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Artykuły w czasopismach na temat „Fatty acid metabolism”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Harwood, J. L. "Fatty Acid Metabolism." Annual Review of Plant Physiology and Plant Molecular Biology 39, no. 1 (1988): 101–38. http://dx.doi.org/10.1146/annurev.pp.39.060188.000533.

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de las Fuentes, Lisa, Pilar Herrero, Linda R. Peterson, Daniel P. Kelly, Robert J. Gropler, and Víctor G. Dávila-Román. "Myocardial Fatty Acid Metabolism." Hypertension 41, no. 1 (2003): 83–87. http://dx.doi.org/10.1161/01.hyp.0000047668.48494.39.

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SPIECKERMANN, P., J. HUTTER, and C. ALVES. "Myocardial fatty acid metabolism." Journal of Molecular and Cellular Cardiology 18 (1986): 68. http://dx.doi.org/10.1016/s0022-2828(86)80233-4.

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Katafuchi, Takeshi, and Makoto Makishima. "Fatty Acid Metabolism during Exercise." Journal of Nihon University Medical Association 80, no. 1 (2021): 15–19. http://dx.doi.org/10.4264/numa.80.1_15.

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SATO, Ryuichiro. "Fatty Acid Metabolism and SREBP." Oleoscience 1, no. 11 (2001): 1065–72. http://dx.doi.org/10.5650/oleoscience.1.1065.

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LONDON, BARRY. "Fatty Acid Metabolism and Arrhythmias." Journal of Cardiovascular Electrophysiology 15, no. 11 (2004): 1317–18. http://dx.doi.org/10.1046/j.1540-8167.2004.04576.x.

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7

Giovannini, M., C. Agostoni, G. Biasucci, et al. "Fatty acid metabolism in phenylketonuria." European Journal of Pediatrics 155, S1 (1996): S132—S135. http://dx.doi.org/10.1007/pl00014230.

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Innis, Sheila M., Howard Sprecher, David Hachey, John Edmond, and Robert E. Anderson. "Neonatal polyunsaturated fatty acid metabolism." Lipids 34, no. 2 (1999): 139–49. http://dx.doi.org/10.1007/s11745-999-0348-x.

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9

Handler, Jeffrey A., and Ronald G. Thurman. "Fatty acid-dependent ethanol metabolism." Biochemical and Biophysical Research Communications 133, no. 1 (1985): 44–51. http://dx.doi.org/10.1016/0006-291x(85)91839-x.

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Jensen, Michael D., Karin Ekberg, and Bernard R. Landau. "Lipid metabolism during fasting." American Journal of Physiology-Endocrinology and Metabolism 281, no. 4 (2001): E789—E793. http://dx.doi.org/10.1152/ajpendo.2001.281.4.e789.

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These studies were conducted to understand the relationship between measures of systemic free fatty acid (FFA) reesterification and regional FFA, glycerol, and triglyceride metabolism during fasting. Indirect calorimetry was used to measure fatty acid oxidation in six men after a 60-h fast. Systemic and regional (splanchnic, renal, and leg) FFA ([3H]palmitate) and glycerol ([3H]glycerol) kinetics, as well as splanchnic triglyceride release, were measured. The rate of systemic FFA reesterification was 366 ± 93 μmol/min, which was greater ( P < 0.05) than splanchnic triglyceride fatty acid ou

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11

Stinkens, R., G. H. Goossens, J. W. E. Jocken, and E. E. Blaak. "Targeting fatty acid metabolism to improve glucose metabolism." Obesity Reviews 16, no. 9 (2015): 715–57. http://dx.doi.org/10.1111/obr.12298.

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12

Hulver, Matthew W., Jason R. Berggren, Ronald N. Cortright, et al. "Skeletal muscle lipid metabolism with obesity." American Journal of Physiology-Endocrinology and Metabolism 284, no. 4 (2003): E741—E747. http://dx.doi.org/10.1152/ajpendo.00514.2002.

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The objectives of this study were to 1) examine skeletal muscle fatty acid oxidation in individuals with varying degrees of adiposity and 2) determine the relationship between skeletal muscle fatty acid oxidation and the accumulation of long-chain fatty acyl-CoAs. Muscle was obtained from normal-weight [ n = 8; body mass index (BMI) 23.8 ± 0.58 kg/m2], overweight/obese ( n = 8; BMI 30.2 ± 0.81 kg/m2), and extremely obese ( n = 8; BMI 53.8 ± 3.5 kg/m2) females undergoing abdominal surgery. Skeletal muscle fatty acid oxidation was assessed in intact muscle strips. Long-chain fatty acyl-CoA conce

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13

Yamamoto, Tsunehisa, and Motoaki Sano. "Deranged Myocardial Fatty Acid Metabolism in Heart Failure." International Journal of Molecular Sciences 23, no. 2 (2022): 996. http://dx.doi.org/10.3390/ijms23020996.

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The heart requires fatty acids to maintain its activity. Various mechanisms regulate myocardial fatty acid metabolism, such as energy production using fatty acids as fuel, for which it is known that coordinated control of fatty acid uptake, β-oxidation, and mitochondrial oxidative phosphorylation steps are important for efficient adenosine triphosphate (ATP) production without unwanted side effects. The fatty acids taken up by cardiomyocytes are not only used as substrates for energy production but also for the synthesis of triglycerides and the replacement reaction of fatty acid chains in cel

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14

Xu, Huan, Yanbo Chen, Meng Gu, et al. "Fatty Acid Metabolism Reprogramming in Advanced Prostate Cancer." Metabolites 11, no. 11 (2021): 765. http://dx.doi.org/10.3390/metabo11110765.

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Prostate cancer (PCa) is a carcinoma in which fatty acids are abundant. Fatty acid metabolism is rewired during PCa development. Although PCa can be treated with hormone therapy, after prolonged treatment, castration-resistant prostate cancer can develop and can lead to increased mortality. Changes to fatty acid metabolism occur systemically and locally in prostate cancer patients, and understanding these changes may lead to individualized treatments, especially in advanced, castration-resistant prostate cancers. The fatty acid metabolic changes are not merely reflective of oncogenic activity,

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15

KISHINO, Shigenobu, and Jun OGAWA. "Novel Fatty Acid Metabolism of Lactic Acid Bacteria:Application to Functional Fatty Acid Production and Gut Lipid Metabolism Control." KAGAKU TO SEIBUTSU 51, no. 11 (2013): 738–44. http://dx.doi.org/10.1271/kagakutoseibutsu.51.738.

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16

Koundouros, Nikos, and George Poulogiannis. "Reprogramming of fatty acid metabolism in cancer." British Journal of Cancer 122, no. 1 (2019): 4–22. http://dx.doi.org/10.1038/s41416-019-0650-z.

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AbstractA common feature of cancer cells is their ability to rewire their metabolism to sustain the production of ATP and macromolecules needed for cell growth, division and survival. In particular, the importance of altered fatty acid metabolism in cancer has received renewed interest as, aside their principal role as structural components of the membrane matrix, they are important secondary messengers, and can also serve as fuel sources for energy production. In this review, we will examine the mechanisms through which cancer cells rewire their fatty acid metabolism with a focus on four main

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17

Cheng, Qiang, Zhongxuan Li, Jing Zhang, et al. "Soybean Oil Regulates the Fatty Acid Synthesis Ⅱ System of Bacillus amyloliquefaciens LFB112 by Activating Acetyl-CoA Levels." Microorganisms 11, no. 5 (2023): 1164. http://dx.doi.org/10.3390/microorganisms11051164.

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[Background] Bacillus LFB112 is a strain of Bacillus amyloliquefaciens screened in our laboratory. Previous studies found that it has a strong ability for fatty acid metabolism and can improve the lipid metabolism of broilers when used as feed additives. [Methods] This study aimed to confirm the fatty acid metabolism of Bacillus LFB112. Sterilized soybean oil (SSO) was added to the Beef Peptone Yeast (BPY) medium, and its effect on fatty acid content in the supernatant and bacteria, as well as expression levels of genes related to fatty acid metabolism, were studied. The control group was the

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18

Burdge, G. C. "Polyunsaturated fatty acid intakes and -linolenic acid metabolism." American Journal of Clinical Nutrition 93, no. 3 (2010): 665–66. http://dx.doi.org/10.3945/ajcn.110.008169.

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19

Luo, Yuting, Hanbing Wang, Baorui Liu, and Jia Wei. "Fatty Acid Metabolism and Cancer Immunotherapy." Current Oncology Reports 24, no. 5 (2022): 659–70. http://dx.doi.org/10.1007/s11912-022-01223-1.

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20

Strandvik, Birgitta. "Fatty Acid Metabolism in Cystic Fibrosis." New England Journal of Medicine 350, no. 6 (2004): 605–7. http://dx.doi.org/10.1056/nejme038217.

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21

Muoio, Deborah M. "Metabolism and Vascular Fatty Acid Transport." New England Journal of Medicine 363, no. 3 (2010): 291–93. http://dx.doi.org/10.1056/nejmcibr1005397.

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22

Eckardt, Nancy A. "Tocopherols and ER Fatty Acid Metabolism." Plant Cell 20, no. 2 (2008): 246. http://dx.doi.org/10.1105/tpc.108.200212.

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23

Buchan, Gregory J., Gustavo Bonacci, Marco Fazzari, Sonia R. Salvatore, and Stacy Gelhaus Wendell. "Nitro-fatty acid formation and metabolism." Nitric Oxide 79 (September 2018): 38–44. http://dx.doi.org/10.1016/j.niox.2018.07.003.

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24

Strandvik, Birgitta. "Fatty acid metabolism in cystic fibrosis." Prostaglandins, Leukotrienes and Essential Fatty Acids 83, no. 3 (2010): 121–29. http://dx.doi.org/10.1016/j.plefa.2010.07.002.

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25

Poulsen, Lars la Cour, Majken Siersbæk, and Susanne Mandrup. "PPARs: Fatty acid sensors controlling metabolism." Seminars in Cell & Developmental Biology 23, no. 6 (2012): 631–39. http://dx.doi.org/10.1016/j.semcdb.2012.01.003.

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26

Saleh, Jumana, Allan D. Sniderman, and Katherine Cianflone. "Regulation of plasma fatty acid metabolism." Clinica Chimica Acta 286, no. 1-2 (1999): 163–80. http://dx.doi.org/10.1016/s0009-8981(99)00099-6.

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27

Semenkovich, C. "Fatty Acid Metabolism and Vascular Disease." Trends in Cardiovascular Medicine 14, no. 2 (2004): 72–76. http://dx.doi.org/10.1016/j.tcm.2003.12.004.

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28

Walch, Laurence, Alenka Čopič, and Catherine L. Jackson. "Fatty Acid Metabolism Meets Organelle Dynamics." Developmental Cell 32, no. 6 (2015): 657–58. http://dx.doi.org/10.1016/j.devcel.2015.03.008.

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Kushwaha, Priyanka, Michael J. Wolfgang, and Ryan C. Riddle. "Fatty acid metabolism by the osteoblast." Bone 115 (October 2018): 8–14. http://dx.doi.org/10.1016/j.bone.2017.08.024.

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30

Chen, Yuanying, and Peng Li. "Fatty acid metabolism and cancer development." Science Bulletin 61, no. 19 (2016): 1473–79. http://dx.doi.org/10.1007/s11434-016-1129-4.

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31

Currie, Erin, Almut Schulze, Rudolf Zechner, Tobias C. Walther, and Robert V. Farese. "Cellular Fatty Acid Metabolism and Cancer." Cell Metabolism 18, no. 2 (2013): 153–61. http://dx.doi.org/10.1016/j.cmet.2013.05.017.

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32

Liu, Bin, and Zhiyu Dai. "Fatty Acid Metabolism in Endothelial Cell." Genes 13, no. 12 (2022): 2301. http://dx.doi.org/10.3390/genes13122301.

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The endothelium is a monolayer of cells lining the inner blood vessels. Endothelial cells (ECs) play indispensable roles in angiogenesis, homeostasis, and immune response under normal physiological conditions, and their dysfunction is closely associated with pathologies such as cardiovascular diseases. Abnormal EC metabolism, especially dysfunctional fatty acid (FA) metabolism, contributes to the development of many diseases including pulmonary hypertension (PH). In this review, we focus on discussing the latest advances in FA metabolism in ECs under normal and pathological conditions with an

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33

Ruidera, E., C. E. Irazu, P. R. Rajagopalan, J. K. Orak, C. T. Fitts, and I. Singh. "Fatty acid metabolism in renal ischemia." Lipids 23, no. 9 (1988): 882–84. http://dx.doi.org/10.1007/bf02536209.

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34

van Roermund, C. W. T., H. R. Waterham, L. Ijlst, and R. J. A. Wanders. "Fatty acid metabolism in Saccharomyces cerevisiae." Cellular and Molecular Life Sciences (CMLS) 60, no. 9 (2003): 1838–51. http://dx.doi.org/10.1007/s00018-003-3076-x.

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35

Spener, F. "Fatty Acid Metabolism and its Regulation." Chemistry and Physics of Lipids 36, no. 4 (1985): 395. http://dx.doi.org/10.1016/0009-3084(85)90047-7.

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Prato, Stefano Del, Sabina Marchetto, Antonino Pipitone, Milena Zanon, Saula Vigili De Kreutzenberg, and Antonio Tiengo. "Metformin and free fatty acid metabolism." Diabetes/Metabolism Reviews 11, S1 (1995): S33—S41. http://dx.doi.org/10.1002/dmr.5610110506.

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Ahern, Kevin. "Fatty acid metabolism: A metabolic verse." Biochemistry and Molecular Biology Education 41, no. 5 (2013): 362. http://dx.doi.org/10.1002/bmb.20724.

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Masarwi, Majdi, Abigail DeSchiffart, Justin Ham, and Michaela R. Reagan. "Multiple Myeloma and Fatty Acid Metabolism." JBMR Plus 3, no. 3 (2019): e10173. http://dx.doi.org/10.1002/jbm4.10173.

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Yao, Jiangwei, and Charles O. Rock. "Exogenous fatty acid metabolism in bacteria." Biochimie 141 (October 2017): 30–39. http://dx.doi.org/10.1016/j.biochi.2017.06.015.

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BRUNK, DOUG. "Gender Impacts Myocardial Fatty Acid Metabolism." Ob.Gyn. News 42, no. 21 (2007): 30. https://doi.org/10.1016/s0029-7437(07)70929-1.

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41

Li, Chunlei, Lilong Zhang, Zhendong Qiu, Wenhong Deng, and Weixing Wang. "Key Molecules of Fatty Acid Metabolism in Gastric Cancer." Biomolecules 12, no. 5 (2022): 706. http://dx.doi.org/10.3390/biom12050706.

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Fatty acid metabolism is closely linked to the progression of gastric cancer (GC), a very aggressive and life-threatening tumor. This study examines linked molecules, such as Sterol Regulatory Element-Binding Protein 1 (SREBP1), ATP Citrate Lyase (ACLY), Acetyl-CoA Synthases (ACSs), Acetyl-CoA Carboxylase (ACC), Fatty Acid Synthase (FASN), Stearoyl-CoA Desaturase 1 (SCD1), CD36, Fatty Acid Binding Proteins (FABPs), and Carnitine palmitoyltransferase 1 (CPT1), as well as their latest studies and findings in gastric cancer to unveil its core mechanism. The major enzymes of fatty acid de novo syn

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42

Lopaschuk, Gary D., John R. Ussher, Clifford D. L. Folmes, Jagdip S. Jaswal, and William C. Stanley. "Myocardial Fatty Acid Metabolism in Health and Disease." Physiological Reviews 90, no. 1 (2010): 207–58. http://dx.doi.org/10.1152/physrev.00015.2009.

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There is a constant high demand for energy to sustain the continuous contractile activity of the heart, which is met primarily by the β-oxidation of long-chain fatty acids. The control of fatty acid β-oxidation is complex and is aimed at ensuring that the supply and oxidation of the fatty acids is sufficient to meet the energy demands of the heart. The metabolism of fatty acids via β-oxidation is not regulated in isolation; rather, it occurs in response to alterations in contractile work, the presence of competing substrates (i.e., glucose, lactate, ketones, amino acids), changes in hormonal m

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43

Alves‐Bezerra, Michele, and David E. Cohen. "Triglyceride Metabolism in the Liver." Comprehensive Physiology 8, no. 1 (2018): 1–22. https://doi.org/10.1002/j.2040-4603.2018.tb00008.x.

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ABSTRACTTriglyceride molecules represent the major form of storage and transport of fatty acids within cells and in the plasma. The liver is the central organ for fatty acid metabolism. Fatty acids accrue in liver by hepatocellular uptake from the plasma and by de novo biosynthesis. Fatty acids are eliminated by oxidation within the cell or by secretion into the plasma within triglyceride‐rich very low‐density lipoproteins. Notwithstanding high fluxes through these pathways, under normal circumstances the liver stores only small amounts of fatty acids as triglycerides. In the setting of overnu

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44

Li, Xiaoting, and Xukun Bi. "Integrated Control of Fatty Acid Metabolism in Heart Failure." Metabolites 13, no. 5 (2023): 615. http://dx.doi.org/10.3390/metabo13050615.

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Disrupted fatty acid metabolism is one of the most important metabolic features in heart failure. The heart obtains energy from fatty acids via oxidation. However, heart failure results in markedly decreased fatty acid oxidation and is accompanied by the accumulation of excess lipid moieties that lead to cardiac lipotoxicity. Herein, we summarized and discussed the current understanding of the integrated regulation of fatty acid metabolism (including fatty acid uptake, lipogenesis, lipolysis, and fatty acid oxidation) in the pathogenesis of heart failure. The functions of many enzymes and regu

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45

Atshaves, Barbara P., Stephen M. Storey, Huan Huang, and Friedhelm Schroeder. "Liver fatty acid binding protein expression enhances branched-chain fatty acid metabolism." Molecular and Cellular Biochemistry 259, no. 1/2 (2004): 115–29. http://dx.doi.org/10.1023/b:mcbi.0000021357.97765.f2.

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46

Worgall, Tilla S., and Richard J. Deckelbaum. "Fatty acids: links between genes involved in fatty acid and cholesterol metabolism." Current Opinion in Clinical Nutrition and Metabolic Care 2, no. 2 (1999): 127–33. http://dx.doi.org/10.1097/00075197-199903000-00006.

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47

Tamaki, Nagara, and Masahide Kawamoto. "The use of iodinated free fatty acids for assessing fatty acid metabolism." Journal of Nuclear Cardiology 1, S2 (1994): S72—S78. http://dx.doi.org/10.1007/bf02940072.

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Tanaka, Shota, Akane Hirota, Yoshiaki Okada, Masanori Obana та Yasushi Fujio. "Fatty acid metabolism suppresses neonatal cardiomyocyte proliferation by increasing PDK4 and HMGCS2 expression through PPARδ". PLOS One 20, № 5 (2025): e0318178. https://doi.org/10.1371/journal.pone.0318178.

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Cardiomyocytes lose their capacity to regenerate immediately after birth. Simultaneously, cardiomyocytes change energy metabolism from glycolysis to oxidative phosphorylation, especially using fatty acids. Accumulating evidence has revealed that fatty acid metabolism weakens the proliferative ability of cardiomyocytes. However, its underlying molecular mechanism remains unclear. In this study, we investigated how fatty acid metabolism contributes to cell cycle regulation in neonatal cardiomyocytes. Cultured neonatal rat cardiomyocytes (NRCMs) were treated with a fatty acid mixture (FA) consist

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49

Chen, Jieqing, Bosheng Cai, Changxu Tian, et al. "RNA Sequencing (RNA-Seq) Analysis Reveals Liver Lipid Metabolism Divergent Adaptive Response to Low- and High-Salinity Stress in Spotted Scat (Scatophagus argus)." Animals 13, no. 9 (2023): 1503. http://dx.doi.org/10.3390/ani13091503.

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Spotted scat (Scatophagus argus) can tolerate a wide range of salinity fluctuations. It is a good model for studying environmental salinity adaptation. Lipid metabolism plays an important role in salinity adaptation in fish. To elucidate the mechanism of lipid metabolism in the osmoregulation, the liver transcriptome was analyzed after 22 d culture with a salinity of 5 ppt (Low-salinity group: LS), 25 ppt (Control group: Ctrl), and 35 ppt (High-salinity group: HS) water by using RNA sequencing (RNA-seq) in spotted scat. RNA-seq analysis showed that 1276 and 2768 differentially expressed genes

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Bonen, Arend, Shannon E. Campbell, Carley R. Benton, et al. "Regulation of fatty acid transport by fatty acid translocase/CD36." Proceedings of the Nutrition Society 63, no. 2 (2004): 245–49. http://dx.doi.org/10.1079/pns2004331.

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Fatty acid (FA) translocase (FAT)/CD36 is a key protein involved in regulating the uptake of FA across the plasma membrane in heart and skeletal muscle. A null mutation of FAT/CD36 reduces FA uptake rates and metabolism, while its overexpression increases FA uptake rates and metabolism. FA uptake into the myocyte may be regulated (a) by altering the expression of FAT/CD36, thereby increasing the plasmalemmal content of this protein (i.e. streptozotocin-induced diabetes, chronic muscle stimulation), or (b) by relocating this protein to the plasma membrane, without altering its expression (i.e.

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