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Rozprawy doktorskie na temat „Bones – Growth”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lutego 2023

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1

Yadav, Priti. "Modelling loading and growth of long bones Modelling loading and growth of long bones." Licentiate thesis, KTH, Biomekanik, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-177913.

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The long bones grow by the process of endochondral ossification, which occurs at the growth plate. This process is regulated by biological factors and mechanical factors. The biological factors which contribute to endochondral ossification process are genes, hormones, nutrients etc. The mechanical factor is the load acting on the bone. The major forces on the bone are due to joint contact load and muscle forces, which induce stresses in the bone. Carter and Wong proposed in a theory that cyclic or intermittent octahedral shear stress promotes the bone growth and cyclic or intermittent hydrosta

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2

Chim, Shek Man. "Identification and characterization of novel secreted factors involved in bone remodeling." University of Western Australia. School of Surgery, 2009. http://theses.library.uwa.edu.au/adt-WU2010.0110.

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[Truncated abstract] Bone remodeling is an important process to maintain mechanical integrity. It is accomplished by two important steps, bone resorption followed by new bone formation. Osteoclasts and osteoblasts are the principal cells in bone resorption and bone formation, respectively. A multitude of local and systemic factors regulates this process by controlling the cellular activities in bone remodeling compartments (BRC). An imbalance of osteoblastic bone formation and osteoclastic bone destruction will result in the development of skeletal diseases. Recent studies suggested that angio

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3

Ma, Li, and 马丽. "The influence of nicotine on angiogenesis and osteogenesis in bone regeneration." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B41508440.

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Oest, Megan Elizabeth. "Dual Osteogenic and Angiogenic Growth Factor Delivery as a Treatment for Segmental Bone Defects." Diss., Georgia Institute of Technology, 2007. http://hdl.handle.net/1853/16264.

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A new model of a critically-sized segmental femoral bone defect in rats was developed to enable in vivo imaging and facilitate post-mortem mechanical testing of samples. The critically-sized nature of the model was assessed and confirmed. The efficacy of sustained co-delivery of osteogenic (BMP-2 and TGF- Ò3) and angiogenic (VEGF) growth factors in promoting functional bone repair was assessed. Effects of scaffold modification in terms of geometry and composition were evaluated. The results indicated that co-delivery of BMP-2 and TGF- Ò3 resulted in a dose-dependent improvement in functional b

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5

關健明 and Kin-ming Kwan. "Defining the function of type X collagen in skeletal development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 1997. http://hub.hku.hk/bib/B31237162.

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6

Foster, Bruce Kristian. "Epiphyseal plate repair using fat interposition to reverse physeal deformity : an experimental study." Title page, contents and summary only, 1989. http://web4.library.adelaide.edu.au/theses/09MD/09mdf754.pdf.

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Bibliography: leaves 169-197. Hypothesises that the physis has an internal mechanism of repair to restore physeal function. Aims to establish a defined degree of deformity by partial growth plate excision, then to examine different methods of reversal of such deformity to observe the process of growth plate repair. A secondary aim was to define the percentage of physis that could be resected yet still enable reversal of deformity.

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7

Chayanupatkul, Atinooch. "Bone formation in the temporomandibular joint in response to forward mandibular positioning." Thesis, Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25598776.

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8

Cha, Ming Chuan 1955. "The effect of zinc deficiency on the growth promoting actions of growth hormone and insulin-like growth factor-I /." Thesis, McGill University, 1994. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=55484.

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The effect of zinc deficiency on the growth promoting effect of circulating IGF-I and the direct growth effect of GH on long bone growth were investigated. Food intake was decreased by lack of zinc in the diet. Tissue zinc content and plasma alkaline phosphatase activity were reduced by zinc deficiency. Systemic administration of human IGF-I increased the body weight, tail length and tibia epiphyseal cartilage width of control animals. This somatogenic action was impaired by zinc deficiency, as evidenced by continued weight loss, no increase in tail length and decreased tibial epiphyseal carti

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9

Dai, Zhijie, and 戴志洁. "The role of sodium/myo-inositol cotransporter 1 and myo-inositol in osteogenesis and bone formation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43783533.

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10

Dahlgren, Emma. "Effects of Different Load Magnitudes on Longitudinal Growth of Immature Bones." Thesis, KTH, Skolan för kemi, bioteknologi och hälsa (CBH), 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:kth:diva-230885.

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In vivo studies of mechanical loading on bone have suggested that load magnitude is one of the parameters that play a vital role in bone adaptation. This study examined how longitudinal growth of immature rat metatarsals is affected by different load magnitudes. The main hypotheses were that the longitudinal growth of immature bone would decrease with increased compressive load magnitude, and that the longitudinal growth would be more decelerated the higher the load mag- nitude. The three middle metatarsal bones in the back paws of 19-20 days old Sprague-Dawley rat fetuses were extracted. Meta

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11

Wong, Hoi-leong Xavier, and 王凱亮. "The functional crosstalk between MT1-MMP and ADAMs in craniofacial & vascular development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/197072.

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12

Saxon, Leanne, and mikewood@deakin edu au. "The role of exercise in the development of bone strength during growth." Deakin University. School of Health Sciences, 2002. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051125.095337.

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Exercise during growth may increase peak bone mass; if the benefits are maintained it may reduce the risk of fracture later in life (1). It is hypothesised that exercise will preferentially enhance bone formation on the surface of cortical bone that is undergoing bone modeling at the time (2). Therefore, exercise may increase bone mass accrual on the outer periosteal surface during the pre- and peri-pubertal years, and on the inner endocortical surface during puberty (3). An increase in bone formation on the periosteal surface is, however, more effective for increasing bone strength than medul

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13

Wong, Wing-Kit Ricky, and 黃永傑. "Bone induction using Simvastatin and Gusuibu." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B31246126.

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14

Brooker, Molly J. "The effect of acute exercise on bone metabolism in the pre-pubertal child." Virtual Press, 2000. http://liblink.bsu.edu/uhtbin/catkey/1164852.

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Exercise is known to have a long-term benefit on bone mass in children, but little is known about the underlying mechanisms. The purpose of this investigation was to determine the acute effect of exercise on bone metabolism in pre-pubertal children. Biochemical markers of bone formation were measured in 4 male and 4 female children, 8 to 11 years of age. Each subject performed 50 vertical jumps. Serum osteocalcin and C-telopeptide of type I collagen (CTx), were determined prior to exercise and at 24 and 72 hours post exercise as indicators of bone formation and bone resorption. Osteocalcin con

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15

Liu, Jin. "Increased CKIP-1 suppresses Smad-dependent BMP signaling to inhibit bone formation during aging." HKBU Institutional Repository, 2016. https://repository.hkbu.edu.hk/etd_oa/327.

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Emerging evidence indicates that the dysregulation of protein ubiquitination plays a crucial role in aging-associated diseases. Smad-dependent canonical BMP signaling pathway is indispensable for osteoblastic bone formation, which could be disrupted by the ubiquitination and subsequent proteasomal degradation of Smad1/5, the key molecules for BMP signaling transduction. However, whether the dysregulation of Smad1/5 ubiquitination and disrupted BMP signaling pathway are responsible for the age-related bone formation reduction is still underexplored. Casein kinase-2 interacting protein-1 (CKIP-1

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16

Zheng, Liwu, and 鄭立武. "Biochemical modulation of mandibular distraction osteogenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B31246308.

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17

Alfonso, Durruty Marta Pilar. "Biosignificance of Harris lines as stress markers in relation to moderate undernutrition and bone growth velocity a New Zealand white rabbit model for the study of bone growth /." Diss., Online access via UMI:, 2008.

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18

Gan, Huiyan, and 甘慧妍. "Understanding the role of KIF5B in long bone development and chondrocyte cytokinesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hdl.handle.net/10722/211554.

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Kinesins are motor proteins responsible for the anterograde transport on microtubules. Kinesin-1 is the first characterized kinesin, and it consists of two heavy chains and two light chains. KIF5B is a form of Kinesin-1 heavy chains that is ubiquitously expressed in mammals. The head domain of KIF5B is responsible for ATP-dependent mechanical movement along microtubules, while the tail region is well-known for its interaction with cell specific cargos. Recent studies reveal a second microtubule binding site in the tail, suggesting special functions of KIF5B in microtubule sliding and bundling.

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19

Gluck, Beth. "The Effects of Physical Activity on Bone Density in Adolescent Females." Fogler Library, University of Maine, 2004. http://www.library.umaine.edu/theses/pdf/GluckB2004.pdf.

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20

Blostein, Ashley C. "Effects of running on hormonal growth factors." Virtual Press, 1993. http://liblink.bsu.edu/uhtbin/catkey/865946.

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To determine the influence of running on certain blood-born parameters that are involved in bone metabolism, serum levels of calcium, alkaline phosphatase (ALP, a marker of bone formation), growth hormone (hGH), and parathyroid hormone (PTH), were analyzed in 10 male subjects following a 40 min. run at 70% VO2max. Each trial was preceeded by 1 day of inactivity, a 8-12 hr. fast, and drawing of a baseline blood sample by venipuncture. All other blood samples were taken via an indwelling catheter which was inserted in an antecubital vein immediately following the completion of the exercise bout.

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21

Zhong, Ming. "Apoptotic signaling pathways in mammalian growth plate chondrocytes." Diss., Georgia Institute of Technology, 2010. http://hdl.handle.net/1853/33993.

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The growth plate resting zone consists of hyaline-like chondrocytes disbursed in a proteoglycan rich extracellular matrix. These cells give rise to the columns of the growth zone, consisting of progressively hypertrophic cells. Proliferation of resting zone chondrocytes induced by systemic and local stimuli is the driving force of longitudinal growth of long bones. Therefore, homeostasis of this cell population has great importance. Although the regulation of proliferation and differentiation of these cells has been well studied, little is known about the regulation of their apoptosis. We

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22

Kar, Archana. "Hydroxyapatite deposition onto nanoporous TiO2 and assessment of bone cell growth and proliferation." abstract and full text PDF (free order & download UNR users only), 2007. http://0-gateway.proquest.com.innopac.library.unr.edu/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1447622.

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23

Mohamad, Yusof Loqman. "Longitudinal growth of mammalian bones : a possible role for membrane transporters in mediating chondrocyte hypertrophy." Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/6481.

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Long bone lengthening occurs at the growth plate (GP) by well-regulated chondrocyte proliferation, hypertrophy and terminal matrix deposition. GP chondrocyte (GPC) hypertrophy has been implicated to be the main determinant of bone growth rate; however the mechanism is poorly understood. The work of this thesis examined some of the cellular process that drives the chondrocyte swelling or hypertrophy particularly in a mammalian post natal GPs using living in situ GPC and fixed GP tissues. Confocal scanning microscopy (CLSM) was used to determine living in situ GPC volume and dimension changes in

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24

Moore, Alison Jane. "Quantitative histomorphometric analysis of the bone growth plate in infancy : a comparative study between SIDS and normal subjects /." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09MSM/09msmm821.pdf.

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25

Serrat, Maria A. "Environmentally-determined tissue temperature modulates extremity growth in mammals a potential comprehensive explanation of Allen's Rule /." [Kent, Ohio] : Kent State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=kent1185486409.

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Thesis (Ph.D.)--Kent State University, 2007.<br>Title from PDF t.p. (viewed Mar. 5, 2009). Advisor: C. Owen Lovejoy. Keywords: temperature, bone growth, Allen's Rule, skeletal morphology, limb proportions, environmental effects on bone growth. Includes bibliographical references (p. 157-176).

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26

Zhu, Guixia, and 朱貴霞. "Study of the function of Kinesin-1 (KIF5B) in long bone development." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B41757919.

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27

Zierath, Juleen R. "Bone mineral content in laboratory rats following swim and run training." Virtual Press, 1986. http://liblink.bsu.edu/uhtbin/catkey/472942.

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Increased bone density has been observed following physical training. However, it is not known whether the mechanical forces of muscular contraction, gravitational pull, or a combination of these forces are required to cause this adaptation. Therefore, the purpose of this study was to determine which mechanical force, muscular contraction or gravitational pull, offered the greatest contribution to increased bone mineral content observed following either swim or run training. METHODS: Female Wistar rats were randomly assigned to one of three groups: 1) Sedentary Control (SC; n = 12), 2) Run Tra

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28

Li, Gang Gang. "Biological studies of distraction osteogenesis." Thesis, University of Oxford, 1997. http://ora.ox.ac.uk/objects/uuid:d2976713-d0f6-439a-a70c-9183d44cff81.

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Tsai, Ming-ju Marjorie. "Replicating mesenchymal cells in the condyle in response to normal growth and mandibular protrusion." Hong Kong : University of Hong Kong, 2002. http://sunzi.lib.hku.hk/hkuto/record.jsp?B25575995.

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30

Valverde, Franco Gladys 1972. "The role of fibroblast growth factor receptor 3 in post-natal cartilage and bone metabolism /." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=115917.

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FGFR 3 is one of a family of four high affinity receptors for FGF ligands. Activating mutations in FGFR 3 result in skeletal dysplasias that vary in severity from undetectable to neonatal lethal. Mice with congenital deficiency of FGFR3 develop severe kyphosis and skeletal overgrowth. FGFR3 is also expressed in calvarial pre-osteoblasts, osteoblast and articular chondrocytes, although it biological role in these cells remains undefined. By changing the genetic background of the Fgfr3-/- mice we were able to extend their lifespan and examine its impact on post-natal skeletal growth. To investig

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31

Langeveldt, Carmen Ronel. "Alternative insulin mitogenic signaling pathways in immature osteoblast cell lines." Thesis, Stellenbosch : Stellenbosch University, 2002. http://hdl.handle.net/10019.1/52646.

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Thesis (MSc)--University of Stellenbosch, 2002.<br>ENGLISH ABSTRACT: Insulin is a mitogen for many cells and commonly signals through the classical, mitogenic Raf- MEK-ERK or metabolic PB-kinase pathways. Insulin deficiency or type I diabetes causes severe osteopenia. Obese patients with type II diabetes or insulin resistance, a disease associated with defective insulin signaling pathways and high levels of circulating insulin, have increased or normal bone mineral density. The question of whether hyperinsul inemia preserves bone mass is frequently raised. However, there is still a lot o

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32

Duvall, Craig L. "The Role of osteopontin in postnatal vascular growth functional effects in ischemic limb collateral vessel formation and long bone fracture healing /." Available online, Georgia Institute of Technology, 2006, 2006. http://etd.gatech.edu/theses/available/etd-01102007-130423/.

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Thesis (Ph. D.)--Biomedical Engineering, Georgia Institute of Technology, 2007.<br>David Harrison, Committee Member ; Ravi Bellamkonda, Committee Member ; Larry McIntire, Committee Member ; Oskar Skrinjar, Committee Member ; W. Robert Taylor, Committee Chair ; Robert Guldberg, Committee Chair.

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33

陳卓榮 and Cheuk-wing Wilson Chan. "Molecular basis for increased bone formation in a mouse expressing mutant collagen X." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B31227132.

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34

曹凱韻 and Hoi-wan Tso. "Effects of phagocytosis of apoptotic cells by mesenchymal stem cells on osteogenesis and T cells responses." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39707520.

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35

Xu, Wei. "The impact of rhizoma chuanxiong in fetal bone development." HKBU Institutional Repository, 2016. https://repository.hkbu.edu.hk/etd_oa/253.

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Background and purpose: Rhizoma Chuanxiong (CX), the dry rhizome of Ligusticum chuanxiong Hort., is a commonly used Chinese herbal medicine to treat gynecological diseases. So far, more than 60 chemical components have been identified from CX such as volatile oils (ligustilide, etc.), phenolic acids (ferulic acid, etc.) and alkaloids (chuanxiongzine, etc.). These components in CX are the basis of its wide pharmacodynamic actions including estrogen-like, progesterone-like and anti-coagulant/anti- platelet effects. In our recent survey based on previous published clinical trials, CX was ranked a

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36

Dang, Lei. "Osteoblastic PLEKHO1 contributes to joint inflammation in rheumatoid arthritis." HKBU Institutional Repository, 2019. https://repository.hkbu.edu.hk/etd_oa/687.

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Background: Osteoblasts participating in the inflammation regulation gradually obtain concerns. However, its role in joint inflammation of rheumatoid arthritis (RA) is largely unknown. Here, we investigated the role of osteoblastic pleckstrin homology domain-containing family O member 1 (PLEKHO1), a negative regulator of osteogenic lineage activity, in regulating joint inflammation in RA. Methods: The level of osteoblastic PLEKHO1 in RA patients and collagen-induced arthritis (CIA) mice was examined. The role of osteoblastic PLEKHO1 in joint inflammation was evaluated by a CIA mice model which

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37

蔡明汝 and Ming-ju Marjorie Tsai. "Replicating mesenchymal cells in the condyle in response to normal growth and mandibular protrusion." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31973127.

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Essman, Stephanie Christine. "Effects of ¹⁵³samarium-ethylenediaminetetramethylene phosphonate on physeal and articular cartilage in juvenile rabbits /." Free to MU Campus, others may purchase, 2003. http://wwwlib.umi.com/cr/mo/fullcit?p1418016.

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黃淑興 and Shu-hing Louise Wong. "Replicating mesenchymal cells in the glenoid fossa in response to mandibular advancement." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2002. http://hub.hku.hk/bib/B31973140.

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40

Xiong, Hui, and 熊暉. "Condylar adaptation to active mandibular forward positioning in non-growing rats." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B31374220.

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Poon, Chin-ho, and 潘展豪. "Pushing stem cells toward bone lineage through ultrasound stimulation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47849824.

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When human mesenchymal stem cells (hMSCs) are cultured inside a 3D collagen meshwork, they become a potential tissue engineering bone graft alternative. However, the in vitro osteogenesis rate of hMSCs is slow, leading to a low mineral deposition. To enhance the osteogenic differentiation of hMSCs, low intensity pulsed ultrasound (LIPUS) was employed as an external stumulus. The present study demonstrated the feasibility of employing daily LIPUS exposure for enhancing osteogenesis in vitro. Exposure of seven consecutive days LIPUS, each of 30 minutes duration, did not affect the cell viabi

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42

Monegue, James S. "EVALUATION OF THE EFFECTS OF VITAMIN K ON GROWTH PERFORMANCE AND BONE HEALTH IN SWINE." UKnowledge, 2013. http://uknowledge.uky.edu/animalsci_etds/26.

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The role of vitamin K in the blood clotting cascade has been well documented. Vitamin K has recently been implicated in improving bone health. The current studies were conducted to determine the effects of vitamin K in diets with and without mycotoxin contaminated corn on growth performance, bone characteristics, and related blood metabolites in pigs from weaning to market. Menadione sodium bisulfite complex (MSBC, 33% vitamin K) was chosen as the source of supplemental vitamin K because it is the most common form fed to swine. Vitamin K was tested at 0, 0.5, and 2.0 ppm in a corn-soybean meal

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43

Moreira, Alessandra Arnaud. ""Estudo da utilização clínica das proteínas ósseas morfogenéticas em cirurgia buco-maxilo-facial no Brasil"." Universidade de São Paulo, 2004. http://www.teses.usp.br/teses/disponiveis/23/23143/tde-16032005-161053/.

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A recuperação de partes deficientes do corpo humano por substitutos funcionais, tem suscitado questionamentos por parte de profissionais cirurgiões e pesquisadores da área de cirurgia e traumatologia Bucomaxilofacial. Na tentativa de recuperar o contorno anatômico natural e restaurar a função de áreas com deficiência de tecido ósseo, optou-se inicialmente pela utilização de enxertos ósseos autógenos. Apesar de suas inúmeras vantagens, o uso de enxertos autógenos na reconstrução da face apresenta certos inconvenientes, como a necessidade de hospitalização, intervenção em outra área do organismo

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44

Seo, Hwa-Seon. "The role of TGFß signaling in skeletal development." Thesis, Birmingham, Ala. : University of Alabama at Birmingham, 2008. https://www.mhsl.uab.edu/dt/2008p/seo.pdf.

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45

Burr, Laura Lynn. "Diet enrichment with arachidonic and docosahexaenoic acid during the lactation period attenuates the effects of intrauterine growth restriction from birth to maturity in the guinea pig and improves maternal bone mass." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=112384.

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Intrauterine growth restriction (IUGR) reduces bone mass by 10-30% and impairs arachidonic (AA) and docosahexaenoic (DHA) acid status in infants. Because AA and DHA enhance neonatal bone mass, the aim of this study was to determine the effects of dietary 0.5% AA and 0.2% DHA (w/w) prior to weaning on bone and growth. 40 guinea pigs were randomized to either a control (C) or low-protein diet (LP) during pregnancy and the C diet or the C diet with AA+DHA during lactation. Measurements included bone mass, metabolism, and strength, and erythrocyte lipid of sows and offspring from birth to 16 wk po

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46

Davey, Tamara. "Functional characterisation of a novel osteoclast-derived factor." University of Western Australia. School of Surgery and Pathology, 2008. http://theses.library.uwa.edu.au/adt-WU2008.0219.

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[Truncated abstract] Intracellular communication between osteoclasts and osteoblasts is imperative to maintain bone integrity. A myriad of molecules are responsible for regulating osteoblast and osteoclast activity. In particular, it is well documented that osteoblast-derived factors are crucial in directly controlling osteoclast formation and function. Since bone formation is coupled to bone resorption, it would be expected that osteoclasts also have some role in regulating the growth and function of osteoblast cells. However, despite extensive research upon osteoclast and osteoblast biology,

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47

Schwartz, Filho Humberto Osvaldo [UNESP]. "Osteogênese sobre titânio com nanotopografia." Universidade Estadual Paulista (UNESP), 2011. http://hdl.handle.net/11449/110660.

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Made available in DSpace on 2014-11-10T11:09:56Z (GMT). No. of bitstreams: 0 Previous issue date: 2011-09-19Bitstream added on 2014-11-10T11:57:36Z : No. of bitstreams: 1 000697140_20151015.pdf: 232164 bytes, checksum: 0bed7471a6786fe38e3164319d6deb3e (MD5) Bitstreams deleted on 2015-08-07T12:19:55Z: 000697140_20151015.pdf,. Added 1 bitstream(s) on 2015-08-07T12:20:58Z : No. of bitstreams: 1 000697140.pdf: 6167998 bytes, checksum: bf8732b598c57469944be2ab9eac1b0c (MD5)<br>Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)<br>Os objetivos deste estudo foram avaliar a influên

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Chan, Cheuk-wing Wilson, and 陳卓榮. "ER stress in the pathogenesis of osteochondrodysplasia." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43085192.

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Turner, Justine Marie. "Adolescent onset anorexia nervosa : a model for the effects of inadequate nutrition upon bone size and development." University of Western Australia. School of Paediatrics and Child Health, 2006. http://theses.library.uwa.edu.au/adt-WU2006.0131.

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Despite usual onset during adolescence the cause of low bone density in adolescent onset anorexia nervosa is incompletely understood. Puberty is known to be a crucial time for the development of peak bone mass, due principally to growth plate bone formation and modelling on preformed surfaces. This results in bone formation uncoupled from bone resorption leading to increased bone size due to increase in matrix and bone mineral content. It was hypothesized that low bone density in adolescent anorexia nervosa was caused by malnutrition during puberty suppressing normal bone matrix formation at a

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Mason, Shelley S. "Exploring Tissue Engineering: Vitamin D3 Influences on the Proliferation and Differentiation of an Engineered Osteoblast Precursor Cell Line During Early Bone Tissue Development." PDXScholar, 2013. https://pdxscholar.library.pdx.edu/open_access_etds/1000.

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Most of the load-bearing demand placed on the human body is transduced by skeletal tissue, and the capacity of the skeleton to articulate in various opposing directions is essential for body movement and locomotion. Consequently, cartilage and bone defects due to trauma, disease, and developmental abnormalities result in disabling pain and immobility for millions of people worldwide. A novel way of promoting cartilage and bone regeneration is through the incorporation of either primary cells or multipotent progenitor cells in a three-dimensional (3D) biomaterial scaffold, and/or the addition o

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