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Artykuły w czasopismach na temat „Brain damage”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Larsson, L. "BRAIN DAMAGE, BRAIN REPAIR." Brain 125, no. 12 (2002): 2785–86. http://dx.doi.org/10.1093/brain/awf266.

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2

Raisman, Geoffrey. "Brain Damage, Brain Repair." Journal of the Royal Society of Medicine 96, no. 5 (2003): 249–50. http://dx.doi.org/10.1177/014107680309600517.

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Lanham, Richard A. "Brain Damage, Brain Repair." Journal of Head Trauma Rehabilitation 17, no. 3 (2002): 270–72. http://dx.doi.org/10.1097/00001199-200206000-00012.

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4

Jellinger, K. A. "Brain Damage, Brain Repair." European Journal of Neurology 10, no. 3 (2003): 335. http://dx.doi.org/10.1046/j.1468-1331.2003.00557.x.

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5

Raisman, G. "Brain Damage, Brain Repair." JRSM 96, no. 5 (2003): 249–50. http://dx.doi.org/10.1258/jrsm.96.5.249.

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6

Toledo, C. A. B. "Brain Damage, Brain Repair." Journal of Chemical Neuroanatomy 27, no. 2 (2004): 139. http://dx.doi.org/10.1016/j.jchemneu.2004.01.001.

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7

Floyd, Pink. "Brain Damage." Academic Medicine 83, no. 8 (2008): 742. http://dx.doi.org/10.1097/acm.0b013e318181d965.

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8

Volpe, Joseph J., A. Ernest, and Jane G. Stein. "BRAIN DAMAGE." Pediatric Research 20, no. 10 (1986): 1024–25. http://dx.doi.org/10.1203/00006450-198610000-00039.

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9

Rothwell, Nancy J., and Giamal N. Luheshi. "Brain TNF: Damage limitation or damaged reputation?" Nature Medicine 2, no. 7 (1996): 746–47. http://dx.doi.org/10.1038/nm0796-746.

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10

Lakatos, Andras. "Brain Damage and Brain Repair." Neuropathology and Applied Neurobiology 27, no. 3 (2001): 252–53. http://dx.doi.org/10.1046/j.1365-2990.2001.00336-2.x.

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11

Antonis, Theofilidis. "The Hypothesis of Unexplained Brain Damage and Learning Difficulties." Neuroscience and Neurological Surgery 8, no. 5 (2021): 01–08. http://dx.doi.org/10.31579/2578-8868/165.

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Introduction: The term learning disabilities refers to a complex and multidimensional phenomenon that affects many thousands of students. Due to the rich symptomatology of learning difficulties and the increased differences between individuals, it has not been possible to analyze all cases exclusively from the perspective of the neuropsychological approach. Aim. The aim of this study was to present research conducted in the context of theories on the brain function of people with learning disabilities. Supporting the hypothesis of brain dysfunction. Methodology: Literature review was carried o

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12

Montagu, Ashley. "Sociogenetic Brain Damage." Developmental Medicine & Child Neurology 13, no. 5 (2008): 597–605. http://dx.doi.org/10.1111/j.1469-8749.1971.tb08323.x.

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13

S., L. "Ischemic brain damage." Neurology 41, no. 6 (1991): 953. http://dx.doi.org/10.1212/wnl.41.6.953.

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14

Riva, Daria, Silvana Franceschetti, Alessandra Erbetta, Giovanni Baranello, Silvia Esposito, and Sara Bulgheroni. "Congenital Brain Damage." Journal of Child Neurology 28, no. 4 (2012): 446–54. http://dx.doi.org/10.1177/0883073812447684.

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15

Auer, Roland N. "Hypoglycemic Brain Damage." Metabolic Brain Disease 19, no. 3/4 (2004): 169–75. http://dx.doi.org/10.1023/b:mebr.0000043967.78763.5b.

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16

RUTH, VINETA J., and KARL O. RAIVIO. "Perinatal Brain Damage." Obstetrical & Gynecological Survey 44, no. 4 (1989): 267–68. http://dx.doi.org/10.1097/00006254-198904000-00014.

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17

ROVNER, SOPHIE. "UNDOING BRAIN DAMAGE." Chemical & Engineering News 85, no. 7 (2007): 18. http://dx.doi.org/10.1021/cen-v085n007.p018a.

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18

Hinton, Geoffrey E., David C. Plaut, and Tim Shallice. "Simulating Brain Damage." Scientific American 269, no. 4 (1993): 76–82. http://dx.doi.org/10.1038/scientificamerican1093-76.

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19

Auer, Roland N. "Hypoglycemic brain damage." Forensic Science International 146, no. 2-3 (2004): 105–10. http://dx.doi.org/10.1016/j.forsciint.2004.08.001.

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20

Marchi, Nicola, Peter Rasmussen, Miranda Kapural, et al. "Peripheral markers of brain damage and blood-brain barrier dysfunction." Restorative Neurology and Neuroscience 21, no. 3-4 (2003): 109–21. https://doi.org/10.3233/rnn-2003-00231.

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Purpose: Occurrence of brain damage is frequently associated with abnormal blood-brain barrier (BBB) function. Two brain-specific proteins, S100β and neuron-specific enolase (NSE) are released systemically in a variety of neurological diseases, but S100β levels sometimes rise in the absence of neuronal damage, suggesting that S100β is a marker of BBB rather than neuronal damage. Methods: We measured both proteins in the serum of patients undergoing iatrogenic BBB disruption with intrarterial mannitol, followed by chemotherapy. Results: Serum S100β increased significantly after mannitol infusio

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21

Lv, Yuan, Ling-Ling Zhu, and Gui-Hua Shu. "Relationship between Blood Glucose Fluctuation and Brain Damage in the Hypoglycemia Neonates." American Journal of Perinatology 35, no. 10 (2018): 946–50. http://dx.doi.org/10.1055/s-0038-1626706.

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Objective To investigate the relationship between blood glucose fluctuation and brain damage in the hypoglycemia neonates. Study Design A retrospective study including all neonates hospitalized due to hypoglycemia from September 2013 to August 2016 was performed. All the 58 hypoglycemia infants were divided into two groups—the brain-damaged group and the nonbrain-damaged group, according to head magnetic resonance imaging and/or amplitude-integrated electroencephalogram. Relationship between glucose variability and brain damage and whether these variation indexes could act as early indicators

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22

Wasterlain, Claude G., and Yukiyoshi Shirasaka. "Seizures, brain damage and brain development." Brain and Development 16, no. 4 (1994): 279–95. http://dx.doi.org/10.1016/0387-7604(94)90025-6.

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23

Siesjö, Bo K. "Hypoglycemia, brain metabolism, and brain damage." Diabetes / Metabolism Reviews 4, no. 2 (1988): 113–44. http://dx.doi.org/10.1002/dmr.5610040203.

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24

Iwadate, Kimiharu, Nagahisa Matsuyama, Miwako Aoyagi, Ryo Shimada, and Yohko Ito. "A case of generalized hypoxic brain damage following traumatic brain damage." Legal Medicine 7, no. 2 (2005): 117–21. http://dx.doi.org/10.1016/j.legalmed.2004.10.003.

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25

Ladavas, E. "RIGHT HEMISPHERE DAMAGE." Brain 123, no. 3 (2000): 650–51. http://dx.doi.org/10.1093/brain/123.3.650.

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26

Gratton, Caterina, Emi M. Nomura, Fernando Pérez, and Mark D'Esposito. "Focal Brain Lesions to Critical Locations Cause Widespread Disruption of the Modular Organization of the Brain." Journal of Cognitive Neuroscience 24, no. 6 (2012): 1275–85. http://dx.doi.org/10.1162/jocn_a_00222.

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Although it is generally assumed that brain damage predominantly affects only the function of the damaged region, here we show that focal damage to critical locations causes disruption of network organization throughout the brain. Using resting state fMRI, we assessed whole-brain network structure in patients with focal brain lesions. Only damage to those brain regions important for communication between subnetworks (e.g., “connectors”)—but not to those brain regions important for communication within sub-networks (e.g., “hubs”)—led to decreases in modularity, a measure of the integrity of net

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27

Baumann, Kim. "Brain DNA damage hotspots." Nature Reviews Molecular Cell Biology 22, no. 5 (2021): 304–5. http://dx.doi.org/10.1038/s41580-021-00367-5.

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28

Wright, N. P., J. K. H. Wales;, and T. M. O'Shea. "Brain Damage and Dexamethasone?" PEDIATRICS 106, no. 4 (2000): 864. http://dx.doi.org/10.1542/peds.106.4.864.

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29

Millichap, J. Gordon. "Hypoglycemia and Brain Damage." Pediatric Neurology Briefs 2, no. 11 (1988): 83. http://dx.doi.org/10.15844/pedneurbriefs-2-11-3.

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30

Millichap, J. Gordon. "Brain Damage and Violence." Pediatric Neurology Briefs 7, no. 8 (1993): 57. http://dx.doi.org/10.15844/pedneurbriefs-7-8-1.

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31

DueÃ±as, Alejandro. "Avoiding the Brain Damage." JOURNAL OF ADVANCES IN PHYSICS 10, no. 3 (2015): 2871–73. http://dx.doi.org/10.24297/jap.v10i3.1325.

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The pursuit of low losses and low distortion are necessary conditions to improve the signal integrity. As mentioned in [1], it is sometimes said that thereare two types of engineersâ€”those who have signal integrityproblems and those who will.As clock frequencies increase,magnifying signal integrityproblems, this saying willbecome even more accurate.On the other hand, the usage of the technology is now a day unavoidable. Thus, to avoid the electromagnetic radiationdirectly on the brain, should be recomendablethe useof the hand off device since the integrated inalambric amplifiers of new genera

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32

Fine, Ronald D. "Treatment of brain damage." Medical Journal of Australia 152, no. 8 (1990): 441. http://dx.doi.org/10.5694/j.1326-5377.1990.tb125276.x.

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33

Frisoni, G. B., A. Scuratti, A. Bianchetti, and M. Trabucchi. "Hypergraphia and brain damage." Journal of Neurology, Neurosurgery & Psychiatry 56, no. 5 (1993): 576–77. http://dx.doi.org/10.1136/jnnp.56.5.576-b.

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34

Dammann, Olaf, and Alan Leviton. "Perinatal Brain Damage Causation." Developmental Neuroscience 29, no. 4-5 (2007): 280–88. http://dx.doi.org/10.1159/000105469.

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35

Isaacson, Robert L. "Brain Plasticity Alter Damage." Clinics in Perinatology 17, no. 1 (1990): 67–75. http://dx.doi.org/10.1016/s0095-5108(18)30589-x.

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36

Shieff, Colin. "BRAIN DAMAGE AND HYPONATRAEMIA." Lancet 332, no. 8624 (1988): 1373–74. http://dx.doi.org/10.1016/s0140-6736(88)90919-1.

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37

Lebrun, Yvan. "Cluttering after brain damage." Journal of Fluency Disorders 21, no. 3-4 (1996): 289–95. http://dx.doi.org/10.1016/s0094-730x(96)00031-9.

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38

Lecours, AndréRoch, Jacques Mehler, Maria Alice Parente, et al. "Illiteracy and brain damage." Brain and Cognition 6, no. 3 (1987): 243–65. http://dx.doi.org/10.1016/0278-2626(87)90126-6.

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39

Miller, Jimmy D. "Recovery from Brain Damage." Neurosurgery 36, no. 6 (1995): 1230–33. http://dx.doi.org/10.1227/00006123-199506000-00031.

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40

Bach-y-Rita, P. "Recovery from Brain Damage." Neurorehabilitation and Neural Repair 6, no. 4 (1992): 191–99. http://dx.doi.org/10.1177/136140969200600404.

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41

Miller, Jimmy D. "Recovery from Brain Damage." Neurosurgery 36, no. 6 (1995): 1231. http://dx.doi.org/10.1097/00006123-199506000-00031.

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42

Wilmshurst, P. "Brain damage in divers." BMJ 314, no. 7082 (1997): 689. http://dx.doi.org/10.1136/bmj.314.7082.689.

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43

Yates, P. O., J. R. Broome, A. D. Pitkin, M. Knauth, and S. Ries. "Brain damage in divers." BMJ 314, no. 7096 (1997): 1761. http://dx.doi.org/10.1136/bmj.314.7096.1761.

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44

Barltrop, Donald. "Lead and Brain Damage." Human Toxicology 4, no. 2 (1985): 121. http://dx.doi.org/10.1177/096032718500400201.

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45

Thomson, A. D., O. E. Pratt, M. Jeyasingham, and G. K. Shaw. "Alcohol and Brain Damage." Human Toxicology 7, no. 5 (1988): 455–63. http://dx.doi.org/10.1177/096032718800700513.

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The safe limits of alcohol intake are difficult to define because of individual variations in susceptibility to damage. The present recommendations are based largely on epidemiological studies of liver damage. Recent investigations indicate that alcoholic brain damage is much more common than previously suspected. More information is required about its natural history and the characteristics of individuals most likely to suffer damage. Thiamin (vitamin B 1) deficiency has long been associated with brain damage and may result from a number of additive causes in the alcoholic patient. New inform

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46

Allman, Peter. "Emotionalism Following Brain Damage." Behavioural Neurology 4, no. 1 (1991): 57–62. http://dx.doi.org/10.1155/1991/209837.

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Emotionalism is an heightened tendency to cry, or more rarely, laugh. It is commonly associated with brain damage and is often distressing to both patients and carers. Emotionalism is easily confused with depression, and when severe it can interfere with treatment. The aetiology is poorly understood but its response to drugs with different modes of action suggests that there is more than one underlying mechanism. When the components of emotionalism are studied separately a wide range is observed and they combine in a more complex and varied way than commonly held stereotyped views suggest. Mos

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47

Harper, Clive, and Izuru Matsumoto. "Ethanol and brain damage." Current Opinion in Pharmacology 5, no. 1 (2005): 73–78. http://dx.doi.org/10.1016/j.coph.2004.06.011.

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48

Molnar, Gabriella E., Jessie K. M. Easton, Angeles Badell, et al. "Brain damage causing disability." Archives of Physical Medicine and Rehabilitation 70, no. 5 (1989): S166—S169. http://dx.doi.org/10.1016/0003-9993(89)90023-3.

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49

Hachinski, V. "Brain Damage and Violence." Archives of Neurology 50, no. 8 (1993): 871. http://dx.doi.org/10.1001/archneur.1993.00540080074019.

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50

Siniša, Franjić. "Head Injuries, a general approach." Iberoamerican Journal of Medicine 2, no. 1 (2020): 19–23. https://doi.org/10.5281/zenodo.3626079.

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When a person with a head injury arrives at the hospital, doctors and nurses first check life signs: cardiac work, blood pressure and breathing. For those who do not breathe satisfactorily, a extractor fan may be required. Doctors immediately assess the patient's state of mind and memory. They also test the brain's basic functions by checking the size of the pupils and their response to light, assessing reactions to sensations such as heat and stab of the needle, and testing the ability to move their arms or legs. Computed tomography or magnetic resonance imaging is required to evaluat

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