Gotowe bibliografie tematyczne / Cancer cells

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Gotowa bibliografia na temat „Cancer cells”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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Artykuły w czasopismach na temat "Cancer cells"

Fernández-Lázaro, Diego, César Ignacio Fernández-Lázaro, and Martínez Alfredo Córdova. "Cell Death: Mechanisms and Pathways in Cancer Cells." Cancer Medicine Journal 1, no. 1 (2018): 12–23. http://dx.doi.org/10.46619/cmj.2018.1-1003.

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Programmed cell death is an essential physiological and biological process for the proper development and functioning of the organism. Apoptosis is the term that describes the most frequent form of programmed cell death and derives from the morphological characteristics of this type of death caused by cellular suicide. Apoptosis is highly regulated to maintain homeostasis in the body, since its imbalances by increasing and decreasing lead to different types of diseases. In this review, we aim to describe the mechanisms of cell death and the pathways through apoptosis is initiated, transmitted,

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Srivastava, A. N., Neema Tiwari, Shailendra Yadav, and Suryakant . "LUNG CANCER STEM CELLS-AN UPDATE." Era's journal of medical research 4, no. 1 (2017): 22–31. http://dx.doi.org/10.24041/ejmr2017.4.

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MAS, Bezerra, Ferreira LAM, Kawasaki-Oyama RS, et al. "Effectiveness of Hypoxia-Induced Accumulation of Cancer Stem Cells in Head and Neck Squamous Cell Carcinoma." Cancer Medicine Journal 3, S1 (2020): 13–23. http://dx.doi.org/10.46619/cmj.2020.3.s1-1003.

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INTRODUCTION: The small number of cancer stem cells, which correspond to only 0.01% - 0.1% of total tumor cells, has been the biggest obstacle in understanding their biology and role in the origin and maintenance of tumors, their metastatic and recurrence potentials, and resistance to radio-chemotherapy. Therefore, promoting its accumulation will enable further studies and future advances in the diagnosis and treatment of head and neck cancer squamous cell carcinoma. OBJECTIVE: To induce cancer stem cell accumulation in primary cell cultures of head and neck squamous cell carcinoma using a hyp

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Yin, Wen, Jialing Wang, Linling Jiang, and Y. James Kang. "Cancer and stem cells." Experimental Biology and Medicine 246, no. 16 (2021): 1791–801. http://dx.doi.org/10.1177/15353702211005390.

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Being the second leading cause of death globally, cancer has been a long-standing and rapidly evolving focus of biomedical research and practice in the world. A tremendous effort has been made to understand the origin of cancer cells, the formation of cancerous tissues, and the mechanism by which they spread and relapse, but the disease still remains mysterious. Here, we made an attempt to scrutinize evidences that indicate the role of stem cells in tumorigenesis and metastasis, and cancer relapse. We also looked into the influence of cancers on stem cells, which in turn represent a major cons

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Fujimoto, Naohiro, Bin Han, Masayoshi Nomura, and Tetsuro Matsumoto. "WS1-1-1 Nitrogen-Containing Bisphosphonates Inhibit the Growth of Renal Cell Carcinoma Cells(Renal Cell Cancer)." Japanese Journal of Urology 99, no. 2 (2008): 142. http://dx.doi.org/10.5980/jpnjurol.99.142_1.

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Pratap, Dr Pushpendra D. "CANCER STEM CELLS IN CERVICAL CANCER AS BENEFICIAL GOALS AND BIOMARKERS: A COMPREHENSIVE." Era's Journal of Medical Research 10, no. 2 (2023): 51–55. http://dx.doi.org/10.24041/ejmr2023.36.

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The fourth most prevalent gynaecological malignancy affecting females globally is cervical cancer (CC). HPV (high-risk) infection has been related to the majority of CC cases. Owing to efficient screening through Pap smear and vaccination delivery, the commonness and death rate of CC have significantly decreased. Nevertheless, not all societies have access to this equally. A better therapeutic outcome may be achieved by targeting CSCs, which might play a significant impact in carcinogenesis, metastasis, recurrence, and radio / chemo –resistance of CC. The majority of tumours are made up of a t

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Lee, Cheong J., Joseph Dosch, and Diane M. Simeone. "Pancreatic Cancer Stem Cells." Journal of Clinical Oncology 26, no. 17 (2008): 2806–12. http://dx.doi.org/10.1200/jco.2008.16.6702.

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Cellular heterogeneity in cancer was observed decades ago by studies in mice which showed that distinct subpopulations of cells within a tumor mass are capable of driving tumorigenesis. Conceptualized from this finding was the stem-cell hypothesis for cancer, which suggests that only a specific subset of cancer cells within each tumor is responsible for tumor initiation and propagation, termed tumor initiating cells or cancer stem cells (CSCs). Recent data has been provided to support the existence of CSCs in human blood cell–derived cancers and solid organ tumors of the breast, brain, prostat

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Inderberg, Else Marit, and Sébastien Wälchli. "T cells successfully fighting cancer." Open Access Government 43, no. 1 (2024): 84–85. http://dx.doi.org/10.56367/oag-043-11536.

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T cells successfully fighting cancer Else Marit Inderberg and Sébastien Wälchli from Oslo University Hospital explore what we need to know about T cells successfully fighting cancer. Cell-based immunotherapy uses the patient’s own immune cells to target and kill cancer cells in a specific manner. Lymphocytes called T cells are mainly used for this type of therapy, and to target them more efficiently against cancer cells, they are genetically modified to express chimeric antigen receptors (CAR) or T cell receptors (TCR) that bind proteins or peptides presented on the surface of cancer cells. Th

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Moorthy, Rajesh Kannan, Chandhru Srinivasan, Sridharan Jayamohan, et al. "Knockdown of microRNA-375 suppresses cell proliferation and promotes apoptosis in human breast cancer cells." Indian Journal of Science and Technology 14, no. 43 (2021): 3199–209. http://dx.doi.org/10.17485/ijst/v14i43.1719.

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Logtenberg, Meike E. W., and Johannes Boonstra. "Cancer stem cells and addicted cancer cells." Oncology Discovery 1, no. 1 (2013): 7. http://dx.doi.org/10.7243/2052-6199-1-7.

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Rozprawy doktorskie na temat "Cancer cells"

Sarvi, Sana. "Small cell lung cancer and cancer stem cell-like cells." Thesis, University of Edinburgh, 2014. http://hdl.handle.net/1842/9542.

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Small cell lung cancer (SCLC) is a highly aggressive malignancy with extreme mortality and morbidity. Although initially chemo- and radio-sensitive, almost inevitable recurrence and resistance occurs. SCLC patients often present with metastases, making surgery not feasible. Current therapies, rationally designed on underlying pathogenesis, produce in vitro results, however, these have failed to translate into satisfactory clinical outcomes. Recently, research into cancer stem cells (CSCs) has gained momentum and form an attractive target for novel therapies. Based on this concept, CSCs are the

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Fruka, Tayra. "An evaluation of cancer biomarkers in normal ovarian epithelial cells and ovarian cancer cell lines." University of the Western Cape, 2019. http://hdl.handle.net/11394/6920.

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Philosophiae Doctor - PhD Introduction: Globally, there are over 190,000 new reported cases of ovarian cancers per annum. This comprises 3% to 4% of all cancers in women. Ovarian cancer is one of the leading causes of deaths in women. Ovarian cancer is the second most diagnosed gynaecological malignancy and over all the fifth cause leading to death among all types of cancer in the UK in 2004. More than 70% of epithelial ovarian cancers are diagnosed at an advanced stage. Consequently, the prognosis is poor and the mortality rate high. Thus, the survival rate is affected by how far the disea

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Sasaki, Naoya. "Alpha-fetoprotein-producing pancreatic cancer cells possess cancer stem cell characteristics." Kyoto University, 2012. http://hdl.handle.net/2433/157414.

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Wong, Kit-man Sunny, and 王傑民. "Isolation and characterization of cancer stem cells in non-small cell lung cancer." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47250665.

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Tumor heterogeneity has long been observed and recognized as a challenge to cancer therapy. The cancer stem cell (CSC) model is one of the hypotheses proposed to explain such a phenomenon. A specific cancer stem cell marker has not been determined for non-small cell lung cancers (NSCLC), preventing the definitive evaluation of whether the biology of NSCLC is governed by the CSC model. This study aimed to analyze the expression of candidate CSC markers and using the identified putative marker, to isolate CSC and determine the applicability of the CSC model in NSCLC. The expression or

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Liu, Qing. "Curcumin induces cell inhibition in breast cancer cells." Click to view the E-thesis via HKUTO, 2006. http://sunzi.lib.hku.hk/hkuto/record/B38688608.

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Liu, Qing, and 劉晴. "Curcumin induces cell inhibition in breast cancer cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B38688608.

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Oshima, Nobu. "Induction of Cancer Stem Cell Properties in Colon Cancer Cells by Defined Factors." Kyoto University, 2014. http://hdl.handle.net/2433/192147.

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Oshima N, Yamada Y, Nagayama S, Kawada K, Hasegawa S, et al. (2014) Induction of Cancer Stem Cell Properties in Colon Cancer Cells by Defined Factors. PLoS ONE 9(7): e101735. doi:10.1371/journal.pone.0101735 Kyoto University (京都大学) 0048 新制・課程博士 博士(医学) 甲第18547号 医博第3940号 新制||医||1006(附属図書館) 31447 京都大学大学院医学研究科医学専攻 (主査)教授千葉勉, 教授野田亮, 教授武藤学 学位規則第4条第1項該当

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Coulson-Gilmer, Camilla Lucette. "Cancer stem cells and chemoresistance in ovarian cancer." Thesis, University of Leeds, 2017. http://etheses.whiterose.ac.uk/18470/.

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The high mortality rate associated with epithelial ovarian cancer (EOC) is due to its insidious onset, leading to late diagnosis as well as eventual development of chemoresistance in the majority of patients. Cancer stem-like cells (CSCs) are thought to contribute to development of multi-drug resistant (MDR) tumours partly through their high level of ABC-transporter expression, which enables them to survive chemotherapy. ABC-transporter (MRP1, MRP2, BCRP, Pgp) and putative CSC-marker (ALDH1A1, CD44) expression was therefore evaluated by immunohistochemistry in a paraffin-embedded cohort of 57

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Choi, Mi-Yon. "P53 mediated cell motility in H1299 lung cancer cells." VCU Scholars Compass, 2010. http://scholarscompass.vcu.edu/etd/109.

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Studies have shown that gain-of- function mutant p53, AKT, and NFκB promote invasion and metastasis in tumor cells. Signals transduced by AKT and p53 are integrated via negative feedback between the two pathways. Tumor derived p53 was also indicated to induce NFκB gene expression. Due to the close relationship between p53/AKT and p53/NFκB, we hypothesized that AKT and NFκB can enhance motility in cells expressing mutant p53. Effects on cell motility were determined by scratch assays. CXCL5- chemokine is also known to induce cell motility. We hypothesized that enhanced cell motility by AKT and

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Kapeleris, Joanna C. "Circulating tumour cells in non-small cell lung cancer." Thesis, Queensland University of Technology, 2022. https://eprints.qut.edu.au/228607/1/Joanna_Kapeleris_Thesis.pdf.

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Circulating tumour cells (CTCs) have the potential to transform the management of patients with non-small cell lung cancer (NSCLC). The applications of CTCs can identify clinically actionable targets to predict treatment response and to better understand metastasis. CTCs isolated using microfluidics can be used as prognostic indicators of NSCLC as well as characterizing for markers of immunotherapy (PD-L1), molecular targets (ALK, EGFR). Short term cultures were successfully expanded in 9/70 NSCLC patients and cultured for up to 3 months. Optimization of this novel CTC culture model provides o

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Książki na temat "Cancer cells"

Farrar, William L., ed. Cancer Stem Cells. Cambridge University Press, 2009. http://dx.doi.org/10.1017/cbo9780511605536.

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Yu, John S., ed. Cancer Stem Cells. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-59745-280-9.

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Rajasekhar, Vinagolu K., ed. Cancer Stem Cells. John Wiley & Sons, 2014. http://dx.doi.org/10.1002/9781118356203.

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Wiestler, O. D., B. Haendler, and D. Mumberg, eds. Cancer Stem Cells. Springer Berlin Heidelberg, 2007. http://dx.doi.org/10.1007/978-3-540-70853-7.

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Papaccio, Gianpaolo, and Vincenzo Desiderio, eds. Cancer Stem Cells. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7401-6.

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Bapat, Sharmila, ed. Cancer Stem Cells. John Wiley & Sons, Inc., 2008. http://dx.doi.org/10.1002/9780470391594.

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Pat, Moyer Mary, and Poste George, eds. Colon cancer cells. Academic Press, 1990.

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Papaccio, Federica, and Gianpaolo Papaccio, eds. Cancer Stem Cells. Springer US, 2024. http://dx.doi.org/10.1007/978-1-0716-3730-2.

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Sharmila, Bapat, ed. Cancer stem cells. John Wiley & Sons, 2008.

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L, Farrar William, ed. Cancer stem cells. Cambridge University Press, 2009.

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Części książek na temat "Cancer cells"

Ishii, Hideshi, Naotsugu Haraguchi, Keisuke Ieta, Koshi Mimori, and Masaki Mori. "Cancer Stem Cells: Gastrointestinal Cancers." In Stem Cells and Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-933-8_12.

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Sazeides, Christos, and Anne Le. "Metabolic Relationship Between Cancer-Associated Fibroblasts and Cancer Cells." In The Heterogeneity of Cancer Metabolism. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65768-0_14.

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AbstractCancer-associated fibroblasts (CAFs), a major component of the tumor microenvironment (TME), play an important role in cancer initiation, progression, and metastasis. Recent findings have demonstrated that the TME not only provides physical support for cancer cells but also directs cell-to-cell interactions (in this case, the interaction between cancer cells and CAFs). As cancer progresses, the CAFs also coevolve, transitioning from an inactivated state to an activated state. The elucidation and understanding of the interaction between cancer cells and CAFs will pave the way for new ca

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Chowdhury, Suchandra, and Shyamasree Ghosh. "Cancer Stem Cells." In Stem Cells. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-16-1638-9_7.

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Li, Ting, Christopher Copeland, and Anne Le. "Glutamine Metabolism in Cancer." In The Heterogeneity of Cancer Metabolism. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65768-0_2.

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AbstractMetabolism is a fundamental process for all cellular functions. For decades, there has been growing evidence of a relationship between metabolism and malignant cell proliferation. Unlike normal differentiated cells, cancer cells have reprogrammed metabolism in order to fulfill their energy requirements. These cells display crucial modifications in many metabolic pathways, such as glycolysis and glutaminolysis, which include the tricarboxylic acid (TCA) cycle, the electron transport chain (ETC), and the pentose phosphate pathway (PPP) [1]. Since the discovery of the Warburg effect, it h

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Jung, Jin G., and Anne Le. "Metabolism of Immune Cells in the Tumor Microenvironment." In The Heterogeneity of Cancer Metabolism. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65768-0_13.

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AbstractThe tumor microenvironment (TME) is a complex biological structure surrounding tumor cells and includes blood vessels, immune cells, fibroblasts, adipocytes, and extracellular matrix (ECM) [1, 2]. These heterogeneous surrounding structures provide nutrients, metabolites, and signaling molecules to provide a cancer-friendly environment. The metabolic interplay between immune cells and cancer cells in the TME is a key feature not only for understanding tumor biology but also for discovering cancer cells’ vulnerability. As cancer immunotherapy to treat cancer patients and the use of metab

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Jung, Jin G., and Anne Le. "Targeting Metabolic Cross Talk Between Cancer Cells and Cancer-Associated Fibroblasts." In The Heterogeneity of Cancer Metabolism. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-65768-0_15.

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AbstractAlthough cancer has classically been regarded as a genetic disease of uncontrolled cell growth, the importance of the tumor microenvironment (TME) [1, 2] is continuously emphasized by the accumulating evidence that cancer growth is not simply dependent on the cancer cells themselves [3, 4] but also dependent on angiogenesis [5–8], inflammation [9, 10], and the supporting roles of cancer-associated fibroblasts (CAFs) [11–13]. After the discovery that CAFs are able to remodel the tumor matrix within the TME and provide the nutrients and chemicals to promote cancer cell growth [14], many

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Hung, Jaclyn Y. "Cancer Stem Cells: Lung Cancer." In Stem Cells and Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-933-8_14.

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Dosch, Joseph, Cheong Jun Lee, and Diane M. Simeone. "Cancer Stem Cells: Pancreatic Cancer." In Stem Cells and Cancer. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-933-8_15.

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Ayyildiz, Orhan, and Yusuf Hekimoglu. "What Is Immunotherapy and History of Immunotherapy in Cancers." In Immunotherapy in Human Cancers. Nobel Tip Kitabevleri, 2024. http://dx.doi.org/10.69860/nobel.9786053359388.1.

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Immunotherapy, a transformative cancer treatment, harnesses the immune system’s inherent ability to combat neoplastic cells. Ancient medicine hinted at immunological principles, recognizing natural disease resistance and the body’s defensive capabilities. Over time, the understanding of the immune system evolved, with key contributions from early thinkers like Ibn Sina and later scientific pioneers like Ilya Mechnikov. The immune system, comprising innate and adaptive components, is vital in defending against pathogens and regulating cell proliferation, including cancerous cells. The concept o

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Georgiadis, Konstantinos L., Kathryn Simpson, Mahmood Ayub, et al. "Circulating Tumor Cells." In Pancreatic Cancer. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-7193-0_62.

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Streszczenia konferencji na temat "Cancer cells"

Walsh, Alex J. "Autofluorescence lifetime imaging to identify cancer stem cells from bulk cancer cells." In Multiphoton Microscopy in the Biomedical Sciences XXV, edited by Ammasi Periasamy, Peter T. So, and Karsten König. SPIE, 2025. https://doi.org/10.1117/12.3046370.

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Xiao, Xi, Ran Peng, Feng Pan, Sara Coppola, Pietro Ferraro, and Hao Wang. "Digital Holographic Clinical Medical Studies: Insights into Cancer Cell Morphology and Behavior." In Digital Holography and Three-Dimensional Imaging. Optica Publishing Group, 2024. http://dx.doi.org/10.1364/dh.2024.th2a.2.

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Quantitative morphology and refractive index distribution of cancer cells could be pieces of evidence for clinical detection. We show how holographic tomography improve our treatment strategies by providing deeper insights into cancer cell.

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Mugnano, Martina, Zhe Wang, Vincenza Cerbone, et al. "Amnis Image Stream-Analysis of Tumor Cells." In Digital Holography and Three-Dimensional Imaging. Optica Publishing Group, 2024. http://dx.doi.org/10.1364/dh.2024.w4a.22.

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Imaging flow cytometry is a cutting-edge technology for analyzing cell features. Here we show the advantage of using this system to study cell nucleus of tumor cells, using ovarian cancer A2780 cell line as model. Amnis Image stream it has been used as a standard comparison tool respect to quantitative phase imaging to study cell features.

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Tasmi, Tabassum Ahmad, and Alex J. Walsh. "Propofol Treatment Alters the Metabolic State of MDA-MB-231 Breast Cancer Cells." In Novel Techniques in Microscopy. Optica Publishing Group, 2025. https://doi.org/10.1364/ntm.2025.nw2c.6.

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This study investigates the impact of propofol, an intravenous anesthetic drug, on the metabolic behavior of the breast cancer cell line MDA-MB 231, by utilizing fluorescence lifetime imaging microscopy (FLIM). It revealed significant morphological and metabolic changes in the cells, which may reveal a metabolic-component to propofol’s mechanism.

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Akerkouch, Lahcen, Trung B. Le, and Amanda Haage. "Lateral Migration of Cancer Cells in a Microchannel." In 2024 Design of Medical Devices Conference. American Society of Mechanical Engineers, 2024. http://dx.doi.org/10.1115/dmd2024-1096.

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Abstract This work presents the development of a novel approach to model cancer cell dynamics in microcirculation. The proposed numerical model is based on a hybrid continuum-particle approach. The cancer cell model includes the cell membrane, nucleus, cytoplasm and the cytoskeleton. The Dissipative Particle Dynamics method was employed to simulate the mechanical components. The blood plasma is modeled as a Newtonian incompressible fluid. A Fluid-Structure Interaction coupling, leveraging the Immersed Boundary Method is developed to simulate the cell's response to flow dynamics. The model is a

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Yoshimoto, T., T. Ishikawa, N. Matsuki, et al. "Rheology of Cancer Cells With Different Metastatic Properties." In ASME 2009 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2009. http://dx.doi.org/10.1115/sbc2009-206593.

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Cancer is the leading cause of death in Japan as well as many other countries. One of the most serious problem of cancer is that cancer cells often migrate to a distant part of the body, referred to as metastasis. The rheological properties of cancer cells have been investigated by some reserchers [1,2]. However, the correlations between the metastasis and the rheological properties are still unclear, because of limited number of experimental cases reported so far. In this study, we used two kinds of human breast cancer cell lines, MCF-7 and KPL-4. It is known that KPL-4 has much higher metast

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Salmanzadeh, Alireza, Harsha Kittur, Michael B. Sano, et al. "Investigating Dielectrophoretic Signature of Mouse Ovarian Surface Epithelial Cells, Macrophages and Fibroblasts." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80872.

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Epithelial ovarian carcinomas are the fourth leading cause of death in women in the United States among all cancers and the leading cause of death from gynecological malignancies1. The main reason for this high rate of mortality is the inability to properly detect these carcinomas early. Investigations for diagnosing ovarian cancer in early stages have been hindered by two major obstacles: lack of adequate cell models to study different cancer stages and lack of a reliable technique to isolate these cancer cells from peritoneal fluid. In trying to solve the first challenge, Dr. Schmelz and col

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Kitagawa, H., N. Yamamoto, G. Kosaki, and H. Yamazaki. "AN IMPORTANT ROLE OF CARBOHYDRATE MOIETIES ON CANCER CELL MEMBRANE GLYCOPROTEINS IN CANCER CELL-INDUCED PLATELET AGGREGATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644667.

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Platelet aggregation induced by cancer cells may be an essential process in the development of hematogenous metastasis of cancers. A mechanism in HMV-I (human vaginal melanoma cell line)-induced platelet aggregation was studied by using monoclonal antibodies against membrane proteins of cancer cells or platelets. HMV-I cells or their membrana ractions induced platelet aggregation of human heparinized PRP, to which hirudin had no inhibitory effect. The platelet aggregation by HMV-I was completely lost after the pretreatment of the cells with 0.3U/ml neuraminidase for 60 min at 37°C. Preincubati

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Cohen-Armon, Malka. "An Unveiled Cell Death Mechanism Exclusive to Human Cancer Cells." In Cells 2023. MDPI, 2023. http://dx.doi.org/10.3390/blsf2023021014.

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Di Donato, Marzia, Pia Giovannelli, Antimo Migliaccio, and Gabriella Castoria. "New Approaches Targeting the Invasive Phenotype of Prostate Cancer-Associated Fibroblasts." In Cells 2023. MDPI, 2023. http://dx.doi.org/10.3390/blsf2023021001.

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Raporty organizacyjne na temat "Cancer cells"

Alessa, Mohammed, Tayba Wahedi, Jumanah Alsairafi, et al. Prevalence of Thyroid cancer in Saudi Arabis: Systematic review and Meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.9.0088.

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Review question / Objective: What is the prevalence of Thyroid cancer among population in kingdom of Saudi Arabia?. The aim of this systematic review is to scrutinize the prevalence of thyroid cancer (TC) in Saudi Arabia and assess the relative frequency of subgroups related to types of thyroid cancer, age, and gender. Condition being studied: Thyroid cancer is an abnormal growth of cells that starts in the thyroid gland. There is four types of differentiated thyroid cancer, three of these cancer develop from the follicular cells, the papillary thyroid cancer, follicular thyroid cancer, Hürthl

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Chanvorachote, Pithi. Roles of nitric oxide, reactive oxygen species, and their derivatives in regulation of lung cancer cell metastasis. Chulalongkorn University, 2013. https://doi.org/10.58837/chula.res.2013.28.

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The capability of cancer cells to resist to anoikis, migrate and invade surrounding tissues is associated with high metastatic potential and advanced stage of cancers. Recently, caveolin-1 (Cav-1) protein has garnered increased attention in implicating the aggressive behavior of cancer cells. We demonstrate herein that nitric oxide and hydrogen peroxide play a role in inhibiting anoikis process of lung cancer cells via caveolin-1 dependent mechanism. The Cav-1 function in inhibition of anoikis was demonstrated to be cause through Mcl-1 dependent mechanism. The present study demonstrated that C

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Jones, Jonathan. Cell-Matrix Interactions in Breast Cancer Cells. Defense Technical Information Center, 1995. http://dx.doi.org/10.21236/ada300395.

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Yompakdee, Chulee, and Sittiruk Roytrakul. Molecular target of an anti-cancer compound from leaves of Clausena harmandiana (Pierre). Chulalongkorn University, 2016. https://doi.org/10.58837/chula.res.2016.32.

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Clausena harmandiana (Pierre) Guillaumin or Song faa dong (in Thai), is classified in Family Rutaceae. Previous study, a coumarin compound designated CHA-01 was isolated from leave extract of C. harmandiana with inhibitory activity against calcium signaling in a ZDS1 null mutant yeast Saccharomyces cerevisiae (delta zds1). However, not much has been known on biological activity of this coumarin. In the past, some other coumarins were reported to contain anti-cancer activity. The aim of this research was to study molecular mechanism on antiproliferation activity of CHA-01 in Jurkat T cells. The

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Brooks, James D. Single Cell Characterization of Prostate Cancer-Circulating Tumor Cells. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada596639.

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Brooks, James B. Single Cell Characterization of Prostate Cancer Circulating Tumor Cells. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada550987.

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Quinn, Timothy P. Killing Prostate Cancer Cells and Endothelial Cells with a VEGF-Triggered Cell Death Receptor. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada415526.

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Quinn, Timothy P. Killing Prostate Cancer Cells and Endothelial Cells With a VEGF-Triggered Cell Death Receptor. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada423810.

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Quinn, Timothy P. Killing Prostate Cancer Cells and Endothelial Cells with a VEGF-Triggered Cell Death Receptor. Defense Technical Information Center, 2005. http://dx.doi.org/10.21236/ada476353.

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Markovic, Dubravka, and Edward P. Cohen. Treatment of Breast Cancer with Immunogenic Cells Transfected with DNA from Breast Cancer Cells. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada396744.

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