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Rozprawy doktorskie na temat „Anti ischemic”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Chunder, Rittika [Verfasser]. "Anti-neuronal autoantibodies in ischemic stroke and in anti-MAG IgM Gammopathy / Rittika Chunder." Lübeck : Zentrale Hochschulbibliothek Lübeck, 2018. http://d-nb.info/1153437724/34.

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Yusof, Mozow. "Antecedent hydrogen sulfide elicits an anti-inflammatory phenotype in postischemic murine small intestine." Diss., Columbia, Mo. : University of Missouri-Columbia, 2007. http://hdl.handle.net/10355/4779.

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Thesis (Ph. D.)--University of Missouri-Columbia, 2007.<br>The entire dissertation/thesis text is included in the research.pdf file; the official abstract appears in the short.pdf file (which also appears in the research.pdf); a non-technical general description, or public abstract, appears in the public.pdf file. Vita. Includes bibliographical references.

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Raghavan, Aparna. "Neuroprotective Potential of Withania Somnifera in Cerebral Ischemia." University of Toledo Health Science Campus / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=mco1416570371.

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Parikh, Suchi Vipin. "Ependymin peptide mimetics that assuage ischemic damage increase gene expression of the anti-oxidative enzyme SOD." Link to electronic thesis, 2003. http://www.wpi.edu/Pubs/ETD/Available/etd-0429103-132144.

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Li, Xiaofei. "Improving Stem Cell Survival and Differentiation in Ischemic and Inflammatory Tissues." The Ohio State University, 2016. http://rave.ohiolink.edu/etdc/view?acc_num=osu1469174124.

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Inagaki, Koichi. "Anti-Ischemic Effect of a Novel Cardioprotective Agent JTV519 Is Mediated Through Specific Activation of δ-Isoform of Protein Kinase C in Rat Ventricular Myocardium". Kyoto University, 2000. http://hdl.handle.net/2433/180830.

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Richard, Sébastien. "Explorations des fonctions plaquettaires exposées à l'aspirine au décours de l'accident vasculaire cérébral ischémique." Thesis, Nancy 1, 2011. http://www.theses.fr/2011NAN10132.

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L'aspirine est l'anti-plaquettaire le plus largement prescrit à la phase aiguë de l'accident vasculaire cérébral (AVC) ischémique. Cependant, la survenue de récidives, malgré cette prescription, est fréquente. La description de l'effet de l'aspirine sur l'activité plaquettaire durant cette phase n'a jamais été réalisée. Elle pourrait mettre en évidence une moindre réponse plaquettaire et aider à établir de nouvelles stratégies thérapeutiques. Cinquante patients, ont reçu par voie orale 300 mg d'aspirine, suite à un AVC ischémique. Ensuite, des prélèvements sanguins ont été réalisés : entre 2 e

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Hoxha, M. "THE POTENTIAL THERAPEUTIC ROLE OF MONTELUKAST AND NEW HYBRID AGENTS, TXA2 ANTAGONIST-COX-2 INHIBITORS IN CARDIOVASCULAR EVENTS." Doctoral thesis, Università degli Studi di Milano, 2016. http://hdl.handle.net/2434/347148.

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The main target of my PhD research project was to explore new alternatives to reduce the cardiovascular (CV) risk targeting the arachidonic acid metabolites. Hence, I have been part of two different projects, one studying multitarget compounds with balanced COXIB and TP receptor antagonist properties, and the other evaluating the potential role of a leukotriene (LT) antagonist drug such as montelukast in improving the CV outcome. In reference to the first project, new multitarget compounds were synthesized at the University of Turin, by substituting the carboxylic function of Lumiracoxib.

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Huang, Zhigao. "Implication of anti-apoptotic genes in neuronal death following focal cerebral ischemia in rats." Thesis, University of Ottawa (Canada), 2002. http://hdl.handle.net/10393/6270.

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Accumulating biochemical and morphological evidence suggests that apoptosis contributes to neuronal cell death following cerebral ischemia. Recent research which has examined changes in expression of proapoptotic proteins has further strengthened the important role of apoptosis in ischemic cell death. In this thesis I first addressed the role of apoptosis in ischemic death by examining p53, which is itself a complex multifunctional tumor suppresser gene, following transient focal ischemia. Of particular note were the alterations of the anti-apoptosic gene, naip, that were observed under stress

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10

Bouma, Maarten Godfried. "The anti-inflammatory potential of adenosine an experimental study with emphasis on ischemia-reperfusion injury /." Maastricht : Maastricht : UPM, Universitaire Pers Maastricht ; University Library, Maastricht University [Host], 1997. http://arno.unimaas.nl/show.cgi?fid=5945.

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Keller, Melanie. "Cytoprotective and Anti-Inflammatory Properties of the Atrial Natriuretic Peptide during Ischemia/Reperfusion and Endotoxemia." Diss., lmu, 2005. http://nbn-resolving.de/urn:nbn:de:bvb:19-47771.

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Fernandes, Mara Yone Soares Dias. "Efeito neuroprotetor do α-bisabolol em camundongos submetidos Ã isquemia cerebral focal permanente." Universidade Federal do CearÃ, 2015. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=14886.

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FundaÃÃo Cearense de Apoio ao Desenvolvimento Cientifico e TecnolÃgico<br>O acidente vascular cerebral (AVE) Ã uma das principais causa de mortalidade no Brasil, acometendo cerca de 200.000 indivÃduos anualmente. A fisiopatologia do AVE isquÃmico envolve uma complexa cascata de eventos como a inflamaÃÃo e o estresse oxidativo que podem Ã morte neuronal e dÃficits cognitivos. O α-bisabolol Ã um Ãlcool sesquiterpeno, monocÃclico, que ocorre na natureza e Ã encontrado como constituinte majoritÃrio do Ãleo essencial sintÃtico da Matricaria chamomilla, que possui atividade antiinflamatÃria, an

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13

Wang, Jie. "The Study of the Effects of (1S,2E,4R,6R,-7E,11E)-2,7,11-cembratriene-4,6-diol on Microglia Polarization Using an Ischemia in Vitro Model." University of Cincinnati / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1491559717910191.

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14

Parent-Massin, Dominique. "Necrose ischemique du colon par prise de medicaments anti-hypertenseurs : origine criminelle ? a propos d'un cas." Reims, 1991. http://www.theses.fr/1991REIMM039.

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15

Tang, Yina. "Comparative study on the morphology, chemistry, metabolism and anti-myocardial ischemia activity of three medicinal species of dioscorea." HKBU Institutional Repository, 2015. https://repository.hkbu.edu.hk/etd_oa/166.

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As folk medicines used in China for decades, Dioscorea nipponica Makino (DN, 穿龍薯蕷), D. panthaica Prain et Burkill (DP, 黃山藥), and D. zingiberensis C. H. Wright (DZ, 盾葉薯蕷) are regarded as having more or less similar traditional therapeutic actions, such as regulating qi, relieving pain, and dispersing swelling. It is noteworthy that, of the 49 species of the genus Dioscorea (薯蕷屬) distributed in China, only these three have been successfully developed as effective single-herb medicines for treating cardiovascular diseases by the modern pharmaceutical industry. Usually considered as the bioactive

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16

Kovacic, Hervé. "Contribution a l'etude des proprietes anti-ischemiques du bepridil : action sur la na,k-atpase et l'echangeur na/h." Aix-Marseille 2, 1993. http://www.theses.fr/1993AIX22950.

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Guillou, Sophie. "Évaluation de molécules à activité anti-Xa dans la cardioprotection." Thesis, Poitiers, 2018. http://www.theses.fr/2018POIT1404.

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L’infarctus du myocarde est une des premières causes de décès dans le monde. Sa prise en charge repose sur une reperfusion précoce. De façon paradoxale, la reperfusion induit des lésions délétères qui participent à la nécrose de l’organe. Parmi les stratégies de cardioprotection qui visent à limiter la formation de ces lésions, il a été décrit que l’utilisation d’anticoagulants au moment de la reperfusion avait un effet bénéfique sur la taille finale d’infarctus chez l’animal. La cardioprotection induite par ces molécules serait liée à la modulation des phénomènes thrombo-inflammatoires impliq

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18

Habib, Pardes [Verfasser], Till Akademischer Betreuer] Marquardt, and Marc [Akademischer Betreuer] [Spehr. "Erythropoietin dependent regulation of the anti-apoptotic TMBIM family members FAIM2 and GRINA after murine cerebral ischemia / Pardes Habib ; Till Marquardt, Marc Spehr." Aachen : Universitätsbibliothek der RWTH Aachen, 2020. http://nbn-resolving.de/urn:nbn:de:101:1-2020091114321632513714.

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Habib, Pardes Verfasser], Till [Akademischer Betreuer] Marquardt, and Marc [Akademischer Betreuer] [Spehr. "Erythropoietin dependent regulation of the anti-apoptotic TMBIM family members FAIM2 and GRINA after murine cerebral ischemia / Pardes Habib ; Till Marquardt, Marc Spehr." Aachen : Universitätsbibliothek der RWTH Aachen, 2020. http://d-nb.info/1217503838/34.

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Ritter, Thomas. "Anti-inflammatorische und zytoprotektive Gentherapie am Beispiel der experimentellen Transplantation." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2003. http://dx.doi.org/10.18452/13842.

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Ziel der Arbeit war, zu untersuchen, ob der gezielte Einsatz gentherapeutischer Methoden zu einer Verhinderung der Abstoßung allogener Transplantate bzw. zu einer Verhinderung der Induktion des Ischämie-/Reperfusionsschadens in verschiedenen Transplantationsmodellen der Ratte beitragen kann. Dabei wurden zwei Schwerpunkte gesetzt: Zum einen wurde auf den ex-vivo Gentransfer von therapeutischen Molekülen direkt in das Transplantat mit Hilfe von rekombinanten Adenoviren fokussiert. Zum anderen wurde das Potenzial von retroviral modifizierten, allospezifischen T-Zellen als Träger therapeutischer

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21

LE, PEILLET ELIANE. "Etude des consequences comportementales et histologiques d'une ischemie cerebrale globale chez le rat. Application a la recherche de l'activite anti-ischemique de differents agents pharmacologiques." Paris 6, 1990. http://www.theses.fr/1990PA066207.

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Notre travail a eu pour objet d'etudier les consequences comportementales et histologiques d'ischemies cerebrales globales de durees variables chez le rat. Nous avons mis en evidence, que l'intensite et la reversibilite des troubles comportementaux (fonctions sensori-motrices, motilite, exploration et fonctions mnesiques) sont directement liees a la duree de l'ischemie. De plus, l'intensite des atteintes neuronales, des regions selectivement vulnerables (hoppocampe, striatum et cortex), sont egalement fonction de la duree de l'ischemie. Par ailleurs, il n'existe pas de correlation entre l'impo

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22

D'Amico, Francesco. "Infusione sistemica e loco-regionale di N-ACETIL-CISTEINA nel prelievo di fegato: uno studio prospettico randomizzato controllato." Doctoral thesis, Università degli studi di Padova, 2009. http://hdl.handle.net/11577/3427328.

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Background/Aims. Anti-oxidant agents have the potential to reduce the ischemia-reperfusion damage of organs for liver transplantation (LT). In this randomized prospective study, we tested the impact on the post-LT outcome of the infusion of N-acetilcisteina (NAC) during the liver procurement procedure. Methods. The NAC protocol scheduled: a systemic NAC infusion (30 mg/kg) one hour before the beginning of liver procurement; a loco-regional NAC infusion (300 mg through the portal vein) just before cross-clamping. Between December 2006 and December 2007, 56 grafts, assigned to adult candidates

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23

Freitas, Vera Lorentz de Oliveira. "Efeito do antagonista de receptor do peptídeo liberador de gastrina na resposta inflamatória por lesão de reperfusão pulmonar em ratos." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2014. http://hdl.handle.net/10183/108314.

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A lesão de isquemia e reperfusão pulmonar (LIRI) é a principal causa de disfunção precoce do enxerto após o transplante pulmonar. Diferentes substâncias têm sido utilizadas na tentativa de proteger o pulmão na fase inicial pós-transplante e melhorar o desempenho do enxerto com pouco resultado tanto a curto quanto em longo prazo. O peptídeo liberador gastrina (GRP) está envolvido na indução de respostas imunes inatas adaptativas por induzir a quimiotaxia de mastócitos, migração de macrófagos e proliferação de células T e fibroblastos. Este agente foi capaz de proteger o dano tecidual em outros

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Morrey, Christopher. "Cardioprotective anti-adreneraic effects of histamine H3- receptor activation in myocardial ischemia : GPy-dependent inhibition of voltage-operated Ca2+ channels and prevention of a local cardiac RAS activation via reduction of neuropeptide-mediated mast c." Thesis, University of Surrey, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.520471.

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Feliu, Catherine. "Implication des récepteurs P1 et P2 dans la protection des cellules endothéliales au cours de l’hypoxie-reoxygenation Complementary Role of P2 and adenosine receptors in ATP induced-anti-apoptotic effects against hypoxic injury of HUVECs Current knowledge on the role of P2Y receptors in cardioprotection against ischemia-reperfusion Intra-extracellular quantification of nucleotides and adenosine using UHPLCHRMS: improvement of robustness by the use of ascorbic acid in mobile phase Description of the novel cytoprotective action pathways of ticagrelor against hypoxic lesions at the endothelium." Thesis, Reims, 2019. http://www.theses.fr/2019REIMM202.

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Au cours de l’ischémie cardiaque, la lésion est initiée au niveau de l’endothélium, et progresse aux cardiomyocytes environnants. La signalisation purinergique joue un rôle important au cours d’épisodes l’ischémie/reperfusion (I/R). De multiples travaux ont portés sur l’étude des mécanismes de protection des nucléotides et nucléosides, sans pour autant étudier leurs rôles spécifiques sur l’endothélium. Dans ce travail, nous mettons en évidence une augmentation des concentrations extracellulaires en ATP et adénosine provenant de l’endothélium soumis à une hypoxie. Au niveau endothélial, nous me

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Ajmo, Craig T. "Alternative targets for the treatment of stroke." [Tampa, Fla.] : University of South Florida, 2007. http://purl.fcla.edu/usf/dc/et/SFE0002114.

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Lin, Wan-Ying, and 林宛瑩. "Anti-apoptotic mechanism of ischemic preconditioning against hypoxic-ischemic injury in the neonatal rat brain." Thesis, 2005. http://ndltd.ncl.edu.tw/handle/7j466r.

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碩士<br>國立成功大學<br>分子醫學研究所<br>93<br>Neonatal hypoxic-ischemic (HI) brain injury is a major cause of neonatal mortality and long-term disability. Currently, there are still no effective therapies against neonatal HI brain injury. A sublethal stress, such as a brief episode of ischemia, before a lethal injury may reduce neuronal death against subsequent lethal injury; a phenomenon called “preconditioning”. Elucidating the underlying mechanisms of preconditioning may provide potential neuroprotective therapy for neonatal HI brain injury. Permanent ligation of unilateral carotid artery followed by

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Dunlop, Kate. "Neuroprotective Effects of a Novel Apple Peel Extract AF4 in a Mouse Model of Hypoxic-Ischemic Brain Injury." 2011. http://hdl.handle.net/10222/13997.

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The neuroprotective effects of AF4, a flavonoid-enriched extract derived from the peel of Northern Spy apples (containing quercetin-3-O-glucoside, quercetin-3-O-galactoside, quercetin-3-O-rhamnoside, quercetin-3-O-rutinoside, epicatechin, and cyanidin-3-O-galactoside) were examined by assessing neuronal loss and motor impairment resulting from hypoxic-ischemic (HI) brain injury in adult C57BL/6 mice. Relative to vehicle treatment (water, 10mL/kg/day), oral administration of AF4 (50 mg/kg/day) for 3 days reduces HI-induced neuronal loss in the striatum and hippocampus, motor impairments, and re

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Dunlop, Kate Elizabeth. "Neuroprotective Effects of a Novel Apple Peel Extract AF4 in a Mouse Model of Hypoxic-Ischemic Brain Injury." 2011. http://hdl.handle.net/10222/15722.

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The neuroprotective effects of AF4, a flavonoid-enriched extract derived from the peel of Northern Spy apples (containing quercetin-3-O-glucoside, quercetin-3-O-galactoside, quercetin-3-O-rhamnoside, quercetin-3-O-rutinoside, epicatechin, and cyanidin-3-O-galactoside) were examined by assessing neuronal loss and motor impairment resulting from hypoxic-ischemic (HI) brain injury in adult C57BL/6 mice. Relative to vehicle treatment (water, 10mL/kg/day), oral administration of AF4 (50 mg/kg/day) for 3 days reduces HI-induced neuronal loss in the striatum and hippocampus, motor impairments, and re

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"In vivo and in vitro studies of the anti-oxidative, anti-inflammatory and anti-apoptotic effects of Gastrodiae Rhizoma water extract on ischemic stroke." 2013. http://library.cuhk.edu.hk/record=b5884430.

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Hung, Sze Man.<br>Thesis (M.Phil.)--Chinese University of Hong Kong, 2013.<br>Includes bibliographical references (leaves 186-192).<br>Electronic reproduction. Hong Kong : Chinese University of Hong Kong, [2012] System requirements: Adobe Acrobat Reader. Available via World Wide Web.<br>Abstracts also in Chinese.

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Lin, Ting-An, and 林庭安. "The anti-oxidant activity of ALDH3A1 in ischemic astrocytes is up-regulated by ZC008." Thesis, 2016. http://ndltd.ncl.edu.tw/handle/53603888930208813786.

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碩士<br>國立中興大學<br>生命科學系所<br>104<br>Cerebral ischemia can lead to severe brain damage or even death of the patients. Ischemia/reperfusion (I/R)-induced reactive oxygen species (ROS) and brain''s low antioxidant activity all contribute to the increased oxidative stress in ischemic brain that consequently can lead to severe brain injury. How to down regulate the oxidative stress in ischemic brain has continuously been a hot issue in the control of cerebral ischemia. It is known that brain astrocytes have the highest anti-oxidative activity compared to other brain cell types and ALDH3A1 (aldehyde

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Liang, Philip. "Novel Formulations of Antioxidant and Anti-inflammatory Drugs to Ameliorate Ischemic Damage Measured In Vitro." Thesis, 2009. http://hdl.handle.net/1807/17434.

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The two of major pathways that cause ischemic damage are oxidative stress and inflammation. To decreasing oxidative stress and inflammation, new anti-oxidant and anti-inflammatory agents are tested in ischemic models. In order to study ALRX828C anti-inflammatory properties, an in vivo six-day old air pouch model of inflammation was used to evaluate the anti-inflammatory potential of ALRX828C. Also, the dose response of ALRX828C for TNFα (IC50 = 30 μM) and IL-17 (IC50 = 1.3 μM) were determined by using human peripheral blood mononuclear cell cultures stimulated with ionomycin and PMA. To examin

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Cook, Vanessa L. "The role of novel anti-inflammatory drugs in the repair of ischemic-injured equine jejunum." 2008. http://www.lib.ncsu.edu/theses/available/etd-07082008-220258/unrestricted/etd.pdf.

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HUANG, TZU-LUN, and 黃子倫. "Corticosteroid and mechanism of anti-inflammation in a rodent model of non-arteritic anterior ischemic optic neuropathy." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/11859411280426176489.

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博士<br>慈濟大學<br>醫學科學研究所<br>105<br>Non-arterial anterior ischemic optic neuropathy (NAION) is the most common optic neuropathy in aged adult. The pathogenesis and treatments of NAION have been controversial topics. We supposed key mechanism is that reduction of blood flow around optic nerve (ON) head then lead to subsequent inflammation over ON head and secondary progressive death of retinal ganglion cells (RGCs). Setting up one reliable animal model in order to study the pathogenesis and potential treatment of NAION is the most critical method to get answers. In our experimental design of anteri

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Ching-LanCheng and 鄭靜蘭. "Pharmacoepidemiologic Research for Ischemic Stroke in Elderly Patients Who Taking Cyclo-oxygenase-2 inhibitors or Conventional Non-steroidal Anti-inflammatory Drugs." Thesis, 2010. http://ndltd.ncl.edu.tw/handle/49057700758994998832.

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博士<br>國立成功大學<br>藥學生物科技研究所<br>98<br>Background: An increased risk of cardiovascular events with non-steroid anti-inflammatory drugs (NSAIDs) and cyclo-oxygenase-2 (COX-2) inhibitors were evident in the literature, which have placed the risk-benefit profile of these COX-2 inhibitors under public scrutiny and two COX-2 inhibitors subsequently been withdrawn from the market. As a result, clinicians have been encouraged to weigh the potential benefits and risks of the NSAIDs prior to prescribing them. Nonetheless, aspirin interaction with other NSAIDs has become new concerns recently. Despite a gro

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Carreiro, Carolina Cunha. "Preditores de boa circulação colateral pial no AVC isquémico agudo." Master's thesis, 2017. http://hdl.handle.net/10316/82231.

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Trabalho de Projeto do Mestrado Integrado em Medicina apresentado à Faculdade de Medicina<br>Introdução: A presença de boa circulação colateral pial (CCP) no doente com AVC isquémico (AVCi), submetido a terapêutica de revascularização arterial, associa-se a melhor prognóstico. Desconhece-se a totalidade de fatores que explicam a variabilidade interindividual da CCP. A sua identificação poderá contribuir para melhorar o tratamento do doente com AVCi agudo.Materiais e Métodos: Estudo observacional, de coorte histórica, que incluiu doentes internados no serviço de Neurologia do Centro Hospitalar

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Zerche, Maria. "Einfluss von Anti-NMDA-Rezeptor-NR1-Autoantikörpern bei ApoE4-bedingter chronischer Beeinträchtigung der Blut-Hirn-Schranke." Doctoral thesis, 2018. http://hdl.handle.net/11858/00-1735-0000-002E-E454-5.

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"In vivo investigation of the anti-oxidant, anti-blood coagulation and behavioral studies of danshen-gegen aqueous extract in cerebral ischemia." 2011. http://library.cuhk.edu.hk/record=b5894842.

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Lam, Ming Yiu.<br>"September 2011."<br>Thesis (M.Phil.)--Chinese University of Hong Kong, 2011.<br>Includes bibliographical references (leaves 149-169).<br>Abstracts in English and Chinese.<br>Thesis / Assessment Committee --- p.ii<br>Abstract (English) --- p.iii<br>Abstract (Chinese) --- p.vi<br>Acknowledgements --- p.viii<br>Table of contents --- p.x<br>List of figures --- p.xvi<br>List of tables --- p.xix<br>Abbreviations --- p.xx<br>Chapter Chapter 1 --- Introduction --- p.1<br>Chapter 1.1 --- Cerebral stroke --- p.1<br>Chapter 1.2 --- Epidemiology --- p.2<br>Chapter 1.3 --- Ri

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Huang, Bo-Yau, and 黃柏堯. "Anti-apoptotic role for KMUP-1 in simulated ischemia-inducedmyocardial cell injury." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/05358826020024606979.

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碩士<br>高雄醫學大學<br>藥理學研究所碩士班<br>95<br>Cardiac myocytes undergo apoptosis under condition of ischemia. Myocardial oxidative stress and Ca2+ overload induced by ischemia may be involved in the development and progression of myocardial apoptosis. We hypothesized that KMUP-1 could prevent the ischemia-induced reactive oxygen species production and Ca2+ overload, leading to decreased calcium-responsive signaling in myocardial dysfunction. We showed that serum/glucose deprivation and hypoxia, components of ischemia in vivo, resulted in apoptosis of H9c2 cardiomyoblasts. H9c2 cells apoptosis is evidence

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Chen, Hsuan-Ying, and 陳宣穎. "The synergistic effect of Ginkgo biloba extract (EGb761) combined with anti-ischemia drugs to infarct volume% of gerbil in a transient focal cerebral ischemia." Thesis, 2004. http://ndltd.ncl.edu.tw/handle/91838934015675383633.

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碩士<br>靜宜大學<br>食品營養研究所<br>92<br>Previous studies indicated that Ginkgo biloba extract (EGb761) provides beneficial effects in animal model and patients during acute stroke. The purpose of this study was to investigate the synergistic effect of EGb761 combined with anti-ischemia drugs MK801 & FK506 to infarct volume % of gerbil in a transient focal cerebral ischemia. Experiment 1: the animals were divided into two groups : control and EGb761 group, fed standard chow or EGb761(100 mg/kg/day) for 7 days, then induced ischemia by ligating right middle cerebral artery (MCA) and common carotid artery

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Keller, Melanie [Verfasser]. "Cytoprotective and anti-inflammatory properties of the atrial natriuretic peptide during ischemia-reperfusion and endotoxemia / Melanie Keller." 2005. http://d-nb.info/978868188/34.

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Hsu, Shih-Min, та 徐士閔. "The Mechanism Study Underlying the Anti-oxidant and Anti-inflammation Activities of Ergostatrien-3β-ol from Antrodia camphorata Using Ischemia and Reperfusion Injury Rat Model". Thesis, 2014. http://ndltd.ncl.edu.tw/handle/df946a.

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碩士<br>國立臺灣大學<br>食品科技研究所<br>102<br>Antrodia camphorate has been considered as one of most efficient and precious Chinese herbal medicines, and it has been used as remedy for curing food poisoning, diarrhea, stomachache, hypertension, itchy skin, and liver cancer. Recently, liver protecting characteristic of Antrodia camphorata was believed as the most important bioactivity. Ergostatrien-3β-ol (EK100), which exists in both fruiting body and mycelia, is one of compounds with high liver-protecting property； however, investigation for this compound is scarce. In the present research, animal model w

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Cheng, Chin-Yi, and 程錦宜. "The Anti-Inflammatory and Antioxidative Effects and Mechanisms of Ferulic acid on Cerebral Ischemia-Reperfusion Injury in Rats." Thesis, 2008. http://ndltd.ncl.edu.tw/handle/07585475660656920407.

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博士<br>中國醫藥大學<br>中國醫學研究所博士班<br>97<br>Both Angelica sinensis (Oliv.) Diels (AS) and Ligusticum chuanxiong Hort., (LC) have been used to treat stroke in Traditional Chinese Medicine for centuries. Ferulic acid (4-hydroxy-3-methoxycinnamic acid, FA), a component of both AS and LC, plays a role in neuroprotection. The purpose of this study was to investigate the anti-inflammatory and antioxidative effects and mechnaisms of FA on cerebral ischemia-reperfusion injury in rats. Rats underwent 2, 10, 24 and 36h of reperfusion after 90 min middle cerebral artery occlusion (MCAo). First, the cerebral infa

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Chao, Chia-Sheng, and 趙家聲. "Porcine model intra-abdominal vital organ inflammation induced by abdominal aorta ischemia-reperfusion injury and the anti-inflammation mechanism of hyperin." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/t7jnx5.

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博士<br>高雄醫學大學<br>藥學系博士班<br>105<br>Ischemia-reperfusion injury (IRI) induced complicated inflammation and pathology of vital organs. For seeking possible strategies to treat IRI, we conduct two studies. Part I: Using a porcine model, we evaluated the influence of different endovascular balloon occlusion (EBO) time periods on intra-abdominal pressure (IAP) and the association between various pathophysiologic indicators and reperfusion time. Twelve healthy 3-month-old domestic piglets were subjected to ischemia-reperfusion injury using EBO within the abdominal aorta. Animals were grouped as A, B,

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BARRECA, Liboria. "Polimorfismo a singolo nucleotide dei geni pro-infiammatori /anti-infiammatori e trombotico/fibrinolitici in pazienti con ictus ischemico acuto e relazione con il sottotipo TOAST." Doctoral thesis, 2011. http://hdl.handle.net/10447/95101.

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Yang, Bing. "Signification de la mort cellulaire dans l’insuffisance rénale aiguë et la fibrose causée par ischémie-reperfusion." Thèse, 2017. http://hdl.handle.net/1866/20238.

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