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Artykuły w czasopismach na temat „GRP”

Autor: Grafiati

Data publikacji: 4 czerwca 2021

Data aktualizacji: 25 lipca 2025

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1

Bányai, Krisztián, Ágnes Bogdán, György Szücs, et al. "Assignment of the group A rotavirus NSP4 gene into genotypes using a hemi-nested multiplex PCR assay: a rapid and reproducible assay for strain surveillance studies." Journal of Medical Microbiology 58, no. 3 (2009): 303–11. http://dx.doi.org/10.1099/jmm.0.005124-0.

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The rotavirus non-structural protein NSP4 has been implicated in a number of biological functions during the rotavirus cellular cycle and pathogenesis, and has been addressed as a target for vaccine development. The NSP4 gene has been classified into six genotypes (A–F). A semi-nested triplex PCR was developed for genotyping the major human NSP4 genotypes (A–C), which are common in human rotavirus strains but are also shared among most mammalian rotavirus strains. A total of 192 previously characterized human strains representing numerous G and P type specificities (such as G1P[8], G1P[4], G2P

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2

Glover, Sarah, Rajkumar Nathaniel, Lubna Shakir, et al. "Transient upregulation of GRP and its receptor critically regulate colon cancer cell motility during remodeling." American Journal of Physiology-Gastrointestinal and Liver Physiology 288, no. 6 (2005): G1274—G1282. http://dx.doi.org/10.1152/ajpgi.00108.2004.

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Gastrin-releasing peptide (GRP) is typically viewed as a growth factor in cancer. However, we have suggested that in colon cancer, GRP acts primarily as a morphogen when it and its receptor (GRP-R) are aberrantly upregulated. As such, GRP/GRP-R act(s) primarily to modulate processes contributing to the assumption or maintenance of tumor differentiation. One of the most important such processes is the ability of tumor cells to achieve directed motility in the context of tissue remodeling. Yet the cellular conditions affecting GRP/GRP-R expression, and the biochemical pathways involved in mediat

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3

Koceić Bilan, Nikola, and Ivančica Mirošević. "Functorial reducing pro⁎-Grp category to pro-Grp." Topology and its Applications 263 (August 2019): 74–89. http://dx.doi.org/10.1016/j.topol.2019.05.018.

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4

Jeffry Jamil, Eliza M. Yusup, and Shahrul Azmir Osman. "Non-Destructive Testing (NDT) Method for Defect Detection in Glass Fibre Reinforced Plastic/Polymer (GFRP/GRP) Composite Materials Structures: A Review." Journal of Advanced Research in Micro and Nano Engieering 17, no. 1 (2024): 76–95. http://dx.doi.org/10.37934/armne.17.1.7695.

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Various industries are considering metal product in lieu composite materials. Structural engineers appreciate glass fibre reinforced plastic/polymer (GFRP/GRP) because to its high modulus of elasticity, ratio of strength to weight, and corrosion resistance features. It has been discovered that structural engineering defects to be leading cause for the catastrophic consequences. This paper provides an overview of available NDT methods that can be employed to assess the quality of GFRP/GRP composite materials. The most common NDT method used by researchers and practitioners is also discussed, al

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5

Левкина and Nataliya Levkina. "The Contribution of New Postindustrial Technological Modes to per Capita GRP of the Urals Federal District`s Regions of the Russian Federation." Economics 4, no. 6 (2016): 63–67. http://dx.doi.org/10.12737/22928.

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The article presents the results of the analysis of technological modes&#96; productivity in the economy of the Urals Federal District. The use of evaluation of contribution to per capita GDP industrial (relict and fourth) technological modes and specified data on the gross regional product has allowed to establish that the spread of new postindustrial technological modes in the economy of different regions of the Urals Federal District is uneven. In Kurgan region the postindustrial technological modes are not widespread. The contribution of new modes in per capita GRP in Chelyabinsk regio

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6

Lee, Young-Geun, Sun-Hee Kim, Joon-Seok Park, and Soon-Jong Yoon. "Pipe Stiffness Prediction of GRP Flexible Pipe." Journal of the Korean Society for Advanced Composite Structures 2, no. 3 (2011): 18–24. http://dx.doi.org/10.11004/kosacs.2011.2.3.018.

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7

Hollaway, L. "GRP and Buildings." Composites 16, no. 1 (1985): 56. http://dx.doi.org/10.1016/0010-4361(85)90662-7.

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8

Oh, Jin-Oh, and Sung-Ho Yoon. "Measurements of Thermal Expansion Coefficients in GRP Pipe." Journal of The Korean Society for Composite Materials 25, no. 1 (2012): 26–30. http://dx.doi.org/10.7234/kscm.2012.25.1.026.

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9

Gawin, A. Z., J. N. Baraniuk, J. D. Lundgren, and M. Kaliner. "Effects of gastrin-releasing peptide and analogues on guinea pig nasal mucosal secretion." American Journal of Physiology-Lung Cellular and Molecular Physiology 264, no. 4 (1993): L345—L350. http://dx.doi.org/10.1152/ajplung.1993.264.4.l345.

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The effects of gastrin-releasing peptide (GRP), bombesin, GRP-(1–16) and GRP-(21–27) on guinea pig nasal mucosal secretion were studied in vivo. GRP, bombesin, and GRP-(21–27) induced significant secretion of total protein, albumin, and alkaline phosphatase. GRP induced significant secretion at lower concentrations (10(-11) and 10(-10) M) than were required for bombesin and GRP-(21–27) (10(-7) M). GRP-(1–16) did not stimulate secretion, indicating that the COOH-terminal region of GRP contained the secretagogic principle. Capsaicin, a stimulant of nociceptive sensory nerves, stimulated GRP rele

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10

Zakharzhevskaya, A. Yu. "Disparities of China’s regions development: dependence on the sectoral economic structure." Regional nye issledovaniya 71, no. 1 (2021): 84–95. http://dx.doi.org/10.5922/1994-5280-2021-1-7.

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The article explores the dynamics of changes in disparities of China’s regions’ economic level. According to a number of researchers, economic disparities between China’s regions started to decrease from about the mid-2000s. To clarify this picture the article uses indicator of contribution of the region’s GRP to the country’s total GRP. As a result of the research carried out by the author, it was found that in the PRC there really was a period when the economic gap between regions was narrowing down, but in general, changes in the relative contribution of regions to the country’s GRP are mor

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11

Kramin, T. V., and D. A. Ustyuzhanina. "Impact of GRP Per Capita on the Quality of Life of the Population in Russian Regions." Economy of regions 20, no. 1 (2024): 176–88. http://dx.doi.org/10.17059/ekon.reg.2024-1-12.

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Most countries consider dynamics of gross domestic product (GDP) as an indicator of economic policy success. Despite its importance and usefulness, GDP has limitations when it comes to assessing the situation in society and some economic development characteristics. The paper examines the relationship between gross regional product (GRP) per capita and the quality of life of the population in Russian regions. The impact of GRP on the quality of life was modelled on panel data using spatial fixed effects. As a result, well-specified models explaining differences in the dependent variable were i

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12

Traynor, T. R., and S. M. O'Grady. "Regulation of colonic ion transport by GRP. II. GRP modulates the epithelial response to PGE2." American Journal of Physiology-Cell Physiology 270, no. 3 (1996): C859—C865. http://dx.doi.org/10.1152/ajpcell.1996.270.3.c859.

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The purpose of this study was to examine the potential modulatory effects of gastrin-releasing peptide (GRP) on prostaglandin (PG) E2-stimulated electrolyte transport across the distal colon epithelium. In an earlier study, PGE2 was shown to reduce net Cl absorption without altering the serosal-to-mucosal unidirectional Cl flux in porcine distal colon (19). In the present study, tissues were pretreated with serosal or mucosal GRP and subsequently stimulated with PGE2. The resulting increase in short-circuit current (ISC) was 152% (serosal GRP) and 49% (mucosal GRP) greater than control PGE2 re

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13

Traynor, T. R., and S. M. O'Grady. "Regulation of colonic ion transport by GRP. I. GRP stimulates transepithelial K and Na secretion." American Journal of Physiology-Cell Physiology 270, no. 3 (1996): C848—C858. http://dx.doi.org/10.1152/ajpcell.1996.270.3.c848.

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Regulation of electrolyte transport across porcine distal colon epithelium by gastrin-releasing peptide (GRP) was examined using mucosal sheets mounted in Ussing chambers. Serosal GRP produced a biphasic response consisting of a transient increase in short-circuit current (ISC) followed by a long-lasting decrease. Indomethacin and tetrodotoxin inhibited the ISC increase without affecting the secondary decrease. Addition of GRP to the mucosal solution produced a decrease in ISC similar to that observed with serosal treatment, but no transient increase in ISC was observed. GRP and bombesin (50%

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14

Ladenheim, E. E., J. E. Taylor, D. H. Coy, K. A. Moore, and T. H. Moran. "Hindbrain GRP receptor blockade antagonizes feeding suppression by peripherally administered GRP." American Journal of Physiology-Regulatory, Integrative and Comparative Physiology 271, no. 1 (1996): R180—R184. http://dx.doi.org/10.1152/ajpregu.1996.271.1.r180.

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Bombesin (BN)-like peptides injected peripherally or centrally suppress food intake in rats. The relationship between the central and peripheral actions of BN is unknown. However, experimental evidence supports a critical role for the caudal hindbrain in mediating the feeding effects of BN. To investigate this relationship further, we examined the ability of fourth ventricular infusion of a specific gastrin-releasing peptide (GRP) antagonist, [D-F5, Phe6, D-Ala11]BN-(6-13) methyl ester (BN-ME), to block suppression of glucose intake (0.5 kcal/ml) produced by intraperitoneal administration of G

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15

García, B. Benítez, F. Moreno Ramos, MA González Fernández, et al. "GRP-035 Boceprevir and Telaprevir: Safety: Abstract GRP-035 Table 1." European Journal of Hospital Pharmacy 20, Suppl 1 (2013): A13.1—A13. http://dx.doi.org/10.1136/ejhpharm-2013-000276.035.

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16

Moreno, M., A. Gil, R. Diez, and T. Molina. "GRP-059 Ethanol Content in Chemotherapy: Abstract GRP-059 Table 1." European Journal of Hospital Pharmacy 20, Suppl 1 (2013): A21.3—A22. http://dx.doi.org/10.1136/ejhpharm-2013-000276.059.

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17

Kim, Sun-Hee, Young-Geun Lee, Hyung-Jung Joo, Nam-Jin Jung, and Soon-Jong Yoon. "Prediction of Ring Deflection GRP Pipe Buried Underground." Journal of the Korean Society for Advanced Composite Structures 4, no. 3 (2013): 38–44. http://dx.doi.org/10.11004/kosacs.2013.4.3.038.

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18

Whitley, JC, C. Moore, AS Giraud, and A. Shulkes. "Isolation and characterisation of the ovine gastrin-releasing peptide gene; abundant expression in the pregnant uterus and selective expression in fetal tissues." Journal of Endocrinology 175, no. 2 (2002): 447–57. http://dx.doi.org/10.1677/joe.0.1750447.

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High concentrations of a peptide related to gastrin-releasing peptide (GRP) are produced in the utero-placental unit of the human and sheep and secreted into the general circulation. This suggests an endocrine role in addition to its role as a neurotransmitter/neuromodulator. The GRP is larger than the previously described form GRP(1-27) but it is not known whether the larger form is the product of a related GRP-like gene or differences in post-translational processing. We have therefore cloned the gene for the sheep homologue of the GRP gene and determined its distribution. Only a single GRP

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19

Cardona, C., N. M. Bleehen, and J. G. Reeve. "Characterization of ligand binding and processing by gastrin-releasing peptide receptors in a small-cell lung cancer cell line." Biochemical Journal 281, no. 1 (1992): 115–20. http://dx.doi.org/10.1042/bj2810115.

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The ligand-binding properties of the gastrin-releasing peptide (GRP) receptor and the cellular processing of GRP have been studied in the small-cell lung cancer (SCLC) cell line COR-L42. Scatchard analysis of GRP receptor expression indicated a single class of high-affinity receptors (Kd 1.5 nM) and approx. 6700 receptors/cell. GRP bound to its receptor with a Ki of 2.4 nM. The bombesin-related peptides neuromedin B (NMB) and phyllolitorin also bound to GRP receptors with Ki values of 22.7 and 59.1 nM respectively. Binding of 125I-GRP to COR-L42 cells increased rapidly at 37 degrees, achieved

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20

Holst, J. J., S. Knuhtsen, C. Orskov, et al. "GRP nerves in pig antrum: role of GRP in vagal control of gastrin secretion." American Journal of Physiology-Gastrointestinal and Liver Physiology 253, no. 5 (1987): G643—G649. http://dx.doi.org/10.1152/ajpgi.1987.253.5.g643.

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We extracted gastrin-releasing peptide (GRP) and its C-terminal decapeptide corresponding to 6.4 and 6.8 pmol/g from pig antrum mucosa. By immunohistochemistry GRP was localized to mucosal, submucosal, and myenteric nerve fibers. A few nerve cell bodies were also identified. Using isolated perfused pig antrum with intact vagal innervation, we found concomitant, atropine-resistant release of GRP and gastrin during electrical stimulation of the vagal nerves. Intra-arterial GRP at 10(-11)-10(-10) mol/l caused up to fivefold, dose-dependent increases in gastrin secretion; higher doses were less ef

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21

Fleischmann, Achim, Beatrice Waser, and Jean Claude Reubi. "High expression of gastrin-releasing peptide receptors in the vascular bed of urinary tract cancers: promising candidates for vascular targeting applications." Endocrine-Related Cancer 16, no. 2 (2009): 623–33. http://dx.doi.org/10.1677/erc-08-0316.

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Tumoral gastrin-releasing peptide (GRP) receptors are potential targets for diagnosis and therapy using radiolabeled or cytotoxic GRP analogs. GRP-receptor overexpression has been detected in endocrine-related cancer cells and, more recently, also in the vascular bed of selected tumors. More information on vascular GRP-receptors in cancer is required to asses their potential for vascular targeting applications. Therefore, frequent human cancers (n=368) were analyzed using in vitro GRP-receptor autoradiography on tissue sections with the 125I-[Tyr4]-bombesin radioligand and/or the universal rad

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22

Persson, Kristin, Ronald L. Gingerich, Sonali Nayak, Keiji Wada, Etsuko Wada, and Bo Ahrén. "Reduced GLP-1 and insulin responses and glucose intolerance after gastric glucose in GRP receptor-deleted mice." American Journal of Physiology-Endocrinology and Metabolism 279, no. 5 (2000): E956—E962. http://dx.doi.org/10.1152/ajpendo.2000.279.5.e956.

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By applying a newly developed ELISA technique for determining biologically active intact glucagon-like peptide [GLP-1, GLP-1-(7–36)amide] in mouse, plasma baseline GLP-1 in normal NMRI mice was found to be normally distributed (4.5 ± 0.3 pmol/l; n = 72). In anesthetized mice, gastric glucose (50 or 150 mg) increased plasma GLP-1 levels two- to threefold ( P < 0.01). The simultaneous increase in plasma insulin correlated to the 10-min GLP-1 levels ( r = 0.36, P < 0.001; n = 12). C57BL/6J mice deleted of the gastrin-releasing peptide (GRP) receptor by genetic targeting had impaired glucose

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23

Matkowskyj, Kristina A., Kristin Keller, Sarah Glover, Lori Kornberg, Roger Tran-Son-Tay, and Richard V. Benya. "Expression of GRP and Its Receptor in Well-differentiated Colon Cancer Cells Correlates with the Presence of Focal Adhesion Kinase Phosphorylated at Tyrosines 397 and 407." Journal of Histochemistry & Cytochemistry 51, no. 8 (2003): 1041–48. http://dx.doi.org/10.1177/002215540305100807.

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Gastrin-releasing peptide (GRP) and its receptor (GRP-R) are not normally expressed by epithelial cells lining the colon but are aberrantly expressed in cancer, where they act as morphogens and regulate tumor cell differentiation. Studies of colon cancer formation in mice genetically incapable of synthesizing GRP-R suggested that this receptor's morphogenic properties were mediated via focal adhesion kinase (FAK). We therefore set out to determine the presence of both total and phosphorylated forms of FAK in human colon cancer specimens as a function of tumor cell differentiation and GRP/GRP-R

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24

Guo, Y. S., L. Mok, C. W. Cooper, G. H. Greeley, J. C. Thompson, and P. Singh. "Effect of gastrin-releasing peptide analogues on gastrin and somatostatin release from isolated rat stomach." American Journal of Physiology-Gastrointestinal and Liver Physiology 253, no. 2 (1987): G206—G210. http://dx.doi.org/10.1152/ajpgi.1987.253.2.g206.

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A differential biological potency of bombesin (BBS), compared with its mammalian counterpart gastrin-releasing peptide (GRP), has been reported in several biological systems in rodents. In the present study we have examined the relative potency of BBS, GRP-(1-27) (GRP-L), and GRP-(14-27) (GRP-S) on the release of gastrin and somatostatin (SRIF) from the isolated perfused rat stomachs. Male rats were fasted overnight and the stomachs perfused via the celiac artery. Increasing doses of BBS, GRP-L, and GRP-S were perfused for 15 min each and the effluent collected for measurement of gastrin and S

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25

Ostrovskii, Andrei V. "Regional Economic Development of the PRC through the Data of the World Economic Statistics." Population and Economics 8, no. 4 (2024): 123–37. https://doi.org/10.3897/popecon.8.e121879.

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The article analyzes the results of a forecast on the socio-economic development of Chinese regions made ten years ago in a collective monograph by the Institute of the Far East of the Russian Academy of Sciences. These results are compared with data from various countries in terms of gross domestic product (GDP) and GDP per capita. Based on statistical data from the National Bureau of Statistics (NBS) of China for 2022, it was determined that China’s GDP ranks first globally in terms of purchasing power parity (PPP) and second in terms of the dollar exchange rate. More than half of China’s GD

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26

Ostrovskii, Andrei V. "Regional Economic Development of the PRC through the Data of the World Economic Statistics." Population and Economics 8, no. (4) (2024): 123–37. https://doi.org/10.3897/popecon.8.e121879.

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The article analyzes the results of a forecast on the socio-economic development of Chinese regions made ten years ago in a collective monograph by the Institute of the Far East of the Russian Academy of Sciences. These results are compared with data from various countries in terms of gross domestic product (GDP) and GDP per capita. Based on statistical data from the National Bureau of Statistics (NBS) of China for 2022, it was determined that China's GDP ranks first globally in terms of purchasing power parity (PPP) and second in terms of the dollar exchange rate. More than half of China's GD

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27

Gontcharov, George A., and Aleksandr V. Mosenkov. "Gaia DR2 giants in the Galactic dust – II. Application of the reddening maps and models." Monthly Notices of the Royal Astronomical Society 500, no. 2 (2020): 2607–19. http://dx.doi.org/10.1093/mnras/staa2728.

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ABSTRACT We exploit a complete sample of 101 810 Gaia DR2 giants, selected in Paper I in the space cylinder with a radius of 700 pc around the Sun and a height of |Z| = 1800 pc, using the Gaia DR2 parallaxes, GBP and GRP photometry, and WISE W3 photometry. We explain the spatial variations of the modes of the observables GBP − GRP and GRP − W3 by the spatial variations of the corresponding reddenings described in the GM20 3D dust distribution model. Presented in this paper, GM20 is an advanced version of the model introduced by Gontcharov in 2009. GM20 proposes two intersecting dust layers, al

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Carroll, Robert E., Kristina A. Matkowskyj, Subrata Chakrabarti, Thomas J. McDonald, and Richard V. Benya. "Aberrant expression of gastrin-releasing peptide and its receptor by well-differentiated colon cancers in humans." American Journal of Physiology-Gastrointestinal and Liver Physiology 276, no. 3 (1999): G655—G665. http://dx.doi.org/10.1152/ajpgi.1999.276.3.g655.

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Epithelial cells lining the adult human colon do not normally express gastrin-releasing peptide (GRP) or its receptor (GRPR). In contrast, approximately one-third of human colon cancers and cancer cell lines have been shown to express GRP-binding sites. Because GRPR activation causes the proliferation of many cancer cell lines, GRP has been presumed to act as a clinically significant growth factor. Yet GRP has not been shown to be expressed by colon cancers in humans nor has the effect of GRP and/or GRPR coexpression on tumor behavior been investigated. We therefore determined GRP and GRPR exp

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Lundgren, J. D., J. N. Baraniuk, N. L. Ostrowski, M. A. Kaliner, and J. H. Shelhamer. "Gastrin-releasing peptide stimulates glycoconjugate release from feline trachea." American Journal of Physiology-Lung Cellular and Molecular Physiology 258, no. 2 (1990): L68—L74. http://dx.doi.org/10.1152/ajplung.1990.258.2.l68.

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The effect of gastrin-releasing peptide (GRP) on respiratory glycoconjugate (RGC) secretion was investigated in a feline tracheal organ culture model. RGC secretion was stimulated by GRP in a dose-dependent fashion at concentrations from 10(-8) to 10(-5) M (range 15-38% increase above control) with a peak effect within 0.5-1 h of incubation. GRP-(14-27), the receptor binding portion of GRP, and the related molecule, bombesin, also stimulated RGC secretion by approximately 20% above control. Acetyl-GRP-(20-27) stimulated RGC release by 10%, whereas GRP-(1-16) was inactive. Autoradiographic stud

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30

LeSauter, Joseph, Rae Silver, Robin Cloues, and Paul Witkovsky. "Light exposure induces short- and long-term changes in the excitability of retinorecipient neurons in suprachiasmatic nucleus." Journal of Neurophysiology 106, no. 2 (2011): 576–88. http://dx.doi.org/10.1152/jn.00060.2011.

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The suprachiasmatic nucleus (SCN) is the locus of a hypothalamic circadian clock that synchronizes physiological and behavioral responses to the daily light-dark cycle. The nucleus is composed of functionally and peptidergically diverse populations of cells for which distinct electrochemical properties are largely unstudied. SCN neurons containing gastrin-releasing peptide (GRP) receive direct retinal input via the retinohypothalamic tract. We targeted GRP neurons with a green fluorescent protein (GFP) marker for whole cell patch-clamping. In these neurons, we studied short (0.5–1.5 h)- and lo

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31

Arzhenovskiy, Sergey. "Estimate of shadow economy dynamics in Russia and regions: The inflationary aspect." Applied Econometrics 69 (2023): 121–40. http://dx.doi.org/10.22394/1993-7601-2023-69-121-140.

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The share of the shadow economy in the GDP of Russia and by region — in the GRP of the regions, was estimated in the study, based on quarterly data. The methodology included two approaches — structural modeling and using of the cash demand model. Estimation of consumer price index elasticity factors by the share of shadow economy in GDP/GRP was carried out. Comparison of empirical results of the study with obtained other authors and other methods, both by country and by region, showed their comparability.

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FELDMAN, M. "GRP and Pavlov's dogs." Gut 49, no. 1 (2001): 5–6. http://dx.doi.org/10.1136/gut.49.1.5.

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Pearce, Colin. "Carbon footprint of GRP." Reinforced Plastics 51, no. 8 (2007): 8. http://dx.doi.org/10.1016/s0034-3617(07)70229-1.

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Banks, W. M. "GRP in structural engineering." Thin-Walled Structures 3, no. 1 (1985): 88–89. http://dx.doi.org/10.1016/0263-8231(85)90025-4.

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Bashford, David. "GRP in structural engineering." Materials & Design 6, no. 3 (1985): 137. http://dx.doi.org/10.1016/0261-3069(85)90059-7.

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Roose, Jeroen P. "Lost GRP on cytotoxicity?" Nature Immunology 17, no. 12 (2016): 1339–40. http://dx.doi.org/10.1038/ni.3620.

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HOLST, J. J., L. BJØRNSKOV HANSEN, T. W. SCHWARTZ, M. HANSEN, and E. BORCH. "Nature and Release of Products of the GRP Precursor Other than GRP." Annals of the New York Academy of Sciences 547, no. 1 Bombesin-Like (1988): 443–44. http://dx.doi.org/10.1111/j.1749-6632.1988.tb23911.x.

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Holst, JJ, L. Bjørnskov Hansen, TW Schwartz, and M. Hansen. "Nature and release of products of the GRP-precursor apart from GRP." Regulatory Peptides 19, no. 1-2 (1987): 114. http://dx.doi.org/10.1016/0167-0115(87)90105-4.

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Lebacq-Verheyden, Anne-Marie, Shoshana Segal, Frank Cuttitta, and James F. Battey. "Swiss 3T3 mouse embryo fibroblasts transfected with a human prepro-GRP gene synthesize and secrete pro-GRP rather than GRP." Journal of Cellular Biochemistry 36, no. 3 (1988): 237–48. http://dx.doi.org/10.1002/jcb.240360305.

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Dumesny, Chelsea, Jane C. Whitley, Graham S. Baldwin, Andrew S. Giraud, and Arthur Shulkes. "Developmental expression and biological activity of gastrin-releasing peptide and its receptors in the kidney." American Journal of Physiology-Renal Physiology 287, no. 3 (2004): F578—F585. http://dx.doi.org/10.1152/ajprenal.00416.2003.

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Mammalian gastrin-releasing peptide (GRP) has a widespread distribution and multiple stimulating effects on metabolism, release of regulatory peptides, gastrointestinal and pancreatic secretions, and behavior. GRP is a potent mitogen for a number of tumor types, including colon and lung. Although GRP is known to stimulate the growth of renal tumors, little is known of its synthesis, distribution, and receptors in the developing and mature kidney. Both Northern blot analysis and RT-PCR revealed the presence of GRP mRNA in ovine kidney from midgestation through to adulthood. GRP mRNA was detecte

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Kusano, K., H. Gainer, J. F. Battey, Z. Fathi, and E. Wada. "Receptor-activated currents in mouse fibroblasts expressing transfected bombesin receptor subtype cDNAs." American Journal of Physiology-Cell Physiology 265, no. 4 (1993): C869—C876. http://dx.doi.org/10.1152/ajpcell.1993.265.4.c869.

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BALB/c 3T3 cells do not normally express receptors for bombesin-like peptides [bombesin (Bn), gastrin-releasing peptide (GRP), and neuromedin B (NmB)]. Transfection of BALB/c 3T3 cells with complementary DNA-encoding GRP receptors or NmB receptors leads to stable expression of functional GRP receptors (GRP Rt) or NmB receptors (NmB Rt), respectively, which are coupled to cell membrane ion channels. Whole cell current analysis using patch electrodes shows that the activation of these newly expressed receptors induces cation conductance increases, most frequently a Ca(2+)-activated plasma membra

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Qiao, Jingbo, Junquan Liu, Jillian C. Jacobson, et al. "Anti-GRP-R monoclonal antibody antitumor therapy against neuroblastoma." PLOS ONE 17, no. 12 (2022): e0277956. http://dx.doi.org/10.1371/journal.pone.0277956.

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Standard treatment for patients with high-risk neuroblastoma remains multimodal therapy including chemoradiation, surgical resection, and autologous stem cell rescue. Immunotherapy has demonstrated success in treating many types of cancers; however, its use in pediatric solid tumors has been limited by low tumor mutation burdens. Gastrin-releasing peptide receptor (GRP-R) is overexpressed in numerous malignancies, including poorly-differentiated neuroblastoma. Monoclonal antibodies (mAbs) to GRP-R have yet to be developed but could serve as a potential novel immunotherapy. This preclinical stu

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Ladenheim, EE, LL Hampton, AC Whitney, WO White, JF Battey, and TH Moran. "Disruptions in feeding and body weight control in gastrin-releasing peptide receptor deficient mice." Journal of Endocrinology 174, no. 2 (2002): 273–81. http://dx.doi.org/10.1677/joe.0.1740273.

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Bombesin (BN) interacts with two mammalian receptor subtypes termed gastrin-releasing peptide (GRP)-preferring (GRP-R) and neuromedin B (NMB)-preferring (NMB-R) that may mediate the satiety action of BN. We examined the feeding behavior of mice that were deficient in the GRP-R (GRP-R KO) to assess the overall contribution of this receptor subtype in the feeding actions of BN-related peptides. GRP-R KO mice failed to suppress glucose intake in response to systemically administered BN and GRP(18-27), whereas both peptides elicited a potent reduction of intake in wild-type (WT) mice. Neither GRP-

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Pinski, J., T. Yano, K. Groot, R. Z. Cai, S. Radulovic, and A. V. Schally. "Endocrine effects of new bombesin/gastrin-releasing peptide antagonists in rats." American Journal of Physiology-Endocrinology and Metabolism 263, no. 4 (1992): E712—E717. http://dx.doi.org/10.1152/ajpendo.1992.263.4.e712.

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Four new and specific pseudononapeptide bombesin/gastrin-releasing peptide (GRP) receptor antagonists, containing the D-forms of Trp or Trp analogue (Tpi) at position 6, were studied for their effects on the endocrine pancreas and GRP-(14-27)-induced gastrin release in pentobarbital-anesthetized rats. One of the analogues, D-Tpi6,Leu13-psi (CH2NH)Leu14-bombesin-(6-14) (RC-3095), was injected into the lateral brain ventricle just preceding intracerebroventricular administration of GRP-(14-27) to evaluate its antagonistic effect on GRP-induced serum growth hormone (GH) suppression. Analogues RC-

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Fleischmann, A. M., B. Waser, and J. C. Reubi. "Gastrin-releasing peptide receptors in the tumor vascular bed of various human cancers: high incidence in urinary tract cancers." Journal of Clinical Oncology 27, no. 15_suppl (2009): e14575-e14575. http://dx.doi.org/10.1200/jco.2009.27.15_suppl.e14575.

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e14575 Background: Tumoral Gastrin-releasing peptide (GRP) receptors are potential targets for diagnosis and therapy using radiolabeled or cytotoxic GRP analogs. GRP-receptor overexpression has been detected in cancer cells and, more recently, also in the vascular bed of selected tumors. More information on vascular GRP-receptors in cancer is required to asses their potential for vascular targeting applications. Methods: Frequent human cancers from the breast (n=134), lung (n=57), prostate (n=50), kidney (n=32), colon (n=46), urinary tract (n=26) and biliary tract (n=23) were analyzed using in

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Schjoldager, B., S. S. Poulsen, P. Schmidt, D. H. Coy, and J. J. Holst. "Gastrin-releasing peptide is a transmitter mediating porcine gallbladder contraction." American Journal of Physiology-Gastrointestinal and Liver Physiology 260, no. 4 (1991): G577—G585. http://dx.doi.org/10.1152/ajpgi.1991.260.4.g577.

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We studied the role of gastrin-releasing peptide (GRP) for porcine gallbladder motility. Immunohistochemistry visualized nerve fibers containing GRP-like immunoreactivity in muscularis. GRP concentration dependently stimulated contractions of muscularis strips (ED50, 2.9 nM). Neuromedin B was less potent (ED50, 0.1 microM), suggesting existence of GRP-preferring receptors. GRP-induced contractions were unaffected by muscarinic antagonism (1 microM atropine), axonal blockade (1 microM tetrodotoxin), cholecystokinin (CCK) receptor antagonism (10 microM MK-329), or substance P desensitization (1

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Lenz, H. J. "CNS regulation of gastric and autonomic functions in dogs by gastrin-releasing peptide." American Journal of Physiology-Gastrointestinal and Liver Physiology 255, no. 3 (1988): G298—G303. http://dx.doi.org/10.1152/ajpgi.1988.255.3.g298.

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The central nervous system effects of canine gastrin-releasing peptide (GRP) were studied on gastric acid secretion, emptying, blood flow, and the autonomic nervous system in conscious dogs. GRP injected into the third cerebral ventricle significantly (P less than 0.01) increased plasma epinephrine but not norepinephrine concentrations. GRP (0.1-1.0 nmol/kg) significantly decreased gastric acid secretion stimulated by an 8% peptone meal, delayed gastric emptying of the liquid peptone meal, and increased left gastric artery flow. Ganglionic blockade, truncal vagotomy, or adrenalectomy did not a

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Kim, Mi-Kyoung, Hyun-Joo Park, Yeon Kim, Soo-Kyung Bae, Hyung Kim, and Moon-Kyoung Bae. "Involvement of Gastrin-Releasing Peptide Receptor in the Regulation of Adipocyte Differentiation in 3T3-L1 Cells." International Journal of Molecular Sciences 19, no. 12 (2018): 3971. http://dx.doi.org/10.3390/ijms19123971.

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Gastrin-releasing peptide (GRP), a member of bombesin-like peptides, and its receptor (GRP-R) play an important role in various physiological and pathological conditions. In this work, we investigated the role of GRP-R on adipogenesis in 3T3-L1 adipocytes. The expression of GRP-R was significantly increased during the adipocyte differentiation of 3T3-L1 cells. The inhibition of GRP-R by the antagonist RC-3095 affected adipogenesis in 3T3-L1 cells, which reduced lipid accumulation and regulated the expression of adipogenic genes. Moreover, cyclic AMP response element-binding protein (CREB) dire

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Kawai, Koichi, Yukinobu Chiba, Hidehito Mukai, Eisuke Munekata, and Kamejiro Yamashita. "Effects of neuromedin B, gastrin-releasing peptide-10 and their fragment peptides on secretion of gastrointestinal and pancreatic hormones in dogs." Acta Endocrinologica 117, no. 2 (1988): 205–13. http://dx.doi.org/10.1530/acta.0.1170205.

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Abstract. The effects of neuromedin B (NMB), gastrin-releasing peptide (GRP)-10 and their C-terminal fragment peptides on the pancreatic and gastrointestinal hormone release were studied in dogs. Intravenous bolus injections of NMB and GRP-10 (4.5 nmol/kg) into conscious dogs elicited a sharp and statistically significant rise in plasma gastrin and insulin levels, but only GRP-10 brought on a significant rise in the plasma glucagon and enteroglucagon levels. The degree of stimulation of gastrin and insulin secretion by NMB and GRP-10 was dose-dependent. With a dose of 4.5 nmol/kg, the minimum

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Whitley, JC, A. Shulkes, LA Salamonsen, D. Vogiagis, M. Familari, and AS Giraud. "Temporal expression and cellular localization of a gastrin-releasing peptide-related gene in ovine uterus during the oestrous cycle and pregnancy." Journal of Endocrinology 157, no. 1 (1998): 139–48. http://dx.doi.org/10.1677/joe.0.1570139.

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Synthesis of both mRNA and peptide for gastrin-releasing peptide (GRP) has been demonstrated in the pregnant endometrium of sheep and women. However, it is not known whether GRP is synthesized in the sheep uterus during the oestrous cycle. Furthermore the cellular site of GRP mRNA synthesis in the uterus has not been determined. Therefore we examined the synthesis of GRP and determined the cellular location of GRP peptide and mRNA in sheep uterus taken at different times during the oestrous cycle (duration 17 days) and pregnancy (duration 145 days). Northern blot analysis of RNA isolated from

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