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Artykuły w czasopismach na temat „Anti-IL-17”

Autor: Grafiati
Data publikacji: 2 czerwca 2025
Data aktualizacji: 31 lipca 2025

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1

Ly, Karen, Mary P. Smith, Quinn Thibodeaux, Vidhatha Reddy, Wilson Liao, and Tina Bhutani. "Anti IL-17 in psoriasis." Expert Review of Clinical Immunology 15, no. 11 (2019): 1185–94. http://dx.doi.org/10.1080/1744666x.2020.1679625.

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Buckland, Jenny. "Anti-TNF and anti-IL-17 antibodies—better together!" Nature Reviews Rheumatology 10, no. 12 (2014): 699. http://dx.doi.org/10.1038/nrrheum.2014.183.

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Daude, M., and S. Barbarot. "Maladie de Darier et anti IL 17." Annales de Dermatologie et de Vénéréologie - FMC 3, no. 8 (2023): A282. http://dx.doi.org/10.1016/j.fander.2023.09.448.

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Ndongo-Thiam, Ndieme, Alice Clement, Jean-Jacques Pin, Diane Razanajaona-Doll, and Pierre Miossec. "Negative association between autoantibodies against IL-17, IL-17/anti-IL-17 antibody immune complexes and destruction in rheumatoid arthritis." Annals of the Rheumatic Diseases 75, no. 7 (2016): 1420–22. http://dx.doi.org/10.1136/annrheumdis-2016-209149.

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Sales-Campos, Helioswilton, Chamberttan Souza Desidério, Rafael Obata Trevisan, et al. "Anti-IL-4, Anti-IL-17, and Anti-IFN-Gamma Activity in the Saliva of Amblyomma sculptum Ticks." International Journal of Molecular Sciences 26, no. 10 (2025): 4734. https://doi.org/10.3390/ijms26104734.

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The saliva of hematophagous arthropods, such as ticks and triatomines, contains bioactive ligands capable of modulating immune molecules, including cytokines. Cytokines play a critical role in immune regulation and have therapeutic relevance in inflammatory and immune-mediated diseases. Despite recent advances, identifying cytokine-binding molecules remains a significant challenge. Interferon-gamma (IFN-γ), interleukin-4 (IL-4), and interleukin-17 (IL-17) are key cytokines involved in inflammation, adaptive immunity, and host defense. This study evaluated the ability of salivary components fro
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Selimov, Pavel, Rositsa Karalilova, Ljubinka Damjanovska, et al. "Rheumatoid arthritis and the proinflammatory cytokine IL-17." Folia Medica 65, no. 1 (2023): 53–59. http://dx.doi.org/10.3897/folmed.65.e72448.

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Introduction: Rheumatoid arthritis (RA) is the most common inflammatory joint disease. Various proinflammatory cytokines are involved in the pathogenesis of this chronic disorder. It is characterized by the presence of autoantibodies, such as rheumatoid factor and antibodies against citrullinated peptides. The present study focuses on investigation of possible association between the proinflammatory cytokine interleukin 17 and anti-CCP, anti-MCV, and anti-CarP antibodies seropositivity in RA patients. Aim: To assess serum levels of interleukin 17 (IL-17) in patients with rheumatoid arthritis a
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Burchill, Matthew A., Dean T. Nardelli, Douglas M. England, et al. "Inhibition of Interleukin-17 Prevents the Development of Arthritis in Vaccinated Mice Challenged with Borrelia burgdorferi." Infection and Immunity 71, no. 6 (2003): 3437–42. http://dx.doi.org/10.1128/iai.71.6.3437-3442.2003.

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ABSTRACT We showed that Borrelia burgdorferi-vaccinated interferon gamma-deficient (IFN-γ0) mice challenged with the Lyme spirochete developed a prominent chronic severe destructive osteoarthropathy. The immune response underlying the development of the severe destructive arthritis involves interleukin-17 (IL-17). Treatment of vaccinated IFN-γ0 mice challenged with B. burgdorferi with anti-IL-17 antibody delayed the onset of swelling of the hind paws but, more importantly, inhibited the development of arthritis. Histopathologic examination confirmed that treatment with anti-IL-17 antibody prev
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8

Maniati, Eleni, and Thorsten Hagemann. "IL-17 mediates resistance to anti-VEGF therapy." Nature Medicine 19, no. 9 (2013): 1092–94. http://dx.doi.org/10.1038/nm.3333.

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Lux, Sebastian. "Psoriasis: Anti-IL-17-Inhibitor als Rescue-Therapie?" hautnah dermatologie 35, no. 2 (2019): 30. http://dx.doi.org/10.1007/s15012-019-3036-2.

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Cooper, Andrea M. "IL-17 and anti-bacterial immunity: Protectionversustissue damage." European Journal of Immunology 39, no. 3 (2009): 649–52. http://dx.doi.org/10.1002/eji.200839090.

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Wendling, Daniel, Clément Prati, Mickael Chouk, and Frank Verhoeven. "Effects of anti-IL-23 and anti-IL-17: The hidden side of spondyloarthritis polymorphism?" Joint Bone Spine 87, no. 1 (2020): 5–7. http://dx.doi.org/10.1016/j.jbspin.2019.06.012.

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Sato, Shuzo, Xian Zhang, Haruki Matsumoto, et al. "Transcription factor Fli-1 impacts IL-17 expression and affects immune cell infiltration into the kidney in lupus-prone mice." Journal of Immunology 204, no. 1_Supplement (2020): 59.15. http://dx.doi.org/10.4049/jimmunol.204.supp.59.15.

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Abstract Objectives Transcription factor Fli-1 affects lupus nephritis development through regulating several cytokines and chemokines. Th17 immune response is important to maintain inflammation in lupus nephritis. The purpose of this study is to elucidate whether Fli-1 affects renal IL-17 expression and influence immune cell infiltration into the kidney. Methods Sera were collected from Fli-1-heterozygous (Fli-1+/−) MRL/lpr mice and wild-type (WT) littermates, and the concentration of IL-17 was measured by enzyme-linked immunosorbent assay. Expression of IL-17 and related molecules were measu
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13

Hamada, Kaoru, Yu Sawada, Ryosuke Hino, and Motonobu Nakamura. "HTLV-1 carrier psoriasis patients treated by anti-IL-23/IL-17." Australasian Journal of Dermatology 59, no. 2 (2017): e154-e154. http://dx.doi.org/10.1111/ajd.12701.

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Lee, Hajeong, Jae Wook Lee, Kyung Don Yoo, et al. "Cln 3-requiring 9 is a negative regulator of Th17 pathway-driven inflammation in anti-glomerular basement membrane glomerulonephritis." American Journal of Physiology-Renal Physiology 311, no. 3 (2016): F505—F519. http://dx.doi.org/10.1152/ajprenal.00533.2015.

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T helper 17 (Th17) lymphocytes promote renal inflammation in anti-glomerular basement membrane glomerulonephritis (anti-GBM GN), and signal transducer and activator of transcription 3 (STAT3) mediates activation of Th17 lymphocytes by IL-6 and transforming growth factor-β (TGF-β). Cln 3-requiring 9 (Ctr9), a subunit of RNA polymerase-associated factor complex (PAFc), regulates the transcription of IL-6/STAT3-dependent genes. Here, we investigated the role of Ctr9 in regulating Th17-driven inflammation in anti-GBM GN. In mice, STAT3β or IL-17 knockout ameliorated anti-GBM autoantibody-induced r
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Indah Jayanthi, Anak Agung, Luh Made Mas Rusyati, Ni Made Dwi Puspawati, I. Gusti Ayu Agung Elis Indira, I. Gusti Nyoman Darmaputra, and I. Gusti Ayu Agung Dwi Karmila. "Kadar interleukin-17 plasma berkorelasi positif dengan kadar imunoglobulin M (IgM) anti phenolic glycolipid-1 (PGL-1) pada narakontak serumah pasien kusta tipe multibasiler." Intisari Sains Medis 14, no. 1 (2023): 445–51. http://dx.doi.org/10.15562/ism.v14i1.1694.

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Introduction: Household contacts are the group most at risk of contracting subclinical leprosy, which acts as a transmission source. Immunoglobulin M (IgM) anti-Phenolic Glycolipid-1 (PGL-1) serological indicates the abundance of M. leprae in person and Interleukin-17 (IL-17) levels play an important role in preventing clinical leprosy. Thus, this study aims to determine the correlation of IL-17 to IgM-anti PGL-1 in household contacts of multibacillary type leprosy patients. Methods: This study was an analytic observational study with a cross-sectional approach. The research was conducted from
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16

Siloşi, Isabela, Mihail Virgil Boldeanu, Manole Cojocaru, et al. "The Relationship of Cytokines IL-13 and IL-17 with Autoantibodies Profile in Early Rheumatoid Arthritis." Journal of Immunology Research 2016 (2016): 1–10. http://dx.doi.org/10.1155/2016/3109135.

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Aims.In the present study, we aimed to assess the concentrations of IL-13 and IL-17 in serum of patients with early rheumatoid arthritis (eRA), the investigation of correlation between the concentrations of these cytokines and disease activity score, and the concentration of some autoantibodies and the evaluation of the utility of IL-13 and -17 concentration measurements as markers of disease activity.Materials and Methods. Serum samples were collected from 30 patients and from 28 controls and analysed parameters.Results. The serum concentrations of IL-13, IL-17, anti-CCP, and IgM-RF were stat
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17

Cho, Mila, Seon-Yeong Lee, Jin-Sil Park, et al. "Up-Regulation of Stromal Cell-Derived Factor by IL-17 and IL-18 via Phosphatidylinositol 3-Kinase Dependent Pathway (171.8)." Journal of Immunology 188, no. 1_Supplement (2012): 171.8. http://dx.doi.org/10.4049/jimmunol.188.supp.171.8.

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Abstract IL-17 producing Th17 has a key role in the pathogenesis of autoimmune inflammation. Among various cytokines which are interwoven with the IL-17 pathway, members of IL-1beta family including IL-18 have recently gained significance. In the present study, we stimulated synovial fibroblasts with the combination of IL-17 and IL-18, and quantified the cellular production of stromal cell-derived factor-1 (SDF-1) with ELISA and its mRNA with RT-PCR. Both IL-17 and IL-18 significantly increased the level of SDF-1, not only individually but also synergistically (p<0.05). The synergism wa
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18

Garber, Ken. "Anti-IL-17 mAbs herald new options in psoriasis." Nature Biotechnology 30, no. 6 (2012): 475–76. http://dx.doi.org/10.1038/nbt0612-475.

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Zelante, Teresa, Antonella De Luca, Carmen D' Angelo, Silvia Moretti, and Luigina Romani. "IL-17/Th17 in anti-fungal immunity: What's new?" European Journal of Immunology 39, no. 3 (2009): 645–48. http://dx.doi.org/10.1002/eji.200839102.

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Hölttä, Veera, Taina Sipponen, Mia Westerholm-Ormio та ін. "In Crohn's Disease, Anti-TNF-α Treatment Changes the Balance between Mucosal IL-17, FOXP3, and CD4 Cells". ISRN Gastroenterology 2012 (14 червня 2012): 1–6. http://dx.doi.org/10.5402/2012/505432.

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Aim. In Crohn's disease (CD), anti-TNF-α treatment is a potent medication. We aimed to characterize the effect of anti-TNF-α treatment on T effector and regulatory cells. Material and Methods. We studied T-effector and regulatory cells on cellular and mRNA levels in intestinal biopsy samples from 13 Crohn's disease patient. Biopsies were obtained at baseline and 3 months after anti-TNF-α treatment, and from 14 inflammation-free control subjects. Results. Patients had higher numbers of ileal IL-17+ and forkhead box P3 (FOXP3)+ cells than did control subjects, both before ( P≤0.001 and P≤0.05, r
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Miyagawa, Hanae, Hiromichi Hara, Jun Araya, et al. "Characteristics of anti-IL-17/23 biologics-induced interstitial pneumonia in patients with psoriasis." PLOS ONE 16, no. 1 (2021): e0245284. http://dx.doi.org/10.1371/journal.pone.0245284.

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Objectives Anti-IL-17/23 biologics are increasingly used to treat psoriasis. We aimed to elucidate characteristics of drug-induced interstitial pneumonia (DIIP) caused by anti-IL-17/23 biologics. Methods We retrospectively analyzed the clinical data of psoriasis patients treated with anti-IL-17/23 biologics. Chest CT was performed to evaluate DIIP. Serum KL-6 levels were measured before treatment (baseline) and during treatment. Results A total of 603 psoriasis patients were treated with anti-IL-17/23 biologics with mean follow-up of 21.1 months. Six patients developed DIIP at mean 14 months a
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Frieder, Jillian, Dario Kivelevitch, Isabel Haugh, Ian Watson, and Alan Menter. "Anti-IL-23 and Anti-IL-17 Biologic Agents for the Treatment of Immune-Mediated Inflammatory Conditions." Clinical Pharmacology & Therapeutics 103, no. 1 (2017): 88–101. http://dx.doi.org/10.1002/cpt.893.

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Zuniga, Luis, Wen-Jun Shen, Barbara Joyce-Shaikh, et al. "IL-17 — a novel adipose tissue cytokine with anti-adipogenic and pro-osteogenic properties (114.6)." Journal of Immunology 186, no. 1_Supplement (2011): 114.6. http://dx.doi.org/10.4049/jimmunol.186.supp.114.6.

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Abstract Inflammatory mediators have the potential to impact a surprising range of diseases, including obesity, obesity associated metabolic syndrome, and autoimmunity. Adipocyte development and their function in lipid storage and metabolism are regulated in a complex fashion by inflammatory cytokines. Although IL-17 appears to mediate inflammation-associated bone loss, studies indicate IL-17 also has important homeostatic roles in facilitating wound repair and promoting cell proliferation. Recent evidence indicates IL-17 has an anti-adipogenic role while at the same time having an unexpected
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CHIBA, Kenji. "A new orally active anti-rheumatic drug targets IL-15 and IL-17." Japanese Journal of Clinical Immunology 30, no. 5 (2007): 375–82. http://dx.doi.org/10.2177/jsci.30.375.

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Sheet, Madha Mohammed, Laith Abdul-Elah Kamel та Jamal Nasser Farhood. "The Role of Anti-TNFα Therapy in the Amelioration of Disease Burden in Patients with Refractory Rheumatoid Arthritis". Al Mustansiriyah Journal of Pharmaceutical Sciences 14, № 1 (2014): 14–22. http://dx.doi.org/10.32947/ajps.v14i1.121.

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To evaluate the changes in anti cyclic citrullinated peptide (Anti-CCP) antibodies,rheumatoid factor (RF), interleukins 17 and 10 (IL-17 and IL-10), high sensitivity C- reactiveprotein (hs CRP) and T regulatory cells (Treg) count following an anti TNF-α biological agent(etanercept) therapy in patients with rheumatoid arthritis.Refractory rheumatoid arthritis patients who failed treatment with DMARDs (diseasemodifying anti rheumatic drugs) were treated with etanercept for three months. Serum and bloodsamples were tested before and after therapy for six markers including ACCP, RF, IL- 10, IL 17,
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Xu, Jiangnan, Yamei Zhang, Jordi Ochando, Liwu Li, and Yaozhong Ding. "IL-17 plays a critical role in airway epithelial injury after transplantation." Journal of Immunology 198, no. 1_Supplement (2017): 82.24. http://dx.doi.org/10.4049/jimmunol.198.supp.82.24.

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Abstract Bronchiolitis obliterans syndrome (BOS) is the major obstacle limiting long-term allograft survival of lung transplantation. Airway epithelium is the primary target in obliterative airway diseases, and the degree of epithelial injury is closely correlated with the development of BOS. IL-17 plays a critical role in mediating inflammatory responses and is involved in transplant graft rejection. In this study, we performed murine orthotopic allogeneic trachea transplantations and examined the role of IL-17 in airway epithelial injury after transplantation. Expression of Th1 and Th17 cyto
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Mou, Zhirong, Ping Jia, Shiby Kuriakose, Forough Khadem, and Jude E. Uzonna. "Interleukin-17-Mediated Control of Parasitemia in Experimental Trypanosoma congolense Infection in Mice." Infection and Immunity 78, no. 12 (2010): 5271–79. http://dx.doi.org/10.1128/iai.00168-10.

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ABSTRACT BALB/c mice are highly susceptible to experimental Trypanosoma congolense infections, whereas C57BL/6 mice are relatively resistant. Infected highly susceptible BALB/c mice die of systemic inflammatory response syndrome. Because interleukin-17 (IL-17) and Th17 cells regulate inflammatory responses, we investigated their role in the pathogenesis of experimental African trypanosomiasis in mice. We show that the production of IL-17 by spleen and liver cells and the serum IL-17 level increased after T. congolense infection in mice. Interestingly, infected highly susceptible BALB/c mice pr
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Kuroi, Taiga, Sachiyo Okamoto, Kyosuke Saeki та ін. "Anti-IL-12/23 p40 Antibody Attenuates Chronic Graft Versus Host Disease Via Suppression of IFN-γ/IL-17-Producing Cells". Blood 124, № 21 (2014): 1095. http://dx.doi.org/10.1182/blood.v124.21.1095.1095.

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Abstract Chronic graft-versus-host disease (GVHD) remains a major cause of late death and morbidity following allogeneic hematopoietic cell transplantation. Recently, in addition to Th2 cells, Th1 and Th17 cells have been shown to contribute to chronic GVHD progression. IL-12 induces Th1 cells and IL-23 plays a role in stabilizing and/or amplifying Th17 cells as well as in inducing IFN-γ/IL-17 double-producing cells. Because monoclonal antibody (mAb) targeting the p40 subunit common to both IL-12 and IL-23 can inhibit both IL-12 and IL-23 receptor-mediated signaling, we investigated the effect
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Gargiulo, Luigi, Luciano Ibba, Alessandra Narcisi, et al. "Anti-IL-17/23 Drugs for the Treatment of Moderate-to-Severe Hidradenitis Suppurativa in Patients With Concomitant Psoriasis: A Multicenter Retrospective Study." Dermatology Practical & Conceptual 14, no. 4 (2024): e2024250. http://dx.doi.org/10.5826/dpc.1404a250.

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Introduction: Psoriasis and hidradenitis suppurativa (HS) are chronic inflammatory diseases with significant overlap in their immunologic pathways, which involve cytokines such as tumor necrosis factor-alfa, interleukin (IL)-17, and IL-23. Current treatment options for HS are limited, as only adalimumab and secukinumab are approved for severe cases. Given the overlapping pathogenetic features between HS and psoriasis, anti-IL-17 and anti-IL-23 drugs could represent valuable treatments for the management of HS. Objectives: We sought to evaluate the effectiveness and safety of anti-IL-17 and ant
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Velden, Joachim, Hans-Joachim Paust, Elion Hoxha, et al. "Renal IL-17 expression in human ANCA-associated glomerulonephritis." American Journal of Physiology-Renal Physiology 302, no. 12 (2012): F1663—F1673. http://dx.doi.org/10.1152/ajprenal.00683.2011.

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Interleukin-17A (IL-17) promotes inflammatory renal tissue damage in mouse models of crescentic glomerulonephritis, including murine experimental autoimmune anti-myeloperoxidase glomerulonephritis, which most likely depends on IL-17-producing Th17 cells. In human anti-neutrophil cytoplasmic antibody (ANCA)-associated glomerulonephritis, however, the cellular sources of IL-17 remain to be elucidated. Therefore, we analyzed human kidney biopsies of active necrotizing and crescentic ANCA-associated glomerulonephritis by immunohistochemistry using an IL-17-specific antibody and by immunofluorescen
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Caspi, Rachel, Phyllis Silver, Daniel Cua, Zoe Chen, Chi-Chao Chan, and Dror Luger. "IL-23 and IL-17 in pathogenesis of experimental ocular autoimmunity: requirement for IL–23 may extend beyond its role in sustaining the IL-17 effector response (129.26)." Journal of Immunology 178, no. 1_Supplement (2007): S222. http://dx.doi.org/10.4049/jimmunol.178.supp.129.26.

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Abstract Experimental autoimmune uveitis (EAU) represents autoimmune uveitis in humans. Here we examined the role of the IL-23/IL-17 pathway in EAU. We immunized IL-23p19, IL-12p35, IL-12p40, IFN-γ and IL-17 KO mice with the uveitogenic Ag IRBP. Alternatively, we treated wild type mice immunized for EAU with Abs to IL-23p19 or to IL-17 throughout the disease course (prevention), or only during the effector phase (reversal). IL-23p19 KO were resistant to EAU, showing reduced Ag-specific DTH, IL-2, IFN-γ, IL-17, IL-1β, IL-6 and IL-5. In contrast, IL-12p35 and IFN-γ KO mice developed exacerbated
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Chai, Lichao, Jing Wang, and Yan Wei. "Fucoxanthin improves functional recovery of orbitopathy in Graves’ disease by downregulating IL-17 mRNA expression in a mouse model." Tropical Journal of Pharmaceutical Research 19, no. 5 (2020): 933–41. http://dx.doi.org/10.4314/tjpr.v19i5.4.

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Purpose: To explore the efficacy of fucoxanthin (FX), a carotenoid, against inflammation via inhibition of IL-17 mRNA expression, and its anti-oxidant activity in Graves’ orbitopathy (GO)-induced mice model.Methods: The effects of FX on IL-6, IL-8, IL-17, MCP-1, and TNF-α, in orbital fibroblast tissues extracted from GO-induced BALB/c mice was investigated. Anti-oxidative stress markers, 8-hydroxy-2’- deoxyguanosine (8-OHdG) and malondialdehyde (MDA) levels were quantified in tear samples collected from GO-induced FX treated mice.Results: FX administration in cultured human orbital fibroblast
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Chabaud, Martine, François Fossiez, Jean-Luc Taupin, and Pierre Miossec. "Enhancing Effect of IL-17 on IL-1-Induced IL-6 and Leukemia Inhibitory Factor Production by Rheumatoid Arthritis Synoviocytes and Its Regulation by Th2 Cytokines." Journal of Immunology 161, no. 1 (1998): 409–14. http://dx.doi.org/10.4049/jimmunol.161.1.409.

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Abstract IL-17 is a cytokine produced by CD4 T cells that activates the production of inflammatory mediators by synoviocytes. To study the contribution of soluble factors in the interaction between T cells and synoviocytes in rheumatoid arthritis (RA), we looked at the effect of IL-17 on these cells in the presence of cytokines classified as pro (IL-1)- and anti-inflammatory (IL-4, IL-13, IL-10). Both human rIL-1β and rIL-17 induced IL-6 and leukemia inhibitory factor (LIF) production by synovial fibroblasts in a dose-dependent manner. After 7 days of culture, optimal concentrations of IL-1β i
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Amlong, Corey A., Dean T. Nardelli, Sara Heil Peterson, Thomas F. Warner, Steven M. Callister, and Ronald F. Schell. "Anti-Interleukin-15 Prevents Arthritis in Borrelia-Vaccinated and -Infected Mice." Clinical and Vaccine Immunology 13, no. 2 (2006): 289–96. http://dx.doi.org/10.1128/cvi.13.2.289-296.2006.

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ABSTRACT We showed previously that interleukin-17 (IL-17) plays a significant role in the induction of arthritis associated with Borrelia vaccination and challenge. Little information, however, is available about the chain of immunologic events that leads to the release of IL-17. The production of IL-17 has been linked to stimulation of memory cells by IL-15. Therefore, we hypothesized that IL-15 is involved in the induction of arthritis associated with Borrelia vaccination and infection of mice. Here we present evidence that treatment of Borrelia-vaccinated and -infected mice with anti-IL-15
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Misra, Durga Prasanna, Smriti Chaurasia, and Ramnath Misra. "Increased Circulating Th17 Cells, Serum IL-17A, and IL-23 in Takayasu Arteritis." Autoimmune Diseases 2016 (2016): 1–8. http://dx.doi.org/10.1155/2016/7841718.

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Introduction.Th17,γδT, NK, and NKT cells in peripheral blood and serum IL-17 and IL-23 in Takayasu arteritis (TA) were measured and correlated with disease activity.Methods.Th17 (anti-CD3APC, CD4PECy7, and IL-17PE), NKT, NK (anti-CD3APC, CD56FITC), andγδT (anti-CD3FITC andγδTCRAPC) cells were enumerated by flow cytometry in peripheral blood of 30 patients with TA (ACR1990 criteria) and 20 healthy controls, serum IL-17 and IL-23 measured by ELISA. Relation with disease activity (NIH criteria, ITAS2010) was analyzed (using nonparametric tests, median with interquartile range).Results.Mean age of
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Pitmon, Elise V., Ju Chen, Xiaoyang Ye, and Kepeng Wang. "A Direct Link between IL-17 and Regulatory T cells in Autoimmune Colitis." Journal of Immunology 204, no. 1_Supplement (2020): 143.17. http://dx.doi.org/10.4049/jimmunol.204.supp.143.17.

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Abstract Inflammatory bowel diseases are rising in prevalence globally. While current treatments aim to achieve remission, there remains no cure for this disease. IL-17 family cytokines are important players in inflammation, but clinical trials using anti-IL-17A or IL-17RA blocking antibodies have failed for Crohn’s disease, due to exacerbated inflammation in some patients. While anti-IL-17 antibodies have shown efficacy in the treatment of other autoimmune diseases like psoriasis, patients can often develop Crohn’s disease as a side effect of these drugs. These data suggest a protective role
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José, Anthar Ávalos Narváez, Daniel Solís Dittrich Kevin, Elisa González Robles María, Jiménez Zaragoza Michelle, Arturo Ovilla Alvarez Ricardo, and Aréchiga López Mayra. "Advances in Monoclonal Antibody Therapy for Moderate to Severe Psoriasis: Targeting TNF, IL-17, And IL-23 Pathways for Optimal Disease Management." International Journal of Medical Science and Clinical Research Studies 5, no. 05 (2025): 766–69. https://doi.org/10.5281/zenodo.15502235.

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Psoriasis, a chronic immune-mediated dermatological disorder, significantly impacts patients' quality of life, particularly in its moderate to severe forms. The advent of biologic therapies, specifically monoclonal antibodies (mAbs), has revolutionized the treatment landscape. This article provides an in-depth analysis of the efficacy, safety, and mechanistic insights of anti-TNF (e.g., adalimumab, infliximab), anti-IL-17 (e.g., secukinumab, ixekizumab), and anti-IL-23 (e.g., guselkumab, risankizumab) agents in the management of moderate to severe psoriasis. Clinical trials and real-world evid
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Atzeni, Fabiola, Antonio Carriero, Laura Boccassini, and Salvatore D'Angelo. "Anti-IL-17 Agents in the Treatment of Axial Spondyloarthritis." ImmunoTargets and Therapy Volume 10 (May 2021): 141–53. http://dx.doi.org/10.2147/itt.s259126.

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Ke, Yan, Ke Liu, Guo-Qiang Huang, et al. "Anti-Inflammatory Role of IL-17 in Experimental Autoimmune Uveitis." Journal of Immunology 182, no. 5 (2009): 3183–90. http://dx.doi.org/10.4049/jimmunol.0802487.

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Puig, L. "Brodalumab: The first anti-IL-17 receptor agent for psoriasis." Drugs of Today 53, no. 5 (2017): 283. http://dx.doi.org/10.1358/dot.2017.53.5.2613690.

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Noell, Claire, Brianna McQuade, Alice Gottlieb, and David Rosmarin. "Anti IL-17 flared psoriasis in a patient on secukinumab." Dermatologic Therapy 30, no. 4 (2017): e12505. http://dx.doi.org/10.1111/dth.12505.

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Zhang, Rui, Jiang Qian, Jie Guo, Yi-fei Yuan, and Kang Xue. "Suppression of Experimental Autoimmune Uveoretinitis by Anti-IL-17 Antibody." Current Eye Research 34, no. 4 (2009): 297–303. http://dx.doi.org/10.1080/02713680902741696.

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Brown, Gabrielle, Mona Malakouti, Eva Wang, John Y. Koo, and Ethan Levin. "Anti-IL-17 phase II data for psoriasis: A review." Journal of Dermatological Treatment 26, no. 1 (2014): 32–36. http://dx.doi.org/10.3109/09546634.2013.878448.

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El-Lessy, Fatma. "Downregulation of proinflammatory cytokines and dynamic expression of TGF-β1 molecules induced by anti-IL-17 mAb in murine schistosomiasis mansoni". Egyptian journal of Immunology 32, № 1 (2025): 16–26. https://doi.org/10.55133/eji.320102.

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Hepato-intestinal schistosomiasis is characterized by severe pathological changes at advanced chronic stages, including granulomatous lesions and liver fibrosis. The objective of our research was to assess the dynamic expression of profibrotic molecules, the transforming growth factor beta 1 (TGF-β1), and proinflammatory cytokines immunomodulation induced by interleukin 17 (IL-17) neutralization in murine Schistosomiasis mansoni. The study included 56 specific pathogen-free male C57BL/6 mice, divided into 3 main groups: GI uninfected normal controls, GII S. mansoni infected with 70±5 cercariae
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Banuelos, Jesus, Soon Shin, and Nick Lu. "Distinct glucocorticoid sensitivity of Th17 cytokines in murine T hybridomas and primary cells (IRC11P.428)." Journal of Immunology 194, no. 1_Supplement (2015): 197.10. http://dx.doi.org/10.4049/jimmunol.194.supp.197.10.

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Abstract Cytokine suppression contributes to the anti-inflammatory actions of glucocorticoids (GCs). However, IL-17 has been reported to be resistant to GC regulation in asthma, nasal polyps, and Crohn’s disease. In contrast, a negative GC-response element has been identified in the promoter region of the IL-17 gene and IL-17 was sensitive to GC suppression in a murine asthma model. To clarify whether IL-17 and other Th17 cytokines are sensitive to GC regulation, we generated Th17 hybridoma cells by fusing in vitro differentiated murine (bl/6) Th17 cells with BW5147 thymoma. Two clones selecti
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Prabhala, Rao H., Mariateresa Fulcinitti, Dheeraj Pelluru, et al. "Multiple Myeloma Cells Express IL-17A Creating An Autocrine Loop: An Attractive Therapeutic Target." Blood 122, no. 21 (2013): 3113. http://dx.doi.org/10.1182/blood.v122.21.3113.3113.

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Abstract We have previously demonstrated that Th17 cells, which produce IL-17A, are significantly elevated in peripheral blood and bone marrow (BM) of patients with Multiple Myeloma (MM) and IL-17A promotes MM cell growth and survival, both in vitro and in vivo via IL-17A receptor. We have recently evaluated and observed that anti-IL-17A monoclonal antibody (mAb) significantly inhibited MM cell growth in vitro, while IL-17A induced proliferation of MM cells compared to control. We have also observed significant down-regulation of IL-6 production by anti-IL-17A mAb in MM-BMSC co-culture. Import
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Fried, Richard, Mark Lebwohl, Miriam Bettencourt, John Koo, and Abby Jacobson. "Onset of Plaque Psoriasis Treatment Responses With Anti-IL-17/IL-23 Biologic Therapies." Journal of Drugs in Dermatology 21, no. 8 (2022): 854–60. http://dx.doi.org/10.36849/jdd.66791.

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Matusiak, Łukasz, Justyna Szczęch, Andrzej Bieniek, Danuta Nowicka-Suszko, and Jacek C. Szepietowski. "Increased interleukin (IL)-17 serum levels in patients with hidradenitis suppurativa: Implications for treatment with anti-IL-17 agents." Journal of the American Academy of Dermatology 76, no. 4 (2017): 670–75. http://dx.doi.org/10.1016/j.jaad.2016.10.042.

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Yin, Yuanqin, Fei Li, Jing Shi, Songlin Li, Jingjing Cai, and Youhong Jiang. "MiR-146a Regulates Inflammatory Infiltration by Macrophages in Polymyositis/Dermatomyositis by Targeting TRAF6 and Affecting IL-17/ICAM-1 Pathway." Cellular Physiology and Biochemistry 40, no. 3-4 (2016): 486–98. http://dx.doi.org/10.1159/000452563.

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Background/Aims: The primary objective of this study was to investigate the role of miR-146a in inducing the inflammatory infiltration of macrophages in polymyositis/dermatomyositis (PM/DM) through targeting TNF receptor associated factor 6 (TRAF6), which may further down-regulate the Interleukin-17 (IL-17)/Intercellular Adhesion Molecule 1 (ICAM-1) pathway. Methods: Biopsies were collected from PM/DM patients and healthy volunteers. PM/DM model establishment and macrophage isolation were performed on Sprague Dawley (SD) rats. Model rats and macrophages were treated with anti-IL-17, anti-ICAM-
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El-Zawawy, Hanaa Tarek, Huda Fahmy Farag, Mona Mohamed Tolba, and Hanaa Abdalbasit Abdalsamea. "Improving Hashimoto’s thyroiditis by eradicating Blastocystis hominis: Relation to IL-17." Therapeutic Advances in Endocrinology and Metabolism 11 (January 2020): 204201882090701. http://dx.doi.org/10.1177/2042018820907013.

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Background: Hashimoto’s thyroiditis (HT) is a common autoimmune disorder that causes significant morbidity. Interleukin (IL)-17 was identified as a major contributing factor in the pathogenesis of HT. Blastocystis hominis (BH) is a very common infection and has been shown to be associated with several diseases. Our aim was to determine serum IL-17 level in HT patients with and without BH infection and the effect of eradicating BH in patients with HT. Methods: A prospective cohort study was conducted on 20 HT patients not infected with BH (group I), 20 HT patients infected with BH (group II), a
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