Gotowe bibliografie tematyczne / Ghrelin signaling

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Gotowa bibliografia na temat „Ghrelin signaling”

Autor: Grafiati

Data publikacji: 2 listopada 2024

Data aktualizacji: 31 lipca 2025

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Artykuły w czasopismach na temat "Ghrelin signaling"

Hougland, James L. "Ghrelin octanoylation by ghrelin O-acyltransferase: Unique protein biochemistry underlying metabolic signaling." Biochemical Society Transactions 47, no. 1 (2019): 169–78. http://dx.doi.org/10.1042/bst20180436.

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Abstract Ghrelin is a small peptide hormone that requires a unique post-translational modification, serine octanoylation, to bind and activate the GHS-R1a receptor. Ghrelin signaling is implicated in a variety of neurological and physiological processes, but is most well known for its roles in controlling hunger and metabolic regulation. Ghrelin octanoylation is catalyzed by ghrelin O-acyltransferase (GOAT), a member of the membrane-bound O-acyltransferase (MBOAT) enzyme family. From the status of ghrelin as the only substrate for GOAT in the human genome to the source and requirement for the

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Holst, Birgitte, Erik Brandt, Anders Bach, Anders Heding, and Thue W. Schwartz. "Nonpeptide and Peptide Growth Hormone Secretagogues Act Both as Ghrelin Receptor Agonist and as Positive or Negative Allosteric Modulators of Ghrelin Signaling." Molecular Endocrinology 19, no. 9 (2005): 2400–2411. http://dx.doi.org/10.1210/me.2005-0059.

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Abstract Two nonpeptide (L692,429 and MK-677) and two peptide [GH-releasing peptide (GHRP)-6 and ghrelin] agonists were compared in binding and in signal transduction assays: calcium mobilization, inositol phosphate turnover, cAMP-responsive element (CRE), and serum-responsive element (SRE) controlled transcription, as well as arrestin mobilization. MK-677 acted as a simple agonist having an affinity of 6.5 nm and activated all signal transduction systems with similar high potency (0.2–1.4 nm). L-692,429 also displayed a very similar potency in all signaling assays (25–60 nm) but competed with

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Heldsinger, Andrea, Gintautas Grabauskas, Xiaoyin Wu, et al. "Ghrelin Induces Leptin Resistance by Activation of Suppressor of Cytokine Signaling 3 Expression in Male Rats: Implications in Satiety Regulation." Endocrinology 155, no. 10 (2014): 3956–69. http://dx.doi.org/10.1210/en.2013-2095.

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Abstract The anorexigenic adipocyte-derived hormone leptin and the orexigenic hormone ghrelin act in opposition to regulate feeding behavior via the vagal afferent pathways. The mechanisms by which ghrelin exerts its inhibitory effects on leptin are unknown. We hypothesized that ghrelin activates the exchange protein activated by cAMP (Epac), inducing increased SOCS3 expression, which negatively affects leptin signal transduction and neuronal firing in nodose ganglia (NG) neurons. We showed that 91 ± 3% of leptin receptor (LRb) –bearing neurons contained ghrelin receptors (GHS-R1a) and that gh

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Holliday, Nicholas D., Birgitte Holst, Elena A. Rodionova, Thue W. Schwartz, and Helen M. Cox. "Importance of Constitutive Activity and Arrestin-Independent Mechanisms for Intracellular Trafficking of the Ghrelin Receptor." Molecular Endocrinology 21, no. 12 (2007): 3100–3112. http://dx.doi.org/10.1210/me.2007-0254.

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Abstract The ghrelin receptor (GhrelinR) and its related orphan GPR39 each display constitutive signaling, but only GhrelinRs undergo basal internalization. Here we investigate these differences by considering the roles of the C tail receptor domains for constitutive internalization and activity. Furthermore the interaction between phosphorylated receptors and β-arrestin adaptor proteins has been examined. Replacement of the FLAG-tagged GhrelinR C tail with the equivalent GPR39 domain (GhR-39 chimera) preserved Gq signaling. However in contrast to the GhrelinR, GhR-39 receptors exhibited no ba

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Wu, Chia-Shan, Jiyeon Noh, Ellie Tuchaai, et al. "SUPPRESSION OF GHRELIN SIGNALING EXACERBATES ULCERATIVE COLITIS IN OLDER MICE." Innovation in Aging 3, Supplement_1 (2019): S87. http://dx.doi.org/10.1093/geroni/igz038.334.

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Abstract The aging process is characterized by increased chronic low-grade inflammation, aka inflamm-aging, which offend is accompanied by ‘leaky gut’ syndrome. Inflamm-aging is a highly significant risk factor for both morbidity and mortality in the older adult population (&gt;65 years of age). In addition, there is a growing prevalence of inflammatory bowel disease (IBD), a chronic inflammatory condition of the gastrointestinal tract in the older adult population. The pathogenesis of late-onset IBD is suggested to be more complex compared with younger IBD patients; the causes determining

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Lin, Tsung-Chieh, Yuan-Ming Yeh, Wen-Lang Fan, et al. "Ghrelin Upregulates Oncogenic Aurora A to Promote Renal Cell Carcinoma Invasion." Cancers 11, no. 3 (2019): 303. http://dx.doi.org/10.3390/cancers11030303.

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Ghrelin is a peptide hormone, originally identified from the stomach, that functions as an endogenous ligand of the growth hormone secretagogue receptor (GHSR) and promotes growth hormone (GH) release and food intake. Increasing reports point out ghrelin’s role in cancer progression. We previously characterized ghrelin’s prognostic significance in the clear cell subtype of renal cell carcinoma (ccRCC), and its pro-metastatic ability via Snail-dependent cell migration. However, ghrelin’s activity in promoting cell invasion remains obscure. In this study, an Ingenuity Pathway Analysis (IPA)-base

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Madison, Lisa D., Jarrad M. Scarlett, Peter Levasseur, et al. "Prostacyclin signaling regulates circulating ghrelin during acute inflammation." Journal of Endocrinology 196, no. 2 (2007): 263–73. http://dx.doi.org/10.1677/joe-07-0478.

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Ghrelin is an octanoylated 28 amino acid peptide predominantly secreted by the stomach, and has potent stimulatory effects on appetite. Several laboratories, including our own, have demonstrated that ghrelin levels fall in states of acute inflammation brought about by injection of bacterial lipopolysaccharide (LPS). We now demonstrate that the decrease in circulating ghrelin is not due to a decrease in ghrelin gene expression, but is instead likely to be due to an acute decrease in ghrelin secretion. Furthermore, we have found that the change in circulating ghrelin during acute inflammation re

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Xu, Geyang, Yin Li, Wenjiao An, et al. "Gastric Mammalian Target of Rapamycin Signaling Regulates Ghrelin Production and Food Intake." Endocrinology 150, no. 8 (2009): 3637–44. http://dx.doi.org/10.1210/en.2009-0372.

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Ghrelin, a gastric hormone, provides a hunger signal to the central nervous system to stimulate food intake. Mammalian target of rapamycin (mTOR) is an intracellular fuel sensor critical for cellular energy homeostasis. Here we showed the reciprocal relationship of gastric mTOR signaling and ghrelin during changes in energy status. mTOR activity was down-regulated, whereas gastric preproghrelin and circulating ghrelin were increased by fasting. In db/db mice, gastric mTOR signaling was enhanced, whereas gastric preproghrelin and circulating ghrelin were decreased. Inhibition of the gastric mTO

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Gluck, Elizabeth F., Natalie Stephens, and Steven J. Swoap. "Peripheral ghrelin deepens torpor bouts in mice through the arcuate nucleus neuropeptide Y signaling pathway." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 291, no. 5 (2006): R1303—R1309. http://dx.doi.org/10.1152/ajpregu.00232.2006.

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Many small mammals have the ability to enter torpor, characterized by a controlled drop in body temperature (Tb). We hypothesized that ghrelin would modulate torpor bouts, because torpor is induced by fasting in mice coincident with elevated circulating ghrelin. Female National Institutes of Health (NIH) Swiss mice were implanted with a Tb telemeter and housed at an ambient temperature (Ta) of 18°C. On fasting, all mice entered a bout of torpor (minimum Tb: 23.8 ± 2.0°C). Peripheral ghrelin administration (100 μg) during fasting significantly deepened the bout of torpor (Tb minimum: 19.4 ± 0.5

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Hosoda, Hiroshi. "Effect of Ghrelin on the Cardiovascular System." Biology 11, no. 8 (2022): 1190. http://dx.doi.org/10.3390/biology11081190.

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Ghrelin, an n-octanoyl-modified 28-amino-acid-peptide, was first discovered in the human and rat stomach as an endogenous ligand for the growth hormone secretagogue receptor (GHS-R). Ghrelin-GHS-R1a signaling regulates feeding behavior and energy balance, promotes vascular activity and angiogenesis, improves arrhythmia and heart failure, and also protects against cardiovascular disease by suppressing cardiac remodeling after myocardial infarction. Ghrelin’s cardiovascular protective effects are mediated by the suppression of sympathetic activity; activation of parasympathetic activity; allevia

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Rozprawy doktorskie na temat "Ghrelin signaling"

Marion, Candice. "Novel insights on ghrelin receptor signaling in energy homeostasis and feeding behavior using the GhsrQ343X mutant rat model." Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCB109.

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La ghréline acylée, une hormone produite par l’estomac, favorise la prise de poids corporel, majoritairement sous forme de masse grasse, par le biais de divers mécanismes centraux et périphériques via le récepteur sécrétagogue de l’hormone de croissance (GHSR). Le GHSR est un récepteur couplé aux protéines G qui, en plus de répondre à la ghréline acylée, possède une signalisation indépendante de la ghréline par le biais de son activité constitutive ou par une modulation de réponses dopaminergiques via oligomérisation du GHSR avec des récepteurs dopaminergiques. Malgré les puissantes réponses p

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de, Amorim Laura Miranda. "The roles of the preproghrelin-derived peptides - ghrelin, desacyl ghrelin and obestatin - in prostate cancer." Thesis, Queensland University of Technology, 2012. https://eprints.qut.edu.au/53260/1/Laura_de_Amorim_thesis.pdf.

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Prostate cancer is the second most common cause of cancer related deaths in Western men. Despite the significant improvements in current treatment techniques, there is no cure for advanced metastatic, castrate-resistant disease. Early detection and prevention of progression to a castrate-resistant state may provide new strategies to improve survival. A number of growth factors have been shown to act in an autocrine/paracrine manner to modulate prostate cancer tumour growth. Our laboratory has previously shown that ghrelin and its receptors (the functional GHS-R1a and the non-functional GHS-R1b

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Lassman, Daniel James. "Gut-Brain Signalling in Man: The Roles of Lipid, Cholecystokinin and Ghrelin." Thesis, University of Manchester, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.492929.

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Ingested lipid releases the gut peptide cholecystokinin (CCK) which limits food intake by modulating gut function and signalling to the brain. Current understanding of this pathway and its interaction with other gut-brain signalling peptides such as ghrelin is incomplete. To clarify the roles of lipid, CCK and ghrelin in gut-brain signalling pathway in man, a series of human studies were performed in order to answer the following questions.

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Qu, Mengdi. "Molecular mechanism underlying CaMK1D-dependent function in AgRP neurons." Electronic Thesis or Diss., Strasbourg, 2024. http://www.theses.fr/2024STRAJ029.

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La perturbation des mécanismes de réponse au stress chez les organismes peut entraîner une dysfonction cellulaire et des maladies telles que le syndrome métabolique. L'équilibre énergétique est principalement régulé par le système nerveux central (SNC), qui intègre des signaux hormonaux, neuronaux et alimentaires provenant de divers tissus. Une dysfonction de ce système est liée à l'obésité et au syndrome métabolique, qui sont tous deux des précurseurs du diabète de type 2 (T2D). Notre laboratoire a découvert que la protéine kinase ID dépendante du calcium/calmoduline (CaMK1D), un gène associé

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Walpole, Carina Maree. "The function and mechanisms of action of ghrelin and obestatin in ovarian cancer." Thesis, Queensland University of Technology, 2012. https://eprints.qut.edu.au/63497/1/Carina_Walpole_Thesis.pdf.

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In this study, we have demonstrated that the preproghrelin derived hormones, ghrelin and obestatin, may play a role in ovarian cancer. Ghrelin and obestatin stimulated an increase in cell migration in ovarian cancer cell lines and may play a role in cancer progression. Ovarian cancer is the leading cause of death among gynaecological cancers and is the sixth most common cause of cancer-related deaths in women in developed countries. As ovarian cancer is difficult to diagnose at a low tumour grade, two thirds of ovarian cancers are not diagnosed until the late stages of cancer development resul

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Kaiser, Julia Marion [Verfasser]. "Ghrelin beeinflusst die pankreatische Betazelle durch Modulation von KATP-Kanälen über den cAMP/PKA Signalweg / Julia Marion Kaiser." Tübingen : Universitätsbibliothek Tübingen, 2021. http://nbn-resolving.de/urn:nbn:de:bsz:21-dspace-968490.

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Cunningham, Peter Stephen. "The ghrelin receptor isoforms (GHS-R1a and GHS-R1b) and GPR39 : an investigation into receptor dimerisation." Thesis, Queensland University of Technology, 2010. https://eprints.qut.edu.au/39443/1/Peter_Cunningham_Thesis.pdf.

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Prostate cancer is the second most common cause of cancer-related deaths in Western males. Current diagnostic, prognostic and treatment approaches are not ideal and advanced metastatic prostate cancer is incurable. There is an urgent need for improved adjunctive therapies and markers for this disease. GPCRs are likely to play a significant role in the initiation and progression of prostate cancer. Over the last decade, it has emerged that G protein coupled receptors (GPCRs) are likely to function as homodimers and heterodimers. Heterodimerisation between GPCRs can result in the formation of no

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Börner, Sabina. "Untersuchungen zum Zusammenhang zwischen Fettmobilisierung und futteraufnahmesteigernden Signalen bei der Milchkuh im peripartalen Zeitraum." Doctoral thesis, Universitätsbibliothek Leipzig, 2014. http://nbn-resolving.de/urn:nbn:de:bsz:15-qucosa-144728.

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Die Belastung des Energiestoffwechsels der Hochleistungskuh ist in der peripartalen Phase am größten. Die Regulation der Futteraufnahme und des Energiestoffwechsels durch den Nucleus arcuatus (ARC) des Hypothalamus spielt eine entscheidende Rolle während dieser Phase. Zahlreiche Metabolite und Hormone, wie z.B. das Peptidhormon Ghrelin, beeinflussen die Expression des orexigenen (futteraufnahmesteigernden) Neuropeptids Agouti-related Protein (AgRP) im ARC des Hypothalamus. Das Ziel dieser Arbeit war es, den Zusammenhang zwischen Körperfettmobilisierung und orexigenen Signalen im peripartalen Z

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Ribeiro, Luís Filipe da Silva. "A Link Between Metabolic Signaling and Cognition: The Hippocampal Function of Ghrelin." Doctoral thesis, 2013. http://hdl.handle.net/10316/24357.

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Tese de doutoramento em Biologia, na especialidade de Biologia Celular, apresentada à Faculdade de Ciências e Tecnologia da Universidade de Coimbra.<br>Peptide hormones such as insulin, leptin and ghrelin are well known for their role in the regulation of appetite. Recent evidence suggests that these peptides, in addition to acting on the hypothalamus to modulate food intake, may play wider roles in modulating brain functions. Ghrelin, a 28 amino acids peptide, is a regulator of growth hormone release, and an appetite-stimulating hormone, secreted from the empty stomach. Recent data show t

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Shah, Samiksha. "A hunger hormone that attenuates conditioned fear?: investigating the role of ghrelin receptor signaling in the acquisition and consolidation of fear memory." Thesis, 2017. https://hdl.handle.net/2144/23989.

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Ghrelin has been established as a hunger hormone because of its role in weight regulation and appetite stimulation. However, recent studies have uncovered a role for ghrelin in the modulation of negative emotional states like fear, anxiety and depression. The unusually high constitutive activity of the ghrelin receptor, growth hormone secretagogue receptor type 1a (GHSR1a) and its extensive ability to dimerize with other neuromodulatory receptors highlights the complexity of ghrelin receptor signaling. This led us to examine one of the essential constituents of this signaling mechanism. We exo

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Książki na temat "Ghrelin signaling"

Beninger, Richard J. Dopamine receptor subtypes and incentive learning. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198824091.003.0007.

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Dopamine receptor subtypes and incentive learning explains that dopamine receptors are G protein-coupled and form two families: D1-like receptors, including D1 and D5, stimulate adenylyl cyclase and cyclic adenosine monophosphate (cAMP); D2-like receptors, including D2, D3, and D4, inhibit cAMP. Antipsychotic medications are dopamine receptor antagonists and their clinical potency is strongly correlated with blockade of D2 receptors, implicating overactivity of D2 receptors in psychosis in schizophrenia. D1- and D2-like receptors appear to be involved in unconditioned locomotor activity and in

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Części książek na temat "Ghrelin signaling"

Fang, Chuo, Hang Xu, Shaodong Guo, Susanne U. Mertens-Talcott, and Yuxiang Sun. "Ghrelin Signaling in Immunometabolism and Inflamm-Aging." In Advances in Experimental Medicine and Biology. Springer Singapore, 2018. http://dx.doi.org/10.1007/978-981-13-1286-1_9.

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Dezaki, Katsuya, Boldbaatar Damdindorj, Tomoyuki Kurashina, and Toshihiko Yada. "Ghrelin’s Novel Signaling in Islet β-Cells to Inhibit Insulin Secretion and Its Blockade As a Promising Strategy to Treat Type 2 Diabetes." In Ghrelin in Health and Disease. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-903-7_3.

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Salcido, Alexis A., Neftali F. Reyes, Andrea Y. Macias, et al. "An Emerging Role for Gut-Brain Signaling Involving Ghrelin in Chronic Stress." In Advances in Experimental Medicine and Biology. Springer Nature Switzerland, 2025. https://doi.org/10.1007/978-3-031-89525-8_7.

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"Ghrelin Receptor." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101438.

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Mendiratta, Meenal Shewale, Shivam Manilal Patel, and Jose Manuel Garcia. "Ghrelin: Effects and Mechanisms of Action in Tumor-Induced Cachexia." In BASIC/TRANSLATIONAL - Growth Factors, Cytokines & Intracellular Signaling. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p24.p2-90.

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Dezaki, Katsuya, and Toshihiko Yada. "Islet β-Cell Ghrelin Signaling for Inhibition of Insulin Secretion." In Methods in Enzymology. Elsevier, 2012. http://dx.doi.org/10.1016/b978-0-12-381272-8.00020-9.

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Evron, Tama, Nikhil M. Urs, Laurie Sutton, Yushi Bai, Marc G. Caron, and Larry S. Barak. "β-arrestin Regulation of Ghrelin Signaling in modulating Addictive Behavior." In Catecholamine Research in the 21st Century. Elsevier, 2014. http://dx.doi.org/10.1016/b978-0-12-800044-1.00167-7.

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Andrade, Sara, Marcos Couselo Carreira, Andreia Ribeiro, et al. "Development of an Anti-Ghrelin Vaccine for Obesity Treatment." In BASIC/TRANSLATIONAL - Hypothalamic Signaling in Feeding Behavior & Metabolic Regulation. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p34.p2-305.

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Nass, Ralf, Jianhua Liu, Suzan Pezzoli, Leon Farhi, Bruce Gaylinn, and Michael Thorner. "Inhibition of Acyl-Ghrelin Release in Healthy Young Adults during Euglycemic Hyperinsulinemic Clamp." In BASIC/TRANSLATIONAL - Hypothalamic Signaling in Feeding Behavior & Metabolic Regulation. The Endocrine Society, 2011. http://dx.doi.org/10.1210/endo-meetings.2011.part2.p34.p2-308.

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Lambert, Charles. "Attenuating Cancer Cachexia-Prolonging Life." In Frailty and Sarcopenia - Recent Evidence and New Perspectives. IntechOpen, 2022. http://dx.doi.org/10.5772/intechopen.101250.

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Death by cancer cachexia is dependent on the time allotted to cancer to cause muscle and fat wasting. If clinicians, nurses, researchers can prolong the life of a cancer patient other therapeutic interventions such as radiation and chemotherapy may be given the time to work and rid the cancer patient of tumors and save lives. Three areas by which cancer induces cachexia is through impaired insulin-like growth factor signaling, elevations in the proinflammatory cytokines TNF-α and IL-6 and subsequent reductions in muscle protein synthesis and increases in muscle protein degradation. Therefore,

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Streszczenia konferencji na temat "Ghrelin signaling"

Patlis, AsiaLuna. "Cellular and molecular effects of weight stigma: The effect of cortisol on hypothalamic neurons in vitro." In 10th Annual International Weight Stigma Conference. Weight Stigma Conference, 2024. https://doi.org/10.31076/2024.p22.

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Weight stigma has been associated with changes to the stress-responsive hypothalamic-pituitary-adrenocortical (HPA) axis. Chronic activation of the HPA axis leads to elevated levels of circulating glucocorticoids such as cortisol. This poster will show a project proposal to study the cellular and molecular effects of cortisol on human arcuate-like hypothalamic neurons in vitro. The arcuate nucleus of the hypothalamus (ARC) is involved in the homeostatic regulation of body weight and feeding. The ARC is home to two important neuronal populations: proopiomelanocortin (POMC) and agouti-related ne

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Pinheiro, Amanda Pereira Sindeaux, Pedro Vitor Ferreira Rodrigues, Raoni de Oliveira da Silva Domingues, and Leonardo José Rodrigues Araújo Melo. "Gastrointestinal dysmotility associated with Parkinson’s disease’s mechanism." In XIV Congresso Paulista de Neurologia. Zeppelini Editorial e Comunicação, 2023. http://dx.doi.org/10.5327/1516-3180.141s1.461.

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Introduction: Parkinson’s Disease (PD) is a condition of the brain that consiste of the death of dopaminergic neurons in the substantia nigra, therefore causing dyskinesias and dystonias. Besides the motor symptoms, the neurogastro motility is affected by the disease, since gastrointestinal dysfunction is a frequent and clinically relevant symptom of PD. Objectives: To link the neural pathways and neurotransmitters that involve the neuroenteric system control and the PD’s pathology. Methods: A systematic literature review was performed based on data extraction through the advanced research eng

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Patil, Rashmi, Urmila M. Aswar, and Nishant Vyas. "Pterostilbene alleviates Cafeteria diet-induced Obesity and underlying Depression in Adolescent mice through SIRT1 mediated Leptin-Ghrelin signalling pathway." In ASPET 2024 Annual Meeting Abstract. American Society for Pharmacology and Experimental Therapeutics, 2024. http://dx.doi.org/10.1124/jpet.305.934770.

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