Gotowe bibliografie tematyczne / Ca++ antagonist

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Ca++ antagonist”

Autor: Grafiati
Data publikacji: 2 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Ca++ antagonist"

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Yoneyama, Fumiya, Keisuke Satoh, and Norio Taira. "SD-3211, a new non-dihydropyridine Ca-antagonist, is as vasoselective as dihydropyrldine Ca-antagonists." Japanese Journal of Pharmacology 52 (1990): 282. http://dx.doi.org/10.1016/s0021-5198(19)55670-0.

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Bkaily, Ghassan, Sanaa Choufani, Levon Avedanian, et al. "Nonpeptidic antagonists of ETA and ETB receptors reverse the ET-1-induced sustained increase of cytosolic and nuclear calcium in human aortic vascular smooth muscle cellsThis article is one of a selection of papers published in the special issue (part 2 of 2) on Forefronts in Endothelin." Canadian Journal of Physiology and Pharmacology 86, no. 8 (2008): 546–56. http://dx.doi.org/10.1139/y08-048.

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Our previous work showed that ET-1 induced a concentration-dependent increase of cytosolic Ca2+ ([Ca]c) and nuclear Ca2+ ([Ca]n) in human aortic vascular smooth muscle cells (hVSMCs). In the present study, using hVSMCs and 3-dimensional confocal microscopy coupled to the Ca2+ fluorescent probe Fluo-3, we showed that peptidic antagonists of ETA and ETB receptors (BQ-123 (10−6 mol/L) and BQ-788 (10−7 mol/L), respectively) prevented, but did not reverse, ET-1-induced sustained increase of [Ca]c and [Ca]n. In contrast, nonpeptidic antagonists of ETA and ETB (respectively, BMS-182874 (10−8–10−6 mol
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Conway, William S., Wojciech J. Janisiewicz, Joshua D. Klein, and Carl E. Sams. "Strategy for Combining Heat Treatment, Calcium Infiltration, and Biological Control to Reduce Postharvest Decay of `Gala' Apples." HortScience 34, no. 4 (1999): 700–704. http://dx.doi.org/10.21273/hortsci.34.4.700.

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The viability of Penicillium expansum Link conidia in sporulating culture declined rapidly when exposed to 38 °C, and when conidia were exposed to 38 °C prior to inoculation of apple fruits (Malus ×domestica Borkh.), the resulting lesions were smaller than those on fruit inoculated with nonheated conidia. `Gala' apples were heated after harvest (38 °C for 4 days), pressure infiltrated with a 2% solution of CaCl2, or treated with the antagonist Pseudomonas syringae van Hall, alone or in combinations to reduce postharvest decay caused by Penicillium expansum. After up to 6 months in storage at 1
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Page, Stephen J., Mingyan Zhu, and Suzanne M. Appleyard. "Effects of acute and chronic nicotine on catecholamine neurons of the nucleus of the solitary tract." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 316, no. 1 (2019): R38—R49. http://dx.doi.org/10.1152/ajpregu.00344.2017.

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Nicotine is an addictive drug that has broad effects throughout the brain. One site of action is the nucleus of the solitary tract (NTS), where nicotine initiates a stress response and modulates cardiovascular and gastric function through nicotinic acetylcholine receptors (nAChRs). Catecholamine (CA) neurons in the NTS influence stress and gastric and cardiovascular reflexes, making them potential mediators of nicotine’s effects; however nicotine’s effect on these neurons is unknown. Here, we determined nicotine’s actions on NTS-CA neurons by use of patch-clamp techniques in brain slices from
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Gilliam, F. R., P. A. Rivas, D. J. Wendt, C. F. Starmer, and A. O. Grant. "Extracellular pH modulates block of both sodium and calcium channels by nicardipine." American Journal of Physiology-Heart and Circulatory Physiology 259, no. 4 (1990): H1178—H1184. http://dx.doi.org/10.1152/ajpheart.1990.259.4.h1178.

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Slowing of the recovery of the Na channel from local anesthetic block at low external pH has been well described. In contrast the Ca channel has been reported not to show such an effect. Because of the absence of this response, a two-site model for the interaction of Ca antagonists with the Ca channel has been proposed. We sought to determine whether these results were a consequence of utilizing the poorly lipid-soluble Ca antagonist diltiazem as the test drug. We have measured the time constants of recovery (tau r) of Na (INa) and Ca currents (ICa) recorded from rabbit atrial myocytes during
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BUSHI, KIMIHIKO. "Drug interaction of digitalis and Ca antagonist." Rinsho yakuri/Japanese Journal of Clinical Pharmacology and Therapeutics 24, no. 1 (1993): 237–38. http://dx.doi.org/10.3999/jscpt.24.237.

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HIRZEL, H. "Clinical profile of a Ca++-CaM-Antagonist." Journal of Molecular and Cellular Cardiology 19 (1987): S35. http://dx.doi.org/10.1016/s0022-2828(87)80111-6.

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Lamouche, Stéphane, and Nobuharu Yamaguchi. "Role of PAC1 receptor in adrenal catecholamine secretion induced by PACAP and VIP in vivo." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 280, no. 2 (2001): R510—R518. http://dx.doi.org/10.1152/ajpregu.2001.280.2.r510.

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The present study was conducted to investigate the functional implication of the pituitary adenylate cyclase-activating polypeptide (PACAP) type I (PAC1) receptor in the adrenal catecholamine (CA) secretion induced by either PACAP-27 or vasoactive intestinal polypeptide (VIP) in anesthetized dogs. PACAP-27, VIP, and their respective antagonists were locally infused to the left adrenal gland via the left adrenolumbar artery. Plasma CA concentrations in adrenal venous and aortic blood were determined by means of a high-performance liquid chromatograph coupled with an electrochemical detector. Ad
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Janisiewicz, Wojciech J., William S. Conway, D. Michael Glenn, and Carl E. Sams. "Integrating Biological Control and Calcium Treatment for Controlling Postharvest Decay of Apples." HortScience 33, no. 1 (1998): 105–9. http://dx.doi.org/10.21273/hortsci.33.1.0105.

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Two nonfungicidal postharvest treatments and N fertility were evaluated for their effectiveness, singly and in combination, in controlling decay (“blue mold”) of apple (Malus ×domestica Borkh.) by Penicillium expansum Link. The antifungal treatments were biological control using the antagonist Pseudomonas syringae van Hall (isolate ESC-11 used in BioSave-11 and BioSave-110 products), and pressure infiltration of fruit with 0.27 M CaCl2. Fruit susceptibility to decay was evaluated using fruit after 3 and 6 months of storage over 2 years. Calcium infiltration resulted in a 3-fold increase in Ca
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Negulescu, P. A., and T. E. Machen. "Intracellular Ca regulation during secretagogue stimulation of the parietal cell." American Journal of Physiology-Cell Physiology 254, no. 1 (1988): C130—C140. http://dx.doi.org/10.1152/ajpcell.1988.254.1.c130.

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Intracellular [Ca] ([Ca]i) was measured following secretagogue stimulation of rabbit gastric glands loaded with the Ca-sensitive dye fura-2. Glands were mounted on cover slips and placed either in a perfused cuvette (for spectrofluorimetric measurements on whole glands) or in a chamber on the stage of a microscope (for microspectrofluorimetric measurements on single parietal cells within a gland). In parietal cells resting [Ca]i = 91 nM. Either histamine or carbachol caused [Ca]i to increase (spike) rapidly (within 5 s) to greater than 425 nM by releasing Ca from an intracellular store. The tw
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Rozprawy doktorskie na temat "Ca++ antagonist"

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Nett, Leonore. "Beeinflussung der Expression endothelialer Adhäsionsmoleküle durch Ca++-Antagonisten, NO-Donoren und Antioxidantien." Doctoral thesis, Universitätsbibliothek Leipzig, 2006. http://nbn-resolving.de/urn:nbn:de:swb:15-2006101229.

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Adhäsionsmoleküle spielen eine entscheidende Rolle bei der Arterioskleroseentstehung. Sie bedingen die Anheftung und nachfolgende Migration von Leukozyten in den subendothelialen Raum (siehe 2.2.2). Eine vermehrte Expression ist Ausdruck einer Aktivierung des Gefäßendothels und kann zum einen durch physikalische Faktoren, wie zum Beispiel erhöhtem shear stress, ausgelöst werden. Zum anderen setzen verschiedene Substanzen Signalkaskaden der Endothelzelle in Gang, welche in einer vermehrten Expression der Oberflächenadhäsionsmoleküle gipfelt. Neben Zytokinen (IL-1, TNFα) und Bakterientoxinen wie
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Ai, Tomohiko. "Accentuated antagonism by angiotenism II on guinea pig cardiac L-type Ca-currents enhanced by β-adrenergic stimulation". Kyoto University, 1999. http://hdl.handle.net/2433/181725.

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Cardi, Delphine. "Etude du mutant E255L de l'ATPase Ca²+ SERCA1a de lapin et de l'ATPase Ca²+ PfATP6 de Plasmodium falciparum : Expression chez la levure S, cervisiae, purification, characterisation et essai d'inhibition par un antipaludéen puissant, l'artemisinine." Paris 11, 2009. http://www.theses.fr/2009PA11T046.

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Crowley, John J. "Cholesterol and Phospholipid Modulation of BK[subscript Ca] Channel Activity and Ethanol Sensitivity: a dissertation." eScholarship@UMMS, 2003. https://escholarship.umassmed.edu/gsbs_diss/107.

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The large conductance Ca++-activated K+ channel (BKCa) regulates neuronal excitability through the efflux of K+, in response to membrane depolarization and increases in intracellular Ca++. The activity of the BKCa channel is increased by acute exposure to ethanol (EtOH), which is thought to underlie, in part, the influence of the drug on peptide hormone release from neurohypophysial nerve terminals (Dopico et al., 1996, 1998). Moreover, chronic EtOH exposure attenuates acute drug action on hormone release, and reduces the sensitivity of BKCa channels to acute EtOH exposure (Knott et al., 2002)
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Kozlowski, Junior Vitoldo Antonio. "Efeito de antagonistas de receptores H1 e H2, sobre os niveis de Ca, P, Mg, Si, B e Sr, atraves de espectroscopia de emissão atomica com plasma induzido(ICP-AES), no soro de ratos parcialmente hepatectomizados." [s.n.], 1995. http://repositorio.unicamp.br/jspui/handle/REPOSIP/290173.

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Orientador: Thales Rocha de Mattos Filho<br>Tese(doutorado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba<br>Made available in DSpace on 2018-07-20T23:29:18Z (GMT). No. of bitstreams: 1 KozlowskiJunior_VitoldoAntonio_D.pdf: 2805679 bytes, checksum: f41eb64ef505024519936acf4a453fab (MD5) Previous issue date: 1995<br>Resumo: Ratos albinos machos foram utilizados para investigar os efeitos de antagonista específico de receptores Hl periféricos, Terfenadina e receptores H2, Cimetidina sobre as concentrações dos elementos Ca, P, Mg, Si, B e Sr, através de Espectrosco
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Nett, Leonore [Verfasser]. "Beeinflussung der Expression endothelialer Adhäsionsmoleküle durch Ca++-Antagonisten, NO-Donoren und Antioxidantien / eingereicht von Leonore Nett." 2006. http://d-nb.info/981531253/34.

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Książki na temat "Ca++ antagonist"

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Köhler, Mikro Robert. Versuche zum Nachweis der Wirkung unterschiedlicher Konzentrationen des Ca²⁺-Antagonisten Nifedipin auf die In-vitro-Befruchtungsergebnisse boviner Oozyten sowie auf die Cholesterin-Anteile boviner Spermienmembranen. [s.n.], 1998.

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Schürrle, Norbert. Hämodynamische Untersuchungen beim Hund unter intraarteriellen Calziumgaben und ihre Beeinflussung durch Prämedikation mit dem [beta]1-Blocker [Beta-Blocker] Metoprolol oder dem Ca-Antagonisten Gallopamil. [s.n.], 1985.

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Effects of [CA²+] and verapamil on muscle injury immediately after exercise. 1991.

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Effects of [CA²⁺] and verapamil on muscle injury immediately after exercise. 1991.

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Saurer, Christian. Einfluss der perioperativen Gabe von Digoxin oder Ca-Antagonisten auf die postoperative Arrhythmie-Inzidenz nach Koronaroperationen. 1995.

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Radlinger, Klaus. Elektronenemikroskopisch-morphometrische Analyse der Gefässwand von Koronararterien spontan hypertensiver Ratten nach Behandlung mit dem Ca-Antagonisten Nitrendipin. 1993.

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Breisgau, Universität Freiburg im, ed. Elektronenmikroskopisch-morphometrische Analyse der Gefässwand der Arteria femoralis spontan hypertensiver Ratten (SHR) nach Behandlung mit dem Ca-Antagonisten Nitrendipin. 1995.

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Keul, Radovan. Modulierende Effekte von Manidipin und anderen Ca²⁺-Antagonisten auf die Transkription der mRNAs für die Zytokine IL-1[beta] , IL-6 und GM-CSF und die Transkriptionsfaktoren Fos und Jun in humanen Mesangialzellen. 1992.

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Koppers, Dagmar. Elektrophysiologische Untersuchungen der Aktionspotentialkonfiguration, der maximalen Anstiegsgeschwindigkeit (dV/dt)max und der Kaliumströme Ik und Ik₁ an Purkinje Fasern des Kaninchens unter dem Einfluss des Ca²⁺-Antagonisten Diltiazem und der ACE-Hemmer Fosinopril, Fosinoprilat und Captopril. 1994.

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Części książek na temat "Ca++ antagonist"

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Watanabe, Yasuo, Koji Taya, Ichie Hara, Yusuke Ohgami, and Hiroya Utsumi. "Neuronal Changes In Ca2+ And Ph Regulated By A Ca Antagonist Under Extracellular Acidosis." In Calcium Ion Modulators. CRC Press, 2024. http://dx.doi.org/10.1201/9781003580768-5.

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Yamamoto, Jyunji. "Eeg Analysis Of Ca Antagonists In Rabbits." In Calcium Ion Modulators. CRC Press, 2024. http://dx.doi.org/10.1201/9781003580768-13.

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Ošťádal, B., F. Kolář, V. Pelouch, et al. "Ca Antagonists in Cardiopulmonary Changes Induced by High Altitude Hypoxia." In Developments in Cardiovascular Medicine. Springer US, 1987. http://dx.doi.org/10.1007/978-1-4613-2057-9_14.

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Hanck, D. A., M. F. Sheets, and H. A. Fozzard. "State-Dependent Block of Cardiac Na Currents by Ca Channel Antagonists." In Adrenergic System and Ventricular Arrhythmias in Myocardial Infarction. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-74317-7_13.

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Diemer, N. H., T. Balchen, T. Bruhn, et al. "Extended Studies on the Effect of Glutamate Antagonists on Ischemic CA-1 Damage." In Mechanisms of Secondary Brain Damage in Cerebral Ischemia and Trauma. Springer Vienna, 1996. http://dx.doi.org/10.1007/978-3-7091-9465-2_13.

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Fleckenstein-Grün, G., F. Thimm, M. Frey, and A. Czirfusz. "Role of calcium in arteriosclerosis — Experimental evaluation of antiarteriosclerotic potencies of Ca antagonists." In Arteriosclerosis. Steinkopff, 1994. http://dx.doi.org/10.1007/978-3-642-85660-0_14.

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Fleckenstein-Grün, G., Y. Makita, Y. K. Byon, and A. Fleckenstein. "Effects of Ca++-Agonistic and Ca++-Antagonistic 1,4-Dihydropyridine Compounds on Tonic and Phasic Activation of Extramural Coronary Vasculature." In Bayer-Symposium. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-70499-4_18.

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Lüscher, E. F. "Die Rolle der Blutplättchen bei der Atherogenese und ihre Beeinflussung durch Ca++-Antagonisten." In Therapie und Prävention mit Kalziumantagonisten. Steinkopff, 1988. http://dx.doi.org/10.1007/978-3-642-85362-3_10.

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Kitamura, K., and H. Kuriyama. "Molecular Mechanisms of Action of Antihypertensive Agents Ca-Antagonists and K-Channel Openers on Vascular Smooth Muscle." In Handbook of Experimental Pharmacology. Springer Berlin Heidelberg, 1994. http://dx.doi.org/10.1007/978-3-642-78920-5_16.

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Yamada, Shizuo, Shiny A. Uchida, Yoshiharu Deguchi, Minoru Yamamoto, and Ryohei Kimura. "Receptor Binding Characteristics Of Nimodipine And Other 1,4-Dihydropyridine Ca++ Antagonists In Relation To Their Pharmacokinetics In Senescent Brain." In Calcium Ion Modulators. CRC Press, 2024. http://dx.doi.org/10.1201/9781003580768-3.

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Streszczenia konferencji na temat "Ca++ antagonist"

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Bevan, Jane, and S. Heptinstall. "BOW CAN WE INHIBIT 5HT-INDUCED PLATELET AGGREGATION AND WHY SHOULD WE BOTHER?" In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643853.

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Platelets are induced to aggregate when 5-hydraxytryptamine (5HT) is added to citrated whole blood and the extent of aggregation can be measured using a Whole Blood Platelet Counter. We have used this method to study a) 5HT-induced platelet aggregation in normal human blood and the effects of 14 5HT receptor antagonists and 2 Ca++-channel blockers, and b) aggregation in blood from patients with peripheral vascular disease (PVD). Previous studies of platelet aggregation in platelet-rich plasma have indicated an increased platelet sensitivity to 5HT in PVD, and a multicentre study of ketanserin
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Ribeiro, Fernanda Cristina Poscai, and Everton Lopes Rodrigues. "Integrative review of the use of NMDA antagonists for TBI treatment." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.190.

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Introduction: The kinetic energy of TBI generates mechanical deformation, which causes release of glutamate, activating ionotropic receptors, principally NMDA receptors, favoring the flow of Ca++ and Na+ into the cell, producing edema. Then, the neurotoxicity generated by glutamate release can be avoided by NMDA antagonists. Objectives: To define if NMDA antagonists are promising for the treatment of TBI by literature analysis and to verify if there are reports of adverse reactions. Methodology: The review utilized the Scielo and Pubmed databases and the keywords used were: NMDA antagonist, Br
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Matsuyama, T., K. Machida, H. Matsuyama, Y. Dotake, K. Takagi, and H. Inoue. "Long-Acting Muscarinic Antagonist Attenuated Human Basophil-Derived IL-4 Production." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1257.

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Ahmad, S., M. Biggart, F. Sassano, A. Ghosh та R. Tarran. "The Orai1 Antagonist, ELD607, Reduces Chronic Neutrophilic Inflammation in βENaC Mice". У American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a6682.

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Kishimoto, Shuichi, Erika Bou, Kaho Higashi, Ryosuke Suzuki, and Shoji Fukushima. "Abstract 840: Enhancement of antitumor effect of platinum complexes by PXR antagonist." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-840.

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Basu, A., M. Arif, N. Johnson, et al. "Repositioning of Basimglurant (mGluR5 Antagonist) for the Treatment of Idiopathic Pulmonary Fibrosis." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a2603.

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Rold, Tammy L., Ashley F. Berendzen, Nicole E. Bernskoetter, Jillian C. Rushin, Lixin Ma, and Timothy J. Hoffman. "Abstract 4927:68Ga/177Lu-Peptide antagonist targeting of breast cancer: A xenograft evaluation." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4927.

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Sassano, F., S. Ahmad, J. Iskarpatyoti, et al. "The Orai1 Antagonist ELD607 Safely Reduces Lung Inflammation in G551D Cystic Fibrosis Ferrets." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a6352.

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Elzi, David J., Meihua Song, Kevin Hakala, Susan T. Weintraub, and Yuzuru Shiio. "Abstract A16: Wnt antagonist SFRP1 functions as secreted mediator of senescence." In Abstracts: Second AACR International Conference on Frontiers in Basic Cancer Research--Sep 14-18, 2011; San Francisco, CA. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.fbcr11-a16.

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Wrennall, J., A. Ghosh, M. Strenta, M. F. Sassano, and R. Tarran. "The Novel Orai1 Antagonist ELD607 Rescues the Hyperinflammatory Ca2+ Signaling Seen in CF Neutrophils." In American Thoracic Society 2023 International Conference, May 19-24, 2023 - Washington, DC. American Thoracic Society, 2023. http://dx.doi.org/10.1164/ajrccm-conference.2023.207.1_meetingabstracts.a4784.

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Raporty organizacyjne na temat "Ca++ antagonist"

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Altstein, Miriam, and Ronald J. Nachman. Rational Design of Insect Control Agent Prototypes Based on Pyrokinin/PBAN Neuropeptide Antagonists. United States Department of Agriculture, 2013. http://dx.doi.org/10.32747/2013.7593398.bard.

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The general objective of this study was to develop rationally designed mimetic antagonists (and agonists) of the PK/PBAN Np class with enhanced bio-stability and bioavailability as prototypes for effective and environmentally friendly pest insect management agents. The PK/PBAN family is a multifunctional group of Nps that mediates key functions in insects (sex pheromone biosynthesis, cuticular melanization, myotropic activity, diapause and pupal development) and is, therefore, of high scientific and applied interest. The objectives of the current study were: (i) to identify an antagonist bioph
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Fromm, A., Avihai Danon, and Jian-Kang Zhu. Genes Controlling Calcium-Enhanced Tolerance to Salinity in Plants. United States Department of Agriculture, 2003. http://dx.doi.org/10.32747/2003.7585201.bard.

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The specific objectives of the proposed research were to identify, clone and characterize downstream cellular target(s) of SOS3 in Arabidopsis thaliana, to analyze the Ca2+-binding characteristics of SOS3 and the sos3-1 mutant and their interactions with SOS3 cellular targets to analyze the SOS3 cell-specific expression patterns, and its subcellular localization, and to assess the in vivo role of SOS3 target protein(s) in plant tolerance to salinity stress. In the course of the study, in view of recent opportunities in identifying Ca2+ - responsive genes using microarrays, the group at Weizman
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