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Artykuły w czasopismach na temat „Cognition disorders”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Woodruff, Bryan. "Disorders of Cognition." Seminars in Neurology 31, no. 01 (2011): 018–28. http://dx.doi.org/10.1055/s-0031-1271314.

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Chamard, Ludivine, Sabrina Ferreira, Alexa Pijoff, Manon Silvestre, Eric Berger, and Eloi Magnin. "Cognitive Impairment Involving Social Cognition in SPG4 Hereditary Spastic Paraplegia." Behavioural Neurology 2016 (2016): 1–6. http://dx.doi.org/10.1155/2016/6423461.

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Objectives. To describe cognitive assessment including social cognition in SPG4 patients.Methods. We reported a series of nine patients with SPG4 mutation with an extensive neuropsychological examination including social cognition assessment.Results. None of our patients presented with mental retardation or dementia. All presented with mild cognitive impairment with a high frequency of attention deficit (100%), executive disorders (89%), and social cognition impairment (78%). An asymptomatic patient for motor skills presented with the same cognitive profile. No correlation was found in this sm

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de Sousa, Paulo, William Sellwood, Martin Griffiths, and Richard P. Bentall. "Disorganisation, thought disorder and socio-cognitive functioning in schizophrenia spectrum disorders." British Journal of Psychiatry 214, no. 2 (2018): 103–12. http://dx.doi.org/10.1192/bjp.2018.160.

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BackgroundPoor social cognition is prevalent in schizophrenia spectrum disorders. Some authors argue that these effects are symptom-specific and that socio-cognitive difficulties (e.g. theory of mind) are strongly associated with thought disorder and symptoms of disorganisation.AimsThe current review tests the strength of this association.MethodWe meta-analysed studies published between 1980 and 2016 that tested the association between social cognition and these symptoms in schizophrenia spectrum disorders.ResultsOur search (PsycINFO, MEDLINE and Web of Science) identified 123 studies (N = 910

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Jones, Barbara Pendleton, Connie C. Duncan, Pim Brouwers, and Allan F. Mirsky. "Cognition in eating disorders." Journal of Clinical and Experimental Neuropsychology 13, no. 5 (1991): 711–28. http://dx.doi.org/10.1080/01688639108401085.

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Gjelsvik, Bergljot, Darko Lovric, and J. Mark G. Williams. "Embodied cognition and emotional disorders." Journal of Experimental Psychopathology 9, no. 3 (2018): pr.035714. http://dx.doi.org/10.5127/pr.035714.

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Research into embodied cognition (EC) in cognitive neuroscience and psychology has risen exponentially over the last 25 years, covering a vast area of research; from understanding how ability to judge speech sounds depends on an intact motor cortex, to why people perceive hills as steeper when carrying a heavy backpack. Although there are many theories addressing these phenomena, increasing evidence across EC studies suggests simulation (i.e., re-enactment of the motor-sensory aspects of meaning) as an important basis of knowledge. The authors 1) review evidence for the EC paradigm’s claim to

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Uchino, Takashi, Ryo Okubo, Youji Takubo, et al. "Mediation Effects of Social Cognition on the Relationship between Neurocognition and Social Functioning in Major Depressive Disorder and Schizophrenia Spectrum Disorders." Journal of Personalized Medicine 13, no. 4 (2023): 683. http://dx.doi.org/10.3390/jpm13040683.

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Background: In schizophrenia spectrum disorders (SSD), social cognition mediates the relationship between neurocognition and social functioning. Although people with major depressive disorder (MDD) also exhibit cognitive impairments, which are often prolonged, little is known about the role of social cognition in MDD. Methods: Using data obtained through an internet survey, 210 patients with SSD or MDD were selected using propensity score matching based on their demographics and illness duration. Social cognition, neurocognition, and social functioning were evaluated using the Self-Assessment

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Haywood, H. Carl, and Stéphane Raffard. "Cognition and Psychopathology: Overview." Journal of Cognitive Education and Psychology 16, no. 1 (2017): 3–8. http://dx.doi.org/10.1891/1945-8959.16.1.3.

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The bidirectional relation of cognition and psychopathology is discussed in historical context as an introduction to the special issue of Journal of Cognitive Education and Psychology devoted to contemporary research on this topic. Cognition refers to the processes by which information is transformed, elaborated, stored, recovered, and used. Psychopathology refers to disorders in behavior, cognition, and/or perception. Although rooted in studies of major psychiatric disorders and general intelligence, the field has moved to include developmental disabilities, neurological impairment, and less

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Holland, Negin, Trevor W. Robbins, and James B. Rowe. "The role of noradrenaline in cognition and cognitive disorders." Brain 144, no. 8 (2021): 2243–56. http://dx.doi.org/10.1093/brain/awab111.

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Abstract Many aspects of cognition and behaviour are regulated by noradrenergic projections to the forebrain originating from the locus coeruleus, acting through alpha and beta adrenoreceptors. Loss of these projections is common in neurodegenerative diseases and contributes to their cognitive and behavioural deficits. We review the evidence for a noradrenergic modulation of cognition in its contribution to Alzheimer’s disease, Parkinson’s disease and other cognitive disorders. We discuss the advances in human imaging and computational methods that quantify the locus coeruleus and its function

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Umarova, Sabohon Minavvarovna Mashrabbaeva Gulliza Muhammadali kizi. "DISORDERS OF SENSORY FUNCTIONS." ACADEMIC RESEARCH IN MODERN SCIENCE 2, no. 22 (2023): 75–79. https://doi.org/10.5281/zenodo.8416359.

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According to the science of world psychology, perception is considered as a simple mental cognitive process consisting of reflecting some features of things and events of the existing world, as well as the internal states of the human body through the direct influence of material stimuli on certain receptors - this is the first stage of cognition

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Başar, Erol. "Special issue: Brain oscillations in cognition and cognitive disorders." Brain Research 1235 (October 2008): 1. http://dx.doi.org/10.1016/j.brainres.2008.09.010.

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Humphreys, Glyn. "Disorders of Visuo-spatial Cognition." Neurocase 11, no. 2 (2005): 146–47. http://dx.doi.org/10.1080/13554790590925547.

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Desmarais, Philippe, Krista L. Lanctôt, Mario Masellis, Sandra E. Black, and Nathan Herrmann. "Social inappropriateness in neurodegenerative disorders." International Psychogeriatrics 30, no. 2 (2017): 197–207. http://dx.doi.org/10.1017/s1041610217001260.

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ABSTRACTBackground:New onset of mood and behavioral changes in middle-aged patients are frequently the first manifestations of an unrecognized neurocognitive disorder. Impairment of social cognition, the cognitive ability to process social information coming from others, such as emotions, to attribute mental states to others, and to respond appropriately to them, is often at the origin of behavioral manifestations in neurodegenerative disorders.Methods:This paper reviews the current literature on social cognition impairment in neurocognitive disorders, particularly in prodromal stages of behav

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Temple, Elise. "The developmental cognitive neuroscience approach to the study of developmental disorders." Behavioral and Brain Sciences 25, no. 6 (2002): 771. http://dx.doi.org/10.1017/s0140525x02420134.

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Functional magnetic resonance imaging studies of developmental disorders and normal cognition that include children are becoming increasingly common and represent part of a newly expanding field of developmental cognitive neuroscience. These studies have illustrated the importance of the process of development in understanding brain mechanisms underlying cognition and including children in the study of the etiology of developmental disorders.

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Fourrier, Célia, Gaurav Singhal, and Bernhard T. Baune. "Neuroinflammation and cognition across psychiatric conditions." CNS Spectrums 24, no. 1 (2019): 4–15. http://dx.doi.org/10.1017/s1092852918001499.

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Cognitive impairments reported across psychiatric conditions (ie, major depressive disorder, bipolar disorder, schizophrenia, and posttraumatic stress disorder) strongly impair the quality of life of patients and the recovery of those conditions. There is therefore a great need for consideration for cognitive dysfunction in the management of psychiatric disorders. The redundant pattern of cognitive impairments across such conditions suggests possible shared mechanisms potentially leading to their development. Here, we review for the first time the possible role of inflammation in cognitive dys

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Eum, Seenae, Scot Kristian Hill, and Jeffrey R. Bishop. "Considering medication exposure in genomic association studies of cognition in psychotic disorders." Pharmacogenomics 23, no. 14 (2022): 791–806. http://dx.doi.org/10.2217/pgs-2022-0070.

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Cognitive dysfunction is a core feature of psychosis-spectrum illnesses, and the characterization of related genetic mechanisms may provide insights regarding the disease pathophysiology. Substantial efforts have been made to determine the genetic component of cognitive symptoms, without clear success. Illness-related moderators and environmental factors such as medications hinder the detection of genomic association with cognition. Polypharmacy is common in psychotic disorders, and the cumulative effects of medication regimens can confound gene–cognition associations. A review of the relative

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Ramey, Tatiana, and Paul S. Regier. "Cognitive impairment in substance use disorders." CNS Spectrums 24, no. 1 (2018): 102–13. http://dx.doi.org/10.1017/s1092852918001426.

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Cognitive impairments in substance use disorders have been extensively researched, especially since the advent of cognitive and computational neuroscience and neuroimaging methods in the last 20 years. Conceptually, altered cognitive function can be viewed as a hallmark feature of substance use disorders, with documented alterations in the well-known “executive” domains of attention, inhibition/regulation, working memory, and decision-making. Poor cognitive (sometimes referred to as “top-down”) regulation of downstream motivational processes—whether appetitive (reward, incentive salience) or a

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Briscoe, J. "The beauty of models for developmental disorders." Behavioral and Brain Sciences 25, no. 6 (2002): 750–52. http://dx.doi.org/10.1017/s0140525x02220130.

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Cognitive frameworks provide important means for uniting concepts of specificity, cognition, and dynamic change in development. Two points are challenged by evidence from special populations: (1) that boundary constraints such as Residual Normality and a cognitive “endstate” compromise the use of cognitive models; and (2) the developmental process itself automatically rejects either Residual Normality or residual deviance from typical development.

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Kauliņa, Anda. "Cognitive Analysis of 9 to 11-Year-Old Children With Intellectual Development Disorders." Journal of Pedagogy and Psychology "Signum Temporis" 9, no. 1 (2017): 16–22. http://dx.doi.org/10.1515/sigtem-2017-0006.

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Abstract Cognitive development significantly influences efficiency and results of child’s understanding and comprehension of the world. Attention and cognition play a significant role to ensure academic achievement and success. Attention is essential for purposeful planning of action and systematic work. Attention is necessary to follow the study material and for physical survival in everyday life. Cognition is significant in decision making and evaluating possible outcomes, being especially important in children with cognitive development disorders. The aim of the present study was to find ou

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McDonald, Skye. "What's New in the Clinical Management of Disorders of Social Cognition?" Brain Impairment 18, no. 1 (2017): 2–10. http://dx.doi.org/10.1017/brimp.2017.2.

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Social cognition refers to the ability to use social cues to infer the meaning and intentions behind the behaviour of others in order to respond in a socially adaptive manner. It is increasingly recognised that disorders of social cognition, including problems with emotion perception, theory of mind, conversational inference, morality judgements, decision making and social inhibition, characterise many developmental and psychiatric disorders and are highly relevant to many with acquired brain injuries or diseases, especially the frontotemporal dementias. This review provides an introduction an

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Troisi, Alfonso. "Psychiatric disorders and the social brain: Distinguishing mentalizing and empathizing." Behavioral and Brain Sciences 31, no. 3 (2008): 279–80. http://dx.doi.org/10.1017/s0140525x08004408.

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AbstractSocial cognition is a broad term, incorporating all aspects of social functioning from perceiving emotional stimuli to attributional style and theory of mind. Not distinguishing between these different capacities may confound the interpretation of the data deriving from studies of the relationship between psychiatric disorders and the social brain. The distinction between cognitive and affective components of social cognition is clearly exemplified by the abnormalities observed in psychopathy and Williams syndrome.

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Operto, Francesca Felicia, Grazia Maria Giovanna Pastorino, Chiara Padovano, Chiara Scuoppo, Valentina Vivenzio, and Giangennaro Coppola. "Social Cognition in Neuropsychiatric Disorders in Pediatric Age." BRAIN. Broad Research in Artificial Intelligence and Neuroscience 11, no. 3sup1 (2020): 81–88. http://dx.doi.org/10.18662/brain/11.3sup1/124.

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Objective: The purpose of our study was to assess social cognition in adolescents and children with epilepsy or Specific Learning Disorder (SLD) compared to typical individuals. It was verified whether the age of onset, duration and drug therapy of epileptics can influence this ability and if there is a correlation between Social Cognition, intelligence and executive functions. Methods: This is an observational cross-sectional study that included a total of 125 subjects between 7 and 16 years (62 with focal epilepsy and 63 with SLD). The control group included 32 healthy subjects. Study subjec

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OPERTO, Francesca Felicia, Grazia Maria Giovanna PASTORINO, Chiara PADOVANO, Chiara SCUOPPO, Valentina VIVENZIO, and Giangennaro COPPOLA. "Social Cognition in Neuropsychiatric Disorders in Pediatric Age." BRAIN. Broad Research in Artificial Intelligence and Neuroscience 11, no. 3 Sup.1 (2020): 81–88. https://doi.org/10.18662/brain/11.3Sup1/124.

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 Objective: The purpose of our study was to assess social cognition in ad-olescents and children with epilepsy or Specific Learning Disorder (SLD) compared to typical individuals. It was verified whether the age of onset, duration and drug therapy of epileptics can influence this ability and if there is a correlation between Social Cognition, intelligence and executive functions.Methods: This is an observational cross-sectional study that included a total of 125 subjects between 7 and 16 years (62 with focal epilepsy and 63 with SLD). The control group included 32 healthy subjects. Study

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Keifer, Cara M., Amori Yee Mikami, James P. Morris, Erin J. Libsack, and Matthew D. Lerner. "Prediction of social behavior in autism spectrum disorders: Explicit versus implicit social cognition." Autism 24, no. 7 (2020): 1758–72. http://dx.doi.org/10.1177/1362361320922058.

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Deficient social communication and interaction behaviors are a hallmark feature of individuals with autism spectrum disorder. These social communication and interaction deficits potentially stem from problems with explicit social cognition (i.e. processes that are controlled and largely conscious) as well as with implicit social cognition (i.e. processes that are fast, spontaneous, and primarily unconscious). This study aimed to investigate the relative contributions of implicit and explicit social cognition factors as predictors of multi-informant measures of social communication and interact

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Doyle, Alysa E., Pieter J. Vuijk, Nathan D. Doty, et al. "Cross-Disorder Cognitive Impairments in Youth Referred for Neuropsychiatric Evaluation." Journal of the International Neuropsychological Society 24, no. 1 (2017): 91–103. http://dx.doi.org/10.1017/s1355617717000601.

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AbstractObjectives: Studies suggest that impairments in some of the same domains of cognition occur in different neuropsychiatric conditions, including those known to share genetic liability. Yet, direct, multi-disorder cognitive comparisons are limited, and it remains unclear whether overlapping deficits are due to comorbidity. We aimed to extend the literature by examining cognition across different neuropsychiatric conditions and addressing comorbidity. Methods: Subjects were 486 youth consecutively referred for neuropsychiatric evaluation and enrolled in the Longitudinal Study of Genetic I

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Hottman, David A., Dustin Chernick, Shaowu Cheng, Zhe Wang, and Ling Li. "HDL and cognition in neurodegenerative disorders." Neurobiology of Disease 72 (December 2014): 22–36. http://dx.doi.org/10.1016/j.nbd.2014.07.015.

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van den Bosch, R. J. "Exploring cognition and frontal lobe disorders." European Psychiatry 17 (May 2002): 12. http://dx.doi.org/10.1016/s0924-9338(02)80060-8.

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Pratt, Helen D., Debra O'Donnell, and Marilyn F. Orfuss. "Disorders of cognition, attention and learning." Indian Journal of Pediatrics 66, no. 3 (1999): 401–14. http://dx.doi.org/10.1007/bf02845534.

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Shively, C. A., and C. L. Bethea. "Cognition, Mood Disorders, and Sex Hormone." ILAR Journal 45, no. 2 (2004): 189–99. http://dx.doi.org/10.1093/ilar.45.2.189.

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Chutko, L. S., S. Yu Surushkina, and E. A. Yakovenko. "Disorders of social cognition in children." Zhurnal nevrologii i psikhiatrii im. S.S. Korsakova 123, no. 1 (2023): 34. http://dx.doi.org/10.17116/jnevro202312301134.

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Dimitrova-Kirilova, Vladina Miroslavova, Aleksandra Krasimirova Yankova, Dimitrinka Rosenova Dimitrova, Mihael Tsalta-Mladenov, and Veselinka Nestorova. "Asymptomatic ischemic cerebrovascular disorders and cognition." Varna Medical Forum 12, no. 1 (2023): 21. http://dx.doi.org/10.14748/vmf.v12i1.8741.

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Eifert, Georg H., and Lyn Craill. "The Relationship between Affect, Behaviour, and Cognition in Behavioural and Cognitive Treatments of Depression and Phobic Anxiety." Behaviour Change 6, no. 2 (1989): 96–103. http://dx.doi.org/10.1017/s0813483900007634.

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Cognitive theories and therapies of emotional dysfunctions are based on the premise that the affective, behavioural, and cognitive response repertoires are fused and highly interdependent. Such views have been criticised with the argument that affect and cognition are relatively independent and that there is a much more direct and stronger link between affect and behaviour. In an attempt to clarify potential differences in the interplay between affect, behaviour, and cognition in unipolar depression and phobic anxiety, a quantitative review of the relative efficacy of performance-based (behavi

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Howard, CW, and MD Fox. "P.007 Web-based monitoring for cognitive decline following deep brain stimulation." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 50, s2 (2023): S59. http://dx.doi.org/10.1017/cjn.2023.112.

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Background: There is a pressing need to monitor the cognitive outcomes of patients undergoing deep brain stimulation (DBS) for movement disorders, despite the prevalence of pre-operative cognitive evaluations. Previous research has demonstrated the potential for DBS to induce reversible cognitive decline, highlighting the need for post-operative cognitive monitoring. Methods: To address this issue, the present study sought to improve upon the existing Autonomous Cognitive Examination through the development of a 5-minute web-based exam. This examination leverages the capacity of machine-learni

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Wu, Sally, Nicolette Stogios, Margaret Hahn, et al. "Outcomes and clinical implications of intranasal insulin on cognition in humans: A systematic review and meta-analysis." PLOS ONE 18, no. 6 (2023): e0286887. http://dx.doi.org/10.1371/journal.pone.0286887.

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Background Aberrant brain insulin signaling has been posited to lie at the crossroads of several metabolic and cognitive disorders. Intranasal insulin (INI) is a non-invasive approach that allows investigation and modulation of insulin signaling in the brain while limiting peripheral side effects. Objectives The objective of this systematic review and meta-analysis is to evaluate the effects of INI on cognition in diverse patient populations and healthy individuals. Methods MEDLINE, EMBASE, PsycINFO, and Cochrane CENTRAL were systematically searched from 2000 to July 2021. Eligible studies wer

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Dionysopoulou, Sofia, Evangelia Charmandari, Alexandra Bargiota, Nikolaos F. Vlahos, George Mastorakos, and Georgios Valsamakis. "The Role of Hypothalamic Inflammation in Diet-Induced Obesity and Its Association with Cognitive and Mood Disorders." Nutrients 13, no. 2 (2021): 498. http://dx.doi.org/10.3390/nu13020498.

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Obesity is often associated with cognitive and mood disorders. Recent evidence suggests that obesity may cause hypothalamic inflammation. Our aim was to investigate the hypothesis that there is a causal link between obesity-induced hypothalamic inflammation and cognitive and mood disorders. Inflammation may influence hypothalamic inter-connections with regions important for cognition and mood, while it may cause dysregulation of the Hypothalamic-Pituitary-Adrenal (HPA) axis and influence monoaminergic systems. Exercise, healthy diet, and glucagon-like peptide receptor agonists, which can reduc

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Velit-Salazar, Mario Renato, Paulo R. Shiroma, and Eloise Cherian. "A Systematic Review of the Neurocognitive Effects of Psychedelics in Healthy Populations: Implications for Depressive Disorders and Post-Traumatic Stress Disorder." Brain Sciences 14, no. 3 (2024): 248. http://dx.doi.org/10.3390/brainsci14030248.

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Objective: This study aims to provide an overview of pharmacological trials that examine the neurocognitive effects of psychedelics among healthy individuals and patients with post-traumatic stress disorder (PTSD) or major depressive disorder (MDD). Methods: The Preferred Reporting Items for Systematic Reviews (PRISMA) was used as a guide to structure and report the findings for this review. A literature search included the MEDLINE database up until December 2022. We included randomized or open-label human studies of MDMA, psilocybin, mescaline, LSD, DMT, or cannabis reporting non-emotionally

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Carvalho, Luciane Bizari Coin de, Lucila Bizari Fernandes do Prado, Luciana Silva, et al. "Cognitive dysfunction in children with sleep disorders." Arquivos de Neuro-Psiquiatria 62, no. 2a (2004): 212–16. http://dx.doi.org/10.1590/s0004-282x2004000200004.

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Sleep is basic for physical and cognitive development and some studies have suggested that there may be an association between sleep disorders (SD) and cognitive dysfunction (CD) in children. Little is known, however, about SD and cognition in 7-10-year-old children, a fact that motivated the present study. METHOD: We applied an SD questionnaire in 1180 children, 547 with SD and 633 without SD (CG), to assess cognition with a screening test (Bender Visual Motor Gestalt Test - BT). RESULTS: We observed a similar frequency of CD in the children with SD (39%) and that ot the CG (40%). The 8-year-

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Filley, Christopher M., and R. Douglas Fields. "White matter and cognition: making the connection." Journal of Neurophysiology 116, no. 5 (2016): 2093–104. http://dx.doi.org/10.1152/jn.00221.2016.

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Whereas the cerebral cortex has long been regarded by neuroscientists as the major locus of cognitive function, the white matter of the brain is increasingly recognized as equally critical for cognition. White matter comprises half of the brain, has expanded more than gray matter in evolution, and forms an indispensable component of distributed neural networks that subserve neurobehavioral operations. White matter tracts mediate the essential connectivity by which human behavior is organized, working in concert with gray matter to enable the extraordinary repertoire of human cognitive capaciti

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Sharashkina, Natalia V., Valentina S. Ostapenko, and Nadezda K. Runikhina. "Features of cognitive disorders in elderly and old patients with hypertension." Russian Family Doctor 22, no. 1 (2018): 36–40. http://dx.doi.org/10.17816/rfd2018136-40.

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Aim. To assess cognitive status of the elderly and old patients with arterial hypertension, in outpatient setting. Material and methods. Totally, 356 patients included, age 74,9 ± 6,1 y., 80,4% females. For cognition assessment before and after the treatment, neuropsychological test was done, with MMSE - short scale of psychic status assessment, drawing test; literal and categoric associations test. Results. In the group <80 y. o. The prominence of MMSE disorders correlated with blood pressure values (BP): for systolic BP (r = -0,22, p = 0,0003), for diastolic BP (r = -0,13, p = 0,03), i.e.

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Dave, Prachi, S. Sudha, Vijaya Raghavan, and P. Poornachandrika. "Association Between Built Space and Cognition in Humans: A Scoping Review." INDIAN JOURNAL OF MENTAL HEALTH AND NEUROSCIENCES 3, no. 01 (2020): 23–35. http://dx.doi.org/10.32746/10.32746/ijmhns.2020.v3.i1.46.

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Background: Various built environment characteristics have been shown to have varied effects on mental health. There is evidence for a positive relationship between certain BE characteristics and mental disorders. The talk on cognition is inevitable in any argument on mental health. This review looks at various BE characteristics and cognition and their interrelationship. Materials and Methods: A Boolean search of PubMed articles with keywords like ‘built environment’, ‘built spaces’, ‘green spaces’, greenspace,’neighbourhood environment’ and ‘attention’, ‘concentration’, ‘cognition’, ‘cogniti

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Dave, Prachi, S. Sudha, Vijaya Raghavan, and P. Poornachandrika. "Association Between Built Space and Cognition in Humans: A Scoping Review." INDIAN JOURNAL OF MENTAL HEALTH AND NEUROSCIENCES 3, no. 01 (2020): 23–35. http://dx.doi.org/10.32746/ijmhns.2020.v3.i1.46.

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Background: Various built environment characteristics have been shown to have varied effects on mental health. There is evidence for a positive relationship between certain BE characteristics and mental disorders. The talk on cognition is inevitable in any argument on mental health. This review looks at various BE characteristics and cognition and their interrelationship. Materials and Methods: A Boolean search of PubMed articles with keywords like ‘built environment’, ‘built spaces’, ‘green spaces’, greenspace,’neighbourhood environment’ and ‘attention’, ‘concentration’, ‘cognition’, ‘cogniti

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Nautiyal, Anuj. "Drug Abuse & Cognitive Functioning." Mental Health & Human Resilience International Journal 7, no. 2 (2023): 1–11. http://dx.doi.org/10.23880/mhrij-16000227.

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Extensive research has been conducted on cognitive impairments in substance use disorders, particularly with the advancements in cognitive and computational neuroscience as well as neuroimaging techniques over the past two decades. It has been noticed that, impaired cognition functioning is the common factor in person with substance abuse most precisely in case of attention, memory, recalling, decision making etc. Addiction has been showing basic impairment in brain and brain related processes through improper regulation and decreasing motivation and development of apathy. This special issue a

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Palser, Eleanor. "Discussion paper: The role of sensorimotor signals in cognition and its relevance to autism." PsyPag Quarterly 1, no. 108 (2018): 26–29. http://dx.doi.org/10.53841/bpspag.2018.1.108.26.

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Cognitive science has traditionally championed the separation of processing into the constituent domains of perception, action, and higher cognition, with very little crosstalk either amongst scientists researching different domains or conceptually, between nodes in the network. Cognitive reasoning, for example, is widely considered to occur in segregation to motor processing. However, increasingly, evidence is suggesting that this artificial separation of function may not be reflected in the computational organisation of the brain. Theories of embodied cognition argue that sensorimotor cues f

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Gao, Yan, Dandan Wang, Qian Wang, et al. "Causal Impacts of Psychiatric Disorders on Cognition and the Mediating Effect of Oxidative Stress: A Mendelian Randomization Study." Antioxidants 14, no. 2 (2025): 162. https://doi.org/10.3390/antiox14020162.

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Many psychiatric disorders are associated with major cognitive deficits. However, it is uncertain whether these deficits develop as a result of psychiatric disorders and what shared risk factors might mediate this relationship. Here, we utilized the Mendelian randomization (MR) analysis to investigate the complex causal relationship between nine major psychiatric disorders and three cognitive phenotypes, while also examining the potential mediating role of oxidative stress as a shared biological underpinning. Schizophrenia (SZ), major depressive disorder (MDD), and attention deficit hyperactiv

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Bora, E., and C. Pantelis. "Meta-analysis of social cognition in attention-deficit/hyperactivity disorder (ADHD): comparison with healthy controls and autistic spectrum disorder." Psychological Medicine 46, no. 4 (2015): 699–716. http://dx.doi.org/10.1017/s0033291715002573.

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BackgroundImpairment in social cognition is an established finding in autism spectrum disorders (ASD). Emerging evidence suggests that attention-deficit/hyperactivity disorder (ADHD) might be also associated with deficits in theory of mind (ToM) and emotion recognition. However, there are inconsistent findings, and it has been debatable whether such deficits persist beyond childhood and how similar social cognitive deficits are in ADHD v. ASD.MethodWe conducted a meta-analysis of social cognition, including emotion recognition and ToM, studies in ADHD compared with healthy controls and ASD. Th

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Abdi, Zeinab, and Tonmoy Sharma. "Social Cognition and Its Neural Correlates in Schizophrenia and Autism." CNS Spectrums 9, no. 5 (2004): 335–43. http://dx.doi.org/10.1017/s1092852900009317.

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AbstractThe study of social cognition in psychiatric disorders has become increasingly popular in recent years. This is due to the its proposed link to social functioning and the inability of general neurocognitive skills to explain the spectrum of impairments observed in patients. This article reviews research into two of the processes thought to underlie social cognition (emotion perception and theory of mind) in schizophrenia and autism. This is followed by a look at neuroimaging studies and their efforts to localize the neural correlates of emotion perception and theory of mind in the two

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Vladusich, Tony. "Towards a computational neuroscience of autism-psychosis spectrum disorders." Behavioral and Brain Sciences 31, no. 3 (2008): 282–83. http://dx.doi.org/10.1017/s0140525x08004433.

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AbstractCrespi & Badcock (C&B) hypothesize that psychosis and autism represent opposite poles of human social cognition. I briefly outline how computational models of cognitive brain function may be used as a resource to further develop and experimentally test hypotheses concerning “autism-psychosis spectrum disorders.”1

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Oliver, Lindsay, Iska Moxon-Emre, Aristotle Voineskos, and Stephanie Ameis. "M49. BEHAVIOURAL SOCIAL COGNITION IN SCHIZOPHRENIA SPECTRUM DISORDERS IN COMPARISON TO AUTISM SPECTRUM DISORDER: A SYSTEMATIC REVIEW AND META-ANALYSIS." Schizophrenia Bulletin 46, Supplement_1 (2020): S152—S153. http://dx.doi.org/10.1093/schbul/sbaa030.361.

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Abstract Background Schizophrenia spectrum disorders (SSDs) and autism spectrum disorder (ASD) both feature social cognitive deficits, which are highly debilitating. These include lower-level processes (e.g. emotion recognition), thought to be subserved by a frontoparietal mirroring network, and higher-level mentalizing processes (e.g. theory of mind), involving cortical midline and lateral temporal brain regions. Across both disorders, impairments in social cognition persist over time, drive disability, and predict functional outcome. Overlapping symptoms in SSDs and ASD have long been recogn

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Lence, Miloseva, Milosev Vladimir, and Rihter Kneginja. "COGNITION AND SUICIDE: EFFECTIVENESS OF COGNITIVE BEHAVIOUR THERAPY." International Journal of Cognitive Research in Science, Engineering and Education (IJCRSEE) 4, no. 1 (2016): 79–84. https://doi.org/10.5937/IJCRSEE1601079M.

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The aim of this review paper is to show an overview of the empirical evidence of effectiveness of cognitive behavioral therapy (CBT) in reducing suicidal cognitions and suicidal behavior. The topic of suicidal cognition and suicidal behavior is of special importance to clinicians and practitioners. Analyses of empirical findings from the oldest, first systematic review and meta-analysis and the newest one shown that there not enough evidence from clinical trials to suggest that CBT focusing on mental illness reduces suicidal cognitions and behaviors. But, from the other hand, CBT focusing on s

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Rubinsten, Orly, and Avishai Henik. "Comorbidity: Cognition and biology count!" Behavioral and Brain Sciences 33, no. 2-3 (2010): 168–70. http://dx.doi.org/10.1017/s0140525x10000695.

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AbstractWe agree with Cramer et al. that pure cases of behavioral disorders with no symptom overlaps are rare. However, we argue that disorders do exist and the network idea is limited and limiting. Networks of symptoms are observed mainly at behavioral levels. The core deficit is commonly at the cognitive or brain levels, and there the story is completely different.

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Drugas, Marius. "REVIEW OF THE BOOK “EMBODIED HOT COGNITIVE VULNERABILITY TO EMOTIONAL DISORDERS. FROM THEORY TO TREATMENT”, WRITTEN BY ALEXANDRU TIBA." Psychological Thought 14, no. 1 (2021): 265–68. http://dx.doi.org/10.37708/psyct.v14i1.608.

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This review presents a book that brings a fresh perspective on emotional disorders. The author of the book starts from renowned models in psychological counseling and psychotherapy and identifies the need for a different focus on factors that lead to vulnerability, namely the embodied hot cognition. Each chapter of the book carefully indicates the current state of research in the field (hot cognitions, distorted hot cognition, stress-related neuroadaptations, disturbed motivation, irrational beliefs), including recent neuropsychological findings. The final chapter presents an evidence-based tr

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