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Artykuły w czasopismach na temat „CD169”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 1 lutego 2022

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1

Bogie, Jeroen FJ, Ellen Boelen, Els Louagie, et al. "CD169 is a marker for highly pathogenic phagocytes in multiple sclerosis." Multiple Sclerosis Journal 24, no. 3 (2017): 290–300. http://dx.doi.org/10.1177/1352458517698759.

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Background: Phagocytes, such as macrophages and microglia, are key effector cells in the pathophysiology of multiple sclerosis (MS). It is widely accepted that they instigate and promote neuroinflammatory and neurodegenerative events in MS. An increasing amount of studies indicate that Siglec-1, also known CD169, is a marker for activated phagocytes in inflammatory disorders. Objective: In this study, we set out to define how CD169+ phagocytes contribute to neuroinflammation in MS. Methods: CD169-diphtheria toxin receptor (DTR) mice, which express human DTR under control of the CD169 promoter,

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Affandi, Alsya J., Joanna Grabowska, Katarzyna Olesek, et al. "Selective tumor antigen vaccine delivery to human CD169+antigen-presenting cells using ganglioside-liposomes." Proceedings of the National Academy of Sciences 117, no. 44 (2020): 27528–39. http://dx.doi.org/10.1073/pnas.2006186117.

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Priming of CD8+T cells by dendritic cells (DCs) is crucial for the generation of effective antitumor immune responses. Here, we describe a liposomal vaccine carrier that delivers tumor antigens to human CD169/Siglec-1+antigen-presenting cells using gangliosides as targeting ligands. Ganglioside-liposomes specifically bound to CD169 and were internalized by in vitro-generated monocyte-derived DCs (moDCs) and macrophages and by ex vivo-isolated splenic macrophages in a CD169-dependent manner. In blood, high-dimensional reduction analysis revealed that ganglioside-liposomes specifically targeted

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van Dinther, Dieke, Miguel Lopez Venegas, Henrike Veninga, et al. "Activation of CD8+ T Cell Responses after Melanoma Antigen Targeting to CD169+ Antigen Presenting Cells in Mice and Humans." Cancers 11, no. 2 (2019): 183. http://dx.doi.org/10.3390/cancers11020183.

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The lack of tumor-reactive T cells is one reason why immune checkpoint inhibitor therapies still fail in a significant proportion of melanoma patients. A vaccination that induces melanoma-specific T cells could potentially enhance the efficacy of immune checkpoint inhibitors. Here, we describe a vaccination strategy in which melanoma antigens are targeted to mouse and human CD169 and thereby induce strong melanoma antigen-specific T cell responses. CD169 is a sialic acid receptor expressed on a subset of mouse splenic macrophages that captures antigen from the blood and transfers it to dendrit

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Twilhaar, Maarten Nijen, Lucas Czentner Colomo, Joanna Grabowska, et al. "849 Optimization of a GM3-containing liposomal vaccine that delivers antigen to CD169+ splenic macrophages." Journal for ImmunoTherapy of Cancer 8, Suppl 3 (2020): A902. http://dx.doi.org/10.1136/jitc-2020-sitc2020.0849.

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BackgroundAlthough promising developments in cancer vaccination have been made, therapeutic effectiveness is often insufficient. Liposomal vaccine effectiveness could be enhanced by antigen encapsulation and incorporation of molecules that actively target to antigen presenting cells to enhance T cell activation. CD169-expressing splenic macrophages are located in the marginal zone and efficiently capture particulate antigens such as viruses and exosomes from the blood circulation. Upon antigen capture CD169+ macrophages transfer antigen to cross-presenting dendritic cells that are responsible

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5

Ludewig, Burkhard, and Luisa Cervantes-Barragan. "CD169+ macrophages take the bullet." Nature Immunology 13, no. 1 (2011): 13–14. http://dx.doi.org/10.1038/ni.2189.

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Nijen Twilhaar, Maarten K., Lucas Czentner, Joanna Grabowska, et al. "Optimization of Liposomes for Antigen Targeting to Splenic CD169+ Macrophages." Pharmaceutics 12, no. 12 (2020): 1138. http://dx.doi.org/10.3390/pharmaceutics12121138.

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Despite promising progress in cancer vaccination, therapeutic effectiveness is often insufficient. Cancer vaccine effectiveness could be enhanced by targeting vaccine antigens to antigen-presenting cells, thereby increasing T-cell activation. CD169-expressing splenic macrophages efficiently capture particulate antigens from the blood and transfer these antigens to dendritic cells for the activation of CD8+ T cells. In this study, we incorporated a physiological ligand for CD169, the ganglioside GM3, into liposomes to enhance liposome uptake by CD169+ macrophages. We assessed how variation in t

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7

Li, Wei, Yaomei Wang, Huizhi Zhao, et al. "Identification, Isolation and Transcriptome Analyses of Mouse, Rat and Man Erythroblastic Island Central Macrophages." Blood 132, Supplement 1 (2018): 841. http://dx.doi.org/10.1182/blood-2018-99-114188.

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Abstract Erythroblastic island (EBI), composed of a central macrophage and surrounding erythroid cells, is the first hematopoietic niche discovered for erythropoiesis. Yet, the identity of the central macrophage has so far remained elusive. Based on the previous findings that F4/80, VCAM1 and CD169 are potential mouse central macrophage markers, we first calculated the number of F4/80+VCAM1+CD169+ mouse macrophages in the mouse bone marrow and compared it to the number of Ter119+ erythroblasts. We found that the ratio of F4/80+VCAM1+CD169+ macrophage and erythroblasts is about 1:2. Given the f

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8

Topf, Michael C., Larry Harshyne, Madalina Tuluc, et al. "Loss of CD169+ Subcapsular Macrophages during Metastatic Spread of Head and Neck Squamous Cell Carcinoma." Otolaryngology–Head and Neck Surgery 161, no. 1 (2019): 67–73. http://dx.doi.org/10.1177/0194599819829741.

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Objective The purpose of this study is to assess CD169 expression in metastatic and nearby tumor-free lymph nodes of patients with head and neck squamous cell carcinoma (SCC). Study Design Retrospective analysis based on immunohistochemistry. Setting Tertiary care center. Subjects and Methods The abundance of CD169+ cells in the subcapsular sinuses (SCSs) of lymph nodes was assessed immunohistochemically in paraffin-embedded tissue samples derived from 22 patients with oral cavity and oropharyngeal SCC. Results SCSs of lymph nodes harboring metastatic SCC contained significantly fewer CD169+ m

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9

Grabowska, Joanna, Dorian A. Stolk, Maarten K. Nijen Twilhaar та ін. "Liposomal Nanovaccine Containing α-Galactosylceramide and Ganglioside GM3 Stimulates Robust CD8+ T Cell Responses via CD169+ Macrophages and cDC1". Vaccines 9, № 1 (2021): 56. http://dx.doi.org/10.3390/vaccines9010056.

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Successful anti-cancer vaccines aim to prime and reinvigorate cytotoxic T cells and should therefore comprise a potent antigen and adjuvant. Antigen targeting to splenic CD169+ macrophages was shown to induce robust CD8+ T cell responses via antigen transfer to cDC1. Interestingly, CD169+ macrophages can also activate type I natural killer T-cells (NKT). NKT activation via ligands such as α-galactosylceramide (αGC) serve as natural adjuvants through dendritic cell activation. Here, we incorporated ganglioside GM3 and αGC in ovalbumin (OVA) protein-containing liposomes to achieve both CD169+ ta

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10

Doehn, Jan-Moritz, Christoph Tabeling, Robert Biesen, et al. "CD169/SIGLEC1 is expressed on circulating monocytes in COVID-19 and expression levels are associated with disease severity." Infection 49, no. 4 (2021): 757–62. http://dx.doi.org/10.1007/s15010-021-01606-9.

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AbstractCoronavirus disease 2019 (COVID-19) is caused by infection with severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2). Type I interferons are important in the defense of viral infections. Recently, neutralizing IgG auto-antibodies against type I interferons were found in patients with severe COVID-19 infection. Here, we analyzed expression of CD169/SIGLEC1, a well described downstream molecule in interferon signaling, and found increased monocytic CD169/SIGLEC1 expression levels in patients with mild, acute COVID-19, compared to patients with severe disease. We recommend further

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11

Friedrich, Sarah-Kim, Rosa Schmitz, Michael Bergerhausen, et al. "Usp18 Expression in CD169+ Macrophages is Important for Strong Immune Response after Vaccination with VSV-EBOV." Vaccines 8, no. 1 (2020): 142. http://dx.doi.org/10.3390/vaccines8010142.

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Ebola virus epidemics can be effectively limited by the VSV-EBOV vaccine (Ervebo) due to its rapid protection abilities; however, side effects prevent the broad use of VSV-EBOV as vaccine. Mechanisms explaining the efficient immune activation after single injection with the VSV-EBOV vaccine remain mainly unknown. Here, using the clinically available VSV-EBOV vaccine (Ervebo), we show that the cell-intrinsic expression of the interferon-inhibitor Usp18 in CD169+ macrophages is one important factor modulating the anti-Ebola virus immune response. The absence of Usp18 in CD169+ macrophages led to

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12

Alvarez, Belén, Paloma Martínez, María Yuste, et al. "Phenotypic and functional heterogeneity of CD169+ and CD163+ macrophages from porcine lymph nodes and spleen." Developmental & Comparative Immunology 44, no. 1 (2014): 44–49. http://dx.doi.org/10.1016/j.dci.2013.11.010.

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13

Jacobsen, Rebecca, Allison R. Pettit, Liza J. Raggatt, et al. "Mobilizing Doses Of G-CSF Stop Medullary Erythropoiesis By Depleting F4/80+ VCAM1+ ER-HR3+ CD169+ Erythroid-Island Macrophages." Blood 122, no. 21 (2013): 309. http://dx.doi.org/10.1182/blood.v122.21.309.309.

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Abstract G-CSF mobilizes hematopoietic stem cells (HSCs) from the bone marrow (BM) into the blood by suppressing a subset of HSC niche supportive macrophages. As macrophages are the central component of erythropoietic islands in BM, spleen and liver, we examined the effect of G-CSF on erythropoiesis in C57BL/6 mice. Mobilizing doses of G-CSF caused a marked whitening of the BM, a 15-fold decrease in the number of phenotypic erythroblasts, a 1.5-fold decrease in polychromatic and orthochromatic erythroblasts, and a 4.5-fold reduction in reticulocytes in the BM. Conversely, more immature pro-ery

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14

Bernhard, Caroline A., Christine Ried, Stefan Kochanek, and Thomas Brocker. "CD169+ macrophages are sufficient for priming of CTLs with specificities left out by cross-priming dendritic cells." Proceedings of the National Academy of Sciences 112, no. 17 (2015): 5461–66. http://dx.doi.org/10.1073/pnas.1423356112.

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Dendritic cells (DCs) are considered the most potent antigen-presenting cells (APCs), which directly prime or cross-prime MHC I-restricted cytotoxic T cells (CTLs). However, recent evidence suggests the existence of other, as-yet unidentified APCs also able to prime T cells. To identify those APCs, we used adenoviral (rAd) vectors, which do not infect DCs but selectively accumulate in CD169+ macrophages (MPs). In mice that lack DCs, infection of CD169+ MPs was sufficient to prime CTLs specific for all epitopes tested. In contrast, CTL responses relying exclusively on cross-presenting DCs were

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15

GAO, Xin, Philip Boulais, Masato Tanaka, Christopher Richard Marlein, and Paul S. Frenette. "Macrophage Transfer to HSCs Assigns Residence in Bone Marrow." Blood 134, Supplement_1 (2019): 276. http://dx.doi.org/10.1182/blood-2019-123762.

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Hematopoietic stem cells (HSCs), residing at the apex of the hematopoietic hierarchy, are a rare and heterogeneous cell population. Although HSC subsets with various repopulation capacities and lineage bias have been identified, there is no available information about whether each HSC has an equal chance of being mobilized or whether there are specific pools of mobilizable or non-mobilizable HSCs in bone marrow (BM). Here, we identify a BM resident HSC subset based on the expression of the macrophage marker F4/80. Flow cytometry analysis revealed that F4/80 was expressed on 75% of HSCs (Lin- S

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16

Martinez-Pomares, Luisa, and Siamon Gordon. "CD169+ macrophages at the crossroads of antigen presentation." Trends in Immunology 33, no. 2 (2012): 66–70. http://dx.doi.org/10.1016/j.it.2011.11.001.

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Chow, Andrew, Matthew Huggins, Jalal Ahmed, et al. "CD169+ Macrophages Regulate Erythropoiesis Under Homeostasis, Recovery From Erythron Injury and in JAK2V617F-Induced Polycythemia Vera." Blood 120, no. 21 (2012): 80. http://dx.doi.org/10.1182/blood.v120.21.80.80.

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Abstract Abstract 80 The role of macrophages (MΦ) in erythropoiesis was suggested several decades ago with the description of “erythroblastic islands” in the bone marrow (BM) composed of a central MΦ surrounded by developing erythroblasts. This hypothesis was strengthened by in vitro observations using cell culture systems showing that MΦ promote erythroblast proliferation and survival. However, the in vivo role of MΦ in erythropoiesis under homeostasis or disease remains unclear. Central MΦ reportedly express CD169 (or Sialoadhesin), an antigen that specifically marks tissue resident MΦ among

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18

Jang, Hee-Seong, Hamid Rabb, and Babu J. Padanilam. "CD169+ Macrophages: Regulators of Neutrophil Trafficking to Injured Kidneys." Journal of the American Society of Nephrology 26, no. 4 (2014): 769–71. http://dx.doi.org/10.1681/asn.2014090848.

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Hiemstra, Ida H., Marieke R. Beijer, Henrike Veninga, et al. "The identification and developmental requirements of colonic CD169+macrophages." Immunology 142, no. 2 (2014): 269–78. http://dx.doi.org/10.1111/imm.12251.

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Teo, Yi Juan, See Liang Ng, Keng Wai Mak, et al. "Renal CD169++ resident macrophages are crucial for protection against acute systemic candidiasis." Life Science Alliance 4, no. 5 (2021): e202000890. http://dx.doi.org/10.26508/lsa.202000890.

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Disseminated candidiasis remains as the most common hospital-acquired bloodstream fungal infection with up to 40% mortality rate despite the advancement of medical and hygienic practices. While it is well established that this infection heavily relies on the innate immune response for host survival, much less is known for the protective role elicited by the tissue-resident macrophage (TRM) subsets in the kidney, the prime organ for Candida persistence. Here, we describe a unique CD169++ TRM subset that controls Candida growth and inflammation during acute systemic candidiasis. Their absence ca

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21

Xia, Yuan, Ling-min Tian, Yu Liu, et al. "Low Dose of Cyanidin-3-O-Glucoside Alleviated Dextran Sulfate Sodium–Induced Colitis, Mediated by CD169+ Macrophage Pathway." Inflammatory Bowel Diseases 25, no. 9 (2019): 1510–21. http://dx.doi.org/10.1093/ibd/izz090.

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Abstract Background Inflammatory bowel disease (IBD) is a chronic disease of the intestinal tract in which excessive activation of inflammatory response is correlated. Cyanidin-3-O-glucoside (C3G) is a powerful anti-inflammatory agent, widely existing in fruits and vegetables. However, the role of C3G has rarely been investigated in dextran sulfate sodium (DSS)–induced colitis. Methods In an attempt to elucidate the possible mechanism of IBD and develop new efficient therapeutic methods for colitis, we evaluated the effects of C3G on DSS-induced colitis. DSS-induced colitic C57BL/6 mice were i

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Chen, Weihsu C., Norihito Kawasaki, Corwin M. Nycholat, et al. "Antigen Delivery to Macrophages Using Liposomal Nanoparticles Targeting Sialoadhesin/CD169." PLoS ONE 7, no. 6 (2012): e39039. http://dx.doi.org/10.1371/journal.pone.0039039.

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Marmey, Béatrice, Charlotte Boix, Jean-Baptiste Barbaroux, et al. "CD14 and CD169 expression in human lymph nodes and spleen: specific expansion of CD14+CD169− monocyte-derived cells in diffuse large B-cell lymphomas." Human Pathology 37, no. 1 (2006): 68–77. http://dx.doi.org/10.1016/j.humpath.2005.09.016.

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Kaur, Simranpreet, Liza J. Raggatt, Susan M. Millard, et al. "Self-repopulating recipient bone marrow resident macrophages promote long-term hematopoietic stem cell engraftment." Blood 132, no. 7 (2018): 735–49. http://dx.doi.org/10.1182/blood-2018-01-829663.

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Key Points Recipient macrophages persist in hematopoietic tissues and self-repopulate via in situ proliferation after syngeneic transplantation. Targeted depletion of recipient CD169+ macrophages after transplant impaired long-term bone marrow engraftment of hematopoietic stem cells.

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Purtha, Whitney E., Karen A. Chachu, Herbert W. Virgin, and Michael S. Diamond. "Early B-Cell Activation after West Nile Virus Infection Requires Alpha/Beta Interferon but Not Antigen Receptor Signaling." Journal of Virology 82, no. 22 (2008): 10964–74. http://dx.doi.org/10.1128/jvi.01646-08.

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ABSTRACT The B-cell response against West Nile virus (WNV), an encephalitic Flavivirus of global concern, is critical to controlling central nervous system dissemination and neurological sequelae, including death. Here, using a well-characterized mouse model of WNV infection, we examine the factors that govern early B-cell activation. Subcutaneous inoculation with a low dose of replicating WNV results in extensive B-cell activation in the draining lymph node (LN) within days of infection as judged by upregulation of the surface markers CD69, class II major histocompatibility complex, and CD86

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Ren, Y. W., Y. Y. Zhang, N. A. Affara, et al. "The polymorphism analysis of CD169 and CD163 related with the risk of porcine reproductive and respiratory syndrome virus (PRRSV) infection." Molecular Biology Reports 39, no. 11 (2012): 9903–9. http://dx.doi.org/10.1007/s11033-012-1857-8.

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Seu, Katie Giger, Julien Papoin, Rose Fessler, et al. "Unravelling Macrophage Heterogeneity in Erythroblastic Islands Between Species." Blood 128, no. 22 (2016): 2436. http://dx.doi.org/10.1182/blood.v128.22.2436.2436.

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Abstract Erythroblastic islands (EBIs) are a hallmark of mammalian erythropoiesis consisting of a central macrophage surrounded by and interacting closely with maturing erythroblasts. While it is generally accepted that the island macrophages play an important role in erythropoiesis, the inability to identify and isolate this macrophage subpopulation has limited our understanding of their functional involvement. Previous studies have relied on immunohistochemistry/immunofluorescence in situ or in vitro. More recently, flow cytometry was used to characterize EBI formation and the immunophenotyp

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Xu, Fangda, Asanga Bandara, Hisashi Akiyama, et al. "Membrane-wrapped nanoparticles probe divergent roles of GM3 and phosphatidylserine in lipid-mediated viral entry pathways." Proceedings of the National Academy of Sciences 115, no. 39 (2018): E9041—E9050. http://dx.doi.org/10.1073/pnas.1804292115.

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Gold nanoparticles (NPs) wrapped in a membrane can be utilized as artificial virus nanoparticles (AVNs) that combine the large nonblinking or bleaching optical cross-section of the NP core with the biological surface properties and functionalities provided by a self-assembled lipid membrane. We used these hybrid nanomaterials to test the roles of monosialodihexosylganglioside (GM3) and phosphatidylserine (PS) for a lipid-mediated targeting of virus-containing compartments (VCCs) in macrophages. GM3-presenting AVNs bind to CD169 (Siglec-1)–expressing macrophages, but inclusion of PS in the GM3-

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Hayashi, Yoshihiro, Jieyu Wang, Zefeng Xu, et al. "EPO Signaling Triggers Erythrocytosis By Expanding Erythrocytes and Also Subsets of Macrophages." Blood 128, no. 22 (2016): 542. http://dx.doi.org/10.1182/blood.v128.22.542.542.

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Abstract Activated EPO receptor (EPOR) signaling (EPO-EPOR-JAK2-STATs) leads to erythrocytosis, which is defined as an absolute increase in red cell mass (RCM). Erythrocytosis is classified as primary versus secondary. Primary erythrocytosis is defined by erythroid cell-intrinsic EPO-independent mechanism due to constitutively activated EPOR signaling by gain-of function EPOR or JAK2mutations. Hypoxia inducible factor-2α (HIF-2α) is a master regulator of EPO gene expression. A defect in the oxygen-sensing VHL-PHD2-HIF-2α signaling pathway due to stress conditions or mutations leads to excess E

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Ikezumi, Yohei, Toshiaki Suzuki, Shinichi Hayafuji, et al. "The sialoadhesin (CD169) expressing a macrophage subset in human proliferative glomerulonephritis." Nephrology Dialysis Transplantation 20, no. 12 (2005): 2704–13. http://dx.doi.org/10.1093/ndt/gfi105.

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Chow, Andrew, Matthew Huggins, Jalal Ahmed, et al. "CD169+ macrophages provide a niche promoting erythropoiesis under homeostasis and stress." Nature Medicine 19, no. 4 (2013): 429–36. http://dx.doi.org/10.1038/nm.3057.

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Zhang, Yi, Jin-Qing Li, Ze-Zhou Jiang, Lian Li, Yan Wu, and Limin Zheng. "CD169 identifies an anti-tumour macrophage subpopulation in human hepatocellular carcinoma." Journal of Pathology 239, no. 2 (2016): 231–41. http://dx.doi.org/10.1002/path.4720.

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Saunderson, Sarah C., Amy C. Dunn, Paul R. Crocker, and Alexander D. McLellan. "CD169 mediates the capture of exosomes in spleen and lymph node." Blood 123, no. 2 (2014): 208–16. http://dx.doi.org/10.1182/blood-2013-03-489732.

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Key Points This study has identified a novel capture mechanism for host-derived vesicles within the spleen and lymph node. This pathway modulates the immune response to circulating particulate antigens.

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Costa-Hurtado, Mar, Alexandre Olvera, Verónica Martinez-Moliner, et al. "Changes in Macrophage Phenotype after Infection of Pigs with Haemophilus parasuis Strains with Different Levels of Virulence." Infection and Immunity 81, no. 7 (2013): 2327–33. http://dx.doi.org/10.1128/iai.00056-13.

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ABSTRACTHaemophilus parasuisis a colonizer of healthy piglets and the etiological agent of Glässer's disease. Differences in virulence among strains ofH. parasuishave been widely observed. In order to explore the host-pathogen interaction, snatch-farrowed colostrum-deprived piglets were intranasally infected with 4 strains ofH. parasuis: reference virulent strain Nagasaki, reference nonvirulent strain SW114, field strain IT29205 (from a systemic lesion and virulent in a previous challenge), and field strain F9 (from the nasal cavity of a healthy piglet). At different times after infection, two

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Xu, Haifeng C., Jun Huang, Vishal Khairnar, et al. "Deficiency of the B Cell-Activating Factor Receptor Results in Limited CD169+Macrophage Function during Viral Infection." Journal of Virology 89, no. 9 (2015): 4748–59. http://dx.doi.org/10.1128/jvi.02976-14.

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ABSTRACTThe B cell-activating factor (BAFF) is critical for B cell development and humoral immunity in mice and humans. While the role of BAFF in B cells has been widely described, its role in innate immunity remains unknown. Using BAFF receptor (BAFFR)-deficient mice, we characterized BAFFR-related innate and adaptive immune functions following infection with vesicular stomatitis virus (VSV) and lymphocytic choriomeningitis virus (LCMV). We identified a critical role for BAFFR signaling in the generation and maintenance of the CD169+macrophage compartment. Consequently,Baffr−/−mice exhibited

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Sun, Bing, Linda Abadjian, Alexander Monto, Heather Freasier, and Lynn Pulliam. "Hepatitis C Virus Cure in Human Immunodeficiency Virus Coinfection Dampens Inflammation and Improves Cognition Through Multiple Mechanisms." Journal of Infectious Diseases 222, no. 3 (2020): 396–406. http://dx.doi.org/10.1093/infdis/jiaa109.

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Abstract Background Chronic inflammation in human immunodeficiency virus (HIV)/hepatitis C virus (HCV) coinfection increases cognitive impairment. With newer, direct-acting antiviral therapies for HCV, our objective was to determine whether chronic inflammation would be decreased and cognition improved with HCV sustained viral response (SVR) in coinfection. Methods We studied 4 groups longitudinally: 7 HCV-monoinfected and 12 HIV/HCV-coinfected persons before and after treatment for HCV, 12 HIV-monoinfected persons, and 9 healthy controls. We measured monocyte activation and gene expression, m

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Niu, Pengxia, Sang-Wook Kim, Won-Il Kim, and Kwan-Suk Kim. "Association analyses of DNA polymorphisms in immune-related candidate genes GBP1, GBP2, CD163, and CD169 with porcine growth and meat quality traits." Journal of Biomedical Research 16, no. 2 (2015): 40–46. http://dx.doi.org/10.12729/jbr.2015.16.2.040.

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Ravishankar, B., R. Shinde, H. Liu, et al. "Marginal zone CD169+ macrophages coordinate apoptotic cell-driven cellular recruitment and tolerance." Proceedings of the National Academy of Sciences 111, no. 11 (2014): 4215–20. http://dx.doi.org/10.1073/pnas.1320924111.

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Asano, Kenichi, Kenta Kikuchi, and Masato Tanaka. "CD169 macrophages regulate immune responses toward particulate materials in the circulating fluid." Journal of Biochemistry 164, no. 2 (2018): 77–85. http://dx.doi.org/10.1093/jb/mvy050.

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Sewald, X., M. S. Ladinsky, P. D. Uchil, et al. "Retroviruses use CD169-mediated trans-infection of permissive lymphocytes to establish infection." Science 350, no. 6260 (2015): 563–67. http://dx.doi.org/10.1126/science.aab2749.

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Jacobsen, Rebecca, Allison Pettit, Valerie Barbier, et al. "Mobilising doses of G-CSF stop medullary erythropoiesis by depleting CD169+ macrophages." Experimental Hematology 41, no. 8 (2013): S59. http://dx.doi.org/10.1016/j.exphem.2013.05.230.

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Gummuluru, S., N. G. P. Ramirez, and H. Akiyama. "CD169-Dependent Cell-Associated HIV-1 Transmission: A Driver of Virus Dissemination." Journal of Infectious Diseases 210, suppl 3 (2014): S641—S647. http://dx.doi.org/10.1093/infdis/jiu442.

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Asano, Touko, Koji Ohnishi, Takuya Shiota, et al. "CD169-positive sinus macrophages in the lymph nodes determine bladder cancer prognosis." Cancer Science 109, no. 5 (2018): 1723–30. http://dx.doi.org/10.1111/cas.13565.

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Ohnishi, Koji, Yoichi Saito, Yoshihiro Komohara, et al. "Clinical significance of CD169-positive lymph node macrophages in human malignant tumors." Journal of Clinical Oncology 32, no. 15_suppl (2014): 11118. http://dx.doi.org/10.1200/jco.2014.32.15_suppl.11118.

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Gupta, Pravesh, Si Min Lai, Jianpeng Sheng, et al. "Tissue-Resident CD169 + Macrophages Form a Crucial Front Line against Plasmodium Infection." Cell Reports 16, no. 6 (2016): 1749–61. http://dx.doi.org/10.1016/j.celrep.2016.07.010.

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Asano, Kenichi, Ami Nabeyama, Yasunobu Miyake, et al. "CD169-Positive Macrophages Dominate Antitumor Immunity by Crosspresenting Dead Cell-Associated Antigens." Immunity 34, no. 1 (2011): 85–95. http://dx.doi.org/10.1016/j.immuni.2010.12.011.

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Muhsin-Sharafaldine, Morad-Remy, Sarah C. Saunderson, Amy C. Dunn, and Alexander D. McLellan. "Melanoma growth and lymph node metastasis is independent of host CD169 expression." Biochemical and Biophysical Research Communications 486, no. 4 (2017): 965–70. http://dx.doi.org/10.1016/j.bbrc.2017.03.138.

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Tacconi, Carlotta, Catharina D. Commerford, Lothar C. Dieterich, et al. "CD169+ lymph node macrophages have protective functions in mouse breast cancer metastasis." Cell Reports 35, no. 2 (2021): 108993. http://dx.doi.org/10.1016/j.celrep.2021.108993.

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Ortillon, Marine, Remy Coudereau, Martin Cour, et al. "Monocyte CD169 expression in COVID ‐19 patients upon intensive care unit admission." Cytometry Part A 99, no. 5 (2021): 466–71. http://dx.doi.org/10.1002/cyto.a.24315.

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Hamdan, Thamer A., Hilal Bhat, Lamin B. Cham, et al. "Map3k14 as a Regulator of Innate and Adaptive Immune Response during Acute Viral Infection." Pathogens 9, no. 2 (2020): 96. http://dx.doi.org/10.3390/pathogens9020096.

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Streszczenie:

The replication of virus in secondary lymphoid organs is crucial for the activation of antigen-presenting cells. Balanced viral replication ensures the sufficient availability of antigens and production of cytokines, and both of which are needed for virus-specific immune activation and viral elimination. Host factors that regulate coordinated viral replication are not fully understood. In the study reported here, we identified Map3k14 as an important regulator of enforced viral replication in the spleen while performing genome-wide association studies of various inbred mouse lines in a model o

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