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Rozprawy doktorskie na temat „HBV co-infection”

Autor: Grafiati

Data publikacji: 3 czerwca 2025

Data aktualizacji: 31 lipca 2025

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1

Rana, Urvi. "Characteristics and Clinical Outcomes in Antiretroviral Treated HIV-HBV Co-infection." Thesis, Université d'Ottawa / University of Ottawa, 2018. http://hdl.handle.net/10393/38118.

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Objective: The objective of this thesis was to compare demographic and clinical characteristics and factors associated with advanced hepatic fibrosis between HIV and HIV-hepatitis B (HBV) co-infected patients. Methods: Proportional odds models were developed to investigate socio-demographic and clinical variables’ association on liver fibrosis determined by AST-to-Platelet-Ratio-Index (APRI). Results: HBV status and APRI values were available for 2,419 of 9,289 (26%) participants. 199 (9%) were HBV co-infected. Compared to HIV infected, HIV-HBV co-infected individuals were 2.19 (95% CI: 1.63

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Wang, Xiao Qian. "The Impact of Hepatitis B co-infection on the HIV Reservoir." Thesis, University of Sydney, 2020. https://hdl.handle.net/2123/22895.

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Hepatitis B virus (HBV) co-infection increases overall and liver-related mortality in people living with HIV, yet the relationship between these two viruses is still poorly understood. We determined whether levels of HIV were associated with markers of HBV infection, immune activation and inflammation or liver damage. We also designed an assay to sequence full-length HIV RNA in order to further validate our findings. HIV-HBV co-infected and HIV mono-infected participants, naïve to ART, were recruited in Bangkok, Thailand as part of a prospective observational cohort study. HIV proviruses were

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3

Powell, Eleanor A. "Viral Factors Associated with Occult HBV in HIV-positive South Africans." University of Cincinnati / OhioLINK, 2015. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1439561912.

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Martin, Quigley Christina M. "Characterization of Occult Hepatitis B Virus Infection in HIV-Positive Individuals." University of Cincinnati / OhioLINK, 2011. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1307441402.

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Schmelas, Carolin [Verfasser], and Ralf [Akademischer Betreuer] Bartenschlager. "One for all, all for one: Combining and improving AAV, RNAi and CRISPR technologies to combat HBV infection and HBV/HDV co-infections / Carolin Schmelas ; Betreuer: Ralf Bartenschlager." Heidelberg : Universitätsbibliothek Heidelberg, 2020. http://d-nb.info/1212512278/34.

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Oliveira, Marina Pedroso de. "Estudo molecular da infecção pelo vírus da hepatite B em pacientes coinfectados pelo HIV, virgens de tratamento, em Goiânia-Goiás." Universidade Federal de Goiás, 2013. http://repositorio.bc.ufg.br/tede/handle/tede/3470.

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Submitted by Luciana Ferreira (lucgeral@gmail.com) on 2014-10-29T14:55:41Z No. of bitstreams: 2 Dissertação - Marina Pedroso de Oliveira - 2013.pdf: 1546029 bytes, checksum: 047574069d3a0ee543cf485aa4e61edd (MD5) license_rdf: 23148 bytes, checksum: 9da0b6dfac957114c6a7714714b86306 (MD5)<br>Approved for entry into archive by Luciana Ferreira (lucgeral@gmail.com) on 2014-10-30T10:52:04Z (GMT) No. of bitstreams: 2 Dissertação - Marina Pedroso de Oliveira - 2013.pdf: 1546029 bytes, checksum: 047574069d3a0ee543cf485aa4e61edd (MD5) license_rdf: 23148 bytes, checksum: 9da0b6dfac957114c6a7714714b86306

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7

Martel, Nora. "Variabilité génétique du virus de l'hépatite B et implication sur le diagnostic et la pathogénèse." Phd thesis, Université Claude Bernard - Lyon I, 2012. http://tel.archives-ouvertes.fr/tel-00865253.

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L'infection chronique ou occulte par le virus de l'hépatite B (HBV) est l'un des facteurs de risque les plusimportants pour le développement de carcinome hépatocellulaire viro-induit. Malgré la petite taille dugénome et les contraintes imposées par l'organisation de ce génome, le HBV présente une grandevariabilité génétique qui est le résultat des erreurs générées lors de l'étape de reverse transcription maisaussi, lors des processus de sélection et d'adaptation. Durant l'infection, une relation étroite hôte-viruss'établit. La population virale est caractérisée par la présence de quasi-espèces

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8

England, Kirsty Anne. "Paediatric HCV and HIV infection : mode of acquisition, progression and co-infection." Thesis, University College London (University of London), 2008. http://discovery.ucl.ac.uk/1444156/.

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In this thesis a diverse range of topics related to paediatric HIV and HCV infection are investigated information is provided on the more specific areas of coinfection, disease monitoring methodologies and the impact of mode of acquisition of infection. Four unique epidemiological cohorts of vertically and parenterally HIV, HCV and HIV/IICV coinfcctcd children are analysed. ALT reference ranges adjusted for age and sex in children under five years of age show that ALT levels greater than 60U/1 in boys and 55U/1 in girls should be regarded as elevated in the first 18 months of life while therea

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9

Falconer, Karolin. "HIV-1/HCV co-infection immunity and viral dynamics /." Stockholm, 2010. http://diss.kib.ki.se/2010/978-91-7409-762-7/.

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10

Fialho, Renata. "Neuropsychiatric manifestations of hepatitis C treatment in HIV/HCV co-infection." Thesis, University of Sussex, 2017. http://sro.sussex.ac.uk/id/eprint/71260/.

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Hepatitis C (HCV) infection is associated with high rates of mortality and morbidity. Interferon alpha based treatment for HCV offers a good rate of viral clearance, however the associated neuropsychiatric side effects increase the risk of treatment interruption and disease progression. The HIV/HCV coinfection is of particular interest due to association with higher rates of HCV treatment side effects and earlier treatment discontinuation when compared with HCV mono-infection. Therefore, the aim of the thesis was to further explore the effect of coinfection on mood and cognition and how HCV in

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11

Gupta, Priyanka. "Host gene expression and its regulation by microRNA in HCV and HIV/HCV co-infection." Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/11998.

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HCV infection is prevalent and long-term infection may lead to liver cirrhosis and hepatocellular carcinoma (HCC). HIV/HCV co-infection has been shown to accelerate progression of HCV associated liver disease, and HCV in exacerbating hepatotoxicity of antiretroviral therapy. Both viruses are able to manipulate the host gene expression (mRNA) and regulatory (miRNA) machinery. It remains unclear how the regulation of mRNA expression is governed in HCV-infected Peripheral Blood Mononuclear Cells (PBMC) and liver tissue (tumor and non-tumor). This may help explain the cross-talk between PBMC and l

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12

Burke, Stephanie. "Generalized Impairment of CD8+ T-cells in HCV Mono- and HIV-HCV Co-infection." Thesis, Université d'Ottawa / University of Ottawa, 2015. http://hdl.handle.net/10393/32770.

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Chronic hepatitis C virus (HCV) infection has global effects on the immune system. CD8+ T-cells, responsible for viral clearance and control, are dysfunctional for as yet unknown reasons. It is hypothesized that IL-7 signaling pathway deficiencies contribute to this impairment. Blood-derived CD8+ T-cells in chronic HCV mono- and HIV-HCV co-infection had lower IL-7-induced activation of STAT5 and production of Bcl-2, and lower proliferation in co-infection, compared to controls. Lower Bcl-2 production was also associated with increased fibrosis. These changes were independent of the IL-7 recep

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13

Moorman, Jonathan P., Matthew R. Krolikowski, Stephanie M. Mathis, and Robert P. Pack. "HIV/HCV Co-infection: Burden of Disease and Care Strategies in Appalachia." Digital Commons @ East Tennessee State University, 2018. https://dc.etsu.edu/etsu-works/2766.

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Purpose of Review: The purpose of this review is to address infection with HIV and hepatitis C in the Appalachian region of the USA and the driving forces underlying this epidemic. We seek to discuss epidemiology of disease and the possible interventions to reduce incidence and burden of disease in this resource-limited area. Recent Findings: The rise of the opioid crisis has fueled a rise in new hepatitis C infection, and a rise in new HIV infection is expected to follow. Injection drug use has directly contributed to the epidemic and continues to remain a risk factor. Men who have sex with m

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14

Shenje, Justin Tapiwa. "Tuberculous pericarditis in HIV co-infected compared to those without HIV co-infection." Diss., University of Pretoria, 2013. http://hdl.handle.net/2263/33504.

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Introduction: Tuberculosis (TB) pericarditis is a relatively rare form of tuberculosis which has been on the decline. However, the advent of the human immunodeficiency virus (HIV) pandemic has brought about the resurgence of tuberculosis pericarditis and an even poorer prognosis for patients with HIV co-infection. Objectives: The aim is to describe the baseline characteristics of tuberculous pericarditis patients and to assess the impact of HIV on the clinical presentation of this disease. Methods: The study describes baseline data from a randomised clinical trial which explored the use o

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15

Matsukura, Motoi. "Highly active anti-retroviral therapy and liver mitochondrial toxicity in human immunodeficiency virus / hepatitis C virus co-infection." Thesis, University of British Columbia, 2008. http://hdl.handle.net/2429/2517.

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Background: A third of HIV-infected patients are co-infected with HCV in the developed world, and more of co-infected patients than ever before are dying because of liver related diseases today. Drug-related hepatotoxicity is a growing concern among human immunodeficiency virus (HIV) / hepatitis C virus (HCV) co-infected population. Nucleotide analogues containing HIV antiretroviral therapy, namely highly active anti-retroviral therapy (HAART), can induce mitochondrial toxicity. However, little is known about the effect of nucleotide analogues on the liver at the cellular and molecular level,

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16

David, Jummy Funke. "Mathematical epidemiology of HIV/AIDS and tuberculosis co-infection." Thesis, University of British Columbia, 2015. http://hdl.handle.net/2429/54295.

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The project deals with the analysis of a general dynamical model for the spread of HIV/AIDS and tuberculosis Co-infection. We capture in the model the dynamics of HIV/AIDS infected individuals and investigate their impacts in the progression of tuberculosis with and without TB treatment. It is shown that TB-only model and HIV-only model have locally asymptotically stable disease-free equilibrium when the basic reproduction number is less than unity and a unique endemic equilibrium exists when the basic reproduction number is greater than unity. We analyze the full HIV/AIDS-TB coinfection mod

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17

Matthews, Gail Veronica National Centre in HIV Epidemiology &amp Clinical Research Faculty of Medicine UNSW. "Therapeutic strategies in HIV and Hepatitis B co-infection." Awarded by:University of New South Wales. National Centre in HIV Epidemiology & Clinical Research, 2009. http://handle.unsw.edu.au/1959.4/44538.

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Individuals with HIV-HBV coinfection are at risk of accelerated hepatic fibrosis, and end stage liver disease is now a major cause of morbidity and mortality in this population. For many years lamivudine (3TC) monotherapy was the only effective treatment for HBV in coinfected individuals, but its use was complicated by the frequent development of HBV drug resistance, and loss of virological suppression. With the availability of new potent antivirals for HBV, including tenofovir (TDF), therapeutic options for HIV-HBV coinfected individuals have increased, although the management of HBV disease

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18

Hughes, R. "Herpesvirus and HIV-1 co-infection of human macrophages." Thesis, University College London (University of London), 2017. http://discovery.ucl.ac.uk/1563691/.

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HSV-1 and HIV-1 co-infection of human macrophages represents a clinically relevant model with which to investigate the host-pathogen interactions between macrophages and viruses. In this thesis, I demonstrate that HSV-1 productively infects human monocyte derived macrophages, with associated cell death and type I IFN responses, and additionally, that a proportion of macrophages support latent HSV-1 infection. I de ne latency as the absence of lytic gene transcription, virion production and cell death, in the presence of persistent expression of the HSV-1 latency associated transcript (LAT). I

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19

Victoria, Marilu Barbieri. "Implicações clínicas e imunobiológicas da co-infecção HIV e vírus da hepatite C em uma população atendida na fundação de medicina tropical do Amazonas." Universidade Federal do Amazonas, 2009. http://tede.ufam.edu.br/handle/tede/3113.

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Made available in DSpace on 2015-04-20T12:31:42Z (GMT). No. of bitstreams: 1 PRE-TEXTUAL.pdf: 394027 bytes, checksum: d5e64d94f3ade196d4079ab1173940e8 (MD5) Previous issue date: 2009-06-30<br>Fundação de Amparo à Pesquisa do Estado do Amazonas<br>The global epidemic of AIDS began in 1981, the United States, and after the introduction of antiretroviral drugs to patients infected with HIV / AIDS have been given a higher survival emerged a new challenge, co-infection with the hepatitis C virus (HCV), which is already the leading cause of death in these individuals. The HCV was discovered in 1

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20

Auma, Ann Winniefred Nangobi. "THE IMPACT OF DIRECT-ACTING ANTI-VIRAL THERAPY ON NAIVE CD4+ T CELL LYMPHOPENIA AND CELLULAR IMMUNE ACTIVATION IN HCV INFECTION AND HCV/HIV CO-INFECTION." Case Western Reserve University School of Graduate Studies / OhioLINK, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1625764728651756.

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21

Wang, Bin. "Biological and molecular mechanisms in HIV disease progression and co-infection." Thesis, The University of Sydney, 2000. https://hdl.handle.net/2123/27745.

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The present thesis has undertaken two major areas of HIV pathogenesis for investigation. The first part of the thesis consists of viral and host factors that may account for slow progression or non-progression of HIV disease. A number of viral factors such as nef gene, genetic variability over time, V2 region extension and the defect in the vpr gene/ other HIV-1 genes have been critically analyzed with the sequencing of viral strains from both progressing and non-progressing HIV:1 infected members of well established cohorts. A detailed full-length (9.7kb) sequencing of HIV strains has

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22

Herreru-MartiÌnez, Esteban. "Hepatitis C and HIV co-infection in patients with haemophilia." Thesis, University College London (University of London), 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.404936.

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Judge, Chelsey J. "IL-7-MEDIATED CD56BRIGHT NK CELL FUNCTION IS IMPAIRED IN HCV IN PRESENCE AND ABSENCE OF CONTROLLED HIV INFECTION, WHILE CD14BRIGHTCD16- MONOCYTES NEGATIVELY CORRELATE WITH CD4 MEMORY T CELLS AND HCV DECLINE DURING HCV-HIV CO-INFECTION." Case Western Reserve University School of Graduate Studies / OhioLINK, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=case1481187921533387.

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Broadbent, Andrew James. "Chlamydia trachomatis : quantification, immunological investigation and co-infection with HIV-1." Thesis, Imperial College London, 2010. http://hdl.handle.net/10044/1/5929.

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Three aspects of Chlamydia trachomatis translational research were explored in this thesis. Firstly, as over 75% of patients with LGV are also HIV-1 sero-positive, a cell-culture model was established to investigate whether HIV-1 altered the replication of C. trachomatis serovar L2 in vitro. Inclusion size was significantly increased in the presence of HIV-1, but there was no significant alteration in chlamydial growth kinetics, infectivity, morphology, or transcription of 16sRNA, ompA or euo, suggesting that viral co-infection did not induce chlamydial persistence. It is, therefore, unlikely

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25

Du, Toit Eben Francois. "Modelling the co-infection dynamics of HIV-1 and M. tuberculosis." Diss., Pretoria : [s.n.], 2008. http://upetd.up.ac.za/thesis/available/etd-08172008-213855.

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Danta, Mark. "Acute hepatitis C infection in HIV co-infection : epidemiology, natural history and the host-viral responses." Thesis, University College London (University of London), 2006. http://discovery.ucl.ac.uk/1445482/.

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Aim: To analyse the epidemiological, clinical and immunological aspects of a recent epidemic of acute HCV in HIV co-infected individuals.;Methods: The epidemiology was characterised using combined molecular and clinical studies. A phylogenetic analysis of the E1/E2 region of the HCV genome was performed to determine HCV transmission. Transmission risk factors were then explored in a multicentre case-control study. Data was collected on the early clinical course of HCV in HIV. Longitudinal cell-mediated responses to HCV and HCV evolution were studied with PBMCs and serum collected during the ac

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27

Ncube, Wenzokuhle S. "People’s understanding of TB in a setting of high HIV/TB prevalence: case studies in Gugulethu Township, Western Cape Province." University of the Western Cape, 2014. http://hdl.handle.net/11394/4170.

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Magister Artium (Development Studies) - MA(DVS)<br>Tuberculosis (TB) infection is present in many people but it is sometimes latent until one’s immune system is compromised. As such, it increasingly manifests in people, especially those whose immune system has been compromised by e.g. HIV, as an opportunistic disease. TB is thus closely interlinked with HIV and efforts to eradicate TB have been integrated with the fight against HIV in South Africa. The study revealed that factors such as poverty and stigma - be it enacted or perceived - has an impact on how people with TB deal with the burden

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Trinh, Quynh Mai. "Epidemiology and genomics of Mycobacterium tuberculosis in TB/HIV co-infection in Vietnam." Thesis, The University of Sydney, 2018. http://hdl.handle.net/2123/20231.

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TB and human HIV infection have mutually detrimental effects that complicate patient management and global TB control efforts. The overall aim was to improve our understanding of the TB epidemiology, drug resistance profile, and transmission dynamics among people living with HIV in Vietnam. The global epidemiology of TB, HIV and TB/HIV co-infection in low and high incidence countries with a specific focus on the Asia-Pacific region and relevant disease trends within Vietnam was described. Findings from a retrospective study with 200 M. tuberculosis isolates from TB/HIV co-infected patients in

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29

Lucca, Maria Elvira Santos de. "Análise epidemiológica da tuberculose e co-infecção HIV/TB, em Ribeirão Preto-SP, de 1998-2006." Universidade de São Paulo, 2008. http://www.teses.usp.br/teses/disponiveis/17/17139/tde-01042008-151419/.

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A pesquisa teve como objetivo analisar o Programa de Controle da Tuberculose (PCT) no município de Ribeirão Preto -São Paulo, no período de 1998 a 2006. Utilizou-se para este propósito indicadores epidemiológicos e de desempenho construídos a partir de dados das fichas de notificação de tuberculose (TB) armazenadas no sistema de informação o EPI-Tb, da Secretaria municipal de saúde deste município e os dados populacionais de estimativas do DATASUS, do período de estudo. Selecionaram-se para o estudo casos novos de TB notificados e residentes no município, por ano de diagnóstico, excluindo-se c

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Mumba, Chibale K. "Mathematical modelling and analysis of HIV/AIDS and trichomonas vaginalis co-infection." Diss., University of Pretoria, 2017. http://hdl.handle.net/2263/65185.

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Deterministic models for the transmission dynamics of HIV/AIDS and trichomonas vaginalis (TV) in a human population are formulated and analysed. The models which assumed standard incidence formulations are shown to have globally asymptotically stable (GAS) disease-free equilibria whenever their associated reproduction number is less than unity. Furthermore, both models possess a unique endemic equilibrium that is GAS whenever the associated reproduction number is greater than unity. An extended model for the co-infection of TV and HIV in a human population is also designed and rigorously

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Makatsa, Mohau Steven. "Characterization of Mycobacterium tuberculosis-specific Th22 cells in HIV-TB co-infection." Doctoral thesis, Faculty of Health Sciences, 2020. http://hdl.handle.net/11427/32398.

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Tuberculosis (TB) remains the infectious disease causing the greatest global mortality, with an estimated 10 million incident cases of TB and 1.45 million deaths in 2018. Although there is a cure for TB, the success of the treatment is hampered by multidrug resistant TB and HIV infection. There is an urgent need for an effective TB vaccine to prevent ongoing transmission. The development of a new and efficacious TB vaccine will likely be dependent on our understanding of protective immunity to TB. Although it is well established that Th1 cells are crucial in the response against Mycobacterium

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Slade, Jessica A. "In Vitro and In Vivo Characterization of Chlamydia and HSV Co-infection." Digital Commons @ East Tennessee State University, 2016. https://dc.etsu.edu/etd/3026.

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The obligate intracellular bacterium, Chlamydia trachomatis, and Herpes Simplex Virus Type-2 (HSV-2) are the leading sexually transmitted pathogens in the world. These infections are usually asymptomatic and clinically mild, but complications can be severe. Reports of dual detection of Chlamydia and HSV within the genital tracts of humans led our laboratory to develop an in vitro Chlamydia/HSV co-infection model. Little is known regarding the specific pathogenesis of Chlamydia and HSV co-infections, but HSV-super-infection of Chlamydia-infected cells caused the chlamydiae to deviate from their

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Batista, Mariana Dias [UNIFESP]. "Prevalência e correlações clínicas da infecção pelo herpesvírus humano tipo 8 em indivíduos recém-infectados pelo HIV." Universidade Federal de São Paulo (UNIFESP), 2009. http://repositorio.unifesp.br/handle/11600/9968.

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Made available in DSpace on 2015-07-22T20:50:38Z (GMT). No. of bitstreams: 0 Previous issue date: 2009-03-25<br>A infecção pelo herpesvírus humano tipo 8 (HHV-8) e suas correlações clínicas não foram até o presente bem caracterizadas em indivíduos recém-infectados pelo HIV. Esse estudo avalia a prevalência e correlações clínicas da infecção pelo HHV-8 em uma coorte de 228 indivíduos recém-infectados pelo HIV (vírus da imunodeficiência humana), além de estudar a incidência desta infecção após um ano de acompanhamento. As sorologias para HHV-8 foram realizadas através de imunofluorescência in

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34

Griesbeck, Morgane. "Dissecting mechanisms underlying increased TLR7-mediated IFNα production in pDCs in physiological and pathophysiological settings : between sex differences and HIV-1-HCV co-infection". Thesis, Paris 6, 2015. http://www.theses.fr/2015PA066444/document.

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Les interférons de type I (IFN-I) peuvent être produits par toutes les cellules mais les rares cellules dendritiques plasmacytoïdes en sont les principales cellules productrices. L’IFNa orchestre de nombreux mécanismes pathogéniques dans l’infection par le virus de l’immunodéficience humaine de type 1 (VIH-1). L’étude de modèles physiologiques et pathophysiologiques peut fournir des informations cruciales sur les moyens d’exploiter la signalisation de l’IFNa dans un but thérapeutique. Les pDCs de femmes produisent plus d’IFNa en réponse à la stimulation du récepteur Toll-like 7 (TLR7) que les

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35

Salisch, Nadine Christina. "Roles of co-stimulatory and co-inhibitory receptor expression in T cell dysfunction in HIV-1 and SIV infection." kostenfrei, 2010. http://d-nb.info/1002589851/34.

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Long, Elizabeth Michelle. "Genetic and co-receptor characterization of viral diversity early in human immunodeficiency virus type 1 infection /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/4997.

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Shiu, Yuen-chi Eunice, and 邵琬詞. "A systematic review of the population prevalence of HIV and STD co-infection." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/193832.

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Background: Sexually transmitted diseases (STD) remain a public health concern in worldwide. Human immunodeficiency virus (HIV), one of the STDs, is associated with the increase risk of other STD infections. According to Centers for Disease Control and Prevention (CDC), individuals who are infected with STDs are more likely to be infected with HIV than uninfected individuals. As HIV and other STDs share the same transmission route, the co-infection may be observed more frequently in the population. To control the number of co-infection, screening programs are essential in all areas. While scre

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38

Rubi, Gérald. "Neurosyphilis révélatrice d'une co-infection à VIH : à propos d'un cas." Montpellier 1, 1997. http://www.theses.fr/1997MON11151.

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Mwandumba, Henry Charles. "Modulation of human alveolar microphage function during mycobacterium tuberculosis and HIV co-infection." Thesis, University of Liverpool, 2007. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.443938.

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Thawer, Narjis Khatoon G. "Investigating defects in monocyte responses to Mycobacterium tuberculosis in HIV-TB co-infection." Master's thesis, University of Cape Town, 2014. http://hdl.handle.net/11427/5923.

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Paula, Denise Píccoli de. "Prevalência da co-infecção em pacientes HIV soropositivos com o HTLV 1 e 2 em dois centros de referência de Goiânia-GO." Pontifícia Universidade Católica de Goiás, 2014. http://localhost:8080/tede/handle/tede/3010.

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Made available in DSpace on 2016-08-10T10:54:46Z (GMT). No. of bitstreams: 1 DENISE PICCOLI DE PAULA.pdf: 7499605 bytes, checksum: acd6b8560cb237be607f00ebc352a85c (MD5) Previous issue date: 2014-08-14<br>Co-infection with Human T Lymphotropic (HTLV) in individuals infected by the human immunodeficiency virus (HIV) occurs because these viruses share similar routes of transmission is through sexual, parenteral or vertical route, besides infecting the same cell type, and can accelerate as much retard the development of diseases related to them. Rates of HIV (HTLV) coinfection vary according t

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Mbunyuza, Lungelwa. "Treatment adherence in TB/HIV co-infected patients in Mount Frere, Eastern Cape." University of Western Cape, 2020. http://hdl.handle.net/11394/8051.

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Master of Public Health - MPH<br>Adequate levels of adherence to treatment for tuberculosis (TB) and HIV at the same time poses a problem for public health in South Africa. TB/HIV co-infected patients face many potential barriers to adherence to treatment for both conditions. There is a need for more knowledge about factors influencing treatment adherence in co-infected patients on concomitant treatment. The aim of this study was to explore the barriers and facilitators to treatment adherence among people co-infected with TB/HIV living in the Alfred Nzo District, Eastern Cape, in order to iden

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Cuadros, Diego Fernando. "THE ROLE OF CO-INFECTION IN THE SPREAD OF HIV IN SUB-SAHARAN AFRICA." UKnowledge, 2011. http://uknowledge.uky.edu/biology_etds/1.

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The cause of the high HIV prevalence in sub-Saharan Africa is incompletely understood, with heterosexual penile-vaginal transmission proposed as the main mechanism. Heterosexual HIV transmission has a very low probability; further, a single estimation of heterosexual probability of HIV transmission fails to reproduce the variation associated with important biological cofactors. In particular, studies of HIV incidence suggest that co-infection with other infectious diseases influence the HIV transmission, and therefore might substantially vary the pattern of the spread of the infection. To asse

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Tang, Hing-cheung, and 鄧慶璋. "Syphilis co-infection with HIV/AIDS : study of a local cohort in Hong Kong." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2014. http://hdl.handle.net/10722/206941.

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Background In the recent decade resurgence of syphilis was noted in a global scale, especially in high-risk communities like men who have sex with men (MSM) and commercial sex workers (CSW). Syphilis co-infection is important in people living with human immunodeficiency virus and acquired immunodeficiency syndrome (HIV/AIDS) because of their similar transmission routes, mutual interactions at pathophysiology level, relation to socioeconomic factors and public health implications. Methods Retrospective analysis of syphilis co-infections among the HIV/AIDS patients who have been under the c

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Dupont, Maeva. "Identification of novel factors involved in the exacerbation of HIV-1 infection and spread among macrophages in the tuberculosis context." Thesis, Toulouse 3, 2019. http://www.theses.fr/2019TOU30211.

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Mycobacterium tuberculosis (Mtb), la bactérie responsable de la tuberculose (TB), et le virus de l'immunodéficience humaine (VIH-1), l'agent du syndrome de l'immunodéficience acquise (SIDA), accélèrent leurs progressions mutuelles chez les patients co-infectés. Alors que de nombreuses données cliniques rapportent une augmentation de la charge virale dans les sites anatomiques co-infectés, les mécanismes qui en sont responsables restent insuffisamment décrits. Mtb cible principalement les macrophages. Nous émettons l'hypothèse que l'infection des macrophages par Mtb créé un microenvironnement p

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Bimerew, Million S. "Analysing implementation of the integrated tuberculosis, HIV and AIDS policy in a rural sub-district, Western Cape." University of the Western Cape, 2015. http://hdl.handle.net/11394/5244.

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Magister Curationis - MCur<br>Globally, tuberculosis (TB) and AIDS are the leading causes of morbidity and mortality of people, particularly in many developing countries. South Africa is among those countries with a high double burden of TB and HIV infections in the world. Although policy guidelines have been developed to mitigate the problems of TB and HIV coinfection, there are still challenges with their implementation. The aim of this study was to analyse the implementation of the integrated TB and HIV policy in a rural sub-district of the Western Cape. The study applied a descriptive surv

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Cox, Jennifer H. "The function of the CD4 co-receptor for T helper cell activation and HIV infection." Thesis, University of Oxford, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.413989.

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Carvalho, Flávia Lopes. "CO-INFECÇÃO POR Leishmania sp. EM INDIVÍDUOS VIVENDO COM HIV/Aids." Universidade Federal do Maranhão, 2009. http://tedebc.ufma.br:8080/jspui/handle/tede/1129.

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Made available in DSpace on 2016-08-19T18:15:59Z (GMT). No. of bitstreams: 1 FLAVIA LOPES CARVALHO.pdf: 442162 bytes, checksum: b9d2b399e741a894bb9550e5094975a0 (MD5) Previous issue date: 2009-11-18<br>Co-infection Leishmania-HIV/Aids is a serious public health problem in almost of the world. The visceral leishmaniasis is the clinical form of leishmaniosis hat is most associated with HIV/Aids cases being the co-infection understated, since the leishmaniasis is not AIDS-defining illness. This was a descriptive cross sectional study from March 2006 to December 2008, aiming to investigate the

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Nguyen, Truong Tam. "Co-infection VIH/VHC : développement et mise en oeuvre d’outils paracliniques pour la prise en charge dans le pays à ressource limitée et la personnalisation thérapeutique." Thesis, Montpellier, 2015. http://www.theses.fr/2015MONT3508/document.

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Les hépatites virales constituent un enjeu majeur de santé public dans le domaine des maladies infectieuses. L’enjeu pour les prochaines années sera de mettre en œuvre des stratégies de dépistage et de de prise en charge des hépatites virales et du VIH, en particulier chez les usagers de drogue par voie intraveineuse et dans les pays à ressources limitées. Dans ce travail de thèse nous avons évalué des outils et des stratégies nouvelles pour améliorer le diagnostic et la prise en charge des infections par le VIH et le VHC dans les pays du Sud. Notre première étude porte sur l’évaluation de la

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Mackey, Maureen Taylor. "Inhibition of HIV infection by co-ordinated blockade of multiple steps in the virus life-cycle." Thesis, King's College London (University of London), 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.415839.

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