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Artykuły w czasopismach na temat „Bilirubin-induced neurologic dysfunction”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Bhutani, Vinod K., and Ronald Wong. "Bilirubin-induced neurologic dysfunction (BIND)." Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 1. http://dx.doi.org/10.1016/j.siny.2014.12.010.

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Johnson, Lois, and Vinod K. Bhutani. "The Clinical Syndrome of Bilirubin-Induced Neurologic Dysfunction." Seminars in Perinatology 35, no. 3 (2011): 101–13. http://dx.doi.org/10.1053/j.semperi.2011.02.003.

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Bhutani, Vinod K., and Lois Johnson-Hamerman. "The clinical syndrome of bilirubin-induced neurologic dysfunction." Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 6–13. http://dx.doi.org/10.1016/j.siny.2014.12.008.

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Morioka, Ichiro, Sota Iwatani, Tsubasa Koda, Kazumoto Iijima, and Hajime Nakamura. "Disorders of bilirubin binding to albumin and bilirubin-induced neurologic dysfunction." Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 31–36. http://dx.doi.org/10.1016/j.siny.2014.11.001.

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Wusthoff, Courtney J., and Irene M. Loe. "Impact of bilirubin-induced neurologic dysfunction on neurodevelopmental outcomes." Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 52–57. http://dx.doi.org/10.1016/j.siny.2014.12.003.

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Wong, Ronald J., and David K. Stevenson. "Neonatal hemolysis and risk of bilirubin-induced neurologic dysfunction." Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 26–30. http://dx.doi.org/10.1016/j.siny.2014.12.005.

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Lakatos, Lajos, and György Balla. "Dysregulation of brain metal homeostasis in bilirubin-induced neurologic dysfunction." Journal of Trace Elements in Medicine and Biology 36 (July 2016): 90. http://dx.doi.org/10.1016/j.jtemb.2016.03.004.

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Alaba, Emmanuel Oluwaseun, Iyabode Olabisi Florence Dedeke, and Dominic Donatus Agbo. "Utilizing the Bilirubin-Albumin Ratio as a Predictive Marker for Bilirubin-Induced Neurologic Dysfunction: A Comparative Analysis in Two Referral Hospitals in Abeokuta." Journal of Advances in Medicine and Medical Research 36, no. 2 (2024): 125–34. http://dx.doi.org/10.9734/jammr/2024/v36i25373.

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Aim: We aim to assess the predictive value of Bilirubin Albumin Ratio (BAR) for evaluating bilirubin-induced neurologic dysfunction (BIND). Bilirubin-induced neurologic dysfunction is a major cause of morbidity in neonates with highest burden in Sub-Saharan Africa and other resource-limited settings. Bilirubin Albumin Ratio (BAR) has been purported to correlate better with BIND than total serum bilirubin (TSB) which is the conventional tool used in evaluating babies with severe hyperbilirubinemia. There is however paucity of studies utilizing BAR in evaluating BIND. Study Design: The stu

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Bhutani, Vinod K., and David K. Stevenson. "The Need for Technologies to Prevent Bilirubin-Induced Neurologic Dysfunction Syndrome." Seminars in Perinatology 35, no. 3 (2011): 97–100. http://dx.doi.org/10.1053/j.semperi.2011.02.002.

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Shapiro, Steven M., and Gerald R. Popelka. "Auditory Impairment in Infants at Risk for Bilirubin-Induced Neurologic Dysfunction." Seminars in Perinatology 35, no. 3 (2011): 162–70. http://dx.doi.org/10.1053/j.semperi.2011.02.011.

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Shapiro, Steven M. "Definition of the Clinical Spectrum of Kernicterus and Bilirubin-Induced Neurologic Dysfunction (BIND)." Journal of Perinatology 25, no. 1 (2004): 54–59. http://dx.doi.org/10.1038/sj.jp.7211157.

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Haywood, Susan. "Answer to: Dysregulation of brain metal homeostasis in bilirubin-induced neurologic dysfunction (BIND)." Journal of Trace Elements in Medicine and Biology 36 (July 2016): 91–92. http://dx.doi.org/10.1016/j.jtemb.2016.03.005.

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Zipursky, Alvin, and Vinod K. Bhutani. "Impact of Rhesus disease on the global problem of bilirubin-induced neurologic dysfunction." Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 2–5. http://dx.doi.org/10.1016/j.siny.2014.12.001.

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Asfari, Ahmed, Carolyn R. Ahlers-Schmidt, and Barry T. Bloom. "Total and Fractionated Bilirubin during the First Week in the Neonatal Intensive Care Unit." Kansas Journal of Medicine 7, no. 4 (2014): 129–31. http://dx.doi.org/10.17161/kjm.v7i4.11500.

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Background. Fractionated bilirubin requires more blood (0.6 ml) than total bilirubin alone (0.2 ml). Our focus during the first week in the Neonatal Intensive Care Unit (NICU) is on prevention of Bilirubin Induced Neurologic Dysfunction and kernicterus, which do not require fractionation. We wanted to determine the benefit of knowing fractionated bilirubin in the first week. Methods. In this retrospective study, data were obtained from the first week for 1202 NICU inborn admissions. Results. Direct bilirubin was more than 2.0 mg/dl in only six infants (0.6%). Five had multisystem injury from h

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Daood, Monica J., Mitchell Hoyson, and Jon F. Watchko. "Lipid peroxidation is not the primary mechanism of bilirubin-induced neurologic dysfunction in jaundiced Gunn rat pups." Pediatric Research 72, no. 5 (2012): 455–59. http://dx.doi.org/10.1038/pr.2012.111.

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Watchko, Jon F., Michael J. Painter, and Ashok Panigrahy. "Are the neuromotor disabilities of bilirubin-induced neurologic dysfunction disorders related to the cerebellum and its connections?" Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 47–51. http://dx.doi.org/10.1016/j.siny.2014.12.004.

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DelFavero, Julie Jensen, Amy J. Jnah, and Desi Newberry. "Glucose-6-Phosphate Dehydrogenase Deficiency and the Benefits of Early Screening." Neonatal Network 39, no. 5 (2020): 270–82. http://dx.doi.org/10.1891/0730-0832.39.5.270.

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Glucose-6-phosphate dehydrogenase (G6PD) deficiency, the most common enzymopathy worldwide, is an insufficient amount of the G6PD enzyme, which is vital to the protection of the erythrocyte. Deficient enzyme levels lead to oxidative damage, hemolysis, and resultant severe hyperbilirubinemia. If not promptly recognized and treated, G6PD deficiency can potentially lead to bilirubin-induced neurologic dysfunction, acute bilirubin encephalopathy, and kernicterus. Glucose-6-phosphate dehydrogenase deficiency is one of the three most common causes for pathologic hyperbilirubinemia. A change in migra

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Nair, Jayasree, and Vasantha Kumar. "Liver Failure and Conjugated Hyperbilirubinemia in a Preterm Neonate: Role of Early IVIG and Exchange Transfusion." American Journal of Perinatology Reports 08, no. 02 (2018): e95-e98. http://dx.doi.org/10.1055/s-0038-1649339.

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AbstractNeonatal liver failure (NLF) is a rare diagnosis but carries with it significant risks of mortality and morbidity. Common etiologies for NLF include metabolic causes, gestational alloimmune liver disease (GALD or neonatal hemochromatosis), and viral infections. We report a case of liver failure in a premature infant with abnormal iron profile within 48 hours of birth. Lack of accepted guidelines for the initial management of severe jaundice with a high direct component in the first week after birth made treatment challenging. The infant underwent intensive phototherapy along with four

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Duarte, Jacinto, Santiago Macias, Francisco Coria, Enrique Fernandez, and Luis Erick Clavería. "Valproate-Induced Coma: Case Report and Literature Review." Annals of Pharmacotherapy 27, no. 5 (1993): 582–83. http://dx.doi.org/10.1177/106002809302700510.

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OBJECTIVE: To report a case of hyperammonemia without hepatic dysfunction as a possible cause of lethargy, stupor, and coma in a woman after valproic acid (VPA) administration, and discuss the possible different mechanisms of ammonia elevation and coma. CASE SUMMARY: A woman diagnosed with complex partial seizures that secondarily generalize was treated with phenytoin (PHT) 250 mg/d for 18 years. Three months before admission, this dosage was increased to 300 mg/d and phenobarbital (PB) 100 mg/d was added because the seizures were incompletely controlled. The patient developed a progressive in

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Victor Idowu, Joel-Medewase, Adebami Olusegun Joseph, and Alabi Ayobami Oyetunji. "Maternal and Neonatal Factors Associated with the Incidence of Acute Bilirubin Encephalopathy in Nigerian Population." International Journal of Health Sciences and Research 14, no. 9 (2024): 14–24. http://dx.doi.org/10.52403/ijhsr.20240903.

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Background: Acute bilirubin encephalopathy (ABE) is still a common cause of morbidity and mortality in developing countries despite the availability, and efficacy of phototherapy and exchange blood transfusion. The prevalence of kernicterus has not shown any significant reduction in low and middle-income countries (LMICs) because many neonates with jaundice present very late with overt features of ABE. Objective: The study was done to determine maternal and neonatal factors associated with ABE and factors responsible for the late presentation of neonatal jaundice among the patients presenting

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van Toorn, Ronald, Philip Brink, Johan Smith, Christelle Ackermann, and Regan Solomons. "Bilirubin-Induced Neurological Dysfunction." Journal of Child Neurology 31, no. 14 (2016): 1579–83. http://dx.doi.org/10.1177/0883073816666473.

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The clinical expression of bilirubin-induced neurological dysfunction varies according to severity and location of the disease. Definitions have been proposed to describe different bilirubin-induced neurological dysfunction subtypes. Our objective was to describe the severity and clinico-radiological-neurophysiological correlation in 30 consecutive children with bilirubin-induced neurological dysfunction seen over a period of 5 years. Thirty children exposed to acute neonatal bilirubin encephalopathy were included in the study. The mean peak total serum bilirubin level was 625 μmol/L (range 48

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Good, William V., and Chuan Hou. "Visuocortical bilirubin-induced neurological dysfunction." Seminars in Fetal and Neonatal Medicine 20, no. 1 (2015): 37–41. http://dx.doi.org/10.1016/j.siny.2014.12.007.

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Geiger, Angela S., Ann C. Rice, and Steven M. Shapiro. "Minocycline Blocks Acute Bilirubin-Induced Neurological Dysfunction in Jaundiced Gunn Rats." Neonatology 92, no. 4 (2007): 219–26. http://dx.doi.org/10.1159/000103740.

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Wang, Nanqin, Yongzhu Jia, Xuanzi Zhou, Xia Wang, Huyao Zhou, and Nong Xiao. "Effects of Repetitive Transcranial Magnetic Stimulation on Pallidum GABAergic Neurons and Motor Function in Rat Models of Kernicterus." Brain Sciences 13, no. 9 (2023): 1252. http://dx.doi.org/10.3390/brainsci13091252.

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Kernicterus is a serious complication of hyperbilirubinemia, caused by neuronal injury due to excessive unconjugated bilirubin (UCB) in specific brain areas. This injury induced by this accumulation in the globus pallidus can induce severe motor dysfunction. Repetitive transcranial magnetic stimulation (rTMS) has shown neuroprotective effects in various neurological diseases. This study aimed to investigate the effects of rTMS on pallidal nerve damage and motor dysfunction in a rat model of kernicterus. Rats were divided into a sham group (n = 16), a model group (bilirubin with sham rTMS; n =

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Parfenova, Helena, Charles W. Leffler, Shyamali Basuroy, Jianxiong Liu, and Alexander L. Fedinec. "Antioxidant Roles of Heme Oxygenase, Carbon Monoxide, and Bilirubin in Cerebral Circulation during Seizures." Journal of Cerebral Blood Flow & Metabolism 32, no. 6 (2012): 1024–34. http://dx.doi.org/10.1038/jcbfm.2012.13.

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Postictal cerebrovascular dysfunction is an adverse effect of seizures in newborn piglets. The brain heme oxygenase (HO) provides protection against cerebrovascular dysfunction. We investigated the contribution of reactive oxygen species (ROS) to seizure-induced vascular damage and the mechanism of HO vasoprotection. In a bicuculline model of seizures, we addressed the hypotheses: (1) seizures increase brain ROS; (2) ROS contribute to cerebral vascular dysfunction; (3) ROS initiate a vasoprotective mechanisms by activating endogenous HO; and (4) HO products have antioxidant properties. As asse

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Rice, Ann C., Victoria L. Chiou, Sarah B. Zuckoff, and Steven M. Shapiro. "Profile of minocycline neuroprotection in bilirubin-induced auditory system dysfunction." Brain Research 1368 (January 2011): 290–98. http://dx.doi.org/10.1016/j.brainres.2010.10.052.

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Palanigounder, Raju, Karthikeyan Raju, Nithya Parasuraman, Supasakthi Sundaraswamy, and Ramila Rajgopal. "Hemolytic Jaundice in Newborn with Risk of Neurotoxicity: Unravelling the Dilemma in Management." SAR Journal of Medical Case Reports 6, no. 01 (2025): 10–14. https://doi.org/10.36346/sarjmcr.2025.v06i01.003.

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Hemolytic disease of the fetus & newborn (HDFN) is a disorder where RBCs are destroyed by maternal erythrocyte alloantibodies and there is increased production of unconjugated bilirubin and anemia and in severe cases, hydrops in the fetus. Rh & ABO incompatibility are the most common causes of severe unconjugated hyperbilirubinemia in newborn babies. Though many treatment options are available for this condition, phototherapy stands first and most important of these all. This article signifies the successful management of OB incompatibility with hemolytic jaundice and clinical sepsis i

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Ahlfors, Charles E., Richard P. Wennberg, J. Donald Ostrow, and Claudio Tiribelli. "Unbound (Free) Bilirubin: Improving the Paradigm for Evaluating Neonatal Jaundice." Clinical Chemistry 55, no. 7 (2009): 1288–99. http://dx.doi.org/10.1373/clinchem.2008.121269.

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Abstract Background: The serum or plasma total bilirubin concentration (BT) has long been the standard clinical laboratory test for evaluating neonatal jaundice, despite studies showing that BT correlates poorly with acute bilirubin encephalopathy (ABE) and its sequelae including death, classical kernicterus, or bilirubin-induced neurological dysfunction (BIND). The poor correlation between BT and ABE is commonly attributed to the confounding effects of comorbidities such as hemolytic diseases, prematurity, asphyxia, or infection. Mounting evidence suggests, however, that BT inherently perform

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Llido, John Paul, Emanuela Fioriti, Aleš Dvořák, et al. "Severe motor dysfunction in Gunn rats, a model for neonatal bilirubin-induced damage, is not due to changes in brain bilirubin and total serum bilirubin." Journal of the Neurological Sciences 455 (December 2023): 121605. http://dx.doi.org/10.1016/j.jns.2023.121605.

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Matzdorff, Axel, Andrej Wojciechowski, Paulina Zyluk-Gadowska, Firat Baluken, and Annerose Meier. "A Patient with Target Cells and Hyperkalemia - or why Hematologists Should Still be Able to ‘Fly by Sight‘." Blood 142, Supplement 1 (2023): 5223. http://dx.doi.org/10.1182/blood-2023-173109.

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INTRODUCTION: Hematology has changed. Both benign and malignant hematology rely heavily on new diagnostic techniques, such as flow-cytometry, cytogenetics and most recently single gene analysis by next generation sequencing. Examination of a blood smear has come out of favor and is usually performed by lab technicians and not by the hematologist. This case report describes how examination of the blood smear which showed an unexpected red blood cell morphology eventually led after 40 years to the right diagnosis. CASE: R. C. is a 42-year-old male (100 kg = 220 lbs, 1,83 m = 6 ft, BMI 29,9) diag

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Kumar, Surendra, Karnika Agrawal, Sunil Kumar, and Anupam Chaturvedi. "Risk Stratification of Term Neonates for Development of Significant Hyperbilirubinemia by Cord Serum Bilirubin and Albumin Ratio." Medical Journal of Dr. D.Y. Patil Vidyapeeth 17, no. 3 (2023): 600–604. http://dx.doi.org/10.4103/mjdrdypu.mjdrdypu_1075_22.

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ABSTRACT Background: Neonatal hyperbilirubinemia is a common neonatal problem. Although benign in the majority of the cases, few neonates develop bilirubin-induced neurological dysfunction further progressing to kernicterus. Due to early hospital discharge of healthy-term neonates, there is need of a reliable tool for early prediction of significant hyperbilirubinemia in them. Aim: The aim of this study is to determine the utility of cord total serum bilirubin/albumin ratio as an early predictor of significant neonatal hyperbilirubinemia in healthy-term newborns. Settings and Design: This is a

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Maksymenko, Mykhailo, and Roman Havryliuk. "COGNITIVE IMPAIRMENT CAUSED BY HYPERBILIRUBINEMIA IN ADULT PATIENTS WITH MECHANICAL JAUNDICE SECONDARY TO CHOLEDOCHOLITHIASIS." Eastern Ukrainian Medical Journal 13, no. 1 (2025): 64–69. https://doi.org/10.21272/eumj.2025;13(1):64-69.

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Background. The neurotoxic effects of bilirubin are well-documented in neonatal jaundice, where hyperbilirubinemia can lead to kernicterus and long-term neurodevelopmental deficits. However, the cognitive impact of hyperbilirubinemia in adults, particularly in the context of mechanical jaundice, remains poorly understood. With the rising prevalence of choledocholithiasis due to lifestyle changes and an aging population, understanding the neurological consequences of this condition is becoming increasingly important. Objectives. This study aimed to characterize the extent and nature of cognitiv

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Zidan, Lamiaa Khaled, Mohamed Ahmed Rowisha, Rasha Ahmed Elshafey, Trandil Hassan Elmehallawi, and Heba Saied Elmahdy. "Correlation between Magnetic Resonance Spectroscopy and Auditory Brain-Stem Response Audiometry in Acute Bilirubin Encephalopathy in Full Term Neonates." Journal of Advances in Medicine and Medical Research 35, no. 22 (2023): 110–17. http://dx.doi.org/10.9734/jammr/2023/v35i225252.

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Background: This research aimed to correlate between MRS and ABR to see whether MRS offers new perspectives on the pathophysiology of hyperbilirubinemia and the early diagnosis of acute bilirubin encephalopathy (ABE) in full-term neonates. Methods: This observational cohort study was done at Tanta University Hospitals between March 2019 and March 2021. It enrolled 26 newborns with ABE who were diagnosed with BIND-M score (Modified Bilirubin Induced Neurological Dysfunction Score), In addition, 20 healthy, age-matched neonates were enrolled as control subjects. Brain MRS and ABR were done

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Shrestha, Abhigan B., Bikash Karki, Prakash Acharya, et al. "Liver injury: the therapeutic dilemma of homeopathy – a case report from Bangladesh." Annals of Medicine & Surgery 85, no. 7 (2023): 3642–45. http://dx.doi.org/10.1097/ms9.0000000000000918.

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Despite the lack of scientific evidence supporting its effectiveness, homeopathic treatment is increasingly being used as a form of alternative medicine, with many people taking homeopathic remedies instead of drug therapies. It is based on the principle of ‘like cures like’, meaning that a remedy similar to the illness can be used to treat it. However, there have been several reports suggesting the risks of homeopathic remedies, among which homeopathy-induced liver injury is widely discussed. Here, we report a case of a 35-year-old well-oriented male patient with a typical clinical presentati

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Ghorui, Arnab, Bhabesh Kant Chowdhry, Pramod Kumar Manjhi, Pradeep Kumar, and Chandra Mohan Kumar. "Evaluation of efficacy of oral calcium phosphate as an adjunct to standard-of-care regular phototherapy in cases of neonatal jaundice: a hospital-based double-blind, randomised, placebo-controlled trial." BMJ Paediatrics Open 8, no. 1 (2024): e002902. http://dx.doi.org/10.1136/bmjpo-2024-002902.

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ObjectiveNeonatal jaundice is the most common cause of neonatal morbidity and rehospitalisation in the first week of life, affecting approximately 60% of term and 80% of preterm neonates, with 10% requiring phototherapy to prevent bilirubin-induced neurological dysfunction. Enterohepatic circulation contributes 10%–20% of the body’s bilirubin load, and oral calcium-phosphate can inhibit this process by binding to unconjugated bilirubin and acting as a bilirubin-trapping agent in the gut. This study aimed to evaluate the efficacy of oral calcium phosphate as an adjunct to phototherapy in reduci

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Lakshmi, C. V. S., Syed Adnan Ali, Uppin Narayan Reddy, Farhana Nazneen, and Muzammil. "A prospective study on etiological factors and clinical indicators in term and near term neonates admitted with hyperbilirubinemia in nicu in a tertiary care hospital in Hyderabad, India." IP International Journal of Medical Paediatrics and Oncology 7, no. 2 (2021): 51–56. http://dx.doi.org/10.18231/j.ijmpo.2021.011.

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1.To study the etiology and risk factors of neonatal hyperbilirubinemia in term and near-term infants. 2. To study the clinical course of these infants during NICU stayThe present study was conducted at NICU, Department of Pediatrics, Princess Esra hospital, Deccan Medical College, Hyderabad, India, from October 2019 to October 2020. Term and late preterm infants admitted in NICU with Serum Bilirubin levels more than 12mg/dl were included in the study. The risk factors, etiology and clinical profile of these infants during NICU stay were studied. 210 neonates were admitted in NICU with hyperbi

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J., Aaseth, Bjorklund G., Dadar M., and Lakatos L. "NEUROPROTECTIVE EFFECTS OF D - PENICILLAMINEIN THE NEONATAL PERIOD." Наука и здравоохранение, no. 5(21) (November 29, 2019): 116–20. http://dx.doi.org/10.34689/sh.2019.5.54366.

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Originally D-penicillamine (D-PA) was used due to its potential benefits in the treatment of neonatal hyperbilirubinemia caused by hemolytic diseases in newborn infants or immaturity of the liver enzyme uridine 5-diphospho-glucuronosyltransferase (UDP-glucuronosyltransferase). In that period, the prevalence of retinopathy of prematurity (ROP) in infants who received treatment with D-PA. Later, studies were performed by different institutes in Poland, Hungary, the USA, Mexico, and India. Short or long-term use of the medication didnt seem to have caused toxicity or intolerance, even D-PA in the

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Mosallam, Dalia, Reem N. Said, Marwa A. Abd Elsamad, and Nada M. Abdelfatah. "Use of serum bilirubin/albumin ratio for early prediction of bilirubin induced neurological dysfunction." Egyptian Pediatric Association Gazette 67, no. 1 (2019). http://dx.doi.org/10.1186/s43054-019-0010-x.

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Abstract Background Kernicterus or bilirubin encephalopathy is a preventable cause of handicap, still occurs in our country. The aim of the current study was to assess the role of bilirubin/albumin ratio in improving the morbidity of the cases with unconjugated hyperbilirubinemia and to estimate of the cutoff value for B/A ratio for prevention and early management of bilirubin-induced neurological dysfunction. Results The mean gestational age was 37.1 ± 2.11 weeks; the mean age of onset of jaundice was 2.36 ± 1.04 days; the mean level of total bilirubin was 26.14 ± 7.36 mg/dl. At chosen cutoff

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L, Lakatos, and Balla G. "Bilirubin-induced neurologic dysfunction (BIND): appearances are fairly often deceptive." Birth Defects 2, no. 1 (2017). http://dx.doi.org/10.15761/bdj.1000109.

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Manni, Isabella, Rocco Giuliana Di, Salvatore Fusco, et al. "Monitoring the response of hyperbilirubinemia in the mouse brain by in vivo bioluminescence imaging." MDPI, June 19, 2017. https://doi.org/10.5281/zenodo.813877.

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Increased levels of unconjugated bilirubin are neurotoxic, but the mechanism leading to neurological damage has not been completely elucidated. Innovative strategies of investigation are needed to more precisely define this pathological process. By longitudinal in vivo bioluminescence imaging, we noninvasively visualized the brain response to hyperbilirubinemia in the MITO-Luc mouse, in which light emission is restricted to the regions of active cell proliferation. We assessed that acute hyperbilirubinemia promotes bioluminescence in the brain region, indicating an increment in the cell prolif

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Lakatos, Lajos. "A Dangerous Vitious Circle of the Neonatal Brain in Bilirubin- Induced Neurologic Dysfunction (BIND)." MOJ Bioequivalence & Bioavailability 3, no. 3 (2017). http://dx.doi.org/10.15406/mojbb.2017.03.00038.

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Usman, Fatima, Zubaida Ladan Farouk, Abdulaziz Ahmed, and Muutassim Ibrahim. "Comparison of Bilirubin Induced Neurologic Dysfunction Score with Brainstem Auditory Evoked Response in Detecting Acute Bilirubin Encephalopathy in Term Neonates." JOURNAL OF CLINICAL AND DIAGNOSTIC RESEARCH, 2019. http://dx.doi.org/10.7860/jcdr/2019/41670.13382.

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Zidan, Lamiaa Khaled, Mohamed Ahmed Rowisha, Mohammed Abd Ellatif Nassar, Rasha Ahmed Elshafey, Trandil Hassan El Mahallawi, and Heba Saied Elmahdy. "Magnetic resonance spectroscopy and auditory brain-stem response audiometry as predictors of bilirubin-induced neurologic dysfunction in full-term jaundiced neonates." European Journal of Pediatrics, November 18, 2023. http://dx.doi.org/10.1007/s00431-023-05246-z.

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AbstractThe purpose of this research was to define the functions of MRS and ABR as predictors of bilirubin-induced neurologic dysfunction (BIND) in full-term neonates who required intervention (phototherapy and/or exchange transfusion). This prospective cohort study was done at the NICU of Tanta University Hospitals over a 2-year duration. Fifty-six full-term neonates with pathological unconjugated hyperbilirubinemia were divided according to MRS and ABR findings into 2 groups: group (1) included 26 cases with mild acute bilirubin encephalopathy (BIND-M score 1–4). Group (2) included 30 cases

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Liu, Hua, Ruolin Zhang, Min Zhang, Huanji Zheng, Xiuxiu Li, and Jun Chen. "Diagnostics Value of Quantitative Magnetic Resonance Imaging (MRI) in Neonatal Acute Bilirubin Encephalopathy." Journal of Child Neurology, April 16, 2023, 088307382311685. http://dx.doi.org/10.1177/08830738231168514.

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Background: This study was designed to investigate the diagnostic value of relative signal intensity of globus pallidus on T1-weighted magnetic resonance imaging (MRI) in neonatal acute bilirubin encephalopathy (ABE). Methods: Participants who were recruited in hospital from April 2019 to May 2020 were grouped into mildly increased total serum bilirubin (TSB) group (n = 30), severely increased TSB group (n = 25), or extremely increased TSB group (n = 10) based on the total serum bilirubin level. Bilirubin-induced neurologic dysfunction scale score was used to determine if participants had acut

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Radmacher, Paula G., Frank D. Groves, Joshua A. Owa, et al. "A modified Bilirubin-induced neurologic dysfunction (BIND-M) algorithm is useful in evaluating severity of jaundice in a resource-limited setting." BMC Pediatrics 15, no. 1 (2015). http://dx.doi.org/10.1186/s12887-015-0355-2.

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Jalal Al-Mosawi, Aamir. "Kernicterus: The continuing occurrence of a preventable chronic disabling neurologic condition." Journal of Clinical Images and Medical Case Reports 2, no. 1 (2020). http://dx.doi.org/10.52768/2766-7820/1006.

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Background: Kernicterus is a preventable condition that has become increasingly rare in many countries in the world. It is chronic severe form of bilirubin-induced neurological dysfunction that is associated with a variety of metal and neurological abnormalities resulting from bilirubininduced brain damage mostly in the basal ganglia and during the neonatal period. Despite the condition has been increasingly considered as preventable, it is still seen in some geographic areas where diagnostic and preventive interventions continue to experience failures. The aim of this paper is to emphasize th

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Lin, Qihuan, Lanmei Chen, Hongyi Zheng, Hui Tan, Gengbiao Zhang, and Wenbin Zheng. "Imaging of nerve injury in neonatal acute bilirubin encephalopathy using 1H-MRS and Glu-CEST techniques." Frontiers in Neuroscience 17 (March 28, 2023). http://dx.doi.org/10.3389/fnins.2023.1110349.

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ObjectivesTo investigate the significance of proton magnetic resonance spectroscopy (1H-MRS) and glutamate chemical exchange saturation transfer (Glu-CEST) techniques in assessing the condition and prognosis of acute bilirubin encephalopathy patients and to understand the mechanism of nerve injury in this disease.Materials and methodsFrom September 2019 to February 2021, 31 neonates with acute bilirubin encephalopathy and 16 healthy neonates were enrolled in this study. All the quantitative results of 1H-MRS, Glu-CEST, and conventional magnetic resonance imaging (MRI) of all neonates were anal

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Lee, Heng Yang, Azlin Ithnin, Raja Zahratul Azma, Ainoon Othman, Armindo Salvador, and Fook Choe Cheah. "Glucose-6-Phosphate Dehydrogenase Deficiency and Neonatal Hyperbilirubinemia: Insights on Pathophysiology, Diagnosis, and Gene Variants in Disease Heterogeneity." Frontiers in Pediatrics 10 (May 24, 2022). http://dx.doi.org/10.3389/fped.2022.875877.

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Glucose-6-phosphate dehydrogenase (G6PD) deficiency is a prevalent condition worldwide and is caused by loss-of-function mutations in the G6PD gene. Individuals with deficiency are more susceptible to oxidative stress which leads to the classical, acute hemolytic anemia (favism). However, G6PD deficiency in newborn infants presents with an increased risk of hyperbilirubinemia, that may rapidly escalate to result in bilirubin induced neurologic dysfunction (BIND). Often with no overt signs of hemolysis, G6PD deficiency in the neonatal period appears to be different in the pathophysiology from f

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Zhang, Yi, Juan Zhang, Yu-Chen Chen, Jin-Xia Zheng, and Shaohua Ding. "Microstructure and Perfusion Alterations of Globus Pallidus in Neonates with Hyperbilirubinemia." Current Medical Imaging Formerly Current Medical Imaging Reviews 20 (May 8, 2024). http://dx.doi.org/10.2174/0115734056266762231206074419.

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Background:: Previous studies have indicated the abnormality of the globus pallidus in neonates with hyperbilirubinemia. Objective:: This study aims to explore the microstructure and cerebral perfusion of globus pallidus in neonatal hyperbilirubinemia by using Diffusion Tensor Imaging (DTI) and Arterial Spin Labeling (ASL) approaches Methods:: Thirty-seven neonates were enrolled in this study, which were classified into Bilirubin-Induced Neurologic Dysfunction (BIND) group (hyperbilirubinemia with BIND, n=12), non-BIND group (hyperbilirubinemia without BIND, n=15), and healthy controls (HC) gr

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Ebrahim, Nora Abd El-Alim, Madiha Hassan Bayoumi, and Hanan El-Sayed Metwally. "Effect of education program regarding pathological jaundice on nurses’ performance and neonates’ bilirubin-induced neurological dysfunction." European Journal of Pediatrics 184, no. 1 (2025). https://doi.org/10.1007/s00431-024-05921-9.

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AbstractBoth term and preterm infants are susceptible to pathological jaundice, a frequent condition that can cause long-lasting neurological damage. A novel treatment for indirect pathological hyperbilirubinemia is bilisphere phototherapy, which lowers total serum bilirubin just as well as exchange transfusions. A quasi-experimental research design was utilized in the current study. This study was conducted in Neonatal Intensive Care Unit at Benha Specialized Pediatric Hospital. A convenient sample of all available nurses (60) working in the previously mentioned setting. A purposive sample of

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