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Artykuły w czasopismach na temat „Autoimmune process”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Barker, Roger A., and Anthony P. Cahn. "Parkinson's Disease: An Autoimmune Process." International Journal of Neuroscience 43, no. 1-2 (1988): 1–7. http://dx.doi.org/10.3109/00207458808985773.

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Kozlov, V. A. "Tumorigenesis as an autoimmune process." Medical Immunology (Russia) 27, no. 4 (2025): 703–12. https://doi.org/10.15789/1563-0625-taa-3152.

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The data presented in the article reasonably indicate that the immune system responses during the development of autoimmune pathology and in the process of tumor growth are, in fact, autoimmune responses directed at autoantigens of various tissues and organs, and tumor-associated antigens. The latter, in addition to the tumor itself, are also expressed by the cells of normal organs and tissues. At the same time, the differences are characterized by insufficient activity of suppressor cells in the first variant, and enhanced activity in the second variant. One should take into account a potenti

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3

Герасун, Б. А., Р. Ю. Грицко, О. Б. Герасун, and Р. А. Копець. "New method for the treatment autoimmune process." Family Medicine, no. 2(64) (March 21, 2016): 47–51. http://dx.doi.org/10.30841/2307-5112.2(64).2016.101899.

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Fadeev, V. V., I. V. Shevchenko, and G. A. Melnichenko. "Autoimmune polyglandular syndromes." Problems of Endocrinology 45, no. 1 (2019): 47–54. http://dx.doi.org/10.14341/probl11708.

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Autoimmune polyglandular syndromes (APS) are the primary defeat of the autoimmune process of 2 peripheral endocrine glands and more, leading, as a rule, to their insufficiency, often combined with various organ-specific non-endocrine autoimmune diseases.

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Gopal, KVT, GRaghu Rama Rao, YHari Kishan Kumar, MV Appa Rao, P. Vasudev, and Srikant. "Vitiligo: A part of a systemic autoimmune process." Indian Journal of Dermatology, Venereology and Leprology 73, no. 3 (2007): 162. http://dx.doi.org/10.4103/0378-6323.32710.

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Munoz, A., R. Martinez, M. Leon, et al. "Study on autoimmune mechanisms in the neurological process." Annals of the Rheumatic Diseases 70, Suppl 2 (2011): A91. http://dx.doi.org/10.1136/ard.2010.149021.23.

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Galenok, F. A., and Ye A. Zhuk. "Autoimmune process markers in insulin-dependent diabetes mellitus." Problems of Endocrinology 43, no. 3 (2019): 13–16. http://dx.doi.org/10.14341/probl10387.

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Quantitative and functional parameters of the monocyte and В-cell immunity were assessed in patients with insulin-dependent diabetes over the course of disease. Studies of the monocyte component in subjects predisposed to disease and of the humoral one in those with clinical manifestations are valuable for predicting the autoimmune process. Signs predicting a poor outcome are increase of the count of Fc-positive cells, decrease of NBT reduction of monocytes in subjects without signs of the disease, high levels of immunoglobulin G in manifest disease, and increase of IgM and B-lymphopcyte count

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8

Chhabra, Seema, Biman Saikia, Yashwant Kumar, and RanjanaW Minz. "Anti-centriole antibody: An infectious or autoimmune process?" Indian Journal of Allergy, Asthma and Immunology 29, no. 2 (2015): 84. http://dx.doi.org/10.4103/0972-6691.178274.

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Stys, Peter K. "Multiple Sclerosis: Autoimmune Disease or Autoimmune Reaction?" Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 37, S2 (2010): S16—S23. http://dx.doi.org/10.1017/s0317167100022393.

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ABSTRACT:Multiple sclerosis (MS) is traditionally considered an autoimmune inflammatory demyelinating disease of the central nervous system (CNS) with much knowledge available to support this view. However, this characterization implies that the primary event is an aberrant immune response directed at CNS antigens, promoting inflammation and later driving progressive axo-glial degeneration. Trials with potent anti-inflammatory agents and detailed neuropathological studies raise questions about this sequence of events. This hypothetical paper argues that MS may be primarily a “cytodegenerative”

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10

Mayorova, M. A., N. N. Petrova, U. I. Stroev, L. P. Churilov, and Y. Shoenfeld. "Interrelation of auto-immune process, endocrine disorders and depression." V.M. BEKHTEREV REVIEW OF PSYCHIATRY AND MEDICAL PSYCHOLOGY, no. 1 (April 2, 2020): 8–19. http://dx.doi.org/10.31363/2313-7053-2020-1-8-19.

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Disturbances in immune and endocrine regulation are closely interrelated with development of psychiatric diseases, including affective disorders. Depression may be a characteristic of at least 23 autoimmune diseases. Negative influence on the depression development have: an increased level of proinflammatory cytokines, impairment of T-regulatory cells, kynurenine pathway shift towards production of its potentially neurotoxic metabolites, overproduction of stress hormones, hyperleptinemia, hyperprolactinemia, hypothyroidism, as well as vitamin D, calcidiol and calcitriol deficiency. In this rev

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11

Fedoseyeva, Eugenia V., Feng Zhang, Patricia L. Orr, David Levin, Harry J. Buncke, and Gilles Benichou. "De Novo Autoimmunity to Cardiac Myosin After Heart Transplantation and Its Contribution to the Rejection Process." Journal of Immunology 162, no. 11 (1999): 6836–42. http://dx.doi.org/10.4049/jimmunol.162.11.6836.

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Abstract Allograft rejection is initiated by an immune response to donor MHC proteins. We recently reported that this response can result in breakdown of immune tolerance to a recipient self Ag. However, the contribution of this autoimmune response to graft rejection has yet to be determined. Here, we found that after mouse allogeneic heart transplantation, de novo CD4+ T cell and B cell autoimmune response to cardiac myosin (CM), a major contractile protein of cardiac muscle, is elicited in recipients. Importantly, CM is the autoantigen that causes autoimmune myocarditis, a heart autoimmune d

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12

Blumberg, Richard S., Bonnie Dittel, David Hafler, Matthias von Herrath, and Frank O. Nestle. "Unraveling the autoimmune translational research process layer by layer." Nature Medicine 18, no. 1 (2012): 35–41. http://dx.doi.org/10.1038/nm.2632.

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Zeitlin, Abigail A., Matthew J. Simmonds, and Stephen C. L. Gough. "Genetic developments in autoimmune thyroid disease: an evolutionary process." Clinical Endocrinology 68, no. 5 (2008): 671–82. http://dx.doi.org/10.1111/j.1365-2265.2007.03075.x.

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14

Kozhich, A. T., Y. Kawano, C. E. Egwuagu, et al. "A pathogenic autoimmune process targeted at a surrogate epitope." Journal of Experimental Medicine 180, no. 1 (1994): 133–40. http://dx.doi.org/10.1084/jem.180.1.133.

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Immunization with the retinal interphotoreceptor retinoid-binding protein (IRBP) induces in a variety of animals an inflammatory eye disease, experimental autoimmune uveoretinitis (EAU). We have previously shown that sequence 1181-1191 of bovine IRBP (BOV 1181-1191) is immunodominant and highly uveitogenic and immunogenic in Lewis rats. Sequence 1181-1191 of the rat IRBP (RAT 1181-1191) differs from BOV 1181-1191 by two residues, at positions 1188 and 1190, that are pivotal for the immunological activity of the bovine epitope. Here we show that, unlike its bovine homologue, RAT 1181-1191 did n

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15

Hertz, Roy. "Hypothesis: Menstruation is a steroid-regulated, cyclic, autoimmune process." American Journal of Obstetrics and Gynecology 155, no. 2 (1986): 374–75. http://dx.doi.org/10.1016/0002-9378(86)90831-8.

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16

Kirsner, Robert S., and Vincent Falanga. "Features of an autoimmune process in mid-dermal elastolysis." Journal of the American Academy of Dermatology 27, no. 5 (1992): 832–34. http://dx.doi.org/10.1016/0190-9622(92)70259-i.

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Shidlovskyy, O. V., V. O. Shidlovskyy, M. I. Sheremet, et al. "Pathogenetic mechanisms, clinical signs and consequences of the autoimmune thyroiditis impact on body systems (a literature review)." INTERNATIONAL JOURNAL OF ENDOCRINOLOGY (Ukraine) 18, no. 1 (2022): 70–77. http://dx.doi.org/10.22141/2224-0721.18.1.2022.1147.

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Background. The review analyzes the publications on the pathogenetic mechanisms and clinical consequences of the impact of autoimmune thyroiditis and hypothyroidism on body systems. Autoimmune thyroiditis is an organ-specific autoimmune disease of the thyroid gland and the most common cause of hypothyroidism in regions of the world with sufficient iodine. Sources of information. The sources of information were reports in domestic and, mostly, foreign periodicals on immunology, pathological physiology, internal medicine, and endocrinology. Synthesis of evidence. The effect of autoimmune thyroi

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18

Plotnikova, E. Yu. "Autoimmune gastritis." Meditsinskiy sovet = Medical Council, no. 23 (January 19, 2024): 157–67. http://dx.doi.org/10.21518/ms2023-467.

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Diagnosis of autoimmune gastritis (AIG) is often challenging because of the absence of typical symptoms. Clinical guidelines are lacking, which results in inadequate treatment and poor cancer screening. This work presents an overview of current management options and aims at raising awareness for this disease. The paper also describes a clinical case of the full-scale course of AIG with all its complications. Autoimmune gastritis is mostly considered as a disease of elderly with vitamin B12 deficiency and pernicious anaemia. Today it is recognized that AIG is found with a similar prevalence in

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19

Linnoila, Jenny. "Imaging in Autoimmune Neurology." Seminars in Neurology 38, no. 03 (2018): 371–78. http://dx.doi.org/10.1055/s-0038-1660476.

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AbstractAutoimmune disorders are becoming increasingly recognized within the broader field of neurology. The discovery of multiple, novel, neutrally targeted autoantibodies over the past decade and their translation into commercially available testing, in particular, has aided in the more rapid diagnosis of these disorders. When considering imaging in autoimmune neurologic disorders, it is important, when possible, to visualize the autoimmune process itself, as well as to make sure that the patient does not have an associated malignancy driving the overall process. Positron emission tomographi

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20

Nelson, Heather A., Hemant R. Joshi, and Joely A. Straseski. "Mistaken Identity: The Role of Autoantibodies in Endocrine Disease." Journal of Applied Laboratory Medicine 7, no. 1 (2022): 206–20. http://dx.doi.org/10.1093/jalm/jfab128.

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Abstract Background Autoimmune endocrine diseases can be thought of as a case of mistaken identity. The immune system mistakenly attacks one’s own cells, as if they were foreign, which typically results in endocrine gland hypofunction and inadequate hormone production. Type 1 diabetes mellitus and autoimmune thyroid disorders (Hashimoto and Graves diseases) are the most common autoimmune endocrine disorders, while conditions such as Addison disease are encountered less frequently. Autoantibody production can precede clinical presentation, and their measurement may aid verification of an autoim

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21

Krzewska, Aleksandra, and Iwona Ben-Skowronek. "Effect of Associated Autoimmune Diseases on Type 1 Diabetes Mellitus Incidence and Metabolic Control in Children and Adolescents." BioMed Research International 2016 (2016): 1–12. http://dx.doi.org/10.1155/2016/6219730.

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Type 1 diabetes mellitus (T1DM) is one of the most common chronic diseases developing in childhood. The incidence of the disease in children increases for unknown reasons at a rate from 3 to 5% every year worldwide. The background of T1DM is associated with the autoimmune process of pancreatic beta cell destruction, which leads to absolute insulin deficiency and organ damage. Complex interactions between environmental and genetic factors contribute to the development of T1DM in genetically predisposed patients. The T1DM-inducing autoimmune process can also affect other organs, resulting in dev

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22

Kazakova, M. P., A. A. Tskaeva, E. A. Starostina, and E. A. Troshina. "Autoimmune thyroiditis — what is new?" Clinical and experimental thyroidology 19, no. 4 (2024): 4–12. http://dx.doi.org/10.14341/ket12781.

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Recently, the prevalence of autoimmune diseases has been steadily increasing among the population, posing a significant problem for healthcare. One of the most common autoimmune pathologies is autoimmune thyroiditis (AIT). Due to the absence of prognostic markers to predict outcomes of AIT, such as the development of hypothyroidism, there is an increasing necessity for molecular-genetic research into the autoimmune changes occurring within this disease. This research is likely to contribute to the development of new diagnostic methods and the identification of key aspects of pathogenesis, ther

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23

Rahman, MA, IK Datta, R. Bhuyian, et al. "Autoimmune Pancreatitis Presenting as Obstruction Jaundice." Journal of Bangladesh College of Physicians and Surgeons 29, no. 4 (2012): 231–34. http://dx.doi.org/10.3329/jbcps.v29i4.11344.

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Autoimmune pancreatitis is a type of chronic pancreatitis characterized by an autoimmune inflammatory process in which prominent lymphocyte infiltration with associated fibrosis of the pancreas causes organ dysfunction. Clinically it is very important to be aware of autoimmune pancreatitis because autoimmune chronic pancreatitis (AIP) can clinically disguise as pancreaticobiliary malignancies, ordinary chronic or acute pancreatitis. Here we report a case of autoimmune pancreatitis presented with obstructive jaundice with initial diagnosis of carcinoma of head of the pancreas. DOI: http://dx.do

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24

Mirrakhimov, Aibek E., and Erkin M. Mirrakhimov. "Obstructive Sleep Apnea and Autoimmune Disease: A Two-Way Process." Journal of Clinical Sleep Medicine 09, no. 04 (2013): 409. http://dx.doi.org/10.5664/jcsm.2604.

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Bar-Dayan, Y., S. V. Kaveri, M. D. Kazatchkine, and Y. Shoenfeld. "Is cancer an autoimmune process dependent on anti-apoptotic autoantibodies?" Medical Hypotheses 55, no. 2 (2000): 103–8. http://dx.doi.org/10.1054/mehy.1999.1035.

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Leonavičienė, Laima, Rūta Bradūnaitė, Audrius Vasiliauskas, Dalia Vaitkienė, and Algirdas Venalis. "Development of autoimmune process in rats immunized with influenza vaccine." Acta medica Lituanica 16, no. 1 (2009): 23–34. http://dx.doi.org/10.2478/v10140-009-0004-x.

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Gaharu, M., N. Afany, and A. Rahmatya. "Anti-NMDA receptor encephalitis; a psychiatric manifestation of autoimmune process." Journal of the Neurological Sciences 405 (October 2019): 15–16. http://dx.doi.org/10.1016/j.jns.2019.10.790.

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Furlan, Roberto, Gianvito Martino, Francesca Galbiati, et al. "Caspase-1 Regulates the Inflammatory Process Leading to Autoimmune Demyelination." Journal of Immunology 163, no. 5 (1999): 2403–9. http://dx.doi.org/10.4049/jimmunol.163.5.2403.

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Abstract T cell-mediated inflammation is considered to play a key role in the pathogenic mechanisms sustaining multiple sclerosis (MS). Caspase-1, formerly designated IL-1β-converting enzyme, is crucially involved in immune-mediated inflammation because of its pivotal role in regulating the cellular export of IL-1β and IL-18. We studied the role of caspase-1 in experimental autoimmune encephalomyelitis (EAE), the animal model for MS. Caspase-1 is transcriptionally induced during EAE, and its levels correlate with the clinical course and transcription rate of proinflammatory cytokines such as T

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Rose, NR. "Autoimmunity, infection and adjuvants." Lupus 19, no. 4 (2010): 354–58. http://dx.doi.org/10.1177/0961203309360670.

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The effect of infection in initiating autoimmune disease has been debated for many years. There are, even now, few instances of a human autoimmune disease clearly caused by prior infection, probably due to the frequent separation in time and space from the clinical outcomes. As our understanding of the immunologic consequences of the infectious process has deepened, we can re-think some of the issues by focusing attention on the varied adjuvant effects of microbial products. We are now able to distinguish some of the critical steps in progression from virus infection to benign autoimmunity to

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Sarfaraz, Sabahat, and Sabiha Anis. "Multiple Autoimmune Syndrome: An Unusual Combination of Autoimmune Disorders." Reviews on Recent Clinical Trials 15, no. 3 (2020): 240–43. http://dx.doi.org/10.2174/1574887115666200621184110.

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Background: Autoimmune diseases are multifactorial with environmental and heritable factors. Autoimmunity reflects an altered immune status, therefore the presence of more than one disorder is not uncommon. The coexistence of three or more autoimmune diseases in a patient constitutes multiple autoimmune syndrome (MAS). This is an interesting case of a middle-aged female who had celiac disease, primary biliary cholangitis, autoimmune hepatitis and evolving CREST (Calcinosis, Rhaynaud’s phenomenon, Esophageal dysmotility, Sclerodactyly and Telangiectasia) syndrome. Case Report: Fifty years old f

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Abdullaev, X.D, R.A Djalolova, D.M Abdullaev, X.D Abdullaev, and MM Axmedova. "EFFICACY OF ONYCHOMYCOSIS TREATMENT AND EXAMINING THE IMPACT OF PATHOLOGICAL PROCESS DURATION ON PREVALENCE." GOLDEN BRAIN 2, no. 6 (2024): 187–96. https://doi.org/10.5281/zenodo.10705409.

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<em>this article attempts to reveal the main reasons for the influence of the duration of autoimmune pathology on the effectiveness of the treatment of onychomycosis of the feet. To carry out scientific work, the author observed 150 patients in the rheumatology department of the Altai Regional Clinical Hospital aged 18 to 70 years with autoimmune pathology and onychomycosis of the feet. The control group included 115 patients with onychomycosis of the feet without concomitant somatic diseases, examined and treated in the consultative and diagnostic department. The problem in question is still

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Psillas, George, Grigorios George Dimas, Christos Savopoulos, and Jiannis Constantinidis. "Autoimmune Hearing Loss: A Diagnostic Challenge." Journal of Clinical Medicine 11, no. 15 (2022): 4601. http://dx.doi.org/10.3390/jcm11154601.

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Autoimmune hearing loss (AIHL) is a clinical disease and may involve the deposition of immune complexes in the labyrinth vessels, the activation of the complement system, the functional alteration in T-cell subpopulations, or an inflammation process in the inner ear [...]

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Kochieva, Marina Leonidovna. "A case of severe course of Sjögren's disease." Spravočnik vrača obŝej praktiki (Journal of Family Medicine), no. 8 (August 22, 2023): 49–58. http://dx.doi.org/10.33920/med-10-2308-06.

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Sjögren's disease is a systemic disease of unknown etiology, a characteristic feature of which is a chronic autoimmune and lymphoproliferative process in the secreting epithelial glands with the development of parenchymatous sialadenitis with xerostomia and dry keratoconjunctivitis with hypolacrimia. Sjögren's syndrome is a lesion of the salivary and lacrimal glands similar to Sjögren's disease, which develops in 5-25% of patients with systemic connective tissue diseases, more often with rheumatoid arthritis, and in 50-75% of patients with chronic autoimmune liver damage (chronic autoimmune he

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Al'banova, V. I., and M. A. Nefedova. "AUTOIMMUNE BULLOUS DERMATOSES. DIFFERENTIAL DIAGNOSIS." Vestnik dermatologii i venerologii 93, no. 3 (2017): 10–20. http://dx.doi.org/10.25208/0042-4609-2017-93-3-10-20.

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The review presents modern ideas concerning autoimmune bullous dermatoses (pemphigoid group): Duhring disease, bullous pemphigoid, cicatrizing pemphigus, pemphigoid gestationis, linear IgA bullous dermatosis and acquired bullous epidermolysis. Pathogenesis, clinical manifestations and modern methods of disease diagnostics are reported. Particular attention is paid to the diagnosis of acquired bullous epidermolysis, in which the leading role belongs to the reaction of immunofluorescence. The pathogenetic similarity between bullous pemphigoid, linear IgA dermatosis, cicatrizing pemphigoid and pe

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Dulcey S, Luis Andrés, Juan Sebastián Theran, and Raimondo Caltagirone. "Síndrome Poliglandular Autoinmune: Revisión de una condición clínica subestimada." Revista Avances en Salud 6, no. 1 (2023): 55–69. http://dx.doi.org/10.21897/25394622.3222.

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Objective: Literature review about the autoimmune polyglandular syndrome from the epidemiological and clinical-therapeutic point of view. Materials and Methods: A detailed scientific literature searching was done from the last 30 years including the terms Autoimmune Diseases, Endocrinology, Genetics, Genetic Counseling, autoimmune polyglandular syndrome through the Pubmed database. 50 references in English of the greatest impact were selected. Results: Polyglandular autoimmune syndromes are multifactorial diseases with at least two coexisting autoimmune-mediated endocrinopathies. In most endoc

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Slukhanchuk, E. V. "NETs and oncologic process." Obstetrics, Gynecology and Reproduction 15, no. 1 (2021): 107–16. http://dx.doi.org/10.17749/2313-7347/ob.gyn.rep.2021.204.

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Neutrophil Extracellular Traps (NETs) represent the networks consisting of DNA, histones, and proteins produced by activated neutrophils. Such structures have been proved to play a crucial role in inducing neutrophil innate immune response in the pathogenesis of such autoimmune conditions as systemic lupus erythematosus, rheumatoid arthritis, psoriasis, as well as in the pathogenesis of other non-infectious processes, e. g., clotting disorders, thrombosis, diabetes, atherosclerosis, vasculitis and oncology diseases. Recent studies on animal models and human pathologies have uncovered a tremend

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Chen, Jiang, Wen Liu, and Wenjing Zhu. "Foxp3⁺ Treg Cells Are Associated with Pathological Process of Autoimmune Hepatitis by Activating Methylation Modification in Autoimmune Hepatitis Patients." Medical Science Monitor 25 (August 18, 2019): 6204–12. http://dx.doi.org/10.12659/msm.915408.

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Marušić, E., B. Rešić, R. Kuzmanić-Šamija, M. Tomasović, and A. Ursić. "P06.16 Clinical manifestations of the autoimmune process of central nervous system." European Journal of Paediatric Neurology 15 (May 2011): S59. http://dx.doi.org/10.1016/s1090-3798(11)70198-2.

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Brenner, Sarah, and Jacob Mashiah. "Autoimmune blistering diseases in children: signposts in the process of evaluation." Clinics in Dermatology 18, no. 6 (2000): 711–24. http://dx.doi.org/10.1016/s0738-081x(00)00154-1.

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Ferreira, C. A. "Autoimmune diseases: beyond clinical and/or immune parameters to pathogenic process." Pathologie Biologie 54, no. 3 (2006): 119–21. http://dx.doi.org/10.1016/j.patbio.2005.08.004.

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Bonello, Michael, Andrew J. Larner, and Anthony G. Marson. "Profound Amnesia after Temporal Lobectomy: An Autoimmune Process Resembling Patient H.M." Case Reports in Neurology 6, no. 3 (2014): 251–55. http://dx.doi.org/10.1159/000369058.

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Sweeney, Michael. "Autoimmune Neurologic Diseases in Children." Seminars in Neurology 38, no. 03 (2018): 355–70. http://dx.doi.org/10.1055/s-0038-1660520.

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AbstractAutoimmune diseases of the nervous system in children are composed of a heterogeneous group of rare disorders that can affect the central or peripheral nervous system at any level. Presentations may occur in children of any age and are typically acute or subacute in onset. Consideration of an autoimmune process as the etiology of neurologic diseases in children is important, as it may lead to early initiation of immunotherapy and an improvement in long-term neurologic outcomes. The developing nervous and immune systems in children create unique challenges in diagnosis and treatment of

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Vergoossen, Dana L. E., Jaap J. Plomp, Christoph Gstöttner, et al. "Functional monovalency amplifies the pathogenicity of anti-MuSK IgG4 in myasthenia gravis." Proceedings of the National Academy of Sciences 118, no. 13 (2021): e2020635118. http://dx.doi.org/10.1073/pnas.2020635118.

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Human immunoglobulin (Ig) G4 usually displays antiinflammatory activity, and observations of IgG4 autoantibodies causing severe autoimmune disorders are therefore poorly understood. In blood, IgG4 naturally engages in a stochastic process termed “Fab-arm exchange” in which unrelated IgG4s exchange half-molecules continuously. The resulting IgG4 antibodies are composed of two different binding sites, thereby acquiring monovalent binding and inability to cross-link for each antigen recognized. Here, we demonstrate that this process amplifies autoantibody pathogenicity in a classic IgG4-mediated

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Eliseeva, D. D., I. A. Zavalishin, A. V. Karaulov, and S. N. Bykovskaya. "THE ROLE OF REGULATORY T CELLS IN THE DEVELOPMENT OF AUTOIMMUNE PROCESS IN MULTIPLE SCLEROSIS." Annals of the Russian academy of medical sciences 67, no. 3 (2012): 68–74. http://dx.doi.org/10.15690/vramn.v67i3.188.

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In the maintenance of immunological tolerance important role belongs to the recently discovered population of regulatory T-cells CD4 + CD25 + FoxP3 +. These cells have potential in suppressing pathologic immune responses observed at various autoimmune diseases including multiple sclerosis. We have shown a reduction in the number and functional activity of T-reg in peripheral blood of patients with multiple sclerosis in the acute stage, the increase in their number during remission, duration of the relationship of the autoimmune process and the degree of disability of patients with the contents

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Papp, Kata, Eliane Angst, Stefan Seidel, Renata Flury-Frei, and Franc Heinrich Hetzer. "The Diagnostic Challenges of Autoimmune Pancreatitis." Case Reports in Gastroenterology 9, no. 1 (2015): 56–61. http://dx.doi.org/10.1159/000377623.

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Autoimmune pancreatitis is a rare but important differential diagnosis from pancreatic cancer. This autoimmune disease can mimic pancreatic cancer by its clinical symptoms, including weight loss and jaundice. Furthermore imaging findings may include a mass of the pancreas. Here we present the case of a 67-year-old male patient diagnosed with autoimmune pancreatitis but showing the well-known symptoms of pancreatic cancer. This emphasizes the difficulties of histological findings and the importance of the correct diagnostic process.

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Matsukuma, Karen, and Michael Torbenson. "Autoimmune Gastritis: An Underappreciated Entity." AJSP: Reviews and Reports 24, no. 4 (2019): 150–56. http://dx.doi.org/10.1097/pcr.0000000000000320.

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Abstract Autoimmune gastritis is a relatively common but likely underdiagnosed form of chronic gastritis that is associated with iron-deficiency anemia as well as vitamin B12/cobalamin deficiency. This disease confers a 13-fold increased risk of gastric well-differentiated neuroendocrine tumors, due to persistently elevated gastrin levels, and a 3- to 7-fold increased risk of gastric adenocarcinoma. The case described here has a typical presentation of the disease, and the following review highlights key histologic features that aid in the identification of this inflammatory process. Additiona

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Tian, Feng, Hui Chen, Jianmin Zhang, and Wei He. "Reprogramming Metabolism of Macrophages as a Target for Kidney Dysfunction Treatment in Autoimmune Diseases." International Journal of Molecular Sciences 23, no. 14 (2022): 8024. http://dx.doi.org/10.3390/ijms23148024.

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Chronic kidney disease (CKD), as one of the main complications of many autoimmune diseases, is difficult to cure, which places a huge burden on patients’ health and the economy and poses a great threat to human health. At present, the mainstream view is that autoimmune diseases are a series of diseases and complications caused by immune cell dysfunction leading to the attack of an organism’s tissues by its immune cells. The kidney is the organ most seriously affected by autoimmune diseases as it has a very close relationship with immune cells. With the development of an in-depth understanding

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S, Franjić. "In Autoimmune Disease the Immune System Attacks the Body in which it is Located." Vaccines & Vaccination Open Access 8, no. 2 (2023): 1–6. http://dx.doi.org/10.23880/vvoa-16000162.

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Autoimmune diseases are diseases in which the immune system mistakenly attacks the body in which it is located. The function of the immune system is extremely important because it protects against bacteria, parasites, viruses and cancerous mutations. Unfortunately, there are also situations in which the mentioned system can turn against the person himself. There is no clear reason why this happens, which is why autoimmune diseases are very often the subject of many studies today, and there are over a hundred known autoimmune diseases that are mostly accompanied by certain mutations and changes

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Baranski Lamback, Elisa, Grazia Morandi, Eleni Rapti, Georgi Christov, Paul A. Brogan, and Peter Hindmarsh. "Addison’s disease presenting with perimyocarditis." Journal of Pediatric Endocrinology and Metabolism 31, no. 1 (2018): 101–5. http://dx.doi.org/10.1515/jpem-2017-0278.

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AbstractBackground:Polyglandular autoimmune syndrome (PGA) and eosinophilic granulomatosis with polyangiitis (EGPA) do not seem to represent a coincidental association.Case presentation:A case of a 15-year-old boy is reported who presented with severe systemic inflammation, perimyocarditis and cardiogenic shock, in whom EGPA was initially suspected and later diagnosed with autoimmune adrenalitis with PGA.Conclusions:The severity of the systemic inflammation and perimyocarditis suggests a more widespread autoimmune-mediated process. Autoimmune adrenal insufficiency should be considered in all c

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Polonskii, Evgeny L., Dmitriy I. Skulyabin, Sergey V. Lapin, et al. "Polymorphism of autoimmune encephalitis." Annals of Clinical and Experimental Neurology 13, no. 2 (2019): 79–90. https://doi.org/10.25692/acen.2019.2.9.

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This review analyses the current understanding and diagnostic approaches to the management of patients with autoimmune encephalitis. Cellular and synaptic targets, involved in the pathological process in autoimmune encephalitis, are described. The presence of clinical and immunological differences in the pathology is emphasized: on the one hand, non-structural damage to the nervous system is combined with the subacute development of cognitive impairment, epileptic and psychopathological syndromes, which, on the other hand, are associated with polymorphic immunological heterogeneity. The algori

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