Gotowe bibliografie tematyczne / Autism spectrum disorder (A.S.D.)

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Gotowa bibliografia na temat „Autism spectrum disorder (A.S.D.)”

Autor: Grafiati
Data publikacji: 3 czerwca 2025
Data aktualizacji: 19 lipca 2025

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Artykuły w czasopismach na temat "Autism spectrum disorder (A.S.D.)"

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Tulumbacı, Fatih, Emre Korkut, and Hazal Özer. "Comparative Evaluation of Oral Health Status in Healthy Children and Children with Autism Spectrum Disorder." Journal of Pediatric Infectious Diseases 15, no. 05 (2020): 223–27. http://dx.doi.org/10.1055/s-0040-1709659.

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Abstract Objective Autism spectrum disorder (ASD) is regarded a severe neuropsychiatric disorder. The objective of this study was to compare the status of the oral health between children with and without ASDs. Methods A total of 95 children (44 with ASD and 51 without ASD) were included. dmft/DMFT (d/D: decay; m/M: missing; f/F: filling; t/T: teeth) scores and plaque data of all participants were recorded. Saliva Streptococcus mutans and lactobacilli load were determined using CRT bacteria. Results Streptococcus mutans load was >105 CFU in 54.5% of the ASD group and 43.1% in the non-ASD group. Lactobacilli load was >105 CFU in 47.70% of the ASD group and 49.0% in the non-ASD group. There was no statistically significant difference in the S. mutans or lactobacilli loads between the ASD and non-ASD groups. A total of 83.1% of the ASD group had caries, with dmft = 3.25 and DMFT = 5.10. In the non-ASD group, 81.10% of patients had caries, with dmft = 3.10 and DMFT = 4.90; 38.6% of the ASD group and 29.4% of the non-ASD group had no clinical signs of periodontal disease. Conclusion The examination of the amount of saliva S. mutans and lactobacilli at intervals with these patients will provide quantitative data to evaluate the effectiveness of oral health care.
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Trifonova, Ekaterina A., Alexandra I. Klimenko, Zakhar S. Mustafin, Sergey A. Lashin, and Alex V. Kochetov. "The mTOR Signaling Pathway Activity and Vitamin D Availability Control the Expression of Most Autism Predisposition Genes." International Journal of Molecular Sciences 20, no. 24 (2019): 6332. http://dx.doi.org/10.3390/ijms20246332.

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Autism spectrum disorder (ASD) has a strong and complex genetic component with an estimate of more than 1000 genes implicated cataloged in SFARI (Simon′s Foundation Autism Research Initiative) gene database. A significant part of both syndromic and idiopathic autism cases can be attributed to disorders caused by the mechanistic target of rapamycin (mTOR)-dependent translation deregulation. We conducted gene-set analyses and revealed that 606 out of 1053 genes (58%) included in the SFARI Gene database and 179 out of 281 genes (64%) included in the first three categories of the database (“high confidence”, “strong candidate”, and “suggestive evidence”) could be attributed to one of the four groups: 1. FMRP (fragile X mental retardation protein) target genes, 2. mTOR signaling network genes, 3. mTOR-modulated genes, 4. vitamin D3 sensitive genes. The additional gene network analysis revealed 43 new genes and 127 new interactions, so in the whole 222 out of 281 (79%) high scored genes from SFARI Gene database were connected with mTOR signaling activity and/or dependent on vitamin D3 availability directly or indirectly. We hypothesized that genetic and/or environment mTOR hyperactivation, including provocation by vitamin D deficiency, might be a common mechanism controlling the expressivity of most autism predisposition genes and even core symptoms of autism.
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Amy O’ Dell, Allegra Saunders, Julian Gonzalez, and Holly A. Haynes. "Neurobiological and Functional Outcomes of the Interpersonal Whole-Brain Model of Care in Autism Spectrum Disorder: A Mixed-Methods Study." Studies in Psychological Science 3, no. 1 (2025): 1–12. https://doi.org/10.56397/sps.2025.06.01.

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The Interpersonal Whole-Brain Model of Care (IWBMC™) presents a comprehensive intervention for autism spectrum disorder (ASD) targeting neurobiological frameworks. This mixed-methods study evaluated its efficacy through QEEG connectivity analysis of a longitudinal case study and pre-post outcomes from 61 ASD clients. Results demonstrated normalization of brain connectivity patterns alongside statistically significant improvements with large effect sizes across behavioral regulation (d=1.26), emotional recognition (d=0.98), cognitive functioning (d=0.84), and neurophysiological integration (d=1.52). The concurrent improvement in brain connectivity and functional outcomes suggests that the IWBMC™’s integrated approach effectively addresses both neurological underpinnings and behavioral manifestations of ASD, offering promising pathways for intervention.
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Maneeton, *Benchalak, Prapinpatch Maneeton, Yanisa Winichaikul, Suttipong Kawilapat, Nongluck Kienngam, and Narong Maneeton. "ANTIDEPRESSANTS FOR REPETITIVE RESTRICTED BEHAVIORS IN AUTISM SPECTRUM DISORDER: A SYSTEMATIC REVIEW AND META-ANALYSIS OF RANDOMIZED CONTROLLED TRIALS." International Journal of Neuropsychopharmacology 28, Supplement_1 (2025): i253—i254. https://doi.org/10.1093/ijnp/pyae059.446.

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Abstract Background Although previous randomized controlled trials (RCTs) illustrated the efficacy of antidepressants in repetitive restricted behaviors (RRBs) in autism spectrum disorder (ASD), the outcome determining the efficacy of antidepressants in RRBs in ASD remains inconsistent 1-4 Aims & Objectives We aimed to systematically review the efficacy, acceptability, and tolerability of antidepressants in the treatment of RRBs in patients with ASD. Method The authors searched relevant RCTs in the CINAHL, Cochrane Library, PubMed, and Scopus databases from inception to August 16, 2023. The primary outcomes of this review were efficacy, acceptability, and tolerability. The difference in the pooled mean endpoint scores of standardized rating scales for ASD RRBs was estimated to evaluate the efficacious outcomes. An inverse variance, weighing the effect of individual RCT was applied to estimate the pooled mean endpoint scores with 95% confidence intervals (CIs). The RRs with 95% CIs were thoroughly applied for discontinuation rates. All pooled RRs with 95% CIs were calculated using Mantel-Haenszel. Results We included 599 randomized participants from eight RCTs for the synthesis in this review. The overall pooled mean-end score of the RRBs between the antidepressant and placebo-treated groups was not significantly different [SMD (95% CI) of -0.27(-0.56, 0.02), I2=59%]. The pooled mean endpoint for RBB in ASD patients of the clomipramine-treated group was significantly less than desipramine-treated group [SMD (95% CI) of -1.11(-1.80, -0.41), I2=0%]. Considered individually, only clomipramine and fluvoxamine illustrated some efficacy over placebo. There were no significant differences in the overall discontinuation rates and discontinuation due to adverse events. Additionally, adverse events were not significantly different between the antidepressant and placebo-treated groups. Discussion & Conclusion The synthesis of results shows the various outcomes of antidepressants in the treatment of RRB symptoms. Clomipramine and fluvoxamine appear to have some efficacy in the reduction of RRBs in ASD but are not found in other antidepressants. Although antidepressants are well tolerated with minimal side effects, their acceptability is not better than placebo. Further well-defined and large sample-size studies could verify these outcomes. References 1.GORDON, C. T., NELSON, J. E., HAMBURGER, S. D. &RAPOPORT, J. L. 1993. A double-blind comparison of clomipramine, desipramine, and placebo in the treatment of autistic disorder. Archives of General Psychiatry, 50, 441-447. 2.HERSCU, P., HANDEN, B. L., ARNOLD, L. E., SNAPE, M. F., BREGMAN, J. D., GINSBERG, L., HENDREN, R., KOLEVZON, A., MELMED, R. &MINTZ, M. 2020. The SOFIA study: negative multi-center study of low dose fluoxetine on repetitive behaviors in children and adolescents with autistic disorder. Journal of autism and developmental disorders, 50, 3233-3244. 3.KING, B. H., HOLLANDER, E., SIKICH, L., MCCRACKEN, J. T., SCAHILL, L., BREGMAN, J. D., DONNELLY, C. L., ANAGNOSTOU, E., DUKES, K. &SULLIVAN, L. 2009. Lack of efficacy of citalopram in children with autism spectrum disorders and high levels of repetitive behavior: citalopram ineffective in children with autism. Archives of general psychiatry, 66, 583-590. 4.MCDOUGLE, C. J., NAYLOR, S. T., COHEN, D. J., VOLKMAR, F. R., HENINGER, G. R. &PRICE, L. H. 1996. A double-blind, placebo-controlled study of fluvoxamine in adults with autistic disorder. Archives of general psychiatry, 53, 1001-1008.
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Pastari, Marta, Sri Endriyani, Sri Martini, and Gina Khairaatun Hisaan. "Implementasi Keperawatan Terapi Hipnosis Lima Jari Pada Orang Tua Yang Memiliki Anak Autism Spectrum Disorder." JKM : Jurnal Keperawatan Merdeka 4, no. 1 (2024): 77–83. https://doi.org/10.36086/jkm.v4i1.2241.

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Background: Autism is a serious neurobiological developmental disorder that occurs in children, causing problems for children to communicate and relate to their environment. Most parents have various emotional reactions when they find out that their child has autism. So it has a negative impact on parents both physically and psychologically, including parents who experience feelings of worry or anxiety (Mulyadi & Sutadi, 2014). Method: The design of this case study is descriptive with a nursing care approach that focuses on nursing implementation and evaluation. The subjects used in this case study were four patients with the same case and nursing problem, namely anxiety. The research location in this case study was at Nu_Kids Integrated Therapy, carried out for seven days for each patient, varying according to the date of visit from March to April 2023. Data collection methods used were interviews, observation, physical examination, implementation and assessment scales. The results of data collection are presented in the form of tables and narratives. Results: Before the researcher implemented five finger hypnosis therapy on clients, the researcher needed to build a relationship of mutual trust (BHSP) with the four clients in order to gain the trust of the clients. After BHSP, researchers used the SRQ questionnaire to determine the level of anxiety experienced by clients. It was found that Mrs. D value is 7 (there is a psychological problem of anxiety). To Mrs. S value is 6 (there is a psychological problem of anxiety). To Mrs. N value is 7 (there is a psychological problem of anxiety). To Mrs. NA value is 6 (there is a psychological problem of anxiety). The researcher then carried out the implementation of five-finger hypnosis nursing therapy for 7 days, once a day for 10-15 minutes, then the client would repeat it at home independently. After 7 days of implementing five finger hypnosis, the researcher assessed the client's response; where is Mrs. D can now speak relaxedly, his face is no longer tense, Mrs. S can concentrate again, Mrs. NA really understands and can carry out five finger hypnosis therapy without being facilitated or assisted by researchers, Mrs. N can now calm down and relax, and is starting to get used to relaxing when doing anything. Conclusion: The results of this study indicate that after being given the implementation of Five Finger Hypnosis Therapy, the four clients (100%) no longer experienced anxiety disorders with a score of Mrs. D and Mrs. N = five, Mrs. NA and Mrs. S = 4 (Score < 6 means there is no psychological problem of anxiety). So it can be concluded that five finger hypnosis therapy is effective in reducing anxiety.
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Cheremnykh, E. G., A. N. Pozdnyakova, E. E. Balakireva, et al. "Complement System Marker for Assessing the Effectiveness of Therapy in Patients with Autism Spectrum Disorder and Childhood Schizophrenia." Psychiatry (Moscow) (Psikhiatriya) 22, no. 5 (2025): 38–48. https://doi.org/10.30629/2618-6667-2024-22-5-38-48.

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Background: assessment of the effectiveness of antipsychotic therapy is generally based on the knowledge, experience of the psychiatrist, and psychometric scales. However, all changes in a person’s mental state can be both a consequence and a cause of changes in biochemical processes, one of which is the complement system (CS). In addition to the fact that CS is the most important factor in innate immunity, its proteins, especially C3, are ubiquitous and directly involved in organismal and cellular homeostasis. CS includes its connection with coagulation, which compensates for the lack of activation potential of the cascade proteolytic complement system, as the first link of immunity. The deficiency is associated with excessive consumption of CS proteins in pathologically altered processes, which also determine mental pathologies. A change in the level of this connection allows one to determine changes in the overall level of homeostasis, for example, as a result of treatment. A new marker for assessing the level of coagulation-complement coupling, equal to the difference in the functional activity of CS in plasma and serum (d), may increase the reliability of assessing the effectiveness of therapy. Objective: justification for the use of this marker of the complement system in combination with mental state assessments on psychometric scales and electroencephalography results to analyze the effectiveness of therapy for patients with autism and schizophrenia aged 8–14 years. Patients and Methods: the study included 7 patients aged 8 to 14 years (10 ± 2.2) with a diagnosis “Childhood autism, F84.0 (ICD-10)” and 11 patients aged 11 to 14 years (13 ± 1.4) with a diagnosis “Schizophrenia, F20 (ICD-10)”. The assessment of the condition before and after treatment was carried out using the following scales: for patients with autism — CARS (Childhood Autism Rating Scale), BFCRS (Bush–Francis Catatonia Rating Scale), CGI-S (General Clinical Severity Scale); for patients with schizophrenia — PANSS (Positive and Negative Syndrome Scale) and CGI-S. A neurophysiological study of all patients was carried out with spectral analysis and topographic mapping using the NeuroKM computer system (Russia). A calculated parameter equal to the difference (d) of the functional activity of the complement system (faCS) in plasma and serum was used as a biological marker of CS. FaCS was assessed using the method on the ciliates Tetrahymena pyriformis and the Biolat device. Results: in 14 out of 18 patients, positive changes in mental state were observed, which corresponded to positive changes in marker d, as well as positive changes in ratings on scales and EEG results, in accordance with all assessments, 14 out of 18 patients from both groups showed positive changes in their condition, in 3 patients resistance to psychopharmacotherapy and minimal positive dynamics of the condition were noted; the marker of the complement system in these patients decreased slightly upon discharge; in one patient assessments of the biological marker and scales were contradictory. Conclusion: in most patients (14 out of 18 people), the selected therapy was effective, and the new candidate marker d of the complement system changed in the same direction (its normalization was noted) with clinical symptoms and neurophysiological parameters. The results of the study suggest that the candidate marker d reflects changes in the patients’ conditions as a result of treatment.
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Tei, *Shisei, and Junya Fujino. "THE NEURAL BASIS OF GAMING DISORDER AND THEIR COMORBIDITIES." International Journal of Neuropsychopharmacology 28, Supplement_1 (2025): i93. https://doi.org/10.1093/ijnp/pyae059.160.

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Abstract Background Given the growing popularity of online gaming and the rapid rise of societal and mental health crises, it is crucial to identify the neurocognitive features that delineate problematic gaming. Digital technologies are continually developing, and gaming has become much more social, immersive, and also monetized [1]. Furthermore, preoccupation with games frequently leads to behavioral addiction and aggravates physical and mental problems, similar to other psychiatric disorders. In response to this situation, the World Health Organization included gaming disorder (GD) in its latest revision of the International Classification of Diseases, 11th edition (ICD 11). However, the neuropsychological mechanisms underlying GD and addiction-related mindsets have not been sufficiently understood. Aims & Objectives To better understand GD, we aimed to explore the current research on GD by focusing primarily on neuropsychological features. We intended to investigate the brain imaging findings to illuminate the cognitive mechanisms underlying the symptoms of GD and its common features with other psychiatric disorders. In particular, we focused on two key features of GD: (1) EF dysfunction, and (2) altered game-related cue reactivity. Method In this preliminary review, we performed a literature search to identify the current state-of-the-art literature on GD, addiction, and other psychiatric comorbidities. We combined the results of recent findings to provide a more coherent explanation for the possible cognitive mechanisms of GD. We mainly searched the PubMed and Web of Science databases using relevant keywords. All retrieved studies were assessed for eligibility to reduce the selection bias. Results GD is frequently associated with general EF dysfunction and addiction-related reward processing [2, 3]. It may hinder controlled, planned, and conscious processing of daily behaviors while enhancing automatic processing that aggravates craving by habitual reactions to gaming cues. These abnormalities can be linked to structural changes and neuroadaptations in the brain, which may be due to persistent gaming and suppression of daily activities [2, 4]. These GD-related characteristics resemble those of other addictions. Moreover, it is possible that such shared features of addiction might also be observed in seemingly unrelated psychiatric disorders such as autism spectrum disorder (ASD), including inflexible self-control, atypical empathy, and reduced social skills [5,6]. Discussion & Conclusion The possible similarities in cognitive features between GD and other psychiatric conditions warrant more empirical investigation, as this conclusion is based on relatively few studies. Moreover, there were substantial variations among experimental participants, necessitating new research strategies. Future studies with both GD and ASD cohorts may provide novel theoretical and clinical insights. This may provide additional clues for more practical interventions. References [1]King, D. L., Wö lfling, K., &Potenza, M. N. (2020). Taking gaming disorder treatment to the next level. JAMA psychiatry, 77(8), 869-870. [2]Weinstein, A., Livny, A., &Weizman, A. (2017). New developments in brain research of internet and gaming disorder. Neuroscience &Biobehavioral Reviews, 75, pp. 314-330. [3]Wang, M., Zheng, H., Zhou, W., Yang, B., Wang, L., Chen, S., &Dong, G. H. (2022). Disrupted dynamic network reconfiguration of the executive and reward networks in internet gaming disorder. Psychological Medicine, pp.1-10. [4]Dong, G., Wang, L., Du, X., &Potenza, M. N. (2017). Gaming increases craving to gaming-related stimuli in individuals with Internet gaming disorder. Biological Psychiatry: Cognitive Neuroscience and Neuroimaging, 2(5), pp. 404-412. [5]Murray, A., Koronczai, B., Kirá ly, O., Griffiths, M. D., Mannion, A., Leader, G., &Demetrovics, Z. (2021). Autism, problematic internet use and gaming disorder: A systematic review. Review Journal of Autism and Developmental Disorders, pp.1- 21. [6]Fujino, J., Tei, S., Hashimoto, R. I., Itahashi, T., Ohta, H., Kanai, C.,... &Takahashi, H. (2017). Attitudes toward risk and ambiguity in patients with autism spectrum disorder. Molecular autism, 8, pp. 1-10.
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Ashraf, S., A. Bachu, S. Srinivas, W. Tankersley, and K. Shah. "Use of Methylphenidate to Improve Cognition in Autism Spectrum Disorder (ASD)." European Psychiatry 66, S1 (2023): S202. http://dx.doi.org/10.1192/j.eurpsy.2023.476.

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IntroductionMethylphenidate (MPH) is used to treat attention deficit hyperactivity disorder (ADHD) and has shown unique benefits in children and adolescents with autism spectrum disorder (ASD). In addition to improving attention, hyperactivity, and repetitive movement, it also improves cognition in ASD. Our focus is to explore the understudied benefits and safety of methylphenidate use for cognition in ASD children.Objectives1) To study the response and benefit of methylphenidate in autistic kids to improve cognition and life function.2) To assess the safety profile and risks of using methylphenidate at different doses in children with autism as a guiding tool for prescribing physicians.MethodsA literature search was conducted using relevant medical subject headings (MeSH) terms in PubMed, Medline, and PubMed Central. We identified all published articles from inception until September 30, 2022. Initial search results found 41 studies, of which 15 were excluded as they did not meet inclusion and exclusion criteria. After a thorough full-text review, we included 5 studies in our final qualitative synthesis review.ResultsMethylphenidate was found to have the unique benefit of increasing cognitive processing speed, thus improving everyday life function in ASD and ADHD children compared with only ADHD, with a clinical significance of p<0.001. Stimulants may improve the processing of social situations and interactions or social functions due to this unique response (Peled, J et al. Nordic journal of psychiatry 2020; 74(3), 163–167) A clinically significant performance gain on cognitive tasks was identified with a linear dose-response at three different doses (10 mg, 15 mg, 20 mg) of methylphenidate. ASD children with ADHD made significantly fewer omission errors, [F(3,69) = 7.21, p < 0.001], and commission errors [F(3,69) = 4.64, p = 0.005] on MPH, compared to placebo. They also showed more response at higher MPH doses, [F(3,69) = 10.45, p < 0.001]. Thus, the children were faster, more accurate, and had fewer errors at higher MPH doses (Pearson, D. A. et al. Journal of child and adolescent psychopharmacology 2020; 30(7), 414–426). There were no serious side effects or suicidality reported for low dose and a medium dose of MPH in ASD children assessed with Response Impressions and Side Effects Checklist-Kids (RISC-K), A 38-item parent rating scale (Kim, S. J. et al. Journal of autism and developmental disorders 2017; 47(8), 2307–2313).ConclusionsThe findings support the positive response of methylphenidate on cognitive function in ASD children. No serious adverse effects or suicidality were noted. Multi-center well-designed studies are recommended to determine further efficacy and safety of MPH in ASD children for cognition.Disclosure of InterestNone Declared
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Jacob, Marianne, and Marisa Censani. "13-Year-Old Boy Presenting with Bilateral Femur Fractures in the Setting of Severe Vitamin D Deficiency." Case Reports in Pediatrics 2021 (August 4, 2021): 1–4. http://dx.doi.org/10.1155/2021/2440999.

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Background. Femur fractures in adolescents are rare. Severe vitamin D deficiency has important implications for bone health. We describe the case of a 13-year-old boy with autism spectrum disorder (ASD) who presented with low-impact bilateral femur fractures in the setting of severe vitamin D deficiency. Case Presentation. A 13-year-old boy with ASD presented with bilateral leg pain after an unwitnessed fall. Laboratory investigations revealed severe hypocalcemia (S. calcium 4.9 mg/dL) and severe vitamin D deficiency (25(OH)D < 4 ng/mL). Lower extremity X-rays revealed bilateral distal femoral metaphyseal fractures. Conclusion. This is the youngest known case of bilateral femoral fractures in the setting of severe 25(OH)D deficiency. Children with ASD are especially at risk for 25(OH)D deficiency as many have inadequate nutritional intake. As such, primary care providers may provide a pivotal role in the routine laboratory screening of 25(OH)D in this population.
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Cole, Richard, Michael Elmalem, Esha Abrol, and Panayiota Petrochilos. "36 Study of the prevalence of autistic traits and alexithymia, with associated psychiatric comorbidity, in an outpatient program of patients with functional neurological symptom disorder (FNSD)." Journal of Neurology, Neurosurgery & Psychiatry 93, no. 12 (2022): e3.30. http://dx.doi.org/10.1136/jnnp-2022-bnpa.36.

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IntroductionWhilst higher rates of alexithymia have previously been reported in FNSD, little is known about the prevalence of autistic traits in adults with FNSD. We aim to:Report on the prevalence of autistic traits in an outpatient group of adults diagnosed with FNSD using the Autism Spectrum Quotient (AQ-10)Report on the prevalence of alexithymia using the Toronto Alexithymia Scale (TAS-20)Report on differences in symptom severity of psychiatric comorbidity between those scoring <6 or ≥6 on the AQ-10, and those with or without alexithymiaMethodOut of 105 consecutive patients reviewed in an outpatient FNSD program, 91 completed self-report assessments for autistic traits, alexithymia, generalised anxiety, depression, social phobia, somatic symptom severity, attention deficit hyperactivity disorder (ADHD) and dyslexia. Patients were grouped by AQ-10 scores of <6 or ≥6 and compared for significant differences in tested variables using a Mann-Whitney U test. Kruskal-Wallis H tested differences in alexithymia status. Simple effects were tested using pairwise comparisons.Results40% screened positive on the AQ-10 (scoring ≥6), and 40% screen positive for alexithymia. When comparing those scoring < or ≥6 on the AQ-10, those with the higher number of autistic traits scored significantly higher on scales of alexithymia, depression, generalised anxiety, social phobia,ADHD, and dyslexia. Positive alexithymia status was significantly associated with a higher number of autistic traits as well symptoms of generalised anxiety, depression, somatic symptoms severity, social phobia and dyslexia.ConclusionWhilst higher rates of neurodevelopmental disorders have previously been reported in FNSD, we report new evidence for a high proportion of autistic traits and further evidence of a high prevalence of alexithymia in a group of adults with FNSD.1–10Mechanistic insights are limited however autistic traits may be associated with FNSD due to altered sensitivity to sensory data, as well as cognitive or affective biases, or increased susceptibility to panic. There may be an additional contribution from psychosocial stressors. Clinically, the AQ-10 and TAS-20 may be important tools in the management of FNSD, and a higher prevalence of autistic traits may highlight a need for specialised communication styles in the MDT.11This builds on research exploring the relationship between autistic traits, alexithymia and FNSD. Previous research suggests that alexithymia and altered interoceptive awareness may be modifying factors in the relationship between autistic traits and FNSD,12and further research is required to clarify the nature of these relationships.ReferencesDemartini B, Nisticò V, Goeta D, Tedesco R, Giordano B, Faggioli R,et al. Clinical overlap between functional neurological disorders and autism spectrum disorders: A preliminary study.Journal of the Neurological Sciences2021 Oct;429:117648.Freedmanet al. Psychogenic nonepileptic events in pediatric patients with autism.Hatta K, Hosozawa M, Tanaka K, Shimizu T. Exploring traits of autism and their impact on functional disability in children with somatic symptom disorder.Journal of Autism and Developmental Disorders2019 Feb 15;49(2):729–37.Jester KA, Londino DL, Hayman J. 2.68 examining the occurrence of conversion disorder diagnoses and asd among adolescents and young adults in the emergency department.Journal of the American Academy of Child & Adolescent Psychiatry [Internet]. 2019 Oct 1 [cited 2022 Feb 7];58(10):S193. Available from: http://www.jaacap.org/article/S089085671931617X/fulltextMcWilliams A, Reilly C, Gupta J, Hadji-Michael M, Srinivasan R, Heyman I. Autism spectrum disorder in children and young people with non-epileptic seizures.Seizure2019 Dec 1;73:51–5.Mierschet al. A retrospective study of 131 patients with psychogenic non-epileptic seizures (PNES)- Comorbid diagnoses and outcome after inpatient treatment.Nimmo-Smith V, Heuvelman H, Dalman C, Lundberg M, Idring S, Carpenter P,et’al. Anxiety disorders in adults with autism spectrum disorder: a population-based study.Journal of Autism and Developmental Disorders. 2020 Jan 1;50(1):308–18.Pun P, Frater J, Broughton M, Dob R, Lehn A. Psychological profiles and clinical clusters of patients diagnosed with functional neurological disorder.Frontiers in Neurology2020 Oct 15;11.Zdankiewicz-Scigała E, Scigała D, Sikora J, Kwaterniak W, Longobardi C. Relationship between interoceptive sensibility and somatoform disorders in adults with autism spectrum traits. The mediating role of alexithymia and emotional dysregulation.PLoS ONE. 2021 Aug 1;16(8 August).Demartini B, Petrochilos P, Ricciardi L, Price G, Edwards MJ, Joyce E. The role of alexithymia in the development of functional motor symptoms (conversion disorder).Journal of Neurology, Neurosurgery & Psychiatry [Internet]. 2014 Oct 1 [cited 2022 Feb 8];85(10):1132–7. Available from: https://jnnp.bmj.com/content/85/10/1132Cooper M, Gale K, Langley K, Broughton T, Massey TH, Hall NJ,et al. Neurological consultation with an autistic patient.Practical Neurology [Internet]. 2021 Oct 8 [cited 2022 Jan 14];practneurol-2020-002856. Available from: https://pn.bmj.com/content/early/2021/10/07/practneurol-2020-002856Shah P, Hall R, Catmur C, Bird G. Alexithymia, not autism, is associated with impaired interoception.Cortex2016 Aug 1;81:215–20.
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Rozprawy doktorskie na temat "Autism spectrum disorder (A.S.D.)"

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Liemersdorf, Christian [Verfasser]. "Neuronal Systems Potentially Involved in Autism Spectrum Disorder / Christian Liemersdorf." Bonn : Universitäts- und Landesbibliothek Bonn, 2017. http://d-nb.info/1160594627/34.

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BRAZ, PRISCILLA FONSECA DE ABREU. "AN ANALYSIS OF THE END USER DEVELOPMENT S PROBLEM SPACE IN THE DOMAIN OF TECHNOLOGIES FOR AUTISM SPECTRUM DISORDER S THERAPISTS." PONTIFÍCIA UNIVERSIDADE CATÓLICA DO RIO DE JANEIRO, 2017. http://www.maxwell.vrac.puc-rio.br/Busca_etds.php?strSecao=resultado&nrSeq=31001@1.

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PONTIFÍCIA UNIVERSIDADE CATÓLICA DO RIO DE JANEIRO<br>COORDENAÇÃO DE APERFEIÇOAMENTO DO PESSOAL DE ENSINO SUPERIOR<br>PROGRAMA DE EXCELENCIA ACADEMICA<br>Esta tese apresenta um estudo sobre o espaço de problema no contexto do desenvolvimento de tecnologias para terapeutas do Transtorno do Espectro do Autismo (TEA) e visa contribuir para o design de tecnologias nessa área. Apesar de haver uma grande quantidade de tecnologias para o público com TEA, tais tecnologias possibilitam em geral um uso restrito, tendo em vista a amplitude do chamado Espectro do Autismo e a variedade de Transtornos que as pessoas com um diagnóstico de TEA podem manifestar. Desse modo, torna-se essencial o desenvolvimento de tecnologias mais flexíveis que atendam às necessidades de cada indivíduo com TEA. Além disso, a literatura apresenta relatos pontuais (de sucesso ou insucesso) sobre as tecnologias desenvolvidas, mas não há uma estruturação maior do domínio. Diante disso, consideramos que há uma lacuna com relação à caracterização do problema nessa área, sendo importante explorar o espaço de problema antes de propor novas soluções. Diversos são os desafios envolvidos na adoção de tecnologias para geração de software adaptável por terapeutas de TEA, tais como a falta ou o pouco conhecimento dos terapeutas sobre esse tipo de tecnologia, o tempo escasso para o aprendizado e uso dessas tecnologias e até mesmo a falta de interesse nelas. Realizamos estudos para explorar o espaço de problema no contexto de End User Development (EUD) e levantar os principais significados, questionamentos e dificuldades dos terapeutas relacionados às tecnologias adaptáveis e/ou extensíveis e eles nos mostraram que o uso do conceito de sondas de design foi essencial para a aproximação dos terapeutas com esse tipo de tecnologia e para um aprofundamento, por parte do designer, das dificuldades e necessidades deles e os desafios inseridos nesse contexto. Além disso, esses estudos nos permitiram levantar um conjunto de possíveis alterações e adaptações que uma tecnologia voltada para esse público poderia fazer de modo a atender as necessidades dos terapeutas com seus atendidos. A partir desse conjunto levantado, analisamos essas possíveis alterações com base na teoria da Engenharia Semiótica para o contexto de EUD e investigamos como tecnologias existentes possibilitam ou não fazer esses tipos de alterações. Desse modo, essa pesquisa nos permitiu identificar o domínio de TEA como uma área de aplicação de EUD com diversos desafios, analisá-los com maior profundidade e apontar possíveis caminhos para superá-los, contribuindo para o Design de tecnologias para o público de TEA.<br>This thesis presents a study on the problem space in the context of development of technologies for Autism Spectrum Disorder s (ASD) therapists and it aims to contribute to the design of technologies in this area. Although there is a large number of technologies to the audience with ASD, such technologies usually allow a limited use, given the extent of the Autism Spectrum and the variety of disorders that people with ASD diagnosis can have. Thus, it is essential to develop more flexible technologies that meet the needs of each individual with ASD. Moreover, the literature presents specific reports (of success or failure) about the developed technologies, but there is no organization of the domain. Therefore, we consider that there is a gap regarding the problem characterization in this area, and it is important to explore the problem space before proposing new solutions. There are several challenges related to the adoption of technologies for generating adaptable software by ASD therapists, such as the lack or little knowledge of the therapists about this kind of technology, the limited time for learning and using these technologies and even the lack of interest. We conducted studies in order to explore the problem space in the context of the End User Development (EUD) and to raise the main meanings, questions and difficulties of the therapists related to adaptable and/or extensible technologies and they showed us that the use of the design probes concept was essential for the approximation of the therapists with such technology type and a deepening understanding of their own difficulties and needs, and the challenges placed in this context. Moreover, these studies have allowed us to raise a set of possible changes and adaptations that a technology targeted for this audience could do in order to address the needs of therapists and their patients. From this achieved set, we have analyzed these possible changes based on the Semiotic Engineering theory for EUD context and we have investigated how existing technologies enable or not these kinds of changes. Thus, this research has enabled us to identify the ASD domain as an EUD application area with several challenges, to analyze them in greater depth and to identify possible ways to overcome them, contributing to the design of technologies for the ASD audience.
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Paye, Anna-Lena [Verfasser], and Inge [Akademischer Betreuer] Kamp-Becker. "The Female Variation of Autism Spectrum Disorder - An fMRI Study / Anna-Lena Paye ; Betreuer: Inge Kamp-Becker." Marburg : Philipps-Universität Marburg, 2017. http://d-nb.info/1130323064/34.

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Borowiak, Kamila [Verfasser]. "Brain Mechanisms for the Perception of Visual and Auditory Communication Signals – Insights from Autism Spectrum Disorder / Kamila Borowiak." Berlin : Humboldt-Universität zu Berlin, 2020. http://d-nb.info/1216103720/34.

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Eltokhi, Ahmed [Verfasser], and Gudrun A. [Akademischer Betreuer] Rappold. "Dissecting hormonal and transient effects in Shankopathies associated with autism spectrum disorder / Ahmed Eltokhi ; Betreuer: Gudrun A. Rappold." Heidelberg : Universitätsbibliothek Heidelberg, 2020. http://d-nb.info/1219303089/34.

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Schelinski, Stefanie [Verfasser], Katharina von [Gutachter] Kriegstein, Stefan [Gutachter] Schweinberger, and Isabel [Gutachter] Dziobek. "Mechanisms of Voice Processing: Evidence from Autism Spectrum Disorder / Stefanie Schelinski ; Gutachter: Katharina von Kriegstein, Stefan Schweinberger, Isabel Dziobek." Berlin : Humboldt-Universität zu Berlin, 2018. http://d-nb.info/1185497439/34.

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Kliemann, Dorit [Verfasser]. "Implicit and explicit facial emotion recognition in autism spectrum disorder : Insights from behavior, gaze and functional magnetic resonance imaging / Dorit Kliemann." Berlin : Freie Universität Berlin, 2013. http://d-nb.info/1031667075/34.

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Sungur, Ayse Özge [Verfasser], and Markus [Akademischer Betreuer] Wöhr. "Disentangling the Role of SHANK1 in a Mouse Model for Autism Spectrum Disorder: From Brain to Behavior / Ayse Özge Sungur ; Betreuer: Markus Wöhr." Marburg : Philipps-Universität Marburg, 2017. http://d-nb.info/1141904489/34.

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Kempfer, Rieke [Verfasser]. "Chromatin folding in health and disease: exploring allele-specific topologies and the reorganization due to the 16p11.2 deletion in autism-spectrum disorder. / Rieke Kempfer." Berlin : Humboldt-Universität zu Berlin, 2020. http://d-nb.info/1221128973/34.

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Yousaf, Afsheen [Verfasser], Ina [Gutachter] Koch, and Christine M. [Gutachter] Freitag. "Integrative bioinformatics pipeline for genome-wide association studies in neuropsychiatry and the subsequent application in Autism Spectrum Disorder cohorts / Afsheen Yousaf ; Gutachter: Ina Koch, Christine M. Freitag." Frankfurt am Main : Universitätsbibliothek Johann Christian Senckenberg, 2020. http://d-nb.info/1210555689/34.

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Książki na temat "Autism spectrum disorder (A.S.D.)"

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Chris, Barson, ed. The autism spectrum in the 21st century: Exploring psychology, biology, and practice. Jessica Kingsley Publishers, 2010.

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Autism Spectrum Disorder. SAGE Publications Ltd, 2017.

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Bross, Leslie, Kaye L. Otten, and Sonja R. de Boer. Educator′s Guide to Autism Spectrum Disorder: Interventions and Treatments. Corwin Press, 2023.

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Cannell, John Jacob. Autism causes, prevention and treatment: Vitamin D deficiency and the explosive rise of autism spectrum disorder. 2015.

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Autism Spectrum Disorders: Characteristics, Causes and Practical Issues. SAGE Publications, Limited, 2017.

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Ogando, Inna, and Valeria Savrasova. At David´s Birthday Party: Book with Pictograms. Autism Spectrum Disorder Recommended. Social Skill Story. Independently Published, 2022.

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Ogando, Inna, and Valeria Savrasova. Pat´s Day at School: Social Skill Story. Book with Pictograms. Autism Spectrum Disorder Recommended. Children´s Book with Visual Aid. Independently Published, 2022.

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Lipsky, Deborah, and Hope Richards. Managing Meltdowns: Using the S. C. A. R. E. D. Calming Technique with Children and Adults with Autism. Kingsley Publishers, Jessica, 2009.

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Weisman de Mamani, Amy, Merranda McLaughlin, Olivia Altamirano, Daisy Lopez, and Salman Shaheen Ahmad. Culturally Informed Therapy for Schizophrenia. Oxford University Press, 2020. http://dx.doi.org/10.1093/med-psych/9780197500644.001.0001.

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This book is primarily designed for clinicians and researchers interested in learning how to conduct an empirically supported culturally informed therapy for schizophrenia (CIT-S) that integrates core components of evidenced-based family therapy. It is estimated that approximately 1% of adults in the United States will be diagnosed with schizophrenia or a related schizophrenia spectrum disorder. Without treatment, prognosis is generally poor. Fortunately, traditional family therapies have shown increasing promise in reducing relapse rates and improving mental health for this population. As more and more societies become multicultural, however, there is an increasing expectation that mental health providers will also be prepared to meet the needs of unique and culturally diverse clients in an efficient, skillful, and culturally relevant manner. CIT-S is a 15-week, family-focused, cognitive behavioral approach for managing schizophrenia spectrum disorders. The intervention draws upon clients’ cultural beliefs, practices, and traditions to help them conceptualize and manage mental illness. It aims to improve the quality of clients’ lives in a manner that is in line with their values and takes into account their cultural norms when discussing important issues and addressing challenges (such as mental illness) within the family. CIT-S contains five distinct modules: (a) family collectivism, (b) psychoeducation, (c) spirituality, (d) communication training, and (e) problem-solving. For each module, a detailed rationale, background information, therapy instructions, suggested homework assignments, and a sample case vignette are provided in an accessible, easy-to-use manner.
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Części książek na temat "Autism spectrum disorder (A.S.D.)"

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Lejuste, Florian, Ryad Tamouza, and Marion Leboyer. "Is Autism Spectrum Disorder Related to Immune Dysfunction(s)?" In Immuno-Psychiatry. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-71229-7_13.

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Holley, Meredith I., Sheri Ravenscroft, Briana Brukilacchio, Morgan Engelmann, and Autumn Fikki. "Dell Children’s (S)TAAR Model of Early Autism Assessment." In Interprofessional Care Coordination for Pediatric Autism Spectrum Disorder. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-46295-6_6.

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Kwon, Eun Young, Caroline Guardino, and Joanna E. Cannon. "d/Deaf and Hard of Hearing Multilingual Learners With Disabilities: A Case Study of a Learner Who Is Deaf With Autism Spectrum Disorder and From an Immigrant Family." In Deaf and Hard of Hearing Multilingual Learners. Routledge, 2022. http://dx.doi.org/10.4324/9781003259176-7.

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Myers, Scott M., and Thomas D. Challman. "Autism spectrum disorder." In Capute and Accardo' s Neurodevelopmental Disabilities in Infancy and Childhood. Elsevier, 2025. http://dx.doi.org/10.1016/b978-0-12-824060-1.00038-9.

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Vegelin, Maud, Gosia Teodorowicz, and Huub F.J. Savelkoul. "Vitamin D and Autism Spectrum Disorder." In Vitamin D. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.96928.

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1,25(OH)2D is the hormonally active form of vitamin D known for its pleiotropic immunomodulatory effects. Via altering gene transcription, 1,25(OH)D exerts immunosuppressive effects and stimulates immune regulation. Recently, the interest in vitamin D in association with autism spectrum disorder (ASD) has been triggered. The prevalence of ASD has increased excessively over the last few decades, emphasizing the need for a better understanding of the etiology of the disorder as well as to find better treatments. Vitamin D levels in ASD patients are observed to be lower compared to healthy individuals and maternal vitamin D deficiency has been associated with an increased risk of ASD. Moreover, vitamin D supplementation improves ASD symptoms. These recent clinical findings strongly suggest that vitamin D is a factor in ASD onset and progression. Yet, possible mechanisms behind this association remain unknown. This review summarizes immunomodulatory properties of vitamin D and peripheral immune dysregulation in ASD, after which possible mechanisms via which vitamin D could rebalance the immune system in ASD are discussed. Although promising clinical results have been found, further research is necessary to draw conclusions about the effect and mechanisms behind the effect of vitamin D on ASD development.
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Del Giudice, Marco. "The Autism Spectrum." In Evolutionary Psychopathology, edited by Marco Del Giudice. Oxford University Press, 2018. http://dx.doi.org/10.1093/med-psych/9780190246846.003.0010.

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The chapter discusses autism spectrum disorder (ASD). Autism is defined by a triad of symptoms: impairments in social interaction, impairments in communication, and restricted/repetitive behaviors and interests. After an overview of this disorder, its developmental features, and the main risk factors identified in the epidemiological literature, the chapter critically reviews existing evolutionary models and suggests new directions for research. The final section applies the criteria developed earlier in the book to classify the disorder within the fast-slow-defense (FSD) model and identify functionally distinct subtypes. The author proposes to distinguish between a slow spectrum subtype with normal or high IQ and a major role of common alleles (S-ASD) and a subtype unrelated to life history variation, with high rates of intellectual disability and a major role of rare and de novo mutations (O-ASD).
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"D. Medication Flowchart." In Autism Spectrum Disorder: What Every Parent Needs to Know, 2nd ed. American Academy of Pediatrics, 2019. http://dx.doi.org/10.1542/9781610022705-appendix_d.

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Volkmar, Fred R., Marc Woodbury-Smith, Suzanne L. Macari, and Roald A. Øien. "Diagnostic Issues and Complexities in Autism and Related Conditions." In Differential Diagnosis of Autism Spectrum Disorder. Oxford University PressNew York, 2022. http://dx.doi.org/10.1093/med-psych/9780197516881.003.0001.

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Abstract Since the first recognition of autism and Asperger’s disorder, there have been important debates about the boundaries of these conditions and the degree to which they ‘shade off’ into normalcy. These debates have centered on the relevance of a narrow versus broad view of the clinical disorder(s) as well as on the implications of its complex genetic underpinnings and increased knowledge of the social neuroscience of autism and related conditions. With the recognition of the complexity of the genetics of autism there has been much debate regarding the broader autism phenotype and the potential adaptive function of some aspects of the condition. In addition, concerns with boundary issues in relation to cognitive and communicative ability, and the relevance of gender, diverse backgrounds, and cultural issues have all arisen. The current DSM-5 definition was developed using research instruments and has proven overly stringent in important respects. Clarification of these issues will enhance both clinical work and research.
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El-Ansary, Afaf, Khaled Saad, Ramesa Shafi Bhat, Amina Al-Haidan, and Laila Al-Ayadhi. "Role of vitamin D in modulating gut microbiota, and certain etiological mechanisms of autism spectrum disorder." In Understanding Autism. Elsevier, 2025. http://dx.doi.org/10.1016/b978-0-443-27366-7.00017-1.

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Xavier Santos, João, Célia Rasga, and Astrid Moura Vicente. "Exposure to Xenobiotics and Gene-Environment Interactions in Autism Spectrum Disorder: A Systematic Review." In Autism Spectrum Disorder - Profile, Heterogeneity, Neurobiology and Intervention. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.95758.

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Heritability estimates indicate that genetic susceptibility does not fully explain Autism Spectrum Disorder (ASD) risk variance, and that environmental factors may play a role in this disease. To explore the impact of the environment in ASD etiology, we performed a systematic review of the literature on xenobiotics implicated in the disease, and their interactions with gene variants. We compiled 72 studies reporting associations between ASD and xenobiotic exposure, including air pollutants, persistent and non-persistent organic pollutants, heavy metals, pesticides, pharmaceutical drugs and nutrients. Additionally, 9 studies reported that interactions between some of these chemicals (eg. NO2, particulate matter, manganese, folic acid and vitamin D) and genetic risk factors (eg. variants in the CYP2R1, GSTM1, GSTP1, MET, MTHFR and VDR genes) modulate ASD risk. The chemicals highlighted in this review induce neuropathological mechanisms previously implicated in ASD, including oxidative stress and hypoxia, dysregulation of signaling pathways and endocrine disruption. Exposure to xenobiotics may be harmful during critical windows of neurodevelopment, particularly for individuals with variants in genes involved in xenobiotic metabolization or in widespread signaling pathways. We emphasize the importance of leveraging multilevel data collections and integrative approaches grounded on artificial intelligence to address gene–environment interactions and understand ASD etiology, towards prevention and treatment strategies.
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Streszczenia konferencji na temat "Autism spectrum disorder (A.S.D.)"

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Runge, K., L. Tebartz van Elst, and D. Endres. "Cerebrospinal fluid findings in adult patients with autism spectrum disorder." In Abstracts of the 1st Symposium of the Arbeitsgemeinschaft für Neuropsychopharmakologie und Pharmakopsychiatrie (AGNP) and Deutsche Gesellschaft für Biologische Psychiatrie (DGBP). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1679145.

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Rodrigues, Yan Victor Araújo, and Renata Orlandi Rubim. "Perception of family physicians regarding identification of autism spectrum disorder." In SBN Conference 2022. Thieme Revinter Publicações Ltda., 2023. http://dx.doi.org/10.1055/s-0043-1774488.

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Alonzo, Paola, and Rosalba Roccolini. "AUTISM SPECTRUM DISORDER: FROM THEORY TO PRACTICE AND CREATIVITY." In 9th SWS International Scientific Conferences on SOCIAL SCIENCES - ISCSS 2022. SGEM WORLD SCIENCE, 2022. http://dx.doi.org/10.35603/sws.iscss.2022/s06.055.

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Definition, theories, characteristics, specifiers, learning and support methods for autism are the elements on which we have focused in this paper. The aim of the work is to compose a synthesis of the main theories on the subject, giving greater priority to the qualitative method. Thanks to the theoretical reference and the knowledge acquired through the studies selected and conducted so far, and in the certainty of positive answers, we intend to draw suggestions, guide and promote interventions and aids for learning in subjects with autism spectrum disorder. This type of disorder includes a "persistent deficit of social communication and social interaction in multiple contexts ..." (DSM-5, 2014, p.57)[1]. Among the theories reported, those of L. Kanner, H. Asperger, R.P. Hobson, U. Frith, G. Rizzolatti and S. Baron-Cohen. Specialists arrive at the diagnosis of autism spectrum disorder thanks to specific characteristics that are indicated, through codes, as specifiers in the ICF. From these it is possible to deduce and identify the best approach methods for each individual helping person. The interventions that are compared here are essentially related to music therapy, the ABA method (Applied Behavioral Analysis), the ToM method (Theory of Mind), the FTP method (Floor Time Play) and the enactive mind. Examples, these, more fitting with the purpose and the reference method.
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Mehra, Rohan. "The Impact of Vitamin Supplementation (D, B12, B9) on Behaviors Associated with Autism Spectrum Disorder." In 28th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2024. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.187_2024.

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Background: One in 36 children in the United States are diagnosed with Autism Spectrum Disorder (ASD). Although heritability of the condition ranges from 40 to 80%, other factors such as vitamin levels, may have a significant impact on the risk of development. These vitamins include D, B12, and B9. Purpose: To assess the impact vitamin supplementation has on behaviors associated with ASD, and to determine which specific aspects of ASD may be improved with vitamin supplementation. Methods: A literature review was performed. The search was utilized PubMed, JSTOR and Web of Science. Keyword strings included: “Vitamin D B12 B9 folate cobalamin supplementation brain autism spectrum disorder neurological development”, “Vitamin D B12 B9 folate cobalamin supplementation brain autism spectrum disorder neurological development”, and “Vitamin D B12 B9 folate cobalamin supplementation brain autism spectrum disorder neurological development” Results: Supplementation with Vitamin D was associated with decreases in both irritability and hyperactivity. Vitamin B12 Supplementation showed improvements in several behaviors including hyperactivity, tantrumming and receptive language. Vitamin B9 Supplementation was associated with an improvement in verbal communication and may be indicated as an effective adjunct therapy to structured teaching programs. Conclusions: Although each form of supplementation was associated with positive behavior changes, the research included contradicting outcomes and further research is warranted to strengthen existing conclusions.
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Heidelberg, Friederike, Deyana Valcheva, Thorsten Rosenbaum, and Angela Abicht. "Autism Spectrum Disorder as First Symptom of Neuronal Ceroid Lipofuscinosis Type 7 (NCL7)." In Abstracts of the 45th Annual Meeting of the Society for Neuropediatrics. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1698239.

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Gikovate, Carla Gruber, and Clara Gruber Telles. "Families with children in the autism spectrum disorder: tracing difficulties and support strategies." In SBN Conference 2022. Thieme Revinter Publicações Ltda., 2023. http://dx.doi.org/10.1055/s-0043-1774487.

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de Vries, Heike, Dietlind Klaus, Katrin Leister, Ulrich Brandl, and Peter Huppke. "Panic Attacks and Anxiety Disorder in a Child with Autism Spectrum Disorder and ADHD Triggered by GAD65 Autoimmune Encephalitis." In Abstracts of the 46th Annual Meeting of the Society for Neuropediatrics. Georg Thieme Verlag KG, 2021. http://dx.doi.org/10.1055/s-0041-1739596.

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Lima, Vinicius Alves, Louise Scridelli Tavares, Felipe Arthur Almeida Jorge, et al. "Unilateral retinoblastoma, autism spectrum disorder and macrocrania in 13q deletion syndrome: a case report." In SBN Conference 2022. Thieme Revinter Publicações Ltda., 2023. http://dx.doi.org/10.1055/s-0043-1774608.

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Koh, Magdalena Yvonne, and Aishworiya Ramkumar. "334 Iron and vitamin D deficiency in young children with autism spectrum disorder in singapore." In RCPCH Conference Singapore. BMJ Publishing Group Ltd, 2021. http://dx.doi.org/10.1136/bmjpo-2021-rcpch.184.

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Hanaoka, S. "The Treatment by Small Dose of Valproic Acid for Auditory Hypersensitivity in Autism Spectrum Disorder." In Abstracts of the 47th Annual Meeting of the SENP (Société Européenne De Neurologie Pédiatrique). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1685436.

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