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Artykuły w czasopismach na temat „Arthritis Molecular aspects”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 31 stycznia 2023

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1

Gross, D. M., and B. T. Huber. "Cellular and molecular aspects of Lyme arthritis." Cellular and Molecular Life Sciences 57, no. 11 (October 2000): 1562–69. http://dx.doi.org/10.1007/pl00000641.

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Chetina, E. V., and E. P. Sharapova. "Rheumatic pain management: molecular aspects." Modern Rheumatology Journal 14, no. 1 (March 22, 2020): 93–100. http://dx.doi.org/10.14412/1996-7012-2020-1-93-100.

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Rheumatic diseases (RDs), including osteoarthritis and rheumatoid arthritis, are non-infectious slowly progressive incurable inflammatory diseases that lead to prolonged disability due to damage to the musculoskeletal system. Pain is a dominant symptom at any stage of these diseases, is directly related to joint functioning, and determines the quality of life in patients. Moreover, despite the significant successes of studying the role of inflammation and regulation of autoimmune processes, the pathogenetic mechanisms for the development and maintenance of pain in RDs are little investigated.

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Neeck, Gunther, Rainer Renkawitz, and Martin Eggert. "Molecular aspects of glucocorticoid hormone action in rheumatoid arthritis." Cytokines, Cellular & Molecular Therapy 7, no. 2 (January 2002): 61–69. http://dx.doi.org/10.1080/13684730412331302081.

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Kobayashi, Shu, Shigeki Momohara, Naoyuki Kamatani, and Hiroshi Okamoto. "Molecular aspects of rheumatoid arthritis: role of environmental factors." FEBS Journal 275, no. 18 (July 25, 2008): 4456–62. http://dx.doi.org/10.1111/j.1742-4658.2008.06581.x.

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Okamoto, Hiroshi, Thomas P. Cujec, Hisashi Yamanaka, and Naoyuki Kamatani. "Molecular aspects of rheumatoid arthritis: role of transcription factors." FEBS Journal 275, no. 18 (July 25, 2008): 4463–70. http://dx.doi.org/10.1111/j.1742-4658.2008.06582.x.

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García-Hernández, Mariana H., Roberto González-Amaro, and Diana Patricia Portales-Pérez. "Specific therapy to regulate inflammation in rheumatoid arthritis: molecular aspects." Immunotherapy 6, no. 5 (May 2014): 623–36. http://dx.doi.org/10.2217/imt.14.26.

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Lotz, M., and J. Roudier. "Epstein-Barr virus and rheumatoid arthritis: cellular and molecular aspects." Rheumatology International 9, no. 3-5 (November 1989): 147–52. http://dx.doi.org/10.1007/bf00271872.

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Sacre, Sandra M., Evangelos Andreakos, Peter Taylor, Marc Feldmann, and Brian M. Foxwell. "Molecular therapeutic targets in rheumatoid arthritis." Expert Reviews in Molecular Medicine 7, no. 16 (August 24, 2005): 1–20. http://dx.doi.org/10.1017/s1462399405009488.

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In an attempt to combat the pain and damage generated by rheumatoid arthritis (RA), new drugs are being developed to target molecular aspects of the disease process. Recently, a major development has been the use of biologicals (antibodies and soluble receptors) that neutralise the activity of tumour necrosis factor α (TNF-α) and interleukin 1 (IL-1), both of which are involved in disease progression. An increase in our understanding of cell and molecular biology has resulted in the identification and investigation of potential new targets, and also the refinement and improvement of current th

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9

RULLI, NESTOR E., JULIAN MELTON, ANJA WILMES, GARY EWART, and SURESH MAHALINGAM. "The Molecular and Cellular Aspects of Arthritis Due to Alphavirus Infections." Annals of the New York Academy of Sciences 1102, no. 1 (April 2007): 96–108. http://dx.doi.org/10.1196/annals.1408.007.

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Okamoto, Hiroshi. "Molecular aspects of rheumatoid arthritis: chemokines, environmental factors and transcription factors." FEBS Journal 275, no. 18 (July 25, 2008): 4447. http://dx.doi.org/10.1111/j.1742-4658.2008.06579.x.

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Iwamoto, Takuji, Hiroshi Okamoto, Yoshiaki Toyama, and Shigeki Momohara. "Molecular aspects of rheumatoid arthritis: chemokines in the joints of patients." FEBS Journal 275, no. 18 (July 25, 2008): 4448–55. http://dx.doi.org/10.1111/j.1742-4658.2008.06580.x.

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Dulyapin, V. A. "Morphological aspects of chondroclasis in rheumatoid arthritis." Bulletin of Experimental Biology and Medicine 112, no. 1 (July 1991): 1040–44. http://dx.doi.org/10.1007/bf00841168.

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Stoustrup, P., K. D. Kristensen, A. Küseler, T. K. Pedersen, and T. Herlin. "AB1210 Aspects of temporomandibular joint arthritis-related orofacial symptoms in juvenile idiopathic arthritis." Annals of the Rheumatic Diseases 71, Suppl 3 (June 2013): 707.2–707. http://dx.doi.org/10.1136/annrheumdis-2012-eular.1208.

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Ishikawa, Larissa Lumi Watanabe, Priscila Maria Colavite, Larissa Camargo da Rosa, Bianca Balbino, Thais Graziela Donegá França, Sofia Fernanda Gonçalves Zorzella-Pezavento, Fernanda Chiuso-Minicucci, and Alexandrina Sartori. "Commercial Bovine Proteoglycan Is Highly Arthritogenic and Can Be Used as an Alternative Antigen Source for PGIA Model." BioMed Research International 2014 (2014): 1–12. http://dx.doi.org/10.1155/2014/148594.

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Rheumatoid arthritis (RA) is the most common systemic autoimmune disease. It affects mainly the joints, causing synovitis, cartilage destruction, and bone erosion. Many experimental models are used to study the mechanisms involved in immunopathogenesis and new therapies for this disease. Proteoglycan-induced arthritis (PGIA) is a widely used model based on the cross-reactivity of injected foreign (usually human) PG and mice self-PG. Considering the complexity of the extraction and purification of human PG, in this study we evaluated the arthritogenicity of bovine PG that is commercially availa

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15

Visser, H. "Sarcoid arthritis: clinical characteristics, diagnostic aspects, and risk factors." Annals of the Rheumatic Diseases 61, no. 6 (June 1, 2002): 499–504. http://dx.doi.org/10.1136/ard.61.6.499.

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Askari, Gholamreza, Abdolreza Norouzy, Vahid Hadi, Naseh Pahlavani, Mahsa Malekahmadi, Elyas Nattagh-Eshtivani, JamshidGholizadeh Navashenaq, Saeid Hadi, GordonA Ferns, and Majid Ghayour-Mobarhan. "Nigella sativa in controlling Type 2 diabetes, cardiovascular, and rheumatoid arthritis diseases: Molecular aspects." Journal of Research in Medical Sciences 26, no. 1 (2021): 20. http://dx.doi.org/10.4103/jrms.jrms_236_20.

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Márquez, Ana, Javier Martín, and F. David Carmona. "Emerging aspects of molecular biomarkers for diagnosis, prognosis and treatment response in rheumatoid arthritis." Expert Review of Molecular Diagnostics 16, no. 6 (April 18, 2016): 663–75. http://dx.doi.org/10.1080/14737159.2016.1174579.

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Macaubas, Claudia, Khoa Nguyen, Kuang-Hung Pan, Tzielan Lee, Chetan Deshpande, Christy Sandborg, Stanley Cohen, and Elizabeth Mellins. "Distinct Molecular and Cellular Aspects of Systemic Juvenile Idiopathic Arthritis (SJIA) and Polyarticular (PolyJIA)." Clinical Immunology 123 (2007): S94—S95. http://dx.doi.org/10.1016/j.clim.2007.03.450.

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Hoffmann, Henrik, and Cordelia Schiene-Fischer. "Functional aspects of extracellular cyclophilins." Biological Chemistry 395, no. 7-8 (July 1, 2014): 721–35. http://dx.doi.org/10.1515/hsz-2014-0125.

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Abstract The cyclophilin family of peptidyl prolyl cis/trans isomerases includes several isoforms found to be secreted in response to different stimuli, thus existing both in the interior and the exterior of cells. The extracellular fractions of the cyclophilins CypA and CypB are involved in the control of cell-cell communication. By binding to the cell membrane receptor CD147 and cell surface heparans they elicit a variety of intracellular signaling cascades involved in inflammatory processes. Increased levels of cyclophilins in inflammatory tissues and body fluids are considered as an inflam

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Laragione, Teresina, Max Brenner, Amit Lahiri, Erjing Gao, Carolyn Harris, and Percio S. Gulko. "Huntingtin-interacting protein 1 (HIP1) regulates arthritis severity and synovial fibroblast invasiveness by altering PDGFR and Rac1 signalling." Annals of the Rheumatic Diseases 77, no. 11 (July 26, 2018): 1627–35. http://dx.doi.org/10.1136/annrheumdis-2018-213498.

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ObjectivesWhile new treatments for rheumatoid arthritis (RA) have markedly improved disease control by targeting immune/inflammatory pathways, current treatments rarely induce remission, underscoring the need for therapies that target other aspects of the disease. Little is known about the regulation of disease severity and joint damage, which are major predictors of disease outcome, and might be better or complementary targets for therapy. In this study, we aimed to discover and characterise a new arthritis severity gene.MethodsAn unbiased and phenotype-driven strategy including studies of un

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21

Galozzi, Paola, Sara Bindoli, Andrea Doria, Francesca Oliviero, and Paolo Sfriso. "Autoinflammatory Features in Gouty Arthritis." Journal of Clinical Medicine 10, no. 9 (April 26, 2021): 1880. http://dx.doi.org/10.3390/jcm10091880.

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In the panorama of inflammatory arthritis, gout is the most common and studied disease. It is known that hyperuricemia and monosodium urate (MSU) crystal-induced inflammation provoke crystal deposits in joints. However, since hyperuricemia alone is not sufficient to develop gout, molecular-genetic contributions are necessary to better clinically frame the disease. Herein, we review the autoinflammatory features of gout, from clinical challenges and differential diagnosis, to the autoinflammatory mechanisms, providing also emerging therapeutic options available for targeting the main inflammato

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Boers, M., A. M. Croonen, B. A. Dijkmans, F. C. Breedveld, F. Eulderink, A. Cats, and J. J. Weening. "Renal findings in rheumatoid arthritis: clinical aspects of 132 necropsies." Annals of the Rheumatic Diseases 46, no. 9 (September 1, 1987): 658–63. http://dx.doi.org/10.1136/ard.46.9.658.

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van de Laar, M. A., M. Aalbers, F. G. Bruins, A. C. van Dinther-Janssen, J. K. van der Korst, and C. J. Meijer. "Food intolerance in rheumatoid arthritis. II. Clinical and histological aspects." Annals of the Rheumatic Diseases 51, no. 3 (March 1, 1992): 303–6. http://dx.doi.org/10.1136/ard.51.3.303.

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Cavagna, Lorenzo, Sara Monti, Vittorio Grosso, Nicola Boffini, Eva Scorletti, Gloria Crepaldi, and Roberto Caporali. "The Multifaceted Aspects of Interstitial Lung Disease in Rheumatoid Arthritis." BioMed Research International 2013 (2013): 1–13. http://dx.doi.org/10.1155/2013/759760.

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Interstitial lung disease (ILD) is a relevant extra-articular manifestation of rheumatoid arthritis (RA) that may occur either in early stages or as a complication of long-standing disease. RA related ILD (RA-ILD) significantly influences thequoad vitamprognosis of these patients. Several histopathological patterns of RA-ILD have been described: usual interstitial pneumonia (UIP) is the most frequent one, followed by nonspecific interstitial pneumonia (NSIP); other patterns are less commonly observed. Several factors have been associated with an increased risk of developing RA-ILD. The genetic

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Brand, Linda, De Wet Wolmarans, and Sarel J. Brand. "A Quick and Painless Reminder: The Pharmacotherapy of Rheumatoid Arthritis in Primary Practice." South African Family Practice 60, no. 2 (June 7, 2018): 38–42. http://dx.doi.org/10.4102/safp.v60i2.4839.

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Rheumatoid arthritis, an auto-immune disorder, is characterized by chronic inflammation of the joints, synovial hyperplasia and bone erosion. These pathological features are promoted by a synovial microenvironment featuring B-cell and T-cell infiltrate, synovial fibroblasts and an intricate network of pro-inflammatory cellular messengers – prominent molecular role-players that represent critical targets in the pharmacotherapy of the disease. This review offers a brief overview of the etiopathology of rheumatoid arthritis while focussing on the practical aspects of methotrexate and glucocortico

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Sha, Naijun, Marina Vannucci, Philip J. Brown, Michael K. Trower, Gillian Amphlett, and Francesco Falciani. "Gene Selection in Arthritis Classification with Large-Scale Microarray Expression Profiles." Comparative and Functional Genomics 4, no. 2 (2003): 171–81. http://dx.doi.org/10.1002/cfg.264.

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The use of large-scale microarray expression profiling to identify predictors of disease class has become of major interest. Beyond their impact in the clinical setting (i.e. improving diagnosis and treatment), these markers are also likely to provide clues on the molecular mechanisms underlining the diseases. In this paper we describe a new method for the identification of multiple gene predictors of disease class. The method is applied to the classification of two forms of arthritis that have a similar clinical endpoint but different underlying molecular mechanisms: rheumatoid arthritis (RA)

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Loréal, Olivier, Thibault Cavey, François Robin, Moussa Kenawi, Pascal Guggenbuhl, and Pierre Brissot. "Iron as a Therapeutic Target in HFE-Related Hemochromatosis: Usual and Novel Aspects." Pharmaceuticals 11, no. 4 (November 26, 2018): 131. http://dx.doi.org/10.3390/ph11040131.

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Genetic hemochromatosis is an iron overload disease that is mainly related to the C282Y mutation in the HFE gene. This gene controls the expression of hepcidin, a peptide secreted in plasma by the liver and regulates systemic iron distribution. Homozygous C282Y mutation induces hepcidin deficiency, leading to increased circulating transferrin saturation, and ultimately, iron accumulation in organs such as the liver, pancreas, heart, and bone. Iron in excess may induce or favor the development of complications such as cirrhosis, liver cancer, diabetes, heart failure, hypogonadism, but also comp

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Hashimoto, Teppei, Kohsuke Yoshida, Akira Hashiramoto, and Kiyoshi Matsui. "Cell-Free DNA in Rheumatoid Arthritis." International Journal of Molecular Sciences 22, no. 16 (August 19, 2021): 8941. http://dx.doi.org/10.3390/ijms22168941.

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Endogenous DNA derived from the nuclei or mitochondria is released into the bloodstream following cell damage or death. Extracellular DNA, called cell-free DNA (cfDNA), is associated with various pathological conditions. Recently, multiple aspects of cfDNA have been assessed, including cfDNA levels, integrity, methylation, and mutations. Rheumatoid arthritis (RA) is the most common form of autoimmune arthritis, and treatment of RA has highly varied outcomes. cfDNA in patients with RA is elevated in peripheral blood and synovial fluid and is associated with disease activity. Profiling of cfDNA

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Poddighe, Dimitri, Micol Romano, Maurizio Gattinara, and Valeria Gerloni. "Biologics for the Treatment of Juvenile Idiopathic Arthritis." Current Medicinal Chemistry 25, no. 42 (February 6, 2019): 5860–93. http://dx.doi.org/10.2174/0929867325666180522085716.

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Juvenile Idiopathic Arthritis (JIA) is one of the most common chronic diseases in children. Recently, the management of JIA has substantially changed, thanks to the availability of new treatment options, represented by biological drugs or biologics. These drugs modulate the specific mechanisms of the immune systems, such as TNF-α, IL-1 and IL-6 signaling, or lymphocyte activation and/or functioning. In this review, we provide a comprehensive discussion on the current recommendations and clinical evidence regarding the use of the available biologics in the treatment of JIA; moreover, the main p

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Berardi, S., A. Corrado, N. Maruotti, D. Cici, and F. P. Cantatore. "Osteoblast role in the pathogenesis of rheumatoid arthritis." Molecular Biology Reports 48, no. 3 (March 2021): 2843–52. http://dx.doi.org/10.1007/s11033-021-06288-y.

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AbstractIn the pathogenesis of several rheumatic diseases, such as rheumatoid arthritis, spondyloarthritis, osteoarthritis, osteoporosis, alterations in osteoblast growth, differentiation and activity play a role. In particular, in rheumatoid arthritis bone homeostasis is perturbed: in addition to stimulating the pathologic bone resorption process performed by osteoclasts in course of rheumatoid arthritis, proinflammatory cytokines (such as Tumor Necrosis factor-α, Interleukin-1) can also inhibit osteoblast differentiation and function, resulting in net bone loss. Mouse models of rheumatoid ar

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Caporali, Roberto, and Josef S. Smolen. "Back to the future: forget ultrasound and focus on clinical assessment in rheumatoid arthritis management." Annals of the Rheumatic Diseases 77, no. 1 (August 2, 2017): 18–20. http://dx.doi.org/10.1136/annrheumdis-2017-211458.

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Ultrasound (US) unquestionably improves many aspects of rheumatoid arthritis (RA) diagnosis and management, but no consensus has been reached regarding the optimal US methodology that should be used, and high levels of standardisation have not yet been attained. Current evidence from two randomised controlled trials does not support the US in directing treatment decisions. A return to the endorsement of clinical assessment and the adoption of T2T strategies aiming at ACR/EULAR remission still represent the standard of care for RA and should be pursued through appropriate educational programmes

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Inman, R. D. "Immunogenetic aspects of host immune response." Canadian Journal of Microbiology 34, no. 3 (March 1, 1988): 319–22. http://dx.doi.org/10.1139/m88-058.

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The central role of histocompatibility leukocyte antigens (HLA) class II molecules in antigen presentation has received great attention in recent years, yet class I molecules have been defined as primarily functioning as a restriction element for cytotoxic T cell killing of virus-infected cells. Extensive clinical evidence, however, indicates that the HLA class I genes are strongly associated with nonseptic complications of enteric and genitourinary bacterial infections. Ninety percent of patients with Reiter's syndrome and reactive arthritis are positive for HLA-B27, yet the mechanism of dise

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Kreutz, G. "European regulatory aspects on new medicines targeted at treatment of rheumatoid arthritis." Annals of the Rheumatic Diseases 58, Supplement 1 (November 1, 1999): i92—i95. http://dx.doi.org/10.1136/ard.58.2008.i92.

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Doghish, Ahmed S., Ahmed Ismail, Hesham A. El-Mahdy, Samy Y. Elkhawaga, Elsayed G. E. Elsakka, Eman A. Mady, Mahmoud A. Elrebehy, Mahmoud A. F. Khalil, and Hussein M. El-Husseiny. "miRNAs insights into rheumatoid arthritis: Favorable and detrimental aspects of key performers." Life Sciences 314 (February 2023): 121321. http://dx.doi.org/10.1016/j.lfs.2022.121321.

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Pandolfi, Franco, Laura Franza, Valentina Carusi, Simona Altamura, Gloria Andriollo, and Eleonora Nucera. "Interleukin-6 in Rheumatoid Arthritis." International Journal of Molecular Sciences 21, no. 15 (July 23, 2020): 5238. http://dx.doi.org/10.3390/ijms21155238.

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The role of interleukin (IL)-6 in health and disease has been under a lot of scrutiny in recent years, particularly during the recent COVID-19 pandemic. The inflammatory pathways in which IL-6 is involved are also partly responsible of the development and progression of rheumatoid arthritis (RA), opening interesting perspectives in terms of therapy. Anti-IL-6 drugs are being used with variable degrees of success in other diseases and are being tested in RA. Results have been encouraging, particularly when anti-IL-6 has been used with other drugs, such as metothrexate (MTX). In this review we d

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Doumen, M., S. Pazmino, D. Bertrand, D. De Cock, J. Joly, R. Westhovens, and P. Verschueren. "POS0266-HPR PATIENT-PERCEIVED ASPECTS OF RA FLARE EVOLVE OVER TIME, AS REFLECTED BY THE FLARE-RA QUESTIONNAIRE: POST-HOC ANALYSIS OF TAPERA." Annals of the Rheumatic Diseases 80, Suppl 1 (May 19, 2021): 356.1–356. http://dx.doi.org/10.1136/annrheumdis-2021-eular.2003.

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Background:Flares are common in rheumatoid arthritis (RA). While flares negatively impact clinical and patient-reported outcomes, different aspects of disease activity may constitute a flare to patients. Flare Assessment in RA (FLARE-RA) is a patient-reported questionnaire aiming to detect active or recent RA flares (1). During its validation, arthritis and general health subscales were identified and the instrument was adapted from 13 questions (1-6 Likert scale) to 11 questions (0-10).Objectives:To investigate which patient-perceived aspects of flare are assessed by FLARE-RA in the context o

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Zheng, Yijun, and Duming Zhu. "Molecular Hydrogen Therapy Ameliorates Organ Damage Induced by Sepsis." Oxidative Medicine and Cellular Longevity 2016 (2016): 1–6. http://dx.doi.org/10.1155/2016/5806057.

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Since it was proposed in 2007, molecular hydrogen therapy has been widely concerned and researched. Many animal experiments were carried out in a variety of disease fields, such as cerebral infarction, ischemia reperfusion injury, Parkinson syndrome, type 2 diabetes mellitus, metabolic syndrome, chronic kidney disease, radiation injury, chronic hepatitis, rheumatoid arthritis, stress ulcer, acute sports injuries, mitochondrial and inflammatory disease, and acute erythema skin disease and other pathological processes or diseases. Molecular hydrogen therapy is pointed out as there is protective

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Shadnoush, Mahdi, Vida Nazemian, Homa Manaheji, and Jalal Zaringhalam. "Research Paper: The Effect of Orally Administered Probiotics on the Behavioral, Cellular, and Molecular Aspects of Adjuvant-Induced Arthritis." Basic and Clinical Neuroscience Journal 9, no. 5 (September 30, 2018): 325–36. http://dx.doi.org/10.32598/bcn.9.5.325.

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Nazemian, Vida, Homa Manaheji, Ali Mohammad Sharifi, and Jalal Zaringhalam. "Long term treatment by mesenchymal stem cells conditioned medium modulates cellular, molecular and behavioral aspects of adjuvant-induced arthritis." Cellular and Molecular Biology 64, no. 1 (January 31, 2018): 19. http://dx.doi.org/10.14715/cmb/2018.64.2.5.

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Corbet, Marlene, Miguel A. Pineda, Kun Yang, Anuradha Tarafdar, Sarah McGrath, Rinako Nakagawa, Felicity E. Lumb, Colin J. Suckling, William Harnett, and Margaret M. Harnett. "Suppression of inflammatory arthritis by the parasitic worm product ES-62 is associated with epigenetic changes in synovial fibroblasts." PLOS Pathogens 17, no. 11 (November 8, 2021): e1010069. http://dx.doi.org/10.1371/journal.ppat.1010069.

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ES-62 is the major secreted protein of the parasitic filarial nematode, Acanthocheilonema viteae. The molecule exists as a large tetramer (MW, ~240kD), which possesses immunomodulatory properties by virtue of multiple phosphorylcholine (PC) moieties attached to N-type glycans. By suppressing inflammatory immune responses, ES-62 can prevent disease development in certain mouse models of allergic and autoimmune conditions, including joint pathology in collagen-induced arthritis (CIA), a model of rheumatoid arthritis (RA). Such protection is associated with functional suppression of “pathogenic”

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Schoels, Monika, John Wong, David L. Scott, Angela Zink, Pamela Richards, Robert Landewé, Josef S. Smolen, and Daniel Aletaha. "Economic aspects of treatment options in rheumatoid arthritis: a systematic literature review informing the EULAR recommendations for the management of rheumatoid arthritis." Annals of the Rheumatic Diseases 69, no. 6 (May 6, 2010): 995–1003. http://dx.doi.org/10.1136/ard.2009.126714.

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ObjectiveTo review the cost effectiveness of rheumatoid arthritis (RA) treatments and inform the clinical recommendations by the European League Against Rheumatism.MethodsA systematic literature search and review of the health economic evidence on RA treatment options was performed.ResultsDespite diverse methodological approaches, health economic analyses are concordant: at onset of disease, traditional disease-modifying antirheumatic drugs (DMARDs) are cost effective—that is, treatment merits outweigh treatment costs. If DMARDs fail, therapeutic escalation with tumour necrosis factor α inhibi

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Deng, Jing, Handan Tan, Jiayue Hu, Guannan Su, Qingfeng Cao, Xinyue Huang, Chunjiang Zhou, Yao Wang, Aize Kijlstra, and Peizeng Yang. "Genetic aspects of idiopathic paediatric uveitis and juvenile idiopathic arthritis associated uveitis in Chinese Han." British Journal of Ophthalmology 104, no. 3 (April 2, 2019): 443–47. http://dx.doi.org/10.1136/bjophthalmol-2018-313200.

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BackgroundIdiopathic paediatric uveitis (IPU) and juvenile idiopathic arthritis associated uveitis (JIA-U) are the two most common entities in paediatric uveitis. This study addressed the possible association of IPU and JIA-U with genes that had been shown earlier to be associated with juvenile idiopathic arthritis.MethodsWe carried out a case-control association study involving 286 IPU, 134 JIA-U patients and 743 healthy individuals. A total of 84 candidate single nucleotide polymorphisms (SNPs) in 60 genes were selected for this study. The MassARRAY platform and iPLEX Gold Genotyping Assay w

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Kostine, Marie, Léa Rouxel, Thomas Barnetche, Rémi Veillon, Florent Martin, Caroline Dutriaux, Léa Dousset, et al. "Rheumatic disorders associated with immune checkpoint inhibitors in patients with cancer—clinical aspects and relationship with tumour response: a single-centre prospective cohort study." Annals of the Rheumatic Diseases 77, no. 3 (November 16, 2017): 393–98. http://dx.doi.org/10.1136/annrheumdis-2017-212257.

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ObjectivesTo evaluate the prevalence and type of rheumatic immune-related adverse events (irAEs) in patients receiving immune checkpoint inhibitors (ICIs), as well as the correlation with tumour response.MethodsThis was a single-centre prospective observational study including all cancer patients receiving ICIs. The occurrence of irAEs and tumour response was assessed on a regular basis. Patients who experienced musculoskeletal symptoms were referred to the department of rheumatology for clinical evaluation and management.ResultsFrom September 2015 to May 2017, 524 patients received ICIs and 3

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Nikitina, N., I. Afanaciev, and A. Rebrov. "AB0410 Use of disease-modifying treatment (dmard) in patients with rheumatoid arthritis: clinical aspects." Annals of the Rheumatic Diseases 71, Suppl 3 (June 2013): 661.1–661. http://dx.doi.org/10.1136/annrheumdis-2012-eular.410.

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Louis-Sidney, F., D. Morillon, M. Blettery, L. Brunier, P. Numeric, and M. De Bandt. "POS0945 NON-RADIOGRAPHIC AXIAL SPONDYLOARTHRITIS IN THE AFRO-CARIBBEAN POPULATION, CLINICAL ASPECTS AND PARTICULARITIES." Annals of the Rheumatic Diseases 80, Suppl 1 (May 19, 2021): 735.2–736. http://dx.doi.org/10.1136/annrheumdis-2021-eular.260.

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Background:Spondyloarthritis is a polymorphic disease and the absence of diagnostic marker has led to propose diagnostic criteria for recognition. All the criteria, established in Caucasian populations, place at the center of the approach sacroiliac imaging and genetic terrain (HLA B27). For this reason, these criteria are not appropriate in populations lacking HLA B27. SPA is known to be rare in African populations and this rarity correlates with that of HLA B27.Prevalence of B27 in French West Indies is 2% (identical to the African populations).Objectives:We report clinical manifestations of

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Pelosi, Andrea, Claudio Lunardi, Piera Filomena Fiore, Elisa Tinazzi, Giuseppe Patuzzo, Giuseppe Argentino, Francesca Moretta, Antonio Puccetti, and Marzia Dolcino. "MicroRNA Expression Profiling in Psoriatic Arthritis." BioMed Research International 2018 (2018): 1–15. http://dx.doi.org/10.1155/2018/7305380.

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Background. Psoriatic arthritis (PsA) is an inflammatory arthritis, characterized by bone erosions and new bone formation. MicroRNAs (miRNAs) are key regulators of the immune responses. Differential expression of miRNAs has been reported in several inflammatory autoimmune diseases; however, their role in PsA is not fully elucidated. We aimed to identify miRNA expression signatures associated with PsA and to investigate their potential implication in the disease pathogenesis. Methods. miRNA microarray was performed in blood cells of PsA patients and healthy controls. miRNA pathway analyses were

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Rodríguez, Sandra, Andrés Muñoz, Rosa-Helena Bustos, and Diego Jaimes. "Pharmacovigilance of Biopharmaceuticals in Rheumatic Diseases, Adverse Events, Evolution, and Perspective: An Overview." Biomedicines 8, no. 9 (August 23, 2020): 303. http://dx.doi.org/10.3390/biomedicines8090303.

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Since we have gained an understanding of the immunological pathophysiology of rheumatic diseases such as rheumatoid arthritis and systemic lupus erythematosus, treatment based on biological drugs has become a fundamental axis. These therapies are oriented towards the regulation of cytokines such as tumour necrosis factor-alpha (TNF-α), interleukin (IL)-6, IL-1, and the modulation of cell-mediated immunity (B cells and T cells) by anti CD20 or anti CTAL-4 agents, and can increase the risk of associated infections or adverse events (AE). In this context, the entry of biotherapeutics represented

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Gerami, Reza, Ramezan Jafari, Niloufar Nazeri, and Amin Saburi. "The Anti-inflammatory Role of Curcumin in Osteoarthritis: An Overview of Molecular and Radiologic Changes." Hospital Practices and Research 7, no. 1 (December 1, 2021): 1–3. http://dx.doi.org/10.34172/hpr.2022.01.

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Osteoarthritis (OA) is the most common form of arthritis, causing pain and progressive disability in millions of people worldwide. The commonly prescribed medications for OA, including non-steroidal anti-inflammatory drugs, have many side effects which has led the scientists to consider safer drugs as an alternative. Therapeutic effects of Curcumin on OA are increasingly declared, and its various aspects in suppressing inflammation and reducing the disease progression are examined more thoroughly. This study aims to discuss curcumin and OA to help scientists working in these fields. In this br

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Skovsgaard Itenov, K., N. Søe, E. M. Bartels, H. Bliddal, and M. Andersen. "AB0103 SITE SPECIFICITY OF RHEUMATOID ARTHRITIS INFLAMMATION: A SECONDARY ANALYSIS OF BIOPSIES FROM RADIAL AND ULNAR ASPECTS OF MCP JOINTS." Annals of the Rheumatic Diseases 81, Suppl 1 (May 23, 2022): 1182.2–1182. http://dx.doi.org/10.1136/annrheumdis-2022-eular.5196.

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BackgroundUlnar drift is a common complication of Rheumatoid Arthritis (RA) (1,2). There is no clear consensus regarding the etiology of the hand deformity. Observations from corrective hand surgery and other studies have noted more pronounced inflammation in the radial site of the MCP-joints (3,4). This could partly explain the pathophysiology behind the ulnar deviation.ObjectivesTo determine if there is more pronounced inflammation, measured by increased CD-68 expression (5) and Krenn-synovitis score (6), at the radial side of the MCP joints when compared to the ulnar side, in patients with

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Patenaude, S., and W. Gerhart. "AB0923-PARE WHAT IS THE IMPACT ON THE QUALITY OF LIFE (QOL) OF PEOPLE LIVING WITH SPONDYLOARTHRITIS (SPA*)?" Annals of the Rheumatic Diseases 80, Suppl 1 (May 19, 2021): 1482.1–1483. http://dx.doi.org/10.1136/annrheumdis-2021-eular.1615.

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Background:SpA describes a group of chronic inflammatory arthritic diseases with common features including inflammation of the spine, eyes, skin and gastrointestinal tract. These conditions can be painful and debilitating for many. Delayed diagnosis and treatment can lead to irreversible damage to the spine and other joints. Diagnosis of these conditions can take, on average, 7 years or more. We don’t know what causes SpA and there is no cure. The onset of symptoms can be in early childhood and expands throughout one’s lifespan. It affects children, women and men worldwide.Objectives:To unders

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