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Rozprawy doktorskie na temat „Cell cycle”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Chauhan, Anuradha. "Cell cycle." Doctoral thesis, Humboldt-Universität zu Berlin, Mathematisch-Naturwissenschaftliche Fakultät I, 2011. http://dx.doi.org/10.18452/16301.

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Die Zellreplikation ein kontrollierter Prozess aus sequentieller und zeitlich koordinierter Aktivierung und Abbau von Zyklinen, die einen schnellen Übergang zwischen den Zyklusphasen ermöglichen. Dabei ist der Erfolg bei der Ermittlung der wichtigsten Komponenten und Aufgliederung der Schaltmechanismen im Wesentlichen auf die gleichzeitige Anwendung von Modellsystemen wie Hefe, Frosch und Fliege zurückzuführen. Das heutige Verständnis des Zellzyklus muss erweitert werden, um zu überprüfen ob die Erkenntnisse auch auf in-vivo Modelle von Säugetieren wie der Maus zutreffen. Es existieren solche

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2

Radmaneshfar, Elahe. "Mathematical modelling of the cell cycle stress response." Thesis, University of Aberdeen, 2012. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=192232.

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Thanky, Niren Rasik. "The mycobacterial cell cycle." Thesis, Imperial College London, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405727.

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4

Chaffey, Gary S. "Modelling the cell cycle." Thesis, University of Surrey, 2015. http://epubs.surrey.ac.uk/807189/.

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This thesis may be divided into two related parts. The first of which considers a population balance approach to modelling a population of cells, with particular emphasis on how the cells pass between the G1 and S phases of the cell cycle. In the second part of the thesis a model is described which combines a cell cycle model with a simple Pharmacokinetic/Pharmacodynamic (PKPD) drug model. This model is then discussed in detail. Knowledge of how a population of cancerous cells progress through the cell cycle is vital if the population is to be treated effectively, as treatment outcome is depen

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5

Li, Victor Chun. "The Cell Cycle and Differentiation in Stem Cells." Thesis, Harvard University, 2012. http://dissertations.umi.com/gsas.harvard:10536.

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The relationship between cellular proliferation and differentiation is a major topic in cell biology. What we know comes from models of somatic cell differentiation, where it is widely viewed that cycling and differentiation are coupled, antagonistic phenomena linked at the G1 phase. The extension of this view to stem cells, however, is unclear. One potential possibility is that stem cells also tightly link their G1 phase with their differentiation, indicating a similarity between the differentiation of stem cells and the differentiation of more mature somatic cells. On the other hand, stem ce

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6

Gauger, Michele Ann Sancar Aziz. "Cryptochrome, circadian cycle, cell cycle checkpoints, and cancer." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,971.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2007.<br>Title from electronic title page (viewed Dec. 18, 2007). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Biochemistry and Biophysics." Discipline: Biochemistry and Biophysics; Department/School: Medicine.

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7

Gad, Annica. "Cell cycle control by components of cell anchorage /." Stockholm : Division of Pathology, Karolinska institutet, 2005. http://diss.kib.ki.se/2005/91-7140-359-0/.

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8

Cadart, Clotilde. "Cell size homeostasis in animal cells." Thesis, Université Paris-Saclay (ComUE), 2017. http://www.theses.fr/2017SACLS103/document.

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Le mécanisme d’homéostasie de taille chez les cellules animales est très peu compris actuellement. Cette question est pourtant d’un intérêt majeur car le maintien de l’homéostasie de taille dans une population de cellules prolifératives doit se faire par une coordination entre la croissance et la division. Chez la levure S. pombe, il a ainsi été montré que la taille est une information cruciale pour déclencher l’entrée en mitose (Fantes, 1977). Chez plusieurs bactéries et les cellules filles de la levure S. cerevisiae au contraire, de récentes études ont au contraire montré que l’homéostasie d

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9

Poplawski, Andrzej. "Cell cycle analysis of archaea." Doctoral thesis, Uppsala University, Department of Cell and Molecular Biology, 2000. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-1078.

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<p>In my thesis, the cell cycle analysis of archaea and hyperthermophilic organisms is presented for the first time. Crenarchaea from the genus <i>Sulfolobus</i> were used as a model system. Plow cytometry and light microscopy were applied to investigate the timing and coordination of different cell cycle events. Furthermore, DNA content, nucleoid structure, and nucleoid distribution at different stages during the cell cycle were studied. The <i>Sulfolobus</i> cell cycle was characterized as having a short pre-replication and a long post-replication period. The presence of a low proportion of

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10

Shirazi, Fard Shahrzad. "The Heterogenic Final Cell Cycle of Retinal Horizontal Cells." Doctoral thesis, Uppsala universitet, Medicinsk utvecklingsbiologi, 2014. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-222559.

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The cell cycle is a highly complex process that is under the control of several pathways. Failure to regulate and/or complete the cell cycle often leads to cell cycle arrest, which may be followed by programmed cell death (apoptosis). One cell type that has a variety of unique cell cycle properties is the horizontal cell of the chicken retina. In this thesis we aimed to characterize the final cell cycle of retinal horizontal cells. In addition, the regulation of the cell cycle and the resistance to apoptosis of retinal horizontal cells are investigated. Our results show that the final cell cy

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11

Lomazzi, Marina. "Regulation of cell cycle by E2F1 in primary cells." Thesis, Open University, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.397894.

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12

Giraddi, Rajashekharagouda. "Cell cycle kinetics of mammary stem and progenitor cells." Thesis, University of Cambridge, 2013. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607789.

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13

Wang, Li. "CELL CYCLE REGULATION IN THE POST-MITOTIC NEURONAL CELLS." Case Western Reserve University School of Graduate Studies / OhioLINK, 2007. http://rave.ohiolink.edu/etdc/view?acc_num=case1184254319.

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14

France, Stephen Andrew. "Transcription and cell cycle control." Thesis, University of Glasgow, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340264.

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15

Sheppard, Catherine Louise. "Phosphodiesterases in the cell cycle." Thesis, University of Glasgow, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.392426.

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16

Zuo, Yuting. "Trichomonas vaginalis cell cycle studies /." Thesis, Connect to this title online; UW restricted, 1999. http://hdl.handle.net/1773/9301.

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17

Lantela, Daniel. "CUX1 and the Cell Cycle." Thesis, McGill University, 2013. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=119509.

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CUX1 is a transcription factor implicated in the control of cell proliferation. Over-expression of CUX1 is observed in many human tumors and cancer cell lines. Cells constitutively over-expressing the p110 isoform of CUX1 proliferate faster and spend less time in G1 following quiescence. We studied three aspects of CUX1 function and regulation during the cell cycle. In the first part of this study we investigated how over-expression of p110 CUX1 results in a shortened G1. Using quantitative PCR we showed that over-expression of p110 CUX1 caused an increase in the transcription of DDK gene

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18

Batsivari, Antoniana. "Studying the cell cycle status during haematopoietic stem cell development." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25802.

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In adults blood stem cells, called haematopoietic stem cells (HSC), give rise to all blood cells throughout life. The origin and biology of HSCs during embryo development has been an intensely studied topic. Definitive HSCs are generated intra-embryonically in the aorta-gonad-mesonephros (AGM) region of the mid-gestation embryo. Recent research revealed that HSCs emerge through multistep maturation of precursors: proHSC → preHSC I → preHSC II → definitive HSC (dHSC). A hallmark of the HSC emergence is the appearance of intra-aortic haematopoietic clusters that are considered to be sites of hae

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19

Lyman, Rachel C. "Cell cycle control and its modulation in HPV infected cells /." St Andrews, 2009. http://hdl.handle.net/10023/863.

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20

Ching, Ada Sik-Lun. "Cell cycle studies in Paramecium : effects of abrupt changes of nutritional state on cell cycle regulation." Thesis, University of British Columbia, 1985. http://hdl.handle.net/2429/24595.

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The controls over initiation of DNA synthesis, initiation of cell division, regulation of macronuclear DNA content, and the relationship between cell mass and growth rate were examined in cells growing under nutrient constraint, or in cells experiencing a change in growth conditions through nutritional enrichment (shift-up) or nutritional shift-down. Reduction in both cell mass and DNA content was achieved by growing Paramecium cells under nutritional limitation in the chemostat. Under the extreme condition in the chemostat, the normally balanced relationship between DNA content and cell mass

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21

Richardson, Deborah Susan. "Drug-induced apoptosis and cell cycle modulation in leukaemia cells." Thesis, Queen Mary, University of London, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.417928.

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22

Hosseini, Shirazi Seyed Farshad. "Cell cycle dependency of cisplatin cytotoxicity on ovarian cancer cells." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1998. http://www.collectionscanada.ca/obj/s4/f2/dsk2/tape17/PQDD_0028/NQ36776.pdf.

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23

Wade, Mark. "p53 independent apoptosis and cell cycle checkpoints in human cells." Thesis, Imperial College London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.251742.

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24

Lyman, Rachel C. "Cell cycle control and its modulation in HPV infected cells." Thesis, University of St Andrews, 2010. http://hdl.handle.net/10023/863.

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A key effect of human papillomavirus (HPV) infection is to disrupt the normal cell cycle in order to subvert the cellular DNA replication machinery. Morphologically, condylomata induced by high and low risk HPV types cannot be distinguished and many studies have shown that the pattern of viral gene expression is similar in condylomata caused by both high risk and low risk HPV types. Detailed morphological study of cell cycle protein expression has not previously been performed on condylomata infected with low risk HPV types. The findings presented suggest that the mechanisms employed by low ri

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25

Yildirim, Salih. "Cell cycle responses of glioma stem cells to ionizing radiation." Thesis, University of Sussex, 2011. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589993.

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The Cancer Stem Cell (CSC) hypothesis has provided a novel theory of tumorigenesis by suggesting that mechanisms of organogenesis in developmental processes may be aberrantly active in neoplasms. This hypothesis proposes that CSCS within a tumour play the role of stem cells in a tissue. This novel approach not only leads to new insights into the origination of cancer, but also suggests that CSCs may be responsible for the resistance of several cancer types to current therapies. Thus, CSCs may also be targets for novel therapies. This study interrogates the proposed role of Glioma Stem Cells (G

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26

Thompson, Christopher C. M. "Cell cycle-associated thymidine kinase regulation in friend erythroleukaemia cells." Thesis, University of Ulster, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.260517.

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27

Yiangou, Loukia. "Investigating the role of cell cycle regulators in mesoderm specification." Thesis, University of Cambridge, 2018. https://www.repository.cam.ac.uk/handle/1810/276182.

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Mesoderm is one of the three primary germ layers from which the cardiovascular system, muscle and bone originate and derivatives of the mesoderm lineage are affected in a number of pathologies. Therefore, understanding the mechanisms regulating formation of mesoderm is interesting for a diversity of diseases and clinical application. In vivo study of human development beyond gastrulation is technically challenging and the mechanisms controlling mesoderm specification are difficult to study since the maximum number of days allowed to grow human embryos is 14 days. Thus, in this dissertation I u

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28

Lundgren, Magnus. "Exploring the Cell Cycle of Archaea." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7848.

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29

Zakeri, Ghazal. "Prioritization of cell cycle regulated genes." Thesis, Norges teknisk-naturvitenskapelige universitet, Institutt for biologi, 2013. http://urn.kb.se/resolve?urn=urn:nbn:no:ntnu:diva-23242.

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The cell cycle is an important biological process in which a set of events occurs in a sequential manner progressing to the cell division. Cell cycle is regulated by periodic fluctuations in the expression levels of several genes referred to as cell cycle regulated genes. In this study, we apply machine learning techniques to prioritize a list of candidate genes with respect to being involved in the cell cycle regulation process. We focus on the data obtained from different expression experiments on which partial least squares regression (PLS) models have been previously developed to identify

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30

Bolger, Brendan Stephen. "Cell cycle kinetics in cervical tumours." Thesis, Imperial College London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.294984.

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31

Brown, N. R. "Structural studies of cell cycle proteins." Thesis, University of Oxford, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.299526.

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32

Woollard, Alison. "Cell cycle control in fission yeast." Thesis, University of London, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.318479.

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33

Wrighton, Katharine Helen. "TP53 mutation and cell cycle regulation." Thesis, King's College London (University of London), 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405794.

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34

Firby, D. J. "Regulation of the sycamore cell cycle." Thesis, De Montfort University, 1985. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304219.

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35

Crossland, V. M. "Cell cycle specific recruitment of PKCε". Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1352790/.

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Protein kinase C (PKC) comprises a family of serine/threonine kinases which play central roles in intracellular signal transduction typically triggered by recruitment to membraneous compartments. The epsilon isoform of PKC (PKCå) has been shown to localize to cell-cell contacts and to the cytokinetic furrow/midbody, indicative of a role in the cell cycle. Both recruitment patterns can be visualized under conditions of PKCå inhibition, which is selectively achieved using a gatekeeper mutant (PKCå-M486A) and the inhibitor NaPP1. Initial studies indicated that interphase and mitotic cells were no

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36

He, Enuo. "Stochastic modelling of the cell cycle." Thesis, University of Oxford, 2012. http://ora.ox.ac.uk/objects/uuid:04185cde-85af-4e24-8d06-94b865771cf1.

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Precise regulation of cell cycle events by the Cdk-control network is essential for cell proliferation and the perpetuation of life. The unidirectionality of cell cycle progression is governed by several critical irreversible transitions: the G1-to-S transition, the G2-to-M transition, and the M-to-G1 transition. Recent experimental and theoretical evidence has pulled into question the consensus view that irreversible protein degradation causes the irreversibility of those transitions. A new view has started to emerge, which explains the irreversibility of cell cycle transitions as a consequen

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37

Santos, Carlo Steven. "Circadian Control of Cell Cycle Progression." Thesis, Virginia Tech, 2009. http://hdl.handle.net/10919/76987.

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Tumorigenesis is the result of uncontrolled cell growth due to the deregulation of cell cycle checkpoints 1. Period 2 (Per2) is a tumor suppressor that oscillate in expression in a 24-hour cycle 2, 3. Here, we show that Per2 interacts with the tumor suppressor protein p53. Both G1 and G2 checkpoint pathways involve a p53 dependent pathway which can trigger the cell to go through cell arrest or programmed cell death4. Understanding all the mitigating factors involved in regulating cell cycle progression under DNA damage can offer a better idea in how cells become immortal. Initially discovere

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38

Kuleszewicz, Katarzyna. "Cell cycle regulation in mammalian oocytes." Thesis, Imperial College London, 2013. http://hdl.handle.net/10044/1/26148.

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An unusual feature of mammalian female germ cells is that they are arrested in meiotic prophase, equivalent to mitotic G2-phase, for an extended period of time. In this thesis I have investigated two aspects of this arrest. First, I examined whether cohesin replenishment is required for the maintenance of chromosome cohesion during protracted meiotic prophase arrest. Nipbl, an evolutionarily conserved protein, is a component of protein complex called kollerin, whose activity in loading cohesin onto chromosomes is necessary for accurate chromosome segregation during mitosis. However, until now

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39

Holt, Liam J. "Combinatorial control of the cell cycle." Diss., Search in ProQuest Dissertations & Theses. UC Only, 2008. http://gateway.proquest.com/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:3330864.

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40

Delorme, Marilyne. "Downregulation of ATRX disrupts cell proliferation and cell cycle progression." Thesis, University of Ottawa (Canada), 2008. http://hdl.handle.net/10393/27627.

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ATRX is a chromatin remodelling protein of the SNF2 family of chromatin remodelling proteins. Mutations in the ATRX gene have been shown to cause the ATR-X syndrome, an X-linked mental retardation disorder. ATRX is part of a chromatin-remodelling complex with Daxx that localizes to PML nuclear bodies or pericentromeric heterochromatin and is thought to regulate gene expression. In mice, Atrx inactivation results in embryonic lethality whereas conditional forebrain specific Atrx ablation showed impaired development and disorganization of the cortex. Furthermore, ATRX phosphorylation was shown t

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41

Jahnke, Ulrike. "Cell cycle de-regulation and cell death in leukaemia chemotherapy." Thesis, Queen Mary, University of London, 2006. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.439424.

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Anderson, Jon E. "Cell cycle regulation in the early porcine embryo /." free to MU campus, to others for purchase, 2000. http://wwwlib.umi.com/cr/mo/fullcit?p9974607.

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Caldon, Catherine Elizabeth Garvan Institute of Medical Research Faculty of Medicine UNSW. "Cell cycle control by ID1 and WT1 in breast cancer cells." Awarded by:University of New South Wales. Garvan Institute of Medical Research, 2007. http://handle.unsw.edu.au/1959.4/33125.

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Loss of proliferative control is a cornerstone of cancer development, induced by deregulation of mitogenic signalling, insensitivity to anti-proliferative signals and direct changes in cell cycle proteins. In breast cancer these alterations are frequently targeted through cyclins D1 and E, leading to defects in G1/S transition. I have investigated the role of two potential pro-proliferative oncogenes in breast cancer, id1 andwt1. Each protein promotes proliferation in distinct contexts, with unique consquences for breast cancer cells. Using a 3D culture model of non-transformed mammary epithel

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Blakemore, Louise Margaret. "Curcumin-induced G2/M cell cycle arrest in colorectal cancer cells." Thesis, University of Leicester, 2011. http://hdl.handle.net/2381/9809.

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Curcumin, a diet-derived chemopreventive and chemotherapeutic agent has been shown to induce G2/M cell cycle arrest, and previous studies suggested that microtubule depolymerisation may be linked to M-phase arrest. However, mechanisms involved have not been elucidated and often non-physiological concentrations of curcumin were used. The goal of this study was to characterise in more detail curcumin-induced cell cycle arrest using a panel of human colorectal cancer cell (CRC) lines, HT-29, SW480, HCT116 p53+/+, HCT116 p53-/- and HCT116 p21-/-. Using physiologically relevant concentrations of cu

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Izzard, Tanya. "Extracellular matrix and the cell cycle in vascular smooth muscle cells." Thesis, University of Bristol, 2000. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322616.

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Ouertani, A. "Determinants of cell cycle progression in human mammary epithelial MCF12 cells." Thesis, University College London (University of London), 2012. http://discovery.ucl.ac.uk/1362848/.

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Cancer of the mammary gland is the most common type of cancer in women worldwide, and the vast majority of breast cancers originate from a cluster of malignant cells in the epithelial tissue of the breast, which initially confines the ductal carcinoma in situ. Research has shown that the signalling pathways that increase differentiation and maintain proliferation in normal epithelial cells are of utmost importance for sustaining this barrier against malignant cells. As a model for normal mammary epithelial cells, the MCF-12A cell line was used to determine factors that are required for cell cy

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47

Calegari, Federico, and Julieta Aprea. "Bioelectric State and Cell Cycle Control of Mammalian Neural Stem Cells." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-185623.

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The concerted action of ion channels and pumps establishing a resting membrane potential has been most thoroughly studied in the context of excitable cells, most notably neurons, but emerging evidences indicate that they are also involved in controlling proliferation and differentiation of nonexcitable somatic stem cells. The importance of understanding stem cell contribution to tissue formation during embryonic development, adult homeostasis, and regeneration in disease has prompted many groups to study and manipulate the membrane potential of stem cells in a variety of systems. In this paper

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Calegari, Federico, and Julieta Aprea. "Bioelectric State and Cell Cycle Control of Mammalian Neural Stem Cells." Sage-Hindawi, 2012. https://tud.qucosa.de/id/qucosa%3A27972.

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The concerted action of ion channels and pumps establishing a resting membrane potential has been most thoroughly studied in the context of excitable cells, most notably neurons, but emerging evidences indicate that they are also involved in controlling proliferation and differentiation of nonexcitable somatic stem cells. The importance of understanding stem cell contribution to tissue formation during embryonic development, adult homeostasis, and regeneration in disease has prompted many groups to study and manipulate the membrane potential of stem cells in a variety of systems. In this paper

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49

Ashford, Anne Louise. "The role of the protein kinase DYRK1B in cancer cell survival and cell cycle control." Thesis, University of Cambridge, 2014. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.648671.

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50

Fredlund, Jan O. "The role of polyanimes in cell cycle progression." Lund : Lund University Dept. of Animal Physiology, 1996. http://catalog.hathitrust.org/api/volumes/oclc/38100686.html.

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