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Artykuły w czasopismach na temat „Antigliadin antibodies”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Wong, D., M. Dwinnel, M. Schulzer, M. Nimmo, B. R. Leavitt, and S. D. Spacey. "Ataxia and the Role of Antigliadin Antibodies." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 34, no. 2 (2007): 193–96. http://dx.doi.org/10.1017/s031716710000603x.

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Background:Although it is acknowledged that patients with celiac disease can develop neurological complications such as ataxia, the association of antigliadin antibodies in the etiology of sporadic ataxia and the usefulness of this testing in diagnosis of ataxia is controversial.Methods:We investigated this association by testing for the presence of IgG and IgA antigliadin antibodies in 56 ataxic patients and 59 controls. The ataxia patients were subsequently classified into three groups: sporadic, hereditary and MSA.Results:Of the total ataxic patients, 6/56 (11%) were positive for either IgG

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2

TÜRKOĞLU, Recai, Nuriye ÇÖMEZ, Mehmet GENCER, Yılmaz ÇETİNKAYA, Züleyha AKKAN ÇETİNKAYA, and Hülya TİRELİ. "Antigliadin Antibodies and Multiple Sclerosis." Turkiye Klinikleri Journal of Medical Sciences 30, no. 2 (2010): 577–83. http://dx.doi.org/10.5336/medsci.2008-9050.

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Sultan, Sheikh Javeed, Qazi Masood Ahmad, and Sheikh Tariq Sultan. "Antigliadin antibodies in psoriasis." Australasian Journal of Dermatology 51, no. 4 (2010): 238–42. http://dx.doi.org/10.1111/j.1440-0960.2010.00648.x.

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Zachor, Ditza A., Elizabeth Mroczek‐Musulman, and Pamela Brown. "Prevalence of Celiac Disease in Down Syndrome in the United States." Journal of Pediatric Gastroenterology and Nutrition 31, no. 3 (2000): 275–79. http://dx.doi.org/10.1002/j.1536-4801.2000.tb07116.x.

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ABSTRACTBackgroundNumerous studies in Europe have documented a high prevalence of celiac disease in Down syndrome. This study was undertaken to estimate the prevalence of celiac disease in Down syndrome in the southeastern United States.MethodsSeventy‐five patients with Down syndrome were screened using immunoglobulin (Ig)A‐anti antiendomysium antibodies, IgA‐antigliadin antibodies, and total IgA level. When either antiendomysium or antigliadin antibodies produced positive findings, patients were referred to a pediatric gastroenterologist for consideration of a duodenal biopsy.ResultsThirteen

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5

Bushara, K. O., M. Nance, and C. M. Gomez. "Antigliadin antibodies in Huntington's disease." Neurology 62, no. 1 (2004): 132–33. http://dx.doi.org/10.1212/wnl.62.1.132.

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Barta, Z., G. Mekkel, M. Zeher, and K. O. Bushara. "Antigliadin antibodies in Huntington's disease." Neurology 63, no. 4 (2004): 762. http://dx.doi.org/10.1212/wnl.63.4.762-a.

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7

Kalayciyan, A., and A. Kotogyan. "Psoriasis, enteropathy and antigliadin antibodies." British Journal of Dermatology 154, no. 4 (2006): 778–79. http://dx.doi.org/10.1111/j.1365-2133.2006.07132.x.

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8

Schlesinger, I., and R. Hering. "Antigliadin Antibodies in Migraine Patients." Cephalalgia 17, no. 6 (1997): 712. http://dx.doi.org/10.1177/033310249701700616.

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El-Alameey, Inas R., Hanaa H. Ahmed, Sawsan M. Tawfik, Fawzia Hassaballa, Ayman M. Abdel Gawad, and Eman Eltahlawy. "Gluten Sensitivity among Egyptian Infants with Congenital Heart Disease." Open Access Macedonian Journal of Medical Sciences 5, no. 1 (2017): 58–63. http://dx.doi.org/10.3889/oamjms.2017.026.

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BACKGROUND: Gastrointestinal symptoms are a common feature in infants with congenital heart disease.AIM: This study was designed to evaluate age-dependent serum levels of antigliadin antibodies among malnourished Egyptian infants with congenital heart disease (CHD) and gastrointestinal symptoms.SUBJECTS AND METHODS: This case-control study conducted on 60 infants with established congenital heart disease. They were subdivided into cyanotic and acyanotic groups, and each group includes 30 patients compared with thirty apparently healthy infants of matched age, sex, and social class. Serum antig

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10

Juto, Per, Bo Fredrikzon, and Olle Hernell. "Gliadin‐Specific Serum Immunoglobulins A, E, G, and M in Childhood." Journal of Pediatric Gastroenterology and Nutrition 4, no. 5 (1985): 723–29. http://dx.doi.org/10.1002/j.1536-4801.1985.tb08945.x.

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Summary:An enzyme‐linked immunosorbent assay technique was developed to determine serum antigliadin antibodies of the IgA, IgE, IgG, and IgM classes. The antibody level of each serum specimen was expressed as an index value, i.e., optical density of test serum/optical density of cutoff, where cutoff was calculated for each immunoglobulin class as the mean + 3 SD for six healthy controls. Indices for each immunoglobulin class were determined in 69 children who were admitted for their first small intestinal mucosal biopsy due to either symptoms of malabsorption compatible with celiac disease, or

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11

Bushara, Khalaf, and Mark Hallett. "Prevalence of antigliadin antibodies in ataxia patients." Neurology 62, no. 7 (2004): 1237.2–1238. http://dx.doi.org/10.1212/wnl.62.7.1237-a.

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12

Abele, M., L. Schols, S. Schwartz, and T. Klockgether. "Prevalence of antigliadin antibodies in ataxia patients." Neurology 60, no. 10 (2003): 1674–75. http://dx.doi.org/10.1212/01.wnl.0000069606.82919.04.

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Unsworth, D. J., J. Dias, and J. A. Walker-Smith. "ANTIGLIADIN AND ANTIRETICULIN ANTIBODIES IN COELIAC DISEASE." Lancet 331, no. 8587 (1988): 705. http://dx.doi.org/10.1016/s0140-6736(88)91503-6.

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Garcia, Yolanda Hidalgo, Santiago Gómez Díez, Lourdes Tricas Aizpún, and Narciso Pérez Oliva. "Antigliadin Antibodies Associated with Chronic Mucocutaneous Candidiasis." Pediatric Dermatology 19, no. 5 (2002): 415–18. http://dx.doi.org/10.1046/j.1525-1470.2002.00117.x.

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15

Korponay‐Szabó, Ilma R., Judit B. Kovács, Antal Czinner, Gyula Gorácz, Adrienn Vámos, and Teréz Szabó. "High Prevalence of Silent Celiac Disease in Preschool Children Screened with IgA/IgG Antiendomysium Antibodies." Journal of Pediatric Gastroenterology and Nutrition 28, no. 1 (1999): 26–30. http://dx.doi.org/10.1002/j.1536-4801.1999.tb01999.x.

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ABSTRACTBackground:Because of the different sensitivity and specificity of serologic tests, the search for silent celiac disease is usually performed with the combined or sequential use of several tests. Among these, the IgA‐class endomysium antibody test has the highest specificity and positive predictive value, but it may overlook IgA‐deficient patients.Methods:To test a new one‐step screening approach, serum samples from 427 apparently healthy, 3‐ to 6‐year‐old Hungarian children were investigated for IgA‐class and IgG‐class endomysium antibodies using monkey esophagus and human jejunum as

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16

Koehne, Victor de Barros, Magda Bahia, Cristina Costa Duarte Lanna, Maria Raquel da Costa Pinto, Eduardo Alves Bambirra, and Aloísio Sales da Cunha. "Prevalence of serological markers for celiac disease (IgA and IgG class antigliadin antibodies and IgA class antiendomysium antibodies) in patients with autoimmune rheumatologic diseases in Belo Horizonte, MG, Brazil." Arquivos de Gastroenterologia 47, no. 3 (2010): 250–56. http://dx.doi.org/10.1590/s0004-28032010000300008.

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CONTEXT: Patients with autoimmune rheumatologic conditions and celiac disease tend to have a variety of autoantibodies, many of which have no clear pathogenic role. The literature contains frequent reports of celiac disease being more prevalent in patients with rheumatologic diseases, although this remains controversial. OBJECTIVES: To investigate the prevalence of positive serum tests for celiac disease, particularly IgA and IgG antigliadin (AGA) antibodies and IgA antiendomysium antibodies (EmA) in patients with autoimmune rheumatologic diseases. A second aim was to correlate positive serum

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17

Hadjivassiliou, Marios, Richard A. Grünewald, David S. Sanders, Priya Shanmugarajah, and Nigel Hoggard. "Effect of gluten-free diet on cerebellar MR spectroscopy in gluten ataxia." Neurology 89, no. 7 (2017): 705–9. http://dx.doi.org/10.1212/wnl.0000000000004237.

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Objective:To evaluate the effect of gluten free diet (GFD) on magnetic resonance spectroscopy (MRS) of the cerebellum in patients with gluten ataxia (GA).Methods:Patients with GA, defined as sporadic ataxia with positive antigliadin antibodies in the absence of an alternative cause, routinely undergo MRS at baseline and after the introduction of GFD as part of their clinical care. We present our experience of the effect of GFD on MRS of the cerebellum.Results:A total of 117 consecutive patients with GA were included in this report. Sixty-three were on strict GFD with elimination of antigliadin

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18

Dumitrascu, Dinu Iuliu, and Aurel Popa Wagner. "Ageing and comorbidities in humans with antigliadin antibodies." Journal of Gastrointestinal and Liver Diseases 31, no. 3 (2022): 368–58. http://dx.doi.org/10.15403/jgld-4465.

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Chinnery, P. F., P. J. Reading, D. Milne, D. Gardner-Medwin, and D. M. Turnbull. "CSF antigliadin antibodies and the Ramsay Hunt syndrome." Neurology 49, no. 4 (1997): 1131–33. http://dx.doi.org/10.1212/wnl.49.4.1131.

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Catassi, C., A. Guerrieri, E. Bartolotta, G. V. Coppa, and P. L. Giorgi. "ANTIGLIADIN ANTIBODIES AT ONSET OF DIABETES IN CHILDREN." Lancet 330, no. 8551 (1987): 158. http://dx.doi.org/10.1016/s0140-6736(87)92357-9.

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Cacciari, E., S. Salardi, U. Volta, and G. R. Corazza. "ANTIGLIADIN ANTIBODIES IN COELIAC CHILDREN WITH SHORT STATURE." Lancet 326, no. 8469-8470 (1985): 1434. http://dx.doi.org/10.1016/s0140-6736(85)92608-x.

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Lahteenoja, Toivanen, Raiha, Syrjanen, and Viander. "Salivary Antigliadin and Antiendomysium Antibodies in Coeliac Disease." Scandinavian Journal of Immunology 50, no. 5 (1999): 528–35. http://dx.doi.org/10.1046/j.1365-3083.1999.00606.x.

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Calero, P., C. Ribes‐Koninckx, V. Albiach, C. Carles, and J. Ferrer. "IgA Antigliadin Antibodies as a Screening Method for Nonovert Celiac Disease in Children with Insulin‐Dependent Diabetes Mellitus." Journal of Pediatric Gastroenterology and Nutrition 23, no. 1 (1996): 29–33. http://dx.doi.org/10.1002/j.1536-4801.1996.tb00298.x.

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SummaryOne hundred forty‐one children with insulindependent diabetes mellitus were screened for serum immunoglobulin A (IgA) antigliadin antibodies by means of an enzyme‐linked immunosorbent assay (ELISA) method. None of them had gastrointestinal symptoms, and no major nutritional disturbances were detected except for a girl with moderate growth delay. Twelve patients with positive IgA antigliadin antibodies on two or more consecutive measurements underwent a small intestinal biopsy; four of them had a subtotal villous atrophy, and celiac disease was diagnosed; in another patient, a partial vi

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24

Vainio, E., K. Kalimo, M. Viander, and T. Reunala. "Antigliadin antibodies and gluten-free diet in dermatitis herpetiformis." Acta Dermato-Venereologica 65, no. 4 (1985): 291–97. http://dx.doi.org/10.2340/0001555565291297.

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IgA and IgG class antigliadin antibodies (AGA) were analysed with ELISA technique from serum samples of 30 dermatitis herpetiformis patients. Jejunal biopsies were performed to all patients before any treatment and high levels of IgA class AGA were found to be associated with subtotal villous atrophy. Fourteen patients started gluten-free diet (GFD) which caused a significant decrease in both IgA and IgG class AGA. The decrease of IgA AGA was faster than that of IgG AGA and IgA antibody levels fell to normal range during the GFD treatment in all but one patient. In contrast, 5 out of 8 patient

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25

Cavataio, F., G. Iacono, A. Carroccio, and G. Montalto. "Diagnostic Accuracy of a New Stick Micromethod with Which to Measure Antigliadin Antibodies." Journal of Pediatric Gastroenterology and Nutrition 19, no. 4 (1994): 401–2. http://dx.doi.org/10.1002/j.1536-4801.1994.tb11459.x.

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SummaryA new, simplified micromethod for semiquantitative determination of the antigliadin antibody (AGA) has been proposed. When the new stick method was used in a population of infants hospitalized for gastrointestinal problems, the results were virtually identical to those of the traditional method

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26

Vainio, Eeva, Istvan Kosnai, Olavi Hällström, Sarolta Karpati, Markku Mäki, and Timo Reunala. "Antigliadin and Antireticulin Antibodies in Children with Dermatitis Herpetiformis." Journal of Pediatric Gastroenterology and Nutrition 5, no. 5 (1986): 735–39. http://dx.doi.org/10.1097/00005176-198609000-00012.

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Fornasieri, A., R. A. Sinico, P. Maldifassi, P. Bernasconi, M. Vegni, and G. D'Amico. "IgA-antigliadin antibodies in IgA mesangial nephropathy (Berger's disease)." BMJ 295, no. 6590 (1987): 78–80. http://dx.doi.org/10.1136/bmj.295.6590.78.

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Weisenseel, P., A. V. Kuznetsov, T. Ruzicka, and J. C. Prinz. "Palmoplantar pustulosis is not inevitably associated with antigliadin antibodies." British Journal of Dermatology 156, no. 6 (2007): 1399–400. http://dx.doi.org/10.1111/j.1365-2133.2007.07908.x.

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Cipolli, M., EA Valletta, C. Zampieri, and G. Mastella. "Increased serum levels of antigliadin antibodies in cystic fibrosis." Acta Paediatrica 82, no. 1 (1993): 95–97. http://dx.doi.org/10.1111/j.1651-2227.1993.tb12526.x.

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Weile, B., E. Grodzinsky, T. Skogh, R. Jordal, B. Cavell, and PA Krasilnikoff. "Screening Danish blood donors for antigliadin and antiendomysium antibodies." Acta Paediatrica 85, s412 (1996): 46. http://dx.doi.org/10.1111/j.1651-2227.1996.tb14248.x.

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Dias, J., D. J. Unsworth, and J. A. Walker-Smith. "ANTIGLIADIN AND ANTIRETICULIN ANTIBODIES IN SCREENING FOR COELIAC DISEASE." Lancet 330, no. 8551 (1987): 157–58. http://dx.doi.org/10.1016/s0140-6736(87)92356-7.

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Rodriguez-Soriano, J., A. Arrieta, A. Vallo, M. J. Sebastian, J. C. Vitoria, and M. D. Masdevall. "IgA ANTIGLIADIN ANTIBODIES IN CHILDREN WITH IgA MESANGIAL GLOMERULONEPHRITIS." Lancet 331, no. 8594 (1988): 1109–10. http://dx.doi.org/10.1016/s0140-6736(88)91928-9.

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Rostoker, G., M. T. Chaumette, E. Wirquin, et al. "IgA mesangial nephritis, IgA antigliadin antibodies, and coeliac disease." Lancet 336, no. 8718 (1990): 824–25. http://dx.doi.org/10.1016/0140-6736(90)93300-e.

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Patinen, Pertti, Fred Björksten, Maria Malmström, Erkki Savilahrtl, and Timo Reunala. "Salivary and serum IgA antigliadin antibodies in dermatitis herpetiformis." European Journal of Oral Sciences 103, no. 5 (1995): 280–84. http://dx.doi.org/10.1111/j.1600-0722.1995.tb00027.x.

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Vainio, Eeva, Istvan Kosnai, Olavi Hällström, Sarolta Karpati, Markku Mäki, and Timo Reunala. "Antigliadin and Antireticulin Antibodies in Children with Dermatitis Herpetiformis." Journal of Pediatric Gastroenterology and Nutrition 5, no. 5 (1986): 735–39. http://dx.doi.org/10.1002/j.1536-4801.1986.tb09171.x.

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SummaryThe serum samples of 27 children with dermatitis herpetiformis (DH) were examined for the presence of antigliadin (AGA) and antireticulin (ARA) antibodies. AGA were determined with an enzyme‐linked immuno‐sorbent assay (ELISA) and ARA with an immunofluorescence method. Increased IgA or IgG class AGA levels were found in four of ten children on a normal diet, in two of 25 on a gluten‐free diet (GFD), and in two of four children on gluten challenge. The corresponding figures for ARA were nine of ten, two of 25, and four of four, respectively. All nine patients with ARA on a normal diet ha

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Kolho, Kaija‐Leena, and Erkki Savilahti. "IgA Endomysium Antibodies on Human Umbilical Cord: An Excellent Diagnostic Tool for Celiac Disease in Childhood." Journal of Pediatric Gastroenterology and Nutrition 24, no. 5 (1997): 563–67. http://dx.doi.org/10.1002/j.1536-4801.1997.tb00652.x.

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Background:An improvement in screening for celiac disease has recently been described that uses human umbilical cord as a substitute for monkey esophagus to determine IgA endomysium antibodies in adults. As using monkey esophagus is ethically questionable for large‐scale screening, we studied whether substitution of umbilical cord would be suitable for pediatric patients as well.Methods:Serum from 53 children with untreated celiac disease, 22 in remission and 13 on challenge, were screened for antigliadin IgA, antigliadin IgG, and IgA reticulin antibodies, in addition to IgA endomysium antibod

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Sacchetti, L., A. Ferrajolo, G. Salerno, et al. "Diagnostic value of various serum antibodies detected by diverse methods in childhood celiac disease." Clinical Chemistry 42, no. 11 (1996): 1838–42. http://dx.doi.org/10.1093/clinchem/42.11.1838.

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Abstract The diagnostic performances of antiendomysium IgA detected on monkey esophagus and human umbilical cord smooth muscle, of antireticulin IgA, and of antigliadin IgA and IgG were calculated in 74 children with celiac disease (CD) or other gastrointestinal disorders. We also compared four methods for gliadin antibody detection. With a diagnostic specificity of 100%, diagnostic sensitivity was 94% for antireticulin IgA, 93% for antiendomysium IgA when detected on human umbilical cord smooth muscle, and 97% when detected on monkey esophagus. The diagnostic sensitivity for gliadin antibody

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Yuce, Aysel, Hulya Demir, Nurten Kocak, Figen Gurakan, and Hasan Ozen. "Antiendomysium and antigliadin antibodies for the diagnosis of celiac disease." American Journal of Gastroenterology 95, no. 5 (2000): 1366–67. http://dx.doi.org/10.1111/j.1572-0241.2000.02042.x.

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Kumar, V., N. Jain, A. Lerner, E. H. Beutner, T. P. Chorzelski, and E. Lebenthal. "Comparative Studies of Different Gliadin Preparations in Detecting Antigliadin Antibodies." Journal of Pediatric Gastroenterology and Nutrition 5, no. 5 (1986): 730–34. http://dx.doi.org/10.1097/00005176-198609000-00011.

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Yüce, A. "Antiendomysium and antigliadin antibodies for the diagnosis of celiac disease." American Journal of Gastroenterology 95, no. 5 (2000): 1366–67. http://dx.doi.org/10.1016/s0002-9270(00)00834-0.

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Rosales-Rivera, L. C., J. L. Acero-Sánchez, P. Lozano-Sánchez, I. Katakis, and C. K. O'Sullivan. "Electrochemical immunosensor detection of antigliadin antibodies from real human serum." Biosensors and Bioelectronics 26, no. 11 (2011): 4471–76. http://dx.doi.org/10.1016/j.bios.2011.05.004.

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Marinello, Daniela, Anna Rapa, Raffaella Osello, et al. "Celiac Disease Screening: Exploring the Iceberg With Salivary Antigliadin Antibodies." Journal of Pediatric Gastroenterology and Nutrition 32, no. 2 (2001): 227–28. http://dx.doi.org/10.1097/00005176-200102000-00032.

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Almaguer-Mederos, L. E., Y. R. Almira, E. M. Gongora, et al. "Antigliadin antibodies in Cuban patients with spinocerebellar ataxia type 2." Journal of Neurology, Neurosurgery & Psychiatry 79, no. 3 (2008): 315–17. http://dx.doi.org/10.1136/jnnp.2006.113985.

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Marinello, Daniela, Anna Rapa, Raffaella Osello, et al. "Celiac Disease Screening: Exploring the Iceberg With Salivary Antigliadin Antibodies." Journal of Pediatric Gastroenterology and Nutrition 32, no. 2 (2001): 227–28. http://dx.doi.org/10.1002/j.1536-4801.2001.tb07255.x.

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Kumar, V., N. Jain, A. Lerner, E. H. Beutner, T. P. Chorzelski, and E. Lebenthal. "Comparative Studies of Different Gliadin Preparations in Detecting Antigliadin Antibodies." Journal of Pediatric Gastroenterology and Nutrition 5, no. 5 (1986): 730–34. http://dx.doi.org/10.1002/j.1536-4801.1986.tb09170.x.

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SummaryAntigliadin antibodies (AGA) have been used as indicators of celiac disease. The presence of these antibodies in other gastrointestinal and liver disorders and even in normal healthy controls casts a shadow on the diagnostic significance of AGA. We examined 91 normal controls of varying ages and 97 patients with various gastrointestinal and liver disorders. Forty‐eight of 97 nonceliac patients were positive for AGA, and diagnosis‐specific incidences ranged as high as 75% in patients with small bowel disease. In addition, the levels of AGA were dependent upon age as their presence increa

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46

Elfar, NashwaN, and AmalS Elbandari. "Serum level of celiac disease-associated antigliadin antibodies in psoriatic patients." Egyptian Journal of Dermatology and Venerology 34, no. 2 (2014): 130. http://dx.doi.org/10.4103/1110-6530.150274.

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Economidou, J., G. Avgerinou, A. Tsiroyianni, P. Stavropoulos, A. Vareltzidis, and A. Katsambas. "Endomysium and antigliadin antibodies in dermatitis herpetiformis and other bullous diseases." Journal of the European Academy of Dermatology and Venereology 11, no. 2 (1998): 184–85. http://dx.doi.org/10.1111/j.1468-3083.1998.tb00778.x.

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