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Gotowe bibliografie tematyczne / Bone Diseases, Metabolic – physiopathology / Artykuły w czasopismach

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Artykuły w czasopismach na temat „Bone Diseases, Metabolic – physiopathology”

Autor: Grafiati

Data publikacji: 29 lipca 2024

Data aktualizacji: 31 lipca 2025

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1

Izzo, Marcello, Albino Carrizzo, Carmine Izzo, et al. "Vitamin D: Not Just Bone Metabolism but a Key Player in Cardiovascular Diseases." Life 11, no. 5 (2021): 452. http://dx.doi.org/10.3390/life11050452.

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Vitamin D is the first item of drug expenditure for the treatment of osteoporosis. Its deficiency is a condition that affects not only older individuals but also young people. Recently, the scientific community has focused its attention on the possible role of vitamin D in the development of several chronic diseases such as cardiovascular and metabolic diseases. This review aims to highlight the possible role of vitamin D in cardiovascular and metabolic diseases. In particular, here we examine (1) the role of vitamin D in diabetes mellitus, metabolic syndrome, and obesity, and its influence on

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Iaconis, Antonella, Francesco Molinari, Roberta Fusco, and Rosanna Di Paola. "Mitochondria as a Disease-Relevant Organelle in Rheumatoid Arthritis: A Key Breakout in Fight Against the Disease." Biomedicines 13, no. 7 (2025): 1708. https://doi.org/10.3390/biomedicines13071708.

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Rheumatoid arthritis (RA) is one of the most representative autoimmune diseases. The peculiarity of this disease is synovial inflammation, which results in joint destruction and often disability. Although there are still several pathogenetic mechanisms to be clarified, lately, most studies have highlighted the involvement of mitochondria in the onset and progression of the disease. Mitochondrial functions are connected to many metabolic processes and the delivery of proinflammatory mediators. Mitochondria play a crucial role in the physiopathology of RA, contributing to chronic inflammation, c

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Biver, Emmanuel, Pierre Hardouin, and Joseph Caverzasio. "The “bone morphogenic proteins” pathways in bone and joint diseases: Translational perspectives from physiopathology to therapeutic targets." Cytokine & Growth Factor Reviews 24, no. 1 (2013): 69–81. http://dx.doi.org/10.1016/j.cytogfr.2012.06.003.

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Smiljić, Lj, M. Radunović, M. Muratović, and T. Smilić. "ADIPOKINES AS FOR RUNNERS OF INFLAMMATORY CITOKINE SUPERFAMILY AND ITS INVOLVEMENT IN INFLAMMATORY AND IMMUNE DISEASES." Praxis medica 37, no. 2 (2009): 135–41. http://dx.doi.org/10.70949/pramed200902311s.

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&lt;p&gt;Leptin was discovered in 1994, finding, that marked the beginning of a new understanding about white adipose tissue. Up to end of 20th century this tissue was viewed as an inert tissue, devoted to body protection from heat loss and to passively store energy. This static vision, changed with identification of the product of the gene obese (ob) wich accentuated the role of adipose tissue in the physiopathology of obesity-linked diseases, and led to the discovery of various adipokines, many of a pro-inflammatory nature. With emerged knowledge of (WAT)-derived adipokines, it is ob

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Grill, Vivian, and T. John Martin. "Metabolic bone diseases." Medical Journal of Australia 163, no. 1 (1995): 38–41. http://dx.doi.org/10.5694/j.1326-5377.1995.tb126087.x.

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SEINO, YOSHIKI. "Metabolic bone diseases." Pediatrics International 39, no. 4 (1997): 478. http://dx.doi.org/10.1111/j.1442-200x.1997.tb03623.x.

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Horvai, Andrew E., and Brendan F. Boyce. "Metabolic bone diseases." Seminars in Diagnostic Pathology 28, no. 1 (2011): 13–25. http://dx.doi.org/10.1053/j.semdp.2011.02.004.

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Dubois-Deruy, Emilie, Yara El Masri, Annie Turkieh, Philippe Amouyel, Florence Pinet, and Jean-Sébastien Annicotte. "Cardiac Acetylation in Metabolic Diseases." Biomedicines 10, no. 8 (2022): 1834. http://dx.doi.org/10.3390/biomedicines10081834.

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Lysine acetylation is a highly conserved mechanism that affects several biological processes such as cell growth, metabolism, enzymatic activity, subcellular localization of proteins, gene transcription or chromatin structure. This post-translational modification, mainly regulated by lysine acetyltransferase (KAT) and lysine deacetylase (KDAC) enzymes, can occur on histone or non-histone proteins. Several studies have demonstrated that dysregulated acetylation is involved in cardiac dysfunction, associated with metabolic disorder or heart failure. Since the prevalence of obesity, type 2 diabet

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9

Dumond Bourie, Aurore, Jean-Baptiste Potier, Michel Pinget, and Karim Bouzakri. "Myokines: Crosstalk and Consequences on Liver Physiopathology." Nutrients 15, no. 7 (2023): 1729. http://dx.doi.org/10.3390/nu15071729.

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Non-alcoholic fatty liver disease (NAFLD) is a chronic liver disease mainly characterized by the hepatic accumulation of lipid inducing a deregulation of β-oxidation. Its advanced form is non-alcoholic steatohepatitis (NASH), which, in addition to lipid accumulation, induces hepatocellular damage, oxidative stress and fibrosis that can progress to cirrhosis and to its final stage: hepatocellular carcinoma (HCC). To date, no specific therapeutic treatment exists. The implications of organ crosstalk have been highlighted in many metabolic disorders, such as diabetes, metabolic-associated liver d

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10

Sánchez-Oliver, Antonio Jesús. "Obesity Phisiopathology: Current Perspectives." Journal of Nutritional Biology 4, no. 1 (2017): 21. http://dx.doi.org/10.18314/jnb.v4i1.160.

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Obesity is a global health problem, being considered one of the most serious and prevalent non-communicable diseases of the 21st century. It is currently understood as a multifactorial chronic condition associated with potentially serious complications whose treatment requires a multidisciplinary approach, given its huge clinical impact and associated health-carecost. Besides, properly tackling obesity requires a founded knowledge of its specific physiopathology is required. Together withthe rise in adiposity, a series of cellular processes happen, which cause several metabolic changes that dr

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11

Rossi, Francesca, Chiara Tortora, Marco Paoletta, et al. "Osteoporosis in Childhood Cancer Survivors: Physiopathology, Prevention, Therapy and Future Perspectives." Cancers 14, no. 18 (2022): 4349. http://dx.doi.org/10.3390/cancers14184349.

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The improvement of chemotherapy, radiotherapy, and surgical interventions, together with hematopoietic stem cell transplantation, increased childhood cancer survival rate in the last decades, reaching 80% in Europe. Nevertheless, anti-cancer treatments are mainly responsible for the onset of long-term side effects in childhood cancer survivors (CCS), including alterations of the endocrine system function and activity. In particular, the most frequent dysfunction in CCS is a metabolic bone disorder characterized by low bone mineral density (BMD) with increased skeletal fragility. BMD loss is al

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12

Sinigaglia, L. "Metabolic bone diseases: an overview." Reumatismo 66, no. 2 (2014): 109. http://dx.doi.org/10.4081/reumatismo.2014.783.

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13

Bazzocchi, Alberto, and Giuseppe Guglielmi. "Imaging of Metabolic Bone Diseases." Seminars in Musculoskeletal Radiology 28, no. 05 (2024): 513–14. http://dx.doi.org/10.1055/s-0044-1789256.

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Guglielmi, Giuseppe, Silvana Muscarella, Antonio Leone, and Wilfred C. G. Peh. "Imaging of Metabolic Bone Diseases." Radiologic Clinics of North America 46, no. 4 (2008): 735–54. http://dx.doi.org/10.1016/j.rcl.2008.04.010.

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15

Polyzos, Stergios A., and Christos S. Mantzoros. "Outliers of bone metabolic diseases." Metabolism 80 (March 2018): 1–4. http://dx.doi.org/10.1016/j.metabol.2017.09.009.

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Haugeberg, Glenn. "Imaging of metabolic bone diseases." Best Practice & Research Clinical Rheumatology 22, no. 6 (2008): 1127–39. http://dx.doi.org/10.1016/j.berh.2008.09.016.

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Canalis, Ernesto. "Growth Factors, Bone Metabolism, and Metabolic Bone Diseases." Endocrinologist 6, no. 2 (1996): 89–94. http://dx.doi.org/10.1097/00019616-199603000-00005.

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18

Varlet, Alice-Anaïs, Emmanuèle Helfer, and Catherine Badens. "Molecular and Mechanobiological Pathways Related to the Physiopathology of FPLD2." Cells 9, no. 9 (2020): 1947. http://dx.doi.org/10.3390/cells9091947.

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Laminopathies are rare and heterogeneous diseases affecting one to almost all tissues, as in Progeria, and sharing certain features such as metabolic disorders and a predisposition to atherosclerotic cardiovascular diseases. These two features are the main characteristics of the adipose tissue-specific laminopathy called familial partial lipodystrophy type 2 (FPLD2). The only gene that is involved in FPLD2 physiopathology is the LMNA gene, with at least 20 mutations that are considered pathogenic. LMNA encodes the type V intermediate filament lamin A/C, which is incorporated into the lamina me

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19

Delgadillo-Velázquez, Jaime, Herminia Mendivil-Alvarado, Carlos Daniel Coronado-Alvarado, and Humberto Astiazaran-Garcia. "Extracellular Vesicles from Adipose Tissue Could Promote Metabolic Adaptation through PI3K/Akt/mTOR." Cells 11, no. 11 (2022): 1831. http://dx.doi.org/10.3390/cells11111831.

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Extracellular vesicles (EVs) are nanoparticles secreted by cells under physiological and pathological conditions, such as metabolic diseases. In this context, EVs are considered potential key mediators in the physiopathology of obesity. It has been reported that EVs derived from adipose tissue (ADEVs) contribute to the development of a local inflammatory response that leads to adipose tissue dysfunction. In addition, it has been proposed that EVs are associated with the onset and progression of several obesity-related metabolic diseases such as insulin resistance. In particular, characterizing

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20

Parkman, Robertson, and Crooks. "BONE MARROW TRANSPLANTATION FOR METABOLIC DISEASES." Radiologic Clinics of North America 16, no. 2 (1996): 429–38. http://dx.doi.org/10.1016/s0033-8389(22)00219-6.

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Sobh, Mahmoud M., Mohamed Abdalbary, Sherouk Elnagar, et al. "Secondary Osteoporosis and Metabolic Bone Diseases." Journal of Clinical Medicine 11, no. 9 (2022): 2382. http://dx.doi.org/10.3390/jcm11092382.

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Fragility fracture is a worldwide problem and a main cause of disability and impaired quality of life. It is primarily caused by osteoporosis, characterized by impaired bone quantity and or quality. Proper diagnosis of osteoporosis is essential for prevention of fragility fractures. Osteoporosis can be primary in postmenopausal women because of estrogen deficiency. Secondary forms of osteoporosis are not uncommon in both men and women. Most systemic illnesses and organ dysfunction can lead to osteoporosis. The kidney plays a crucial role in maintaining physiological bone homeostasis by control

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Sobh, Mahmoud M., Mohamed Abdalbary, Sherouk Elnagar, et al. "Secondary Osteoporosis and Metabolic Bone Diseases." Journal of Clinical Medicine 11, no. 9 (2022): 2382. http://dx.doi.org/10.3390/jcm11092382.

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Fragility fracture is a worldwide problem and a main cause of disability and impaired quality of life. It is primarily caused by osteoporosis, characterized by impaired bone quantity and or quality. Proper diagnosis of osteoporosis is essential for prevention of fragility fractures. Osteoporosis can be primary in postmenopausal women because of estrogen deficiency. Secondary forms of osteoporosis are not uncommon in both men and women. Most systemic illnesses and organ dysfunction can lead to osteoporosis. The kidney plays a crucial role in maintaining physiological bone homeostasis by control

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23

Kara Gedik, Gonca. "Radionuclide Imaging in Metabolic Bone Diseases." Nuclear Medicine Seminars 8, no. 1 (2022): 25–31. http://dx.doi.org/10.4274/nts.galenos.2022.0004.

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Allan, P. J., and S. Lal. "Metabolic bone diseases in intestinal failure." Journal of Human Nutrition and Dietetics 33, no. 3 (2019): 423–30. http://dx.doi.org/10.1111/jhn.12726.

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Hannan, Fadil M., Paul J. Newey, Michael P. Whyte, and Rajesh V. Thakker. "Genetic approaches to metabolic bone diseases." British Journal of Clinical Pharmacology 85, no. 6 (2018): 1147–60. http://dx.doi.org/10.1111/bcp.13803.

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Whyte, Michael P. "Heritable Metabolic and Dysplastic Bone Diseases." Endocrinology and Metabolism Clinics of North America 19, no. 1 (1990): 133–73. http://dx.doi.org/10.1016/s0889-8529(18)30342-6.

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Lipkin, Edward W. "METABOLIC BONE DISEASE IN GUT DISEASES." Gastroenterology Clinics of North America 27, no. 2 (1998): 513–23. http://dx.doi.org/10.1016/s0889-8553(05)70016-9.

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Parkman, Robertson, and Gay Crooks. "BONE MARROW TRANSPLANTATION FOR METABOLIC DISEASES." Immunology and Allergy Clinics of North America 16, no. 2 (1996): 429–38. http://dx.doi.org/10.1016/s0889-8561(05)70254-x.

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Wang, Yan, Fan Wu, and Yukun Li. "TRACP-5b and metabolic bone diseases." Bone 43 (October 2008): S91—S92. http://dx.doi.org/10.1016/j.bone.2008.07.124.

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Mikosch, Peter. "Bone scintigraphy for the diagnosis of metabolic bone diseases." Wiener Medizinische Wochenschrift 154, no. 5-6 (2004): 119–26. http://dx.doi.org/10.1007/s10354-004-0053-4.

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Aggarwal, Juhi, Mansi Modi, RajNarayan Gupta, and EramHussain Pasha. "Utility of bone turnover markers in metabolic bone diseases." Santosh University Journal of Health Sciences 9, no. 1 (2023): 48. http://dx.doi.org/10.4103/sujhs.sujhs_38_23.

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Testini, Valentina, Laura Eusebi, Umberto Tupputi, Francesca Anna Carpagnano, Francesco Bartelli, and Giuseppe Guglielmi. "Metabolic Bone Diseases in the Pediatric Population." Seminars in Musculoskeletal Radiology 25, no. 01 (2021): 094–104. http://dx.doi.org/10.1055/s-0040-1722566.

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AbstractBone plays an important role in regulating mineral balance in response to physiologic needs. In addition, bone is subject to a continuous remodeling process to maintain healthy bone mass and growth. Metabolic bone diseases are a heterogeneous group of diseases caused by abnormalities of bone mass, mineral structure homeostasis, bone turnover, or bone growth. In pediatrics, several significant advances have been made in recent years in the diagnosis of metabolic bone diseases (e.g., osteogenesis imperfecta, hyperparathyroidism, rickets, renal osteodystrophy, pediatric osteoporosis, and

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Bonora, Massimo, Simone Patergnani, Daniela Ramaccini, et al. "Physiopathology of the Permeability Transition Pore: Molecular Mechanisms in Human Pathology." Biomolecules 10, no. 7 (2020): 998. http://dx.doi.org/10.3390/biom10070998.

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Mitochondrial permeability transition (MPT) is the sudden loss in the permeability of the inner mitochondrial membrane (IMM) to low-molecular-weight solutes. Due to osmotic forces, MPT is paralleled by a massive influx of water into the mitochondrial matrix, eventually leading to the structural collapse of the organelle. Thus, MPT can initiate outer-mitochondrial-membrane permeabilization (MOMP), promoting the activation of the apoptotic caspase cascade and caspase-independent cell-death mechanisms. The induction of MPT is mostly dependent on mitochondrial reactive oxygen species (ROS) and Ca2

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Zhang, Rubin. "Metabolic bone diseases in kidney transplant recipients." World Journal of Nephrology 1, no. 5 (2012): 127. http://dx.doi.org/10.5527/wjn.v1.i5.127.

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Moya-Angeler, Joaquin, Joseph M. Lane, and Jose A. Rodriguez. "Metabolic Bone Diseases and Total Hip Arthroplasty." Journal of the American Academy of Orthopaedic Surgeons 25, no. 11 (2017): 725–35. http://dx.doi.org/10.5435/jaaos-d-16-00067.

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Laxer, Eric B., and Max Aebi. "Metabolic bone diseases of the spinal column." Current Opinion in Orthopaedics 5, no. 2 (1994): 58–68. http://dx.doi.org/10.1097/00001433-199404000-00010.

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Di Donna, Antonio, Salvatore Masala, Gianluca Muto, Stefano Marcia, Flavio Giordano, and Mario Muto. "Metabolic Bone Diseases: Recommendations for Interventional Radiology." Seminars in Musculoskeletal Radiology 28, no. 05 (2024): 641–50. http://dx.doi.org/10.1055/s-0044-1788580.

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AbstractMetabolic bone diseases (MBDs), including osteoporosis, osteomalacia, and Paget's disease, represent a significant challenge in medical imaging and intervention. Interventional radiologists play a pivotal role in managing these conditions, due to the crucial role of detailed and precise imaging in diagnosing and treating MBDs. This article offers comprehensive recommendations aimed at optimizing interventional radiologic practices for patients with MBDs, highlighting the importance of using advanced imaging modalities, including dual-energy X-ray absorptiometry and magnetic resonance i

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Wendlová, Jaroslava. "Metabolic bone diseases: basic and clinical aspects." Wiener Medizinische Wochenschrift 157, no. 23-24 (2007): 581. http://dx.doi.org/10.1007/s10354-007-0488-5.

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Hamdy, Neveen A. T. "Role of bisphosphonates in metabolic bone diseases." Trends in Endocrinology & Metabolism 4, no. 1 (1993): 19–25. http://dx.doi.org/10.1016/1043-2760(93)90059-n.

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Torri, Francesca, Piervito Lopriore, Vincenzo Montano, Gabriele Siciliano, Michelangelo Mancuso, and Giulia Ricci. "Pathophysiology and Management of Fatigue in Neuromuscular Diseases." International Journal of Molecular Sciences 24, no. 5 (2023): 5005. http://dx.doi.org/10.3390/ijms24055005.

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Fatigue is a major determinant of quality of life and motor function in patients affected by several neuromuscular diseases, each of them characterized by a peculiar physiopathology and the involvement of numerous interplaying factors. This narrative review aims to provide an overview on the pathophysiology of fatigue at a biochemical and molecular level with regard to muscular dystrophies, metabolic myopathies, and primary mitochondrial disorders with a focus on mitochondrial myopathies and spinal muscular atrophy, which, although fulfilling the definition of rare diseases, as a group represe

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Losasso, Marina Ribas, Maria Luiza Cesto Parussolo, Antony Oliveira Silva, et al. "Unraveling the Metabolic Pathways Between Metabolic-Associated Fatty Liver Disease (MAFLD) and Sarcopenia." International Journal of Molecular Sciences 26, no. 10 (2025): 4673. https://doi.org/10.3390/ijms26104673.

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Metabolic-Associated Fatty Liver Disease (MAFLD) is a public health concern that is constantly expanding, with a fast-growing prevalence, and it affects about a quarter of the world’s population. This condition is a significant risk factor for cardiovascular, hepatic, and oncologic diseases, such as hypertension, hepatoma, and atherosclerosis. Sarcopenia was long considered to be an aging-related syndrome, but today, it is acknowledged to be secondarily related to chronic diseases such as metabolic syndrome, cardiovascular conditions, and liver diseases, among other comorbidities associated wi

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Rey-Reñones, Cristina, Jose Miguel Baena-Díez, Isabel Aguilar-Palacio, Cristina Miquel, and María Grau. "Type 2 Diabetes Mellitus and Cancer: Epidemiology, Physiopathology and Prevention." Biomedicines 9, no. 10 (2021): 1429. http://dx.doi.org/10.3390/biomedicines9101429.

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Individuals with type 2 diabetes mellitus are at greater risk of developing cancer and of dying from it. Both diseases are age-related, contributing to the impact of population aging on the long-term sustainability of health care systems in European Union countries. The purpose of this narrative review was to describe, from epidemiological, pathophysiological and preventive perspectives, the links between type 2 diabetes mellitus and the most prevalent cancers in these patients. Multiple metabolic abnormalities that may occur in type 2 diabetes mellitus, particularly obesity, could explain the

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Cordani, Marco, Miguel Sánchez-Álvarez, Raffaele Strippoli, Alexandr V. Bazhin, and Massimo Donadelli. "Sestrins at the Interface of ROS Control and Autophagy Regulation in Health and Disease." Oxidative Medicine and Cellular Longevity 2019 (May 7, 2019): 1–11. http://dx.doi.org/10.1155/2019/1283075.

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Reactive oxygen species (ROS) and autophagy are two highly complex and interrelated components of cell physiopathology, but our understanding of their integration and their contribution to cell homeostasis and disease is still limited. Sestrins (SESNs) belong to a family of highly conserved stress-inducible proteins that orchestrate antioxidant and autophagy-regulating functions protecting cells from various noxious stimuli, including DNA damage, oxidative stress, hypoxia, and metabolic stress. They are also relevant modulators of metabolism as positive regulators of the key energy sensor AMP-

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Oton-Gonzalez, Lucia, Chiara Mazziotta, Maria Rosa Iaquinta, et al. "Genetics and Epigenetics of Bone Remodeling and Metabolic Bone Diseases." International Journal of Molecular Sciences 23, no. 3 (2022): 1500. http://dx.doi.org/10.3390/ijms23031500.

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Bone metabolism consists of a balance between bone formation and bone resorption, which is mediated by osteoblast and osteoclast activity, respectively. In order to ensure bone plasticity, the bone remodeling process needs to function properly. Mesenchymal stem cells differentiate into the osteoblast lineage by activating different signaling pathways, including transforming growth factor β (TGF-β)/bone morphogenic protein (BMP) and the Wingless/Int-1 (Wnt)/β-catenin pathways. Recent data indicate that bone remodeling processes are also epigenetically regulated by DNA methylation, histone post-

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Undale, Anita H., Jennifer J. Westendorf, Michael J. Yaszemski, and Sundeep Khosla. "Mesenchymal Stem Cells for Bone Repair and Metabolic Bone Diseases." Mayo Clinic Proceedings 84, no. 10 (2009): 893–902. http://dx.doi.org/10.4065/84.10.893.

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Sollmann, Nico, Michael Dieckmeyer, Julio Carballido-Gamio, et al. "Magnetic Resonance Assessment of Bone Quality in Metabolic Bone Diseases." Seminars in Musculoskeletal Radiology 28, no. 05 (2024): 576–93. http://dx.doi.org/10.1055/s-0044-1788693.

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AbstractMetabolic bone diseases (MBDs) are a diverse group of diseases, affecting the mass or structure of bones and leading to reduced bone quality. Parameters representing different aspects of bone health can be obtained from various magnetic resonance imaging (MRI) methods such as proton MR spectroscopy, as well as chemical shift encoding-based water-fat imaging, that have been frequently applied to study bone marrow in particular. Furthermore, T2* mapping and high-resolution trabecular bone imaging have been implemented to study bone microstructure. In addition, quantitative susceptibility

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Cook, Gary J. R., Gopinath Gnanasegaran, and Sue Chua. "Miscellaneous Indications in Bone Scintigraphy: Metabolic Bone Diseases and Malignant Bone Tumors." Seminars in Nuclear Medicine 40, no. 1 (2010): 52–61. http://dx.doi.org/10.1053/j.semnuclmed.2009.08.002.

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Sousa, Patrícia, and Mariana Verdelho Machado. "MAFLD under the lens: the role of gut microbiota." Metabolism and Target Organ Damage 2, no. 4 (2022): 14. http://dx.doi.org/10.20517/mtod.2022.15.

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Obesity, the metabolic syndrome, and metabolic dysfunction-associated fatty liver disease (MAFLD) can be portrayed as transmissible diseases. Indeed, they can be induced, in animal models, by cohabitation or by transplantation of fecal microbiota from other animals or humans with those diseases. As such, to get a 10,000-foot view, we need to see under the lens the microbes that populate our gut. Gut microbiota participates in the harvesting of energy from nutrients, it allows the digestion of otherwise indigestible nutrients such as fibers, and it also produces short chain fatty acids and some

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Shiraki, M. "Recent advances in metabolic bone diseases of the elderly. Calcium requirement and bone metabolic marker." Nippon Ronen Igakkai Zasshi. Japanese Journal of Geriatrics 29, no. 2 (1992): 101–4. http://dx.doi.org/10.3143/geriatrics.29.101.

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Arantes, Henrique Pierotti, André Gonçalves da Silva, and Marise Lazaretti-Castro. "Bisphosphonates in the treatment of metabolic bone diseases." Arquivos Brasileiros de Endocrinologia & Metabologia 54, no. 2 (2010): 206–12. http://dx.doi.org/10.1590/s0004-27302010000200017.

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Osteoporosis is a disease characterized by low bone mass associated with the deterioration of microarchitecture, due to an imbalance either in high bone resorption or low bone formation or in both, leading to a high risk of fractures. Bisphosphonates are medications which reduce the ability of osteoclasts to induce bone resorption and consequently improve the balance between resorption and formation. There are bisphosphonates approved for the prevention and treatment of osteoporosis. Administration can be oral (daily, weekly or monthly) or intravenous (quarterly or yearly). These medications a

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