Gotowe bibliografie tematyczne / Gotto Kakizaki rat

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  1. Artykuły w czasopismach
  2. Rozprawy doktorskie
  3. Części książek
  4. Streszczenia konferencji

Gotowa bibliografia na temat „Gotto Kakizaki rat”

Autor: Grafiati
Data publikacji: 2 czerwca 2025
Data aktualizacji: 19 lipca 2025

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Artykuły w czasopismach na temat "Gotto Kakizaki rat"

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Jin, Yong-Xie, Haeng-Ryan Kim, and So-Young Kim. "Effect of Mineral-rich Salt Intake on Diabetic Goto-Kakizaki Rats." Journal of the Korean Society of Food Science and Nutrition 43, no. 3 (2014): 355–59. http://dx.doi.org/10.3746/jkfn.2014.43.3.355.

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Abd-Elrahman, Khaled S., Olaia Colinas, Emma J. Walsh, et al. "Abnormal myosin phosphatase targeting subunit 1 phosphorylation and actin polymerization contribute to impaired myogenic regulation of cerebral arterial diameter in the type 2 diabetic Goto-Kakizaki rat." Journal of Cerebral Blood Flow & Metabolism 37, no. 1 (2016): 227–40. http://dx.doi.org/10.1177/0271678x15622463.

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The myogenic response of cerebral resistance arterial smooth muscle to intraluminal pressure elevation is a key physiological mechanism regulating blood flow to the brain. Rho-associated kinase plays a critical role in the myogenic response by activating Ca2+ sensitization mechanisms: (i) Rho-associated kinase inhibits myosin light chain phosphatase by phosphorylating its targeting subunit myosin phosphatase targeting subunit 1 (at T855), augmenting 20 kDa myosin regulatory light chain (LC20) phosphorylation and force generation; and (ii) Rho-associated kinase stimulates cytoskeletal actin pol
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Zong-Chao, Ling, Suad Efendic, Rolf Wibom, et al. "Glucose Metabolism in Goto-Kakizaki Rat Islets*." Endocrinology 139, no. 6 (1998): 2670–75. http://dx.doi.org/10.1210/endo.139.6.6053.

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Abstract Islets from Goto-Kakizaki (GK) rats from our colony, despite marked impairment of glucose-induced insulin release, used glucose and produced CO2 at a rate 3 times that of islets from control Wistar rats. Almost all glucose used was accounted for in CO2 and lactate production. The percentages of glucose carbon used collected in CO2 and lactate were similar for control and GK islets. GK islets also oxidized 40% more acetate and leucine to CO2 than did control islets. The fraction of carbon leaving the Krebs cycle relative to CO2 production was the same in GK and control islets. The capa
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El-Omar, Magdi M., Zhao-Kang Yang, Aled O. Phillips, and Ajay M. Shah. "Cardiac dysfunction in the Goto-Kakizaki rat." Basic Research in Cardiology 99, no. 2 (2004): 133–41. http://dx.doi.org/10.1007/s00395-004-0440-4.

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Rose, Tobias, Suad Efendic, and Marjan Rupnik. "Ca2+–Secretion Coupling Is Impaired in Diabetic Goto Kakizaki rats." Journal of General Physiology 129, no. 6 (2007): 493–508. http://dx.doi.org/10.1085/jgp.200609604.

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The Goto Kakizaki (GK) rat is a widely used animal model to study defective glucose-stimulated insulin release in type-2 diabetes (T2D). As in T2D patients, the expression of several proteins involved in Ca2+-dependent exocytosis of insulin-containing large dense-core vesicles is dysregulated in this model. So far, a defect in late steps of insulin secretion could not be demonstrated. To resolve this apparent contradiction, we studied Ca2+–secretion coupling of healthy and GK rat β cells in acute pancreatic tissue slices by assessing exocytosis with high time-resolution membrane capacitance me
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Guest, PC, SM Abdel-Halim, DJ Gross, et al. "Proinsulin processing in the diabetic Goto-Kakizaki rat." Journal of Endocrinology 175, no. 3 (2002): 637–47. http://dx.doi.org/10.1677/joe.0.1750637.

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The biosynthesis and processing of proinsulin was investigated in the diabetic Goto-Kakizaki (GK) rat. Immunofluorescence microscopy comparing GK and Wistar control rat pancreata revealed marked changes in the distribution of alpha-cells and pronounced beta-cell heterogeneity in the expression patterns of insulin, prohormone convertases PC1, PC2, carboxypeptidase E (CPE) and the PC-binding proteins 7B2 and ProSAAS. Western blot analyses of isolated islets revealed little difference in PC1 and CPE expression but PC2 immunoreactivity was markedly lower in the GK islets. The processing of the PC2
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Kim, Dae-Jung, Mi-Ja Chung, Jin-Kyoun You, Dong-Joo Seo, Jeong-Mi Kim, and Myeon Choe. "Effect of Medicinal Plant Water Extracts on Glucose-regulating Enzyme Activities in Goto-Kakizaki Rat Liver Cytosol." Journal of the Korean Society of Food Science and Nutrition 38, no. 10 (2009): 1331–35. http://dx.doi.org/10.3746/jkfn.2009.38.10.1331.

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Agardh, Carl-David, Elisabet Agardh, Hui Zhang, and Claes-Göran Östenson. "Altered endothelial/pericyte ratio in Goto-Kakizaki rat retina." Journal of Diabetes and its Complications 11, no. 3 (1997): 158–62. http://dx.doi.org/10.1016/s1056-8727(96)00049-9.

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Ahmad, T., C. Ohlsson, M. Saaf, CG Ostenson, and A. Kreicbergs. "Skeletal changes in type-2 diabetic Goto-Kakizaki rats." Journal of Endocrinology 178, no. 1 (2003): 111–16. http://dx.doi.org/10.1677/joe.0.1780111.

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We characterized appendicular and axial bones in rats with type-2 diabetes in five female Goto-Kakizaki (GK) rats, a strain developed from the Wistar rat showing spontaneous type-2 diabetes, and five age- and sex-matched non-diabetic Wistar rats. The humerus, tibia, metatarsals and vertebral bodies were analysed by peripheral quantitative computerized tomography (pQCT). In diabetic rats, the height of the vertebral bodies and length of the humerus were decreased while the length of the metatarsals was increased. A decreased cross-sectional area was found in the vertebral end-plate region and t
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D'Souza, Alicia, Frank C. Howarth, Joseph Yanni, et al. "Left ventricle structural remodelling in the prediabetic Goto-Kakizaki rat." Experimental Physiology 96, no. 9 (2011): 875–88. http://dx.doi.org/10.1113/expphysiol.2011.058271.

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Rozprawy doktorskie na temat "Gotto Kakizaki rat"

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Sloan, Ruben Carnell Gavin Timothy P. "Effect of duodenal-jejunal bypass on skeletal muscle insulin signaling in Goto-Kakizaki rats." [Greenville, N.C.] : East Carolina University, 2009. http://hdl.handle.net/10342/1893.

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Thesis (M.S.)--East Carolina University, 2009.<br>Presented to the faculty of the Department of Exercise and Sport Science. Advisor: Timothy P. Gavin. Title from PDF t.p. (viewed May 25, 2010). Includes bibliographical references.
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Kondo, Hiroyo. "Effects of exercise on capillary regression and inhibitory expression of angiogenic factors in the rat skeletal muscle of type 2 diabetes." Kyoto University, 2015. http://hdl.handle.net/2433/200474.

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Kyoto University (京都大学)<br>0048<br>新制・論文博士<br>博士(人間・環境学)<br>乙第12951号<br>論人博第42号<br>新制||人||178(附属図書館)<br>27||論人博||42(吉田南総合図書館)<br>32250<br>三重大学大学院医学系研究科生命医科学専攻<br>(主査)教授 石原 昭彦, 教授 神﨑 素樹, 准教授 久代 恵介, 准教授 月浦 崇, 教授 藤野 英己<br>学位規則第4条第2項該当
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Pitasi, Caterina Luana. "Implication of GSK3β in Islet Inflammation During Diabetes". Thesis, Sorbonne Paris Cité, 2017. http://www.theses.fr/2017USPCC251.

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Le diabète est une maladie chronique avec une progression alarmante. L’insuline-résistance et la diminution de la masse fonctionnelle des cellules beta, associée à l'inflammation des îlots, sont les principaux défauts impliqués dans la pathogenèse du diabète de type 2 (DT2). La compréhension des mécanismes impliqués dans l'inflammation des îlots pancréatiques, et l'identification de cibles moléculaires à visée anti-inflammatoire, sont des approches intéressantes pour le traitement du diabète. La glycogène synthase kinase 3 (GSK3), est une sérine-thréonine kinase qui régule des fonctions cellul
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Kominato, Rieko. "Src activation generates reactive oxygen species and impairs metabolism-secretion coupling in diabetic Goto-Kakizaki and ouabain-treated rat pancreatic islets." Kyoto University, 2008. http://hdl.handle.net/2433/124247.

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Brierley, Rachel Claire Mary. "Effects of type 2 diabetes mellitus on cognitive performance, synaptic plasticity and glutamate receptors in Goto–Kakizaki rats." Thesis, University of Bristol, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.495650.

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Jype 2 diabetes (2DM) is linked with cognitive deficits in aged human patients with poor diabetic control. Conflicting evidence exists from studies Into cognitive deficits using streptozotocin (ST Z) induced and spontaneous diabetic models; furthermore, the mechanism for cognitive deficits is unclear. The Goto-Kakizaki (GK) rat, a spontaneous non-obese model of 2DM, was selected to isolate the effects of prolonged hyperglycaemia on brains of aged GK rats using behavioural tasks, electrophysiology and immunochemistry, compared with age-matched Wistar control rats.
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Kundrotienė, Jurgita. "Ischemic brain damage following transient and moderate compression of sensorimotor cortex in Sprague-Dawley and diabetic Goto-Kakizaki rats /." Stockholm, 2004. http://diss.kib.ki.se/2004/91-7349-819-X/.

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Wallis, Robert H. "Marker assisted construction of congenic lines for the Goto Kakizaki rat in order to elucidate the genetic control of type 2 diabetes mellitus." Thesis, University of Oxford, 2001. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.393991.

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Persdotter, Hedlund Gabriella. "Protein and mRNA Studies of Rat FA1/Pref-1/dlk." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-7773.

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Eickhoff, Hans Christian August. "Impact of bariatric surgery on control of type 2 diabetes : neuroendocrine mechanisms and clinical significance." Doctoral thesis, 2015. http://hdl.handle.net/10316/26409.

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Tese de doutoramento em Ciências da Saúde (Pré-Bolonha), Ramo de Medicina, especialidade de Cirurgia (Cirurgia), apresentada à Faculdade de Medicina da Universidade de Coimbra<br>Background: Bariatric and metabolic surgery is an accepted treatment for obese patients with type 2 diabetes (T2D). However, the potential of gastrointestinal surgery regarding glycemic control in non-severely obese diabetic patients has yet to be defined. Along with weight loss itself, changes in gut hormone profiles after surgery play an important role in diabetes remission. Pathophysiology of T2D includes insulin r
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Części książek na temat "Gotto Kakizaki rat"

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Östenson, Claes-Göran, Samy M. Abdel-Halim, Arne Andersson, and Suad Efendic. "Studies on the pathogenesis of NIDDM in the GK (Goto-Kakizaki) rat." In Lessons from Animal Diabetes VI. Birkhäuser Boston, 1996. http://dx.doi.org/10.1007/978-1-4612-4112-6_17.

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Guest, Paul C. "Characterization of the Goto-Kakizaki (GK) Rat Model of Type 2 Diabetes." In Methods in Molecular Biology. Springer New York, 2018. http://dx.doi.org/10.1007/978-1-4939-8994-2_19.

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Santos, Dario Loureiro, Carlos Marques Palmeira, Raquel Seiça, et al. "Diabetes and mitochondrial oxidative stress: A study using heart mitochondria from the diabetic Goto-Kakizaki rat." In Vascular Biochemistry. Springer US, 2003. http://dx.doi.org/10.1007/978-1-4615-0298-2_23.

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"THE GOTO-KAKIZAKI RAT." In Animal Models in Diabetes. CRC Press, 2000. http://dx.doi.org/10.4324/9780203304730-17.

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"The Goto–Kakizaki Rat." In Animal Models of Diabetes. CRC Press, 2007. http://dx.doi.org/10.1201/9781420009453-9.

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Chen, Ai, Myungsoo Ko, Vay Liang W. Go, Moon K. Song, Kaitlyn Liu, and Hong Yang. "Anorexia-Cachexia in Type 2 Diabetic (T2D) Goto-Kakizaki (GK) Rats: Role of Impaired Brainstem Thyrotropin-Releasing Hormone (TRH) Regulation of Vagal Function." In The Endocrine Society's 92nd Annual Meeting, June 19–22, 2010 - San Diego. Endocrine Society, 2010. http://dx.doi.org/10.1210/endo-meetings.2010.part1.p10.p1-483.

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Streszczenia konferencji na temat "Gotto Kakizaki rat"

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Guanying Piao, Bangguo Qian, Shigeru Saito, et al. "Phenotype-difference oriented identification of molecular functions for diabetes progression in Goto-Kakizaki rat." In 2011 IEEE International Conference on Systems Biology (ISB). IEEE, 2011. http://dx.doi.org/10.1109/isb.2011.6033130.

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Saito, Shigeru, Yidan Sun, Zhi-Ping Liu, et al. "Identification of master regulator candidates for diabetes progression in Goto-Kakizaki Rat by a computational procedure." In 2011 IEEE International Conference on Systems Biology (ISB). IEEE, 2011. http://dx.doi.org/10.1109/isb.2011.6033155.

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