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Gotowe bibliografie tematyczne / Bone resorption / Książki

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Książki na temat „Bone resorption”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Bronner, Felix, Mary C. Farach-Carson, and Janet Rubin, eds. Bone Resorption. Springer-Verlag, 2005. http://dx.doi.org/10.1007/b136184.

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2

Felix, Bronner, Farach-Carson Mary C. 1958-, and Rubin Janet, eds. Bone resorption. Springer, 2005.

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3

Harvey, Wilson. Prostaglandins in bone resorption. CRC Press, 1988.

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4

Pierce, Angela Mary. Cellular mechanisms in bone and tooth resorption: Morphological studies in rats and monkeys. Kongl. Carolinska Medico Chirurgiska Institutet, 1988.

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5

Brown, R. James. Histological and compositional responses of bone to immobilization and other experimental conditions: Semi-annual report. Institute of Chemical Biology, University of San Francisco, 1985.

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6

Thamsborg, Gorm. Effect of nasal salmon calcitonin on calcium and bone metabolism. Lægeforeningens Forlag, 1999.

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7

Maria, Bijvoet Olav Leonardus, ed. Bisphosphonate on bones. Elsevier, 1995.

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8

Arun, Shanbhag, Rubash Harry E, and Jacobs Joshua J. 1956-, eds. Joint replacement and bone resorption: Pathology, biomaterials, and clinical practice. Taylor & Francis, 2005.

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9

Åkesson, Louise. Panoramic radiography in the assessment of the marginal bone level. Department of Oral Radiology, Faculty of Odontology, Lund University, 1991.

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10

Ljunggren, Östen. Experimental studies on bone resorption: Effects of parathyroid hormone and vitamin D. Univ., 1993.

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11

Fleisch, Herbert. Bisphosphonates in bone disease: From the laboratory to the patient. H. Fleish, 1993.

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12

Ransjö, Maria. Regulation of bone resorption by the adenylate cyclase-cyclic AMP system: A biochemical study on mouse calvarial bones and isolated bone cells. University of Umeå, 1988.

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13

Loomer, Peter Michael. The direct effects of Porphyromonas Gingivalis 2561 on bone formation and mineral resorption in vitro. University of Toronto, Faculty of Dentistry], 1997.

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14

D, Rubens R., and European Conference on Clinical Oncology (5th : 1989 : London, England), eds. The Management of bone metastases and hypercalcaemia by osteoclast inhibition: An international symposium held during the 5th European Conference on Clinical Oncology (ECCO 5), London, September 1989. Hogrefe & Huber, 1990.

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15

Karhukorpi, Eeva-Kaisa. The mode of action of osteoclasts in bone resorption: An ultrastructural study of enzymes possibly involved. University of Oulu], Department of Anatomy, 1991.

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16

Clemens Waltherus Gerardus Maria Löwik. The mechanism of action of parathyroid hormone in bone resorption: The role of intracellular calcium and cAMP. [s.n.], 1987.

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17

Congress, European Society for Medical Oncology. Disodium pamidronate (APD) in the treatment of malignancy-related disorders: An international symposium held during the 13th Congress of the European Society for Medical Oncology (ESMO), Lugano, Switzerland, October 1988. Hogrefe & Huber, 1990.

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18

Meyer, James L. I loved a boy: Confessions of a Roman Catholic priest. Van Antwerp and Beale Publishers, 2007.

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19

Meyer, James L. I loved a boy: Confessions of a Roman Catholic priest. Van Antwerp and Beale Publishers, 2007.

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20

Peter, Burckhardt, ed. Disodium pamidronate (APD) in the treatment of malignancy-related disorders. Huber, 1989.

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21

Shorey, Seema. Differences in the degree to which osteoclasts from different parts of the skeleton employ cathepsin K and matrix metalloproteinases for bone resorption. National Library of Canada, 2002.

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22

Tai, Victoria. The effects of leukotriene Bb4s on osteoclast formation and osteoclastic bone resorption and the role of osteoblastic cells in these processes. National Library of Canada = Bibliothèque nationale du Canada, 1999.

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23

Mundy, Gregory R. Bone remodeling and its disorders. Martin Dunitz, 1994.

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24

Bronner, Felix, Janet Rubin, and Mary C. Farach-Carson. Bone Resorption. Springer London, Limited, 2006.

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25

Bronner, Felix, Janet Rubin, and Mary C. Farach-Carson. Bone Resorption. Springer London, Limited, 2010.

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26

Harvey, Wilson, and Alan Bennett. Prostaglandins in Bone Resorption. CRC Press, 2020. http://dx.doi.org/10.1201/9780429279195.

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27

Harvey, Wilson, and Alan Bennett. Prostaglandins in Bone Resorption. Taylor & Francis Group, 2020.

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28

Prostaglandins in Bone Resorption. Taylor & Francis Group, 2020.

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29

Harvey, Wilson, and Alan Bennett. Prostaglandins in Bone Resorption. Taylor & Francis Group, 2020.

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30

Harvey, Wilson, and Alan Bennett. Prostaglandins in Bone Resorption. Taylor & Francis Group, 2020.

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31

Harvey, Wilson, and Alan Bennett. Prostaglandins in Bone Resorption. Taylor & Francis Group, 2019.

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32

Osteoblasts: Morphology, Functions and Clinical Implications. Nova Science Pub Inc, 2013.

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33

Shanbhag, Arun, Harry E. Rubash, and Joshua J. Jacobs, eds. Joint Replacement and Bone Resorption. CRC Press, 2005. http://dx.doi.org/10.1201/b14138.

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34

Rubash, Harry E., Arun Shanbhag, and Joshua J. Jacobs. Joint Replacement and Bone Resorption. Taylor & Francis Group, 2019.

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35

Bone resorption, metastasis, and diphosphonates. Raven Press, 1985.

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36

(Editor), Kurt W. Brunner, Herbert Fleisch (Editor), and Hans-Jörg Senn (Editor), eds. Bisphosphonates and Tumor-Osteolysis (Recent Results in Cancer Research). Springer, 1995.

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37

Anderson, Paul, Borje Edgar Christopher Nordin, and Howard Arthur Morris. Physiological Basis of Metabolic Bone Disease. Taylor & Francis Group, 2014.

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38

Anderson, Paul, Borje Edgar Christopher Nordin, and Howard Arthur Morris. Physiological Basis of Metabolic Bone Disease. Taylor & Francis Group, 2014.

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39

Anderson, Paul. Physiological Basis of Metabolic Bone Disease. Taylor & Francis Group, 2014.

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40

Anderson, Paul. Physiological Basis of Metabolic Bone Disease. Taylor & Francis Group, 2017.

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41

Rubash, Harry E., Arun Shanbhag, and Joshua J. Jacobs. Joint Replacement and Bone Resorption: Pathology, Biomaterials and Clinical Practice. Taylor & Francis Group, 2005.

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42

(Editor), Arun Shanbhag, Harry E. Rubash (Editor), and Joshua J. Jacobs (Editor), eds. Joint Replacement and Bone Resorption: Pathology, Biomaterials and Clinical Practice. Informa Healthcare, 2005.

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43

Rubash, Harry E., Arun Shanbhag, and Joshua J. Jacobs. Joint Replacement and Bone Resorption: Pathology, Biomaterials and Clinical Practice. Taylor & Francis Group, 2005.

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44

Mansfield, Ian David. The synthesis of novel agents which inhibit tumour-stimulated bone resorption. 1999.

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45

The relationship between bone mineral density and selected variables: An epidemiological perspective. 1990.

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46

Wordsworth, B. P. Skeletal dysplasias. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0150.

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Streszczenie:

Bone is metabolically active throughout life and metabolic disturbances may have wide-ranging consequences that are not restricted to altering its mechanics. The study of some genetic bone diseases has already provided remarkable insights into the normal regulation of bone metabolism. Skeletal dysplasias are developmental disorders of the chondro-osseous tissues commonly resulting in short stature, which is often disproportionate. The underlying mutations are often in the structural genes encoding components of the matrix but may also involve growth factors or cell signalling. In contrast, the

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47

Javaid, Kassim. Paget’s disease of bone. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0274.

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Paget’s disease of bone is an uncommon bone disorder with increased bone resorption and disorganized bone formation of woven bone. Its cause is unclear; there is a clear genetic component but additional environmental factors are important, given the reduction in severity and prevalence in the UK. Paget’s disease is usually asymptomatic and detected by an unexplained raised alkaline phosphatase on routine biochemistry. Symptoms include focal bone pain, including headache. Other symptoms include bone deformity and complications such as fracture and nerve conduction. Paget’s disease can sometimes

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48

Ralston, Stuart H. Paget’s disease of bone. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0144.

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Paget's disease of bone (PDB) affects up to 1% of people of European origin aged 55 years and above. It is characterized by focal abnormalities of bone remodelling which disrupt normal bone architecture, leading to expansion and reduced mechanical strength of affected bones. This can lead to various complications including deformity, fracture, nerve compression syndromes, and osteoarthritis, although many patients are asymptomatic. Genetic factors play a key role in the pathogenesis of PDB. This seems to be mediated by a combination of rare genetic variants which cause familial forms of the di

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49

Ralston, Stuart H. Paget’s disease of bone. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0144_update_001.

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Paget’s disease of bone (PDB) affects up to 1% of people of European origin aged 55 years and above. It is characterized by focal abnormalities of bone remodelling which disrupt normal bone architecture, leading to expansion and reduced mechanical strength of affected bones. This can lead to various complications including deformity, fracture, nerve compression syndromes, and osteoarthritis, although many patients are asymptomatic. Genetic factors play a key role in the pathogenesis of PDB. This seems to be mediated by a combination of rare genetic variants which cause familial forms of the di

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50

Wheeler, Donna L. The short- and long-term effects of methotrexate on the rat skeleton. 1993.

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