Gotowa bibliografia na temat „Fillagrin”

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Artykuły w czasopismach na temat "Fillagrin"

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Kochergin, N. G., and N. G. Kochergin. "Xerosis and xemose." Vestnik dermatologii i venerologii 87, no. 4 (2011): 121–24. http://dx.doi.org/10.25208/vdv1063.

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This article covers current concepts of the barrier function of skin, major pathophysiological mechanisms of skin xerosis development, its fillagrin-dependent mutations and major clinical signs by the example of atopic dermatitis. The author gives recommendations for using moisturizers on the basis of Uriage Thermal Water comprising Xemose Cream, Xemose Cerate and Xemose Gel-Cream for efficient reduction of extra skin dryness.
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Uchi, H., M. Kiyomatsu-Oda, and M. Furue. "466 The tryptophan photoproduct FICZ improves atopic dermatitis by enhancement of fillagrin production." Journal of Investigative Dermatology 137, no. 10 (2017): S272. http://dx.doi.org/10.1016/j.jid.2017.07.662.

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Sakshi, Sharma* Anchal Sankhyan Dr. Dev Prakash Dahiya Shelly Indu. "A Review of Eczema: Current Therapies and Emerging Treatments." International Journal of Pharmaceutical Sciences 3, no. 5 (2025): 2718–26. https://doi.org/10.5281/zenodo.15440679.

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Atopic dermatitis, commonly known as eczema, is a chronic relapsing skin disease characterized by pruritus, disruption of the epidermal barrier function, and immunoglobulin E-mediated sensitization to food and environmental allergens, which can lead to morbidity. Eczema is not always easy to control and every physician should know the essential principles of treatment. Symptoms, such as inflamed papules, plaques, and itching, and associated consequences, such as sleep disturbances, can have a significant impact on the quality of life of the patient and his family. Basic pharmacological treatme
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Puppels, Gerwin, Claudio Nico, Dhanis Lad, et al. "Strategy for neonatal discrimination between Filaggrin-wildtype neonates and Fillagrin loss of function mutation carriers for the purpose of preventative AD-treatment." Journal of Allergy and Clinical Immunology 151, no. 2 (2023): AB151. http://dx.doi.org/10.1016/j.jaci.2022.12.471.

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Circiumaru, A., Y. Kisten, M. Hansson, et al. "SAT0584 SPECIFIC ACPA REACTIVITIES AND INFLAMMATORY BIOMARKERS ALONG WITH ULTRASOUND TENOSYNOVITIS ARE ASSOCIATED WITH ARTHRITIS ONSET IN A POPULATION AT RISK FOR RHEUMATOID ARTHRITIS." Annals of the Rheumatic Diseases 79, Suppl 1 (2020): 1251.2–1251. http://dx.doi.org/10.1136/annrheumdis-2020-eular.5578.

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Background:Anti-citrullinated protein antibodies (ACPA) are characteristic markers for rheumatoid arthritis (RA), developing years before disease onset. Early clinical and biological biomarkers could provide useful information on the onset of RA in predisposed individuals.Objectives:The aim of the study was to investigate whether ACPA along with inflammatory markers and musculoskeletal ultrasound changes could predict arthritis development in individuals at risk for RA.Methods:ACPA-positive individuals with musculoskeletal complaints were referred from primary care to a rheumatology clinic, re
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Becker drs., M., P. Koelink, W. Bemelman, G. D'Haens, C. Buskens, and M. Wildenberg. "P107 Decreased epithelial keratinization contributes to defective wound healing in Crohn's disease fistula." Journal of Crohn's and Colitis 17, Supplement_1 (2023): i271—i272. http://dx.doi.org/10.1093/ecco-jcc/jjac190.0237.

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Abstract Background Up to 30% of Crohn's Disease (CD) patients suffer from perianal fistula creating a high disease burden. Treating CD fistula is challenging compared to cryptoglandular fistula, which have a significantly better wound healing response. Previous studies suggested epithelial to mesenchymal transition as an inducer of fistula formation, as markers were observed in fistula tracts. However, most studies compared CD fistula to normal mucosa, thus disregarding whether any observed difference was in fact a sign of healing (as CD fistula will have more active healing than uninjured mu
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Mohammad, fm fr alajmi. "THE SKIN BARRIER FUNCTION AND THE DEVELOPMENT OF DERMATITIS." March 15, 2018. https://doi.org/10.5281/zenodo.1237454.

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The skin barrier is a very important element of the body as it plays crucial roles in the immune surveillance and epidermal homeostasis, and in stopping entrance of microorganisms and allergens. The capacity of the skin to protect the body relies on its structure that includes different layers of cells containing components such proteins and lipids organized in such a way that they provide a tight and strong structure to the skin. Due to various factors, impairments of the structural integrity of the skin occur and compromise its barrier functions provoking thereby immune responses that lead t
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Al Shaqaq, Azhar Abdullah, Lucydx Li, Daniele Merico, and Julia Upton. "Novel FLG mutation associated with severe atopy." LymphoSign Journal, March 12, 2024. http://dx.doi.org/10.14785/lymphosign-2024-0001.

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ABSTRACT
 Background: The prevalence of atopic disease, which consists of conditions such as atopic dermatitis, allergies, and asthma, has been on the rise in recent decades. In children, atopic dermatitis often acts as an initial manifestation of atopic disease and frequently precedes the development of food allergies, asthma, and allergic rhinitis. Mutations in the FLG gene, encoding the fillagrin precursor profillagrin, serve as a genetic risk factor for these diseases. Approximately 25-50% of individuals with atopic dermatitis carry FLG mutations. It has been proposed that FLG mutatio
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Вербенко, Д. А., А. Э. Карамова, В. В. Чикин, et al. "Mutacii 2282del4, R501X, R2447X, S3247X fillagrina pri atopicheskom dermatite." Вестник Российского государственного медицинского университета, no. 2024(1) (February 2024). http://dx.doi.org/10.24075/vrgmu.2024.006.

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Atopicheskij dermatit (AtD) — shiroko rasprostranennoe mul'tifaktornoe geneticheski determinirovannoe vospalitel'noe zabolevanie kozhi, obuslovlennoe sredi prochih prichin narusheniem funkcij epidermal'nogo bar'era. Nulevye mutacii gena filaggrina — vazhnogo komponenta sistemy natural'nogo uvlazhnyayushchego faktora, privodyashchie k otsutstviyu vyrabotki polnocennogo belka-predshestvennika, associirovany s AtD. Cel' issledovaniya — ocenit' chastotu naibolee rasprostranennyh v evropejskih populyaciyah nulevyh mutacij gena filaggrina 2282delACTG (rs558269137), R501X (rs61816761), S3247X (rs1505
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Rozprawy doktorskie na temat "Fillagrin"

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Clarenfjord, Johan, and Christian Göransson. "Säker fillagring i hemmiljö : Hur säkrar man upp fillagring i en hemmiljö?" Thesis, Linnéuniversitetet, Institutionen för datavetenskap (DV), 2015. http://urn.kb.se/resolve?urn=urn:nbn:se:lnu:diva-46831.

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I det här arbetet har författarna valt att fokusera på lagringssäkerhet ur synvinkelnhemmiljö. För att kunna ge målgruppen någon form av riktlinje definieras olika kravpå säkerhet och lagring från författarnas sida att utgå från vid diskussion och arbetetsupplägg. Arbetet inriktar sig på vad det finns för möjligheter för att kunna säkra uppde olika formerna av lagringsmöjligheter. Är det möjligt att göra? Vilka källor kanman förvänta sig att målgruppen använder för att lösa de här problemen? Genom attstudera tidigare forskning och göra omfattande dokumentstudier kommer författarnafram till vad
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Hansen, Peter, and Olov Norell. "DistroFS: En lösning för distribuerad lagring av filer." Thesis, University of Gävle, Department of Mathematics, Natural and Computer Sciences, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:hig:diva-177.

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<p>Nuvarande implementationer av distribuerade hashtabeller (DHT) har en begränsad storlek för data som kan lagras, som t.ex. OpenDHTs datastorleks gräns på 1kByte. Är det möjligt att lagra filer större än 1kByte med DHT-tekniken? Finns det någon lösning för att skydda de data som lagrats utan att försämra prestandan? Vår lösning var att utveckla en klient- och servermjukvara. Mjukvaran använder sig av DHT tekniken för att dela upp filer och distribuera delarna över ett serverkluster. För att se om mjukvaran fungerade som tänkt, gjorde vi ett test utifrån de inledande frågorna. Testet visade a
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