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Rozprawy doktorskie na temat „Carcinoma breast”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

LeBedis, Christina. "Lymph node involvement in breast carcinoma metastasis." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31255.

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Since lymph node stromal cells remain largely uncharacterized with respect to cell surface markers and function, their role in regulating the growth and invasion of disseminated cancer cells, including breast carcinoma has, to date, been virtually unexplored. In the present study, we asked whether peripheral lymph node cells could modulate the growth of breast carcinoma cells and, thereby, contribute to the progression of the metastatic process. Primary cultures of rat peripheral lymph node stromal cells were obtained by limiting dilution and two sublines, STA4 and STB12, with breast carcinoma

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2

Hosein, Abdel Nasser. "Fundamental investigations into breast carcinoma-associated fibroblasts." Thesis, McGill University, 2012. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=107576.

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Breast cancer stroma is a heterogenous mixture of cells types which, in addition to the carcinomatous epithelium itself, consists predominately of endothelium, adipocytes, immune infiltrates and fibroblasts. Recently there has been a movement towards characterizing each of these cells types found within the cancer stroma and understanding how they ultimately impact the disease. The cancer-associated fibroblast (CAF) has garnered a great deal of this attention and numerous in vivo models models have shown the predominance of several pathways CAFs may regulate in order to promote breast cancer p

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3

Talvensaari-Mattila, A. (Anne). "MMP-2 immunoreactive protein in breast carcinoma and neoplastic cervical lesions:mMP-2 is a new prognostic factor in breast carcinoma." Doctoral thesis, University of Oulu, 1999. http://urn.fi/urn:isbn:9514254023.

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Abstract Tumor invasion and metastasis are the major causes of treatment failure or death for carcinoma patients. Matrix metalloproteinases (MMPs) are zinc dependent endopeptidases implicated in tumor invasion and metastasis. The expression of MMP-2 has been previously linked to invasiveness of carcinoma cells. The MMP-2 immunoreactive protein was studied here in squamous cell carcinoma of the utrine cervix and in adenocarcinoma of the breast by using a specific monoclonal antibody in immunohistochemical stainings. Immunoreactive protein of latent MMP-2 was found to be an early event in neopla

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4

Wärnberg, Fredrik. "Prognosis in carcinoma in situ of the breast." Doctoral thesis, Uppsala University, Department of Surgical Sciences, 2000. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-484.

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<p>The incidence of breast cancer is rising steadily in Sweden and the proportion of carcinoma in situ (CIS) has increased appreciably, most likely due to mammography screening. The aim of this study was twofold: (1) to examine risk factors for subsequent invasive breast carcinoma and breast cancer death after primary ductal carcinoma in situ (DCIS) and (2) to study the biology in the progress between in situ and invasive carcinoma.</p><p> In a cohort-study based on 3,398 women with a primary CIS reported to the Swedish Cancer Registry (SCR) 1980-1992, women diagnosed in 1989-1992 ran a

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5

Karihtala, P. (Peeter). "Oxidative damage and counteracting mechanisms in breast carcinoma." Doctoral thesis, University of Oulu, 2006. http://urn.fi/urn:isbn:9514279530.

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Abstract Breast cancer is the leading cause of death from cancer among Finnish women, but the ultimate causation of carcinogenesis still remains unclear. Reactive oxygen species (ROS) is a collective term for several types of reactive oxygen metabolites that are continuously generated in human cells mainly as by-products of aerobic respiration. ROS, including nitric oxide and its derivatives, play highly important roles in cell physiology. If ROS production exceeds the capacity of detoxification systems, principally antioxidant enzymes, oxidative stress is said to occur. This state is known to

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6

Harbord, Sara Helen Alison. "X chromosome studies and breast and ovarian carcinoma." Thesis, University of British Columbia, 2007. http://hdl.handle.net/2429/212.

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Skewed somatic X inactivation (XCI), X-linked gene overexpression and abnormal X content have been associated with breast and ovarian cancer. Partial or complete reactivation of the inactive X in females may be a step in breast and ovarian cancer progression, leading to overexpression of some tumour enhancing gene. Markers of an X reactivation event were examined: X gene dosage, expression, and methylation in 8 ovarian cancer cell lines. Another marker of an X reactivation event, skewed XCI, was assayed in peripheral blood DNA from 106 breast and/or ovarian cancer patients (52 BRCA1 mutation c

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7

Rakha, Emad Abd El-A. "Phenotypic and genotypic characteristics of human breast carcinoma." Thesis, University of Nottingham, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.417122.

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8

Lee, Andrew Hilary Schokman. "Inflammation and vascularity in carcinoma of the breast." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612226.

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9

Manouchehri, Jasmine Marie. "RhTRAIL with Sensitizing Agents for Breast Carcinoma Treatment." Cleveland State University / OhioLINK, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=csu1543316657492919.

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10

Zhou, Wenjing. "Aspects of Progression in Breast Carcinoma : from ductal carcinoma in situ to invasive cancer." Doctoral thesis, Uppsala universitet, Institutionen för kirurgiska vetenskaper, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-166745.

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In the past decades our knowledge concerning breast cancer progression from ductal carcinoma in situ (DCIS) to invasive cancer has grown rapidly. However, molecular factors driving the progression are still largely unknown. In the first study, we investigated tumor evolution in breast cancer by analyzing TP53 mutation status in tumors from various stages of the disease. Presence of the same TP53 mutations in both DCIS and invasive components from the same tumor indicates same cellular origin. The role of mutant TP53 in the progression of breast cancer is less clear and may vary between subtype

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11

Webster, Lucy R. "Diagnostic molecular profiling of ductal carcinoma in situ of the breast." Thesis, The University of Sydney, 2007. https://hdl.handle.net/2123/28109.

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The introduction of population-based mammographic screening has led to a dramatic increase in the diagnosis of ductal carcinoma in situ (DCIS) of the breast; with DCIS now accounting for approximately 25-35% of all screen-detected breast cancers. Epidemiological, histopathological and molecular studies have provided compelling evidence to support the notion that DCIS is a direct precursor of invasive cancer; although such data also indicate that DCIS represents a highly heterogeneous group of lesions. Some women with DCIS will develop life-threatening invasive cancer, whereas others may

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12

Wang, Chao. "The role of hyaluronan receptor RHAMM in breast carcinoma." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape10/PQDD_0004/NQ41526.pdf.

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13

Ali, Shahina. "Molecular mechanisms of eosinophil recruitment to breast carcinoma sites." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2001. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/MQ64939.pdf.

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14

Sinha, Prakash. "Investigation into the Factors Influencing Outcomes in Breast Carcinoma." Thesis, University of Kent, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.499672.

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15

Warzecha, Hind [Verfasser], and Falko [Akademischer Betreuer] Fend. "Invasive Micropapillary Breast Carcinoma / Hind Warzecha ; Betreuer: Falko Fend." Tübingen : Universitätsbibliothek Tübingen, 2018. http://d-nb.info/1199268836/34.

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16

Yang, Jie. "Identification of Paclitaxel-induced Cytokines in Breast Carcinoma Cells." OpenSIUC, 2019. https://opensiuc.lib.siu.edu/theses/2587.

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Inflammatory cytokines and chemokines are known to promote tumor cell survival, invasion, the formation of blood and lymphatic vessels, and hence, metastasis. We previously showed that a pro-inflammatory pathway regulated by Toll-like Receptor-4 (TLR4) can be activated in human breast cancer (BC) cell lines by a clinical chemotherapeutic drug, paclitaxel (PXL). Prior data showed that PXL treatment of TLR4+ tumors in vivo increased inflammation and tumor spread. Here, we used two BC models based on MDA-MB-231 and HCC1806 cell lines. Transcript expression of 123 cytokines in vitro and in vivo wa

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17

Raymond, Wendy Ann. "Immunohistochemcial prognostic parameters in breast carcinoma / Wendy Ann Raymond." 1990, 1990. http://hdl.handle.net/2440/38405.

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18

Uva, V. "FOXP3 EXPRESSION IN BREAST CARCINOMA CELLS AND METASTATIC SPREAD." Doctoral thesis, Università degli Studi di Milano, 2014. http://hdl.handle.net/2434/229557.

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Forkhead box P3 (FOXP3), a gene member of the forkhead/winged-helix family of transcription regulators, is implicated in regulating immune system development and function. This gene has been found to be of crucial importance for the generation of CD4+CD25+ regulatory T cells (Tregs). Tregs express both the full-length FOXP3 and Δ2FOXP3 natural splice variant. In addition to its expression in the lymphocyte lineage, studies have recently described FOXP3 expression in non-hematopoietic-derived cells, including cancerous or normal epithelial cells of multiple lineages and origins. The role of FO

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19

Cowan, William Keith. "Observations on the pathology of screen-detected mammary carcinoma." Thesis, University of Newcastle Upon Tyne, 1994. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.238784.

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20

Habel, Laurel A. "Risk of recurrence following ductal carcinoma in situ of the breast /." Thesis, Connect to this title online; UW restricted, 1996. http://hdl.handle.net/1773/10931.

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21

McManus, Damian Terence. "Somatic genetic and chromosomal abnormalities in ovarian and breast carcinoma." Thesis, Queen's University Belfast, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.263336.

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22

CAMPIONI, GLORIA. "Monolayers and three-dimensional cultures to investigate metabolic reprogramming in breast and bladder cancer." Doctoral thesis, Università degli Studi di Milano-Bicocca, 2022. http://hdl.handle.net/10281/375413.

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La riprogrammazione metabolica è stata osservata in molti tipi di cancro ed è considerata un tratto distintivi di questa malattia eterogenea multifattoriale. Comprendere i meccanismi che portano al riarrangiamento metabolico e come queste attività promuovono l’attivazione di proprietà maligne nel cancro può aiutare a sfruttare le alterazioni metaboliche a beneficio terapeutico. Nei tumori solidi le cellule tumorali interagiscono con il complesso habitat del microambiente tumorale (TME) che può modulare il metabolismo delle cellule tumorali e la loro sensibilità o resistenza al trattamento farm

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23

Kaira, Mustapha. "In situ molecular profilling of the microenvironment of breast carcinoma." Thesis, Uppsala universitet, Institutionen för biologisk grundutbildning, 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-265258.

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High stromal PDGF receptor B expression was shown to have strong prognostic value in a studyinvolving over 600 breast cancer patients however, the molecular role of the receptor in tumordevelopment remains unclear. In this project we studied the spatial distribution and expressionlevels of a panel genes and markers associated with PDGF signaling, in breast cancer tumormicroenvironment (TME) using a newly developed technique -in situ sequencing. The techniquerelies on padlock probes which we validated with corresponding RNA sequencing, microarray,and immunohistochemistry data. Our results showe

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24

Garimella, Veerabhadram. "Molecular heterogeneity of multifocal invasive ductal carcinoma of the breast." Thesis, University of Hull, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.445303.

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25

Petridis, Christos. "Molecular genetics of lobular breast cancer ductal carcinoma in situ." Thesis, King's College London (University of London), 2018. https://kclpure.kcl.ac.uk/portal/en/theses/molecular-genetics-of-lobular-breast-cancer-ductal-carcinoma-in-situ(788866a0-2bd3-4fa7-82b0-4135f4823d1a).html.

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Ductal carcinoma in situ (DCIS) and lobular carcinoma in situ (LCIS) are clinically undetectable forms of non-invasive breast cancer. DCIS is considered a non-obligate precursor of invasive ductal carcinoma (IDC). LCIS shares many of the same genetic aberrations as invasive lobular breast cancer (ILC), which accounts for 10-15% of all invasive breast cancer. With the advent of screening mammography, the diagnosis of pure DCIS (with no invasive component) and LCIS has become more common, and approximately 20% of screen detected tumours are pure DCIS. The aim of this project is to test the hypot

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26

Koterba, Kristen L. "Regulation of Autophagy and Cell Death in Breast Carcinoma Cells." University of Toledo Health Science Campus / OhioLINK, 2010. http://rave.ohiolink.edu/etdc/view?acc_num=mco1276005638.

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27

Starobinska, Ella. "Is HPV a Culprit For Breast Carcinoma? A Retrospective Study." Thesis, The University of Arizona, 2017. http://hdl.handle.net/10150/623542.

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A Thesis submitted to The University of Arizona College of Medicine - Phoenix in partial fulfillment of the requirements for the Degree of Doctor of Medicine.<br>The role of viruses in most common cancers is undoubtedly important, yet highly underestimated. Human Papilloma Virus (HPV) has been implicated with 99.7% of cervical cancers and its oncogenic mechanism has been clearly identified. The association of cervical cancer with 90% of triple negative breast cancer (TNBC) cases in African countries creates an intriguing possibility of HPV being a candidate oncovirus for breast cancer. In fact

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28

Moriarty, Charlotte M. Harwood. "Functional Analysis of MicroRNA-10b in Breast Carcinoma: A Dissertation." eScholarship@UMMS, 2009. https://escholarship.umassmed.edu/gsbs_diss/426.

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MicroRNAs (miRNAs) represent a class of small noncoding RNAs that regulate gene expression. Recent studies have shown that miRNAs are mis-expressed in various human cancers and that some miRNAs have the potential to act as tumor suppressors or oncogenes. MiR-10b is one miRNA that has been shown to be deregulated in breast cancer. However, current findings regarding miR-10b’s role in breast cancer are controversial. MiR-10b was originally reported to be downregulated in breast cancer compared to normal breast tissue. Subsequently, miR-10b was argued to be upregulated in metastatic breast cancer

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29

Gerson, Kristin D. "Analysis of Integrin α6β4 Function in Breast Carcinoma: A Dissertation". eScholarship@UMMS, 2012. https://escholarship.umassmed.edu/gsbs_diss/588.

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The development and survival of multicellular organisms depends upon the ability of cells to move. Embryogenesis, immune surveillance, wound healing, and metastatic disease are all processes that necessitate effective cellular locomotion. Central to the process of cell motility is the family of integrins, transmembrane cell surface receptors that mediate stable adhesions between cells and their extracellular environment. Many human diseases are associated with aberrant integrin function. Carcinoma cells in particular can hijack integrins, harnessing their mechanical and signaling potential to

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30

Mannu, Gurdeep Singh. "Epidemiology of ductal carcinoma in situ." Thesis, University of Oxford, 2017. http://ora.ox.ac.uk/objects/uuid:c78ce9f9-b5f6-4166-a53e-5d79819c7a28.

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<b>Introduction:</b> Almost 7,000 people are diagnosed with ductal carcinoma in situ (DCIS) in the United Kingdom each year, but there remains uncertainty regarding its natural history and optimal management. The aim of this thesis was to evaluate factors contributing to the epidemiology of DCIS and its outcomes. <b>Methods:</b> 1) A cohort study comparing risk factors for DCIS and invasive breast cancer (IBC) using UK Biobank; 2) A cohort study examining the accuracy of preoperative biopsy in DCIS using clinical records from the Netherlands Cancer Institute; 3) A cohort study examining the ra

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31

Bianchessi, Susana Trigo. "Ressonancia magnetica na avaliação da extensão local do carcinoma mamario." [s.n.], 2008. http://repositorio.unicamp.br/jspui/handle/REPOSIP/310657.

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Orientadores: Marcelo Alvarenga, Cesar Cabello dos Santos<br>Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciencias Medicas<br>Made available in DSpace on 2018-08-10T14:34:16Z (GMT). No. of bitstreams: 1 Bianchessi_SusanaTrigo_M.pdf: 5977471 bytes, checksum: 691416790e0a9f01b1b82619e5b6e1f3 (MD5) Previous issue date: 2008<br>Resumo: Introdução: A correta avaliação da extensão local do câncer de mama é necessária para a escolha do tratamento local entre cirurgia conservadora e mastectomia. Objetivo: Avaliar se a ressonância magnética (RM) é um bom preditor do tamanh

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32

Kiltie, Anne Elizabeth. "DNA damage as a predictor of normal tissue response to radiotherapy." Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.244711.

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33

Lui, Lik-hang Eric. "Aberrant methylation of E-cadherin gene (ECAD) in invasive ductal breast carcinoma /." View the Table of Contents & Abstract, 2005. http://sunzi.lib.hku.hk/hkuto/record/B31361912.

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Prochazka, Michaela. "The risk of second primary lung carcinoma in breast cancer patients /." Stockholm, 2006. http://diss.kib.ki.se/2006/91-7140-649-2/.

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35

Williams, Cory S. M. "The role of heat shock cognate 70 in human breast carcinoma." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.365303.

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36

O'Sullivan, Colette. "Epithelial mucins associated with breast carcinoma : purification, characterisation and immunological investigation." Thesis, University of Nottingham, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.290401.

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37

Schlippe, Maria von. "The relationship between E cadherin and integrins in breast carcinoma cells." Thesis, King's College London (University of London), 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.271648.

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38

Teo, Katy Ann. "Elucidating biological mechanisms associated with invasive lobular carcinoma of the breast." Thesis, University of Edinburgh, 2017. http://hdl.handle.net/1842/23628.

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Breast cancer is a heterogeneous disease, and can be classified according to histological subtypes based on cellular morphology. Invasive ductal carcinoma (IDC) and invasive lobular carcinoma (ILC) are the most common histological subtypes, accounting for approximately 80% and 12% of cases respectively. ILC exhibits a number of distinct clinico-pathological features in comparison with IDC, and is understudied as a breast cancer subtype. ILC tumours are typically oestrogen receptor positive, HER2 negative, and frequently demonstrate early loss of Ecadherin expression, which is a hallmark of the

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39

Shkandala, A. Yu. "Effect of ionizing radiation on growth and apoptosis of breast carcinoma." Thesis, Видавництво СумДУ, 2012. http://essuir.sumdu.edu.ua/handle/123456789/27507.

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Mahoney, Brent P. "The chemotherapeutic consequences of tumor pH in breast carcinoma model systems." Diss., The University of Arizona, 2002. http://hdl.handle.net/10150/280195.

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We hypothesize that acid-outside transplasmalemmalar pH gradients, typical of those found in drug-insensitive breast tumors, will result in drug resistance by the exclusion of weakly basic chemotherapeutic agents via ion trapping. We tested our hypothesis in vitro by evaluating pH dependent cytotoxicity of various clinically relevant chemotherapeutic drugs in the MCF-7 human breast adenocarcinoma cell line. The results of this work were consistent with our hypothesis, namely that (i) weak acids are more toxic in acidic extracellular media, (ii) weak bases are more toxic in basic extracellular

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41

Conde, Sandro José [UNESP]. "Inter-relação da modulação gênica entre triidotironina(T3) e estradiol(E2) em cultura primária de adrenocarcinoma de mama." Universidade Estadual Paulista (UNESP), 2008. http://hdl.handle.net/11449/100932.

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Made available in DSpace on 2014-06-11T19:31:04Z (GMT). No. of bitstreams: 0 Previous issue date: 2008-02-22Bitstream added on 2014-06-13T19:40:52Z : No. of bitstreams: 1 conde_sj_dr_botfm.pdf: 1083233 bytes, checksum: a328bfb8396940e543c7e1865edd1942 (MD5)<br>Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)<br>Sabe-se que o E2 e o status hormonal da paciente são importantes para a proliferação e o tratamento do câncer de mama, quanto ao T3, apesar dos estudos epidemiológicos serem ainda contraditórios em relação a sua influência no câncer de mama, estudos laboratoriais demonstr

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42

Rehman, Shazza. "A study of the molecular pathology of ductal carcinoma in situ and invasive ductal carcinoma of the breast." Thesis, University College London (University of London), 2005. http://discovery.ucl.ac.uk/1445073/.

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The biological validity of the histopathological classification of ductal carcinoma in situ (DCIS) of the breast was evaluated in this study by correlating the three histopathological grades of DCIS to immunohistochemical expression of Ki67, p53, cerbB-2, markers of poor prognosis in invasive ductal carcinoma (IDC) and also to bcl2 and ER, markers of good prognosis in invasive breast cancer. DCIS grades correlated positively to Ki67, p53, cerbB-2 and negatively to bcl2 and ER, suggesting validity of the classification. The incidence of bax protein expression was determined immunohistochemicall

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43

Ringberg, Hagberg Anita. "In situ carcinoma of the breast aspects on natural history and treatment with special reference to subcutaneous mastectomy /." Malmö : Dept. of Plastic and Reconstructive Surgery, Lund University, Malmö General Hospital, 1992. http://books.google.com/books?id=qHVsAAAAMAAJ.

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44

King, John F. "Comparisons of gene expression patterns in progressive breast carcinoma and the adjacent stromal microenvironment." Fairfax, VA : George Mason University, 2007. http://hdl.handle.net/1920/2949.

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Thesis (M.S.)--George Mason University, 2007.<br>Title from PDF t.p. (viewed Jan. 18, 2008). Thesis directors: Donald Seto, Anna Baranova. Submitted in partial fulfillment of the requirements for the degree of Master of Science in Bioinformatics. Vita: p. 105. Includes bibliographical references (p. 100-104). Also available in print.

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45

Biazús, Jorge Villanova. "Valor prognóstico e preditivo dos marcadores imunoistoquímicos no carcinoma invasor de mama." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2007. http://hdl.handle.net/10183/12105.

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Base teórica: Nas últimas décadas, o prognóstico do câncer inicial da mama tem sido baseado nas características clínicas das pacientes e em aspectos histológicos dos tumores. Nos últimos anos, subtipos moleculares de carcinoma de mama foram identificados através de estudos de perfil genético por “DNA microarray”. Esta nova classificação pode melhorar a avaliação prognóstica, porém ainda não está disponível e é de alto custo. A possibilidade de identificar subtipos moleculares através de métodos mais simples e de menor custo é particularmente importante em países com recursos limitados. A ident

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46

Denton, Graeme. "Immunological recognition of the tumour related mucin MUC1." Thesis, University of Nottingham, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284052.

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47

Harihar, Sitaram. "The Role of Phosphoinositide Signaling in Breast Cancer Metastasis Suppressor 1-Mediated Metastasis Suppression of Human Breast Carcinoma Cells." DigitalCommons@USU, 2011. https://digitalcommons.usu.edu/etd/870.

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Breast cancer is the most common non-skin cancer in women and the second most common cause of cancer-related death in U.S. women. Despite numerous advances in treatment strategies against breast cancer, the presence of undetected distant metastasis of the primary tumor remains the main cause of mortality. Current screening and detection methods such as mammograms are simply not sensitive enough to detect formation of metastasis. Further, currently available therapies against metastatic breast cancer do not provide a complete cure for the disease. Thus, understanding the biology and molecular f

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Worthington, Tim Rees. "The role of thrombomodulin in carcinoma of the human pacreas and breast." Thesis, Imperial College London, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.396306.

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Lui, Lik-hang Eric, and 雷力恒. "Aberrant methylation of E-cadherin gene (ECAD) in invasive ductal breast carcinoma." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B4501016X.

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Mahmood, Sardar. "Identification of New Oncogenes Involved in the Tumoral Progression of Breast Carcinoma." Thesis, Paris 11, 2012. http://www.theses.fr/2012PA11T021.

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La disponibilité à la fois des données à grande échelle du transcriptome et du génome de tumeurs permet maintenant d'identifier assez facilement des oncogènes candidats, gènes qui sont surexprimés en conséquence de l'amplification d'ADN. Ces oncogènes candidats doivent alors être fonctionnellement validés et leur rôle dans la cellule normale et tumorale doit être étudié.Dans cette étude, nous nous sommes principalement focalisés sur le cancer du sein, le cancer le plus fréquent chez les femmes et la deuxième cause de décès par cancer chez les femmes à travers le monde. En France, 52.000 nouvea

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