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Gotowa bibliografia na temat „Empyema of Gall Bladder”

Autor: Grafiati

Data publikacji: 2 czerwca 2025

Data aktualizacji: 22 czerwca 2025

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Artykuły w czasopismach na temat "Empyema of Gall Bladder"

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Vineeth, V. K., Jagadeesh Chandrasekaran, and Aarthee Asokan. "Empyema of Gall Bladder- Typhoid." International Journal of Health Sciences and Research 11, no. 8 (2021): 194–97. http://dx.doi.org/10.52403/ijhsr.20210828.

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Enteric fever involves reticuloendothelial system. Most common complications are intestinal perforation, hepatitis, encephalopathy and bleeding ileal ulcers. Apart from known complications we present an interesting case of middle aged female with uncontrolled diabetes presenting with empyema of gall bladder secondary to Salmonella typhi. Both blood and gallbladder tissue bacteriological cultures had Salmonella typhi confirming our diagnosis. It was noteworthy that empyema was found intraoperatively. Patient recovered after cholecystectomy and antibiotics. Considering this rare complication we advise Typhoid conjugate vaccine in tropical countries. This case report paves way to recognize this complication to watch for. Key words: Enteric fever, Empyema, complications, Typhoid vaccine, gall bladder.

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ZAKRIA, Muhammad. "Empyema gall bladder in cirrhotic patient." Annals of Hepato-Biliary-Pancreatic Surgery 25, no. 1 (2021): S319. http://dx.doi.org/10.14701/ahbps.ep-121.

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Mir, IqbalSaleem. "Empyema gall bladder and laparoscopic cholecystectomy." Journal of Minimal Access Surgery 3, no. 4 (2007): 178. http://dx.doi.org/10.4103/0972-9941.38913.

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Ferdousi, Shamima, and Sadia Armin Khan. "Primary Squamous Cell Carcinoma of Gall Bladder: A Case Report." Ibrahim Medical College Journal 8, no. 2 (2016): 61–63. http://dx.doi.org/10.3329/imcj.v8i2.26681.

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Squamous cell carcinoma of the gall bladder is rare. It accounts for less than 12.7 % of all cases of gall bladder cancer. Pure squamous cell carcinoma is even less common with a reported incidence of 3.3%. We present a case of 70 years-old man with decreased appetite, vomiting and fever associated with right upper quadrant pain for two months. Ultrasonography of the abdomen revealed a distended gallbladder with multiple calculi along with large hyperechoic area of sludge. Provisional diagnosis was cholelithiasis with empyema of gall bladder. Cholecystectomy was done. Histopathological examination revealed well to moderately differentiated squamous cell carcinoma of the gall bladder without evidence of metastasis.Ibrahim Med. Coll. J. 2014; 8(2): 61-63

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Dr, Usman Ali Dr Muhammad Zubair. "POST CHOLECYSTECTOMY HISTOPATHOLOGICAL PATTERN OF GALL BLADDER." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES o6, no. 03 (2019): 5133–37. https://doi.org/10.5281/zenodo.2590607.

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<strong><em>Objective: </em></strong><em>The main objective of this research work was to find out the pattern of histopathology of the specimens of gall bladder in the patients after surgical removal of gall bladder. </em> <strong><em>Methodology: </em></strong><em>This was a retrograde research work conducted in a hospital of private university & two others private hospitals which were non-teaching in the city of Hyderabad. This research work covered a period of complete three years from the start of June 2005 to the end of May 2008. The analysis of the reports of histopathology of all patients who were undergoing open cholecystectomy or laparoscopic cholecystectomy carried out. The inspection of the documents of these patients carried out with specific prominence on appearance, findings of ultrasound before operation, findings during surgery & results of histopathology. </em> <strong><em>Results: </em></strong><em>Two hundred</em><em> and eighty-two samples of gall bladder were put under histopathology in the period of this study duration. Seventy-five were the males & two hundred and seven were the female participants. Chronic inflammation of the gall bladder was very dominant outcome of histopathology observed in more than sixty four percent patients. Cholecystitis was followed by the empyema in more than thirty three percent patients whereas gall bladder carcinoma was present in only 1.4% patients.</em> <strong><em>Conclusion: </em></strong><em>Chronic cholecystitis was the most common feature as discovered by histopathology.</em> <strong>Key Words: </strong><em>Empyema, carcinoma, inflammation, chronic, gall bladder, cholelithiasis, histopathology. </em>

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Tauro, LeoF, JohnJS Martis, and HejmadiD Shenoy. "Tuberculosis of gall bladder presenting as empyema." Saudi Journal of Gastroenterology 14, no. 2 (2008): 101. http://dx.doi.org/10.4103/1319-3767.39630.

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Mishra, Dr Anurag, Dr Md Abu Masud Ansari, and Dr Shivanshu Misra. "Laparoscopic management of rare “ H type” duplication of gall bladder." Surgical update: International Journal of Surgery and Orthopedics 6, no. 6 (2020): 354–57. http://dx.doi.org/10.17511/ijoso.2020.i06.03.

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A duplicated gallbladder is a rare congenital anomaly with an incidence of 1:4000 live births. Theycan remain asymptomatic and identified incidentally or present as acute cholecystitis, empyema,torsion, cholecystoenteric fistula, Gall bladder lump, or carcinoma. Here the current case is aboutdiscussing a case of a 25-year-old female who presented with symptomatic gallstone disease with aduplicated gallbladder having multiple stones in both the gallbladders. MRCP performedpreoperatively revealed Y type duplication (double Gall bladder with common cystic duct).Laparoscopic cholecystectomy was performed and it finally revealed H type duplication (double Gallbladder with separate cystic ducts for each Gall Bladder).

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Iqbal, Muhammad Nouman, Muhammad Arslan Iqbal, Rukhsar Javaid, and Muhammad Waseem Abbas. "Gall stones: a fundamental clinical review." International Journal of Research in Medical Sciences 7, no. 7 (2019): 2869. http://dx.doi.org/10.18203/2320-6012.ijrms20192938.

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Formation of stones in the gall bladder is known as cholelithiasis. About 10% to 20% of Western population are suffering from gall stones and this percentage is increasing day by day. Biochemically gall stones are classified into black pigment stones, brown pigment stones and cholesterol stones. Gall stones can be anatomically located at two possible sites; in the gall bladder known as cholelithiasis and in the common bile duct known as choledocholithiasis. Gall stones may present with symptoms known as symptomatic gallstones or without symptoms known as asymptomatic gallstones. The major causes of gallstones are high cholesterol diet, low bile salt levels, decreased gall bladder motility etc. Obesity, female gender, family history, rapid weight loss and vitamin B12 or folic acid deficiency are considered as important risk factors in the development of gall stones. The clinical presentations include acute cholecystitis and febrile illness with pain and tenderness in the right upper quadrant (Murphy sign). Generalized body weakness and weight loss are considered as generalized symptoms of gallstones. The complications include cholangitis, empyema of gall bladder, pancreatitis, abscess formation, porcelain gall bladder and gall bladder perforation. The differential diagnosis of gall stones is carried out based on endoscopy, ALT and AST serum levels. Non-surgical treatment for gall stones is oral dissolution therapy. The standard surgical treatment for gall stones is cholecystectomy.

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Dr., Rabia Asif Dr. Shah Faisal Dr. Pir Mubassir shah. "POST CHOLECYSTECTOMY HISTOPATHOLOGICAL PATTERN OF SAMPLES OF GALL BLADDER." INDO AMERICAN JOURNAL OF PHARMACEUTICAL SCIENCES 06, no. 01 (2019): 474–78. https://doi.org/10.5281/zenodo.2532147.

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<strong><em>Objective: </em></strong><em>The objective of this research work was to find out the pattern of histopathology of the specimens of gall bladder in the patients after surgical removal of gall bladder. </em> <strong><em>Methodology: </em></strong><em>This was a retrograde research work conducted in a hospital of private university & two others private hospitals which were non-teaching in the city of Lahore. This research was completed in three years and the duration of this study was from June 2015 to May 2018. The analysis of the reports of histopathology of all patients who were undergoing open cholecystectomy or laparoscopic cholecystectomy carried out. The inspection of the documents of these patients carried out with specific prominence on appearance, findings of ultrasound before operation, findings during surgery & results of histopathology. </em> <strong><em>Results: </em></strong><em>Two hundred</em><em> and eighty two samples of gall bladder were put under histopathology in the period of this study duration. Seventy five were the males & two hundred and seven were the female participants. Chronic inflammation of the gall bladder was very dominant outcome of histopathology observed in more than sixty four percent patients. Cholecystitis was followed by the empyema in more than thirty three percent patients whereas gall bladder carcinoma was present in only 1.4% patients.</em> <strong><em>Conclusion: </em></strong><em>Chronic cholecystitis was the most common feature as discovered by histopathology.</em> <strong>Key Words: </strong><em>Chronic Cholecystitis, Hyperplasia, Chronic, Malignancy, Empyema.</em> <strong>  INDO AMERICAN JOURNAL OF</strong> <strong> PHARMACEU</strong> <strong>QR</strong><strong> code</strong>     <strong>TICAL SCIENCES</strong>  

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Ismail, Muhammad, Nasir Mehmood Watto, Muhammad Qasim Butt, and Fareeha Naz. "VARIOUS FACTORS INVOLVED DURING SAFE LAPAROSCOPIC CHOLECYSTECTOMY IN CASES OF EMPYEMA GALL BLADDER." PAFMJ 71, no. 2 (2021): 540–44. http://dx.doi.org/10.51253/pafmj.v71i2.2791.

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Objective: To evaluate the various factors involved during the safe cholecystectomy performed by laparoscopic method in cases of empyema of gall bladder operated at Pak Emirates Military Hospital Rawalpindi. Study Design: Cross-sectional analytical study. Place and Duration of Study: Pak Emirates Military Hospital, Rawalpindi, from Nov 2017 to Mar 2019. Methodology: This study was performed on 70 patients undergoing cholecystectomy by laparoscopic method for empyema gall bladder during the study period. Adverse effects after the procedure were assessed in detail at 48 hours, at time of discharge and two weeks after the procedure on all the participants. Demographic profile and other factors were compared in the groups with and without the complications by using the chi-square test and binary logistic regression. Results: Out of 70 patients included in the final analysis 24 (34.3%) were male and 46 (65.7%) were female. Mean age of patients who underwent laparoscopic surgery for empyema gall bladder in our study was 42.43 ± 3.161 years. Bile duct perforation and leakage 6 (8.5%) was the commonest complication among the patients included in our study followed by surgical site infection 5 (7.1%). With binary logistic regression we found that presence of co-morbids and high preoperative C reactive protein had a strong association with presence of complications among the patients undergoing cholecystectomy in our study while age, gender, transfusion during surgery were not linked with the complications during or after the study. Conclusion: Limited number of patients faced the complications during or after the surgery. Patients with co-morbid.............

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Rozprawy doktorskie na temat "Empyema of Gall Bladder"

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Curtis-Barrett, Carmel Mary. "Gene transfer to the murine gall bladder." Thesis, University of Cambridge, 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624932.

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Moutsopoulos, Konstantinos. "Physically deformable models for simulation of laparoscopic surgery." Thesis, Imperial College London, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.339157.

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Маркевич, Ольга Василівна, Ольга Васильевна Маркевич, Olha Vasylivna Markevych, Е. А. Лазебник, Л. И. Величко та Л. Е. Карпенко. "Применение "Холивера" при заболеваниях желчевыводящей системы у детей". Thesis, Изд-во СумГУ, 2006. http://essuir.sumdu.edu.ua/handle/123456789/6977.

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Opiyo, Monica Naomi. "The role of glucocorticoid metabolism in bile acid homeostasis." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/25673.

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Alterations in glucocorticoid (GC) biosynthesis and metabolism are associated with a variety of pathophysiological disorders including cholestasis, diabetes and other metabolic disorders. Bile acids (BA) are also important modulators of metabolic functions and regulate cholesterol, triglyceride and glucose homeostasis as well as being critical for dietary fat digestion, enterohepatic function, and postprandial thermogenesis. In intact cells and in vivo, the 11β-hydroxysteroid dehydrogenase type 1 (11β-HSD1) enzyme converts inactive GC precursors (cortisone in humans, and 11-dehydrocorticosterone in mice and rats) into their active forms (cortisol and corticosterone, respectively) thereby amplifying local intracellular GC levels. Interconversion by 11β-HSD1 of other sterols has also been described. These include conversions of 7keto-cholesterol to 7β-hydroxycholesterol, 7-oxodehydroepiandrosterone (7-oxo-DHEA) to 7α-hydroxy- and 7β-hydroxy DHEA, 7- oxo-lithocholic acid (LCA, a bile acid; BA) to chenodeoxycholic acid (CDCA, a 7α- hydroxylated BA) and ursodeoxycholic acid (UDCA, a 7β-hydroxylated BA) in human liver microsomes. In the liver, BA inhibit 11β-HSD1 but whether 11β-HSD1 regulates BA homeostasis is unclear. Evidence of molecular regulation of the enterohepatic recycling of bile acids by liver glucocorticoid receptor (GR) in mice does suggest a role for 11β-HSD1. It was therefore hypothesised that disruption of 11β-HSD1 expression in mice would impair BA recycling and might affect the relative concentrations of BA within the enterohepatic circuit. The primary objective of the current work was to investigate the impact of altered 11β-HSD1 on BA homeostasis. This was achieved using genetically modified mouse models with altered 11β-HSD1 expression, either globally or restricted to hepatocytes. BA are stored in the gall bladder and are released postprandially, to aid digestion. It was hypothesised that 11β-HSD1 deficiency might the affect the process of postprandial gall bladder emptying/refilling. Mice with global 11β-HSD1 knockout (Hsd11b1-/-) and age-matched control mice (C57Bl/6) were either fasted for 4h and culled or fasted for 4h and re-fed for another 4h before culling. Their response to fasting and re-feeding was assessed with specific focus on organs associated with BA recycling in the enterohepatic circuit (liver, gall bladder, serum and small intestine). Gall bladders of fasted Hsd11b1-/- and C57Bl/6 mice had similar volumes of bile but in fasted Hsd11b1-/- mice, BA concentrations were higher in serum and liver. As expected, re-feeding caused gall bladder emptying in C57Bl/6 mice with consequent increased serum and liver bile acid concentrations. In Hsd11b1-/- mice, the gall bladder did not empty and serum and liver BA concentrations were similar to the fasted state. To explore possible reasons for this, levels of mRNA encoding proteins known to be involved in hepatic BA transport were quantified using real-time q-PCR. Levels of mRNA encoding NTCP/ SCL10A1/ SCL10A1, the transporter responsible for most hepatocyte BA uptake, were increased in livers of fasted Hsd11b1-/- mice whereas levels of Slc51b mRNA, encoding the OST- transporter that facilitates BA removal from liver to the systemic circulation, and levels of Mrp2 and Atp8b1/FIC1 mRNAs (both encoding proteins which transport BA from liver into gall bladder) were decreased. This suggests that in fasted Hsd11b1-/- mice, BA transporter expression is altered to increase BA influx into hepatocytes and decrease efflux, to compensate for reduced levels of liver BA. These data together imply that bile acid recycling is controlled by 11β-HSD1 activity which regulates gall bladder emptying, hepatic BA concentration and BA transporter activity to ensure continuity of BA recycling within the enterohepatic circuit compartments. These changes may also affect digestion of lipids and fat-soluble micronutrients. Because 11β-HSD1 can directly metabolise secondary BA, it was predicted that 11β-HSD1 deficiency would lead to changes in the BA profile. Profiling of BA in the gall bladder was performed using mass spectrophotometry. In Hsd11b1-/- mice, 7α-hydroxylated BA predominated (cholic acid [CA]>α-muricholic acid [α- MCA]>CDCA>others), in contrast to C57Bl/6 mice in which 7β-hydroxylated BA predominated (ω-MCA>β-MCA>UDCA>others). The ratio of 7α:7β acids was therefore >100-fold greater in Hsd11b1-/- mice. This suggests that 11β-HSD1 either directly or indirectly controls the epimerisation of 7α- to 7β- hydroxylated BAs. Measurement of mRNAs encoding proteins important for hepatic BA biosynthesis in livers of fasted Hsd11b1-/- mice showed decreased expression of Scarb1/SR-B1, Cyp39a1 and Cyp27a1 (though with no change in levels of CDCA, the product of CYP27A1, in liver or bile fluid), compared to fasted control mice. Hepatic levels of Gpbar1/TGR5/GPBAR1 and Cyp3a11 mRNAs, encoding proteins important in BA detoxification, were increased and decreased, respectively. This suggests that Gpbar1/TGR5/GPBAR1, encoding G-protein coupled bile acid receptor (also called TGR5/GPBAR1) and an FXR target, could be induced to detoxify 7α-hydroxylated BA whereas expression of Cyp3a11, which catalyses the conversion of LCA to hyodeoxycholic acid (HDCA) is decreased; bile fluid of Hsd11b1-/- mice contained lower levels of LCA and little to no HDCA, though LCA and HDCA levels in liver were unaltered. Currently, the functional differences between 7α- and 7β- hydroxylated BA are not clear. However, these findings could have significant implications for bile acid-mediated transcription which, in turn, might affect lipid and sterol metabolism. Also, alterations in BA composition may have other physiological consequences via other pathways. Because cholesterol is the precursor of BA synthesis, it was hypothesised that western diet (WD) (containing cholesterol) would exacerbate and/or alter the phenotype of Hsd11b1-/- mice. Gall bladder weights of fasted Hsd11b1-/- and control C57Bl/6 mice did not change with western diet compared to chow diet. In control C57Bl/6 mice, the total BA concentration in the gall bladder increased in response to WD in comparison to chow diet. In contrast, Hsd11b1-/- mice showed no change in total BA concentration when fed on WD in comparison to chow. These data indicate that 11β-HSD1 is required by mice for the normal increase in total BA concentration in bile in response to dietary cholesterol. BA profiling of bile from control mice fed on WD showed no difference in the relative amounts of 7β-hydroxylated BA and 7α-hydroxylated BA to littermates fed on chow diet with the exception of β–MCA which increased, and α–MCA which decreased. Like chow-fed Hsd11b1-/- mice, BA profiling of bile from WD-fed Hsd11b1-/- mice showed a significant decrease in relative levels of 7β-hydroxylated BA (UDCA < β-MCA < others) and an increase in percentage of 7α-hydroxylated BAs (CA>α-MCA>CDCA>others) compared to C57Bl/6 controls. These data show that Hsd11b1-/- mice fail to show the normal increase in 7β-hydroxylated BA and decrease in 7α-hydroxylated BA observed in control mice in response to a cholesterol containing diet, suggesting 11β-HSD1 deficiency blunts the influence of cholesterol on BA composition. Measurement of hepatic mRNAs encoding BA transporters suggest that hepatocyte uptake of BA is decreased in C57Bl/6 on WD compared to those mice on chow diet, whereas this was not the case in Hsd11b1-/- mice where hepatic expression did not change with diet. Thus, Hsd11b1-/- mice failed to increase expression of Ntcp/ Scl10a1/ Scl10a1 appropriately, suggesting impaired hepatic BA uptake, while Slc51b (encoding OST-β) expression was increased, compared to control mice, possibly to reduce hepatic BA concentration by transporting BA out of hepatocytes into the systemic circulation. Therefore, Hsd11b1-/- mice may adapt to a cholesterol-induced increase in hepatic BA by blunting hepatic BA uptake via NTCP/ SCL10A1/ SCL10A1 and increasing hepatic efflux via OST-β. The effects of 11β-HSD1 deficiency upon BA recycling and BA profile and concentration within the enterohepatic circuit, could reflect 11β-HSD1 action within the liver or could be due to actions in other tissues.<br>To investigate the role of hepatic 11β-HSD1 specifically, 11β-HSD1 liver-specific knockout (Hsd11b1LKO), 11β- HSD1 liver-specific over-expressors (Hsd11b1LOE) and control mice with exon 3 of the Hsd11b1 gene “floxed” (Hsd11b1F) were studied. Findings from this study indicate a role for 11β-HSD1 in adaption to dietary cholesterol and suggest that hepatic 11β-HSD1 (as opposed to 11β-HSD1 in extra-hepatic tissues) is the main factor regulating BA metabolism. Also, work from this thesis demonstrates 11β-HSD1 is an important regulator of gall bladder emptying and filling, an important component of enterohepatic bile acid recycling. Based on these findings it is anticipated that therapeutic use of 11β-HSD1 inhibitors will result in BA imbalances within the enterohepatic circuit and therefore BA homeostasis. Care must therefore be observed when implementing therapeutic use of 11β-HSD1 inhibitors, with particular focus on patients with cholestasis, Addison’s disease and critically ill patients who already have known BA imbalances in their enterohepatic system.

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Кравець, Олександр Валерійович, В. О. Братушка, Александр Валерьевич Кравец та Oleksandr Valeriiovych Kravets. "Досвід діагностики та лікування поліпів жовчного міхура". Thesis, Сумський державний університет, 2014. http://essuir.sumdu.edu.ua/handle/123456789/36441.

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Нами проведено аналіз історій хвороби 32 хворих, оперованих з приводу поліпів (П) жовчного міхура (ЖМ). Середній вік – 45,1 ± 6,5 років. При цитуванні документа, використовуйте посилання http://essuir.sumdu.edu.ua/handle/123456789/36441

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Chiblen', S. V., I. Yu Olijnyk, and O. V. Tsyhykalo. "Features of the vascular bed anlage in the gall bladder and the cystic duct in human early prenatal ontogenesis." Thesis, БДМУ, 2017. http://dspace.bsmu.edu.ua:8080/xmlui/handle/123456789/16782.

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Москаленко, Роман Андрійович, Роман Андреевич Москаленко, Roman Andriiovych Moskalenko та ін. "Морфологічний аналіз 3 випадків порцелянового жовчного міхура". Thesis, Сумський державний університет, 2016. http://essuir.sumdu.edu.ua/handle/123456789/45117.

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Порцеляновий жовчний міхур є рідкісним проявом хронічних захворювань жовчного міхура, характеризується кальцифікацією своєї стінки і зустрічається у 0,06-0,8% холецистектомій (Palermo M, 2011). За результатами різних досліджень, у 0-62% випадків пацієнти з ПЖМ виникає рак жовчного міхура (Stephen A.E, 2001, Cunningham S.C., 2007). Метою нашої роботи був морфологічний аналіз 3 клінічних випадків порцелянового жовчного міхура.

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Юзько, Р. В. "Топографія печінково-дванадцятипалокишкової звʼязки плодів у третьому триместрі вагітності". Thesis, Сумський державний університет, 2017. http://essuir.sumdu.edu.ua/handle/123456789/64356.

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Вступ. Дослідження літературних джерел виявило нами деякі суперечності, щодо трактування положень про топографію судин печінково-дванадцятипалокишкової звʼязки та характеру кровопостачання позапечінкових жовчних проток.

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Гньотов, М. І., та О. В. Хабаль. "Оптимізація лікувальної тактики холестерозу жовчного міхура під впливом розувастатину, мосаприду та урсодезоксихолієвої кислоти". Thesis, Сумський державний університет, 2017. http://essuir.sumdu.edu.ua/handle/123456789/54846.

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Захворювання жовчного міхура (ЖМ) за частотою випадків поступаються хіба що атеросклерозу. Раннє виявлення та адекватне лікування холестерозу ЖМ має велике клінічне значення внаслідок можливості позитивно вплинути на перебіг захворювання. Мета: вивчення впливу розувастатину, мосаприду та урсодезоксихолієвої кислоти (УДХК) на перебіг хронічного холециститу та холестерозу жовчного міхура у хворих на ІХС, кардіосклероз та ожиріння.

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Mustacchi, Zan. "Incidência de colecistolitíase em Síndrome de Down; aspectos específicos de diagnóstico: genético, clínico e laboratorial." Universidade de São Paulo, 1997. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-06112009-144406/.

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Foi realizado o estudo prospectivo de uma amostra da população portadora de Síndrome de Down que procurou o Departamento de Genética do Hospital Infantil Darcy Vargas,no período de 1959 a 1996. Dos 2816 portadores de Síndrome de Down que compareceram a este Serviço, neste período , a amostra estudada consistiu de 518 pacientes que mantiveram períodos de retomo ambulatorial conforme previsto pelo Protocolo de segmento clínico adotado. O objetivo do estudo foi caracterizar a incidência de colecistopatia litiásica em pacientes portadores de Síndrome de Down . Para desenvolver este estudo, após o diagnóstico citogenético e clínico da Síndrome de Down, procurou-se correlacionar faixa etária dos genitores, uso de anticoncepcionais e principalmente promover a utilização de exames subsidiários específicos que permitiram melhores condições diagnósticas e definição de mecanismos fisiopatológicos eventuais relacionados à colilitiase. Verificou-se a presença de cálculos de vesícula biliar em 27 pacientes ( 3,28%) comparando-se à incidência descrita na população como um todo (0,07%). Estes dados caracterizam a prevalência de cálculos de vesícula biliar em portadores de Síndrome de Down ( a distribuição das probabilidades da frequência de cálculo de vesícula biliar em Sindrome de Down está dentro do intervalo de 95%, entre 3,6% e 7,7%). As correlações realizadas vieram a excluir,na amostra estudada, algumas etiologias comumente descritas para colelitíase e provavelmente vincula o fenômeno da colelitíase a mecanismos fisioembriopatológicos ligados à hipotonia e estase do conteúdo da vesícula biliar( e/ou redução na velocidade do seu esvaziamento). Em consequência torna-se importante propor uma investigação rotineira para esta patologia, com ênfase nos mecanismos fisiopatológicos, prováveis responsáveis pela incidência aumentada na população com Síndrome de Down.<br>The purpose of this study has been the prospective analysis of the Down syndrome population received by the Genetic Department of \"Hospital Infantil Darcy Vargas\", in the period from 1959 to 1996. The Department has received 2816 Down Syndrome patients\' in this period, and this study has selected 518 patients, clinical and cytogenetically diagnosed, which were studied following the specific clinical protocol, in periodical evaluations. The incidence of cholelithiasis was verified and it has been analysed the possible relationships among this disease and parentaI age, contraceptive use and clinical features leading to the discussion of eventual aetiologic mechanisms causing gallstones. It was verified 27 patients affected by Down Syndrome and presenting cholelithiasis in the survey of 518 patients ( 3.28 %) compared to the frequency of 7/10000 in the non Down population ( 0.07% ). The statistical analysis has shown that the distribution of the frequency probabilities have been between 3.6% and 7.7%, These studies have verified that the aetiology of the gallstones in the Down syndrome patients could be related to the hypotonic characteristic of the patients, leading to cholestasis, It is very important the suggestion of routine evaluation for this pathology, related to the eventual aetiologic features.

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Książki na temat "Empyema of Gall Bladder"

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Kapoor, Vinay K., ed. A Pictorial Treatise on Gall Bladder Cancer. Springer Singapore, 2021. http://dx.doi.org/10.1007/978-981-15-5289-2.

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Albores-Saavedra, Jorge. Histological typing of tumours of the gallbladder and extrahepatic bile ducts. 2nd ed. Springer-Verlag, 1991.

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Keshav, Satish, and Alexandra Kent. Gall bladder disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0200.

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The gall bladder is a sac which lies underneath the liver and stores and concentrates bile produced by the liver. As food enters the duodenum, it stimulates the release of cholecystokinin, which in turn stimulates the release of bile, which passes via the cystic duct to the common bile duct, which connects to the duodenum at the sphincter of Oddi. Bile is required in digestion, especially for the emulsification and absorption of fat. Biliary disease can take several forms. Cholelithiasis refers to the presence of gallstones in the gall bladder, whereas choledocholithiasis refers to gallstones in the biliary tree. Cholecystitis is inflammation and infection of the gall bladder. Cholangitis is inflammation and infection of the biliary tree. Sphincter of Oddi dysfunction (SOD) is characterized by symptoms of biliary obstruction, with no structural cause. Other forms of biliary disease are gall bladder polyps, primary biliary cholangitis, and primary sclerosing cholangitis.

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Moran. Liver and Gall Bladder. Lea & Febiger, 1989.

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Feldman, Mark, and Nicholas F. Larusso. Gall Bladder and Bile Ducts. Elsevier Health Sciences, 1997.

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Rela, Mohamed. Liver, Gall Bladder, and Bile Ducts. Oxford University Press, 2023.

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Kapoor, Vinay K. Pictorial Treatise on Gall Bladder Cancer. Springer Singapore Pte. Limited, 2022.

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Intestinal obstruction from gall-stone. s.n., 1985.

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Intestinal obstruction from gall-stone. s.n., 1985.

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Robson, Arthur William Mayo, and Farquhar MacRae. Diseases of the Gall-Bladder and Bile Ducts: Including Gall-Stones. Franklin Classics Trade Press, 2018.

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Części książek na temat "Empyema of Gall Bladder"

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Herling, Andreas W. "Gall Bladder Function." In Drug Discovery and Evaluation: Pharmacological Assays. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-05392-9_59.

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Allen, Derek C. "Gall Bladder Carcinoma." In Histopathology Reporting. Springer London, 2000. http://dx.doi.org/10.1007/978-1-4471-3671-2_8.

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Ertel, Audrey E., David Bentrem, and Daniel E. Abbott. "Gall Bladder Cancer." In Cancer Treatment and Research. Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-34244-3_6.

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McVeigh, Gerard. "Gall Bladder Carcinoma." In Histopathology Reporting. Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-27828-1_9.

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Allen, Derek C. "Gall Bladder Carcinoma." In Histopathology Reporting. Springer London, 2013. http://dx.doi.org/10.1007/978-1-4471-5263-7_8.

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Zatonski, Witold, and Nikolaus Becker. "Gall Bladder — ICD 156." In Atlas of Cancer Mortality in Poland 1975–1979. Springer Berlin Heidelberg, 1988. http://dx.doi.org/10.1007/978-3-642-72607-1_8.

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Merrick, Malcolm V. "Liver and gall-bladder." In Essentials of Nuclear Medicine. Springer London, 1998. http://dx.doi.org/10.1007/978-1-4471-0907-5_10.

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Rushton, D. I. "Liver and Gall Bladder." In Fetal and Neonatal Pathology. Springer London, 1987. http://dx.doi.org/10.1007/978-1-4471-3523-4_17.

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Rushton, D. Ian. "Liver and Gall Bladder." In Fetal and Neonatal Pathology. Springer London, 2001. http://dx.doi.org/10.1007/978-1-4471-3682-8_16.

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Rushton, D. Ian. "Liver and Gall Bladder." In Fetal and Neonatal Pathology. Springer London, 1993. http://dx.doi.org/10.1007/978-1-4471-3802-0_16.

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Streszczenia konferencji na temat "Empyema of Gall Bladder"

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Aditya Chowdary, Tripuraneni Venkata, and Ramalingam Trivikraman. "IDDF2018-ABS-0045 Incidental gall bladder cancer in laparoscopic cholecystectomy." In International Digestive Disease Forum (IDDF) 2018, Hong Kong, 9–10 June 2018. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2018. http://dx.doi.org/10.1136/gutjnl-2018-iddfabstracts.81.

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Kokaj, Jahja O. "High-speed photography during laser-based gall bladder stone lithotripsy." In 24th International Congress on High-Speed Photography and Photonics, edited by Kazuyoshi Takayama, Tsutomo Saito, Harald Kleine, and Eugene V. Timofeev. SPIE, 2001. http://dx.doi.org/10.1117/12.424256.

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Singh, V. R. "Study of Gall Bladder Stones as a New Piezoelectric Sensor Material." In 2008 IEEE International Workshop on Medical Measurement and Applications (MeMeA '08). IEEE, 2008. http://dx.doi.org/10.1109/memea.2008.4543008.

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Kumar, Abhyanand, Lokesh Phagna, Sagar Rawat, and Neha Gupta. "Analysis of Gall Bladder & Kidney Stone Using Spectroscopic Technique: A Review." In 2023 4th International Conference on Signal Processing and Communication (ICSPC). IEEE, 2023. http://dx.doi.org/10.1109/icspc57692.2023.10125670.

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Kapoor, Vinay K., Vinita Agrawal, Amit Goel, Narendra Krishnani, Rakesh Pandey, and Suraksha Agrawal. "Abstract A26: CEA and CA 19‐9 as biomarkers for prevention of gall bladder cancer." In Abstracts: AACR International Conference on Frontiers in Cancer Prevention Research‐‐ Dec 6–9, 2009; Houston, TX. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1940-6207.prev-09-a26.

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Shemis, M. A., M. A. Gondal, and A. A. I. Khalil. "Development of Laser Induced Breakdown Spectrometer for detection of carcinogenic metals in gall bladder stones." In 2014 11th Annual High-capacity Optical Networks and Emerging/Enabling Technologies (HONET). IEEE, 2014. http://dx.doi.org/10.1109/honet.2014.7029389.

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Vanitha, M., T. R. Ganesh Babu, R. Praveena, and M. Sathyapriya. "Segmentation and classification of Gallstone in ultrasound images of gall bladder using Support Vector Machine." In 2022 13th International Conference on Computing Communication and Networking Technologies (ICCCNT). IEEE, 2022. http://dx.doi.org/10.1109/icccnt54827.2022.9984361.

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AL-zuwainni, Sama. "CHARACTERIZATION OF SOME BACTERIA AND MEASUREMENT OF IL12 CONCENTERATION IN PATIENTS WITH GALL BLADDER INFLAMMATION." In International Conference of Natural Science 2017. College of Basic Education, Charmo University, Chamchamal, Sulaimani/Iraq, 2018. http://dx.doi.org/10.31530/17017.

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Freise, NF, A. Kunstein, V. Keitel, B. Jensen, O. Böhmer, and D. Häussinger. "Gall Bladder Wall Thickening (GBWT) im Rahmen eines Dengue-Schock-Syndroms – vollständige Regredienz unter konservativem Procedere." In Interdisziplinärer Kongress | Ultraschall 2019 – 43. Dreiländertreffen DEGUM | ÖGUM | SGUM. Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1695961.

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Nandagopal, Lakshminarayanan, Martin J. Heslin, John R. Porterfield, et al. "Abstract 3283: Adjuvant therapy associated with improved survival in gall bladder cancer: A single institution retrospective study." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3283.

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