucleoside triphosphatases in energy transduction, cellular regulation and information transfer in biological systems: A discussion meeting of the Royal Society. Portland Press, 1992.

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1932-, Mukohata Yasuo, Morales Manuel F, Fleischer Sidney, Yamada Kagaku Shinko Zaidān, and Yamada Conference on Energy Transduction in ATPases (11th : 1985 : Kōbe-shi, Japan), eds. Perspectives of biological energy transduction. Academic Press, 1987.

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1945-, Richter Ch, ed. ADP-ribosylation of proteins: Enzymology and biological significance. Springer-Verlag, 1987.

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1949-, Althaus F. R., Hilz H. 1924-, Shall S. 1932-, and International Symposium on ADP-Ribosylation Reactions (7th : 1984 : Vitznau, Switzerland), eds. ADP-ribosylation of proteins. Springer-Verlag, 1985.

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K, Jacobson Myron, and Jacobson Elaine L, eds. ADP-ribose transfer reactions: Mechanisms andbiological significance. Springer-Verlag, 1989.

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Części książek na temat "Adenosine deaminase and Metabolism"

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Kelley, William N., Peter E. Daddona, and Martin B. van der Weyden. "Characterization of Human Adenosine Deaminase." In Ciba Foundation Symposium 48 - Purine and Pyrimidine Metabolism. John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720301.ch16.

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Simmonds, H. Anne, A. Sahota, C. F. Potter, D. Perrett, K. Hugh-Jones, and J. G. Watson. "Purine Metabolism in Adenosine Deaminase Deficiency." In Ciba Foundation Symposium 68 - Enzyme Defects and Immune Dysfunction. John Wiley & Sons, Ltd., 2008. http://dx.doi.org/10.1002/9780470720516.ch16.

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Berkvens, Th M., D. Valerio, M. G. C. Duyvesteyn, et al. "The Human Gene for Adenosine Deaminase." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5104-7_33.

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Geiger, Jonathan D., and James I. Nagy. "Adenosine Deaminase and [3H] Nitrobenzylthioinosine as Markers of Adenosine Metabolism and Transport in Central Purinergic Systems." In Adenosine and Adenosine Receptors. Humana Press, 1990. http://dx.doi.org/10.1007/978-1-4612-4504-9_7.

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Wortmann, R. L., J. A. Veum, and H. S. Cheung. "Adenosine Deaminase Deficiency and Chondro-Osseous Dysplasia." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5104-7_13.

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Daddona, Peter E., Stuart H. Orkin, and William N. Kelley. "Expression Defects of Mutant Human Adenosine Deaminase." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5104-7_34.

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Gallagher, Kim P., Thomas B. McClanahan, Bradley J. Martin, et al. "Does Adenosine Deaminase Inhibition Protect Ischemic Myocardium?" In Purine and Pyrimidine Metabolism in Man VIII. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-2584-4_62.

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Strauss, Phyllis R. "Murine Lymphocytes and Lymphocyte Cell Lines Secrete Adenosine Deaminase." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-1248-2_44.

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Marshall, M. Ernest, Lela K. Riley, and Mary Sue Coleman. "Detection of mRNA for Adenosine Deaminase Using Biotinylated Probes." In Purine and Pyrimidine Metabolism in Man V. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5104-7_39.

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Williams, David A., and Thomas Moritz. "Genetic Manipulation of Hematopoietic Stem Cells in Adenosine Deaminase Deficiency." In Purine and Pyrimidine Metabolism in Man VIII. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4615-2584-4_86.

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Streszczenia konferencji na temat "Adenosine deaminase and Metabolism"

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Batubara, Winda Rizki Pebrina, Tri Nugraha Susilawati, and Dono Indarto. "Analysis of Naringin and Hesperidin Compounds in Berastagi Sweet Orange (<i>Citrus Sinensis L</i>) Peel Extract." In 8th International Conference on Advanced Material for Better Future. Trans Tech Publications Ltd, 2025. https://doi.org/10.4028/p-aj6obu.

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Lipid metabolism is the process of breaking down triglycerides into fatty acids and glycerol, which are oxidized to provide energy. Peroxisome proliferation-activated receptor (PPAR)- alpha and adenosine monophosphate protein kinase (AMPK) are key proteins to increase endogenous lipolysis, which become important targets for weight loss management. Orange peels generally contain hesperidin and naringin compounds which can improve lipid metabolism through lipolysis, cholesterol regulation, inhibition of fat absorption, antioxidants and anti-inflammation. This study aimed to identify hesperidin a
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Fajt, James, Alyn Jenkins, and Anna Murphy. "Development and Field Application of a New Bacteria Monitoring Technique." In CORROSION 2019. NACE International, 2019. https://doi.org/10.5006/c2019-13158.

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Absrract Accurate bacteria population monitoring is an important tool in oil and gas production to mitigate microbiologically influenced corrosion (MIC), prevent reservoir souring and optimize biocide treatment strategies. Serum serial dilution testing (SSDT) is a flawed yet widely accepted traditional method for bacteria testing in the oil and gas industry. The serum bottle test is difficult to perform correctly in the best of conditions and impractical to use in most if not all oil and gas field applications. The SSDT culture method returns an indirect estimate of bacteria levels that are of
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Schlund, N. J., K. Zabelny, and J. I. Peters. "Deceptive Deaminase: Coccidioides Empyema With Elevated Adenosine Deaminase." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a3881.

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Pereira, Flávio Ribeiro, Blanca Helena Rios Gomes Bica, Maria Pompeya Olmedo de Lopes de Figueiredo, et al. "Deficiency of Adenosine Deaminase 2: a rare disease." In XXXIX Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2022. http://dx.doi.org/10.47660/cbr.2022.2212.

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Sharoyan, S., L. Karapetyan, R. Harutyunyan, S. Mardanyan, and A. Antonyan. "SAT0051 Citrullination of adenosine deaminase isoforms in rheumatoid arthritis." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.1433.

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Tofovic, Stevan P., Victor Bilan, Edwin K. Jackson, and OLGA Rafikova. "Role Of Plasma Adenosine Deaminase In Hemolysis-Induced Pulmonary Hypertension." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6335.

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PARENTE, JOSÉ SÁVIO MENEZES, AMANDA VIRGINIA BATISTA CAVALCANTE, and THAÍS GUERREIRO JORGE ROCHA. "POLYARTERITIS NODOSA OR DEFICIENCY OF ADENOSINE DEAMINASE 2?: CASE REPORT." In 36º Congresso Brasileiro de Reumatologia. Editora Blucher, 2019. http://dx.doi.org/10.5151/sbr2019-207.

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Cho, Yu Ji, Jong Deog Lee, Eun Young Yun, et al. "Clinical Characteristics Of Tuberculous Pleurisy Patients With Low Adenosine Deaminase Levels." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a1781.

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Kiryukhina, Larisa, Marina Dyakova, and Diljara Esmedlyaeva. "Adenosine deaminase in the COPD pathogenesis in patients with pulmonary tuberculosis." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2718.

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Trajcevska, Mirjana, and Aleksandar Sandevski. "Activity of lysozyme and adenosine deaminase in patients with pulmonary tuberculosis." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa4762.

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Raporty organizacyjne na temat "Adenosine deaminase and Metabolism"

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Jander, Georg, Gad Galili, and Yair Shachar-Hill. Genetic, Genomic and Biochemical Analysis of Arabidopsis Threonine Aldolase and Associated Molecular and Metabolic Networks. United States Department of Agriculture, 2010. http://dx.doi.org/10.32747/2010.7696546.bard.

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Streszczenie:
Since the amino acids threonine and isoleucine can be limiting in mammalian diet and there is interest in increasing their abundance in certain crop plants. To meet this need, a BARD proposal was written with two main research objectives: (i) investigate new avenues for manipulating threonine and isoleucine content in plants and (ii) study the role of threonine aldolase in plant metabolism. Research conducted to meet these goals included analysis of the sub-cellular localization of threonine aldolase in the plant, analysis of metabolic flux in developing embryos, over- and under-expression of
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