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Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Holmberg, Lars. "Den frånvarande intellektuelle : En diskursteoretisk analys av en kollektiv representation." Doctoral thesis, Uppsala universitet, Sociologiska institutionen, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-236306.

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From an institutionalized conception of contemporary intellectuals as silent or absent, the aim of this thesis is to analyse how the representation of the intellectual changed from a hero of reason to a more or less absent actor. Descriptions of intellectuals that were initially analysed which showed that the representation of the intellectual, besides being linked to modernity, contained a relationship between the individual actor and political power, a relationship which could be traced back to ancient myths. Using discourse theory and concepts primarily from Ernesto Laclau and Chantal Mouff

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2

Kay, Robert James. "Expression of C. elegans heat shock genes in mouse cells." Thesis, University of British Columbia, 1986. http://hdl.handle.net/2429/27118.

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The transcriptional control elements of a pair of divergently transcribed C. eleqans heat shock-indueible hsp16 genes were examined by expressing the genes in mouse cells. For this purpose, transfection vectors containing elements necessary for bovine papillomavirus-mediated extrachromosomal replication were constructed. Genes inserted into these vectors could be efficiently transcribed upon introduction into mouse fibroblasts. Elevated copy numbers of these vectors in transfected cells were dependent on expression of BPV gene products in the same cells, but were not dependent on the presence

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3

Claus, Carol L. "Inhibition of troponin C expression in C¦2C¦12 mouse skeletal muscle cells by an antisense oligodeoxyribonucleotide." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0021/MQ47318.pdf.

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4

Akabane, Hiroto. "Protein kinase C activity in mouse eggs regulates gamete membrane interaction." Huntington, WV : [Marshall University Libraries], 2006. http://www.marshall.edu/etd/descript.asp?ref=655.

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5

Dehghani, Hesam. "Embryonic alkaline phosphatase and protein kinase C in preimplantation mouse development." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/NQ58304.pdf.

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6

Soare, Catalina P. "Characterization of Liver Damage Mechanisms Induced by Hepatitis C Virus." Thèse, Université d'Ottawa / University of Ottawa, 2011. http://hdl.handle.net/10393/20343.

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Hepatitis C Virus (HCV) is one of the most important causes of chronic liver disease, affecting more than 170 million people worldwide. The mechanisms of hepatitis C pathogenesis are unknown. Viral cytotoxicity and immune mediated mechanisms might play an important role in its pathogenesis. HCV infection and alcohol abuse frequently coexist and together lead to more rapid progression of liver disease, increasing the incidence and prevalence of cirrhosis and hepatocellular carcinoma. The cytopathic effect of HCV proteins, especially the core, E1 and E2 structural proteins, which induce liver st

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7

Thomas, Christopher. "APC/C processivity and cell cycle regulation in meiosis I mouse oocytes." Thesis, University of Newcastle upon Tyne, 2018. http://hdl.handle.net/10443/4070.

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Accurate cell division is a strictly ordered, highly regulated event. In mitosis, a robust spindle checkpoint ensures that chromosome division errors occur at a relatively low frequency, maintaining high levels of cyclin B1 and securin until chromosomes are accurately aligned. In contrast, in mouse oocytes, cyclin B1 and securin are targeted for destruction in late prometaphase I, at a time when the spindle is yet to fully migrate to the cortex and checkpoint proteins are still at kinetochores. This has been suggested to be symptomatic of an inefficient spindle checkpoint in meiosis I oocytes

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8

Li, Bin. "Characterization of C/EBPδ mRNA stability regulation in mouse mammary epithelial cell". The Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=osu1167885575.

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9

Nardiello, Tricia Lynn. "Protease activity in lymphoid organs of BALB/C and C57BL/6 mice following murine leukemia virus /." Connect to online version, 2007. http://ada.mtholyoke.edu/setr/websrc/pdfs/www/2007/214.pdf.

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10

Guerrero, Israel. "A Comparison of Chikungunya Virus Infection, Dissemination, and Cytokine Induction in Human and Murine Macrophages and Characterization of RAG2-/-γc-/- Mice as an Animal Model to Study Neurotropic Chikungunya Disease". BYU ScholarsArchive, 2020. https://scholarsarchive.byu.edu/etd/8430.

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Chikungunya virus (CHIKV) is classified as an alphavirus in the Togaviridae family. This virus is known to rely on Aedes arthropod vectors for its dissemination. Human infection is characterized by rash, high fever, and severe chronic polyarthritis that can last for years. Recently, efforts in developing animal models have been made in an attempt to better understand CHIKV pathogenesis. CHIKV infection starts with a 7 to 10 day long febrile acute phase, in which most of the symptoms occur (rash, fever, and incapacitating pain in joints and muscle). Once the immune system clears most of the vir

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11

Miyashiro, Samantha Ive. "Caracterização fenotípica do camundongo BALB/c mutante anêmico." Universidade de São Paulo, 2012. http://www.teses.usp.br/teses/disponiveis/10/10133/tde-29042013-155003/.

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Várias linhagens de camundongos resultam de mutações genéticas pontuais espontâneas ou induzidas e estas alterações podem apresentar fenótipos relevantes semelhantes a doenças hereditárias humanas. Massironi et al. (2006) desenvolveram vários camundongos BALB/c mutantes com o agente mutagênico etil-nitroso-uréia (ENU) no Biotério de Experimentação do Departamento de Imunologia do Instituto de Ciências Biomédicas (ICB) da Universidade de São Paulo. Destes, o mutante chamado anêmico foi objeto de estudo desta pesquisa, que tem como objetivo a sua fenotipagem. A avaliação hematológica revelou mod

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12

Wang, Yi-Fang. "Short term effects of c-MYC activation on β-cell physiology and glucose homeostasis in the pIns-c-MycERTAM transgenic mouse". Thesis, University of Warwick, 2012. http://wrap.warwick.ac.uk/45891/.

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Injection of 4-hydroxytamoxifen (4-OHT) activates the oncogene c-myc in transgenic pIns-c-MycERTAM mice, triggering β-cell proliferation in the short term as well as apoptosis and reduced insulin secretion, leading to hyperglycaemia. This hyperglycaemia is preceded by a short period of hypoglycaemia, which may be caused by: (i) increased insulin secretion or release from dying cells; (ii) rapid β-cell proliferation; and (iii) increased insulin sensitivity. This thesis characterizes the initial stages of the expression of c-MYC in the plns-c-MycERTAM mouse model and attempts to identify the cau

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13

Socha, Amanda L. "Differential expression of an endogenous retrovirus in MAIDS susceptible (C57BL/6) versus resistant (BALB/C) mice /." Connect to online version, 2006. http://ada.mtholyoke.edu/setr/websrc/pdfs/www/2006/159.pdf.

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14

Li, Bin. "Characterization of C/EBP[delta] mRNA stability regulation in mouse mammary epithelial cell." Columbus, Ohio : Ohio State University, 2007. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=osu1167885575.

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15

Zhang, Yalun, and 张亚伦. "Pax6/c-Myb regulates neuronal apoptosis in a mouse model of Alzheimer's disease." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47331148.

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Alzheimer’s disease (AD) is the most frequent neurodegenerative disorder which is characterized by impaired mental functions such as memory, language, perception, behavior and personality, as well as cognitive skills. The molecular mechanisms underlying this disease is still largely unknown, but numerous evidence emerge to support a cell cycle hypothesis which implicates the deregulation of cell cycle proteins as key mediators of neuronal dysfunction and loss in AD brains. One of these signals in Aβ-induced neuronal death model is Cdk/Rb/E2F pathway, where Aβ insult evokes activation o

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16

Bancroft, Allison J. "The BALB/c mouse as a model for protective immunity to Brugia pahangi." Thesis, University of Liverpool, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.333631.

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17

Zay, Agnes. "Role of C/EBPβ in two luminal progenitor populations in the mouse mammary gland". Thesis, University of Edinburgh, 2013. http://hdl.handle.net/1842/9522.

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The mammary gland is a branched epithelial organ comprised of myoepithelial, ductal and alveolar cells that are derived from resident stem and progenitor cells. The progression from mammary gland stem cell(s) to the differentiated mammary gland cell types is poorly understood. Here, I describe the identification and characterization of two luminal progenitor cell populations in the mouse mammary gland, and investigate the role of the transcription factor C/EBPβ in their development. In Chapter 2, I describe the isolation of two luminal progenitor cell populations (Sca1+ and Sca1- luminal cells

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18

Leung, Tsz-ki, and 梁子騏. "Transcriptional and post-translational regulations of junctional adhesion molecule-c in mouse germ cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43571992.

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19

Leung, Tsz-ki. "Transcriptional and post-translational regulations of junctional adhesion molecule-c in mouse germ cells." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B43571992.

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20

Noe, Elena Ariane [Verfasser]. "Characterization of Protein Kinase C Alpha Deficiency in a Mouse Model / Elena Ariane Noe." Berlin : Freie Universität Berlin, 2016. http://d-nb.info/111473523X/34.

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21

STALLOCK, JAMES PATRICK. "THE ROLE OF BAX IN APOPTOSIS OF ECTOPIC PRIMORDIAL GERM CELLS IN THE MOUSE." University of Cincinnati / OhioLINK, 2003. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1046891667.

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22

Sabbahi, Abdelaziz. "Transfert de l'eau dans la mousse de polyuréthane à 80 et 100°C : modélisation et expérimentation." Saint-Etienne, 1993. http://www.theses.fr/1993STET4019.

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L'étude du transfert de l'eau dans la mousse de polyuréthane a été réalisée en couplant l'expérience et la modélisation des phénomènes construits en formulant des hypothèses. Le processus de l'absorption de l'eau par la mousse de polyuréthane peut être représenté par une diffusion fickienne avec un coefficient de diffusion constant et un coefficient de transfert de matière à la surface très élevé lors du transfert unidirectionnel. Le processus de séchage a été modélisé en considérant l'évaporation de l'eau et la diffusion de la vapeur dans la partie libre de la mousse de polyuréthane. De même,

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23

Beal, Robert William John. "Investigating the role of Junctional Adhesion Molecule-C (JAM-C) in endothelial cell biology in vitro and in vivo using human and mouse models." Thesis, Queen Mary, University of London, 2018. http://qmro.qmul.ac.uk/xmlui/handle/123456789/44687.

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Junctional adhesion molecule C (JAM-C) is a component of endothelial cell (EC) tight junctions that has been implicated in a number of endothelial functions, such as angiogenesis and trafficking of leukocytes through the endothelium during inflammation. Work within our lab has identified that loss of JAM-C at EC junctions results in increased reverse transendothelial migration (rTEM) of neutrophils back into the circulation, a response that has been associated with the dissemination of inflammation to distant organs. Whilst the mechanism by which JAM-C is lost or redistributed away from EC jun

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24

Soavelomandroso, Anna P., Françoise Gaudin, Sandra Hoys, et al. "Biofilm Structures in a Mono-Associated Mouse Model of Clostridium difficile Infection." FRONTIERS MEDIA SA, 2017. http://hdl.handle.net/10150/626057.

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Clostridium difficile infection (CDI) is a major healthcare-associated disease with high recurrence rates. Host colonization is critical for the infectious process, both in first episodes and in recurrent disease, with biofilm formation playing a key role. The ability of C. difficile to form a biofilm on abiotic surfaces is established, but has not yet been confirmed in the intestinal tract. Here, four different isolates of C. difficile, which are in vitro biofilm producers, were studied for their ability to colonize germ-free mice. The level of colonization achieved was similar for all isolat

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25

Kuwahara, M., T. Ogaeri, R. Matsuura, et al. "In vitro organogenesis of gut-like structures from mouse embryonic stem cells." Blackwell, 2004. http://hdl.handle.net/2237/7446.

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26

Wolfram, Julie Ann. "Delineating the Role of c-Myc in Development and Propagation of Hypertrophic Cardiomyopathy." Case Western Reserve University School of Graduate Studies / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=case1315603810.

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27

Schuster, Stefanie [Verfasser], and Helmut [Gutachter] Brandstätter. "Generation and characterization of a GlyT1b/c knock-in mouse / Stefanie Schuster. Gutachter: Helmut Brandstätter." Erlangen : Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), 2016. http://d-nb.info/1102529079/34.

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28

Han, Jianming. "Roles of PKC isozymes in retinoic acid-induced B16 mouse melanoma cell growth inhibition and differentiation." Huntington, WV : [Marshall University Libraries], 2004. http://www.marshall.edu/etd/descript.asp?ref=385.

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Thesis (Ph. D.)--Marshall University, 2004.<br>Title from document title page. Document formatted into pages; contains p. xvi, 173 p. Includes abstract. Includes bibliographical references (p. 153-173).

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29

Schmid, Manuela. "Eimeria falciformis infection of mouse cells identifies host determinants of parasite development." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2014. http://dx.doi.org/10.18452/17001.

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Eimeria falciformis ist ein Apicomplexa-Parasit, welcher das Blinddarmepithel der Maus befällt. Aufgrund des monoxenen Lebenszyklus in einem exzellent-erforschten Wirt, bietet sich E. falciformis als Modellorganismus an, um Wirts-Parasit-Interaktionen zu untersuchen. Im Rahmen dieser Arbeit wurden mit Hilfe von Genexpressionsanalysen bei E. falciformis-infizierten Zellen und Mäusen Wirtsfaktoren identifiziert, welche für die in vitro bzw. in vivo Entwicklung des Parasiten vonnöten sind. Der Transkriptionsfaktor c-FOS (FBJ osteosarcoma oncogene) zeigte eine erhöhte Expression bei der Infektion

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30

Kabbout, Mohamed Nazih. "ETS1 AND ETS2 ROLE IN RAS ONCOGENIC TRANSFORMATION IN MOUSE EMBRYONIC FIBROBLASTS." The Ohio State University, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=osu1275408102.

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Jones, Torrie Turner. "Age-Related Deficits in Electron Transport Chain Complexes in Rat Neurons and 3xTg-AD Mouse Neurons." Available to subscribers only, 2009. http://proquest.umi.com/pqdweb?did=1797219571&sid=1&Fmt=2&clientId=1509&RQT=309&VName=PQD.

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Thesis (Ph. D.)--Southern Illinois University Carbondale, 2009.<br>"Department of Molecular Biology, Microbiology, and Biochemistry." Keywords: Aging, Cytochrome c oxidase, Electron transport chains, Estrogen, Mitochondria, Neurons. Includes bibliographical references (p. 102-137). Also available online.

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32

Černyšiov, Vitalij. "The effect of melatonin on the antibody production and leukocyte migration in BALB/c line mouse." Doctoral thesis, Lithuanian Academic Libraries Network (LABT), 2014. http://vddb.library.lt/obj/LT-eLABa-0001:E.02~2014~D_20141209_112335-12455.

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Melatonin – is a hormone produced by the pineal gland during the dark time. Light during the night suppresses melatonin production. Melatonin is an important biological body regulator: it controls daily and seasonal biorhythms, glucose metabolism, gonadal activity, cardiovascular system, gastrointestinal tract and the activity of the immune system There is a lot of scientific information about the immunoregulative properties of melatonin. Melatonin modulates the development of some organs of the immune system, cell differentiation, immune response and cytokine production. The imunomodulatory

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33

Gigliotti, Andrew Paul. "CCAAT/enhancer binding protein-delta (C/EBP-delta) expression and function in the mouse mammary gland /." The Ohio State University, 2000. http://rave.ohiolink.edu/etdc/view?acc_num=osu1488203552777002.

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34

Nishikawa, Yuko. "Naringenin Attenuates Metabolic Disturbances in C-26 Cancer Cachexia Mouse Model: Transitional Study for Human Application." The Ohio State University, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1563983598733999.

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35

Sugino, Yoshio. "BALB/c-Fcgr2b-/-Pdcd1-/- mouse expressing anti-urothelial antibody is a novel model of autoimmune cystitis." Kyoto University, 2012. http://hdl.handle.net/2433/159386.

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36

PARRELLA, Edoardo. "From gut to brain: sexually dimorphic pathology in the c-rel-/- mouse mimicking Parkinson’s disease progression." Doctoral thesis, Università degli studi di Brescia, 2022. http://hdl.handle.net/11379/558643.

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La malattia di Parkinson (MP) è la più comune malattia neurodegenerativa del movimento. Rispetto agli uomini, le donne mostrano incidenza e prevalenza più basse, esordio tardivo, e sintomi più blandi. Caratteristiche principali della MP sono la perdita di neuroni dopaminergici nella sostanza nera e l’accumulo di α-sinucleina. Questa proteina, normalmente espressa nel sistema nervoso centrale ed enterico (SNC e SNE), in condizioni patologiche aggrega formando inclusioni proteinacee osservabili nei cervelli e negli intestini dei pazienti affetti da MP. Oltre ai noti deficit motori, la MP è cara

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PARRELLA, Edoardo. "From gut to brain: sexually dimorphic pathology in the c-rel-/- mouse mimicking Parkinson’s disease progression." Doctoral thesis, Università degli studi di Brescia, 2022. http://hdl.handle.net/11379/558640.

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La malattia di Parkinson (MP) è la più comune malattia neurodegenerativa del movimento. Rispetto agli uomini, le donne mostrano incidenza e prevalenza più basse, esordio tardivo, e sintomi più blandi. Caratteristiche principali della MP sono la perdita di neuroni dopaminergici nella sostanza nera e l’accumulo di α-sinucleina. Questa proteina, normalmente espressa nel sistema nervoso centrale ed enterico (SNC e SNE), in condizioni patologiche aggrega formando inclusioni proteinacee osservabili nei cervelli e negli intestini dei pazienti affetti da MP. Oltre ai noti deficit motori, la MP è cara

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PARRELLA, Edoardo. "From gut to brain: sexually dimorphic pathology in the c-rel-/- mouse mimicking Parkinson’s disease progression." Doctoral thesis, Università degli studi di Brescia, 2022. http://hdl.handle.net/11379/558637.

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La malattia di Parkinson (MP) è la più comune malattia neurodegenerativa del movimento. Rispetto agli uomini, le donne mostrano incidenza e prevalenza più basse, esordio tardivo, e sintomi più blandi. Caratteristiche principali della MP sono la perdita di neuroni dopaminergici nella sostanza nera e l’accumulo di α-sinucleina. Questa proteina, normalmente espressa nel sistema nervoso centrale ed enterico (SNC e SNE), in condizioni patologiche aggrega formando inclusioni proteinacee osservabili nei cervelli e negli intestini dei pazienti affetti da MP. Oltre ai noti deficit motori, la MP è cara

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39

Mushens, Rosemary E. "Production, characterization and avidity measurement of novel mouse monoclonal antibodies against erythrocyte glycophorin C and c-myc using synthetic peptides for in vitro and in vivo immunization." Thesis, University of the West of England, Bristol, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392807.

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Lima, Christine A. "Endogenous Localization and Expression Patterns of Aurora Kinases B and C in Mouse Oocytes and Early Embryos." Digital WPI, 2010. https://digitalcommons.wpi.edu/etd-theses/851.

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"The Aurora Kinase proteins are a family of serine/threonine kinases that have been shown to play fundamental roles in controlling M phase progression in somatic cells. Aurora Kinase A protein is known to be vital for proper spindle assembly and therefore, chromosome segregation. Previous reports have shown that Aurora Kinase B is vital for proper completion of karyokinesis and cytokinesis in somatic cells. The role of Aurora Kinase C in somatic cells has been found to be less clear; however it appears to play an important role in spermatogenesis. Little is known about the role of these Aurora

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41

Kaveh, Daryan Alexander. "Mechanisms of BCG-induced CD4 T cell-mediated immunity against mycobacterium bovis in the BALB/c mouse." Thesis, St George's, University of London, 2015. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.676097.

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The only available tuberculosis vaccine is BCG, but this requires urgent replacement or improvement for more efficacious use in the prevention of the large burden of disease in both humans and cattle. The protection provided by BCG remains valuable and in order to facilitate further vaccine development, the underlying protective mechanisms require understanding. In this thesis the putative role of the CD4 T cell response was examined. Collectively it was shown that BCG immunisation of BALB/c mice led to their colonisation by the live BCG vaccine bacilli. By six weeks postimmunisation the vacci

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42

Tesell, Jessica M. "The Notch1-c-Myc Pathway Mediates Leukemia-Initiating Cell Activity in Mouse T-ALL Models: A Dissertation." eScholarship@UMMS, 2013. http://escholarship.umassmed.edu/gsbs_diss/671.

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Although cure rates have significantly improved for children with T-cell acute lymphoblastic leukemia (T-ALL), 20-30% undergo induction failure or relapse with most succumbing to disease. Leukemia-initiating cells (L-ICs) are hypothesized to be resistant to conventional chemotherapy and radiation and are thereby responsible for disease recurrence. Using an in vivo limiting dilution assay, we previously showed that the murine T-ALL L-IC is quite rare, with only 0.003-0.05% of cells capable of initiating disease, and demonstrated that the L-IC is a subset of the leukemic DN3 thymic progenitor po

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43

Totenhagen, John W., Adam Bernstein, Eriko S. Yoshimaru, Robert P. Erickson, and Theodore P. Trouard. "Quantitative magnetic resonance imaging of brain atrophy in a mouse model of Niemann-Pick type C disease." PUBLIC LIBRARY SCIENCE, 2017. http://hdl.handle.net/10150/624480.

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In vivo magnetic resonance imaging (MRI) was used to investigate regional and global brain atrophy in the neurodegenerative Niemann Pick Type C1 (NPC1) disease mouse model. Imaging experiments were conducted with the most commonly studied mouse model of NPC1 disease at early and late disease states. High-resolution in vivo images were acquired at early and late stages of the disease and analyzed with atlas-based registration to obtain measurements of twenty brain region volumes. A two-way ANOVA analysis indicated eighteen of these regions were different due to genotype and thirteen showed a si

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44

Trujillo, Jonathan Anthony. "T cell responses to S-glutathionylated And heteroclitic viral epitopes and CCl2-mediated immune dysregulation in mice infected with a neurotropic coronavirus." Diss., University of Iowa, 2014. https://ir.uiowa.edu/etd/4775.

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Mice infected with neurotropic variants of the murine coronavirus, mouse hepatitis virus, (strains JHMV or J2.2–V–1) develop acute and chronic CNS infections, and provide a model system to study the pathogenesis of virus–induced neuroinflammation, mechanisms of virus persistence, and anti–viral immune responses in the CNS. Using the J2.2–V–1 model of CNS infection, we addressed the role of sustained CCL2 production during viral infection using mice in which CCL2 was expressed transgenically in oligodendrocytes. Tonic CCL2 expression in the CNS resulted in delayed kinetics of virus clearance, a

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Rico, Vargas Sergio Arturo. "Regulation and dysregulation of B lymphopoiesis in mouse bone marrow: in vivo role of macrophage activation (pristane-treatment and malaria-infection), c-myc, c-kit, and immunoglobulin genes." Thesis, McGill University, 1993. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=41340.

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To examine factors influencing normal and disordered genesis of the B lymphocyte lineage in mouse bone marrow, precursor B cell dynamics have been analysed in conditions predisposing to B cell neoplasias and deficiencies. Double immunofluorescence labeling and stathmokinetic techniques have been used to quantitate the population size and mitotic activity of pro-B cells before $ mu$ chain expression, pre-B cells expressing cytoplasmic $ mu$ chains, and B lymphocytes expressing surface IgM. Two conditions associated with prolonged macrophage activation and B cell neoplasia, pristane oil-treatmen

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VARANO, GABRIELE. "THE ROLE OF THE B CELL ANTIGEN RECEPTOR IN A MOUSE MODEL OF NON-HODGKIN LYMPHOMA." Doctoral thesis, Università degli Studi di Milano, 2013. http://hdl.handle.net/2434/219066.

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The B cell antigen receptor (BCR) plays a central role both in early B-cell development and in mature B cells, where it controls survival and allows the organism to mount protective immune responses against foreign antigens. Surface BCR expression is retained in most types of Non-Hodgkin B-cell lymphomas (NHL), where it has been proposed to participate to tumor initiation, maintenance and progression. This study has provided for the first time an in vivo genetic proof for the role of the BCR in B-cell lymphomagenesis. Using the λ-MYC Burkitt lymphoma (BL) mouse model, in which conditional a

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Ku, Nam-On. "Inflammatory cytokine regulation of pentraxin genes and tissue- specific expression of the gene for mouse C-reactive protein /." The Ohio State University, 1992. http://rave.ohiolink.edu/etdc/view?acc_num=osu1487780393266327.

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Ohno, Michiko. "Studies on role of the C-terminal region of mouse heat shock protein 72 in biological defense systems." Kyoto University, 2007. http://hdl.handle.net/2433/136532.

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Kyoto University (京都大学)<br>0048<br>新制・課程博士<br>博士(農学)<br>甲第13113号<br>農博第1618号<br>新制||農||941(附属図書館)<br>学位論文||H19||N4239(農学部図書室)<br>UT51-2007-H386<br>京都大学大学院農学研究科食品生物科学専攻<br>(主査)教授北畠直文, 教授吉川正明, 教授河田照雄<br>学位規則第4条第1項該当

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Kondo, Eri. "Skeletal Analysis of the Long Bone Abnormality (lbab/lbab) Mouse, A Novel Chondrodysplastic C-type Natriuretic Peptide Mutant." Kyoto University, 2014. http://hdl.handle.net/2433/189351.

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Kielczewska, Agnieszka. "Natural killer cell receptors and their MHC ligand interactions in innate resistance to mouse cytomegalovirus." Thesis, McGill University, 2007. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=111896.

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Le premier but de mon projet de doctorat a ete la caracterisation moleculaire de l'interface entre les recepteurs activateurs des cellules Natural Killer (NK) et de leurs ligands exprimes dans les cellules infectees ainsi que l'implication de cette interaction sur la reponse a l'infection par le cytomegalovirus (MCMV).<br>J'ai tire un avantage de la variation naturelle au sein des membres lies aux recepteurs Ly49C ainsi que de la disponibilite des structures cristallines des Ly49 afin de comprendre les determinants moleculaires des interactions Ly49H-m157 et egalement identifier les residus de

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