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Artykuły w czasopismach na temat „DC blocker”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 1 lutego 2022

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1

Tittelbach-Helmrich, Klaus. "Digital DC blocker filters." Frequenz 75, no. 9-10 (February 18, 2021): 331–39. http://dx.doi.org/10.1515/freq-2020-0177.

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Abstract This paper mathematically investigates a special kind of digital infinite-impulse response (IIR) filters, suitable for filtering out very low frequencies near zero from digital signals. We investigate the transfer functions of such filters from 1st to 3rd order and provide formulas to calculate the filter coefficients from the desired cutoff frequency.

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Yates, R., and R. Lyons. "DC Blocker Algorithms[DSP Tips & Tricks]." IEEE Signal Processing Magazine 25, no. 2 (March 2008): 132–34. http://dx.doi.org/10.1109/msp.2007.914713.

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3

Nanda Kumar, N. S., Satish K. Singh, and Vazhaikkurichi M. Rajendran. "Mucosal potassium efflux mediated via Kcnn4 channels provides the driving force for electrogenic anion secretion in colon." American Journal of Physiology-Gastrointestinal and Liver Physiology 299, no. 3 (September 2010): G707—G714. http://dx.doi.org/10.1152/ajpgi.00101.2010.

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Intermediate conductance K+ (Kcnn4) channels are present in both mucosal and serosal membranes of colon. However, only serosal Kcnn4 channels have been shown to be essential for agonist-induced (cAMP and Ca2+) anion secretion. The present study sought to determine whether mucosal Kcnn4 channels also play a role in colonic anion secretion. Mucosal-to-serosal and serosal-to-mucosal unidirectional 86Rb (K+ surrogate) fluxes as well as short-circuit current ( Isc; a measure of anion secretion) were measured under voltage-clamp conditions in distal colon from rats fed either a standard or K+-free d

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4

Nazir, Moazzam, Klaehn Burkes, and Johan H. Enslin. "Electrical Safety Considerations of Neutral Blocker Placements for Mitigating DC." IEEE Transactions on Industry Applications 57, no. 1 (January 2021): 1113–21. http://dx.doi.org/10.1109/tia.2020.3032081.

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5

Abdolhamidi, Mostafa, and Mahmoud Moahammad‐Taheri. "Theory and design of phase‐inverted balanced coupled‐line DC‐blocker." IET Microwaves, Antennas & Propagation 13, no. 8 (April 26, 2019): 1188–97. http://dx.doi.org/10.1049/iet-map.2018.5580.

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Wang, Lujun, Boyu Feng, Yu Wang, Tiezhou Wu, and Huipin Lin. "Bidirectional Short-Circuit Current Blocker for DC Microgrid Based on Solid-State Circuit Breaker." Electronics 9, no. 2 (February 10, 2020): 306. http://dx.doi.org/10.3390/electronics9020306.

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In order to solve the imminent problem in that the traditional protection strategy cannot meet time requirements, together with the fact that the rotational inertia of a DC microgrid is small and short-circuit fault develops rapidly, a bidirectional short-circuit current blocker (BSCCB) based on solid-state circuit breaker for a DC microgrid is proposed. Firstly, the bidirectional current blocking circuit structure is proposed based on the analysis of key components. Then, a top-level differential protection strategy is developed based on the aforementioned proposal. Finally, the performance o

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7

Hae-Young, Lee. "A1364 Angiotensin II type 1 receptor blocker, fimasartan, reduces vascular smooth muscle cell senescence by inhibiting CYR61 signaling pathway." Journal of Hypertension 36 (October 2018): e14. http://dx.doi.org/10.1097/01.hjh.0000548042.95520.dc.

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8

Yu, Xiaobao, Ying Song, Zhihua Wang, and Baoyong Chi. "Self‐tuned SAW‐less GNSS receiver front end with blocker filtering and gain‐irrelevant DC offset cancellation." Electronics Letters 51, no. 8 (April 2015): 653–54. http://dx.doi.org/10.1049/el.2015.0170.

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9

Richter, Frank, Oskar Mikulik, Andrea Ebersberger, and Hans-Georg Schaible. "Noradrenergic Agonists and Antagonists Influence Migration of Cortical Spreading Depression in Rat—a Possible Mechanism of Migraine Prophylaxis and Prevention of Postischemic Neuronal Damage." Journal of Cerebral Blood Flow & Metabolism 25, no. 9 (April 13, 2005): 1225–35. http://dx.doi.org/10.1038/sj.jcbfm.9600120.

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Cortical spreading depression (CSD) is thought to be a neuronal mechanism that expands the penumbra zone after focal brain ischemia and that causes migraine aura. Both adrenergic agonists and antagonists significantly influence the size of the penumbra zone and decline the frequency of migraine. To study whether these compounds act by influencing CSD, we applied different drugs topically to an area of the exposed cortex of anesthetized adult rats and observed the migration of CSD-related DC potential deflections across the treated area. The adrenergic agonist norepinephrine (1 mmol/L) and the

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10

Richter, Frank, Sven Rupprecht, Alfred Lehmenkühler, and Hans-Georg Schaible. "Spreading Depression Can Be Elicited in Brain Stem of Immature But Not Adult Rats." Journal of Neurophysiology 90, no. 4 (October 2003): 2163–70. http://dx.doi.org/10.1152/jn.00388.2003.

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Spreading depression (SD), a neuronal mechanism involved in brain pathophysiology, occurs in brain areas with high neuronal density such as the cerebral cortex. By contrast, the brain stem is thought to be resistant to SD. Here we show that DC shifts resembling cortical SD can be elicited in rat brain stem by topical application of KCl but not by pricking the brain stem. However, this was only possible until postnatal day 13, and, in addition, susceptibility for SD had to be enhanced. The latter was achieved by superfusion of the brain stem for 45 min with a solution containing acetate instead

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11

Miura, Masami, Masatomo Yoshioka, Hiroyoshi Miyakawa, Hiroshi Kato, and Ken-Ichi Ito. "Properties of Calcium Spikes Revealed During GABAA Receptor Antagonism in Hippocampal CA1 Neurons From Guinea Pigs." Journal of Neurophysiology 78, no. 5 (November 1, 1997): 2269–79. http://dx.doi.org/10.1152/jn.1997.78.5.2269.

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Miura, Masami, Masatomo Yoshioka, Hiroyoshi Miyakawa, Hiroshi Kato, and Ken-Ichi Ito. Properties of calcium spikes revealed during GABAA receptor antagonism in hippocampal CA1 neurons from guinea pigs. J. Neurophysiol. 78: 2269–2279, 1997. Intracellular electrical responses and changes in intracellular calcium concentration ([Ca2+]i) in response to activation of synaptic inputs and to DC injections were recorded simultaneously from CA1 pyramidal neurons ( n = 42) in guinea pig hippocampal slices. In the presence of the γ-aminobutyric acid-A (GABAA) receptor antagonists, bicuculline (25 μM) and

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12

Lee, Yongho, Shinil Chang, Jungah Kim, and Hyunchol Shin. "A CMOS RF Receiver with Improved Resilience to OFDM-Induced Second-Order Intermodulation Distortion for MedRadio Biomedical Devices and Sensors." Sensors 21, no. 16 (August 5, 2021): 5303. http://dx.doi.org/10.3390/s21165303.

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A MedRadio RF receiver integrated circuit for implanted and wearable biomedical devices must be resilient to the out-of-band (OOB) orthogonal frequency division modulation (OFDM) blocker. As the OFDM is widely adopted for various broadcasting and communication systems in the ultra-high frequency (UHF) band, the selectivity performance of the MedRadio RF receiver can severely deteriorate by the second-order intermodulation (IM2) distortion induced by the OOB OFDM blocker. An analytical investigation shows how the OFDM-induced IM2 distortion power can be translated to an equivalent two-tone-indu

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13

Zhang, Lu, Xing Ouyang, Xiaopeng Shao, and Jian Zhao. "Experimental demonstration of a real-time high-throughput digital DC blocker for compensating ADC imperfections in optical fast-OFDM receivers." Optics Express 24, no. 13 (June 15, 2016): 14215. http://dx.doi.org/10.1364/oe.24.014215.

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14

Gallemore, R. P., and R. H. Steinberg. "Light-evoked modulation of basolateral membrane Cl- conductance in chick retinal pigment epithelium: the light peak and fast oscillation." Journal of Neurophysiology 70, no. 4 (October 1, 1993): 1669–80. http://dx.doi.org/10.1152/jn.1993.70.4.1669.

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1. We studied the ionic mechanism of the light-peak voltage of the DC electroretinogram (DC ERG) in an in vitro preparation of chick neural retina-retinal pigment epithelium (RPE)-choroid. The light peak originates from a depolarization of the RPE basolateral (basal) membrane, associated with an increase in its conductance. Using conventional and Cl(-)-selective microelectrodes, we tested the hypothesis that the light-peak voltage is generated by an increase in Cl- conductance (gCl) of the basolateral (basal) membrane. 2. Perfusion of the RPE basal membrane with 4,4'-diisothiocyanostilbene-2,2

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15

Tapbergenov, S. O., B. S. Sovetov, A. T. Tapbergenov, and Elina Hahn. "Metabolic effects of combined introduction of adrenalin and blocker of methanoprolol beta-adrenophyleters." Biomeditsinskaya Khimiya 63, no. 2 (2017): 154–58. http://dx.doi.org/10.18097/pbmc20176302154.

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The effect of combined administration of adrenaline (0.4 mg/kg, i.p.) and b1-blocker metoprolol (25 mg/kg) on the activity of glutathione peroxidase (GPO), glutathione reductase (GR), catalase, adenosine deaminase (AD), AMP deaminase (AMPD), 5¢-nucleotidase (5¢N), on the level ofmalonic dialdehyde (MDA) and conjugated dienes (CD) was investigated. In blood adrenaline administration to animals caused an increase in the activity of AMPD, AD, 5¢N and GPO, and the increase the level of CD in the blood increases. Metoprolol caused a more pronounced increase in the activity of blood AMPD, AD, 5'N an

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16

Hayashi, Mikio, Jing Wang, Susanne E. Hede, and Ivana Novak. "An intermediate-conductance Ca2+-activated K+ channel is important for secretion in pancreatic duct cells." American Journal of Physiology-Cell Physiology 303, no. 2 (July 15, 2012): C151—C159. http://dx.doi.org/10.1152/ajpcell.00089.2012.

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Potassium channels play a vital role in maintaining the membrane potential and the driving force for anion secretion in epithelia. In pancreatic ducts, which secrete bicarbonate-rich fluid, the identity of K+ channels has not been extensively investigated. In this study, we investigated the molecular basis of functional K+ channels in rodent and human pancreatic ducts (Capan-1, PANC-1, and CFPAC-1) using molecular and electrophysiological techniques. RT-PCR analysis revealed mRNAs for KCNQ1, KCNH2, KCNH5, KCNT1, and KCNT2, as well as KCNN4 coding for the following channels: KVLQT1; HERG; EAG2;

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17

De Greve, Jacques, Teresa Moran, Marie-Pascale Graas, Daniella Galdermans, Peter Vuylsteke, Jean-Luc Canon, Vikram K. Chand, Yali Fu, Dan Massey, and Johan Vansteenkiste. "Phase II study of afatinib, an irreversible ErbB family blocker, in demographically and genotypically defined non-small cell lung cancer (NSCLC) patients." Journal of Clinical Oncology 31, no. 15_suppl (May 20, 2013): 8063. http://dx.doi.org/10.1200/jco.2013.31.15_suppl.8063.

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8063 Background: EGFR mutation+ (M+) NSCLC pts have an improved response to EGFR TKIs vs non-M+ pts. Data on EGFR FISH+ (gene amplified) and HER2 M+ pts are, however, limited. Afatinib is an irreversible ErbB Family Blocker with efficacy in Ph II/III trials in EGFR M+ NSCLC. This exploratory, open-label trial assessed afatinib in 3 genotypically and demographically defined NSCLC groups. Methods: Never/ex-smokers with stage IIIB/IV lung adenocarcinoma with EGFR M+ tumors who had failed prior EGFR TKIs, HER2 M+ tumors independent of prior therapy, or EGFR FISH+ tumors who received ≤3 prior chemo

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18

Rossi, Jean-Francois, Anne-Marie Conge, Catherine Barjot, Mohamed H. Zaki, Marian T. Nakada, Robert Corringham, and Bernard Klein. "Interterleukin-6 (IL-6) Produced by Monocyte Activation Reduces Dendritic Cell (DC) Differentiation in Patients with Multiple Myeloma (MM) and Malignant Lymphoma (ML): Role of CNTO 328, an Anti-IL-6 Monoclonal Antibody (Mab) and Possible Clinical Applications." Blood 104, no. 11 (November 16, 2004): 2662. http://dx.doi.org/10.1182/blood.v104.11.2662.2662.

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Abstract We developed a serum-free process in a closed system using culture cassettes and bags for large-scale and clinical-grade DC vaccination, accepted by the “Afssaps-French drug Agency” (Tarte K. et al. Leukemia2000; 14:2152 & patent). Intermediate mature DCs are generated from mononucleated cells obtained by mobilized leukapheresis, followed by Mo selection using adherence in specific cassettes (CLINIcell, Mabiol). Non-adherent cells are removed and Mo are cultured for 5 days (D) in X-VIVO15 medium (Cambrex) with 2% of human albumin, 100ng/ml of GM-CSF (Leukine, Berlex) and 25 ng/ml

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19

RICHEZ, CHRISTOPHE, THOMAS BARNETCHE, LILIANE KHORYATI, PIERRE DUFFAU, MARIE KOSTINE, CÉCILE CONTIN-BORDES, PATRICK BLANCO, and THIERRY SCHAEVERBEKE. "Tocilizumab Treatment Decreases Circulating Myeloid Dendritic Cells and Monocytes, 2 Components of the Myeloid Lineage." Journal of Rheumatology 39, no. 6 (April 1, 2012): 1192–97. http://dx.doi.org/10.3899/jrheum.111439.

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Objective.Interleukin 6 (IL-6) and tumor necrosis factor-α (TNF-α) are proinflammatory cytokines involved in inflammatory response. Effective TNF-α blocker treatment is associated with an increase in circulating myeloid dendritic cells (mDC), suggesting their release from inflamed synovium. Currently, in vivo effects of IL-6 inhibition on DC are unknown. We monitored the changes in circulating mDC and plasmacytoid DC (pDC) during tocilizumab (TCZ) therapy in patients with rheumatoid arthritis (RA).Methods.DC subset levels were evaluated by flow cytometry in patients with RA (n = 43) and in hea

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20

Heise, Nicole, Ekaterina Shumilina, Meerim K. Nurbaeva, Evi Schmid, Kalina Szteyn, Wenting Yang, Nguyen Thi Xuan, et al. "Effect of dexamethasone on Na+/Ca2+exchanger in dendritic cells." American Journal of Physiology-Cell Physiology 300, no. 6 (June 2011): C1306—C1313. http://dx.doi.org/10.1152/ajpcell.00396.2010.

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Ca+-dependent signaling regulates the function of dendritic cells (DCs), antigen-presenting cells linking innate and adaptive immunity. The activity of DCs is suppressed by glucocorticoids, potent immunosuppressive hormones. The present study explored whether the glucocorticoid dexamethasone influences the cytosolic Ca2+concentration ([Ca2+]i) in DCs. To this end, DCs were isolated from mouse bone marrow. According to fura-2 fluorescence, exposure of DCs to lipopolysaccharide (LPS, 100 ng/ml) increased [Ca2+]i, an effect significantly blunted by overnight incubation with 10 nM dexamethasone be

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21

Valverde, P., T. Kawai, and M. A. Taubman. "Potassium Channel-blockers as Therapeutic Agents to Interfere with Bone Resorption of Periodontal Disease." Journal of Dental Research 84, no. 6 (June 2005): 488–99. http://dx.doi.org/10.1177/154405910508400603.

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Inflammatory lesions of periodontal disease contain all the cellular components, including abundant activated/memory T- and B-cells, necessary to control immunological interactive networks and to accelerate bone resorption by RANKL-dependent and -independent mechanisms. Blockade of RANKL function has been shown to ameliorate periodontal bone resorption and other osteopenic disorders without affecting inflammation. Development of therapies aimed at decreasing the expression of RANKL and pro-inflammatory cytokines by T-cells constitutes a promising strategy to ameliorate not only bone resorption

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22

Islam, S. M. Shamsul, Hae-Ok Byun, Bunsoon Choi, and Seonghyang Sohn. "Inhibition of CD83 Alleviates Systemic Inflammation in Herpes Simplex Virus Type 1-Induced Behçet’s Disease Model Mouse." Mediators of Inflammation 2019 (September 9, 2019): 1–15. http://dx.doi.org/10.1155/2019/5761392.

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Behçet’s disease (BD) is an autoinflammatory disease that can lead to life- and sight-threating complications. Dendritic cells (DCs) are the most potent antigen-presenting cells that can regulate multiple inflammatory pathways. The objective of this study was to investigate the association of the DC stimulatory molecule CD83 with BD. Frequencies of costimulatory molecules expressing DCs in peripheral blood leukocytes (PBL) were measured by flow cytometry (FACS). The severity of symptoms in HSV-1-induced BD symptomatic mice was also assessed. Frequencies of CD83-positive cells were significantl

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23

Ferrari, Davide, Paola Chiozzi, Simonetta Falzoni, Stefania Hanau та Francesco Di Virgilio. "Purinergic Modulation of Interleukin-1β Release from Microglial Cells Stimulated with Bacterial Endotoxin". Journal of Experimental Medicine 185, № 3 (3 лютого 1997): 579–82. http://dx.doi.org/10.1084/jem.185.3.579.

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Microglial cells express a peculiar plasma membrane receptor for extracellular ATP, named P2Z/P2X7 purinergic receptor, that triggers massive transmembrane ion fluxes and a reversible permeabilization of the plasma membrane to hydrophylic molecules of up to 900 dalton molecule weight and eventual cell death (Di Virgilio, F. 1995. Immunol. Today. 16:524–528). The physiological role of this newly cloned (Surprenant, A., F. Rassendren, E. Kawashima, R.A. North and G. Buell. 1996. Science (Wash. DC). 272:735–737) cytolytic receptor is unknown. In vitro and in vivo activation of the macrophage and

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24

Newman, William J., Glen L. Xiong, and Amy V. Barnhorst. "Beta-Blockers." Psychopharm Review 48, no. 10 (October 2013): 73–80. http://dx.doi.org/10.1097/01.psyphr.0000436763.15959.dc.

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25

Salama, G., R. Lombardi, and J. Elson. "Maps of optical action potentials and NADH fluorescence in intact working hearts." American Journal of Physiology-Heart and Circulatory Physiology 252, no. 2 (February 1, 1987): H384—H394. http://dx.doi.org/10.1152/ajpheart.1987.252.2.h384.

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Voltage-sensitive dyes were used to stain intact perfused hearts and to simultaneously measure optical action potentials (APs) from 124 sites on the epicardium. Patterns of electrical depolarization (activation) and repolarization (recovery) along the surface of the heart were determined from the upstrokes and repolarization phases of optical APs. Standard surface extracellular techniques can detect electrical activation but not the recovery or the duration of APs. The optical recordings were previously shown to be equivalent to intracellular electrode measurements (Salama and Morad, Science W

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26

Bisenic, Vesna, Sasa Hinic, Mirjana Krotin, Branislav Milovanovic, Jelena Saric, and Goran Milasinovic. "Brugada syndrome: Case report." Srpski arhiv za celokupno lekarstvo 140, no. 1-2 (2012): 84–90. http://dx.doi.org/10.2298/sarh1202084b.

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Introduction. Brugada syndrome is an arrhythmogenic disease characterized by coved ST segment elevation and J point elevation of at least 2 mm in at least two of the right precordial ECG leads (V1-3) and ventricular arrhythmias, syncope, and sudden death. Risk stratifications of patients with Brugada electrocardiogram are being strongly debated. Case Outline. A 23-year-old man was admitted to the Coronary Care Unit of the Clinical Centre ?Bezanijska kosa? due to weakness, fatigue and chest discomfort. The patient suffered from fainting and palpitations. There was a family history of paternal s

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27

Nair, Jayakumar R., Louise M. Carlson, Noreen Ersing, Asher Alban Chanan-Khan, and Kelvin P. Lee. "Survival Signals, Growth Cytokines, Immunosuppressive Factors and Their Cellular Perpetrators in the Myeloma Tumor Microenvironment." Blood 112, no. 11 (November 16, 2008): 1664. http://dx.doi.org/10.1182/blood.v112.11.1664.1664.

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Abstract Multiple myeloma (MM) is an incurable neoplasia of terminally differentiated plasma cells in the bone marrow. Essential interactions of MM cells with host bone marrow stromal cells (BMSC) induce growth factors essential for MM progression and pathogenesis, as well as induce an immunosuppressive environment that inhibits endogenous and therapeutically-induced immune responses against the MM cells. However, despite their importance, little is known about the identity of these BMSC cells or the molecular basis of their interaction with myeloma cells. A potential MM surface protein that c

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28

Zhang, Si Yi, Donald Robertson, Graeme Yates, and Alan Everett. "Role of L-Type Ca2+ Channels in Transmitter Release From Mammalian Inner Hair Cells I. Gross Sound-Evoked Potentials." Journal of Neurophysiology 82, no. 6 (December 1, 1999): 3307–15. http://dx.doi.org/10.1152/jn.1999.82.6.3307.

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Intracochlear perfusion and gross potential recording of sound-evoked neural and hair cell responses were used to study the site of action of the L-type Ca2+ channel blocker nimodipine in the guinea pig inner ear. In agreement with previous work nimodipine (1–10 μM) caused changes in both the compound auditory nerve action potential (CAP) and the DC component of the hair cell receptor potential (summating potential, or SP) in normal cochleae. For 20-kHz stimulation, the effect of nimodipine on the CAP threshold was markedly greater than the effect on the threshold of the negative SP. This latt

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29

Ahn, Hee-Ran, and Bum-Man Kim. "Perfectly-Matched DC Blocks Terminated in Arbitrary Impedances." Journal of Korean Institute of Electromagnetic Engineering and Science 18, no. 8 (August 31, 2007): 895–903. http://dx.doi.org/10.5515/kjkiees.2007.18.8.895.

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Shumilina, Ekaterina, Meerim K. Nurbaeva, Wenting Yang, Evi Schmid, Kalina Szteyn, Antonella Russo, Nicole Heise, et al. "Altered regulation of cytosolic Ca2+ concentration in dendritic cells from klotho hypomorphic mice." American Journal of Physiology-Cell Physiology 305, no. 1 (July 1, 2013): C70—C77. http://dx.doi.org/10.1152/ajpcell.00355.2012.

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The function of dendritic cells (DCs), antigen-presenting cells regulating naïve T-cells, is regulated by cytosolic Ca2+ concentration ([Ca2+]i). [Ca2+]i is increased by store-operated Ca2+ entry and decreased by K+-independent (NCX) and K+-dependent (NCKX) Na+/Ca2+ exchangers. NCKX exchangers are stimulated by immunosuppressive 1,25-dihydroxyvitamin D3 [1,25(OH)2D3], the biologically active form of vitamin D. Formation of 1,25(OH)2D3 is inhibited by the antiaging protein Klotho. Thus 1,25(OH)2D3 plasma levels are excessive in Klotho-deficient mice ( klotho hm). The present study explored whet

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Baribaud, Frédéric, Stefan Pöhlmann, George Leslie, Frank Mortari, and Robert W. Doms. "Quantitative Expression and Virus Transmission Analysis of DC-SIGN on Monocyte-Derived Dendritic Cells." Journal of Virology 76, no. 18 (September 15, 2002): 9135–42. http://dx.doi.org/10.1128/jvi.76.18.9135-9142.2002.

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ABSTRACT The C-type lectins DC-SIGN and DC-SIGNR efficiently bind human immunodeficiency virus (HIV) and simian immunodeficiency virus (SIV) strains and can transmit bound virus to adjacent CD4-positive cells. DC-SIGN also binds efficiently to the Ebola virus glycoprotein, enhancing Ebola virus infection. DC-SIGN is thought to be responsible for the ability of dendritic cells (DCs) to capture HIV and transmit it to T cells, thus promoting HIV dissemination in vitro and perhaps in vivo as well. To investigate DC-SIGN function and expression levels on DCs, we characterized a panel of monoclonal

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Uehori, Junji, Misako Matsumoto, Shoutaro Tsuji, Takashi Akazawa, Osamu Takeuchi, Shizuo Akira, Tsutomu Kawata, Ichiro Azuma, Kumao Toyoshima, and Tsukasa Seya. "Simultaneous Blocking of Human Toll-Like Receptors 2 and 4 Suppresses Myeloid Dendritic Cell Activation Induced by Mycobacterium bovis Bacillus Calmette-Guérin Peptidoglycan." Infection and Immunity 71, no. 8 (August 2003): 4238–49. http://dx.doi.org/10.1128/iai.71.8.4238-4249.2003.

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ABSTRACT The Mycobacterium bovis bacillus Calmette-Guérin (BCG) cell wall skeleton (CWS) consists of mycolic acids, arabinogalactan, and peptidoglycan (PGN) and activates Toll-like receptor 2 (TLR2) and TLR4. Here we investigated the ability of the essential portion of highly purified BCG CWS to support the TLR agonist function by using the following criteria: myeloid dendritic cell (DC) maturation, i.e., tumor necrosis factor alpha (TNF-α) production and CD83/CD86 up-regulation. The purified PGN region was sufficient to activate TLR2 and TLR4 in mouse DCs and macrophages; in TLR2 and TLR4 do

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Largo, Carlota, Geoffrey C. Tombaugh, Peter G. Aitken, Oscar Herreras, and George G. Somjen. "Heptanol But Not Fluoroacetate Prevents the Propagation of Spreading Depression in Rat Hippocampal Slices." Journal of Neurophysiology 77, no. 1 (January 1, 1997): 9–16. http://dx.doi.org/10.1152/jn.1997.77.1.9.

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Largo, Carlota, Geoffrey C. Tombaugh, Peter G. Aitken, Oscar Herreras, and George G. Somjen. Heptanol but not fluoroacetate prevents the propagation of spreading depression in rat hippocampal slices. J. Neurophysiol. 77: 9–16, 1997. We investigated whether heptanol and other long-chain alcohols that are known to block gap junctions interfere with the generation or the propagation of spreading depression (SD). Waves of SD were triggered by micro-injection of concentrated KCl solution in stratum (s.) radiatum of CA1 of rat hippocampal tissue slices. DC-coupled recordings of extracellular potenti

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Liu, Jifeng, Xiaoning Zhong, Zhiyi He, Jianquan Zhang, Jing Bai, Guangnan Liu, Yi Liang, Leilei Ya, and Xianglin Qin. "Erythromycin Suppresses the Cigarette Smoke Extract-Exposed Dendritic Cell-Mediated Polarization of CD4+ T Cells into Th17 Cells." Journal of Immunology Research 2020 (January 21, 2020): 1–9. http://dx.doi.org/10.1155/2020/1387952.

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Cigarette smoke is a major effector of chronic obstructive pulmonary disease (COPD), and Th17 cells and dendritic cells (DCs) involve in the pathogenesis of COPD. Previous studies have demonstrated the anti-inflammatory effects of macrolides. However, the effects of macrolides on the cigarette smoke extract- (CSE-) induced immune response are unclear. Accordingly, in this study, we evaluated the effects of erythromycin (EM) on CSE-exposed DCs polarizing naïve CD4+ T cells into Th17 cells. DCs were generated from bone marrow-derived mononuclear cells isolated from male BALB/c mice and divided i

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Green, J., and R. Jotte. "Interactions between T helper cells and dendritic cells during the rat mixed lymphocyte reaction." Journal of Experimental Medicine 162, no. 5 (November 1, 1985): 1546–60. http://dx.doi.org/10.1084/jem.162.5.1546.

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This paper describes the interactions between dendritic cells (DC) and T helper (Th) cells in the rat mixed lymphocyte reaction (MLR). Th blasts that are actively proliferating were generated in a 5 d primary MLR; resting Th memory cells were derived from a 10-12 d MLR. The DC were purified from thoracic duct lymph derived from rats whose mesenteric lymph nodes had been removed. The results show that DC are the major stimulators in the primary MLR and also for the restimulation of Th blasts and Th memory cells. Th blasts rapidly formed large clusters when cultured with DC but not with Ia+ macr

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Galy, Anne, Indu Christopherson, Guido Ferlazzo, Guo Liu, Hergen Spits, and Katia Georgopoulos. "Distinct signals control the hematopoiesis of lymphoid-related dendritic cells." Blood 95, no. 1 (January 1, 2000): 128–37. http://dx.doi.org/10.1182/blood.v95.1.128.

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Abstract The molecular and cellular requirements for the development of different populations of human dendritic cells (DC) were studied. Conditions were defined that support DC production from lymphoid progenitors but that fail to induce DC formation from peripheral monocytes. The production of these lymphoid-related DC was severely blocked when hematopoietic progenitors overexpressed Ik7, a mutant dominant-negative Ikaros protein. In contrast, Ik7 did not block the formation of DC in conditions supporting the development of monocyte-derived DC. Furthermore, Ik7 did not block the formation of

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Galy, Anne, Indu Christopherson, Guido Ferlazzo, Guo Liu, Hergen Spits, and Katia Georgopoulos. "Distinct signals control the hematopoiesis of lymphoid-related dendritic cells." Blood 95, no. 1 (January 1, 2000): 128–37. http://dx.doi.org/10.1182/blood.v95.1.128.001k06_128_137.

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The molecular and cellular requirements for the development of different populations of human dendritic cells (DC) were studied. Conditions were defined that support DC production from lymphoid progenitors but that fail to induce DC formation from peripheral monocytes. The production of these lymphoid-related DC was severely blocked when hematopoietic progenitors overexpressed Ik7, a mutant dominant-negative Ikaros protein. In contrast, Ik7 did not block the formation of DC in conditions supporting the development of monocyte-derived DC. Furthermore, Ik7 did not block the formation of monocyte

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Gorji, A., D. Scheller, F. Tegtmeier, R. Köhling, H. Straub, and E.-J. Speckmann. "NiCl2 and Amiloride Induce Spreading Depression in Guinea Pig Hippocampal Slices." Cephalalgia 20, no. 8 (October 2000): 740–47. http://dx.doi.org/10.1111/j.1468-2982.2000.00124.x.

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Spreading depressions (SD) occur in association with ischaemia, epilepsy and migraine. Intracellular calcium oscillations have been suggested to be involved in the generation and propagation of SD. The present study was performed to study the mechanism of conditioning guinea pig hippocampal slices by the T-type calcium channel blockers NiCl2 and amiloride. SD-like fluctuations of DC potential were recorded by inserting microelectrodes into the CA1 and CA3 regions. The SD occurrence was significantly greater with 10 µmol/ l NiCl2 as well as with 25 and 50 µmol/ l amiloride than with other conce

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Tassaneetrithep, Boonrat, Timothy H. Burgess, Angela Granelli-Piperno, Christine Trumpfheller, Jennifer Finke, Wellington Sun, Michael A. Eller, et al. "DC-SIGN (CD209) Mediates Dengue Virus Infection of Human Dendritic Cells." Journal of Experimental Medicine 197, no. 7 (April 7, 2003): 823–29. http://dx.doi.org/10.1084/jem.20021840.

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Dengue virus is a single-stranded, enveloped RNA virus that productively infects human dendritic cells (DCs) primarily at the immature stage of their differentiation. We now find that all four serotypes of dengue use DC-SIGN (CD209), a C-type lectin, to infect dendritic cells. THP-1 cells become susceptible to dengue infection after transfection of DC-specific ICAM-3 grabbing nonintegrin (DC-SIGN), or its homologue L-SIGN, whereas the infection of dendritic cells is blocked by anti–DC-SIGN antibodies and not by antibodies to other molecules on these cells. Viruses produced by dendritic cells a

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Rasgado-Flores, Hector, Vamsi Krishna Mandava, Homayoun Siman, Willy Van Driessche, Joseph M. Pilewski, Scott H. Randell, and Robert J. Bridges. "Effect of apical hyperosmotic sodium challenge and amiloride on sodium transport in human bronchial epithelial cells from cystic fibrosis donors." American Journal of Physiology-Cell Physiology 305, no. 11 (December 1, 2013): C1114—C1122. http://dx.doi.org/10.1152/ajpcell.00166.2013.

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Hypertonic saline (HS) inhalation therapy benefits cystic fibrosis (CF) patients [Donaldson SH, Bennet WD, Zeman KL, Knowles MR, Tarran R, Boucher RC. N Engl J Med 354: 241–250, 2006; Elkins MR, Robinson M, Rose BR, Harbour C, Moriarty CP, Marks GB, Belousova EG, Xuan W, Bye PT; the National Hypertonic Saline in Cystic Fibrosis (NHSCF) Study Group. N Engl J Med 354: 229–240, 2006]. Surprisingly, these benefits are long-lasting and are diminished by the epithelial Na+ channel blocker amiloride (Donaldson SH, Bennet WD, Zeman KL, Knowles MR, Tarran R, Boucher RC. N Engl J Med 354: 241–250, 2006)

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Smit, Egbert F., Solange Peters, Rafal Dziadziuszko, Urania Dafni, Juergen Wolf, Bartosz Wasąg, Wojciech Biernat, et al. "A single-arm phase II trial of afatinib in pretreated patients with advanced NSCLC harboring a HER2 mutation: The ETOP NICHE trial." Journal of Clinical Oncology 35, no. 15_suppl (May 20, 2017): 9070. http://dx.doi.org/10.1200/jco.2017.35.15_suppl.9070.

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9070 Background: HER2mutations are identified in about 2% of lung adenocarcinomas and are critical for lung carcinogenesis. Afatinib is a selective and irreversible erbB family blocker with a manageable toxicity profile and promising results in small retrospective studies targeting HER2 in NSCLC. Methods: NICHE is a single-arm phase II trial exploring the potential of afatinib to control disease (complete or partial response or disease stabilization for ≥12 weeks) in pre-treated patients with advanced NSCLC harboring HER2 exon 20 mutations. Patients were treated with afatinib 40 mg/day p.o. un

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Ayehunie, Seyoum, Eduardo A. Garcia-Zepeda, James A. Hoxie, Richard Horuk, Thomas S. Kupper, Andrew D. Luster, and Ruth M. Ruprecht. "Human Immunodeficiency Virus-1 Entry Into Purified Blood Dendritic Cells Through CC and CXC Chemokine Coreceptors." Blood 90, no. 4 (August 15, 1997): 1379–86. http://dx.doi.org/10.1182/blood.v90.4.1379.

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Abstract Blood dendritic cells (DC) are susceptible to both macrophage (M) and T-cell line (T) tropic human immunodeficiency virus type 1. The CC chemokines RANTES, macrophage inflammatory protein-1α (MIP-1α), MIP-1β, eotaxin, and, to a lesser extent, monocyte chemoattractant protein-1 (MCP-1) and MCP-4 blocked entry of M-tropic virus into blood DC. The CXC chemokine, SDF-1, a fusin (CXCR4 chemokine receptor) ligand, and an antifusin antibody inhibited DC entry by T-tropic virus. Purified blood DC contained CCR1, CCR2, CCR3, and CCR5 as well as the CXCR4 chemokine receptor RNA transcripts and

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Ayehunie, Seyoum, Eduardo A. Garcia-Zepeda, James A. Hoxie, Richard Horuk, Thomas S. Kupper, Andrew D. Luster, and Ruth M. Ruprecht. "Human Immunodeficiency Virus-1 Entry Into Purified Blood Dendritic Cells Through CC and CXC Chemokine Coreceptors." Blood 90, no. 4 (August 15, 1997): 1379–86. http://dx.doi.org/10.1182/blood.v90.4.1379.1379_1379_1386.

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Blood dendritic cells (DC) are susceptible to both macrophage (M) and T-cell line (T) tropic human immunodeficiency virus type 1. The CC chemokines RANTES, macrophage inflammatory protein-1α (MIP-1α), MIP-1β, eotaxin, and, to a lesser extent, monocyte chemoattractant protein-1 (MCP-1) and MCP-4 blocked entry of M-tropic virus into blood DC. The CXC chemokine, SDF-1, a fusin (CXCR4 chemokine receptor) ligand, and an antifusin antibody inhibited DC entry by T-tropic virus. Purified blood DC contained CCR1, CCR2, CCR3, and CCR5 as well as the CXCR4 chemokine receptor RNA transcripts and high leve

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OSORIO, IVAN, and M. G. FREI. "SEIZURE ABATEMENT WITH SINGLE DC PULSES: IS PHASE RESETTING AT PLAY?" International Journal of Neural Systems 19, no. 03 (June 2009): 149–56. http://dx.doi.org/10.1142/s0129065709001926.

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Topological approaches for seizure abatement have received scarce attention. The ability to reset the phase of biological oscillations has been widely exploited in cardiology, as evidenced in part by the usefulness of implantable of defibrillators, but not in epileptology. The aim of this work is to investigate the feasibility of seizure blockage using single or brief monophasic (DC) pulse trains. Single DC or brief (0.1 s) pulse trains were delivered manually or automatically to generalized seizures, induced in rats with the convulsant 3-mercaptoprionic acid, a GABA inhibitor. Treatment outco

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Hwang, Sung In, Jung-Kyun Lee, Dong Hoon Song, Dae Sik Choi, and Do-Kyeong Ko. "Enhancement of the Optical Image with a DC Blocked Spiral Holographic Pattern." Journal of the Korean Physical Society 58, no. 2 (February 15, 2011): 387–91. http://dx.doi.org/10.3938/jkps.58.387.

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Frankel, Sarah S., Ralph M. Steinman, Nelson L. Michael, Silvia Ratto Kim, Nina Bhardwaj, Melissa Pope, Mark K. Louder, et al. "Neutralizing Monoclonal Antibodies Block Human Immunodeficiency Virus Type 1 Infection of Dendritic Cells and Transmission to T Cells." Journal of Virology 72, no. 12 (December 1, 1998): 9788–94. http://dx.doi.org/10.1128/jvi.72.12.9788-9794.1998.

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ABSTRACT Prevention of the initial infection of mucosal dendritic cells (DC) and interruption of the subsequent transmission of HIV-1 from DC to T cells are likely to be important attributes of an effective human immunodeficiency virus type 1 (HIV-1) vaccine. While anti-HIV-1 neutralizing antibodies have been difficult to elicit by immunization, there are several human monoclonal antibodies (MAbs) that effectively neutralize virus infection of activated T cells. We investigated the ability of three well-characterized neutralizing MAbs (IgG1b12, 2F5, and 2G12) to block HIV-1 infection of human

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Santiago-Schwarz, F., DL Coppock, AA Hindenburg, and J. Kern. "Identification of a malignant counterpart of the monocyte-dendritic cell progenitor in an acute myeloid leukemia." Blood 84, no. 9 (November 1, 1994): 3054–62. http://dx.doi.org/10.1182/blood.v84.9.3054.3054.

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Abstract Myeloblasts derived from the peripheral blood of a patient with acute myelogenous leukemia (ORL47) were found to represent the malignant counterpart of the newly elucidated monocyte-dendritic cell colony- forming unit (mono-DC-CFU). The specific cytokine conditions require to achieve intermediate and terminal maturation of DCs and monocytes from these progenitors were defined. With tumor necrosis factor (TNF) + granulocyte-macrophage colony-stimulating factor (GM-CSF) + stem cell factor treatment numerous colony-like clusters developed. In contrast with normal DC development, further

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Santiago-Schwarz, F., DL Coppock, AA Hindenburg, and J. Kern. "Identification of a malignant counterpart of the monocyte-dendritic cell progenitor in an acute myeloid leukemia." Blood 84, no. 9 (November 1, 1994): 3054–62. http://dx.doi.org/10.1182/blood.v84.9.3054.bloodjournal8493054.

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Myeloblasts derived from the peripheral blood of a patient with acute myelogenous leukemia (ORL47) were found to represent the malignant counterpart of the newly elucidated monocyte-dendritic cell colony- forming unit (mono-DC-CFU). The specific cytokine conditions require to achieve intermediate and terminal maturation of DCs and monocytes from these progenitors were defined. With tumor necrosis factor (TNF) + granulocyte-macrophage colony-stimulating factor (GM-CSF) + stem cell factor treatment numerous colony-like clusters developed. In contrast with normal DC development, further advanceme

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Nonaka, Motohiro, Shogo Matsumoto, Bruce Yong Ma, Hiroshi Kido, Nana Kawasaki, Nobuko Kawasaki, and Toshisuke Kawasaki. "Glycan-Dependent and -Independent Dual Recognition between DC-SIGN and Type II Serine Protease MSPL/TMPRSS13 in Colorectal Cancer Cells." Applied Sciences 10, no. 8 (April 13, 2020): 2687. http://dx.doi.org/10.3390/app10082687.

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A class of glycoproteins such as carcinoembryonic antigen (CEA)/CEA-related cell adhesion molecule 1(CEACAM1), CD26 (DPPIV), and mac-2 binding protein (Mac-2BP) harbor tumor-associated glycans in colorectal cancer. In this study, we identified type II transmembrane mosaic serine protease large-form (MSPL) and its splice variant transmembrane protease serine 13 (TMPRSS13) as ligands of Dendritic cell-specific intercellular adhesion molecule-3-grabbing nonintegrin (DC-SIGN) on the colorectal cancer cells. DC-SIGN is a C-type lectin expressed on dendritic cells, serves as a pattern recognition re

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Platzer, Barbara, Almut Jörgl, Sabine Taschner, Bernhard Höcher, and Herbert Strobl. "RelB regulates human dendritic cell subset development by promoting monocyte intermediates." Blood 104, no. 12 (December 1, 2004): 3655–63. http://dx.doi.org/10.1182/blood-2004-02-0412.

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In humans, epithelial Langerhans cells (LCs) and monocyte-derived/interstitial dendritic cells (DCs) constitute 2 myeloid DC sublineages. Molecular mechanisms involved in their development from common myeloid progenitors remain poorly defined. Here we demonstrate that the nuclear factor-κB (NF-κB) transcription factor RelB regulates the generation of monocytic CD14+CD11b+ precursors of interstitial DCs from human hematopoietic progenitors. RelB overexpression promoted, whereas endogenous RelB inhibition (by p100ΔN) blocked, precursor cell development along this DC subset pathway. RelB inhibiti

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