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Artykuły w czasopismach na temat "Articular cartilage – Surgery"

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Gong, Huchen, Yutao Men, Xiuping Yang, Xiaoming Li, and Chunqiu Zhang. "Experimental Study on Creep Characteristics of Microdefect Articular Cartilages in the Damaged Early Stage." Journal of Healthcare Engineering 2019 (November 13, 2019): 1–9. http://dx.doi.org/10.1155/2019/8526436.

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Traumatic joint injury is known to cause cartilage deterioration and osteoarthritis. In order to study the mechanical mechanism of damage evolution on articular cartilage, taking the fresh porcine articular cartilage as the experimental samples, the creep experiments of the intact cartilages and the cartilages with different depth defect were carried out by using the noncontact digital image correlation technology. And then, the creep constitutive equations of cartilages were established. The results showed that the creep curves of different layers changed exponentially and were not coincident
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Weitzel, Paul P. "Complications of Articular Cartilage Surgery." Sports Medicine and Arthroscopy Review 12, no. 3 (September 2004): 160–66. http://dx.doi.org/10.1097/01.jsa.0000131857.12698.65.

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Erggelet, Christoph, and Matthias Steinwachs. "Articular cartilage regeneration techniques." Current Opinion in Orthopedics 10, no. 6 (December 1999): 452–57. http://dx.doi.org/10.1097/00001433-199912000-00006.

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Ulrich-Vinther, Michael, Michael D. Maloney, Edward M. Schwarz, Randy Rosier, and Regis J. OʼKeefe. "Articular Cartilage Biology." Journal of the American Academy of Orthopaedic Surgeons 11, no. 6 (November 2003): 421–30. http://dx.doi.org/10.5435/00124635-200311000-00006.

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Trice, Michael E. "Articular cartilage surgery for the athlete." Current Orthopaedic Practice 19, no. 3 (May 2008): 299–307. http://dx.doi.org/10.1097/bco.0b013e32830349b5.

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Rosenberg, Lawrence C. "Articular Cartilage Lesions." Journal of Bone & Joint Surgery 87, no. 4 (April 2005): 921–22. http://dx.doi.org/10.2106/00004623-200504000-00033.

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Dziak, Rosemary. "Articular cartilage and osteoarthritis." Bone and Mineral 19, no. 1 (October 1992): 99–100. http://dx.doi.org/10.1016/0169-6009(92)90848-8.

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Sah, Robert L., David Amiel, and Richard D. Coutts. "Tissue engineering of articular cartilage." Current Opinion in Orthopaedics 6, no. 6 (December 1995): 52–60. http://dx.doi.org/10.1097/00001433-199512000-00011.

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Vogt, Stephan, and Andreas B. Imhoff. "Injuries to the Articular Cartilage." European Journal of Trauma 32, no. 4 (August 2006): 325–31. http://dx.doi.org/10.1007/s00068-006-6096-z.

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Karpie, John C., and Constance R. Chu. "Imaging of Articular Cartilage." Operative Techniques in Orthopaedics 16, no. 4 (October 2006): 279–85. http://dx.doi.org/10.1053/j.oto.2006.09.005.

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Rozprawy doktorskie na temat "Articular cartilage – Surgery"

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Brockmeier, Peter Macy. "Surgical Navigation for Articular Cartilage Repair: Motivation, Development, and Validation." The Ohio State University, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=osu1250219405.

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Benaroch, Thierry Ezer. "Biosynthetic response of young and adult human articular cartilage to growth factors." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=59530.

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Adult articular cartilage of many species including humans has a limited capacity for repair following injury. The hypothesis that this might be related to a lack of responsiveness to growth factors involved in growth was investigated. Cartilage explants from 4 child and 5 adult human donors were cultured in the presence of various growth factors. Incorporation of $ sp{35}$SO$ sb4$ into proteoglycans and $ sp3$H-thymidine into deoxyribonucleic acid used as measures of the biosynthetic response of cartilage.<br>Young cartilage showed the ability to behave in an autocrine or paracrine manner to
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Esa, Adam. "Characterising the role of articular cartilage progenitor cells in osteoarthritis." Thesis, Cardiff University, 2015. http://orca.cf.ac.uk/90195/.

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Osteoarthritis (OA) is a chronic and highly prevalent degenerative disease of the synovial joint leading to cartilage destruction and bone remodelling. The current management of end-stage OA is joint replacement, however, this procedure is not suitable for a subset of patients hence there is a growing need for alternative treatments and technologies to address this limitation. One such approach to this problem is the application of cell-based therapies that regenerate areas of damaged cartilage. Recently discovered articular cartilage progenitor cells (CPC) have been hallmarked as a potential
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Silva, Anderson Coutinho da. "Estudo da osteoartrose em joelhos de cães secundária à ruptura do ligamento cruzado cranial." Universidade de São Paulo, 2009. http://www.teses.usp.br/teses/disponiveis/5/5145/tde-09062009-165130/.

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INTRODUÇÃO e OBJETIVO: A osteoartrite (OA) embora frequente tem patogênese incerta em humanos. Descrevemos modelo experimental original de OA em cães, analisando em dois tempos diferentes as consequências da Ruptura Espontânea do Ligamento Cruzado Cranial (RLCCr). MÉTODOS: Vinte animais machos com menos de 5 anos ( 20 a 45 Kg) com RLCCr submetidos à artrotomia para estabilização articular tiveram fragmentos articulares removidos para análise. O grupo RLCCr < 20 (10 animais) foi operado antes dos vinte dias e o grupo > 20 (10 animais) após 20 dias do início da lesão. Sete animais com OA pré-exi
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Nguỹên, Quang. "Characterization of proteolytic agents involved in the degradation of human articular cartilage proteoglycan during aging and arthritis." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=74620.

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Some of the age-related changes in the structure of human proteoglycan aggregate, and the depletion of proteoglycan in arthritic cartilage have been attributed to the action of proteolytic agents. However, the identity of these agents is still uncertain. In this study, the cartilage matrix protein, link protein, has been used as an in situ probe of endogenous proteolysis, due to its relative resistance to proteolysis and the accumulation of its proteolytically-modified form with age in the matrix. Stromelysin, a metalloproteinase secreted in a latent form by human articular cartilage in organ
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Whale, Conley Caitlin E. "EFFECT OF A 12-WEEK HOME-BASED NEUROMUSCULAR ELECTRICAL STIMULATION TREATMENT ON CLINICAL OUTCOMES FOLLOWING ARTICULAR CARTILAGE KNEE SURGERY." UKnowledge, 2017. http://uknowledge.uky.edu/rehabsci_etds/40.

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Articular cartilage defects in the knee are common, and can result in pain, decreased function and decreased quality of life. Untreated defects are considered to be a risk factor for developing osteoarthritis, a progressive degenerative joint disease with minimal treatment options. To address these issues, various surgical procedures are available to treat articular cartilage defects in the knee. While these procedures overall have positive results, after surgery patients experience large and persistent deficits in quadriceps strength. A contributing factor to this post-surgical weakness is be
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Ebert, Jay Robert. "Post-operative load bearing rehabilitation following autologous chondrocyte implantation." University of Western Australia. School of Sport Science, Exercise and Health, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0196.

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[Truncated abstract] Autologous Chondrocyte Implantation (ACI) has shown early clinical success as a repair procedure to address focal articular cartilage defects in the knee, and involves isolating and culturing a patient's own chondrocytes in vitro and re-implantation of those cells into the cartilage defect. Over time, repair tissue can develop and remodel into hyaline-like cartilage. A progressive partial weight bearing (PWB) program becomes the critical factor in applying protection and progressive stimulation of the implanted cells, to promote best chondrocyte differentiation and develop
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Chen, Rebecca Y. "Attenuation of the Progression of Articular Cartilage Degeneration by Inhibition of Tgf-β1 Signaling in a Mouse Model of Osteoarthritis". Thesis, Harvard University, 2015. http://nrs.harvard.edu/urn-3:HUL.InstRepos:17331955.

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Background The goal of this study is to understand role of transforming growth factor beta 1 (TGF-β1) in development of osteoarthritis (OA). Results from studies indicate that the genetic inactivation of Smad-3, or the disruption of the interaction of Tgf-β1 with its receptor Tgf-β type II receptor (Tgfbr2), in germline cells results in OA-like knee joints in mice at one month of age. However, other studies suggest that the increased expression of Tgf-β1 in mature knee joints causes OA in animal models. A human genetic study reports that a two-nucleotide deletion, 741-742del AT, and/or a nucle
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Robertson, William Brett. "Functional and radiological evaluation of autologous chondrocyte implantation using a type I/III collagen membrane: from single defect treatment to early osteoarthritis." University of Western Australia. Orthopaedics Unit, 2007. http://theses.library.uwa.edu.au/adt-WU2007.0172.

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[Truncated abstract] Hyaline articular cartilage is a highly specialised tissue consisting of chondrocytes embedded in a matrix of proteoglycan and collagens. Hyaline articular cartilage withstands high levels of mechanical stress and continuously renews its extracellular matrix. Despite this durability, mature articular cartilage is vulnerable to injury and disease processes that cause irreparable tissue damage. Native hyaline articular cartilage has poor regenerative capacity following injury, largely due to the tissue's lack of blood and lymphatic supply, as well as the inability of native
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Rizkalla, Geihan. "Immunochemical studies of aggregating proteoglycans in normal and osteoarthritic human articular cartilages." Thesis, McGill University, 1990. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=59642.

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The aim of the present investigation was to study the structural variability of osteoarthritic human femoral condylar cartilage proteoglycans that can aggregate with hyaluronic acid as compared with site and age matched normal adult cartilage proteoglycans. Using specific antibodies to different parts of the proteoglycan molecule in radioimmunoassays, we were able to detect four proteoglycan populations of different hydrodynamic sizes in normal, as well as in osteoarthritic articular cartilage. These populations were: a large chondroitin 6-sulfate, a smaller chondroitin 4-sulfate, and an even
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Książki na temat "Articular cartilage – Surgery"

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Cartilage tympanoplasty. Stuttgart: Thieme, 2009.

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Minas, Tom. A primer in cartilage repair and joint preservation of the knee. Philadelphia, PA: Elsevier/Saunders, 2011.

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European Congress of Knee Surgery and Arthroscopy (1st 1984 Berlin, Germany). Surgery and arthroscopy of the knee: First European Congress of Knee Surgery and Arthroscopy, Berlin, 9-14.4.1984. Berlin: Springer-Verlag, 1986.

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Bristol-Myers/Zimmer Orthopaedic Symposium (4th 1988 Chicago, Ill.). Articular cartilage and knee joint function: Basic science and arthroscopy. Edited by Ewing J. Whit, Arthroscopy Association of North America., and Bristol-Myers/Zimmer (Firm). New York: Raven Press, 1990.

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1964-, Hendrich Christian, Nöth Ulrich 1967-, and Eulert Jochen, eds. Cartilage surgery and future perspectives. Berlin: Springer, 2003.

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Farr, Jack, and Andreas H. Gomoll. Cartilage Restoration: Practical Clinical Applications. Springer, 2016.

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Farr, Jack, and Andreas H. Gomoll. Cartilage Restoration: Practical Clinical Applications. Springer, 2013.

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Farr, Jack, and Andreas H. Gomoll. Cartilage Restoration: Practical Clinical Applications. Springer, 2018.

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Cartilage surgery: An operative manual. Philadelphia, PA: Elsevier Saunders, 2011.

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F, Houlton John E., and British Small Animal Veterinary Association., eds. BSAVA manual of canine and feline musculoskeletal disorders. Gloucester [England]: British Small Animal Veterinary Association, 2006.

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Części książek na temat "Articular cartilage – Surgery"

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van der Linden, A. J. "Repair of articular cartilage." In Surgery and Arthroscopy of the Knee, 27–30. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71022-3_13.

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Tomford, William W., Christian Ohlendorf, and Henry J. Mankin. "Articular Cartilage Cryopreservation and Transplantation." In Orthopaedic Allograft Surgery, 269–73. Vienna: Springer Vienna, 1996. http://dx.doi.org/10.1007/978-3-7091-6885-1_31.

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Jung, Chan Kwon. "Articular Cartilage: Histology and Physiology." In Techniques in Cartilage Repair Surgery, 17–21. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-41921-8_2.

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Geesink, R. G. T. "Stress response of articular cartilage." In Surgery and Arthroscopy of the Knee, 23–24. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71022-3_11.

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Lansdown, Drew A., Kevin C. Wang, and Brian J. Cole. "Defining Failure in Articular Cartilage Surgery." In Joint Preservation of the Knee, 69–82. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-01491-9_5.

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Aigner, Thomas, and Zhiyong Fan. "Anatomy and Biochemistry of Articular Cartilage." In Cartilage Surgery and Future Perspectives, 3–7. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-19008-7_1.

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Nöth, Ulrich, Arne Berner, Richard Tuli, Achim Battmann, Christian Hendrich, Jochen Eulert, and Rocky S. Tuan. "Fabrication of Cartilage-Polymer Constructs for Articular Cartilage Repair." In Cartilage Surgery and Future Perspectives, 165–70. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-19008-7_19.

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Cavanaugh, John T. "Rehabilitation Strategies Following Articular Cartilage Surgery in the Knee." In Cartilage Repair Strategies, 343–69. Totowa, NJ: Humana Press, 2007. http://dx.doi.org/10.1007/978-1-59745-343-1_20.

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Kim, Seok-Jung, Asode Ananthram Shetty, and Vishvas A. Shetty. "Gel ACI (GACI): Articular Cartilage Repair Technique." In Techniques in Cartilage Repair Surgery, 175–86. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-41921-8_15.

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Bauer, M., R. W. Jackson, and D. Ogilvie-Harris. "Articular cartilage lesions of the femoral condyles." In Surgery and Arthroscopy of the Knee, 31–36. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71022-3_14.

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Streszczenia konferencji na temat "Articular cartilage – Surgery"

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Jaumard, Nicolas V., Joel A. Bauman, William C. Welch, and Beth A. Winkelstein. "Biomechanical Comparison of Contact Pressure in the Cervical Facet Joint During Bending Using a Probe and Pressure-Sensitive Paper." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19498.

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Non-physiologic loading of the facet joint is a potential cause of facet joint pain in the cervical spine [1]. When the local biomechanical environment of the facet joint is altered, like with trauma or after surgery [2], the cartilaginous articular surfaces of the facets can also be damaged. Defining articular contact pressure can provide a metric of altered joint mechanics and the local mechanical environment of the cartilage in the facet joint. However, accessing the articular surface to make such measurements without altering the overall mechanics of the joint remains a substantial challen
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Linder-Ganz, Eran, Gal Zur, Jonathan Shani, Jonathan J. Elsner, Ori Brenner, Steven P. Arnoczky, Gabriel Agar, Elliott B. Hershman, and Avi Shterling. "Can a Polycarbonate-Urethane Meniscal Implant Protect Articular Cartilage? Histopathological Results in a Sheep Model." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-204860.

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The menisci play an important role in the knee joint biomechanics [1]. Clinical studies have shown that the loss of the meniscus leads to degenerative arthritis attributed to the changes in load distribution and the loss of proprioception [2]. Clearly, there is a substantial need to protect the articular cartilage by either repairing or replacing the menisci. There are many difficulties dealing with both fresh frozen or cryopreserved allograft menisci, and the complexities of meniscal repairs may contribute to uneven distribution of load, instability and recurrence of degenerative damage. Henc
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Linder-Ganz, Eran, Jonathan J. Elsner, Gal Zur, Jonathan Shani, Ori Brenner, Elliott Hershman, Avi Shterling, and Farshid Guilak. "Chondroprotective Effects of a Polycarbonate-Urethane Meniscal Implant: Semi-Quantitative Results in a Sheep Model." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19048.

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The menisci play a critical role in load-bearing and stability of the knee joint [1]. Damage or removal of the meniscus leads to alterations in the magnitude and distribution of stresses in the knee, which have been associated with degenerative osteoarthritis [2]. Clearly, there remains a need to develop means of protecting the articular cartilage following meniscal injury by either repairing or replacing the menisci. While allograft meniscal replacements can improve joint stability and function, they often provide little benefit in preventing osteoarthritic changes [3]. The development of an
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Baer, Thomas, Ryan Frisbie, Michael Willey, and Jessica Goetz. "Development of a Simplified Ankle Distractor." In 2017 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dmd2017-3438.

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The physical impairment caused by OA of a single lower extremity joint is comparable to that reported for major life-altering disorders such as end-stage kidney disease and heart failure. (Buckwalter, et al) [1] Ankle distraction arthroplasty has been shown to greatly decrease pain due to end-stage ankle arthritis. Unlike arthrodesis (fusion of the joint), distraction arthroplasty maintains the joint’s natural movement, and it is far less complicated than total joint replacement surgery. There is a considerable body of research supporting the idea that distraction of an end-stage arthritic joi
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Meyer, Eric G., Conor T. Buckley, and Daniel J. Kelly. "The Effect of Cyclic Hydrostatic Pressure on the Functional Development of Cartilaginous Tissues Engineered Using Bone Marrow Derived Mesenchymal Stem Cells." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53634.

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Articular cartilage has a poor capacity for repair. Of the many procedures available to the orthopaedic surgeon, osteochondral grafting is the only technique which reliably produces hyaline cartilage within a defect.1 Bone marrow derived mesenchymal stem cells (MSCs) are an interesting alternative to harvesting cartilage grafts for chondrocytes as they also have the ability to produce cartilaginous tissues in vitro. This suggests that if tissue engineering strategies could be used to develop cartilaginous grafts with mechanical properties approaching that of normal articular cartilage, then hy
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Martínez Bocanegra, Marco A., Javier Bayod Lopez, A. Vidal-Lesso, Ricardo Becerro de Bengoa Vallejo, Raúl Lesso Arroyo, and Humberto Corro Hernández. "Biomechanics Aspects for Silastic Implant Arthroplasty Simulation of the First Metatarsophalangeal Joint." In ASME 2015 International Mechanical Engineering Congress and Exposition. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/imece2015-53525.

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This work focuses on the biomechanical simulation of surgery for total replacement of the first metatarsophalangeal joint (MTPJ) allowed us to identify and analyze several key aspects for finite element simulation of hallux rigidus pathology. Predicting the optimal response of a finite element model (FEM) depends on proper characterization. At this part of the work, those conditions that have a direct or indirect influence on the model that can change its behavior should be considered. For this purpose, we presented in this work a finite element model which include 26 bones: 14 phalanges, 5 me
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