Gotowe bibliografie tematyczne / Cyclosporin

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Cyclosporin”

Autor: Grafiati
Data publikacji: 4 czerwca 2021
Data aktualizacji: 21 lutego 2023

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Artykuły w czasopismach na temat "Cyclosporin"

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Charuk, J. H., P. Y. Wong, and R. A. Reithmeier. "Differential interaction of human renal P-glycoprotein with various metabolites and analogues of cyclosporin A." American Journal of Physiology-Renal Physiology 269, no. 1 (July 1, 1995): F31—F39. http://dx.doi.org/10.1152/ajprenal.1995.269.1.f31.

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Interactions of P-glycoprotein with several analogues and metabolites of cyclosporin A were studied to gain a better understanding of this immunosuppressant's mechanism of excretion and nephrotoxicity. Incorporation of [3H]azidopine into human renal P-glycoprotein in the presence of various concentrations of different cyclosporins was quantitated. Competitive [3H]azidopine photolabeling and 3H drug transport assays of CHRC5 multidrug-resistant cells were also conducted to evaluate effects of cyclosporins on P-glycoprotein function. Cyclosporins A [half-maximal inhibition constant (K0.5) = 20 n
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Hušák, Michal, Bohumil Kratochvíl, Martin Buchta, Ladislav Cvak, and Alexandr Jegorov. "Crystal Structure of Cyclosporin E." Collection of Czechoslovak Chemical Communications 63, no. 1 (1998): 115–20. http://dx.doi.org/10.1135/cccc19980115.

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The structure of cyclosporin E was determined by X-ray diffraction methods and compared with the structure of related cyclosporins. In contrast to cyclosporin A, which crystallizes from acetone as tetragonal dihydrate, cyclosporin E acetone solvate monohydrate (C61H109N11O12·C3H6O·H2O) crystallizes under the same conditions in the monoclinic space group P21 with a = 15.698(2) Å, b = 21.333(3) Å, c = 13.224(2) Å, β = 103.74(1)°, Z = 2, and V = 4 302(1) Å3.
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Traber, R., H. Kobel, H. R. Loosli, H. Senn, B. Rosenwirth, and A. Lawen. "[Melle4]Cyclosporin, a Novel Natural Cyclosporin with anti-HIV Activity: Structural Elucidation, Biosynthesis and Biological Properties." Antiviral Chemistry and Chemotherapy 5, no. 5 (October 1994): 331–39. http://dx.doi.org/10.1177/095632029400500507.

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From fermentations of Tolypocladium niveum supplemented with D-threonine, a novel natural cyclosporin, [Melle4]cyclosporin, was isolated. Its structural elucidation is based on amino acid analysis and spectroscopic data; the amino acid sequence was deduced from two-dimensional NMR investigations applied to the iso-derivative of [Melle4]cyclosporin which, in contrast to the natural product, is present as one homogenous conformation in solution. We show that one of the four N-methyl-L-leucine units of cyclosporin A, namely that in position 4, is replaced by N-methyl-L-isoleucine. The putative me
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Lawen, A., R. Traber, R. Reuille, and M. Ponelle. "In vitro biosynthesis of ring-extended cyclosporins." Biochemical Journal 300, no. 2 (June 1, 1994): 395–99. http://dx.doi.org/10.1042/bj3000395.

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Cyclosporin synthetase, a multifunctional polypeptide, catalyses the biosynthesis of the set of natural cyclosporins. We report that this enzyme is also capable of introducing a beta-alanine into position 7 or 8 of the ring instead of the alpha-alanines present at these positions in cyclosporin A. This leads to 34-membered rings in contrast to the 33-membered ring of the cyclo-undecapeptide cyclosporin A. Both [beta Ala7]CyA and [beta Ala8]CyA show immunosuppressive activity. The cyclosporin synthetase-related enzyme peptolide SDZ 214-103 synthetase, on the other hand, does not incorporate eit
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Schramm, U., G. Fricker, R. Wenger, and D. S. Miller. "P-glycoprotein-mediated secretion of a fluorescent cyclosporin analogue by teleost renal proximal tubules." American Journal of Physiology-Renal Physiology 268, no. 1 (January 1, 1995): F46—F52. http://dx.doi.org/10.1152/ajprenal.1995.268.1.f46.

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The transport of a fluorescent cyclosporin analogue was measured in killifish (Fundulus heteroclitus) proximal tubules by means of epifluorescence microscopy and digital image analysis. Renal cells rapidly accumulated the cyclosporin analogue from the medium and attained steady state within 60 min; luminal fluorescence increased over the first 60-90 min. At steady state, luminal fluorescence intensity was two to three times higher than cellular. Cellular fluorescence intensity was a linear function of medium substrate concentration and was not affected by any treatment used. In contrast, lumin
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Langman, L., D. LeGatt, and R. Yatscoff. "Cross-reactivities of cyclosporin G (NVa2 cyclosporin) and metabolites in cyclosporine A immunoassays." Clinical Biochemistry 26, no. 2 (April 1993): 122. http://dx.doi.org/10.1016/0009-9120(93)90040-d.

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Kratochvíl, Bohumil, Alexandr Jegorov, Svetlana Pakhomova, Michal Hušák, Petr Bulej, Ladislav Cvak, Petr Sedmera, and Vladimír Havlíček. "Crystal Structures of Cyclosporin Derivatives: O-Acetyl-(4R)-4-(E-2-butyl)-4,N-dimethyl-L-threonyl-cyclosporin A and O-Acetyl-(4R)-4-[E-2-(4-bromobutyl)]-4,N-dimethyl-L-threonyl-cyclosporin A." Collection of Czechoslovak Chemical Communications 64, no. 1 (1999): 89–98. http://dx.doi.org/10.1135/cccc19990089.

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The structures of O-acetyl-(4R)-4-(E-2-butyl)-4,N-dimethyl-L-threonyl-cyclosporin A (1) and O-acetyl-(4R)-4-[E-2-(4-bromobutyl)]-4,N-dimethyl-L-threonyl-cyclosporin A (2) were determined by X-ray diffraction methods and compared with the structure of related cyclosporins. In contrast to expectation, neither the acetylation nor the subsequent bromination of 1 affects the conformation and packing of cyclosporins in the solid state. Both compounds are isomorphous and crystallize in the orthorhombic space group P212121 with a = 12.936(2) Å, b = 15.590(2) Å, c = 36.280(3) Å, and a = 12.916(3) Å, b
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Elbakidze, G. M., A. G. Medentsev, and A. G. Elbakidze. "INFLUENCE OF PRODIGIOZAN-DEPENDENT COMUTON ON THE RESISTANCE OF LIVER MITOCHONDRIA AGAINST DAMAGE BY PROTONOFOR." Annals of the Russian academy of medical sciences 69, no. 1-2 (August 20, 2015): 75–79. http://dx.doi.org/10.15690/vramn.v69.i1-2.946.

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An effector of tissue stress of hepatocytes, prodigiozan-dependent comuton (PDC), provokes deenergiezation of liver mitochondria, preloaded by Ca2+ ions. In this case a decrease of membrane potential (MP) and Ca2+ efflux by cyclosporine A sensitive mechanism of megapore is observed. If megapore is blocked by cyclosporin A, protonofor FCCP provoked decrease of MP and Ca2+ efflux by cyclosporin A-insensitive mechanism. It is shown that PDC increases resistance of mitochondria to mentioned protonofor action by inhibition of both these effects. An inhibitory action of PDC is realized by K+ and NAD
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Kim, J. Y., S. H. Park, K. S. Cho, H. J. Kim, C. K. Lee, K. K. Park, S. H. Choi, and W. Y. Chung. "Mechanism of Azithromycin Treatment on Gingival Overgrowth." Journal of Dental Research 87, no. 11 (November 2008): 1075–79. http://dx.doi.org/10.1177/154405910808701110.

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Azithromycin is effective for the remission of cyclosporine A-induced gingival overgrowth (CIGO) in persons who have undergone renal transplant. To explain its mechanism in alleviating the clinical symptoms of these indivduals, we examined the effect of azithromycin on cell proliferation and collagen turnover modified by cyclosporin A in human gingival fibroblasts from healthy persons and from persons who had undergone renal transplant. Cyclosporin A-induced proliferation of renal transplant fibroblasts and normal fibroblasts was inhibited by azithromycin. Azithromycin elevated the reduced met
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&NA;. "Cyclosporin see Minoxidil/cyclosporin." Reactions Weekly &NA;, no. 312 (August 1990): 6. http://dx.doi.org/10.2165/00128415-199003120-00024.

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Rozprawy doktorskie na temat "Cyclosporin"

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Husi, Holger. "Cyclosporins and cyclosporin binding proteins : an insight into the mechanism of immunosuppression /." [S.l.] : [s.n.], 1994. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=10937.

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James, Jacqueline A. "Cyclosporin A and gingival overgrowth." Thesis, Queen's University Belfast, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.282191.

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French, Martin Thomas. "Fluorescence immunoassay for cyclosporin A." Thesis, Loughborough University, 1991. https://dspace.lboro.ac.uk/2134/33279.

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Monodansylcadavarine (MDC) was used to synthesise a fluorescent derivative of cyclosporin A and the product of the reaction was isolated by preparative thin layer chromatography (TLC) and purified by high performance liquid chromatography (HPLC). The fluorescent derivative was shown to bind with a polyclonal antibody to cyclosporin A by submitting the derivative for analysis by cyclosporin radioimmunoassay (RIA). However this derivative did not bind with a monoclonal antibody used in a RIA specific for the parent compound. To achieve this fluorescent derivatives were synthesised using cyclospo
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Gerber, Andreas. "Totalsynthese von Cyclosporin A an der Festphase /." [S.l.] : [s.n.], 2009. http://edoc.unibas.ch/diss/DissB_8755.

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Mereish, Kulthoum A. "Alteration of cyclosporin : a bioavailability through complexation /." Ann Arbor : University Microfilms International, 1985. http://www.gbv.de/dms/bs/toc/01641747x.pdf.

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David, Oliver Jean Claude. "New approaches to improve cyclosporin A monitoring." Thesis, Queen Mary, University of London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.395488.

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Hutchison, Stephen Michael William. "Studies in cyclosporin nephrotoxicity in the rat." Thesis, Queen's University Belfast, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335321.

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Powles, A. V. "Cyclosporin for psoriasis : clinical and immunopathological studies." Thesis, University of Aberdeen, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.253658.

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Ten patients with severe intractable psoriasis were treated with cyclosporin (CyA) at an average dose of 3 mg/kg/day for a period of 12 weeks. At the end of the study, 5 patients had a greater than 90% reduction in their PASI (psoriasis area severity index) score, 3 an 80%, one a 69% and one a 52% reduction. In a long term study, 13 patients with severe psoriasis were treated with CyA for an average duration of 2.5 years. The average dose of CyA was 3 mg/kg/day, with a range of 1 - 5 mg/kg/day. The average reduction in mean PASI score throughout the study was 70 - 80%. Seven of the 13 patients
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Spitzfaden, Claus. "Strukturbestimmung des Cyclophilin/Cyclosporin-Komplexes mittels NMR-Spektroskopie /." [S.l.] : [s.n.], 1993. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=10406.

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Gillam, Elizabeth Maree Jeffery. "The interaction of cyclosporin A with cytochromes P450." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.280968.

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Książki na temat "Cyclosporin"

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Thomson, Angus W., ed. Cyclosporin. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8.

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Carton, Angela Shirleen. The ultrastructural effects of Cyclosporin A. on Trichinella Spriralis. Manchester: University of Manchester, 1994.

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Lian, Jeromy. In vitro induction of P-glycoprotein by cyclosporin A. Ottawa: National Library of Canada, 2003.

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Kappos, Ludwig. Immunsuppressive Therapie der multiplen Sklerose mit Cyclosporin A und Azathioprin. Berlin, Heidelberg: Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74876-9.

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Green, Colin. Recent progress in organ transplantation. Oxford: Medicine Group for the Medicine Publishing Foundation, 1988.

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Edwards, Brian David. Mechanisms and reduction of cyclosporin toxicity in the normal human kidney. Manchester: University of Manchester, 1994.

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Midha, Rajiv. Comparison of regeneration across rat nerve allografts with temporary or continuous cyclosporin A immunosuppression. Ottawa: National Library of Canada = Bibliothèque nationale du Canada, 1993.

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Warner, Ellen. Phase I - II study of vinblastine and oral cyclosporin a in metastatic renal cell carcinoma. Ottawa: National Library of Canada, 1996.

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Wilkie, Martin Erskine. Clinical and laboratory based studies into the effects of calcium antagonists on cyclosporin A nephrotoxicity. Manchester: University of Manchester, 1994.

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Jutta Wehrens. Einfluß der Immunsuppressiva Cyclosporin A und Tacrolimus auf den Gefäßtonus von Nierenarterien der Ratte und des Menschen. Freiburg im Breisgau: Selbstverlag, 2000.

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Części książek na temat "Cyclosporin"

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Kay, John E. "Inhibitory effects of cyclosporin A on lymphocyte activation." In Cyclosporin, 1–23. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_1.

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Powles, Anne V., Barbara S. Baker, and Lionel Fry. "Cyclosporin A and skin disease." In Cyclosporin, 191–212. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_10.

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von Graffenried, Beat, David Friend, Nicholas Shand, Wilfried Scheiss, and Pentti Timonen. "Cyclosporin A (Sandimmun®) in autoimmune disorders." In Cyclosporin, 213–51. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_11.

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Grevel, Joachim, and Barry D. Kahan. "Pharmacokinetics of cyclosporin A." In Cyclosporin, 252–66. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_12.

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Burke, M. Danny, Fiona Macintyre, D. Cameron, and Paul H. Whiting. "Cyclosporin A Metabolism and Drug Interactions." In Cyclosporin, 267–302. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_13.

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Whiting, Paul H., and Angus W. Thompson. "Pathological effects of cyclosporin A in experimental models." In Cyclosporin, 303–23. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_14.

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Thiru, Sathia. "Pathological effects of cyclosporin A in clinical practice." In Cyclosporin, 324–64. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_15.

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Hess, Allan D. "Effect of cyclosporin A on the immune response: pivotal role of the interleukin-2/ interleukin-2 receptor autocrine pathway." In Cyclosporin, 24–33. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_2.

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Motta, Iris, and Paolo Truffa-Bachi. "Influence of CsA on humoral immunity and on B lymphocyte activation." In Cyclosporin, 34–49. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_3.

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Thomson, Angus W., and Janet I. Duncan. "The influence of cyclosporin A on T cell activation, cytokine gene expression and cell-mediated immunity." In Cyclosporin, 50–81. Dordrecht: Springer Netherlands, 1989. http://dx.doi.org/10.1007/978-94-009-0859-8_4.

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Streszczenia konferencji na temat "Cyclosporin"

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Zoja, C., L. Furci, F. Ghilardi, P. Zilio, A. Benigni, and G. Remuzzi. "CYCLOSPORIN A (CyA) INDUCED ENDOTHELIAL CELL INJURY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644123.

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The chronic administration of CyA to animals and humans to prevent graft rejection may induce renal arteriolar damage resembling hemolytic uremic syndrome (HUS). This is a syndrome of vascular damage with thrombotic occlusions of the microcirculation. Endothelial damage is considered the first event in the pathogenetic cascade leading to HUS. We have used bovine aortic endothelial cells in culture to address the issue of CyA-induced arteriolar damage. CyA-induced a time (1-24 hours) and dose (1-50 μM) dependent cell damage. CyA-induced injury was characterized by an early cell detachment follo
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Gerards, AH, RB Landewé, AP Prins, GA Bruijn, HS Goei The, and BA Dijkmans. "FRI0031 Cyclosporin-a plus methotrexate combination therapy is as safe as cyclosporin-a alone in patients with early rheumatoid arthritis." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.1160.

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Koreeda, Y., K. Kubota, T. Matsuda, T. Taira, S. Noma, and T. Kawabata. "Factors Influencing Cyclosporin Blood Concentration in Interstitial Pneumonia Patients." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a4048.

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Han, Weinong, Mei Ming, Tong-Chuan He, and Yu-Ying He. "Abstract 341: Effects of immunosuppressive cyclosporin A on epidermal keratinocytes." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-341.

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Shaw, R. A., Henry H. Mantsch, and Babur Z. Chowdhry. "Solvent and metal ion effects on the conformation of cyclosporin." In Fourier Transform Spectroscopy: Ninth International Conference, edited by John E. Bertie and Hal Wieser. SPIE, 1994. http://dx.doi.org/10.1117/12.166699.

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Schuh, L., and R. Mischke. "Anwendung von Cyclosporin A bei Hunden mit primärer immunvermittelter hämolytischer Anämie." In 27. Jahrestagung der FG „Innere Medizin und klinische Labordiagnostik“ der DVG (InnLab), 2./3. Februar 2019 in München – Teil 2. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1679117.

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Boda, Z., J. Hársfalvi, K. Pecze, and K. Rak. "ACQUIRED HAEMOPHILIA DUE TO FACTOR VIII INHIBITOR WITH SEVERE HAEMORRHAGES IN A 46-YEAR-OLD WOMAN SUCCESSFULLY TREATED WITH CYCLOSPORIN A." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644845.

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A formerly healthy 46-year-old woman suffering from acquired haemophilia caused by factor VIII antibodies was admitted in an unconscious state following subarachnoid haemorrhage. Treatment with prothrombin complex concentrates (taken as a whole 100 000 U of PCC, home made and 10 OOOUofFEIBA, Immuno) , steroid (Prednison 50 mg/day) and cyclophosphamide (100 mg/day) was only partially successful: neurological state improved but the haemorrhagic tendency remained. Significant haematuria, and skin and mucosal bleeding characterized her clinical picture. In the meantime, signs of non-A non-B hepati
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Krajnc, I., K. Lukanoviè, and I. Holc. "SAT0101 Analysis of the efficacy of cyclosporin a in 56 psoriatic patients." In Annual European Congress of Rheumatology, Annals of the rheumatic diseases ARD July 2001. BMJ Publishing Group Ltd and European League Against Rheumatism, 2001. http://dx.doi.org/10.1136/annrheumdis-2001.484.

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Cohen, H., I. J. Mackie, R. Patel, G. Neild, and S. J. Machin. "PERSISTENT DECREASED FIBRINOLYTIC ACTIVITY IN CYCLOSPORIN A (CyA) TREATED RENAL ALLOGRAFT RECIPIENTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644659.

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CyA administration isssociated with histological evidenceof vascular injury. Since vascularendothelial cell components are known to modify fibrinolytic activity, we have examined fibrinolytic changes in renal allograft recipients onCyA and, in addition, markers of vascular endothelial cell damage: plasma vWFRiCof activity and AT-III activity.Parameters of fibrinolytic activity have been assessed serially in 21 renal allograft recipients for 1 year post-transplantation by study of the following: euglobulin clot lysis time (ELT), plasminogen activator activity (PAA) by fibrin plate assay, tissue
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Forstner, M., S. Lin, X. Yang, S. Kinting, I. Rothenaigner, K. Schorpp, Y. Li, K. Hadian, and M. Griese. "High-content screen identifies cyclosporin A as a novel ABCA3-specific molecular corrector." In 43. Jahrestagung der Gesellschaft für Pädiatrische Pneumologie. Georg Thieme Verlag, 2022. http://dx.doi.org/10.1055/s-0042-1754515.

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Raporty organizacyjne na temat "Cyclosporin"

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Drayna, Paul M., Matthew Caldwell, Vasudha Panday, and Charles Reilly. Evaluation of Topical Cyclosporine in Preventing the Development of Corneal Haze after Photorefractive Keratectomy. Fort Belvoir, VA: Defense Technical Information Center, May 2014. http://dx.doi.org/10.21236/ada602027.

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Hofmann, Nina, Robert Ambühl, Suzana Jordan, and Oliver Distler. A systematic literature review- Calcineurin inhibitors treatment in systemic sclerosis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, October 2021. http://dx.doi.org/10.37766/inplasy2021.10.0095.

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Review question / Objective: To systematically review treatment effectiveness and adverse events of calcineurin inhibitors (CNIs) such as cyclosporine A (CsA) and tacrolimus in patients with systemic sclerosis (SSc). Condition being studied: Systemic sclerosis. Eligibility criteria: • Publications with SSc patients treated with CNIs and available information on the outcome of CNI therapy on SSc disease manifestation will be analysed.• Article types: case reports, clinical study, clinical trial, controlled clinical trial, historical article, randomized controlled trial.
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