Gotowe bibliografie tematyczne / Dopamine type I receptor

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „Dopamine type I receptor”

Autor: Grafiati
Data publikacji: 19 października 2024
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Dopamine type I receptor"

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Sarkar, D. K., K. Chaturvedi, S. Oomizu, N. I. Boyadjieva та C. P. Chen. "Dopamine, Dopamine D2 Receptor Short Isoform, Transforming Growth Factor (TGF)-β1, and TGF-β Type II Receptor Interact to Inhibit the Growth of Pituitary Lactotropes". Endocrinology 146, № 10 (2005): 4179–88. http://dx.doi.org/10.1210/en.2005-0430.

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The neurotransmitter dopamine is known to inhibit prolactin secretion and the proliferation of lactotropes in the pituitary gland. In this study, we determined whether dopamine and TGFβ1 interact to regulate lactotropic cell proliferation. We found that dopamine and the dopamine agonist bromocriptine stimulated TGFβ1 secretion and TGFβ1 mRNA expression but inhibited lactotropic cell proliferation both in vivo and in vitro. The dopamine’s inhibitory action on lactotropic cell proliferation was blocked by a TGFβ1-neutralizing antibody. We also found that PR1 cells, which express low amounts of t
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Peiser, Christian, Marcello Trevisani, David A. Groneberg, et al. "Dopamine type 2 receptor expression and function in rodent sensory neurons projecting to the airways." American Journal of Physiology-Lung Cellular and Molecular Physiology 289, no. 1 (2005): L153—L158. http://dx.doi.org/10.1152/ajplung.00222.2004.

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Agonists of the dopamine receptors have been demonstrated to have bronchodilatory properties in pathologically constricted airways. The mechanism by which these agonists induce bronchodilatation is thought to involve airway sensory nerves. In this study, the expression and function of dopamine D2 receptor were examined in sensory ganglia supplying the airways. Neuronal dopamine D2 receptor mRNA expression was demonstrated by single-cell RT-PCR following laser-assisted microdissection. The projection of the neurons to the airways was confirmed by retrograde neuronal labeling. In functional stud
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Myslivecek, Jaromir. "Dopamine and Dopamine-Related Ligands Can Bind Not Only to Dopamine Receptors." Life 12, no. 5 (2022): 606. http://dx.doi.org/10.3390/life12050606.

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The dopaminergic system is one of the most important neurotransmitter systems in the central nervous system (CNS). It acts mainly by activation of the D1-like receptor family at the target cell. Additionally, fine-tuning of the signal is achieved via pre-synaptic modulation by the D2-like receptor family. Some dopamine drugs (both agonists and antagonists) bind in addition to DRs also to α2-ARs and 5-HT receptors. Unfortunately, these compounds are often considered subtype(s) specific. Thus, it is important to consider the presence of these receptor subtypes in specific CNS areas as the functi
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Shariati, G. H., G. Ahangari, M. R. Asadi, F. Poyafard, and H. R. Ahmadkhaniha. "Dopamine Receptor Gene Expression Changes in Peripheral Blood Mononuclear Cells from Schizophrenic Patients Treated with Haloperidol and Olanzapine." European Journal of Inflammation 7, no. 2 (2009): 71–76. http://dx.doi.org/10.1177/1721727x0900700203.

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We investigated dopamine receptor gene expression in peripheral blood mononuclear cells of schizophrenic patients before and after treatment. Also dopamine receptor genes expression profile was compared in two treatment groups including haloperidol and olanzapine. The peripheral blood mononuclear cells were separated from whole blood by Ficoll-hypaque; the total cellular RNA was extracted and the cDNA was synthesized. This process was followed by real-time polymerase chain reaction using primer pairs specific for five dopamine receptor mRNAs and β-actin as internal control. The results show th
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Helms, My N., Xi-Juan Chen, Semra Ramosevac, Douglas C. Eaton, and Lucky Jain. "Dopamine regulation of amiloride-sensitive sodium channels in lung cells." American Journal of Physiology-Lung Cellular and Molecular Physiology 290, no. 4 (2006): L710—L722. http://dx.doi.org/10.1152/ajplung.00486.2004.

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Dopamine increases lung fluid clearance. This is partly due to activation of basolateral Na-K-ATPase. However, activation of Na-K-ATPase by itself is unlikely to produce large changes in transepithelial transport. Therefore, we examined apical and basolateral dopamine's effect on apical, highly selective sodium channels [epithelial sodium channels (ENaC)] in monolayers of an alveolar type 2 cell line (L2). Dopamine increased channel open probability ( Po) without changing the unitary current. The D1 receptor blocker SCH-23390 blocked the dopamine effect, but the D2 receptor blocker sulpiride d
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Ding, Guoliang, Rob F. Wiegerinck, Ming Shen, Anca Cojoc, Carlo M. Zeidenweber та Mary B. Wagner. "Dopamine increases L-type calcium current more in newborn than adult rabbit cardiomyocytes via D1 and β2 receptors". American Journal of Physiology-Heart and Circulatory Physiology 294, № 5 (2008): H2327—H2335. http://dx.doi.org/10.1152/ajpheart.00993.2007.

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Dopamine is used to treat heart failure, particularly after cardiac surgery in infants, but the mechanisms of action are unclear. We investigated differences in the effect of dopamine on L-type calcium current ( ICa) between newborn (NB, 1–4 days) and adult (AD, 3–4 mo) rabbit ventricular myocytes. Myocytes were enzymatically dissociated from NB and AD rabbit hearts. ICa was recorded by using the whole cell patch-clamp technique. mRNA levels of cardiac dopamine receptor type 1 (D1), type 2 (D2), and β-adrenergic receptors (β-ARs) were measured by real-time RT-PCR. Dopamine (100 μM) increased I
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MORA-FERRER, CARLOS, and VOLKER GANGLUFF. "D2-dopamine receptor blockade impairs motion detection in goldfish." Visual Neuroscience 17, no. 2 (2000): 177–86. http://dx.doi.org/10.1017/s0952523800171196.

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Under photopic illumination conditions, motion detection in goldfish is dominated by the long-wavelength-sensitive cone type (L-cone), and under scotopic conditions motion it is determined by rods (Schaerer & Neumeyer, 1996). The switch from rod-dominated to cone-dominated motion detection occurs during light adaptation. It has been suggested that dopamine acts as a neuronal light-adaptative signal. It is known that dopamine affects wavelength discrimination through D1-dopamine receptors (Mora-Ferrer & Neumeyer, 1996), and the dorsal light reflex through D1- and D2-dopamine receptors (
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Hayashida, Yuki, and Andrew T. Ishida. "Dopamine Receptor Activation Can Reduce Voltage-Gated Na+ Current by Modulating Both Entry Into and Recovery From Inactivation." Journal of Neurophysiology 92, no. 5 (2004): 3134–41. http://dx.doi.org/10.1152/jn.00526.2004.

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We tested whether dopamine receptor activation modulates the voltage-gated Na+ current of goldfish retinal ganglion cells, using a fast voltage-clamp amplifier, perforated-patch whole cell mode, and a physiological extracellular Na+ concentration. As found in other cells, activators of D1-type dopamine receptors and of protein kinase A reduced the amplitude of current activated by depolarizations from resting potential without altering the current kinetics or activation range. However, D1-type dopamine receptor activation also accelerated the rate of entry into inactivation during subthreshold
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Milienne-Petiot, Morgane, Lucianne Groenink, Arpi Minassian, and Jared W. Young. "Blockade of dopamine D1-family receptors attenuates the mania-like hyperactive, risk-preferring, and high motivation behavioral profile of mice with low dopamine transporter levels." Journal of Psychopharmacology 31, no. 10 (2017): 1334–46. http://dx.doi.org/10.1177/0269881117731162.

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Background: Patients with bipolar disorder mania exhibit poor cognition, impulsivity, risk-taking, and goal-directed activity that negatively impact their quality of life. To date, existing treatments for bipolar disorder do not adequately remediate cognitive dysfunction. Reducing dopamine transporter expression recreates many bipolar disorder mania-relevant behaviors (i.e. hyperactivity and risk-taking). The current study investigated whether dopamine D1-family receptor blockade would attenuate the risk-taking, hypermotivation, and hyperactivity of dopamine transporter knockdown mice. Methods
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Pedarzani, P., and J. F. Storm. "Dopamine modulates the slow Ca(2+)-activated K+ current IAHP via cyclic AMP-dependent protein kinase in hippocampal neurons." Journal of Neurophysiology 74, no. 6 (1995): 2749–53. http://dx.doi.org/10.1152/jn.1995.74.6.2749.

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1. The effects of dopamine on the slow Ca(2+)-dependent K+ current (IAHP; AHP, afterhyperpolarization) and spike frequency adaptation were studied by whole cell voltage-clamp and sharp microelectrode current-clamp recordings in rat CA1 pyramidal neurons in rat hippocampal slices. 2. Dopamine suppressed IAHP in a dose-dependent manner, under whole cell voltage-clamp conditions. Similarly, under current-clamp conditions, dopamine inhibited spike frequency adaptation and suppressed the slow afterhyperpolarization. 3. The effect of dopamine on IAHP was mimicked by a D1 receptor agonist and blocked
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Rozprawy doktorskie na temat "Dopamine type I receptor"

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Hatcher-Solis, Candice N. "PHARMACOLOGICAL IMPLICATIONS OF ADENOSINE 2A RECEPTOR- DOPAMINE TYPE 2 RECEPTOR HETEROMERIZATION." VCU Scholars Compass, 2016. http://scholarscompass.vcu.edu/etd/4458.

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G protein-coupled receptors (GPCRs) are heptahelical, transmembrane proteins that mediate a plethora of physiological functions by binding ligands and releasing G proteins that interact with downstream effectors. GPCRs signal as monomers, complexes of the same receptor subtype (homomers), or complexes of different receptor subtypes (heteromers). Recently, heteromeric GPCR complexes have become attractive targets for drug development since they exhibit distinct signaling and cell-specific localization from their homomeric counterparts. Yet, the effect of heteromerization on the pharmacology of
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ZANINOVICH, OREL ANTHONY. "THE CLONING OF AN INDR-TYPE DOPAMINE RECEPTOR IN MANDUCA SEXTA." Thesis, The University of Arizona, 2008. http://hdl.handle.net/10150/192256.

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Mann, Miranda Jane. "A neuropsychological investigation of dopamine receptor 4 differences among attention deficit hyperactivity disorder-combined type and control children /." Digital version accessible at:, 2000. http://wwwlib.umi.com/cr/utexas/main.

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Gorji, Hassan. "Role of adenylyl cyclase type 5 in the regulation of the dopamine D3 receptor phosphorylation." Thesis, University of Ottawa (Canada), 2006. http://hdl.handle.net/10393/27364.

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Adenylyl cyclase type 5 (AC5) is expressed in the brain where the highest density of the dopamine D3 receptor (D3R) has been found. The D3R-mediated Gi/o protein activation leads to a specific inhibition of AC5. Therefore, as AC5 is the main signalosome partner of D3R, I hypothesize that D3R phosphorylation is differentially regulated in cells expressing AC5. In HEK293 cells expressing D3R alone, D3R undergo dopamine-induced phosphorylation. Interestingly, in cells co-expressing AC5 and D3R, D3R undergoes a Galphai-dependent dephosphorylation upon dopamine exposure while retaining its ability
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Maier, Annette Louise. "Comparative regional ontogeny of dopamine D₁ receptor binding and mRNA expression in pre- and postnatal rat brain /." Zürich, 1994. http://e-collection.ethbib.ethz.ch/show?type=diss&nr=10902.

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Etchepare, Laetitia. "Role of glutamate N-Methyl-D-Aspartate receptor surface trafficking in the firing pattern of midbrain dopaminergic neurons." Thesis, Bordeaux, 2017. http://www.theses.fr/2017BORD0849/document.

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Les neurones dopaminergiques (DA) mésencéphaliques jouent un rôle prépondérant dans de nombreuses fonctions cérébrales telles que la motivation, mais ils sont également impliques dans l’émergence de pathologies telles que la maladie de Parkinson et l’addiction aux drogues. Ces processus ayant en commun de modifier l’activité de décharge des neurones DA mésencéphaliques, il est d’une importance primordiale de comprendre les mécanismes sous-tendant cette activité. Parmi les différents canaux ioniques et récepteurs impliques dans la génération de l’activité de décharge des neurones DA, les récept
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Roberts-Crowley, Mandy L. "Modulation of Cav1.3 L-Type Calcium Channels by Arachidonic Acid and Muscarinic M1 Receptors: A Dissertation." eScholarship@UMMS, 2007. https://escholarship.umassmed.edu/gsbs_diss/348.

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Membrane excitability, gene expression, and neurotransmitter release are all controlled by voltage-gated L-type Ca2+ (L- )channels. In turn, Ca2+ channels are highly regulated by signal transduction cascades initiated by G protein-coupled receptor (GPCR) activation. In medium spiny neurons of the striatum, both the muscarinic M1 receptors (M1R) and dopaminergic D2 receptors (D2R) specifically inhibit the Cav1.3 L-channel. In Chapters III and IV, the pathways downstream of M1Rs and D2Rs are examined to determine whether an overlap or intersection in inhibition of Cav1.3 occurs by these two rece
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Lucas, Guillaume. "Etude in vivo des modalités d'intervention de la sérotonine et des récepteurs sérotoninergiques de type 5-HT/2A/2C, 5-HT3 et 5-HT4 dans le contrôle de la transmission dopaminergique nigro-striée et mésoaccumbale chez le rat." Bordeaux 2, 1999. http://www.theses.fr/1999BOR28692.

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Hegron, Alan. "Implication des récepteurs de la mélatonine dans les troubles neurologiques et le diabète de type 2 et identification de régions clés du récepteur MT1 responsables de sa sélectivité fonctionnelle." Thesis, Université Paris-Saclay (ComUE), 2018. http://www.theses.fr/2018SACLS555/document.

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La mélatonine est une neurohormone produite principalement par la glande pinéale de manière circadienne et agissant par l’activation de deux récepteurs couplés aux protéines G (RCPGs) appelés MT1 et MT2. La mélatonine régule de nombreuses fonctions physiologiques importantes. La régulation des niveaux de dopamine (DA) et de glucose en font partie mais nous ne savons pas clairement comment la mélatonine les régule.Les niveaux de DA extracellulaire sont principalement régulés par son transporteur (DAT) responsable de sa recapture dans les neurones présynaptiques afin de prévenir d’une hyperactiv
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Thirtamara, Rajamani Keerthi Krishnan. "Animal Models of Drug Addiction and Autism Spectrum Disorders." The Ohio State University, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=osu1386011455.

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Książki na temat "Dopamine type I receptor"

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Tupala, Erkki. Dopamine receptors and transporters in type 1 and 2 alcoholism measured with postmortem human whole hemisphere autoradiography. University of Kuopio, 2001.

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Tiberi, Mario. Dopamine receptor technologies. Humana Press, 2015.

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Tiberi, Mario, ed. Dopamine Receptor Technologies. Springer New York, 2015. http://dx.doi.org/10.1007/978-1-4939-2196-6.

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Donthamsetti, Prashant Chandra. Dissecting Dopamine D2 Receptor Signaling. [publisher not identified], 2015.

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L, Waddington John, ed. D1:D2 dopamine receptor interactions. Academic Press, 1993.

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Boileau, Isabelle, and Ginetta Collo, eds. Therapeutic Applications of Dopamine D3 Receptor Function. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-23058-5.

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Knapp, Mark. Development of dopamine receptor expressing adenoviral vectors. National Library of Canada, 1997.

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R, Demirdamar, and Jenner Peter 1946-, eds. Dopamine receptor subtypes: From basic science to clinical application. IOS Press, 1998.

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Ray, Avi Andrew. SH3 binding domains in the dopamine D(3) receptor. National Library of Canada, 1999.

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Zawarynski, Paul. Dopamine D2 receptor monomers, dimers and higher order oligomers. National Library of Canada, 1998.

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Części książek na temat "Dopamine type I receptor"

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Turco, Raymond. "Dopamine Receptor." In Encyclopedia of Animal Cognition and Behavior. Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-47829-6_1256-1.

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Turco, Raymond. "Dopamine Receptor." In Encyclopedia of Animal Cognition and Behavior. Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-319-55065-7_1256.

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Fuxe, Kjell, Daniel Marcellino, Diego Guidolin, Amina Woods, and Luigi Agnati. "Dopamine Receptor Oligomerization." In The Dopamine Receptors. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-333-6_10.

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Chaudhary, Tanvi, Debashruti Das, Olivia Majhi, and Amrita Mukhopadhyay. "Dopamine Receptor D4." In Encyclopedia of Sexual Psychology and Behavior. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-08956-5_808-1.

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Hazelwood, Lisa A., R. Benjamin Free, and David R. Sibley. "Dopamine Receptor-Interacting Proteins." In The Dopamine Receptors. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-333-6_9.

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Deth, Richard C. "The Dopamine D4 Receptor." In Molecular Origins of Human Attention. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0335-4_4.

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Richtand, Neil M., Laurel M. Pritchard, and Lique M. Coolen. "Dopamine Receptor Alternative Splicing." In Dopamine and Glutamate in Psychiatric Disorders. Humana Press, 2005. http://dx.doi.org/10.1007/978-1-59259-852-6_2.

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Civelli, Olivier, James Bunzow, Paul Albert, Hubert H. M. Van Tol, and David Grandy. "The Dopamine D2 Receptor." In Molecular Biology of G-Protein-Coupled Receptors. Birkhäuser Boston, 1992. http://dx.doi.org/10.1007/978-1-4684-6772-7_7.

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Cepeda, Carlos, Véronique M. André, Emily L. Jocoy, and Michael S. Levine. "Dopamine Receptor Modulation of Glutamatergic Neurotransmission." In The Dopamine Receptors. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-333-6_11.

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Lee, Frankie H. F., and Albert H. C. Wong. "Dopamine Receptor Genetics in Neuropsychiatric Disorders." In The Dopamine Receptors. Humana Press, 2009. http://dx.doi.org/10.1007/978-1-60327-333-6_19.

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Streszczenia konferencji na temat "Dopamine type I receptor"

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Kamasak, Mustafa E., Charles A. Bouman, Bradley T. Christian, and Evan D. Morris. "Imaging D2-Dopamine Receptor using PET." In 2007 IEEE 15th Signal Processing and Communications Applications. IEEE, 2007. http://dx.doi.org/10.1109/siu.2007.4298698.

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Moritz, Amy E., Nora S. Madaras, Kirsten K. Snyder, et al. "Regulation of Dopamine Receptor Subtypes by G Protein-Coupled Receptor Kinase Isoforms." In ASPET 2024 Annual Meeting Abstract. American Society for Pharmacology and Experimental Therapeutics, 2024. http://dx.doi.org/10.1124/jpet.159.988010.

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Liu, X., J. Guan, F. Tao, and B. Mao. "Acupuncture Zusanli Regulate COPD Inflammation Through Dopamine D2 Receptor." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a4755.

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Harrison, J. M., I. Nir, M. Rubinstein, M. J. Low, D. K. Grandy, and P. M. Iuvone. "Retinal Function in Dopamine D4 Receptor Knockout (D4KO) Mice." In Vision Science and its Applications. OSA, 2000. http://dx.doi.org/10.1364/vsia.2000.fd2.

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Javadi, Arman, Nicholas Forsythe, Alaa Refaat, et al. "Abstract 3448: Targeting the dopamine receptor 2 inBRAFmutant colorectal cancer." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-3448.

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Uysal, Mehmet Atilla, Ulgen Sever, and Sacide Pehlivan. "The dopamine receptor D4 VNTR 48bp gene variant in nicotine addiction." In ERS International Congress 2016 abstracts. European Respiratory Society, 2016. http://dx.doi.org/10.1183/13993003.congress-2016.oa1486.

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Raghuraman, Gayatri, Nanduri R. Prabhakar, and Ganesh K. Kumar. "Dopamine D1 Receptor Signaling Mediates Altered GABA Synthesis By Intermittent Hypoxia." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a6630.

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Blake, Keyana, Basant Hens, and Anthony J. Baucum. "Investigating the role of spinophilin in mediating Dopamine D2 Receptor Activity." In ASPET 2024 Annual Meeting Abstract. American Society for Pharmacology and Experimental Therapeutics, 2024. http://dx.doi.org/10.1124/jpet.321.940880.

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Wen, Ruojian, Xiaoqing Chen, Dan Huang, Minjie Chen, and Yuwei Liu. "Sleep Deprivation Increased Dopamine D2 Receptor Expression through Downregulation of miR-9." In 2015 7th International Conference on Information Technology in Medicine and Education (ITME). IEEE, 2015. http://dx.doi.org/10.1109/itme.2015.122.

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Madaras, Nora S., Michele L. Rankin, R. Benjamin Free, et al. "Delineation of the G Protein-Coupled Receptor Kinase Phosphorylation Sites Within the D1 Dopamine Receptor and Their Role in Regulating Receptor Function." In ASPET 2023 Annual Meeting Abstracts. American Society for Pharmacology and Experimental Therapeutics, 2023. http://dx.doi.org/10.1124/jpet.122.154110.

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Raporty organizacyjne na temat "Dopamine type I receptor"

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Dr. Jogeshwar Mukherjee. Development of dopamine receptor radiopharmaceuticals for the study of neurological and psychiatric disorders. Office of Scientific and Technical Information (OSTI), 2009. http://dx.doi.org/10.2172/944919.

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Mukherjee, J. Development of dopamine receptor radiopharmaceuticals for the study of neurological and psychiatric disorders. Progress report 1994--1997. Office of Scientific and Technical Information (OSTI), 1999. http://dx.doi.org/10.2172/764610.

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Reiss, Michael. Type I Receptor Kinase Inhibitors - A Novel Treatment for Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada408030.

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Fagan, Dedra. Type-I Insulin-Like Growth Factor Receptor (IGF1R)-Estrogen Receptor (ER) Crosstalk Contributes to Antiestrogen Therapy Resistance in Breast Cancer Cells. Defense Technical Information Center, 2013. http://dx.doi.org/10.21236/ada575846.

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Reise, Michael. TGF-beta Type I Receptor Kinase Inhibitors - A Novel Treatment for Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada416442.

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Rausch, Matthew. Enhancement of Dendritic Cell-Based Immunotherapy Using a Small Molecule TGF-beta Receptor Type I Kinase Inhibitor. Defense Technical Information Center, 2008. http://dx.doi.org/10.21236/ada487435.

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Plymate, Stephen R. Therapy of Prostate Cancer Using a Human Antibody Targeting the Type 1 Insulin-Like Growth Factor Receptor (IGF-IR). Defense Technical Information Center, 2009. http://dx.doi.org/10.21236/ada524529.

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Thomas, Tania, S. Shappell, S. Kasper, R. A. Serra, and H. L. Moses. The Development and Characterization of a Transgenic Mouse Model Over-Expressing a Truncated TGF(Beta) Type II Receptor in the Prostate. Defense Technical Information Center, 2000. http://dx.doi.org/10.21236/ada390670.

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Deo, Salil, David McAllister, Naveed Sattar, and Jill Pell. The time-varying cardiovascular benefits of glucagon like peptide-1 agonist (GLP-RA)therapy in patients with type 2 diabetes mellitus: A meta-analysis of multinational randomized trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.7.0097.

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Streszczenie:
Review question / Objective: P - patients with type 2 diabetes melllitus already receiving routine medical therapy; I - patients receiving glucagon like peptide 1 receptor agonist (GLP1 receptor agonist) therapy (semaglutide, dulaglutide, liraglutide, exenatide, lixisenatide, efpeglenatide, abiglutide); C - patients receiving standard therapy for diabetes mellitus but not receiving GLP1 agonist therapy; O - composite end point as per invididual trial, cardiovascular mortality, all-cause mortality, myocardial infarction, stoke. Condition being studied: Type 2 diabetes mellitus. Study designs to
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Zhuo, Chuanjun, Hongjun Tian, Lina Wang, Xiangyang Gao, Li Ding, and Ming Liu. Comparative safety of glucagon like peptide‑1 receptor agonists in patients with type 2 diabetes: a network meta-analysis of cardiovascular outcome trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2020. http://dx.doi.org/10.37766/inplasy2020.8.0122.

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