Gotowe bibliografie tematyczne / Astrocytes

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Gotowa bibliografia na temat „Astrocytes”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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Artykuły w czasopismach na temat "Astrocytes"

Nett, Wolfgang J., Scott H. Oloff, and Ken D. McCarthy. "Hippocampal Astrocytes In Situ Exhibit Calcium Oscillations That Occur Independent of Neuronal Activity." Journal of Neurophysiology 87, no. 1 (2002): 528–37. http://dx.doi.org/10.1152/jn.00268.2001.

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Results presented in this study indicate that a large subpopulation (∼65%) of hippocampal astrocytes in situ exhibit calcium oscillations in the absence of neuronal activity. Further, the spontaneous oscillations observed within individual hippocampal astrocytes generally developed asynchronously throughout the astrocyte's fine processes and occasionally spread through a portion of that astrocyte as a calcium wave but do not appear to spread among astrocytes as an intercellular calcium wave. Bath application of cyclopiazonic acid and injection of individual astrocytes with heparin blocked astr

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Huang, Mi, Yixing Du, Conrad Kiyoshi, et al. "Syncytial Isopotentiality: An Electrical Feature of Spinal Cord Astrocyte Networks." Neuroglia 1, no. 1 (2018): 271–79. http://dx.doi.org/10.3390/neuroglia1010018.

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Due to strong electrical coupling, syncytial isopotentiality emerges as a physiological mechanism that coordinates astrocytes into a highly efficient system in brain homeostasis. Although this electrophysiological phenomenon has now been observed in astrocyte networks established by different astrocyte subtypes, the spinal cord remains a brain region that is still unexplored. In ALDH1L1-eGFP transgenic mice, astrocytes can be visualized by confocal microscopy and the spinal cord astrocytes in grey matter are organized in a distinctive pattern. Namely, each astrocyte resides with more directly

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Domingos, Cátia, Franziska E. Müller, Stefan Passlick, et al. "Induced Remodelling of Astrocytes In Vitro and In Vivo by Manipulation of Astrocytic RhoA Activity." Cells 12, no. 2 (2023): 331. http://dx.doi.org/10.3390/cells12020331.

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Structural changes of astrocytes and their perisynaptic processes occur in response to various physiological and pathophysiological stimuli. They are thought to profoundly affect synaptic signalling and neuron-astrocyte communication. Understanding the causal relationship between astrocyte morphology changes and their functional consequences requires experimental tools to selectively manipulate astrocyte morphology. Previous studies indicate that RhoA-related signalling can play a major role in controlling astrocyte morphology, but the direct effect of increased RhoA activity has not been docu

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Amuti, T., I. Ouko, S. Mukonjia, et al. "Role of heterogeneous astrocyte receptor expression in determining astrocytic response to neuronal disorders." Anatomy Journal of Africa 7, no. 1 (2018): 1169–74. http://dx.doi.org/10.4314/aja.v7i1.169490.

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Following neuronal disorders, astrocytes carry out either neuroprotection or neurodegeneration. Previous authors suggest that favoring of neurodegeneration or neuroprotection by astrocytes can be due to many factors such as the influence of cytokines following their binding on their receptors on astrocytes. These receptors have however been shown to be region specific and heterogeneous. Further, research exploiting their role and influence in determining astrocytic response remains partly elucidated. A review of previous and ongoing research on these receptors would be helpful in the disclosur

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Wolfes, Anne C., Saheeb Ahmed, Ankit Awasthi, et al. "A novel method for culturing stellate astrocytes reveals spatially distinct Ca2+ signaling and vesicle recycling in astrocytic processes." Journal of General Physiology 149, no. 1 (2016): 149–70. http://dx.doi.org/10.1085/jgp.201611607.

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Interactions between astrocytes and neurons rely on the release and uptake of glial and neuronal molecules. But whether astrocytic vesicles exist and exocytose in a regulated or constitutive fashion is under debate. The majority of studies have relied on indirect methods or on astrocyte cultures that do not resemble stellate astrocytes found in vivo. Here, to investigate vesicle-associated proteins and exocytosis in stellate astrocytes specifically, we developed a simple, fast, and economical method for growing stellate astrocyte monocultures. This method is superior to other monocultures in t

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Escalada, Paula, Amaia Ezkurdia, María Javier Ramírez, and Maite Solas. "Essential Role of Astrocytes in Learning and Memory." International Journal of Molecular Sciences 25, no. 3 (2024): 1899. http://dx.doi.org/10.3390/ijms25031899.

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One of the most biologically relevant functions of astrocytes within the CNS is the regulation of synaptic transmission, i.e., the physiological basis for information transmission between neurons. Changes in the strength of synaptic connections are indeed thought to be the cellular basis of learning and memory. Importantly, astrocytes have been demonstrated to tightly regulate these processes via the release of several gliotransmitters linked to astrocytic calcium activity as well as astrocyte–neuron metabolic coupling. Therefore, astrocytes seem to be integrators of and actors upon learning-

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Noeker, Jacob, Carmen Nanclares, Alfonso Araque, and Andrés Baraibar. "Astrocyte-neuron signaling in aging." Ageing & Longevity, no. 2. 2025 (February 27, 2025): 136–43. https://doi.org/10.47855/jal9020-2025-2-5.

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Rapid increases in the aging population present a critical challenge to global societies, with a major concern being cognitive decline and brain aging. While neuroscientific research has traditionally focused on neurons, the role of astrocytes in aging is of recent interest but remains underexplored. Astrocytes, the homeostatic cells on the central nervous system, play essential roles in synaptic regulation, neurotransmitter clearance, neuronal support, and homeostasis. Recent studies indicate that astrocytes undergo significant morphological and functional changes during aging, affecting calc

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Nassar, Ajmal, Triveni Kodi, Sairaj Satarker, et al. "Astrocytic MicroRNAs and Transcription Factors in Alzheimer’s Disease and Therapeutic Interventions." Cells 11, no. 24 (2022): 4111. http://dx.doi.org/10.3390/cells11244111.

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Astrocytes are important for maintaining cholesterol metabolism, glutamate uptake, and neurotransmission. Indeed, inflammatory processes and neurodegeneration contribute to the altered morphology, gene expression, and function of astrocytes. Astrocytes, in collaboration with numerous microRNAs, regulate brain cholesterol levels as well as glutamatergic and inflammatory signaling, all of which contribute to general brain homeostasis. Neural electrical activity, synaptic plasticity processes, learning, and memory are dependent on the astrocyte–neuron crosstalk. Here, we review the involvement of

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Emerson, Jacen, Thomas Delgado, Peter Girardi, and Gail V. W. Johnson. "Deletion of Transglutaminase 2 from Mouse Astrocytes Significantly Improves Their Ability to Promote Neurite Outgrowth on an Inhibitory Matrix." International Journal of Molecular Sciences 24, no. 7 (2023): 6058. http://dx.doi.org/10.3390/ijms24076058.

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Astrocytes are the primary support cells of the central nervous system (CNS) that help maintain the energetic requirements and homeostatic environment of neurons. CNS injury causes astrocytes to take on reactive phenotypes with an altered overall function that can range from supportive to harmful for recovering neurons. The characterization of reactive astrocyte populations is a rapidly developing field, and the underlying factors and signaling pathways governing which type of reactive phenotype that astrocytes take on are poorly understood. Our previous studies suggest that transglutaminase 2

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Koyama, Yutaka. "Endothelin ETB Receptor-Mediated Astrocytic Activation: Pathological Roles in Brain Disorders." International Journal of Molecular Sciences 22, no. 9 (2021): 4333. http://dx.doi.org/10.3390/ijms22094333.

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In brain disorders, reactive astrocytes, which are characterized by hypertrophy of the cell body and proliferative properties, are commonly observed. As reactive astrocytes are involved in the pathogenesis of several brain disorders, the control of astrocytic function has been proposed as a therapeutic strategy, and target molecules to effectively control astrocytic functions have been investigated. The production of brain endothelin-1 (ET-1), which increases in brain disorders, is involved in the pathophysiological response of the nervous system. Endothelin B (ETB) receptors are highly expres

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Rozprawy doktorskie na temat "Astrocytes"

Contreras-Sesvold, Carmen Sesvold Carmen Contreras. "Reactive astrocytes : phenotypic and functional characteristics and astrocytes as neural stem cells /." Download the thesis in PDF, 2006. http://www.lrc.usuhs.mil/dissertations/pdf/ContrerasSesvold2006.pdf.

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Feresten, Abigail Helms. "Astrocytes in psychotic disorder." Thesis, University of British Columbia, 2013. http://hdl.handle.net/2429/45435.

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Astrocyte dysregulation has been implicated in the pathophysiology of schizophrenia (SCZ) and bipolar disorder (BPD), however the exact nature of astrocytic alterations remains to be identified. I investigated whether levels of four astrocyte-specific proteins; glial fibrillary acidic protein (GFAP), aldehyde dehydrogenase type 1L1 (ALDH1L1), vimentin, and excitatory amino acid transporter type 1 (EAAT1) are altered in SCZ and BPD. Immunohistochemical staining of ALDH1L1 and GFAP in human grey and white matter was also performed, and staining patterns compared qualitatively. Relative concentra

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Jai-Yoon, Sul. "Calcium signalling in astrocytes." Thesis, King's College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.391921.

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Škovierova, H., S. Mahmood, E. Blahovcova, J. Strnadel, J. Sopkova, and E. Halašova. "Homocystene and human astrocytes." Thesis, Сумський державний університет, 2016. http://essuir.sumdu.edu.ua/handle/123456789/44950.

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Astrocytes are multipotent and serve surprisingly large and diverse variety of functions, providing for the overall brain homeostasis, assisting in neurogenesis, determining the microarchitecture of the grey matter, and defending the brain through evolutionary conserved astrogliosis programs. Astrocytes are specifically involved in various neurodegenerative diseases, including Alzheimer’s and Parkinson’s diseases, and various forms of dementia. Homocysteine is a nonessential sulphur-containing amino acid that had been linked with neurodegenerative diseases and aging.

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James, L. R. "Calcium signal transduction in astrocytes." Thesis, University of Cambridge, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.605022.

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Ca2+ signals can exhibit great spatiotemporal complexity, leading to the hypothesis that the dynamics of Ca2+ signals may allow astrocytes to discriminate between stimuli. An in vitro model system of primary cerebellar and cortical astrocytes was tailored to test this hypothesis, by comparing the kinetics of the Ca2+ signal evoked by different receptor agonists. It was found that known physiological agonists triggered highly heterogeneous responses, but there were no systematic trends in the specific kinetic parameters of Ca2+ signals that dep

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Cambray-Deakin, M. "Astrocytes : targets for neuroactive substances." Thesis, Open University, 1985. http://oro.open.ac.uk/56910/.

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Cahoy, John David. "Genomic analysis of highly purified astrocytes reveals in vivo astrocyte gene expression : a new resource for understanding astrocyte development and function /." May be available electronically:, 2007. http://proquest.umi.com/login?COPT=REJTPTU1MTUmSU5UPTAmVkVSPTI=&clientId=12498.

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Mhyre, Andrew James. "Mechanisms of estrogen signaling in astrocytes /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/6266.

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Siushansian, Ramin. "Vitamin C transport by cerebral astrocytes." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/nq21315.pdf.

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Blaszczyk, Lucie. "Etude des cellules astrocytaires et microgliales thalamiques dans un modèle de douleur neuropathique chez le rat." Thesis, Bordeaux, 2015. http://www.theses.fr/2015BORD0081/document.

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La douleur chronique est une pathologie invalidante de longue durée notamment caractériséepar trois symptômes : l’allodynie (un stimulus non douloureux est perçu comme douloureux),l’hyperalgésie (un stimulus douloureux est perçu comme encore plus douloureux) et desdouleurs ambulatoires. Quand cette douleur est due à une lésion ou une dysfonction du systèmenerveux on parle de douleur neuropathique. Chez les patients et les modèles animaux dedouleurs neuropathiques, les études ont montré que les neurones thalamiques étaienthyperexcitables. Les cellules gliales, astrocytes et microglies, sont des

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Książki na temat "Astrocytes"

Di Benedetto, Barbara, ed. Astrocytes. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9068-9.

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Milner, Richard, ed. Astrocytes. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-452-0.

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Sergey, Fedoroff, and Vernadakis Antonia 1930-, eds. Astrocytes. Academic Press, 1986.

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Di Benedetto, Barbara, ed. Astrocytes. Springer US, 2025. https://doi.org/10.1007/978-1-0716-4366-2.

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1950-, Murphy Seán, ed. Astrocytes: Pharmacology and function. Academic Press, 1993.

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Li, Baoman, Vladimir Parpura, Alexei Verkhratsky, and Caterina Scuderi, eds. Astrocytes in Psychiatric Disorders. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-77375-5.

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1954-, Schipper Hyman M., ed. Astrocytes in brain aging and neurodegeneration. R.G. Landes, 1998.

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Pérez, Oscar González. Astrocytes: Structure, functions and role in disease. Nova Science Publishers, Inc., 2012.

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Gates, Jay Alan. Characterization of calmodulin from C astrocytoma cells in rat cerebrum. s.n.], 1986.

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Haydon, Philip G., and Vladimir Parpura, eds. Astrocytes in (Patho)Physiology of the Nervous System. Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-79492-1.

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Części książek na temat "Astrocytes"

Rojas-Rodríguez, Felipe, Andrés Pinzón, Daniel Fuenmayor, et al. "Multi-Omic Epigenetic-Based Model Reveals Key Molecular Mechanisms Associated with Palmitic Acid Lipotoxicity in Human Astrocyte." In Neurotoxicity - New Advances. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.100133.

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Astrocytes are critical for the metabolic, structural and functional modulatory support of the brain. Lipotoxicity or high levels of saturated fatty acid as Palmitate (PA) has been associated with neurotoxicity, the loss or change of astrocytic functionality, and the etiology and progression of neurodegenerative diseases such as Parkinson or Alzheimer. Several molecular mechanisms of PA’s effect in astrocytes have been described, yet the role of epigenetic regulation and chromatin architecture have not been fully explored. In this study, we developed a multi-omic epigenetic-based model to iden

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Bedner, Peter, Allison R. Peterson, Devin K. Binder, and Christian Steinhäuser. "A Crucial Role for Astrocytes in Epileptogenesis." In Jasper's Basic Mechanisms of the Epilepsies, 5th ed., edited by Annamaria Vezzani and Helen E. Scharfman. Oxford University PressNew York, 2024. http://dx.doi.org/10.1093/med/9780197549469.003.0026.

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Abstract A growing body of evidence suggests that astrocytes are crucial actors in the initiation and progression of epilepsy. Reactive astrocytes in human and experimental epilepsy display marked morphological, transcriptional, and functional changes, but it is still often unclear whether this represents a causative factor, a consequence, or an adaptive response in epileptogenesis. A distinctive feature of astrocytes is their extensive intercellular coupling via gap junction channels. This allows them to form large syncytium-like functional networks that play essential roles in ion and neurot

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Jayasinghe, Ravindri, and Nadun Danushka. "Astrocytes in Glioblastoma Therapy: A Novel Approach to Targeting Tumor Microenvironment." In Challenge of Glioblastoma - From Pathology to Survival [Working Title]. IntechOpen, 2025. https://doi.org/10.5772/intechopen.1008927.

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Glioblastomas (GBM) are one of the most aggressive tumors of the brain with a devastating prognosis despite its standard treatment of chemoradiotherapy and surgery. Astrocytes play a vital role in the tumor microenvironment (TME) of GBM, significantly influencing tumor progression and therapeutic outcomes. This chapter explores the complex interplay between astrocytes and GBM cells, highlighting the potential of targeting astrocytes within the TME as a novel therapeutic approach. Astrocytes undergo astrogliosis in response to GBM, adopting a reactive phenotype that contributes to tumor growth

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Boulbaroud, Samira, Hanane Khalki, and Fatima Zahra Azzaoui. "Cognitive Function Involving Glial Cells." In Physiology and Function of Glial Cells in Health and Disease. IGI Global, 2023. http://dx.doi.org/10.4018/978-1-6684-9675-6.ch003.

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Astrocytes, once considered passive support cells in the central nervous system, are now recognized as dynamic contributors to neuronal processes. They play pivotal roles in regulating synaptic transmission, modulating excitability, and influencing synapse formation. These non-neuronal cells release gliotransmitters like glutamate, affecting synaptic activity. Dysfunctions in astrocytes are linked to neurodegenerative and psychiatric disorders. In neurodegenerative disorders like Alzheimer's and Parkinson's, astrocytic dysfunction plays distinct roles. While astrocytes may not significantly co

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Brustle, O., and M. Dubois-Dalcq. "Stem Cells and their Gliogenic Potential." In Glial Cell Development basic principles and clinical relevance second edition. Oxford University PressOxford, 2001. http://dx.doi.org/10.1093/oso/9780198524786.003.0020.

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Abstract A detailed understanding of the lineage relationships between the three principal cell types of the central nervous system (CNS)—neurons, astrocytes and oligodendrocytes-is a central issue in the study of CNS development and repair. The discovery that in vitro type 2 astrocytes and oligodendrocytes can develop from a common, self-renewing progenitor established the existence of a bipotential precursor for CNS glia (Raff et al., 1983; Bögler et al., 1990). These experiments also showed that the decision of a progenitor cell to differentiate into an oligodendrocyte or a type 2 astrocyte

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"Front Matter." In Astrocytes. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-12-511370-0.50001-1.

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"Copyright." In Astrocytes. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-12-511370-0.50002-3.

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"Contributors." In Astrocytes. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-12-511370-0.50003-5.

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Murphy, Sean. "Preface." In Astrocytes. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-12-511370-0.50004-7.

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LEVISON, STEVEN W., and JAMES E. GOLDMAN. "Astrocyte Origins." In Astrocytes. Elsevier, 1993. http://dx.doi.org/10.1016/b978-0-12-511370-0.50005-9.

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Streszczenia konferencji na temat "Astrocytes"

Gupta, Deven K., Clover Yang, Trey Highland, David A. Miller, and Adam Wax. "Leveraging Kramers-Kronig Relations to Enhance Signal Retrieval of Quantitative Phase Microscopy." In Novel Techniques in Microscopy. Optica Publishing Group, 2025. https://doi.org/10.1364/ntm.2025.nth2c.4.

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We leveraged Kramers-Kronig relations to enhance fluorescence signal retrieval, enabling improved imaging of optic nerve head astrocytes within 3D hydrogel culture. This model uniquely combines microfluidics and interstitial flow to study glaucoma-associated mechanobiology.

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Chen, Wenjing, Xiaotong Gu, and Feifan Zhou. "Photobiomodulation attenuates neuroinflammation in primary astrocytes identified by transcriptome analysis." In Biophotonics and Immune Responses XX, edited by Wei R. Chen and Feifan Zhou. SPIE, 2025. https://doi.org/10.1117/12.3039921.

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Li, Haoran, Jiahua Shi, Huaming Chen, et al. "FDNet: Frequency Domain Denoising Network For Cell Segmentation In Astrocytes Derived From Induced Pluripotent Stem Cells." In 2024 IEEE International Symposium on Biomedical Imaging (ISBI). IEEE, 2024. http://dx.doi.org/10.1109/isbi56570.2024.10635607.

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Patel, Kush C., Mina Shenouda, and Diana Martinex. "The Effects of Circadian Misalignment on Astrocyte Morphology in the Nucleus Tractus Solitarius." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.154_2024.

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Circadian misalignment, as experienced by shift workers, was found to increase the number of astrocytes in the nucleus tractus solitarius (nTS) - a brain region crucial for autonomic control of blood pressure. Sprague Dawley rats subjected to a 6-hour light-dark phase delay every 2 days to simulate shift work exhibited a significant increase in astrocyte cell bodies compared to control rats, both at 9am and 9pm timepoints. The increase in astrocytes may lead to greater glutamate uptake, reducing extracellular glutamate levels in the nTS. Decreased glutamatergic neurotransmission in this region

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Dias, Thales Augusto Oliveira, and Silvia Graciela Ruginsk Leitão. "Participation of calcium channels in the action of angiotensin II in astrocytes." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.299.

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Background: The renin-angiotensin-aldosterone system is the main regulator of blood pressure and blood volume, with most effects being mediated by angiotensin II (Ang-II) - responsible, in the central nervous system, for actions such as thirst and sodium appetite. Astrocytes are believed to mediate such a response, as they express receptors for Ang-II and respond directly to dehydration with impacting morphological changes in the synaptic microenvironment. Many of its functions involve L-type calcium channels (LTCCs). Objectives: Evaluate the participation of LTCCs in the effects induced by An

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Bernick, Kristin B., and Simona Socrate. "Substrate Dependence of Mechanical Response of Neurons and Astrocytes." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53538.

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The response of neural cells to mechanical cues is a critical component of the innate neuroprotective cascade aimed at minimizing the consequences of traumatic brain injury (TBI). Reactive gliosis and the formation of glial scars around the lesion site are among the processes triggered by TBI where mechanical stimuli play a central role. It is well established that the mechanical properties of the microenvironment influence phenotype and morphology in most cell types. It has been shown that astrocytes change morphology [1] and cytoskeletal content [2] when grown on substrates of varying stiffn

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Rocha, Andreia, Bruna Bellaver, Luiza Machado, et al. "TEMPORAL CHANGES IN ASTROCYTES ON A TRANSGENIC RAT MODEL OF AD." In XIII Meeting of Researchers on Alzheimer's Disease and Related Disorders. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1980-5764.rpda023.

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Background: Recent evidences have pointed to astrocytes as important players in the Alzheimer’s Disease (AD) pathogenesis. Objective: With this in mind, we aim to longitudinally investigate astrocyte changes in a new important AD transgenic model, the TgF344-AD rat, the first animal model harboring human APP/PS1 mutations which presents age-dependent amyloid and tau pathology. Methods: TgF344-AD rats and wild type littermates were evaluated in three time points: 3, 6 and 9 months of age. Rats underwent a [18F]FDG-microPET, a spatial-memory, an astrocytes CSF biomarkers (ELISA multiplex) and a

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Ravagnani, Felipe, Hellen Valerio, Jersey Maués, et al. "Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.414.

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Progressive Supranuclear Palsy (PSP) is a neurodegenerative tauopathy and, to date, the pathophysiological mechanisms in PSP that lead to Tau hyperphosphorylation and neurodegeneration are not clear. The development of a model using neural cell lines derived from patients has the potential to identify molecules and possible biomarkers. We developed a model of induced pluripotent stem cells iPSC-derived astrocytes to investigate the pathophysiology of PSP, particularly early events that might contribute to Tau hyperphosphorylation, applying an omics approach to detect differentially expressed g

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Pletnikov, Mikhail V. "ASTROCYTES IN COGNITIVE DYSFUNCTION." In MODERN PROBLEMS IN SYSTEMIC REGULATION OF PHYSIOLOGICAL FUNCTIONS. NPG Publishing, 2019. http://dx.doi.org/10.24108/5-2019-confnf-7.

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Leung, Lai Yee, Pamela J. VandeVord, Warren Hardy, Roche De Guzman, King H. Yang, and Albert I. King. "Effects of Short Duration Overpressure on Astrocytes: An In Vitro Study." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176202.

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Blast wave overpressure from detonations can injure physiological systems ‘silently.’ Experimental and clinical studies have revealed the damaging effects of shock waves on different physiological systems, such as ears, lungs and gastrointestinal tracts [1, 2]. Despite the improved helmet and body armor, many veterans returning from wars suffered from neurological disorders that are being diagnosed as mild traumatic brain injury. Warden (2006) reported that most of these veterans were exposed to blast [3]. In vivo study illustrated neuronal degeneration in the brain after exposure to blast wav

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Raporty organizacyjne na temat "Astrocytes"

Rumbaut Gil, Ana Laura. A Systemic Analysis of Vascular Dysfunction in Parkinson’s Disease: Reviewing the Roles of Astrocytes and COX-2. Florida International University, 2025. https://doi.org/10.25148/fiuurj.3.1.6.

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Although Parkinson’s Disease (PD) is the second most prevalent neurodegenerative disease in the U.S., the pathology remains an enigma. Many neuroinflammation hypotheses have been studied to explain its development. It’s shown that inflammatory markers such as COX-2 activity, which synthesizes the potent vasodilator PGE2, are overexpressed in PD. Additionally, astrocytes, regulators of inflammation in the brain and BBB, undergo important changes during PD. However, the vascular consequences that all this has for PD-led neurodegeneration are relatively unexplored. We conducted a systemic review

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Yang, Lin, Yanzhu Liu, Trudy M. Forte, Jeffrey W. Chisholm, John S. Parks, and Neil S. Shachter. Cultured human astrocytes secrete large cholesteryl ester- andtriglyceride-rich lipoproteins along with endothelial lipase. Office of Scientific and Technical Information (OSTI), 2003. http://dx.doi.org/10.2172/886608.

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Elmann, Anat, Orly Lazarov, Joel Kashman, and Rivka Ofir. therapeutic potential of a desert plant and its active compounds for Alzheimer's Disease. United States Department of Agriculture, 2015. http://dx.doi.org/10.32747/2015.7597913.bard.

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We chose to focus our investigations on the effect of the active forms, TTF and AcA, rather than the whole (crude) extract. 1. To establish cultivation program designed to develop lead cultivar/s (which will be selected from the different Af accessions) with the highest yield of the active compounds TTF and/or achillolide A (AcA). These cultivar/s will be the source for the purification of large amounts of the active compounds when needed in the future for functional foods/drug development. This task was completed. 2. To determine the effect of the Af extract, TTF and AcA on neuronal vulnerabi

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Li, Nianzhen. Nitric Oxide in Astrocyte-Neuron Signaling. Office of Scientific and Technical Information (OSTI), 2002. http://dx.doi.org/10.2172/803739.

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Sulzer, David. Altered Astrocyte-Neuron Interactions and Epileptogenesis in Tuberous Sclerosis Complex Disorder. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada610226.

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Maragakis, Nicholas J., and Hongjun Song. Preclinical Studies of Induced Pluripotent Stem Cell-Derived Astrocyte Transplantation in ALS. Defense Technical Information Center, 2014. http://dx.doi.org/10.21236/ada613757.

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Kahler, David W., and Carmen M. Arroyo. Normal Human Astrocyte Instructions for Initiation of Cultures from Cryopreserved Cells and Subculture. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada442897.

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มณีศรี เลอกรองด์, ศุภางค์, ธนัญญา ทองตัน та อนันต์ ศรีเกียรติขจร. ผลของการได้รับยาพาราเซตตามอลอย่างเรื้อรังที่มีผลต่อการเปลี่ยนแปลง blood-brain barrier : การศึกษาในสัตว์ทดลองและในเซลล์เพาะเลี้ยง : รายงานวิจัยฉบับสมบูรณ์. จุฬาลงกรณ์มหาวิทยาลัย, 2012. https://doi.org/10.58837/chula.res.2012.21.

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ยาพาราเซตามอลเป็นยาที่ได้รับความนิยมใช้กันอย่างแพร่หลาย โดยใช้เป็นยาลดไข้และบรรเทาอาการปวด ผลการวิจัยที่ผ่านมาส่วนใหญ่ยืนยันว่ายาพาราเซตามอลมีฤทธิ์ในการปกป้องเซลล์เมื่อใช้ยาในขนาดเพื่อการรักษา แต่อย่างไรก็ตามในช่วง 10 ปีมานี้เริ่มมีรายงานถึงผลเสียของการใช้ยาชนิดนี้แม้ว่าจะใช้ยาในขนาดเพื่อการรักษา ในหลายระบบ รวมถึงระบบไหลเวียนเลือด และในผลงานวิจัยของคณะเราก่อนหน้านี้พบว่า หนูที่ได้รับยาพาราเซตามอลอย่างเรื้อรังร่วมกับการกระตุ้นให้เกิดปรากฎการณ์คอร์ติคัล สเปรดดิ้ง ดีเปรสชัน มีความไวของเซลล์ประสาทที่บริเวณผิวสมองต่อการกระตุ้นเพิ่มมากขึ้น รวมถึงมีการเปลี่ยนแปลงของปริมาณเลือดในหลอดเลือดสมองด้วย ซึ่ง

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