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1

DAROCA, PHILIP J., and WILLIAM J. GEORGE. "Giant Cell Interstitial Pneumonia." Southern Medical Journal 84, no. 2 (1991): 257–62. http://dx.doi.org/10.1097/00007611-199102000-00027.

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Sisodia, Shantilal M., Kiran Bendale, Wasif Ali ZafarAli Khan, and Vandana Sanklecha. "Giant Cell Interstitial Pneumonia." American Journal of Forensic Medicine and Pathology 34, no. 2 (2013): 110–14. http://dx.doi.org/10.1097/paf.0b013e318288c6f5.

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Kang, Kyeong Woo, Sang Joon Park, Gee Yiung Suh, et al. "A Case of Giant Cell Interstitial Pneumonia." Tuberculosis and Respiratory Diseases 48, no. 2 (2000): 260. http://dx.doi.org/10.4046/trd.2000.48.2.260.

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Hargett, Charles W., Thomas A. Sporn, Victor Roggli, and John W. Hollingsworth. "GIANT CELL INTERSTITIAL PNEUMONIA ASSOCIATED WITH NITROFURANTOIN." Chest 128, no. 4 (2005): 492S. http://dx.doi.org/10.1378/chest.128.4_meetingabstracts.492s.

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MIYAZAKI, Nobuyoshi, Masamitsu KIDO, Akira KAJIKI, and Yasuo MORIMOTO. "A Case of Giant Cell Interstitial Pneumonia." Journal of UOEH 11, no. 1 (1989): 63–67. http://dx.doi.org/10.7888/juoeh.11.63.

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Hargett, Charles William, Thomas A. Sporn, Victor L. Roggli, and John W. Hollingsworth. "Giant Cell Interstitial Pneumonia Associated with Nitrofurantoin." Lung 184, no. 3 (2006): 147–49. http://dx.doi.org/10.1007/s00408-005-2574-z.

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Sgrignoli, Anita R., Deanna R. L. Yen, and Grover M. Hutchins. "Giant Cell and Lymphocytic Interstitial Pneumonia Associated with Fetal Pneumonia." Pediatric Pathology 14, no. 6 (1994): 955–65. http://dx.doi.org/10.3109/15513819409037692.

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Ohori, N. Paul, Frank C. Sciurba, Gregory R. Owens, Michael J. Hodgson, and Samuel A. Yousem. "Giant-Cell Interstitial Pneumonia and Hard-Metal Pneumoconiosis." American Journal of Surgical Pathology 13, no. 7 (1989): 581–87. http://dx.doi.org/10.1097/00000478-198907000-00006.

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Lee, S. M., C. H. Moon, Y. B. Oh, et al. "Giant-cell interstitial pneumonia in a gas station worker." Journal of Korean Medical Science 13, no. 5 (1998): 545. http://dx.doi.org/10.3346/jkms.1998.13.5.545.

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KAKUGAWA, Tomoyuki, Hiroshi MUKAE, Towako NAGATA, et al. "Giant Cell Interstitial Pneumonia in a 15-year-old Boy." Internal Medicine 41, no. 11 (2002): 1007–12. http://dx.doi.org/10.2169/internalmedicine.41.1007.

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Nemery, Benoit, Erik K. Verbeken, and Maurits Demedts. "Giant Cell Interstitial Pneumonia (Hard Metal Lung Disease, Cobalt Lung)." Seminars in Respiratory and Critical Care Medicine 22, no. 04 (2001): 435–48. http://dx.doi.org/10.1055/s-2001-17386.

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Gurel, Duygu, Aydanur Kargi, Mehtat Unlu, Eyup Sabri Ucan, and Ahmet Onen. "Giant Cell Interstitial Pneumonia (Hard Metal Lung Disease, Cobalt Lung): Case Report." Turkish Thoracic Journal/Turk Toraks Dergisi 13, no. 3 (2012): 134–36. http://dx.doi.org/10.5152/ttd.2012.28.

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Fels Elliott, Daffolyn Rachael, Rupal Shah, Catherine Ann Hess, et al. "Giant cell interstitial pneumonia secondary to cobalt exposure from e-cigarette use." European Respiratory Journal 54, no. 6 (2019): 1901922. http://dx.doi.org/10.1183/13993003.01922-2019.

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Lee, Boeun, Arvind Balavenkataraman, Devang Sanghavi, and Kristin Walter. "Recurrent nitrofurantoin-induced giant cell interstitial pneumonia: Case report and literature review." Respiratory Medicine Case Reports 14 (2015): 49–52. http://dx.doi.org/10.1016/j.rmcr.2015.01.002.

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Ramadhanti, Anisa. "Pendekatan Diagnosis Terbaru Vaping Associated Pulmonary Injury (VAPI)." Journal of Health Science and Physiotherapy 2, no. 1 (2020): 74–80. http://dx.doi.org/10.35893/jhsp.v2i1.38.

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Rokok elektronik atau vape diklaim lebih aman dan lebih tidak berbahaya karena lebih sedikit mengandung bahan kimia toksik dibandingkan rokok konvensional. Walaupun demikian, masih banyak pihak yang mempertanyakan keamanan rokok elektronik. Liquid dalam rokok elektronik dan aerosol yang dihasilkan terbukti mengandung sejumlah komponen kimia yang kemungkinan memiliki efek yang berbahaya terhadap kesehatan, terutama paru yang dapat menyebabkan vaping-associated pulmonary injury (VAPI). Berdasarkan laporan kasus sebelumnya, VAPI terbagi menjadi beberapa spektrum penyakit, diantaranya adalah pneum
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Hong, Ji Hyun, Jae Myung Lee, Min Jong Kang, et al. "A Case of Giant Cell Interstitial Pneumonia without History of Exposure to Hard Metal." Tuberculosis and Respiratory Diseases 52, no. 4 (2002): 419. http://dx.doi.org/10.4046/trd.2002.52.4.419.

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Choi, Jin Wook, Kyung Soo Lee, Man Pyo Chung, Joungho Han, Myung Jin Chung, and Jai Soung Park. "Giant Cell Interstitial Pneumonia: High-Resolution CT and Pathologic Findings in Four Adult Patients." American Journal of Roentgenology 184, no. 1 (2005): 268–72. http://dx.doi.org/10.2214/ajr.184.1.01840268.

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Menon, Balakrishnan, Amit Sharma, Jai Kripalani, and Sudhir Jain. "Giant Cell Interstitial Pneumonia in a 60-Year-Old Female without Hard Metal Exposure." Respiration 73, no. 6 (2006): 833–35. http://dx.doi.org/10.1159/000091610.

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Paris, C., J. C. Pairon, M. A. Billon-Galland, et al. "Giant Cell Interstitial Pneumonia: Report of Two Cases with High Titanium Concentration in the Lung." American Journal of Respiratory and Critical Care Medicine 184, no. 11 (2011): 1315–17. http://dx.doi.org/10.1164/ajrccm.184.11.1315a.

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Rodo, A., and W. Bielecki. "Giant Cell Interstitial Pneumonia in a Pet Guinea Pig Due to Exposure to Air Dust." Journal of Comparative Pathology 154, no. 1 (2016): 88. http://dx.doi.org/10.1016/j.jcpa.2015.10.071.

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Kinoshita, Masaharu, Yoshiko Sueyasu, Hisashi Watanabe, et al. "Giant cell interstitial pneumonia in two hard metal workers: The role of bronchoalveolar lavage in diagnosis." Respirology 4, no. 3 (1999): 263–66. http://dx.doi.org/10.1046/j.1440-1843.1999.00186.x.

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Tanaka, Junichi, Hiroshi Moriyama, Masaki Terada, et al. "An observational study of giant cell interstitial pneumonia and lung fibrosis in hard metal lung disease." BMJ Open 4, no. 3 (2014): e004407. http://dx.doi.org/10.1136/bmjopen-2013-004407.

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Du, Xuqin, Jie Liu, Yiran Wang, Mulan Jin, and Qiao Ye. "Cobalt-related interstitial lung disease or hard metal lung disease: A case series of Chinese workers." Toxicology and Industrial Health 37, no. 5 (2021): 280–88. http://dx.doi.org/10.1177/07482337211000989.

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Hard metal lung disease (HMLD) is rarely diagnosed and is caused by the occupational inhalation of hard metal dust, mainly cobalt. The diagnosis of HMLD is based on a thorough occupational dust exposure combined with clinical–radiological–histological findings. We present a series of four Chinese workers who had occupational exposure to cobalt acid lithium or cobalt and tungsten dust. Four patients all complained of intermittent cough, chest tightness, or shortness of breath on exertion. High-resolution computed tomography scans presented bilateral ground-glass attenuation, consolidations, and
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Mizutani, Rafael Futoshi, Mário Terra-Filho, Evelise Lima, et al. "Hard metal lung disease: a case series." Jornal Brasileiro de Pneumologia 42, no. 6 (2016): 447–52. http://dx.doi.org/10.1590/s1806-37562016000000260.

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ABSTRACT Objective: To describe diagnostic and treatment aspects of hard metal lung disease (HMLD) and to review the current literature on the topic. Methods: This was a retrospective study based on the medical records of patients treated at the Occupational Respiratory Diseases Clinic of the Instituto do Coração, in the city of São Paulo, Brazil, between 2010 and 2013. Results: Of 320 patients treated during the study period, 5 (1.56%) were diagnosed with HMLD. All of those 5 patients were male (mean age, 42.0 ± 13.6 years; mean duration of exposure to hard metals, 11.4 ± 8.0 years). Occupati
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Khoor, Andras, Anja C. Roden, Thomas V. Colby, et al. "Giant cell interstitial pneumonia in patients without hard metal exposure: analysis of 3 cases and review of the literature." Human Pathology 50 (April 2016): 176–82. http://dx.doi.org/10.1016/j.humpath.2015.12.004.

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Naqvi, Asghar H., Andrew Hunt, Bryan R. Burnett, and Jerrold L. Abraham. "Pathologic Spectrum and Lung Dust Burden in Giant Cell Interstitial Pneumonia (Hard Metal Disease/Cobalt Pneumonitis): Review of 100 Cases." Archives of Environmental & Occupational Health 63, no. 2 (2008): 51–70. http://dx.doi.org/10.3200/aeoh.63.2.51-70.

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Jacobson, Elliott R., Jack M. Gaskin, Melody Roelke, Ellis C. Greiner, and Jack Allen. "Conjunctivitis, tracheitis, and pneumonia associated with herpesvirus infection in green sea turtles." Journal of the American Veterinary Medical Association 189, no. 9 (1986): 1020–23. https://doi.org/10.2460/javma.1986.189.09.1020.

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SUMMARY Fourteen juvenile (15- to 20-month-old) green sea turtles (Chelonia mydas), representative of a group of sea turtles with clinical signs of respiratory tract disease, were euthanatized and submitted for necropsy. Macroscopically, lesions included periglottal necrosis, tracheitis with intraluminal caseous and laminated necrotic debris, and severe pneumonia. Several turtles had caseous conjunctival exudate covering the eyes. Microscopically, the turtles had fibrinonecrotic inflammation around the glottal opening, tracheitis, and severe bronchopneumonia and interstitial pneumonia. In mult
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Atienza-Mateo, B., A. Serrano-Combarro, N. Del-Val, et al. "OP0126 LONG-TERM FOLLOW UP OF ABATACEPT IN USUAL INTERSTITIAL PNEUMONIA ASSOCIATED TO RHEUMATOID ARTHRITIS. NATIONAL MULTICENTER STUDY OF 172 PATIENTS." Annals of the Rheumatic Diseases 82, Suppl 1 (2023): 82–83. http://dx.doi.org/10.1136/annrheumdis-2023-eular.4912.

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BackgroundInterstitial lung disease (ILD) is a severe extra-articular manifestation of rheumatoid arthritis (RA). Usual interstitial pneumonia (UIP) is the most frequent, and severe ILD pattern in RA. Abatacept (ABA) has demonstrated effectiveness in RA-ILD during a 12-month period of treatment[1-2].ObjectivesTo assess the effectiveness and safety of ABA in RA-ILD patients with radiological pattern of UIP during a long-term follow-up.MethodsFrom a large observational multicenter study of 392 RA-ILD patients treated with ABA, we selected those with UIP. We analyzed from baseline the following o
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deMello, Daphne E. "Interstitial plasma cell pneumonia." Journal of Pediatrics 146, no. 2 (2005): 233. http://dx.doi.org/10.1016/j.jpeds.2004.10.051.

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Hervas, J. A., V. Henales, S. Serra, J. E. Serra, and J. M. Mas. "MEASLES GIANT CELL PNEUMONIA." Pediatric Infectious Disease Journal 9, no. 7 (1990): 529. http://dx.doi.org/10.1097/00006454-199007000-00022.

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Mai, Kien T. "Giant Renomedullary Interstitial Cell Tumor." Journal of Urology 151, no. 4 (1994): 986–88. http://dx.doi.org/10.1016/s0022-5347(17)35143-1.

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Ichimura, Takenori, Miwa Hinata, Daisuke Ichikura, and Shinya Suzuki. "Effectiveness of corticosteroids on immune checkpoint inhibitor-induced interstitial pneumonia among patients with a history of interstitial pneumonia: A case series." SAGE Open Medical Case Reports 9 (January 2021): 2050313X2110313. http://dx.doi.org/10.1177/2050313x211031313.

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There are few reports on the effectiveness of corticosteroids for immune checkpoint inhibitor-induced interstitial pneumonia in patients with a history of interstitial pneumonia. We report on 10 non-small cell lung cancer patients with a history of interstitial pneumonia who experienced immune checkpoint inhibitor-induced interstitial pneumonia. The immune checkpoint inhibitor-induced interstitial pneumonia lasted for a median duration of 41.5 days (range = 22–127 days). Eight of the ten patients responded to corticosteroid monotherapy; one patient responded to corticosteroids and the immunosu
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Ichimura, Takenori, Miwa Hinata, Daisuke Ichikura, and Shinya Suzuki. "Safety of immune checkpoint inhibitors in non-small-cell lung cancer patients with idiopathic interstitial pneumonia: a matched case–control study." Cancer Chemotherapy and Pharmacology 89, no. 1 (2021): 21–30. http://dx.doi.org/10.1007/s00280-021-04362-7.

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Abstract Purpose The immune checkpoint inhibitor nivolumab is commonly used for non-small-cell lung cancer treatment. Immune checkpoint inhibitors cause immune-related adverse events, including interstitial pneumonia. However, there are no studies on the risk factors for interstitial pneumonia exacerbation after immune checkpoint inhibitor administration in patients with a history of different types of interstitial pneumonia. Therefore, we aimed to investigate the risk factors for interstitial pneumonia exacerbation in patients with non-small-cell lung cancer and a history of interstitial pneu
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Ichimura, Takenori, Miwa Hinata, Daisuke Ichikura, and Shinya Suzuki. "Safety of immune checkpoint inhibitors in non-small-cell lung cancer patients with idiopathic interstitial pneumonia: a matched case–control study." Cancer Chemotherapy and Pharmacology 89, no. 1 (2021): 21–30. http://dx.doi.org/10.1007/s00280-021-04362-7.

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Abstract Purpose The immune checkpoint inhibitor nivolumab is commonly used for non-small-cell lung cancer treatment. Immune checkpoint inhibitors cause immune-related adverse events, including interstitial pneumonia. However, there are no studies on the risk factors for interstitial pneumonia exacerbation after immune checkpoint inhibitor administration in patients with a history of different types of interstitial pneumonia. Therefore, we aimed to investigate the risk factors for interstitial pneumonia exacerbation in patients with non-small-cell lung cancer and a history of interstitial pneu
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Churg, Andrew. "Centrilobular Fibrosis in Fibrotic (Chronic) Hypersensitivity Pneumonitis, Usual Interstitial Pneumonia, and Connective Tissue Disease–Associated Interstitial Lung Disease." Archives of Pathology & Laboratory Medicine 144, no. 12 (2020): 1509–16. http://dx.doi.org/10.5858/arpa.2019-0628-ra.

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Context.— Various pulmonary diseases can produce centrilobular (peribronchiolar) fibrosis, which may be isolated or associated with other patterns of more diffuse fibrosis. The major forms of interstitial lung disease in which centrilobular fibrosis is found are fibrotic (chronic) hypersensitivity pneumonitis, connective tissue disease–associated interstitial lung disease, and (a disputed issue) usual interstitial pneumonia/idiopathic interstitial fibrosis. Objective.— To review recent literature that addresses separation of these entities. Data Sources.— Data comprised recent publications. Co
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Yamaguchi, Nobuko, Hiroyuki Takatsuka, Takeshi Wakae, et al. "Idiopathic interstitial pneumonia following stem cell transplantation." Clinical Transplantation 17, no. 4 (2003): 338–46. http://dx.doi.org/10.1034/j.1399-0012.2003.00053.x.

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Kontorshchikov, A. S., A. L. Chernyaev, O. A. Vasyukova, and Zh R. Omarova. "Hypersensitivity pneumonitis complicated by acute respiratory viral infection." CLINICAL AND EXPERIMENTAL MORPHOLOGY 9, no. 3 (2020): 68–73. http://dx.doi.org/10.31088/cem2020.9.3.68-73.

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The article presents a clinical case of hypersensitivity pneumonitis complicated by a viral infection in a 72-year-old woman. The introductory part provides a concise literature review on the etiology, classification, frequency, and possible complications of hypersensitivity pneumonitis. The patient’s computed tomography (CT) lung scan showed bilateral interstitial lesions. The patient was diagnosed with idiopathic pulmonary fibrosis based on the clinical manifestations and radiological findings. The autopsy revealed morphological signs of hypersensitivity pneumonitis (obliterative bronchiolit
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Fujimura, Taku, Tadashi Terui, and Hachiro Tagami. "Disseminated Papular Interstitial Elastolytic Giant Cell Granuloma." Acta Dermato-Venereologica 83, no. 3 (2003): 234–35. http://dx.doi.org/10.1080/00015550310007337.

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Lee, S. H., H. K. Min, and H. R. Kim. "POS0908 NAILFOLD CAPILLAROSCOPY FINDINGS OF INTERSTITIAL PNEUMONIA WITH AUTOIMMUNE FEATURES (IPAF)." Annals of the Rheumatic Diseases 82, Suppl 1 (2023): 763.1–763. http://dx.doi.org/10.1136/annrheumdis-2023-eular.1932.

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BackgroundObjectives:We evaluated nailfold capillaroscopy (NFC) of interstitial pneumonia with autoimmune features (IPAF) and compared it with that of patients with CTD-ILD and idiopathic interstitial pneumonia (IIP).MethodsPatients with newly diagnosed as ILD were evaluated using NFC. Baseline demographic, clinical, serological, and high-resolution CT findings were collected. NFC was semi-quantitatively scored with six domains ranging from 0 to 18. In addition, the overall patterns (scleroderma/non-scleroderma patterns) were determined.ResultsA total of 81 patients (31 with CTD-ILD, 18 with I
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Haram, Kjell, and Knut Jacobsen. "MEASLES AND ITS RELATIONSHIP TO GIANT CELL PNEUMONIA (HECHT PNEUMONIA)." Acta Pathologica Microbiologica Scandinavica Section A Pathology 81A, no. 6 (2009): 761–69. http://dx.doi.org/10.1111/j.1699-0463.1973.tb03569.x.

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Shimizu, Tohru, Noriko Fukushima, Yoshiro Ebihara, Tetsutaro Sata, and Yuzo Aoyama. "GIANT CELL PNEUMONIA IN LETTERER-SIWE DISEASE." Pathology International 37, no. 3 (1987): 493–501. http://dx.doi.org/10.1111/j.1440-1827.1987.tb00383.x.

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Fontana, Vincenzo, Elio Donna, Pamela Dudkiewicz, Gabriella Lander, and Yeon S. Ahn. "Interstitial Pneumonia in Patients with Idhiopatic Thrombocytopenic Purpura." Blood 104, no. 11 (2004): 3953. http://dx.doi.org/10.1182/blood.v104.11.3953.3953.

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Abstract INTRODUCTION: Idhiopatic thrombocytopenic purpura (ITP) is an autoimmune disease characterized by a premature destruction of platelets by macrophage, especially in the spleen. However in some cases, platelet sequestration and destruction may occur in other organs. Chromium labeled platelet sequestration study revealed that liver or precordial area are prominent sites of sequestration in some cases, suggesting that the lung might be the site in certain cases. Some cases of interstitial pneumonia are associated with immunologic injury to the lung and seen in patients with some autoimmun
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Novoa-Bolivar, Erika M., José A. Ros, Sonia Pérez-Fernández, et al. "Diagnostic Utility of Bronchoalveolar Lavage Flow Cytometric Leukocyte Profiling in Interstitial Lung Disease and Infection." Biomolecules 15, no. 4 (2025): 597. https://doi.org/10.3390/biom15040597.

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Interstitial lung diseases (ILD) represent a diverse group of disorders that primarily affect the pulmonary interstitium and, less commonly, involve the alveolar and vascular epithelium. Overlapping clinical, radiological and histopathological features make proper classification difficult, requiring multiple complementary methodologies, including flow cytometry of bronchoalveolar lavages (BAL). This retrospective study analyzed BAL flow cytometry data from 1074 real-life patients, quantifying alveolar macrophages, CD4/CD8 lymphocytes, neutrophils, eosinophils, and CD1a+ Langerhans cells, with
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Majeski, James A., J. Dwight Stinnett, and Deborah J. Cameron. "Suppressor T cell activity in lymphoid interstitial pneumonia." Journal of Surgical Oncology 34, no. 1 (1987): 61–63. http://dx.doi.org/10.1002/jso.2930340115.

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Jiramethee, Norlalak, David Erasmus, Lawrence Nogee, and Andras Khoor. "Pulmonary Neuroendocrine Cell Hyperplasia Associated with Surfactant Protein C Gene Mutation." Case Reports in Pulmonology 2017 (2017): 1–6. http://dx.doi.org/10.1155/2017/9541419.

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Familial interstitial lung disease (ILD) is defined as presence of ILD in 2 or more family members. Surfactant protein C (SFTPC) gene mutations are rare, but well-known cause of familial ILD. We reported a 20-year-old male, who was referred for lung transplantation. He was symptomatic at age 3 and underwent surgical lung biopsy at age 6, which revealed a nonspecific interstitial pneumonia (NSIP) pattern. Genetic workup revealed a novel SFTPC mutation in the first intron with a C to A transversion. At age 21, he underwent bilateral lung transplantation. Explanted lung histology suggested NSIP.
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Zhang, Lei, Tian Xie, Yaqing Li, et al. "Diagnostic value and safety of medical thoracoscopy under local anesthesia for unexplained diffuse interstitial lung disease: A retrospective study." Chronic Respiratory Disease 19 (January 2022): 147997312211333. http://dx.doi.org/10.1177/14799731221133389.

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Objective We aimed to explore the safety and diagnostic value of medical thoracoscopic lung biopsy in patients with unexplained diffuse interstitial lung disease (ILD) in a single center pilot study. Method We retrospectively analyzed clinical and pathological diagnostic data from 52 patients with diffuse ILD undergoing medical thoracoscopic lung biopsy. Results Forty-four cases of diffuse ILD were confirmed pathologically, giving a diagnostic rate of 84.6%. Among these 44 patients, 11 patients were diagnosed with cancer, including eight patients with lung adenocarcinoma, three patients with m
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Kimura-Tsuchiya, Reiko, Eisaku Sasaki, Izumi Nakamura, et al. "A Case of Squamous Cell Carcinoma of Unknown Primary that Responded to the Multi-Tyrosine Kinase Inhibitor Lenvatinib." Case Reports in Oncology 11, no. 1 (2018): 75–80. http://dx.doi.org/10.1159/000486569.

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Lenvatinib is an oral tyrosine kinase inhibitor of vascular endothelial growth factor receptors 1, 2, and 3, fibroblast growth factor receptors 1 through 4, as well as platelet-derived growth factor receptor α, RET, and KIT. At present, lenvatinib is used in the treatment of thyroid cancer and renal cell carcinoma. We herein report a case of a 67-year-old patient with squamous cell carcinoma of unknown primary who was effectively treated with lenvatinib. The patient was initially diagnosed as having undifferentiated thyroid cancer, and after total thyroidectomy and bilateral lymph node dissect
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Ommurugan, Balaji, Amita Priya, Navin Patil, and Joseph Thomas. "BLEOMYCIN-INDUCED REVERSIBLE ACUTE INTERSTITIAL PNEUMONIA." Asian Journal of Pharmaceutical and Clinical Research 10, no. 5 (2017): 6. http://dx.doi.org/10.22159/ajpcr.2017.v10i5.17121.

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As of now, about 380 medications are implicated in causing respiratory reactions and most common among that is drug-induced interstitial disease.Oral, parental as well inhalational drugs are known to cause drug-induced interstitial lung disease. Bleomycin is a chemotherapeutic agent used inthe treatment of lymphomas, germ cell tumors of the testes. Most common pulmonary toxicity is diffuse alveolar damage with nonspecific interstitialpneumonitis being next. We report a case of bleomycin-induced reversible acute interstitial pneumonia in a Hodgkin’s lymphoma patient with adriamycin, bleomycin,
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WADA, Ryuichi, Takumi KANEMARU, Mikihiro KANEKO, Masa-aki OIKAWA, and Toyohiko YOSHIHARA. "Idiopathic Interstitial Pneumonia Associated with Giant Cells in Aborted Equine Fetus: Case Report." Journal of Equine Science 7, no. 1 (1996): 13–15. http://dx.doi.org/10.1294/jes.7.13.

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Chung, Augustine, May Lin Wilgus, Gregory Fishbein, and Joseph P. Lynch. "Pulmonary and Bronchiolar Involvement in Sjogren's Syndrome." Seminars in Respiratory and Critical Care Medicine 40, no. 02 (2019): 235–54. http://dx.doi.org/10.1055/s-0039-1688448.

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AbstractSjogren's syndrome (SS) is a chronic autoimmune disease characterized by mononuclear cells (principally lymphocytes) infiltrating exocrine glands (e.g., salivary and lacrimal glands), leading to destruction of exocrine epithelial cells and dryness of mucosal surfaces. Cardinal symptoms are dry eyes (xerophthalmia) and dry mouth (xerostomia). Extraglandular sites are affected in 30 to 40% of cases of SS (particularly neurological, kidneys, skin, and lungs). B cell hyperactivity, autoantibody production, and hypergammaglobulinemia are cardinal features of SS. Primary SS is not associated
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