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Gotowe bibliografie tematyczne / Cytokinesis Genetic aspects / Rozprawy doktorskie

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Rozprawy doktorskie na temat „Cytokinesis Genetic aspects”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 30 stycznia 2023

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1

O'Keefe, Louise. "Genetic analysis of the role of pebble during cytokinesis in Drosophila." Title page, contents and abstract only, 2001. http://web4.library.adelaide.edu.au/theses/09PH/09pho415.pdf.

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Errata pasted onto back page. Bibliography: p. 133-149. The RhoGEF activity of PBL is shown to be acting predominantly by the activation of Rho1 and downstream signaling pathways required for contractile ring function during cytokinesis. Genetic evidence suggests this could be through the activation of Diaphanous (an FH protein) to reorganize the actin cytoskeleton, as well as through the activation of Rho-kinase which results in the phosphorylation, and activation of myosin. Highlights a possible role for PBL during contractile ring function at a later stage that previously thought. Genetic

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Prior, Leanne Michelle. "Characterization of pebble : a gene required for cytokinesis in Drosophila melanogaster /." Title page, contents and abstract only, 1998. http://web4.library.adelaide.edu.au/theses/09PH/09php9578.pdf.

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3

Mok, Ka-pun Chris, and 莫家斌. "Avian influenza A viral genetic determinants of cytokine hyper-induction in primary human macrophages." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B43941539.

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Huang, Fung-yu. "Pathogenetic aspects of helicobacter pylori infection in gastric cancer : a study on the role of inflammatory cytokine and gene methylation /." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B4370363X.

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Guo, Hong, and 郭紅. "Effects of anti-DNA antibodies on pleural mesothelial cells: in vitro studies to explore thepathogenetic mechanism of pulmonary lupus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B26631945.

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The Best M.Phil Thesis in the Faculties of Dentistry, Engineering, Medicine and Science (University of Hong Kong), Li Ka Shing Prize, 2001-2003.<br>published_or_final_version<br>abstract<br>toc<br>Medicine<br>Master<br>Master of Philosophy

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6

Yim, Chi-ho Howard, and 嚴志濠. "Cytokine dysregulation by human immunodeficiency virus-1 transactivating protein." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2006. http://hub.hku.hk/bib/B36987700.

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7

Silva, Gustavo Milson Fabricio da. "Polimorfismo genético de citocinas na população do Rio de Janeiro." Universidade do Estado do Rio de Janeiro, 2009. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=10210.

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Citocinas são moléculas que controlam e modulam a atividade de numerosas células por se ligarem a seus receptores específicos. As diferenças observadas na produção de citocinas entre indivíduos podem ser, pelo menos em parte, explicadas pelos polimorfismos genéticos como o polimorfismo de um único nucleotídeo (SNP). Em 181 indivíduos saudáveis não-aparentados da cidade do Rio de Janeiro (região Sudeste - Brasil), nós analisamos os polimorfismos de citocinas em genes que codificam para Fator de Necrose Tumoral-alfa (TNF-a), Fator de Crescimento Transformante-beta (TGF-b), Interleucina-10, Inter

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8

Wang, Cathy Ting-Peng. "Molecular dissection of RANKL signaling pathways in osteoclasts." University of Western Australia. School of Surgery and Pathology, 2007. http://theses.library.uwa.edu.au/adt-WU2008.0037.

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[Truncated abstract] Bone remodeling is intricately regulated by osteoclast-mediated bone resorption and osteoblast-mediated bone formation. The elevation in osteoclast number and/or activity is a major hallmark of several common pathological bone disorders including post-menopausal osteoporosis, osteoarthritis, Paget's disease, and tumour-mediated osteolysis. Receptor activator of nuclear factor kappa B ligand (RANKL) is a key cytokine for osteoclast differentiation and activation. The association of RANKL to its cognate receptor, RANK, which is expressed on osteoclast precursors and mature o

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9

Huang, Fung-yu, and 黃鳳如. "Pathogenetic aspects of helicobacter pylori infection in gastric cancer: a study on the role of inflammatorycytokine and gene methylation." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B4370363X.

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10

Cheung, Ka-wa Benny, and 張嘉華. "Mechanism of Bacillus Calmette Guerin-induced immune response." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2003. http://hub.hku.hk/bib/B29488989.

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11

Oliveira, Juliana Garcia de [UNESP]. "Polimorfismos gênicos de citocinas e receptores envolvidos no processo inflamatório da carcinogênese gástrica e alterações nos níveis de expressão gênica." Universidade Estadual Paulista (UNESP), 2011. http://hdl.handle.net/11449/102751.

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Made available in DSpace on 2014-06-11T19:32:15Z (GMT). No. of bitstreams: 0 Previous issue date: 2011-02-25Bitstream added on 2014-06-13T18:43:21Z : No. of bitstreams: 1 oliveira_jg_dr_sjrp.pdf: 1922822 bytes, checksum: 9be7ec21e759c3921cff2ec05792fab4 (MD5)<br>O câncer gástrico é uma doença caracterizada como multifatorial associada a fatores ambientais e genéticos. A carcinogênese do estômago pode progredir de uma inflamação crônica da mucosa gástrica, resultante da infecção pela bactéria Helicobacter pylori que ativa a resposta inflamatória do hospedeiro. Portanto, selecionamos um grupo

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12

Oliveira, Juliana Garcia de. "Polimorfismos gênicos de citocinas e receptores envolvidos no processo inflamatório da carcinogênese gástrica e alterações nos níveis de expressão gênica /." São José do Rio Preto : [s.n.], 2011. http://hdl.handle.net/11449/102751.

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Orientador: Ana Elizabete Silva<br>Banca: Dertia Villalba Freire-Maia<br>Banca: Spencer Luiz Marques Payao<br>Banca: Débora Aparecida Pires de Campos Zuccari<br>Banca: Mariangela Torreglosa Ruiz<br>Resumo: O câncer gástrico é uma doença caracterizada como multifatorial associada a fatores ambientais e genéticos. A carcinogênese do estômago pode progredir de uma inflamação crônica da mucosa gástrica, resultante da infecção pela bactéria Helicobacter pylori que ativa a resposta inflamatória do hospedeiro. Portanto, selecionamos um grupo de polimorfismos presentes em genes de citocinas pró-inflam

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13

O'Keefe, Louise Veronica. "Genetic analysis of the role of pebble during cytokinesis in Drosophila / by Louise O'Keefe." 2001. http://hdl.handle.net/2440/21763.

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Errata pasted onto back page.<br>Bibliography: p. 133-149.<br>149 p., [29] leaves of plates : ill. (chiefly col.) ; 30 cm.<br>Title page, contents and abstract only. The complete thesis in print form is available from the University Library.<br>The RhoGEF activity of PBL is shown to be acting predominantly by the activation of Rho1 and downstream signaling pathways required for contractile ring function during cytokinesis. Genetic evidence suggests this could be through the activation of Diaphanous (an FH protein) to reorganize the actin cytoskeleton, as well as through the activation of Rho-k

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14

O'Keefe, Louise Veronica. "Genetic analysis of the role of pebble during cytokinesis in Drosophila / by Louise O'Keefe." Thesis, 2001. http://hdl.handle.net/2440/21763.

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Errata pasted onto back page.<br>Bibliography: p. 133-149.<br>149 p., [29] leaves of plates : ill. (chiefly col.) ; 30 cm.<br>The RhoGEF activity of PBL is shown to be acting predominantly by the activation of Rho1 and downstream signaling pathways required for contractile ring function during cytokinesis. Genetic evidence suggests this could be through the activation of Diaphanous (an FH protein) to reorganize the actin cytoskeleton, as well as through the activation of Rho-kinase which results in the phosphorylation, and activation of myosin. Highlights a possible role for PBL during contrac

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15

Prior, Leanne Michelle. "Characterization of pebble : a gene required for cytokinesis in Drosophila melanogaster / by Leanne Michelle Prior." Thesis, 1998. http://hdl.handle.net/2440/19317.

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Errata is pasted onto back end paper.<br>Includes bibliographical references (26 leaves).<br>115, [68] leaves, [8] leaves of plates : ill. (some col.) ; 30 cm.<br>This study entailed work towards the isolation of the pbl gene and preliminary characterisation of a candidate pbl transcript. Plasmid rescue of the genomic DNA flanking the inserted P element led to the isolation of a third candidate p61 cDNA, the 1A cDNA. This data suggests that the IA cDNA is encoded by the p61 gene.<br>Thesis (Ph.D.)--University of Adelaide, Dept. of Genetics, 1998

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16

Somers, Wayne Gregory. "Studies of the Drosophila Rho G protein regulators, pebble and RacGAP50C / by W.G. Somers." 2002. http://hdl.handle.net/2440/21906.

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"November 2002"<br>Bibliography: p. 177-194.<br>194 p. : ill., plates (some col.) ; 30 cm.<br>Title page, contents and abstract only. The complete thesis in print form is available from the University Library.<br>Thesis (Ph.D.)--University of Adelaide, Dept. of Molecular Biosciences, 2003

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17

Somers, Wayne Gregory. "Studies of the Drosophila Rho G protein regulators, pebble and RacGAP50C / by W.G. Somers." Thesis, 2002. http://hdl.handle.net/2440/21906.

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18

Reynolds, Brenton James. "Identification of genes involved in leukaemia and differentiation induced by activated mutants of the GM-CSF receptor β subunit". 2005. http://hdl.handle.net/2440/39170.

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Interleukin (IL)-3, IL-5 and granulocyte-macrophage-colony-stimulating factor (GM-CSF) are cytokines that affect the growth, survival and differentiation of many cells within the haematopoietic system. The functions of these factors are mediated by membrane bound receptor complexes that are composed of specific ligand binding subunits (α)and a common signal transducing subunit(hβc). Constitutively activated mutants of hβc have been previously identified that are able to confer factor-independent signalling in a number of haematopoietic cell lines (including FDC-P1 and FDB-1). These activated m

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19

Reynolds, Brenton James. "Identification of genes involved in leukaemia and differentiation induced by activated mutants of the GM-CSF receptor β subunit". Thesis, 2005. http://hdl.handle.net/2440/39170.

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Interleukin (IL)-3, IL-5 and granulocyte-macrophage-colony-stimulating factor (GM-CSF) are cytokines that affect the growth, survival and differentiation of many cells within the haematopoietic system. The functions of these factors are mediated by membrane bound receptor complexes that are composed of specific ligand binding subunits (α)and a common signal transducing subunit(hβc). Constitutively activated mutants of hβc have been previously identified that are able to confer factor-independent signalling in a number of haematopoietic cell lines (including FDC-P1 and FDB-1). These activated m

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20

"CXCL10 and its receptor CXCR3 promote non-alcoholic steatohepatitis through mediating inflammatory cytokines and autophagy." 2014. http://library.cuhk.edu.hk/record=b6115759.

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研究背景及實驗目的: 非酒精性脂肪性肝炎(NASH)使得肥胖和2 型糖尿病變得複雜，肝臟炎症的持續產生是其主要的發病機理。CXCL10 是一種促進炎症的細胞因數，其在肥胖和2 型糖尿病中的表達顯著升高。CXCL10 以及其受體CXCR3 是否在NASH 的發生發展中起作用尚不清楚。在本研究中，我們探索了CXCL10 以及其受體CXCR3 在脂肪性肝炎中的功能，並評估了CXCL10 在NASH 中的臨床價值。<br>實驗方法：CXCL10 基因敲除鼠，CXCR3 敲除鼠以及野生型C57BL/6 小鼠給予蛋氨酸膽鹼缺乏食(MCD)4 周或者8 周。CXCL10 的信號通路以及下游靶點通過細胞因數分析，cDNA array, 蛋白DNA 結合實驗，自噬溶酶體系統分析進行檢測。為了闡明CXCL10 抑制對NASH 的預防治療作用，我們給MCD 餵養的小鼠注射抗CXCL10 抗體。用不同濃度的CXCL10 抗體以及CXCR3 抑制劑NIBR2130 幹預MCD 培養的肝細胞株AML-12。臨床研究中，我們收集了147個非酒精性脂肪肝患者以及73 個健康對照的血清，用酶聯免疫吸附試驗檢測血清中CXCL10 的水準。<br>結果：野生型小鼠給予MCD 餵養後，CXCL10 以及CXCR3 的表達升高，並出現脂肪性肝炎的表現。然而，MCD 飼養的CXCL10 以及CXCR3 基因敲除鼠中，脂肪性

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21

Staser, Karl W. "Erk1 and Erk2 in hematopoiesis, mast cell function, and the management of Nf1-associated leukemia and tumors." Thesis, 2012. http://hdl.handle.net/1805/2892.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Neurofibromatosis type 1 is a genetic disease that results from either heritable or spontaneous autosomal dominant mutations in the NF1 gene, which encodes a protein serving, at least in part, to accelerate the intrinsic hydrolysis of active Ras-GTP to inactive Ras-GDP. A second-hit NF1 mutation precedes predominant NF1 neoplasms, including juvenile myelomoncytic leukemia (JMML) and plexiform neurofibroma formation, potentially fatal conditions with no medical therapy. While NF1 loss of heterozygosity (LOH) in myeloid progenitor cell

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22

Schoof, Nils. "Onkogenomische Aspekte Zytokin-assoziierter Signaltransduktion." Doctoral thesis, 2008. http://hdl.handle.net/11858/00-1735-0000-0006-AD29-1.

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23

Pham, Duy. "Twist1 and Etv5 are part of a transcription factor network defining T helper cell identity." Thesis, 2014. http://hdl.handle.net/1805/4657.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>CD4 T helper cells control immunity to pathogens and the development of inflammatory disease by acquiring the ability to secrete effector cytokines. Cytokine responsiveness is a critical component of the ability of cells to respond to the extracellular milieu by activating Signal Transducer and Activator of Transcription factors that induce the expression of other transcription factors important for cytokine production. STAT4 is a critical regulator of Th1 differentiation and inflammatory disease that attenuates the gene-repressing a

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24

Akhand, Saeed Salehin. "Role of a putative bacterial lipoprotein in Pseudomonas aeruginosa-mediated cytotoxicity toward airway cells." Thesis, 2014. http://hdl.handle.net/1805/5629.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>The patients with Cystic fibrosis (CF), an inherent genetic disorder, suffer from chronic bacterial infection in the lung. In CF, modification of epithelial cells leads to alteration of the lung environment, such as inhibition of ciliary bacterial clearance and accumulation of thickened mucus in the airways. Exploiting these conditions, opportunistic pathogens like Pseudomonas aeruginosa cause lifelong persistent infection in the CF lung by forming into antibiotic-resistant aggregated communities called biofilms. Airway infecti

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