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Rozprawy doktorskie na temat „Cyclosporin”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 21 lutego 2023

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1

Husi, Holger. "Cyclosporins and cyclosporin binding proteins : an insight into the mechanism of immunosuppression /." [S.l.] : [s.n.], 1994. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=10937.

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2

James, Jacqueline A. "Cyclosporin A and gingival overgrowth." Thesis, Queen's University Belfast, 1995. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.282191.

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3

French, Martin Thomas. "Fluorescence immunoassay for cyclosporin A." Thesis, Loughborough University, 1991. https://dspace.lboro.ac.uk/2134/33279.

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Monodansylcadavarine (MDC) was used to synthesise a fluorescent derivative of cyclosporin A and the product of the reaction was isolated by preparative thin layer chromatography (TLC) and purified by high performance liquid chromatography (HPLC). The fluorescent derivative was shown to bind with a polyclonal antibody to cyclosporin A by submitting the derivative for analysis by cyclosporin radioimmunoassay (RIA). However this derivative did not bind with a monoclonal antibody used in a RIA specific for the parent compound. To achieve this fluorescent derivatives were synthesised using cyclospo

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4

Gerber, Andreas. "Totalsynthese von Cyclosporin A an der Festphase /." [S.l.] : [s.n.], 2009. http://edoc.unibas.ch/diss/DissB_8755.

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5

Mereish, Kulthoum A. "Alteration of cyclosporin : a bioavailability through complexation /." Ann Arbor : University Microfilms International, 1985. http://www.gbv.de/dms/bs/toc/01641747x.pdf.

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6

David, Oliver Jean Claude. "New approaches to improve cyclosporin A monitoring." Thesis, Queen Mary, University of London, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.395488.

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7

Hutchison, Stephen Michael William. "Studies in cyclosporin nephrotoxicity in the rat." Thesis, Queen's University Belfast, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335321.

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8

Powles, A. V. "Cyclosporin for psoriasis : clinical and immunopathological studies." Thesis, University of Aberdeen, 1989. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.253658.

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Ten patients with severe intractable psoriasis were treated with cyclosporin (CyA) at an average dose of 3 mg/kg/day for a period of 12 weeks. At the end of the study, 5 patients had a greater than 90% reduction in their PASI (psoriasis area severity index) score, 3 an 80%, one a 69% and one a 52% reduction. In a long term study, 13 patients with severe psoriasis were treated with CyA for an average duration of 2.5 years. The average dose of CyA was 3 mg/kg/day, with a range of 1 - 5 mg/kg/day. The average reduction in mean PASI score throughout the study was 70 - 80%. Seven of the 13 patients

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9

Spitzfaden, Claus. "Strukturbestimmung des Cyclophilin/Cyclosporin-Komplexes mittels NMR-Spektroskopie /." [S.l.] : [s.n.], 1993. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=10406.

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10

Gillam, Elizabeth Maree Jeffery. "The interaction of cyclosporin A with cytochromes P450." Thesis, University of Oxford, 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.280968.

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11

Wastling, Jonathan Mark. "The action of cyclosporin A on helminth parasites." Thesis, University of Aberdeen, 1990. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU031936.

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The influence of the immunomodulatory/antiparasitic drug cyclosporin A (CsA) on parasitic infections is discussed and its effects on three helminths examined. The biology of the tapeworm Hymenolepis microstoma is reviewed and the action of CsA on this parasite investigated. Therapeutic CsA treatment was antagonistic to both juvenile and adult H.microstoma, reducing worm weight, delaying migration into the bile duct, suppressing egg production and lowering parasite survival. CsA also showed significant chemoprophylactic properties against H.microstoma CsA treatment in vivo caused gross and ultr

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12

Khan, Lillian Nasreen. "Effect of cyclosporin A on the immunopathology of asthma." Thesis, Imperial College London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.409081.

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13

Aldridge, R. D. "Modulation of delayed-type hypersensitivity reactions by cyclosporin A." Thesis, University of Aberdeen, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.234482.

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The investigations have given rise to the following findings: 1. CsA is an effective immunosuppressant of both the induction and elicitation phases of tuberculin-like and contact delayed-type hypersensitivity (DTH) and of the induction phase of Jones-Mote hypersensitivity. 2. The effective suppression of tuberculin-like DTH responses in the guinea pig is not dependent upon cyclophosphamide-sensitive suppressor cells. 3. Topically applied CsA inhibits the elicitation of contact dermatitis in experimental animals. 4. The kinetics of percutaneous CsA absorption have been determined, as has the ex

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14

Gudgeon, M. C. "Studies on the mechanism of action of cyclosporin A." Thesis, University of Sussex, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.378279.

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15

Diehl, Rita. "Verträglichkeit und Effektivität Cyclosporin A-vermittelter Immunsuppression beim Schaf für die xenogene, intrazerebrale Transplantation." Doctoral thesis, Universitätsbibliothek Leipzig, 2016. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-214365.

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Einleitung Der Einsatz von Stammzellen als Grundlage neuer therapeutischer Strategien wird bereits seit über 25 Jahren intensiv erforscht. Stammzellen sind in der Lage, in verschiedene funktionale Zelltypen auszudifferenzieren und verfügen über ein enormes Proliferationspotential (NAM et al. 2015). Ausgehend von den Fähigkeiten von Stammzellen sehen Forscher und Kliniker erstmals eine realistische Möglichkeit, kurative Therapieoptionen für Erkrankungen zu entwickeln, die bisher als schwer behandelbar oder sogar unheilbar angesehen wurden. Davon könnten insbesondere Patienten chronisch-degenera

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16

Riether, Carsten. "Peripheral mediatory mechanisms of behaviorally conditioned immunosuppression by cyclosporin A /." [S.l.] : [s.n.], 2008. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=18085.

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17

Zhang, Yixin. "Rational design of cyclosporin A derivatives for selective enzyme inhibition." [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=964279401.

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18

Ormrod, Douglas James. "Modulation of non-specific cellular defence mechanisms by cyclosporin A." Thesis, University of Auckland, 1990. http://hdl.handle.net/2292/3195.

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The immune response modifier Cyclosporin A (CsA) is widely used in the management of organ graft rejection and in the treatment of inflammatory disorders. CsA is a potent suppressor of T-lymphocyte function and it’s biological effects have been defined almost exclusively in these terms. However, recent studies in which the agent was shown to exacerbate a T-lymphocyte independent, experimentally induced bacterial infection of the kidney (pyelonephritis), indicated that CsA had effects on host defence mechanisms other than T-lymphocytes. The present study, using animal models, was undertaken to

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19

Wang, Su. "Transdermal delivery of cyclosporin A by electrically enhanced permeation techniques." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1997. http://www.collectionscanada.ca/obj/s4/f2/dsk3/ftp04/mq23183.pdf.

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20

Misseghers, Byron S. "The histologic characterization of perianal fistulas during treatment with cyclosporin." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0016/NQ47400.pdf.

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21

KONDO, TATSUHEI, HIROSHI TAKAGI, and TAKESHI MORIMOTO. "Canine Pancreatic Allotransplantation with Duodenum (Pancreaticoduodenal Transplantation) Using Cyclosporin A." Nagoya University School of Medicine, 1985. http://hdl.handle.net/2237/17478.

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22

Baumgart, Alessandra. "Cyclosporin A und dessen möglicher Einsatz bei der Tigerschecken-Uveitis." Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-168992.

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23

McNally, P. G. "The influence of calcium channel blockers on cyclosporin A nephrotoxicity." Thesis, University of Leicester, 1990. http://hdl.handle.net/2381/34162.

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Intrarenal vasoconstriction is a characteristic feature of cyclosporin A nephrotoxicity. This thesis investigates the effect of nifedipine, a dihydropyridine calcium channel blocker and potent renal vasodilator, on various aspects of experimental and clinical cyclosporin A nephrotoxicity. In the surgically intact spontaneously hypertensive rat (two-kidney model), short-term administration of cyclosporin A (14 days) induced a marked reduction in glomerular filtration rate and effective renal plasma flow, and an increase in renal vascular resistance. These changes were both reversible on stoppin

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24

Wang, Su. "Transdermal delivery of cyclosporin B by electrically enhanced permeation techniques /." St. John's, NF : [s.n.], 1997.

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25

Richter, Sebastian. "Rapamycin vs. Cyclosporin A : Auswirkungen auf Transplantatabstossung und Tumorwachstum im Tiermodell." kostenfrei, 2009. http://www.opus-bayern.de/uni-regensburg/volltexte/2009/1352/.

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26

Bäuerle, Alexander Lutz [Verfasser]. "Die Therapie der membranösen Glomerulonephritis mit Cyclosporin A / Alexander Lutz Bäuerle." Ulm : Universität Ulm. Medizinische Fakultät, 2012. http://d-nb.info/1028033567/34.

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27

McLauchlan, P. E. "Host-parasite interactions : cellular immune responses and modulation by cyclosporin A." Thesis, University of Aberdeen, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.593084.

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The role of cyclosporin A (CsA) as an immunomodulatory and antiparasitic drug was examined. The immune responses in various host:parasite relationships were examined giving an insight into the complicated nature of this relationship. In addition, analogues of CsA were screened to investigate the antiparasitic mode of action of cyclosporins. Expulsion of intestinal parasites has been proposed to involve the cellular immune response but its precise role is unknown. In CBA/ca mice infected with <I>Hymenolepsis diminuta</I>, peak intestinal mucosal mast cell (IMMC) numbers and release of mast cell

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28

Myrillas, Theofilos T. "Cellular mechanisms underlying the pathogenesis of cyclosporin A-induced gingival overgrowth." Thesis, Queen's University Belfast, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.284391.

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29

Chan, Weng C. "A study of the medicinal chemistry related to the C9-ene amino acid of cyclosporin." Thesis, University of Nottingham, 1988. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.381104.

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30

Gennai, Stéphane. "Effets de la cyclosporine A sur des poumons porcins reperfusés ex vivo." Thesis, Grenoble, 2013. http://www.theses.fr/2013GRENS012/document.

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Objectif De nombreux travaux ont souligné le rôle de la Cyclosporine A (CsA) dans la prévention des lésions d'ischémie-reperfusion (I/R) mais aucun n'a été effectué sur poumons isolés de grands mammifères. Notre objectif était de mesurer pour la première fois les effets de la CsA sur les lésions d'I/R dans un modèle de poumons porcins reperfusés ex vivo, en évaluant plusieurs doses de CsA pour différents temps d'ischémie. Méthodes L'expérimentation A a été conduite sur 4 groupes de 8 paires de poumons chacune : un groupe contrôle et 3 groupes recevant différentes concentrations de CsA (1, 10 o

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31

Saunders, R. N. "The effect of rapamycin after cyclosporin dose reduction on chronic allograft nephropathy." Thesis, University of Leicester, 2003. http://hdl.handle.net/2381/29442.

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Chronic allograft nephropathy (CAN) is the commonest cause of late decline in renal allograft function and subsequent failure. Histopathologically it is underpinned by the accumulation of extracellular matrix. The first chapter provides a thorough review of the current opinions regarding the aetiology, pathophysiology and management of this complex condition. Overexposure to Cyclosporin is a major risk factor for chronic allograft nephropathy and thus Cyclosporin dose reduction has been advocated in some reports. Rapamycin is a relatively new immunosuppressant, recently introduced in renal tra

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32

Horrocks, C. "Immunological studies into the mechanisms of action of cyclosporin A in psoriasis." Thesis, University of Aberdeen, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.592590.

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Seven chronic plaque psoriasis patients were treated with cyclosporin A (CsA, 2.5-5.0 mg/kg/day). A marked resolution of psoriasis was observed within 4 weeks of treatment. Immunophenotypic analysis of patients' peripheral blood mononuclear cells (PBML) before and during CsA therapy revealed no difference in comparison to normal nor any change following CsA therapy. Immunocytochemical analysis of lesional skin, however, revealed marked reductions in infiltrating CD3+ (pan T), CD4+ (T helper), CD25+ (interleukin 2 receptor) and to a lesser extent CD8+ (T cytotoxic/suppressor) cells within 4 wee

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33

Heys, Stephen D. "The potentiation and alleviation of cyclosporin A nephrotoxicity in the Lewis rat." Thesis, University of Aberdeen, 1987. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.254668.

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Cyclosporin A immunosuppression following organ transplantation is associated with a reversible nephrotoxicity as manifested by elevations in serum urea and creatinine concentrations and urinary N-acetyl-B-D-glucosaminidase activities. The metabolism of Cyclosporin A is primarily via the hepatic cytochrome P-450 mono-oxygenase system with previous studies having demonstrated that the short term co-administration of phenobarbitone, an inducer of this enzyme system, ameliorates this nephrotoxicity in the non-renal allografted rat. Initial studies demonstrated that the induction of Cyclosporin A

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34

Takayama, Akira. "Transport of cyclosporin A in kidney epithelial cell line (LLC-PK[1])." Kyoto University, 1995. http://hdl.handle.net/2433/160728.

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本文データは平成22年度国立国会図書館の学位論文(博士)のデジタル化実施により作成された画像ファイルを基にpdf変換したものである<br>Kyoto University (京都大学)<br>0048<br>新制・論文博士<br>博士(医学)<br>乙第8919号<br>論医博第1514号<br>新制||医||613(附属図書館)<br>UT51-95-P410<br>(主査)教授藤田潤, 教授吉田修, 教授乾賢一<br>学位規則第4条第2項該当

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35

Cunningham, Charles. "Aspects of the relationship between the metabolism and toxicity of cyclosporin A." Thesis, University of Aberdeen, 1985. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU356459.

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The fungal metabolite cyclosporin A (CsA) is the first of a new order of immunosuppressant drugs. Unlike conventional immunosuppressive therapy, which produces "blanket suppression", CsA has a selective inhibitory effect on T cell activation. Although CsA does not cause the serious side-effects, such as myelotoxicity, normally associated with cytotoxic agents, the drug is not without its problems, the most clinically important of which are nephro- and hepatotoxicity. The primary aim of the work presented in this thesis was to study mechanisms of CsA- induced toxicity in the rat. In an initial

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36

Sieber, Matthias. "Modulatoren des Calcineurin-NFATc-Signalweges in humanen TH-Zellen." Phd thesis, Universität Potsdam, 2010. http://opus.kobv.de/ubp/volltexte/2010/4467/.

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Die Ca2+/Calmodulin-aktivierte Serin/Threonin-Phosphatase Calcineurin ist ein Schlüsselmolekül des T-Zell-Rezeptorabhängigen Signalnetzwerkes. Calcineurin aktiviert die Transkriptionsfaktoren der NFATc-Familie durch Dephosphorylierung und reguliert darüber die Expression wichtiger Zytokine und Oberflächenproteine. Die Aktivität von Calcineurin wird durch zahlreiche endogene Proteine moduliert und ist Angriffspunkt der immunsuppressiven Substanzen Cyclosporin A und FK506. In dieser Arbeit wurde der alternative niedermolekulare Calcineurin-NFATc-Inhibitor NCI3 hinsichtlich seiner Effekte auf T-

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37

Sedighiani, Fazilat. "Tacrolimus vs. Cyclosporin als primäre immunsuppressive Therapie bei akuten rezidivierenden Abstoßungsreaktionen nach Herztransplantation." Diss., lmu, 2006. http://nbn-resolving.de/urn:nbn:de:bvb:19-60184.

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38

Marlowe, Sharon Nalini Singh. "Azathioprine and cyclosporin A as second line treatments for severe leprosy type reactions." Thesis, London School of Hygiene and Tropical Medicine (University of London), 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.424945.

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39

Cheah, Su-Yin. "Methotrexate, cyclosporin and sulfasalazine in the treatment of rheumatoid arthritis : a systematic review." Thesis, University of Nottingham, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.285683.

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40

Gerlach, Thomas [Verfasser], Markus [Akademischer Betreuer] Neurath, and Benno [Akademischer Betreuer] Weigmann. "Molekulare Analyse des Immunsuppressivums Cyclosporin A / Thomas Gerlach. Gutachter: Markus Neurath ; Benno Weigmann." Erlangen : Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), 2015. http://d-nb.info/1075840473/34.

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41

Fischer, Ricardo Guimaräes. "The ferret in periodontal research clinical features, histology, microbiology and immunosuppression (Cyclosporin-A) /." Malmö : Dept. of Periodontology, Center for Oral Health Sciences, Lund University, 1993. http://catalog.hathitrust.org/api/volumes/oclc/28161627.html.

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42

MITSUYAMA, Hirohito, Fukushi KAMBE, Ryuichiro MURAKAMI, Naoki ISHIGURO, and Hisao SEO. "Analysis of Interieukin-8 Gene Promoter function in Human Osteoblast-like Cells : Regulation by Ca^<2+>-signaling and Cyclosporin A." Research Institute of Environmental Medicine, Nagoya University, 2002. http://hdl.handle.net/2237/2782.

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43

Östraat, Öyvind. "Experimental modulation and suppression of anti-allograft immune response." Malmö : Dept. of Surgery, Lund University, Malmö University Hospital, 1997. http://catalog.hathitrust.org/api/volumes/oclc/38950482.html.

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44

Mangels, Corinna. "Unerwünschte Arzneimittelwirkungen bei der Anwendung von Cyclosporin A (Atopica®) bei Hund und Katze." Diss., lmu, 2012. http://nbn-resolving.de/urn:nbn:de:bvb:19-147364.

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45

Hodgkinson, Brent C. "The effect of cyclosporin A (CsA) on antioxidant status in the male Wistar rat." Thesis, University of Ottawa (Canada), 2000. http://hdl.handle.net/10393/9042.

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Antioxidant utilization potentially is an excellent index of oxidative stress. These studies assessed the effects of two doses of CsA (10 mg/kg/day and 20 mg/kg/day for 14 days) on antioxidant status in numerous tissues, and on the function and structure of testis and kidney in male Wistar rats. Animals were placed on either vitamin E sufficient or deficient diets and injected subcutaneously with either CsA or vehicle. Vitamin E levels in various tissues were measured using the very sensitive technique of gas chomatography and mass spectrometry (GC-MS) to determine the effect of CsA on vitamin

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46

Echtler, Silvia. "Einfluss einer auf Cyclosporin A basierenden immunsuppressiven Therapie auf den Knochenmineralstoffwechsel nach orthotoper Herztransplantation." Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-172602.

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47

Cusack, Rodney Michael. "An investigation of the interaction of metal ions with cyclic octapeptides and cyclosporin A /." [St. Lucia, Qld.], 2001. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe16238.pdf.

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48

Passoth, Peter René. "Das Auftreten einer neurodermitisähnlichen Dermatitis bei Kindern nach Herztransplantation im ersten Lebensjahr unter Cyclosporin A." [S.l.] : [s.n.], 2001. http://deposit.ddb.de/cgi-bin/dokserv?idn=965520749.

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49

Malinowski, Michael. "Systemisches Mycophenolatmofetil versus Cyclosporin A nach perforierender Hochrisiko-Keratoplastik Ergebnisse einer randomisierten, prospektiven klinischen Studie /." [S.l.] : [s.n.], 2002. http://deposit.ddb.de/cgi-bin/dokserv?idn=967644240.

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50

KAMBE, Fukushi, Hisao SEO, and Xia CAO. "Cyclosporin A (CsA)-sensitive Pathway for the Induction of ZAKI-4 Expression by Thyroid Hormone." Research Institute of Environmental Medicine, Nagoya University, 2002. http://hdl.handle.net/2237/2778.

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