Gotowe bibliografie tematyczne / GFAP/Gfap

Spis treści

  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Książki
  4. Części książek
  5. Streszczenia konferencji
  6. Raporty organizacyjne

Gotowa bibliografia na temat „GFAP/Gfap”

Autor: Grafiati
Data publikacji: 2 czerwca 2025
Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "GFAP/Gfap"

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Perng, Ming-Der, Shu-Fang Wen, Terry Gibbon та ін. "Glial Fibrillary Acidic Protein Filaments Can Tolerate the Incorporation of Assembly-compromised GFAP-δ, but with Consequences for Filament Organization and αB-Crystallin Association". Molecular Biology of the Cell 19, № 10 (2008): 4521–33. http://dx.doi.org/10.1091/mbc.e08-03-0284.

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The glial fibrillary acidic protein (GFAP) gene is alternatively spliced to give GFAP-α, the most abundant isoform, and seven other differentially expressed transcripts including GFAP-δ. GFAP-δ has an altered C-terminal domain that renders it incapable of self-assembly in vitro. When titrated with GFAP-α, assembly was restored providing GFAP-δ levels were kept low (∼10%). In a range of immortalized and transformed astrocyte derived cell lines and human spinal cord, we show that GFAP-δ is naturally part of the endogenous intermediate filaments, although levels were low (∼10%). This suggests tha
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Bernardos, Rebecca L., and Pamela A. Raymond. "GFAP transgenic zebrafish." Gene Expression Patterns 6, no. 8 (2006): 1007–13. http://dx.doi.org/10.1016/j.modgep.2006.04.006.

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Messing, Albee, and Michael Brenner. "GFAP at 50." ASN Neuro 12 (January 2020): 175909142094968. http://dx.doi.org/10.1177/1759091420949680.

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Fifty years have passed since the discovery of glial fibrillary acidic protein (GFAP) by Lawrence Eng and colleagues. Now recognized as a member of the intermediate filament family of proteins, it has become a subject for study in fields as diverse as structural biology, cell biology, gene expression, basic neuroscience, clinical genetics and gene therapy. This review covers each of these areas, presenting an overview of current understanding and controversies regarding GFAP with the goal of stimulating continued study of this fascinating protein.
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Eng, Lawrence F., and Roopa S. Ghirnikar. "GFAP and Astrogliosis." Brain Pathology 4, no. 3 (1994): 229–37. http://dx.doi.org/10.1111/j.1750-3639.1994.tb00838.x.

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Brenner, Michael, and Albee Messing. "GFAP Transgenic Mice." Methods 10, no. 3 (1996): 351–64. http://dx.doi.org/10.1006/meth.1996.0113.

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Shafit-Zagardo, B., C. Peterson, and J. E. Goldman. "INCREASES IN GFAP AND GFAP mRNA IN BRINDLED MOUSE CNS." Journal of Neuropathology and Experimental Neurology 46, no. 3 (1987): 361. http://dx.doi.org/10.1097/00005072-198705000-00095.

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Galou, M., E. Colucci-Guyon, D. Ensergueix, et al. "Disrupted glial fibrillary acidic protein network in astrocytes from vimentin knockout mice." Journal of Cell Biology 133, no. 4 (1996): 853–63. http://dx.doi.org/10.1083/jcb.133.4.853.

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Glial fibrillary acidic protein (GFAP) is an intermediate filament protein expressed predominantly in astrocytes. The study of its expression in the astrocyte lineage during development and in reactive astrocytes has revealed an intricate relationship with the expression of vimentin, another intermediate filament protein widely expressed in embryonic development. these findings suggested that vimentin could be implicated in the organization of the GFAP network. To address this question, we have examined GFAP expression and network formation in the recently generated vimentin knockout (Vim-) mi
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Zoltewicz, J. Susie, Dancia Scharf, Boxuan Yang, Aarti Chawla, Kimberly J. Newsom, and Lijuan Fang. "Characterization of Antibodies that Detect Human GFAP after Traumatic Brain Injury." Biomarker Insights 7 (January 2012): BMI.S9873. http://dx.doi.org/10.4137/bmi.s9873.

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After traumatic brain injury (TBI), glial fibrillary acidic protein (GFAP) and other brain-derived proteins and their breakdown products are released into biofluids such as CSF and blood. Recently, a sandwich ELISA was constructed that measured GFAP concentrations in CSF or serum from human mild-moderate TBI patients. The goals of the present study were to characterize the same two antibodies used in this ELISA, and to determine which GFAP bands are detected by this antibody combination. Here, both antibodies recognized GFAP specifically in human brain and post-TBI CSF in a cluster of bands ra
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Chekanova, Ekaterina O., Аlla А. Shabalina, Taras O. Simaniv, et al. "Relapsing Autoimmune GFAP Astrocytopathy: Case Report." Annals of Clinical and Experimental Neurology 17, no. 4 (2024): 89–96. http://dx.doi.org/10.54101/acen.2023.4.11.

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Introduction. Glial fibrillary acidic protein (GFAP) is the main component of intermediate astrocyte filaments. In 2016, anti-GFAP antibodies (Ab) were identified as the specific biomarker for the first established CNS inflammatory disorder subsequently called autoimmune astrocytopathy associated with anti-GFAP Ab (A-GFAP-A). Since GFAP is localized intracellularly, GFAP Ab do not appear to be directly pathogenic though serve as a biomarker of immune inflammation. Although presence of GFAP-Ab in the serum (but not in the CSF) could be observed in various CNS immune-mediated diseases, detection
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Yuan, Wei, Liaoxun Lu, Muding Rao, et al. "GFAP hyperpalmitoylation exacerbates astrogliosis and neurodegenerative pathology in PPT1-deficient mice." Proceedings of the National Academy of Sciences 118, no. 13 (2021): e2022261118. http://dx.doi.org/10.1073/pnas.2022261118.

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The homeostasis of protein palmitoylation and depalmitoylation is essential for proper physiological functions in various tissues, in particular the central nervous system (CNS). The dysfunction of PPT1 (PPT1-KI, infantile neuronal ceroid lipofuscinosis [INCL] mouse model), which catalyze the depalmitoylation process, results in serious neurodegeneration accompanied by severe astrogliosis in the brain. Endeavoring to determine critical factors that might account for the pathogenesis in CNS by palm-proteomics, glial fibrillary acidic protein (GFAP) was spotted, indicating that GFAP is probably
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Rozprawy doktorskie na temat "GFAP/Gfap"

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Wen, Shu-Fang. "The role of GFAP mutation in Alexander disease." Thesis, Durham University, 2008. http://etheses.dur.ac.uk/2064/.

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Alexaneder disease (AxD) is a primary genetic disorder of astrocyte caused by mutations in the type 111 intermediate filament (IF) glial fibrillary acidic protein (GFAP). The pathological hallmark of this disease is the presence of Rosenthal fibres (RF), ubiquitinated protein aggregates with GFAP being the primary constituent. On the basis of age at onset, the disease has been divided into three subtypes: infantile, juvenile and adult. Whilst one of the common mutations R416W is reported in AxD with a wide range in disease severity and age of onset, the mechanisms by which this mutation leads
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Gumprecht, Annett. "Analyse morphometrischer Messungen an Astrozyten des Hippokampus von Wildtypmäusen im Vergleich zu GFAP-/- - Vimentin-/- - Mäusen." Doctoral thesis, Universitätsbibliothek Leipzig, 2013. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-129946.

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Die Forschungsergebnisse der letzten Jahre beweisen, dass die Informationsverarbeitung im ZNS auf eine ausgewogene Interaktion zwischen den Neuronen und den Astrozyten im Sinne eines funktionellen Netzwerkes angewiesen ist. Allerdings liegen nur unzureichende Erkenntnisse über den strukturellen Charakter dieser symbiotischen Beziehung vor. Zu den grundlegenden Aufgaben der Astrozyten gehört die Modulation der synaptischen Aktivität von Neuronen, Aufrechterhaltung der extrazellulären Homoöstase, Ausbildung der Blut-Hirn-Schranke, Pufferung der lokalen Kaliumkonzentration und die Synthese von Zy
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Tackenberg, Mark. "Quantitative Aspekte der Astrozyten von Wildtyp- und GFAP-/- VIM-/- Labormäusen." Doctoral thesis, Universitätsbibliothek Leipzig, 2011. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-69157.

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Astrozyten erfüllen unverzichtbare Aufgaben im ZNS. Sie sorgen im Normalfall unter anderem für eine ausgewogene K+/H2O-Clearence, regulieren den Gefäßdurchmesser, bilden die Blut-/Hirnschranke, betreiben “Transmitter-Recycling” und modulieren die interneuronale Signalweitergabe durch prä- und postsynaptische Mechanismen. Die Funktionen und Einflüsse dieser zentralnervösen Gliazellen unter pathologischen Bedingungen im ZNS sind bei weitem nicht so gut untersucht, aber ebenso vielfältig. Eine ganz entscheidende Frage stellt sowohl unter physiologischen wie auch pathologischen Bedingungen das Vor
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Magerkurth, Olaf. "Nachweis von saurem glialen Faserprotein (GFAP) in humanem Serum und erste klinische Ergebnisse." Diss., lmu, 2003. http://nbn-resolving.de/urn:nbn:de:bvb:19-16561.

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Cardoso, Marie-Céleste. "Implication du gène GFAP dans les pathologies humaines neurodégénératives de la substance blanche." Clermont-Ferrand 1, 2008. http://www.theses.fr/2008CLF1MM22.

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Parmi les maladies génétiques affectant la substance blanche du système nerveux ou leucodystrophies, la maladie d'Alexander est caractérisée par une accumulation diffuse dans les astrocytes d'inclusions cytoplasmiques éosinophiles, les fibres de Rosenthal, accompagnée d'une démyélinisation. Il est clairement établi que le spectre clinique et les caractéristiques radiologiques de cette pathologie sont plus vastes que décrit initialement pour les formes précoces, les formes adultes étant particulièrement hétérogènes. Nous avons développé une méthode diagnostique, rapide et peu onéreuse, basée su
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SUGIHARA, YASUO, MINORU UEDA, HIDEYUKI NAKASHIMA, et al. "INVOLVEMENT OF GLIAL ACTIVATION IN TRIGEMINAL GANGLION IN A RAT MODEL OF LOWER GINGIVAL CANCER PAIN." Nagoya University School of Medicine, 2014. http://hdl.handle.net/2237/20551.

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Higgins, Michael Anthony. "Morphological and Immunocytochemical Investigation of Canine Oligodendrogliomas." Thesis, Virginia Tech, 2006. http://hdl.handle.net/10919/45210.

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Previous studies of human oligodendroglial neoplasms have demonstrated the diagnostic and prognostic values of histomorphologic features and immunocytochemical markers. Primary spontaneous canine intracranial tumors share many of the biologic behaviors and pathologic features of their human counterparts. The objectives of this study were to determine if associations existed between five histomorphologic features (mitoses, cellular atypia, necrosis, vascular hypertrophy, and vascular proliferation), and three immunocytochemical markers (GFAP, EGFR, and Ki-67 labeling index) and the degree of
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Steinriede, Anja. "Eingrenzung der Leichenliegezeit mittels immunhistochemischer Untersuchung des Glial-fibrillary-acidic-Protein (GFAP) in Astrocyten." [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=975443879.

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Bush, T. G. "Ablation of astrocytes in transgenic mice expressing HSV-thymidine kinase from the mouse GFAP promoter." Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.597152.

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To investigate further the functions of astrocytes, a transgenic mouse was generated in which astrocytes can be ablated selectivity. Expression of herpes thymidine kinase (HSV-tk) was targeted to astrocyte using the GFAP promoter. HSV-tk can metabolise anti-herpetic agents, such as ganciclovir (GCV), which results in the formation of toxic moieties that induce cell death. The GFAP promoter cassette was obtained from Lennart Mucke of the Scripps Institute (now UCSF) into which the HSV-tk coding sequence was inserted. A linearised GFAP-HSV-<I>tk </I>construct was microinjected into fertilised mo
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Crawford, Jessica D., Michelle J. Chandley, Katalin Szebeni, Attila Szebeni, Brandon Waters, and Gregory A. Ordway. "Elevated GFAP Protein in Anterior Cingulate Cortical White Matter in Males With Autism Spectrum Disorder." Digital Commons @ East Tennessee State University, 2015. https://doi.org/10.1002/aur.1480.

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Based on evidence of abnormalities in axon thickness and neuronal disorganization, autism spectrum disorder (ASD) is commonly considered to be a condition resulting from neuronal dysfunction. Yet, recent findings suggest that non-neuronal cell types also contribute to ASD pathology. To investigate the role of glial cells in ASD, a combination of protein and gene expression analyses were used to determine levels of two glial markers, glial fibrillary acidic protein (GFAP) and myelin oligodendrocyte glycoprotein (MOG), in the postmortem brain tissue from control and ASD donors. Levels of GFAP im
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Książki na temat "GFAP/Gfap"

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Kranich, Birgit Gerlinde. Die entzündliche Begleitreaktion bei Herpes simplex Encephalitis unter besonderer Berücksichtigung der immunhistologischen Veränderungen des sauren Gliafaserproteins GFAP. [s.n.], 1988.

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Morgenroth, Carlo. Vergleichende immunhistochemische Untersuchung über das Vorkommen von GFAP in der Hypophyse verschiedener Vertebraten. 1990.

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Künzig, Barbara Maria. Licht- und elektronenmikroskopische Untersuchungen von Vimentin, Desmin und GFAP an normalen intrakraniellen Gefässen des Menschen. 1993.

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Części książek na temat "GFAP/Gfap"

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Pekny, M., and U. Wilhelmsson. "GFAP and Astrocyte Intermediate Filaments." In Handbook of Neurochemistry and Molecular Neurobiology. Springer US, 2006. http://dx.doi.org/10.1007/978-0-387-30381-9_14.

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Sabatelli, Eleonora, and Raffaele Iorio. "GFAP Autoimmune Astrocytopathy: Clinical and Immunological Characteristics." In Neuroimmune Diseases. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-60006-7_32.

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Sabatelli, Eleonora, and Raffaele Iorio. "GFAP Autoimmune Astrocytopathy: Clinical and Immunological Characteristics." In Neuroimmune Diseases. Springer International Publishing, 2024. http://dx.doi.org/10.1007/978-3-031-24297-7_32-1.

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Iglesias, J. R., E. Kazner, and C. Aruffo. "GFAP Immunoreactivity of Oligoastrocytomas Study of 120 Cases." In Brain Oncology Biology, diagnosis and therapy. Springer Netherlands, 1987. http://dx.doi.org/10.1007/978-94-009-3347-7_8.

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Tardy, M., G. Le Prince, S. Babajko, H. Riol, C. Fages, and B. Rolland. "GFAP Gene Expression in Normal and Reactive Astrocytes." In Biology and Pathology of Astrocyte-Neuron Interactions. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9486-1_13.

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Yu, Albert C. H., Yuen Ling Lee, and Lawrence F. Eng. "Inhibition of GFAP Synthesis with Antisense Nucleic Acid Constructs." In Biology and Pathology of Astrocyte-Neuron Interactions. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4757-9486-1_28.

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Kokunai, Takashi, Norihiko Tamaki, and Satoshi Matsumoto. "Expression of Intermediate Filaments (GFAP and Vimentin) in Human Gliomas." In Biological Aspects of Brain Tumors. Springer Japan, 1991. http://dx.doi.org/10.1007/978-4-431-68150-2_54.

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Nawashiro, Hiroshi, S. Huang, M. Brenner, K. Shima, and J. M. Hallenbeck. "ICP Monitoring Following Bilateral Carotid Occlusion in GFAP-Null Mice." In Intracranial Pressure and Brain Biochemical Monitoring. Springer Vienna, 2002. http://dx.doi.org/10.1007/978-3-7091-6738-0_69.

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Finch, Caleb E., Todd E. Morgan, Irina Rozovsky, and Min Wei. "The Long Thread of GFAP in Aging, Steroids, and Synaptic Plasticity." In Research and Perspectives in Endocrine Interactions. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-662-07019-2_13.

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Pan, Jia-Bao, Chi-Ming Lee, and Elliott J. Mufson. "Complement Components and GFAP Immunoreactivity within Alzheimer and Pathologic Aged Cortex." In Alzheimer’s and Parkinson’s Diseases. Springer US, 1995. http://dx.doi.org/10.1007/978-1-4757-9145-7_33.

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Streszczenia konferencji na temat "GFAP/Gfap"

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Balaji, P., D. Devasena, P. Rithick, T. Karthi, K. Rishmitha, and R. Roopa. "Microfluidic Lab-on-a-Chip Design and Analysis for GFAP Biomarker Detection Using Machine Learning Algorithms." In 2024 International Conference on Communication, Control, and Intelligent Systems (CCIS). IEEE, 2024. https://doi.org/10.1109/ccis63231.2024.10931904.

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Hellmann, H., S. Brock, and A. Reisberg. "Autoimmunenzephalitis mit GFAP-Autoantikörpern." In RÖKO 2023. Georg Thieme Verlag, 2023. http://dx.doi.org/10.1055/s-0043-1763232.

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Mahat, Bishow Chandra, Katharine Dermigny, Lamees Alzyoud, and Madison Pilato. "Sudden Death in Autoimmune GFAP Astrocytopathy (P5-5.020)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000201975.

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Dias, Thales Augusto Oliveira, and Silvia Graciela Ruginsk Leitão. "Participation of calcium channels in the action of angiotensin II in astrocytes." In XIII Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2021. http://dx.doi.org/10.5327/1516-3180.299.

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Background: The renin-angiotensin-aldosterone system is the main regulator of blood pressure and blood volume, with most effects being mediated by angiotensin II (Ang-II) - responsible, in the central nervous system, for actions such as thirst and sodium appetite. Astrocytes are believed to mediate such a response, as they express receptors for Ang-II and respond directly to dehydration with impacting morphological changes in the synaptic microenvironment. Many of its functions involve L-type calcium channels (LTCCs). Objectives: Evaluate the participation of LTCCs in the effects induced by An
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Kinsella, Fiona C., Lauren M. Byrne, Filipe B. Rodrigues, et al. "D08 Elevated GFAP and UCHL-1 in plasma and CSF in HD." In EHDN 2022 Plenary Meeting, Bologna, Italy, Abstracts. BMJ Publishing Group Ltd, 2022. http://dx.doi.org/10.1136/jnnp-2022-ehdn.64.

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Grandes Moreno, Pedro, Inmaculada Gerrikagoitia Marina, Izaskun Elezgarai Gabantxo, et al. "CB1 hartzaile astrozitikoa mikroskopio elektronikoko prestakinetan detektatzeko markatzaile astroglialak: GFAP vs GLAST." In III. Ikergazte. Nazioarteko ikerketa euskaraz. UEU arg, 2019. http://dx.doi.org/10.26876/ikergazte.iii.04.15.

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Schmid, Ralf S., Ryan E. Bash, Andrea M. Werneke, and C. Ryan Miller. "Abstract 3302: Roles of cortical and subventricular GFAP+ astrocytes in initiation of astrocytomas." In Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-3302.

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Zahid, Anza, Beatriz Thames, Osman Ozel, Mohammad Nakawah, and Sheetal Shroff. "Pseudo-bulbar affect and Tremors in GFAP Astrocytopathy: A Case Report (P4-5.005)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000202968.

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Vasconcelos, Luna Costa, M. Pereira,, and G. Ballough,. "Neuropatologia – análise de HE e GFAP com sequenza gap capilar em modelo de convulsão." In II Encontro do Programa de Pós-Graduação em Ciências Farmacêuticas da Universidade Federal do Ceará e I Simpósio Norte-Nordeste de Ciências Farmacêuticas. Galoa, 2017. http://dx.doi.org/10.17648/ppgcf-2017-66337.

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Suzuki, Kei, Toshihiko Shiraishi, Shin Morishita, and Hiroshi Kanno. "Effects of Mechanical Vibration on Proliferation and Differentiation of Neural Stem Cells." In ASME 2008 International Mechanical Engineering Congress and Exposition. ASMEDC, 2008. http://dx.doi.org/10.1115/imece2008-66831.

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Neural stem cells have been studied to promote neurogenesis in regenerative therapy. The control of differentiation of neural stem cells to nerve cells and the increase of the number of nerve cells are needed. For the purpose of them, it is important to investigate not only chemical factors but also mechanical factors such as hydrostatic pressure in brain and mechanical vibration in walking. In this study, sinusoidal inertia force was applied to cultured neural stem cells and the effects of mechanical vibration on the cells were investigated. After the cells were cultured in culture plates for
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Raporty organizacyjne na temat "GFAP/Gfap"

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Liu, Dianjun, Xiangwen Yao, and Hao Zhang. Effects of general anesthetics on GFAP/Iba-1 expression based on animal experiments: a meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2024. http://dx.doi.org/10.37766/inplasy2024.4.0002.

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